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You searched for +publisher:"University of North Carolina" +contributor:("McCarthy, Ken D."). Showing records 1 – 12 of 12 total matches.

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University of North Carolina

1. Campagnola, Luke. Mapping the functional connectivity of the mouse anteroventral cochlear nucleus.

Degree: 2013, University of North Carolina

 The cochlear nucleus is the first central processor of auditory information and provides afferent input to most of the major brainstem and midbrain auditory nuclei.… (more)

Subjects/Keywords: School of Medicine; UNC Neuroscience Center; Neuroscience Curriculum

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APA (6th Edition):

Campagnola, L. (2013). Mapping the functional connectivity of the mouse anteroventral cochlear nucleus. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:4e1e7988-541d-48d1-9b3c-45955f4c5fa3

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Campagnola, Luke. “Mapping the functional connectivity of the mouse anteroventral cochlear nucleus.” 2013. Thesis, University of North Carolina. Accessed December 05, 2020. https://cdr.lib.unc.edu/record/uuid:4e1e7988-541d-48d1-9b3c-45955f4c5fa3.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Campagnola, Luke. “Mapping the functional connectivity of the mouse anteroventral cochlear nucleus.” 2013. Web. 05 Dec 2020.

Vancouver:

Campagnola L. Mapping the functional connectivity of the mouse anteroventral cochlear nucleus. [Internet] [Thesis]. University of North Carolina; 2013. [cited 2020 Dec 05]. Available from: https://cdr.lib.unc.edu/record/uuid:4e1e7988-541d-48d1-9b3c-45955f4c5fa3.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Campagnola L. Mapping the functional connectivity of the mouse anteroventral cochlear nucleus. [Thesis]. University of North Carolina; 2013. Available from: https://cdr.lib.unc.edu/record/uuid:4e1e7988-541d-48d1-9b3c-45955f4c5fa3

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

2. Petravicz, Jeremy C. Challenging the Dogma: Reevaluating the Role of Astrocyte Calcium Signaling in Physiology.

Degree: 2010, University of North Carolina

 Gliotransmission represents one of the most important conceptual shifts in neuroscience in the past several decades. Gliotransmission refers to the process whereby glial cells release… (more)

Subjects/Keywords: School of Medicine; UNC Neuroscience Center; Neuroscience Curriculum

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APA (6th Edition):

Petravicz, J. C. (2010). Challenging the Dogma: Reevaluating the Role of Astrocyte Calcium Signaling in Physiology. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:ef7cec8c-db8a-4949-8c49-b0b3eade2ce6

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Petravicz, Jeremy C. “Challenging the Dogma: Reevaluating the Role of Astrocyte Calcium Signaling in Physiology.” 2010. Thesis, University of North Carolina. Accessed December 05, 2020. https://cdr.lib.unc.edu/record/uuid:ef7cec8c-db8a-4949-8c49-b0b3eade2ce6.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Petravicz, Jeremy C. “Challenging the Dogma: Reevaluating the Role of Astrocyte Calcium Signaling in Physiology.” 2010. Web. 05 Dec 2020.

Vancouver:

Petravicz JC. Challenging the Dogma: Reevaluating the Role of Astrocyte Calcium Signaling in Physiology. [Internet] [Thesis]. University of North Carolina; 2010. [cited 2020 Dec 05]. Available from: https://cdr.lib.unc.edu/record/uuid:ef7cec8c-db8a-4949-8c49-b0b3eade2ce6.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Petravicz JC. Challenging the Dogma: Reevaluating the Role of Astrocyte Calcium Signaling in Physiology. [Thesis]. University of North Carolina; 2010. Available from: https://cdr.lib.unc.edu/record/uuid:ef7cec8c-db8a-4949-8c49-b0b3eade2ce6

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

3. McMullen, Allison B. Cellular, Histological and Behavioral Changes in the Pathogenesis of Hydrocephalus in the Ro1 Mouse Model.

Degree: Pharmacology, 2011, University of North Carolina

 Hydrocephalus is a highly prevalent neurological disorder characterized by elevated levels of cerebrospinal fluid (CSF) in the brain and subsequent enlargement of the lateral ventricles.… (more)

Subjects/Keywords: School of Medicine; Department of Pharmacology

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APA (6th Edition):

McMullen, A. B. (2011). Cellular, Histological and Behavioral Changes in the Pathogenesis of Hydrocephalus in the Ro1 Mouse Model. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:79730dbd-c913-4972-bbd9-0378ba2f39aa

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

McMullen, Allison B. “Cellular, Histological and Behavioral Changes in the Pathogenesis of Hydrocephalus in the Ro1 Mouse Model.” 2011. Thesis, University of North Carolina. Accessed December 05, 2020. https://cdr.lib.unc.edu/record/uuid:79730dbd-c913-4972-bbd9-0378ba2f39aa.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

McMullen, Allison B. “Cellular, Histological and Behavioral Changes in the Pathogenesis of Hydrocephalus in the Ro1 Mouse Model.” 2011. Web. 05 Dec 2020.

