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You searched for +publisher:"University of North Carolina" +contributor:("Matsushima, Glenn"). Showing records 1 – 14 of 14 total matches.

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University of North Carolina

1. Rackoff, Lauren. Tissue Reservoirs of HIV-1: Insights from the Central Nervous System.

Degree: Microbiology and Immunology, 2016, University of North Carolina

 Cellular and anatomical reservoirs of HIV-1 preclude a cure to infection. Efforts to characterize these reservoirs are an important part of developing a strategy to… (more)

Subjects/Keywords: School of Medicine; Department of Microbiology and Immunology

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APA (6th Edition):

Rackoff, L. (2016). Tissue Reservoirs of HIV-1: Insights from the Central Nervous System. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:48631bb1-0b98-49b9-bbf1-d6f59831a65c

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Rackoff, Lauren. “Tissue Reservoirs of HIV-1: Insights from the Central Nervous System.” 2016. Thesis, University of North Carolina. Accessed October 30, 2020. https://cdr.lib.unc.edu/record/uuid:48631bb1-0b98-49b9-bbf1-d6f59831a65c.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Rackoff, Lauren. “Tissue Reservoirs of HIV-1: Insights from the Central Nervous System.” 2016. Web. 30 Oct 2020.

Vancouver:

Rackoff L. Tissue Reservoirs of HIV-1: Insights from the Central Nervous System. [Internet] [Thesis]. University of North Carolina; 2016. [cited 2020 Oct 30]. Available from: https://cdr.lib.unc.edu/record/uuid:48631bb1-0b98-49b9-bbf1-d6f59831a65c.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Rackoff L. Tissue Reservoirs of HIV-1: Insights from the Central Nervous System. [Thesis]. University of North Carolina; 2016. Available from: https://cdr.lib.unc.edu/record/uuid:48631bb1-0b98-49b9-bbf1-d6f59831a65c

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

2. Chen, Liang. The innate regulatory protein NLRP12 maintains commensal bacterial symbiosis, and mitigates intestinal inflammation and obesity.

Degree: Microbiology and Immunology, 2018, University of North Carolina

 The nucleotide-binding domain, leucine-rich repeat containing proteins, also known as NOD-like receptors (NLRs), are pattern recognition receptors that play important roles in innate immunity (Aganna… (more)

Subjects/Keywords: School of Medicine; Department of Microbiology and Immunology

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APA (6th Edition):

Chen, L. (2018). The innate regulatory protein NLRP12 maintains commensal bacterial symbiosis, and mitigates intestinal inflammation and obesity. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:e755bc43-2da6-4229-b7f8-c4584012348a

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Chen, Liang. “The innate regulatory protein NLRP12 maintains commensal bacterial symbiosis, and mitigates intestinal inflammation and obesity.” 2018. Thesis, University of North Carolina. Accessed October 30, 2020. https://cdr.lib.unc.edu/record/uuid:e755bc43-2da6-4229-b7f8-c4584012348a.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Chen, Liang. “The innate regulatory protein NLRP12 maintains commensal bacterial symbiosis, and mitigates intestinal inflammation and obesity.” 2018. Web. 30 Oct 2020.

Vancouver:

Chen L. The innate regulatory protein NLRP12 maintains commensal bacterial symbiosis, and mitigates intestinal inflammation and obesity. [Internet] [Thesis]. University of North Carolina; 2018. [cited 2020 Oct 30]. Available from: https://cdr.lib.unc.edu/record/uuid:e755bc43-2da6-4229-b7f8-c4584012348a.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Chen L. The innate regulatory protein NLRP12 maintains commensal bacterial symbiosis, and mitigates intestinal inflammation and obesity. [Thesis]. University of North Carolina; 2018. Available from: https://cdr.lib.unc.edu/record/uuid:e755bc43-2da6-4229-b7f8-c4584012348a

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

3. Allard Trout, Denise. Schwann cells promote spontaneous autoimmune peripheral polyneuropathy.

Degree: Microbiology and Immunology, 2018, University of North Carolina

 Chronic inflammatory demyelinating polyneuropathy (CIDP) and Guillain-Barré syndrome (GBS) are autoimmune demyelinating diseases of the peripheral nervous system (PNS). During CIDP and GBS, the immune… (more)

Subjects/Keywords: School of Medicine; Department of Microbiology and Immunology

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APA (6th Edition):

Allard Trout, D. (2018). Schwann cells promote spontaneous autoimmune peripheral polyneuropathy. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:d1ecd067-6919-437e-87d7-c588a8e81d6b

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Allard Trout, Denise. “Schwann cells promote spontaneous autoimmune peripheral polyneuropathy.” 2018. Thesis, University of North Carolina. Accessed October 30, 2020. https://cdr.lib.unc.edu/record/uuid:d1ecd067-6919-437e-87d7-c588a8e81d6b.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Allard Trout, Denise. “Schwann cells promote spontaneous autoimmune peripheral polyneuropathy.” 2018. Web. 30 Oct 2020.

