+publisher:"University of North Texas" +contributor:("Gopal, Kamakshi V.")

University of North Texas

1. Gibson, Jason. Neurotoxicity of the Industrial Solvent 4-Methylcyclohexanemethanol: Involvement of the GABA Receptor.

Degree: 2015, University of North Texas

URL: https://digital.library.unt.edu/ark:/67531/metadc799542/

► A recent chemical spill of 4-Methylcyclohexanemethanol (4-MCHM) in West Virginia left 300,000 people without water. Officials claimed that this compound is not lethally toxic, but… (more)

Subjects/Keywords: GABA; 4-MCHM; cytotoxicity; neurotoxicity; receptor; Solvents – Toxicology.; Neurotoxicology.; GABA – Receptors.

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APA (6^th Edition):

Gibson, J. (2015). Neurotoxicity of the Industrial Solvent 4-Methylcyclohexanemethanol: Involvement of the GABA Receptor. (Thesis). University of North Texas. Retrieved from https://digital.library.unt.edu/ark:/67531/metadc799542/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16^th Edition):

Gibson, Jason. “Neurotoxicity of the Industrial Solvent 4-Methylcyclohexanemethanol: Involvement of the GABA Receptor.” 2015. Thesis, University of North Texas. Accessed January 28, 2021. https://digital.library.unt.edu/ark:/67531/metadc799542/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7^th Edition):

Gibson, Jason. “Neurotoxicity of the Industrial Solvent 4-Methylcyclohexanemethanol: Involvement of the GABA Receptor.” 2015. Web. 28 Jan 2021.

Vancouver:

Gibson J. Neurotoxicity of the Industrial Solvent 4-Methylcyclohexanemethanol: Involvement of the GABA Receptor. [Internet] [Thesis]. University of North Texas; 2015. [cited 2021 Jan 28]. Available from: https://digital.library.unt.edu/ark:/67531/metadc799542/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Gibson J. Neurotoxicity of the Industrial Solvent 4-Methylcyclohexanemethanol: Involvement of the GABA Receptor. [Thesis]. University of North Texas; 2015. Available from: https://digital.library.unt.edu/ark:/67531/metadc799542/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

University of North Texas

2. Rogers, Edmond A. Simultaneous Electrophysiological and Morphological Assessment of Impact Damage to Nerve Cell Networks.

Degree: 2018, University of North Texas

URL: https://digital.library.unt.edu/ark:/67531/metadc1157638/

► A ballistic pendulum impulse generator was used to impact networks in primary culture growing on microelectrode arrays. This approach has the advantage of imparting pure… (more)

Subjects/Keywords: impact trauma; network dynamics; electrophysiology; subcellular damage; nuclear rotation; functional damage; Biology, Neuroscience; Biophysics, Medical; Engineering, Biomedical

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APA (6^th Edition):

Rogers, E. A. (2018). Simultaneous Electrophysiological and Morphological Assessment of Impact Damage to Nerve Cell Networks. (Thesis). University of North Texas. Retrieved from https://digital.library.unt.edu/ark:/67531/metadc1157638/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16^th Edition):

Rogers, Edmond A. “Simultaneous Electrophysiological and Morphological Assessment of Impact Damage to Nerve Cell Networks.” 2018. Thesis, University of North Texas. Accessed January 28, 2021. https://digital.library.unt.edu/ark:/67531/metadc1157638/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7^th Edition):

Rogers, Edmond A. “Simultaneous Electrophysiological and Morphological Assessment of Impact Damage to Nerve Cell Networks.” 2018. Web. 28 Jan 2021.

Vancouver:

Rogers EA. Simultaneous Electrophysiological and Morphological Assessment of Impact Damage to Nerve Cell Networks. [Internet] [Thesis]. University of North Texas; 2018. [cited 2021 Jan 28]. Available from: https://digital.library.unt.edu/ark:/67531/metadc1157638/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Rogers EA. Simultaneous Electrophysiological and Morphological Assessment of Impact Damage to Nerve Cell Networks. [Thesis]. University of North Texas; 2018. Available from: https://digital.library.unt.edu/ark:/67531/metadc1157638/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

University of North Texas

3. Suri, Nikita. "Superbursts": Investigation of Abnormal Paroxysmal Bursting Activity in Nerve Cell Networks In Vitro.

Degree: 2018, University of North Texas

URL: https://digital.library.unt.edu/ark:/67531/metadc1157655/

► Superbursts (SBs) are large, seemingly spontaneous activity fluctuations often encountered in high density neural networks in vitro. Little effort has been put forth to define… (more)

