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You searched for +publisher:"University of North Carolina" +contributor:("Su, Lishan"). Showing records 1 – 12 of 12 total matches.

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University of North Carolina

1. Hench, Virginia Kathleen. Molecular mechanisms of Foxp3-mediated gene regulation and their contribution to T cell and thymic epithelial cell function.

Degree: Microbiology and Immunology, 2010, University of North Carolina

 Severe autoinflammatory disease is associated with mutations in the Foxp3 gene in mice and humans. Foxp3 is required for the development, function, and maintenance of… (more)

Subjects/Keywords: School of Medicine; Department of Microbiology and Immunology

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APA (6th Edition):

Hench, V. K. (2010). Molecular mechanisms of Foxp3-mediated gene regulation and their contribution to T cell and thymic epithelial cell function. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:a63cde9a-a526-48ad-8c08-98752235d19a

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hench, Virginia Kathleen. “Molecular mechanisms of Foxp3-mediated gene regulation and their contribution to T cell and thymic epithelial cell function.” 2010. Thesis, University of North Carolina. Accessed November 28, 2020. https://cdr.lib.unc.edu/record/uuid:a63cde9a-a526-48ad-8c08-98752235d19a.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hench, Virginia Kathleen. “Molecular mechanisms of Foxp3-mediated gene regulation and their contribution to T cell and thymic epithelial cell function.” 2010. Web. 28 Nov 2020.

Vancouver:

Hench VK. Molecular mechanisms of Foxp3-mediated gene regulation and their contribution to T cell and thymic epithelial cell function. [Internet] [Thesis]. University of North Carolina; 2010. [cited 2020 Nov 28]. Available from: https://cdr.lib.unc.edu/record/uuid:a63cde9a-a526-48ad-8c08-98752235d19a.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hench VK. Molecular mechanisms of Foxp3-mediated gene regulation and their contribution to T cell and thymic epithelial cell function. [Thesis]. University of North Carolina; 2010. Available from: https://cdr.lib.unc.edu/record/uuid:a63cde9a-a526-48ad-8c08-98752235d19a

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

2. Washburn, Michael L. Modeling HCV infection, immunopathogenesis, and therapy in humanized mice.

Degree: 2010, University of North Carolina

 Approximately 170 million people worldwide are chronically infected by the hepatitis C virus (HCV), which is often associated with impaired T cell responses to viral… (more)

Subjects/Keywords: School of Medicine; Curriculum in Genetics and Molecular Biology

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APA (6th Edition):

Washburn, M. L. (2010). Modeling HCV infection, immunopathogenesis, and therapy in humanized mice. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:552f98ec-5dc8-4da2-afbe-831b5de5ffc0

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Washburn, Michael L. “Modeling HCV infection, immunopathogenesis, and therapy in humanized mice.” 2010. Thesis, University of North Carolina. Accessed November 28, 2020. https://cdr.lib.unc.edu/record/uuid:552f98ec-5dc8-4da2-afbe-831b5de5ffc0.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Washburn, Michael L. “Modeling HCV infection, immunopathogenesis, and therapy in humanized mice.” 2010. Web. 28 Nov 2020.

Vancouver:

Washburn ML. Modeling HCV infection, immunopathogenesis, and therapy in humanized mice. [Internet] [Thesis]. University of North Carolina; 2010. [cited 2020 Nov 28]. Available from: https://cdr.lib.unc.edu/record/uuid:552f98ec-5dc8-4da2-afbe-831b5de5ffc0.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Washburn ML. Modeling HCV infection, immunopathogenesis, and therapy in humanized mice. [Thesis]. University of North Carolina; 2010. Available from: https://cdr.lib.unc.edu/record/uuid:552f98ec-5dc8-4da2-afbe-831b5de5ffc0

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

3. Arrildt, Kathryn. Phenotypic and Genotypic Features of Macrophage Tropism in HIV-1.

Degree: Microbiology and Immunology, 2016, University of North Carolina

 HIV-1 preferentially attaches and enters specific cell types by recognizing cell-specific receptors. HIV-1 typically targets memory CD4+ T cells by restricting attachment to cells expressing… (more)

Subjects/Keywords: School of Medicine; Department of Microbiology and Immunology

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APA (6th Edition):

Arrildt, K. (2016). Phenotypic and Genotypic Features of Macrophage Tropism in HIV-1. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:650b39d7-9d1e-4d34-8e5b-a219db67813e

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Arrildt, Kathryn. “Phenotypic and Genotypic Features of Macrophage Tropism in HIV-1.” 2016. Thesis, University of North Carolina. Accessed November 28, 2020. https://cdr.lib.unc.edu/record/uuid:650b39d7-9d1e-4d34-8e5b-a219db67813e.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Arrildt, Kathryn. “Phenotypic and Genotypic Features of Macrophage Tropism in HIV-1.” 2016. Web. 28 Nov 2020.

