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You searched for +publisher:"University of North Carolina" +contributor:("Lund, Pauline Kay"). Showing records 1 – 15 of 15 total matches.

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University of North Carolina

1. Scull, Brooks. Intestinal regeneration after irradiation: stem cells and Sox9.

Degree: Cell Biology and Physiology, 2009, University of North Carolina

 Whole body irradiation is a useful challenge to study the response of intestinal epithelial stem cells (IESC) to genetic damage. High dose irradiation causes complete… (more)

Subjects/Keywords: School of Medicine; Department of Cell Biology and Physiology

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APA (6th Edition):

Scull, B. (2009). Intestinal regeneration after irradiation: stem cells and Sox9. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:1d77bd9a-550c-477f-b337-ebdd6f1171ab

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Scull, Brooks. “Intestinal regeneration after irradiation: stem cells and Sox9.” 2009. Thesis, University of North Carolina. Accessed October 30, 2020. https://cdr.lib.unc.edu/record/uuid:1d77bd9a-550c-477f-b337-ebdd6f1171ab.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Scull, Brooks. “Intestinal regeneration after irradiation: stem cells and Sox9.” 2009. Web. 30 Oct 2020.

Vancouver:

Scull B. Intestinal regeneration after irradiation: stem cells and Sox9. [Internet] [Thesis]. University of North Carolina; 2009. [cited 2020 Oct 30]. Available from: https://cdr.lib.unc.edu/record/uuid:1d77bd9a-550c-477f-b337-ebdd6f1171ab.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Scull B. Intestinal regeneration after irradiation: stem cells and Sox9. [Thesis]. University of North Carolina; 2009. Available from: https://cdr.lib.unc.edu/record/uuid:1d77bd9a-550c-477f-b337-ebdd6f1171ab

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

2. Ramocki, Nicole Marie. IRS-1 and SOCS2 are key regulators of insulin-like growth factor signaling in intestinal cancer.

Degree: Cell Biology and Physiology, 2008, University of North Carolina

 The growth hormone/insulin-like growth factor (GH/IGF) axis has been implicated at various stages of tumor progression in colon cancer. Insulin receptor substrate-1 (IRS-1) is a… (more)

Subjects/Keywords: College of Arts and Sciences; School of Medicine; Department of Cell Biology and Physiology

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APA (6th Edition):

Ramocki, N. M. (2008). IRS-1 and SOCS2 are key regulators of insulin-like growth factor signaling in intestinal cancer. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:139568b1-6912-455a-8465-39f2aa05464c

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ramocki, Nicole Marie. “IRS-1 and SOCS2 are key regulators of insulin-like growth factor signaling in intestinal cancer.” 2008. Thesis, University of North Carolina. Accessed October 30, 2020. https://cdr.lib.unc.edu/record/uuid:139568b1-6912-455a-8465-39f2aa05464c.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ramocki, Nicole Marie. “IRS-1 and SOCS2 are key regulators of insulin-like growth factor signaling in intestinal cancer.” 2008. Web. 30 Oct 2020.

Vancouver:

Ramocki NM. IRS-1 and SOCS2 are key regulators of insulin-like growth factor signaling in intestinal cancer. [Internet] [Thesis]. University of North Carolina; 2008. [cited 2020 Oct 30]. Available from: https://cdr.lib.unc.edu/record/uuid:139568b1-6912-455a-8465-39f2aa05464c.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ramocki NM. IRS-1 and SOCS2 are key regulators of insulin-like growth factor signaling in intestinal cancer. [Thesis]. University of North Carolina; 2008. Available from: https://cdr.lib.unc.edu/record/uuid:139568b1-6912-455a-8465-39f2aa05464c

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

3. Hamilton, Kathryn Elizabeth. Role of suppressor of cytokine signaling 3 in colorectal cancer.

Degree: Cell Biology and Physiology, 2010, University of North Carolina

 Patients with inflammatory bowel diseases (IBD) have an increased lifetime risk of developing colorectal cancer (CRC). Suppressors of cytokine signaling (SOCS) are intracellular proteins that… (more)

Subjects/Keywords: School of Medicine; Department of Cell Biology and Physiology

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APA (6th Edition):

Hamilton, K. E. (2010). Role of suppressor of cytokine signaling 3 in colorectal cancer. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:095fdc60-260b-46ee-81c2-299f00899349

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hamilton, Kathryn Elizabeth. “Role of suppressor of cytokine signaling 3 in colorectal cancer.” 2010. Thesis, University of North Carolina. Accessed October 30, 2020. https://cdr.lib.unc.edu/record/uuid:095fdc60-260b-46ee-81c2-299f00899349.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hamilton, Kathryn Elizabeth. “Role of suppressor of cytokine signaling 3 in colorectal cancer.” 2010. Web. 30 Oct 2020.