▼ Superbursts (SBs) are large, seemingly spontaneous activity fluctuations often encountered in high density neural networks in vitro. Little effort has been put forth to define and analyze SBs which are paroxysmal bursting discharges. Through qualitative and quantitative means, I have described specific occurrences of superbursting activity. A complex of paroxysmal bursting has been termed a "superburst episode," and each individual SB is a "superburst event" which is comprises a fine burst structure. Quantitative calculations (employing overall spike summations and coefficient of variation (CV) calculations), reveal three distinct phases. Phase 1 is a "build up" phase of increasingly strong, coordinated bursting with an average of a 17.6% ± 13.7 increase in activity from reference. Phase 2, the "paroxysmal" phase, is comprised of massive coordinated bursting with high frequency spike content. Individual spike activity increases by 52.9% ± 14.6. Phase 3 is a "recovery phase" of lower coordination and an average of a 50.1% ± 35.6 decrease in spike production from reference. SBs can be induced and terminated by physical manipulation of the medium. Using a peristaltic pump with a flow rate of 0.4ml/min, superbursting activity ceases approximately 28.3 min after the introduction of flow. Alternatively, upon cessation of medium flow superbursting activity reemerges after approximately 8.5 min. Additionally, this study explored other methods capable of inducing superbursting activity using osmotic shocks. The induction and termination of SBs demonstrates that the cell culture environment plays a major role in generating this phenomenon. The observations that high density multi-layer neuronal networks in culture are more likely to enter paroxysmal bursting also supports the hypothesis that enrichment and depletion layers of metabolites and ionic species are involved in such unusual activity. The dynamic similarity of the SB phenomenon with epileptiform discharges make further quantification on the spike pattern level pertinent and important. Advisors/Committee Members: Gross, Guenter W., Fuchs, Jannon L., Gopal, Kamakshi V..

Subjects/Keywords: superbursts; electrophysiology; burst fine structure; network dynamics; Biology, Neuroscience; Biophysics, Medical; Engineering, Biomedical

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APA (6^th Edition):

Suri, N. (2018). "Superbursts": Investigation of Abnormal Paroxysmal Bursting Activity in Nerve Cell Networks In Vitro. (Thesis). University of North Texas. Retrieved from https://digital.library.unt.edu/ark:/67531/metadc1157655/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16^th Edition):

Suri, Nikita. “"Superbursts": Investigation of Abnormal Paroxysmal Bursting Activity in Nerve Cell Networks In Vitro.” 2018. Thesis, University of North Texas. Accessed January 28, 2021. https://digital.library.unt.edu/ark:/67531/metadc1157655/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7^th Edition):

Suri, Nikita. “"Superbursts": Investigation of Abnormal Paroxysmal Bursting Activity in Nerve Cell Networks In Vitro.” 2018. Web. 28 Jan 2021.

Vancouver:

Suri N. "Superbursts": Investigation of Abnormal Paroxysmal Bursting Activity in Nerve Cell Networks In Vitro. [Internet] [Thesis]. University of North Texas; 2018. [cited 2021 Jan 28]. Available from: https://digital.library.unt.edu/ark:/67531/metadc1157655/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Suri N. "Superbursts": Investigation of Abnormal Paroxysmal Bursting Activity in Nerve Cell Networks In Vitro. [Thesis]. University of North Texas; 2018. Available from: https://digital.library.unt.edu/ark:/67531/metadc1157655/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

University of North Texas

4. Holmes, Katelyn. Cytotoxicity and Functional Toxicity of Mefloquine and the Search for Protective Compounds.

Degree: 2015, University of North Texas

URL: https://digital.library.unt.edu/ark:/67531/metadc801913/

► Mefloquine hydrochloride is an antimalarial agent that has been used for the past 40 years. Numerous reports of neurological side effects have recently led the… (more)