Vancouver:

Arrildt K. Phenotypic and Genotypic Features of Macrophage Tropism in HIV-1. [Internet] [Thesis]. University of North Carolina; 2016. [cited 2020 Nov 28]. Available from: https://cdr.lib.unc.edu/record/uuid:650b39d7-9d1e-4d34-8e5b-a219db67813e.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Arrildt K. Phenotypic and Genotypic Features of Macrophage Tropism in HIV-1. [Thesis]. University of North Carolina; 2016. Available from: https://cdr.lib.unc.edu/record/uuid:650b39d7-9d1e-4d34-8e5b-a219db67813e

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

4. Maltez, Vivien. Inflammasomes and Cytotoxic Lymphocytes Clear Bacteria From Different Intracellular Niches.

Degree: Microbiology and Immunology, 2017, University of North Carolina

 Programmed cell death is a powerful anti-microbial defense. The innate immune system is our first line of defense against infection, and the induction of cell… (more)

Subjects/Keywords: School of Medicine; Department of Microbiology and Immunology

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APA (6th Edition):

Maltez, V. (2017). Inflammasomes and Cytotoxic Lymphocytes Clear Bacteria From Different Intracellular Niches. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:0eb5d888-178a-4a7c-b6af-78c92db7e502

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Maltez, Vivien. “Inflammasomes and Cytotoxic Lymphocytes Clear Bacteria From Different Intracellular Niches.” 2017. Thesis, University of North Carolina. Accessed November 28, 2020. https://cdr.lib.unc.edu/record/uuid:0eb5d888-178a-4a7c-b6af-78c92db7e502.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Maltez, Vivien. “Inflammasomes and Cytotoxic Lymphocytes Clear Bacteria From Different Intracellular Niches.” 2017. Web. 28 Nov 2020.

Vancouver:

Maltez V. Inflammasomes and Cytotoxic Lymphocytes Clear Bacteria From Different Intracellular Niches. [Internet] [Thesis]. University of North Carolina; 2017. [cited 2020 Nov 28]. Available from: https://cdr.lib.unc.edu/record/uuid:0eb5d888-178a-4a7c-b6af-78c92db7e502.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Maltez V. Inflammasomes and Cytotoxic Lymphocytes Clear Bacteria From Different Intracellular Niches. [Thesis]. University of North Carolina; 2017. Available from: https://cdr.lib.unc.edu/record/uuid:0eb5d888-178a-4a7c-b6af-78c92db7e502

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

5. Morillon, Yves. THYMIC DEVELOPMENT OF AUTOREACTIVE T CELLS IN NOD MICE, AND THE ALTERED TRAFFICKING OF T CELLS FOLLOWING ANTIBODY MEDIATED CROSSLINKING OF THE CD4 CORECEPTOR.

Degree: Microbiology and Immunology, 2014, University of North Carolina

 Two key processes in driving type 1 diabetes are the development and trafficking of β cell-specific T cells into the pancreas. The first part of… (more)

Subjects/Keywords: Immunology; School of Medicine; Department of Microbiology and Immunology

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APA (6th Edition):

Morillon, Y. (2014). THYMIC DEVELOPMENT OF AUTOREACTIVE T CELLS IN NOD MICE, AND THE ALTERED TRAFFICKING OF T CELLS FOLLOWING ANTIBODY MEDIATED CROSSLINKING OF THE CD4 CORECEPTOR. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:6779d3c5-3461-4486-aef2-33ac9de0cd3c

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Morillon, Yves. “THYMIC DEVELOPMENT OF AUTOREACTIVE T CELLS IN NOD MICE, AND THE ALTERED TRAFFICKING OF T CELLS FOLLOWING ANTIBODY MEDIATED CROSSLINKING OF THE CD4 CORECEPTOR.” 2014. Thesis, University of North Carolina. Accessed November 28, 2020. https://cdr.lib.unc.edu/record/uuid:6779d3c5-3461-4486-aef2-33ac9de0cd3c.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Morillon, Yves. “THYMIC DEVELOPMENT OF AUTOREACTIVE T CELLS IN NOD MICE, AND THE ALTERED TRAFFICKING OF T CELLS FOLLOWING ANTIBODY MEDIATED CROSSLINKING OF THE CD4 CORECEPTOR.” 2014. Web. 28 Nov 2020.