Vancouver:

Allard Trout D. Schwann cells promote spontaneous autoimmune peripheral polyneuropathy. [Internet] [Thesis]. University of North Carolina; 2018. [cited 2020 Oct 30]. Available from: https://cdr.lib.unc.edu/record/uuid:d1ecd067-6919-437e-87d7-c588a8e81d6b.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Allard Trout D. Schwann cells promote spontaneous autoimmune peripheral polyneuropathy. [Thesis]. University of North Carolina; 2018. Available from: https://cdr.lib.unc.edu/record/uuid:d1ecd067-6919-437e-87d7-c588a8e81d6b

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

4. Keener, Amanda. REQUIREMENTS FOR MEMORY B CELL AND LONG LIVED PLASMA CELL DEVELOPMENT DURING ADAPTIVE IMMUNE RESPONSES AND STAPHYLOCOCCUS AUREUS INFECTION.

Degree: Microbiology and Immunology, 2014, University of North Carolina

 Memory B cells and plasma cells (PCs) are terminally differentiated B cells that contribute to recall responses vital for protection against infections. Formation of high… (more)

Subjects/Keywords: Immunology; Microbiology; School of Medicine; Department of Microbiology and Immunology

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APA (6th Edition):

Keener, A. (2014). REQUIREMENTS FOR MEMORY B CELL AND LONG LIVED PLASMA CELL DEVELOPMENT DURING ADAPTIVE IMMUNE RESPONSES AND STAPHYLOCOCCUS AUREUS INFECTION. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:579ce358-aff3-43d8-bf97-7c9371c51990

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Keener, Amanda. “REQUIREMENTS FOR MEMORY B CELL AND LONG LIVED PLASMA CELL DEVELOPMENT DURING ADAPTIVE IMMUNE RESPONSES AND STAPHYLOCOCCUS AUREUS INFECTION.” 2014. Thesis, University of North Carolina. Accessed October 30, 2020. https://cdr.lib.unc.edu/record/uuid:579ce358-aff3-43d8-bf97-7c9371c51990.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Keener, Amanda. “REQUIREMENTS FOR MEMORY B CELL AND LONG LIVED PLASMA CELL DEVELOPMENT DURING ADAPTIVE IMMUNE RESPONSES AND STAPHYLOCOCCUS AUREUS INFECTION.” 2014. Web. 30 Oct 2020.

Vancouver:

Keener A. REQUIREMENTS FOR MEMORY B CELL AND LONG LIVED PLASMA CELL DEVELOPMENT DURING ADAPTIVE IMMUNE RESPONSES AND STAPHYLOCOCCUS AUREUS INFECTION. [Internet] [Thesis]. University of North Carolina; 2014. [cited 2020 Oct 30]. Available from: https://cdr.lib.unc.edu/record/uuid:579ce358-aff3-43d8-bf97-7c9371c51990.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Keener A. REQUIREMENTS FOR MEMORY B CELL AND LONG LIVED PLASMA CELL DEVELOPMENT DURING ADAPTIVE IMMUNE RESPONSES AND STAPHYLOCOCCUS AUREUS INFECTION. [Thesis]. University of North Carolina; 2014. Available from: https://cdr.lib.unc.edu/record/uuid:579ce358-aff3-43d8-bf97-7c9371c51990

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

5. Morillon, Yves. THYMIC DEVELOPMENT OF AUTOREACTIVE T CELLS IN NOD MICE, AND THE ALTERED TRAFFICKING OF T CELLS FOLLOWING ANTIBODY MEDIATED CROSSLINKING OF THE CD4 CORECEPTOR.