▼ Mefloquine hydrochloride is an antimalarial agent that has been used for the past 40 years. Numerous reports of neurological side effects have recently led the FDA to issue a strong warning regarding long-term neurological effects. This warning lead to the U.S. Army’s Special Forces and other components to discontinue its use in July of 2013. Despite reported adverse side effects, mefloquine remains in circulation and is recommended to travelers going to specific Asian countries. Mefloquine has been used as a treatment for those already infected with the malaria parasite (blood concentrations ranging from 2.1 to 23 µM), and as prophylaxis (blood concentrations averaging 3.8 µM) (Dow 2003). The purpose of this study was to quantify Mefloquine’s toxicity using spontaneously active nerve cell networks growing on microelectrode arrays in vitro and to identify compounds that alleviate or reduce toxic effects. The current literature on mefloquine toxicity is lacking electrophysiological data. These data will contribute to research on the mechanism of adverse side effects associated with mefloquine use. Sequential titration experiments were performed by adding increasing concentrations of mefloquine solution to cultured neurons. Network responses were quantified and reversibility was examined. In each network, activity decreases were normalized as a percent of reference activity yielding a mean IC50 value of 5.97 ± 0.44 (SD) µM (n=6). After total activity loss, no activity was recovered with two successive medium changes. To test for network response desensitization resulting from sequential applications over 5-6 hr periods, one-point titrations at varying concentrations were conducted with fresh networks. These experiments yielded a single concentration response curve with an IC50 value of 2.97 µM. This represents a statistically significant shift (p < 0.0001) to lower concentrations of mefloquine, demonstrating that sequential applications result in network desensitization. After mefloquine exposures, cells were evaluated for irreversible cytotoxic damage. Over a 12-hour period under 6 µM mefloquine, process beading and granulation of somal cytoplasm were observed. At 8 µM mefloquine cell stress was apparent after only 10 minutes with major glial damage and process beading at 120 minutes. In this study, quinolinic acid served as a neuroprotectant at 20 µM. There have been multiple studies on the endogenous concentrations of quinolinic acid and current literature is quite variable. Immunocompromised individuals have some of the highest blood levels of quinolinic acid (up to 20 µM). With 30 min pre-applications of quinolinic acid, the mefloquine IC50 value shifted from 5.97 ± 0.44 µM (n=6), to 9.28 ± 0.55 µM (n=3). This represents a statistically significant change to higher mefloquine concentrations and demonstrates neuroprotection. Advisors/Committee Members: Gross, Guenter W., Fuchs, Jannon Lou, 1946-, Gopal, Kamakshi V..

Subjects/Keywords: Mefloquine; toxicity; neurotoxicity; malaria; Mefloquine – Toxicology.; Antimalarials – Toxicology.; Neurotoxicology.

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APA (6^th Edition):

Holmes, K. (2015). Cytotoxicity and Functional Toxicity of Mefloquine and the Search for Protective Compounds. (Thesis). University of North Texas. Retrieved from https://digital.library.unt.edu/ark:/67531/metadc801913/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16^th Edition):

Holmes, Katelyn. “Cytotoxicity and Functional Toxicity of Mefloquine and the Search for Protective Compounds.” 2015. Thesis, University of North Texas. Accessed January 28, 2021. https://digital.library.unt.edu/ark:/67531/metadc801913/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7^th Edition):

Holmes, Katelyn. “Cytotoxicity and Functional Toxicity of Mefloquine and the Search for Protective Compounds.” 2015. Web. 28 Jan 2021.

Vancouver:

Holmes K. Cytotoxicity and Functional Toxicity of Mefloquine and the Search for Protective Compounds. [Internet] [Thesis]. University of North Texas; 2015. [cited 2021 Jan 28]. Available from: https://digital.library.unt.edu/ark:/67531/metadc801913/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Holmes K. Cytotoxicity and Functional Toxicity of Mefloquine and the Search for Protective Compounds. [Thesis]. University of North Texas; 2015. Available from: https://digital.library.unt.edu/ark:/67531/metadc801913/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

University of North Texas

5. Carney, Lara E. A preliminary study on the effect of selective serotonin reuptake inhibitors on peripheral and lower brainstem auditory processing.