Vancouver:

Morillon Y. THYMIC DEVELOPMENT OF AUTOREACTIVE T CELLS IN NOD MICE, AND THE ALTERED TRAFFICKING OF T CELLS FOLLOWING ANTIBODY MEDIATED CROSSLINKING OF THE CD4 CORECEPTOR. [Internet] [Thesis]. University of North Carolina; 2014. [cited 2020 Nov 28]. Available from: https://cdr.lib.unc.edu/record/uuid:6779d3c5-3461-4486-aef2-33ac9de0cd3c.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Morillon Y. THYMIC DEVELOPMENT OF AUTOREACTIVE T CELLS IN NOD MICE, AND THE ALTERED TRAFFICKING OF T CELLS FOLLOWING ANTIBODY MEDIATED CROSSLINKING OF THE CD4 CORECEPTOR. [Thesis]. University of North Carolina; 2014. Available from: https://cdr.lib.unc.edu/record/uuid:6779d3c5-3461-4486-aef2-33ac9de0cd3c

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

6. Hagar, Jon. Cytosolic LPS Activates Caspase-11: Implications for Innate Immunity and Management of Septic Shock.

Degree: Microbiology and Immunology, 2016, University of North Carolina

 Caspases are either apoptotic or inflammatory. Amongst inflammatory caspases, caspase-1 and -11 trigger pyroptosis, a form of programmed cell death. Whereas both can be detrimental… (more)

Subjects/Keywords: School of Medicine; Department of Microbiology and Immunology

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APA (6th Edition):

Hagar, J. (2016). Cytosolic LPS Activates Caspase-11: Implications for Innate Immunity and Management of Septic Shock. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:228338b1-5175-41f5-a547-f6e0038498bd

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hagar, Jon. “Cytosolic LPS Activates Caspase-11: Implications for Innate Immunity and Management of Septic Shock.” 2016. Thesis, University of North Carolina. Accessed November 28, 2020. https://cdr.lib.unc.edu/record/uuid:228338b1-5175-41f5-a547-f6e0038498bd.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hagar, Jon. “Cytosolic LPS Activates Caspase-11: Implications for Innate Immunity and Management of Septic Shock.” 2016. Web. 28 Nov 2020.

Vancouver:

Hagar J. Cytosolic LPS Activates Caspase-11: Implications for Innate Immunity and Management of Septic Shock. [Internet] [Thesis]. University of North Carolina; 2016. [cited 2020 Nov 28]. Available from: https://cdr.lib.unc.edu/record/uuid:228338b1-5175-41f5-a547-f6e0038498bd.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hagar J. Cytosolic LPS Activates Caspase-11: Implications for Innate Immunity and Management of Septic Shock. [Thesis]. University of North Carolina; 2016. Available from: https://cdr.lib.unc.edu/record/uuid:228338b1-5175-41f5-a547-f6e0038498bd

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

7. Manzoor, Fatima. Tissue-specific cytokine-based immunotherapy to treat type 1 diabetes.

Degree: Microbiology and Immunology, 2016, University of North Carolina

 Type 1 diabetes (T1D) is characterized by the T cell-mediated destruction of the insulin-producing β cells. Progressive loss of β cell mass results in the… (more)

Subjects/Keywords: School of Medicine; Department of Microbiology and Immunology

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APA (6th Edition):

Manzoor, F. (2016). Tissue-specific cytokine-based immunotherapy to treat type 1 diabetes. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:3d21e01f-87c6-48ae-bbe7-f677faa3f639

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Manzoor, Fatima. “Tissue-specific cytokine-based immunotherapy to treat type 1 diabetes.” 2016. Thesis, University of North Carolina. Accessed November 28, 2020. https://cdr.lib.unc.edu/record/uuid:3d21e01f-87c6-48ae-bbe7-f677faa3f639.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Manzoor, Fatima. “Tissue-specific cytokine-based immunotherapy to treat type 1 diabetes.” 2016. Web. 28 Nov 2020.