Vancouver:

McMullen AB. Cellular, Histological and Behavioral Changes in the Pathogenesis of Hydrocephalus in the Ro1 Mouse Model. [Internet] [Thesis]. University of North Carolina; 2011. [cited 2020 Dec 05]. Available from: https://cdr.lib.unc.edu/record/uuid:79730dbd-c913-4972-bbd9-0378ba2f39aa.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

McMullen AB. Cellular, Histological and Behavioral Changes in the Pathogenesis of Hydrocephalus in the Ro1 Mouse Model. [Thesis]. University of North Carolina; 2011. Available from: https://cdr.lib.unc.edu/record/uuid:79730dbd-c913-4972-bbd9-0378ba2f39aa

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

4. Williams, Kimberly. NEUROTROPHIN-CHEMOKINE RECEPTOR INTERACTIONS ON MACROPHAGES REGULATE HIV NEUROPATHOGENESIS.

Degree: 2015, University of North Carolina

 Invading perivascular macrophages and microglial cells play a pivotal role in the neuropathogenesis of Human Immunodeficiency Virus (HIV) associated neurological disorders. As with many neurodegenerative… (more)

Subjects/Keywords: Neurosciences; Immunology; School of Medicine; UNC Neuroscience Center; Neuroscience Curriculum

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APA (6th Edition):

Williams, K. (2015). NEUROTROPHIN-CHEMOKINE RECEPTOR INTERACTIONS ON MACROPHAGES REGULATE HIV NEUROPATHOGENESIS. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:79d4ec87-ae10-4766-b524-40c7700a3f7e

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Williams, Kimberly. “NEUROTROPHIN-CHEMOKINE RECEPTOR INTERACTIONS ON MACROPHAGES REGULATE HIV NEUROPATHOGENESIS.” 2015. Thesis, University of North Carolina. Accessed December 05, 2020. https://cdr.lib.unc.edu/record/uuid:79d4ec87-ae10-4766-b524-40c7700a3f7e.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Williams, Kimberly. “NEUROTROPHIN-CHEMOKINE RECEPTOR INTERACTIONS ON MACROPHAGES REGULATE HIV NEUROPATHOGENESIS.” 2015. Web. 05 Dec 2020.

Vancouver:

Williams K. NEUROTROPHIN-CHEMOKINE RECEPTOR INTERACTIONS ON MACROPHAGES REGULATE HIV NEUROPATHOGENESIS. [Internet] [Thesis]. University of North Carolina; 2015. [cited 2020 Dec 05]. Available from: https://cdr.lib.unc.edu/record/uuid:79d4ec87-ae10-4766-b524-40c7700a3f7e.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Williams K. NEUROTROPHIN-CHEMOKINE RECEPTOR INTERACTIONS ON MACROPHAGES REGULATE HIV NEUROPATHOGENESIS. [Thesis]. University of North Carolina; 2015. Available from: https://cdr.lib.unc.edu/record/uuid:79d4ec87-ae10-4766-b524-40c7700a3f7e

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

5. Hartung, Jane. Distinct Mechanisms Underlie the Onset and Maintenance of Pain Linked to Catecholamine Dysregulation.

Degree: 2016, University of North Carolina

 A body of evidence links decreased expression and activity of catechol-o-methyltransferase (COMT), an enzyme that metabolizes catecholamines, with idiopathic pain conditions including temporomandibular joint disorder… (more)

Subjects/Keywords: School of Medicine; UNC Neuroscience Center; Neuroscience Curriculum

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APA (6th Edition):

Hartung, J. (2016). Distinct Mechanisms Underlie the Onset and Maintenance of Pain Linked to Catecholamine Dysregulation. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:39168d46-0c8e-4304-826f-f4554de71ad0

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hartung, Jane. “Distinct Mechanisms Underlie the Onset and Maintenance of Pain Linked to Catecholamine Dysregulation.” 2016. Thesis, University of North Carolina. Accessed December 05, 2020. https://cdr.lib.unc.edu/record/uuid:39168d46-0c8e-4304-826f-f4554de71ad0.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hartung, Jane. “Distinct Mechanisms Underlie the Onset and Maintenance of Pain Linked to Catecholamine Dysregulation.” 2016. Web. 05 Dec 2020.