Degree: 2001, University of North Texas

URL: https://digital.library.unt.edu/ark:/67531/metadc2765/

► This study compared auditory behavioral and physiologic measures in normal control subjects and subjects prescribed with a selective serotonin reuptake inhibitor (SSRI) who were yet… (more)

Subjects/Keywords: Auditory perception.; Serotonin uptake inhibitors.; Serotonin; Reuptake; Inhibitors; Auditory processing

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APA (6^th Edition):

Carney, L. E. (2001). A preliminary study on the effect of selective serotonin reuptake inhibitors on peripheral and lower brainstem auditory processing. (Thesis). University of North Texas. Retrieved from https://digital.library.unt.edu/ark:/67531/metadc2765/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16^th Edition):

Carney, Lara E. “A preliminary study on the effect of selective serotonin reuptake inhibitors on peripheral and lower brainstem auditory processing.” 2001. Thesis, University of North Texas. Accessed January 28, 2021. https://digital.library.unt.edu/ark:/67531/metadc2765/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7^th Edition):

Carney, Lara E. “A preliminary study on the effect of selective serotonin reuptake inhibitors on peripheral and lower brainstem auditory processing.” 2001. Web. 28 Jan 2021.

Vancouver:

Carney LE. A preliminary study on the effect of selective serotonin reuptake inhibitors on peripheral and lower brainstem auditory processing. [Internet] [Thesis]. University of North Texas; 2001. [cited 2021 Jan 28]. Available from: https://digital.library.unt.edu/ark:/67531/metadc2765/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Carney LE. A preliminary study on the effect of selective serotonin reuptake inhibitors on peripheral and lower brainstem auditory processing. [Thesis]. University of North Texas; 2001. Available from: https://digital.library.unt.edu/ark:/67531/metadc2765/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

University of North Texas

6. Briley, Kelly Anne. The Effects of Selective Serotonin Reuptake Inhibitors (SSRI) on Auditory Measures in Women.

Degree: 2002, University of North Texas

URL: https://digital.library.unt.edu/ark:/67531/metadc3155/

► This study examined the relationship between selective serotonin reuptake inhibitor (SSRI) medication and auditory measures in clinically depressed women. Experimental subjects were tested in both… (more)

Subjects/Keywords: Auditory perception.; Serotonin uptake inhibitors.; Depression in women.; Women; auditory processing; SSRI; depression; selective serotonin re-uptake inhibitor

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APA (6^th Edition):

Briley, K. A. (2002). The Effects of Selective Serotonin Reuptake Inhibitors (SSRI) on Auditory Measures in Women. (Thesis). University of North Texas. Retrieved from https://digital.library.unt.edu/ark:/67531/metadc3155/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16^th Edition):

Briley, Kelly Anne. “The Effects of Selective Serotonin Reuptake Inhibitors (SSRI) on Auditory Measures in Women.” 2002. Thesis, University of North Texas. Accessed January 28, 2021. https://digital.library.unt.edu/ark:/67531/metadc3155/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7^th Edition):

Briley, Kelly Anne. “The Effects of Selective Serotonin Reuptake Inhibitors (SSRI) on Auditory Measures in Women.” 2002. Web. 28 Jan 2021.

Vancouver:

Briley KA. The Effects of Selective Serotonin Reuptake Inhibitors (SSRI) on Auditory Measures in Women. [Internet] [Thesis]. University of North Texas; 2002. [cited 2021 Jan 28]. Available from: https://digital.library.unt.edu/ark:/67531/metadc3155/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Briley KA. The Effects of Selective Serotonin Reuptake Inhibitors (SSRI) on Auditory Measures in Women. [Thesis]. University of North Texas; 2002. Available from: https://digital.library.unt.edu/ark:/67531/metadc3155/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

University of North Texas

7. Goodale, Elizabeth S. The Effects of Selective Serotonin Reuptake Inhibitors (Ssri) on Auditory Measures in Clinically Depressed Subjects.

Degree: 2002, University of North Texas

URL: https://digital.library.unt.edu/ark:/67531/metadc3146/

► The purpose of this study was to investigate the relationship between selective serotonin reuptake inhibitor (SSRI) medication on auditory skills in clinically depressed subjects. Experimental… (more)

Subjects/Keywords: Auditory perception.; Serotonin uptake inhibitors.; Depressed persons.; Serotonin; auditory processing; N1/P2; SSRI; auditory, depression

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6^th Edition):

Goodale, E. S. (2002). The Effects of Selective Serotonin Reuptake Inhibitors (Ssri) on Auditory Measures in Clinically Depressed Subjects. (Thesis). University of North Texas. Retrieved from https://digital.library.unt.edu/ark:/67531/metadc3146/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16^th Edition):

Goodale, Elizabeth S. “The Effects of Selective Serotonin Reuptake Inhibitors (Ssri) on Auditory Measures in Clinically Depressed Subjects.” 2002. Thesis, University of North Texas. Accessed January 28, 2021. https://digital.library.unt.edu/ark:/67531/metadc3146/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7^th Edition):

Goodale, Elizabeth S. “The Effects of Selective Serotonin Reuptake Inhibitors (Ssri) on Auditory Measures in Clinically Depressed Subjects.” 2002. Web. 28 Jan 2021.