Vancouver:

Manzoor F. Tissue-specific cytokine-based immunotherapy to treat type 1 diabetes. [Internet] [Thesis]. University of North Carolina; 2016. [cited 2020 Nov 28]. Available from: https://cdr.lib.unc.edu/record/uuid:3d21e01f-87c6-48ae-bbe7-f677faa3f639.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Manzoor F. Tissue-specific cytokine-based immunotherapy to treat type 1 diabetes. [Thesis]. University of North Carolina; 2016. Available from: https://cdr.lib.unc.edu/record/uuid:3d21e01f-87c6-48ae-bbe7-f677faa3f639

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

8. Callaway, Justin. Interfering With Inflammatory Cytokine Production During Antibody-Enhanced Dengue Virus Infection.

Degree: Microbiology and Immunology, 2014, University of North Carolina

 Dengue virus (DENV) is the most burdensome arbovirus in the world, infecting nearly 400 million people annually. Patients develop lifelong immunity to the original infecting… (more)

Subjects/Keywords: Immunology; Microbiology; Virology; School of Medicine; Department of Microbiology and Immunology

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APA (6th Edition):

Callaway, J. (2014). Interfering With Inflammatory Cytokine Production During Antibody-Enhanced Dengue Virus Infection. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:fe6f5654-fb88-4343-a1c0-6a93a65e7145

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Callaway, Justin. “Interfering With Inflammatory Cytokine Production During Antibody-Enhanced Dengue Virus Infection.” 2014. Thesis, University of North Carolina. Accessed November 28, 2020. https://cdr.lib.unc.edu/record/uuid:fe6f5654-fb88-4343-a1c0-6a93a65e7145.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Callaway, Justin. “Interfering With Inflammatory Cytokine Production During Antibody-Enhanced Dengue Virus Infection.” 2014. Web. 28 Nov 2020.

Vancouver:

Callaway J. Interfering With Inflammatory Cytokine Production During Antibody-Enhanced Dengue Virus Infection. [Internet] [Thesis]. University of North Carolina; 2014. [cited 2020 Nov 28]. Available from: https://cdr.lib.unc.edu/record/uuid:fe6f5654-fb88-4343-a1c0-6a93a65e7145.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Callaway J. Interfering With Inflammatory Cytokine Production During Antibody-Enhanced Dengue Virus Infection. [Thesis]. University of North Carolina; 2014. Available from: https://cdr.lib.unc.edu/record/uuid:fe6f5654-fb88-4343-a1c0-6a93a65e7145

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

9. Tsao, Li-Chung. Virus-Host Interactions in HIV-1 Pathogenesis and Viral Immune Evasion.

Degree: 2016, University of North Carolina

 Human viruses such as the Human Immunodeficiency virus (HIV-1) causes chronic diseases and is a major public health concern. My dissertation focuses on the molecular… (more)

Subjects/Keywords: School of Medicine; Curriculum in Genetics and Molecular Biology

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APA (6th Edition):

Tsao, L. (2016). Virus-Host Interactions in HIV-1 Pathogenesis and Viral Immune Evasion. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:d21f72b1-b423-4f34-a6f3-a474cb2b261d

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Tsao, Li-Chung. “Virus-Host Interactions in HIV-1 Pathogenesis and Viral Immune Evasion.” 2016. Thesis, University of North Carolina. Accessed November 28, 2020. https://cdr.lib.unc.edu/record/uuid:d21f72b1-b423-4f34-a6f3-a474cb2b261d.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Tsao, Li-Chung. “Virus-Host Interactions in HIV-1 Pathogenesis and Viral Immune Evasion.” 2016. Web. 28 Nov 2020.

Vancouver:

Tsao L. Virus-Host Interactions in HIV-1 Pathogenesis and Viral Immune Evasion. [Internet] [Thesis]. University of North Carolina; 2016. [cited 2020 Nov 28]. Available from: https://cdr.lib.unc.edu/record/uuid:d21f72b1-b423-4f34-a6f3-a474cb2b261d.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Tsao L. Virus-Host Interactions in HIV-1 Pathogenesis and Viral Immune Evasion. [Thesis]. University of North Carolina; 2016. Available from: https://cdr.lib.unc.edu/record/uuid:d21f72b1-b423-4f34-a6f3-a474cb2b261d

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

10. Sivaraman, Vijay. Mechanisms of HIV-1-mediated CD4+ T cell depletion in lymphoid tissue.

Degree: Microbiology and Immunology, 2009, University of North Carolina

 In this dissertation, I investigate the methods employed by HIV-1 to cause depletion of CD4+ cells within the lymphoid organ. Human ex-vivo tissue models for… (more)

Subjects/Keywords: School of Medicine; Department of Microbiology and Immunology

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APA (6th Edition):

Sivaraman, V. (2009). Mechanisms of HIV-1-mediated CD4+ T cell depletion in lymphoid tissue. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:c07e22be-510e-486c-b8a6-16a26491a6f8

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Sivaraman, Vijay. “Mechanisms of HIV-1-mediated CD4+ T cell depletion in lymphoid tissue.” 2009. Thesis, University of North Carolina. Accessed November 28, 2020. https://cdr.lib.unc.edu/record/uuid:c07e22be-510e-486c-b8a6-16a26491a6f8.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Sivaraman, Vijay. “Mechanisms of HIV-1-mediated CD4+ T cell depletion in lymphoid tissue.” 2009. Web. 28 Nov 2020.