Vancouver:

Hartung J. Distinct Mechanisms Underlie the Onset and Maintenance of Pain Linked to Catecholamine Dysregulation. [Internet] [Thesis]. University of North Carolina; 2016. [cited 2020 Dec 05]. Available from: https://cdr.lib.unc.edu/record/uuid:39168d46-0c8e-4304-826f-f4554de71ad0.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hartung J. Distinct Mechanisms Underlie the Onset and Maintenance of Pain Linked to Catecholamine Dysregulation. [Thesis]. University of North Carolina; 2016. Available from: https://cdr.lib.unc.edu/record/uuid:39168d46-0c8e-4304-826f-f4554de71ad0

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

6. Whitley, Sarah Taves. Investigating the Glial Contribution to Persistent Neuropathic Pain.

Degree: 2012, University of North Carolina

 Persistent neuropathic pain is the coordinated activation and sensitization of glial and neuronal elements both peripherally and centrally. Here, we have investigated the role of… (more)

Subjects/Keywords: School of Medicine; UNC Neuroscience Center; Neuroscience Curriculum

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APA (6th Edition):

Whitley, S. T. (2012). Investigating the Glial Contribution to Persistent Neuropathic Pain. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:f4ba56ae-65e4-427b-9206-7f605ea1f039

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Whitley, Sarah Taves. “Investigating the Glial Contribution to Persistent Neuropathic Pain.” 2012. Thesis, University of North Carolina. Accessed December 05, 2020. https://cdr.lib.unc.edu/record/uuid:f4ba56ae-65e4-427b-9206-7f605ea1f039.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Whitley, Sarah Taves. “Investigating the Glial Contribution to Persistent Neuropathic Pain.” 2012. Web. 05 Dec 2020.

Vancouver:

Whitley ST. Investigating the Glial Contribution to Persistent Neuropathic Pain. [Internet] [Thesis]. University of North Carolina; 2012. [cited 2020 Dec 05]. Available from: https://cdr.lib.unc.edu/record/uuid:f4ba56ae-65e4-427b-9206-7f605ea1f039.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Whitley ST. Investigating the Glial Contribution to Persistent Neuropathic Pain. [Thesis]. University of North Carolina; 2012. Available from: https://cdr.lib.unc.edu/record/uuid:f4ba56ae-65e4-427b-9206-7f605ea1f039

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

7. Minton, Suzanne Kurtzer. Stimulating Glial Gq-Coupled GPCR Pathways Blocks Acute Pain and Itch.

Degree: Pharmacology, 2013, University of North Carolina

 Glial G protein-coupled receptor (GPCR) pathways have been linked to synaptic modulation. However, their roles in pain and itch processing are unknown and have been… (more)

Subjects/Keywords: School of Medicine; Department of Pharmacology

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APA (6th Edition):

Minton, S. K. (2013). Stimulating Glial Gq-Coupled GPCR Pathways Blocks Acute Pain and Itch. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:529ba65f-eaa2-457b-94f4-3122199b0ec9

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Minton, Suzanne Kurtzer. “Stimulating Glial Gq-Coupled GPCR Pathways Blocks Acute Pain and Itch.” 2013. Thesis, University of North Carolina. Accessed December 05, 2020. https://cdr.lib.unc.edu/record/uuid:529ba65f-eaa2-457b-94f4-3122199b0ec9.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Minton, Suzanne Kurtzer. “Stimulating Glial Gq-Coupled GPCR Pathways Blocks Acute Pain and Itch.” 2013. Web. 05 Dec 2020.

Vancouver:

Minton SK. Stimulating Glial Gq-Coupled GPCR Pathways Blocks Acute Pain and Itch. [Internet] [Thesis]. University of North Carolina; 2013. [cited 2020 Dec 05]. Available from: https://cdr.lib.unc.edu/record/uuid:529ba65f-eaa2-457b-94f4-3122199b0ec9.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Minton SK. Stimulating Glial Gq-Coupled GPCR Pathways Blocks Acute Pain and Itch. [Thesis]. University of North Carolina; 2013. Available from: https://cdr.lib.unc.edu/record/uuid:529ba65f-eaa2-457b-94f4-3122199b0ec9

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

8. Bachleda, Amelia. SOX2 Is Essential for the Maturation and Maintenance of Retinal Müller Glia.

Degree: 2014, University of North Carolina

 Muller glia (MG) are the principal glial cell of the vertebrate retina. The last cell to divide from a multipotent retinal progenitor cell, they maintain… (more)

Subjects/Keywords: Neurosciences; Developmental biology; School of Medicine; UNC Neuroscience Center; Neuroscience Curriculum