Vancouver:

Goodale ES. The Effects of Selective Serotonin Reuptake Inhibitors (Ssri) on Auditory Measures in Clinically Depressed Subjects. [Internet] [Thesis]. University of North Texas; 2002. [cited 2021 Jan 28]. Available from: https://digital.library.unt.edu/ark:/67531/metadc3146/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Goodale ES. The Effects of Selective Serotonin Reuptake Inhibitors (Ssri) on Auditory Measures in Clinically Depressed Subjects. [Thesis]. University of North Texas; 2002. Available from: https://digital.library.unt.edu/ark:/67531/metadc3146/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

University of North Texas

8. Miller, Benjamin R. Concentration-dependent Effects of D-Methylphenidate on Frontal Cortex and Spinal Cord Networks in vitro.

Degree: 2004, University of North Texas

URL: https://digital.library.unt.edu/ark:/67531/metadc4689/

► Spontaneously active frontal cortex and spinal cord networks grown on microelectrode arrays were used to study effects of D-methylphenidate. These central nervous system tissues have… (more)

Subjects/Keywords: Neural networks (Neurobiology); Methylphenidate.; methylphenidate; dopamine; norepinephrine; neuronal networks

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APA (6^th Edition):

Miller, B. R. (2004). Concentration-dependent Effects of D-Methylphenidate on Frontal Cortex and Spinal Cord Networks in vitro. (Thesis). University of North Texas. Retrieved from https://digital.library.unt.edu/ark:/67531/metadc4689/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16^th Edition):

Miller, Benjamin R. “Concentration-dependent Effects of D-Methylphenidate on Frontal Cortex and Spinal Cord Networks in vitro.” 2004. Thesis, University of North Texas. Accessed January 28, 2021. https://digital.library.unt.edu/ark:/67531/metadc4689/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7^th Edition):

Miller, Benjamin R. “Concentration-dependent Effects of D-Methylphenidate on Frontal Cortex and Spinal Cord Networks in vitro.” 2004. Web. 28 Jan 2021.

Vancouver:

Miller BR. Concentration-dependent Effects of D-Methylphenidate on Frontal Cortex and Spinal Cord Networks in vitro. [Internet] [Thesis]. University of North Texas; 2004. [cited 2021 Jan 28]. Available from: https://digital.library.unt.edu/ark:/67531/metadc4689/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Miller BR. Concentration-dependent Effects of D-Methylphenidate on Frontal Cortex and Spinal Cord Networks in vitro. [Thesis]. University of North Texas; 2004. Available from: https://digital.library.unt.edu/ark:/67531/metadc4689/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

University of North Texas

9. Oli-Rijal, Sabnam. Determination of Dissociation Constants for GABAA Receptor Antagonists using Spontaneously Active Neuronal Networks in vitro.

Degree: 2005, University of North Texas

URL: https://digital.library.unt.edu/ark:/67531/metadc4911/

► Changes in spontaneous spike activities recorded from murine frontal cortex networks grown on substrate-integrated microelectrodes were used to determine the dissociation constant (KB) of three… (more)

Subjects/Keywords: GABA – Antagonists.; Neural circuitry.; Mice – Nervous system.; muscimol; binding affinity; electrophysiology; GABAA receptor; gabazine; TMPP

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APA (6^th Edition):

Oli-Rijal, S. (2005). Determination of Dissociation Constants for GABAA Receptor Antagonists using Spontaneously Active Neuronal Networks in vitro. (Thesis). University of North Texas. Retrieved from https://digital.library.unt.edu/ark:/67531/metadc4911/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16^th Edition):

Oli-Rijal, Sabnam. “Determination of Dissociation Constants for GABAA Receptor Antagonists using Spontaneously Active Neuronal Networks in vitro.” 2005. Thesis, University of North Texas. Accessed January 28, 2021. https://digital.library.unt.edu/ark:/67531/metadc4911/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7^th Edition):

Oli-Rijal, Sabnam. “Determination of Dissociation Constants for GABAA Receptor Antagonists using Spontaneously Active Neuronal Networks in vitro.” 2005. Web. 28 Jan 2021.