Vancouver:

Sivaraman V. Mechanisms of HIV-1-mediated CD4+ T cell depletion in lymphoid tissue. [Internet] [Thesis]. University of North Carolina; 2009. [cited 2020 Nov 28]. Available from: https://cdr.lib.unc.edu/record/uuid:c07e22be-510e-486c-b8a6-16a26491a6f8.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Sivaraman V. Mechanisms of HIV-1-mediated CD4+ T cell depletion in lymphoid tissue. [Thesis]. University of North Carolina; 2009. Available from: https://cdr.lib.unc.edu/record/uuid:c07e22be-510e-486c-b8a6-16a26491a6f8

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

11. Holmes, Derek Allan. Modulation of HIV-1 replication and T cell activation by FoxP3.

Degree: Microbiology and Immunology, 2008, University of North Carolina

 Regulatory T cells (Treg) are defined as the population of CD4+CD25+ that express the forkhead transcription factor family member FoxP3. Treg cells are characterized by… (more)

Subjects/Keywords: School of Medicine; Department of Microbiology and Immunology

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APA (6th Edition):

Holmes, D. A. (2008). Modulation of HIV-1 replication and T cell activation by FoxP3. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:046d89cf-4241-47b0-82a0-3c493ddf9869

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Holmes, Derek Allan. “Modulation of HIV-1 replication and T cell activation by FoxP3.” 2008. Thesis, University of North Carolina. Accessed November 28, 2020. https://cdr.lib.unc.edu/record/uuid:046d89cf-4241-47b0-82a0-3c493ddf9869.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Holmes, Derek Allan. “Modulation of HIV-1 replication and T cell activation by FoxP3.” 2008. Web. 28 Nov 2020.

Vancouver:

Holmes DA. Modulation of HIV-1 replication and T cell activation by FoxP3. [Internet] [Thesis]. University of North Carolina; 2008. [cited 2020 Nov 28]. Available from: https://cdr.lib.unc.edu/record/uuid:046d89cf-4241-47b0-82a0-3c493ddf9869.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Holmes DA. Modulation of HIV-1 replication and T cell activation by FoxP3. [Thesis]. University of North Carolina; 2008. Available from: https://cdr.lib.unc.edu/record/uuid:046d89cf-4241-47b0-82a0-3c493ddf9869

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

12. Loomis, Rebecca Jo. The role of RhoA signaling pathways in regulating HIV-1 replication.

Degree: 2006, University of North Carolina

 RhoGTPases are involved in regulating important cellular processes including cytoskeletal organization, gene transcription and membrane vesicle transport – poising RhoGTPases to link these pathways to… (more)

Subjects/Keywords: School of Medicine; Curriculum in Genetics and Molecular Biology

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APA (6th Edition):

Loomis, R. J. (2006). The role of RhoA signaling pathways in regulating HIV-1 replication. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:00700710-911f-42b0-b877-4deb7a08f50d

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Loomis, Rebecca Jo. “The role of RhoA signaling pathways in regulating HIV-1 replication.” 2006. Thesis, University of North Carolina. Accessed November 28, 2020. https://cdr.lib.unc.edu/record/uuid:00700710-911f-42b0-b877-4deb7a08f50d.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Loomis, Rebecca Jo. “The role of RhoA signaling pathways in regulating HIV-1 replication.” 2006. Web. 28 Nov 2020.

Vancouver:

Loomis RJ. The role of RhoA signaling pathways in regulating HIV-1 replication. [Internet] [Thesis]. University of North Carolina; 2006. [cited 2020 Nov 28]. Available from: https://cdr.lib.unc.edu/record/uuid:00700710-911f-42b0-b877-4deb7a08f50d.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Loomis RJ. The role of RhoA signaling pathways in regulating HIV-1 replication. [Thesis]. University of North Carolina; 2006. Available from: https://cdr.lib.unc.edu/record/uuid:00700710-911f-42b0-b877-4deb7a08f50d

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

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