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APA (6th Edition):

Bachleda, A. (2014). SOX2 Is Essential for the Maturation and Maintenance of Retinal Müller Glia. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:953597a2-829f-4fd1-8be1-0e32f28f4a4d

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Bachleda, Amelia. “SOX2 Is Essential for the Maturation and Maintenance of Retinal Müller Glia.” 2014. Thesis, University of North Carolina. Accessed December 05, 2020. https://cdr.lib.unc.edu/record/uuid:953597a2-829f-4fd1-8be1-0e32f28f4a4d.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Bachleda, Amelia. “SOX2 Is Essential for the Maturation and Maintenance of Retinal Müller Glia.” 2014. Web. 05 Dec 2020.

Vancouver:

Bachleda A. SOX2 Is Essential for the Maturation and Maintenance of Retinal Müller Glia. [Internet] [Thesis]. University of North Carolina; 2014. [cited 2020 Dec 05]. Available from: https://cdr.lib.unc.edu/record/uuid:953597a2-829f-4fd1-8be1-0e32f28f4a4d.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Bachleda A. SOX2 Is Essential for the Maturation and Maintenance of Retinal Müller Glia. [Thesis]. University of North Carolina; 2014. Available from: https://cdr.lib.unc.edu/record/uuid:953597a2-829f-4fd1-8be1-0e32f28f4a4d

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

9. Bonder, Daniel. Astrocytic Gq-GPCR-Linked IP3R-Dependent Ca2+ Signaling Does Not Mediate Neurovascular Coupling in Mouse Visual Cortex in vivo.

Degree: 2014, University of North Carolina

 Local blood flow is modulated in response to changing patterns of neuronal activity (Roy and Sherrington, 1890), a process termed neurovascular coupling. It has been… (more)

Subjects/Keywords: Neurosciences; Cytology; School of Medicine; UNC Neuroscience Center; Neuroscience Curriculum

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APA (6th Edition):

Bonder, D. (2014). Astrocytic Gq-GPCR-Linked IP3R-Dependent Ca2+ Signaling Does Not Mediate Neurovascular Coupling in Mouse Visual Cortex in vivo. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:780de0e7-bf00-411b-88b7-4ca854b38cca

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Bonder, Daniel. “Astrocytic Gq-GPCR-Linked IP3R-Dependent Ca2+ Signaling Does Not Mediate Neurovascular Coupling in Mouse Visual Cortex in vivo.” 2014. Thesis, University of North Carolina. Accessed December 05, 2020. https://cdr.lib.unc.edu/record/uuid:780de0e7-bf00-411b-88b7-4ca854b38cca.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Bonder, Daniel. “Astrocytic Gq-GPCR-Linked IP3R-Dependent Ca2+ Signaling Does Not Mediate Neurovascular Coupling in Mouse Visual Cortex in vivo.” 2014. Web. 05 Dec 2020.

Vancouver:

Bonder D. Astrocytic Gq-GPCR-Linked IP3R-Dependent Ca2+ Signaling Does Not Mediate Neurovascular Coupling in Mouse Visual Cortex in vivo. [Internet] [Thesis]. University of North Carolina; 2014. [cited 2020 Dec 05]. Available from: https://cdr.lib.unc.edu/record/uuid:780de0e7-bf00-411b-88b7-4ca854b38cca.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Bonder D. Astrocytic Gq-GPCR-Linked IP3R-Dependent Ca2+ Signaling Does Not Mediate Neurovascular Coupling in Mouse Visual Cortex in vivo. [Thesis]. University of North Carolina; 2014. Available from: https://cdr.lib.unc.edu/record/uuid:780de0e7-bf00-411b-88b7-4ca854b38cca

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

10. Liang, Katharine. AAV-Nrf2 Mediated Rescue of Oxidative Stress in Mouse Models.

Degree: 2015, University of North Carolina

 Oxidative stress is implicated in many diseases in a variety of different organ systems. While specific antioxidant genes are known to contribute to the regulation… (more)

Subjects/Keywords: Ophthalmology; Neurosciences; Molecular biology; School of Medicine; UNC Neuroscience Center; Neuroscience Curriculum

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APA (6th Edition):

Liang, K. (2015). AAV-Nrf2 Mediated Rescue of Oxidative Stress in Mouse Models. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:224971e0-3a7d-43eb-be67-708d59babcfd

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Liang, Katharine. “AAV-Nrf2 Mediated Rescue of Oxidative Stress in Mouse Models.” 2015. Thesis, University of North Carolina. Accessed December 05, 2020. https://cdr.lib.unc.edu/record/uuid:224971e0-3a7d-43eb-be67-708d59babcfd.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Liang, Katharine. “AAV-Nrf2 Mediated Rescue of Oxidative Stress in Mouse Models.” 2015. Web. 05 Dec 2020.