Vancouver:

Hamilton KE. Role of suppressor of cytokine signaling 3 in colorectal cancer. [Internet] [Thesis]. University of North Carolina; 2010. [cited 2020 Oct 30]. Available from: https://cdr.lib.unc.edu/record/uuid:095fdc60-260b-46ee-81c2-299f00899349.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hamilton KE. Role of suppressor of cytokine signaling 3 in colorectal cancer. [Thesis]. University of North Carolina; 2010. Available from: https://cdr.lib.unc.edu/record/uuid:095fdc60-260b-46ee-81c2-299f00899349

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

4. Newton, Victoria Ann. Suppressor of cytokine signaling-2 (SOCS2) and epidermal growth factor (EGF) modulate insulin-like growth factor-I receptor (IGF-IR) signaling in intestinal cancer.

Degree: Cell Biology and Physiology, 2010, University of North Carolina

 The insulin-like growth factor (IGF-I) pathway is associated with increased risk and progression of colorectal cancer. Understanding mechanisms that inhibit or promote this key pathway… (more)

Subjects/Keywords: School of Medicine; Department of Cell Biology and Physiology

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APA (6th Edition):

Newton, V. A. (2010). Suppressor of cytokine signaling-2 (SOCS2) and epidermal growth factor (EGF) modulate insulin-like growth factor-I receptor (IGF-IR) signaling in intestinal cancer. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:6fc8c0f8-378d-46a4-b570-a706ccfd67ef

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Newton, Victoria Ann. “Suppressor of cytokine signaling-2 (SOCS2) and epidermal growth factor (EGF) modulate insulin-like growth factor-I receptor (IGF-IR) signaling in intestinal cancer.” 2010. Thesis, University of North Carolina. Accessed October 30, 2020. https://cdr.lib.unc.edu/record/uuid:6fc8c0f8-378d-46a4-b570-a706ccfd67ef.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Newton, Victoria Ann. “Suppressor of cytokine signaling-2 (SOCS2) and epidermal growth factor (EGF) modulate insulin-like growth factor-I receptor (IGF-IR) signaling in intestinal cancer.” 2010. Web. 30 Oct 2020.

Vancouver:

Newton VA. Suppressor of cytokine signaling-2 (SOCS2) and epidermal growth factor (EGF) modulate insulin-like growth factor-I receptor (IGF-IR) signaling in intestinal cancer. [Internet] [Thesis]. University of North Carolina; 2010. [cited 2020 Oct 30]. Available from: https://cdr.lib.unc.edu/record/uuid:6fc8c0f8-378d-46a4-b570-a706ccfd67ef.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Newton VA. Suppressor of cytokine signaling-2 (SOCS2) and epidermal growth factor (EGF) modulate insulin-like growth factor-I receptor (IGF-IR) signaling in intestinal cancer. [Thesis]. University of North Carolina; 2010. Available from: https://cdr.lib.unc.edu/record/uuid:6fc8c0f8-378d-46a4-b570-a706ccfd67ef

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

5. Gracz, Adam David. The role of Sry-Box (Sox) transcription factors in epithelial stem cell biology of the gastrointestinal tract.

Degree: Cell Biology and Physiology, 2011, University of North Carolina

 Stem cell biology, though a well-established concept in the scientific zeitgeist, is only beginning to emerge as an independent field of study. An understanding of… (more)

Subjects/Keywords: School of Medicine; Department of Cell Biology and Physiology

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APA (6th Edition):

Gracz, A. D. (2011). The role of Sry-Box (Sox) transcription factors in epithelial stem cell biology of the gastrointestinal tract. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:b263fe64-d6f7-4056-90b7-51d43225a651

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Gracz, Adam David. “The role of Sry-Box (Sox) transcription factors in epithelial stem cell biology of the gastrointestinal tract.” 2011. Thesis, University of North Carolina. Accessed October 30, 2020. https://cdr.lib.unc.edu/record/uuid:b263fe64-d6f7-4056-90b7-51d43225a651.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Gracz, Adam David. “The role of Sry-Box (Sox) transcription factors in epithelial stem cell biology of the gastrointestinal tract.” 2011. Web. 30 Oct 2020.