Degree: Microbiology and Immunology, 2014, University of North Carolina

 Two key processes in driving type 1 diabetes are the development and trafficking of β cell-specific T cells into the pancreas. The first part of… (more)

Subjects/Keywords: Immunology; School of Medicine; Department of Microbiology and Immunology

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APA (6th Edition):

Morillon, Y. (2014). THYMIC DEVELOPMENT OF AUTOREACTIVE T CELLS IN NOD MICE, AND THE ALTERED TRAFFICKING OF T CELLS FOLLOWING ANTIBODY MEDIATED CROSSLINKING OF THE CD4 CORECEPTOR. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:6779d3c5-3461-4486-aef2-33ac9de0cd3c

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Morillon, Yves. “THYMIC DEVELOPMENT OF AUTOREACTIVE T CELLS IN NOD MICE, AND THE ALTERED TRAFFICKING OF T CELLS FOLLOWING ANTIBODY MEDIATED CROSSLINKING OF THE CD4 CORECEPTOR.” 2014. Thesis, University of North Carolina. Accessed October 30, 2020. https://cdr.lib.unc.edu/record/uuid:6779d3c5-3461-4486-aef2-33ac9de0cd3c.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Morillon, Yves. “THYMIC DEVELOPMENT OF AUTOREACTIVE T CELLS IN NOD MICE, AND THE ALTERED TRAFFICKING OF T CELLS FOLLOWING ANTIBODY MEDIATED CROSSLINKING OF THE CD4 CORECEPTOR.” 2014. Web. 30 Oct 2020.

Vancouver:

Morillon Y. THYMIC DEVELOPMENT OF AUTOREACTIVE T CELLS IN NOD MICE, AND THE ALTERED TRAFFICKING OF T CELLS FOLLOWING ANTIBODY MEDIATED CROSSLINKING OF THE CD4 CORECEPTOR. [Internet] [Thesis]. University of North Carolina; 2014. [cited 2020 Oct 30]. Available from: https://cdr.lib.unc.edu/record/uuid:6779d3c5-3461-4486-aef2-33ac9de0cd3c.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Morillon Y. THYMIC DEVELOPMENT OF AUTOREACTIVE T CELLS IN NOD MICE, AND THE ALTERED TRAFFICKING OF T CELLS FOLLOWING ANTIBODY MEDIATED CROSSLINKING OF THE CD4 CORECEPTOR. [Thesis]. University of North Carolina; 2014. Available from: https://cdr.lib.unc.edu/record/uuid:6779d3c5-3461-4486-aef2-33ac9de0cd3c

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

6. Manzoor, Fatima. Tissue-specific cytokine-based immunotherapy to treat type 1 diabetes.

Degree: Microbiology and Immunology, 2016, University of North Carolina

 Type 1 diabetes (T1D) is characterized by the T cell-mediated destruction of the insulin-producing β cells. Progressive loss of β cell mass results in the… (more)

Subjects/Keywords: School of Medicine; Department of Microbiology and Immunology

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APA (6th Edition):

Manzoor, F. (2016). Tissue-specific cytokine-based immunotherapy to treat type 1 diabetes. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:3d21e01f-87c6-48ae-bbe7-f677faa3f639

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Manzoor, Fatima. “Tissue-specific cytokine-based immunotherapy to treat type 1 diabetes.” 2016. Thesis, University of North Carolina. Accessed October 30, 2020. https://cdr.lib.unc.edu/record/uuid:3d21e01f-87c6-48ae-bbe7-f677faa3f639.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Manzoor, Fatima. “Tissue-specific cytokine-based immunotherapy to treat type 1 diabetes.” 2016. Web. 30 Oct 2020.

Vancouver:

Manzoor F. Tissue-specific cytokine-based immunotherapy to treat type 1 diabetes. [Internet] [Thesis]. University of North Carolina; 2016. [cited 2020 Oct 30]. Available from: https://cdr.lib.unc.edu/record/uuid:3d21e01f-87c6-48ae-bbe7-f677faa3f639.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Manzoor F. Tissue-specific cytokine-based immunotherapy to treat type 1 diabetes. [Thesis]. University of North Carolina; 2016. Available from: https://cdr.lib.unc.edu/record/uuid:3d21e01f-87c6-48ae-bbe7-f677faa3f639

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

7. Spidale, Nicholas. Factors regulating thymic dendritic cell homeostasis and function.

Degree: Microbiology and Immunology, 2014, University of North Carolina

 The immune system must balance the generation of a diverse T cell repertoire responsive to various pathogens versus the prevention of tissue destruction by self-antigen-specific… (more)

Subjects/Keywords: Immunology; School of Medicine; Department of Microbiology and Immunology

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APA (6th Edition):

Spidale, N. (2014). Factors regulating thymic dendritic cell homeostasis and function. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:821c13c1-ff67-4848-a3c3-e506874313c9

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Spidale, Nicholas. “Factors regulating thymic dendritic cell homeostasis and function.” 2014. Thesis, University of North Carolina. Accessed October 30, 2020. https://cdr.lib.unc.edu/record/uuid:821c13c1-ff67-4848-a3c3-e506874313c9.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Spidale, Nicholas. “Factors regulating thymic dendritic cell homeostasis and function.” 2014. Web. 30 Oct 2020.