Vancouver:

Oli-Rijal S. Determination of Dissociation Constants for GABAA Receptor Antagonists using Spontaneously Active Neuronal Networks in vitro. [Internet] [Thesis]. University of North Texas; 2005. [cited 2021 Jan 28]. Available from: https://digital.library.unt.edu/ark:/67531/metadc4911/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Oli-Rijal S. Determination of Dissociation Constants for GABAA Receptor Antagonists using Spontaneously Active Neuronal Networks in vitro. [Thesis]. University of North Texas; 2005. Available from: https://digital.library.unt.edu/ark:/67531/metadc4911/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

University of North Texas

10. Robinson, Shirley R. (Shirley Ruth). Monaural and Binaural Speech Reception Thresholds in Normal Children and Those at Risk for Central Auditory Processing Disorders.

Degree: 1997, University of North Texas

URL: https://digital.library.unt.edu/ark:/67531/metadc277814/

► Children with central auditory processing disorders (CAPD) have a normal pure-tone audiogram, however, they have difficulty understanding speech in the presence of background noise. The… (more)

Subjects/Keywords: Word deafness in children.; Hearing disorders in children.; central auditory processing disorders; binaural hearing

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APA (6^th Edition):

Robinson, S. R. (. R. (1997). Monaural and Binaural Speech Reception Thresholds in Normal Children and Those at Risk for Central Auditory Processing Disorders. (Thesis). University of North Texas. Retrieved from https://digital.library.unt.edu/ark:/67531/metadc277814/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16^th Edition):

Robinson, Shirley R (Shirley Ruth). “Monaural and Binaural Speech Reception Thresholds in Normal Children and Those at Risk for Central Auditory Processing Disorders.” 1997. Thesis, University of North Texas. Accessed January 28, 2021. https://digital.library.unt.edu/ark:/67531/metadc277814/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7^th Edition):

Robinson, Shirley R (Shirley Ruth). “Monaural and Binaural Speech Reception Thresholds in Normal Children and Those at Risk for Central Auditory Processing Disorders.” 1997. Web. 28 Jan 2021.

Vancouver:

Robinson SR(R. Monaural and Binaural Speech Reception Thresholds in Normal Children and Those at Risk for Central Auditory Processing Disorders. [Internet] [Thesis]. University of North Texas; 1997. [cited 2021 Jan 28]. Available from: https://digital.library.unt.edu/ark:/67531/metadc277814/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Robinson SR(R. Monaural and Binaural Speech Reception Thresholds in Normal Children and Those at Risk for Central Auditory Processing Disorders. [Thesis]. University of North Texas; 1997. Available from: https://digital.library.unt.edu/ark:/67531/metadc277814/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

University of North Texas

11. Gargan, Lynn. Age-Dependent Effects Of Chronic GABAA Receptor Blockade In Barrel Cortex.

Degree: 2001, University of North Texas

URL: https://digital.library.unt.edu/ark:/67531/metadc2760/

► GABAA receptor binding is transiently increased in rat whisker barrels during the second postnatal week, at a time when neurons in the developing rat cortex… (more)

Subjects/Keywords: GABA – Receptors.; Rats – Nervous system.; GABAA receptor binding; Excitotoxic effects; Postnatal development

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6^th Edition):

Gargan, L. (2001). Age-Dependent Effects Of Chronic GABAA Receptor Blockade In Barrel Cortex. (Thesis). University of North Texas. Retrieved from https://digital.library.unt.edu/ark:/67531/metadc2760/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16^th Edition):

Gargan, Lynn. “Age-Dependent Effects Of Chronic GABAA Receptor Blockade In Barrel Cortex.” 2001. Thesis, University of North Texas. Accessed January 28, 2021. https://digital.library.unt.edu/ark:/67531/metadc2760/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7^th Edition):

Gargan, Lynn. “Age-Dependent Effects Of Chronic GABAA Receptor Blockade In Barrel Cortex.” 2001. Web. 28 Jan 2021.