Vancouver:

Liang K. AAV-Nrf2 Mediated Rescue of Oxidative Stress in Mouse Models. [Internet] [Thesis]. University of North Carolina; 2015. [cited 2020 Dec 05]. Available from: https://cdr.lib.unc.edu/record/uuid:224971e0-3a7d-43eb-be67-708d59babcfd.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Liang K. AAV-Nrf2 Mediated Rescue of Oxidative Stress in Mouse Models. [Thesis]. University of North Carolina; 2015. Available from: https://cdr.lib.unc.edu/record/uuid:224971e0-3a7d-43eb-be67-708d59babcfd

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

11. Sweger, Elizabeth Joy. Expression of the Gi-coupled RASSL Ro1 in GFAP-positive cells: a novel model of hydrocephalus.

Degree: Pharmacology, 2009, University of North Carolina

 Hydrocephalus is a disorder of cerebrospinal fluid dynamics within the central nervous system. Although symptoms of hydrocephalus are often relieved by shunt implantation, complications are… (more)

Subjects/Keywords: School of Medicine; Department of Pharmacology

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APA (6th Edition):

Sweger, E. J. (2009). Expression of the Gi-coupled RASSL Ro1 in GFAP-positive cells: a novel model of hydrocephalus. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:1c73a8ad-fed9-4c4d-bd90-43d69bb17594

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Sweger, Elizabeth Joy. “Expression of the Gi-coupled RASSL Ro1 in GFAP-positive cells: a novel model of hydrocephalus.” 2009. Thesis, University of North Carolina. Accessed December 05, 2020. https://cdr.lib.unc.edu/record/uuid:1c73a8ad-fed9-4c4d-bd90-43d69bb17594.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Sweger, Elizabeth Joy. “Expression of the Gi-coupled RASSL Ro1 in GFAP-positive cells: a novel model of hydrocephalus.” 2009. Web. 05 Dec 2020.

Vancouver:

Sweger EJ. Expression of the Gi-coupled RASSL Ro1 in GFAP-positive cells: a novel model of hydrocephalus. [Internet] [Thesis]. University of North Carolina; 2009. [cited 2020 Dec 05]. Available from: https://cdr.lib.unc.edu/record/uuid:1c73a8ad-fed9-4c4d-bd90-43d69bb17594.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Sweger EJ. Expression of the Gi-coupled RASSL Ro1 in GFAP-positive cells: a novel model of hydrocephalus. [Thesis]. University of North Carolina; 2009. Available from: https://cdr.lib.unc.edu/record/uuid:1c73a8ad-fed9-4c4d-bd90-43d69bb17594

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

12. Djukic, Biljana. Role of the inward rectifying potassium channel, Kir4.1, in astrocyte physiology and neuronal excitability.

Degree: Pharmacology, 2006, University of North Carolina

 During neuronal activity extracellular potassium concentration ([K+]out) becomes elevated and if uncorrected causes neuronal depolarization, hyperexcitability, and seizures. Clearance of K+ from the extracellular space,… (more)

Subjects/Keywords: School of Medicine; Department of Pharmacology

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APA (6th Edition):

Djukic, B. (2006). Role of the inward rectifying potassium channel, Kir4.1, in astrocyte physiology and neuronal excitability. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:99190e15-6088-4850-bc2b-184983fbe7a6

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Djukic, Biljana. “Role of the inward rectifying potassium channel, Kir4.1, in astrocyte physiology and neuronal excitability.” 2006. Thesis, University of North Carolina. Accessed December 05, 2020. https://cdr.lib.unc.edu/record/uuid:99190e15-6088-4850-bc2b-184983fbe7a6.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Djukic, Biljana. “Role of the inward rectifying potassium channel, Kir4.1, in astrocyte physiology and neuronal excitability.” 2006. Web. 05 Dec 2020.

Vancouver:

Djukic B. Role of the inward rectifying potassium channel, Kir4.1, in astrocyte physiology and neuronal excitability. [Internet] [Thesis]. University of North Carolina; 2006. [cited 2020 Dec 05]. Available from: https://cdr.lib.unc.edu/record/uuid:99190e15-6088-4850-bc2b-184983fbe7a6.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Djukic B. Role of the inward rectifying potassium channel, Kir4.1, in astrocyte physiology and neuronal excitability. [Thesis]. University of North Carolina; 2006. Available from: https://cdr.lib.unc.edu/record/uuid:99190e15-6088-4850-bc2b-184983fbe7a6

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

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