Vancouver:

Spidale N. Factors regulating thymic dendritic cell homeostasis and function. [Internet] [Thesis]. University of North Carolina; 2014. [cited 2020 Oct 30]. Available from: https://cdr.lib.unc.edu/record/uuid:821c13c1-ff67-4848-a3c3-e506874313c9.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Spidale N. Factors regulating thymic dendritic cell homeostasis and function. [Thesis]. University of North Carolina; 2014. Available from: https://cdr.lib.unc.edu/record/uuid:821c13c1-ff67-4848-a3c3-e506874313c9

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

8. Callaway, Justin. Interfering With Inflammatory Cytokine Production During Antibody-Enhanced Dengue Virus Infection.

Degree: Microbiology and Immunology, 2014, University of North Carolina

 Dengue virus (DENV) is the most burdensome arbovirus in the world, infecting nearly 400 million people annually. Patients develop lifelong immunity to the original infecting… (more)

Subjects/Keywords: Immunology; Microbiology; Virology; School of Medicine; Department of Microbiology and Immunology

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APA (6th Edition):

Callaway, J. (2014). Interfering With Inflammatory Cytokine Production During Antibody-Enhanced Dengue Virus Infection. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:fe6f5654-fb88-4343-a1c0-6a93a65e7145

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Callaway, Justin. “Interfering With Inflammatory Cytokine Production During Antibody-Enhanced Dengue Virus Infection.” 2014. Thesis, University of North Carolina. Accessed October 30, 2020. https://cdr.lib.unc.edu/record/uuid:fe6f5654-fb88-4343-a1c0-6a93a65e7145.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Callaway, Justin. “Interfering With Inflammatory Cytokine Production During Antibody-Enhanced Dengue Virus Infection.” 2014. Web. 30 Oct 2020.

Vancouver:

Callaway J. Interfering With Inflammatory Cytokine Production During Antibody-Enhanced Dengue Virus Infection. [Internet] [Thesis]. University of North Carolina; 2014. [cited 2020 Oct 30]. Available from: https://cdr.lib.unc.edu/record/uuid:fe6f5654-fb88-4343-a1c0-6a93a65e7145.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Callaway J. Interfering With Inflammatory Cytokine Production During Antibody-Enhanced Dengue Virus Infection. [Thesis]. University of North Carolina; 2014. Available from: https://cdr.lib.unc.edu/record/uuid:fe6f5654-fb88-4343-a1c0-6a93a65e7145

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

9. Freeman, Leslie. A Role for the NLR Family Members NLRC4 And NLRP3 in Astrocytic Inflammasome Activation and Astrogliosis.

Degree: 2016, University of North Carolina

 The inflammasome is implicated in many inflammatory diseases but has been primarily studied in the macrophage-myeloid lineage. Here we demonstrate a physiologic role for nucleotide-binding… (more)

Subjects/Keywords: School of Medicine; Curriculum in Genetics and Molecular Biology

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APA (6th Edition):

Freeman, L. (2016). A Role for the NLR Family Members NLRC4 And NLRP3 in Astrocytic Inflammasome Activation and Astrogliosis. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:ec028a13-7c5a-41ed-af8d-3b5a5865278d

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Freeman, Leslie. “A Role for the NLR Family Members NLRC4 And NLRP3 in Astrocytic Inflammasome Activation and Astrogliosis.” 2016. Thesis, University of North Carolina. Accessed October 30, 2020. https://cdr.lib.unc.edu/record/uuid:ec028a13-7c5a-41ed-af8d-3b5a5865278d.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Freeman, Leslie. “A Role for the NLR Family Members NLRC4 And NLRP3 in Astrocytic Inflammasome Activation and Astrogliosis.” 2016. Web. 30 Oct 2020.