Vancouver:

Gargan L. Age-Dependent Effects Of Chronic GABAA Receptor Blockade In Barrel Cortex. [Internet] [Thesis]. University of North Texas; 2001. [cited 2021 Jan 28]. Available from: https://digital.library.unt.edu/ark:/67531/metadc2760/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Gargan L. Age-Dependent Effects Of Chronic GABAA Receptor Blockade In Barrel Cortex. [Thesis]. University of North Texas; 2001. Available from: https://digital.library.unt.edu/ark:/67531/metadc2760/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

University of North Texas

12. Bishop, Charles E. A preliminary study of the effects of selective-serotonin re-uptake inhibitors (SSRIs) on central auditory processing.

Degree: 2001, University of North Texas

URL: https://digital.library.unt.edu/ark:/67531/metadc2853/

► his study compared auditory behavioral and physiological measures among three subject groups: 1) Normal control subjects, 2) subjects who were on a prescribed SSRI for… (more)

Subjects/Keywords: Auditory perception.; Serotonin uptake inhibitors.; Serotonin; auditory processing; auditory-evoked potentials

Record Details Similar Records Cite « Share

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6^th Edition):

Bishop, C. E. (2001). A preliminary study of the effects of selective-serotonin re-uptake inhibitors (SSRIs) on central auditory processing. (Thesis). University of North Texas. Retrieved from https://digital.library.unt.edu/ark:/67531/metadc2853/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16^th Edition):

Bishop, Charles E. “A preliminary study of the effects of selective-serotonin re-uptake inhibitors (SSRIs) on central auditory processing.” 2001. Thesis, University of North Texas. Accessed January 28, 2021. https://digital.library.unt.edu/ark:/67531/metadc2853/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7^th Edition):

Bishop, Charles E. “A preliminary study of the effects of selective-serotonin re-uptake inhibitors (SSRIs) on central auditory processing.” 2001. Web. 28 Jan 2021.

Vancouver:

Bishop CE. A preliminary study of the effects of selective-serotonin re-uptake inhibitors (SSRIs) on central auditory processing. [Internet] [Thesis]. University of North Texas; 2001. [cited 2021 Jan 28]. Available from: https://digital.library.unt.edu/ark:/67531/metadc2853/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Bishop CE. A preliminary study of the effects of selective-serotonin re-uptake inhibitors (SSRIs) on central auditory processing. [Thesis]. University of North Texas; 2001. Available from: https://digital.library.unt.edu/ark:/67531/metadc2853/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

University of North Texas

13. Parviz, Maryam. Evaluation of Zinc Toxicity Using Neuronal Networks on Microelectrode Arrays: Response Quantification and Entry Pathway Analysis.

Degree: 2007, University of North Texas

URL: https://digital.library.unt.edu/ark:/67531/metadc3928/

► Murine neuronal networks, derived from embryonic frontal cortex (FC) tissue grown on microelectrode arrays, were used to investigate zinc toxicity at concentrations ranging from 20… (more)

Subjects/Keywords: Neurotoxicity; zinc; microelectrode array; Zinc – Toxicology.; Mice – Effect of metals on.; Neural networks (Neurobiology); Mice – Nervous system.

Record Details Similar Records Cite « Share

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6^th Edition):

Parviz, M. (2007). Evaluation of Zinc Toxicity Using Neuronal Networks on Microelectrode Arrays: Response Quantification and Entry Pathway Analysis. (Thesis). University of North Texas. Retrieved from https://digital.library.unt.edu/ark:/67531/metadc3928/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16^th Edition):

Parviz, Maryam. “Evaluation of Zinc Toxicity Using Neuronal Networks on Microelectrode Arrays: Response Quantification and Entry Pathway Analysis.” 2007. Thesis, University of North Texas. Accessed January 28, 2021. https://digital.library.unt.edu/ark:/67531/metadc3928/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7^th Edition):

Parviz, Maryam. “Evaluation of Zinc Toxicity Using Neuronal Networks on Microelectrode Arrays: Response Quantification and Entry Pathway Analysis.” 2007. Web. 28 Jan 2021.

Vancouver:

Parviz M. Evaluation of Zinc Toxicity Using Neuronal Networks on Microelectrode Arrays: Response Quantification and Entry Pathway Analysis. [Internet] [Thesis]. University of North Texas; 2007. [cited 2021 Jan 28]. Available from: https://digital.library.unt.edu/ark:/67531/metadc3928/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Parviz M. Evaluation of Zinc Toxicity Using Neuronal Networks on Microelectrode Arrays: Response Quantification and Entry Pathway Analysis. [Thesis]. University of North Texas; 2007. Available from: https://digital.library.unt.edu/ark:/67531/metadc3928/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

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