Vancouver:

Freeman L. A Role for the NLR Family Members NLRC4 And NLRP3 in Astrocytic Inflammasome Activation and Astrogliosis. [Internet] [Thesis]. University of North Carolina; 2016. [cited 2020 Oct 30]. Available from: https://cdr.lib.unc.edu/record/uuid:ec028a13-7c5a-41ed-af8d-3b5a5865278d.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Freeman L. A Role for the NLR Family Members NLRC4 And NLRP3 in Astrocytic Inflammasome Activation and Astrogliosis. [Thesis]. University of North Carolina; 2016. Available from: https://cdr.lib.unc.edu/record/uuid:ec028a13-7c5a-41ed-af8d-3b5a5865278d

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

10. Taylor, Lorelei Carissa. Sex differences and the protective influence of estrogen in a mouse model of demyelinating disease.

Degree: 2008, University of North Carolina

 Demyelinating diseases such as multiple sclerosis may be influenced by gender dimorphisms and sex hormones. Here, the cuprizone model of toxin-induced oligodendrocyte death and demyelination… (more)

Subjects/Keywords: School of Medicine; UNC Neuroscience Center; Neuroscience Curriculum

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APA (6th Edition):

Taylor, L. C. (2008). Sex differences and the protective influence of estrogen in a mouse model of demyelinating disease. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:9cc84576-e561-4ff4-b0eb-5c28893f3e0a

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Taylor, Lorelei Carissa. “Sex differences and the protective influence of estrogen in a mouse model of demyelinating disease.” 2008. Thesis, University of North Carolina. Accessed October 30, 2020. https://cdr.lib.unc.edu/record/uuid:9cc84576-e561-4ff4-b0eb-5c28893f3e0a.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Taylor, Lorelei Carissa. “Sex differences and the protective influence of estrogen in a mouse model of demyelinating disease.” 2008. Web. 30 Oct 2020.

Vancouver:

Taylor LC. Sex differences and the protective influence of estrogen in a mouse model of demyelinating disease. [Internet] [Thesis]. University of North Carolina; 2008. [cited 2020 Oct 30]. Available from: https://cdr.lib.unc.edu/record/uuid:9cc84576-e561-4ff4-b0eb-5c28893f3e0a.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Taylor LC. Sex differences and the protective influence of estrogen in a mouse model of demyelinating disease. [Thesis]. University of North Carolina; 2008. Available from: https://cdr.lib.unc.edu/record/uuid:9cc84576-e561-4ff4-b0eb-5c28893f3e0a

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

11. Chen, Vivian Susan. Immune mediators of central nervous system demyelination and remyelination.

Degree: Microbiology and Immunology, 2008, University of North Carolina

 The principal goal of an immune response is to protect the host from pathogens, maintain tissue homeostasis and/or to repair damaged tissue. However, an inappropriate… (more)

Subjects/Keywords: School of Medicine; Department of Microbiology and Immunology

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APA (6th Edition):

Chen, V. S. (2008). Immune mediators of central nervous system demyelination and remyelination. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:c751db89-2ae7-4225-88e0-c62eba36f255

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Chen, Vivian Susan. “Immune mediators of central nervous system demyelination and remyelination.” 2008. Thesis, University of North Carolina. Accessed October 30, 2020. https://cdr.lib.unc.edu/record/uuid:c751db89-2ae7-4225-88e0-c62eba36f255.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Chen, Vivian Susan. “Immune mediators of central nervous system demyelination and remyelination.” 2008. Web. 30 Oct 2020.

Vancouver:

Chen VS. Immune mediators of central nervous system demyelination and remyelination. [Internet] [Thesis]. University of North Carolina; 2008. [cited 2020 Oct 30]. Available from: https://cdr.lib.unc.edu/record/uuid:c751db89-2ae7-4225-88e0-c62eba36f255.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Chen VS. Immune mediators of central nervous system demyelination and remyelination. [Thesis]. University of North Carolina; 2008. Available from: https://cdr.lib.unc.edu/record/uuid:c751db89-2ae7-4225-88e0-c62eba36f255

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

12. Williams, Julie Clarke. Regulation of innate immune responses by Mertk.

Degree: 2009, University of North Carolina

 The Tyro3/Axl/Mertk(TAM) family of receptors is important for phagocytosis of apoptotic cells, cytokine regulation, and suppression of autoimmune disease. Mice lacking Mertk present with a… (more)

Subjects/Keywords: School of Dentistry; Oral and Craniofacial Biomedicine PhD Program

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Williams, J. C. (2009). Regulation of innate immune responses by Mertk. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:09a15339-61ed-486b-96cc-0e5f960ed718

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Williams, Julie Clarke. “Regulation of innate immune responses by Mertk.” 2009. Thesis, University of North Carolina. Accessed October 30, 2020. https://cdr.lib.unc.edu/record/uuid:09a15339-61ed-486b-96cc-0e5f960ed718.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Williams, Julie Clarke. “Regulation of innate immune responses by Mertk.” 2009. Web. 30 Oct 2020.

Vancouver:

Williams JC. Regulation of innate immune responses by Mertk. [Internet] [Thesis]. University of North Carolina; 2009. [cited 2020 Oct 30]. Available from: https://cdr.lib.unc.edu/record/uuid:09a15339-61ed-486b-96cc-0e5f960ed718.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Williams JC. Regulation of innate immune responses by Mertk. [Thesis]. University of North Carolina; 2009. Available from: https://cdr.lib.unc.edu/record/uuid:09a15339-61ed-486b-96cc-0e5f960ed718

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

13. Gohlke, Paul Reid. The role of MerTK and BAFF in dendritic cell-B cell interactions.

Degree: Microbiology and Immunology, 2007, University of North Carolina

 Autoimmune disease occurs when the system of cells and molecules designed to protect the host from invading pathogens directs itself towards attacking the host. The… (more)

Subjects/Keywords: School of Medicine; Department of Microbiology and Immunology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Gohlke, P. R. (2007). The role of MerTK and BAFF in dendritic cell-B cell interactions. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:7948886b-0247-4aca-8b06-7ef2548938c8

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Gohlke, Paul Reid. “The role of MerTK and BAFF in dendritic cell-B cell interactions.” 2007. Thesis, University of North Carolina. Accessed October 30, 2020. https://cdr.lib.unc.edu/record/uuid:7948886b-0247-4aca-8b06-7ef2548938c8.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Gohlke, Paul Reid. “The role of MerTK and BAFF in dendritic cell-B cell interactions.” 2007. Web. 30 Oct 2020.

Vancouver:

Gohlke PR. The role of MerTK and BAFF in dendritic cell-B cell interactions. [Internet] [Thesis]. University of North Carolina; 2007. [cited 2020 Oct 30]. Available from: https://cdr.lib.unc.edu/record/uuid:7948886b-0247-4aca-8b06-7ef2548938c8.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Gohlke PR. The role of MerTK and BAFF in dendritic cell-B cell interactions. [Thesis]. University of North Carolina; 2007. Available from: https://cdr.lib.unc.edu/record/uuid:7948886b-0247-4aca-8b06-7ef2548938c8

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

14. Seitz, Heather M. The role of Axl/Mertk/Tyro3 receptor tyrosine kinases in the clearance of apoptotic cells.

Degree: Microbiology and Immunology, 2006, University of North Carolina

 The phagocytosis of apoptotic cells is critical for maintaining normal development, tissue homeostasis, and lymphocyte maturation. While this phenomenon is well documented, a limited number… (more)

Subjects/Keywords: School of Medicine; Department of Microbiology and Immunology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Seitz, H. M. (2006). The role of Axl/Mertk/Tyro3 receptor tyrosine kinases in the clearance of apoptotic cells. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:d5beeac3-041d-43f0-8000-0389ec083f67

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Seitz, Heather M. “The role of Axl/Mertk/Tyro3 receptor tyrosine kinases in the clearance of apoptotic cells.” 2006. Thesis, University of North Carolina. Accessed October 30, 2020. https://cdr.lib.unc.edu/record/uuid:d5beeac3-041d-43f0-8000-0389ec083f67.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Seitz, Heather M. “The role of Axl/Mertk/Tyro3 receptor tyrosine kinases in the clearance of apoptotic cells.” 2006. Web. 30 Oct 2020.

Vancouver:

Seitz HM. The role of Axl/Mertk/Tyro3 receptor tyrosine kinases in the clearance of apoptotic cells. [Internet] [Thesis]. University of North Carolina; 2006. [cited 2020 Oct 30]. Available from: https://cdr.lib.unc.edu/record/uuid:d5beeac3-041d-43f0-8000-0389ec083f67.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Seitz HM. The role of Axl/Mertk/Tyro3 receptor tyrosine kinases in the clearance of apoptotic cells. [Thesis]. University of North Carolina; 2006. Available from: https://cdr.lib.unc.edu/record/uuid:d5beeac3-041d-43f0-8000-0389ec083f67

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

.