Vancouver:

Gracz AD. The role of Sry-Box (Sox) transcription factors in epithelial stem cell biology of the gastrointestinal tract. [Internet] [Thesis]. University of North Carolina; 2011. [cited 2020 Oct 30]. Available from: https://cdr.lib.unc.edu/record/uuid:b263fe64-d6f7-4056-90b7-51d43225a651.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Gracz AD. The role of Sry-Box (Sox) transcription factors in epithelial stem cell biology of the gastrointestinal tract. [Thesis]. University of North Carolina; 2011. Available from: https://cdr.lib.unc.edu/record/uuid:b263fe64-d6f7-4056-90b7-51d43225a651

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

6. Wetzel-Strong, Sarah. Effect of adrenomedullin over-expression in the cardiovascular system during development and disease.

Degree: Cell Biology and Physiology, 2015, University of North Carolina

 Since the discovery of adrenomedullin (Adm - gene; AM - protein) in 1993, many roles for this widely expressed peptide have been described in the… (more)

Subjects/Keywords: Physiology; School of Medicine; Department of Cell Biology and Physiology

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APA (6th Edition):

Wetzel-Strong, S. (2015). Effect of adrenomedullin over-expression in the cardiovascular system during development and disease. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:a0e5c581-09be-45c2-9181-f3dd435e726d

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Wetzel-Strong, Sarah. “Effect of adrenomedullin over-expression in the cardiovascular system during development and disease.” 2015. Thesis, University of North Carolina. Accessed October 30, 2020. https://cdr.lib.unc.edu/record/uuid:a0e5c581-09be-45c2-9181-f3dd435e726d.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Wetzel-Strong, Sarah. “Effect of adrenomedullin over-expression in the cardiovascular system during development and disease.” 2015. Web. 30 Oct 2020.

Vancouver:

Wetzel-Strong S. Effect of adrenomedullin over-expression in the cardiovascular system during development and disease. [Internet] [Thesis]. University of North Carolina; 2015. [cited 2020 Oct 30]. Available from: https://cdr.lib.unc.edu/record/uuid:a0e5c581-09be-45c2-9181-f3dd435e726d.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Wetzel-Strong S. Effect of adrenomedullin over-expression in the cardiovascular system during development and disease. [Thesis]. University of North Carolina; 2015. Available from: https://cdr.lib.unc.edu/record/uuid:a0e5c581-09be-45c2-9181-f3dd435e726d

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

7. Santoro, Maria. OBESITY, HYPERINSULINEMIA, THE INSULIN AND INSULIN-LIKE GROWTH FACTOR 1 RECEPTORS, AND RISK OF COLORECTAL CANCER.

Degree: Cell Biology and Physiology, 2015, University of North Carolina

 Insulin resistance and hyperinsulinemia associated with obesity or type 2 diabetes are strongly associated with increased risk of colorectal cancer (CRC). Elevated plasma insulin can… (more)

Subjects/Keywords: Cytology; Molecular biology; Oncology; School of Medicine; Department of Cell Biology and Physiology

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APA (6th Edition):

Santoro, M. (2015). OBESITY, HYPERINSULINEMIA, THE INSULIN AND INSULIN-LIKE GROWTH FACTOR 1 RECEPTORS, AND RISK OF COLORECTAL CANCER. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:94254ea7-0b9e-46e1-befd-07c9936b703a

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Santoro, Maria. “OBESITY, HYPERINSULINEMIA, THE INSULIN AND INSULIN-LIKE GROWTH FACTOR 1 RECEPTORS, AND RISK OF COLORECTAL CANCER.” 2015. Thesis, University of North Carolina. Accessed October 30, 2020. https://cdr.lib.unc.edu/record/uuid:94254ea7-0b9e-46e1-befd-07c9936b703a.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Santoro, Maria. “OBESITY, HYPERINSULINEMIA, THE INSULIN AND INSULIN-LIKE GROWTH FACTOR 1 RECEPTORS, AND RISK OF COLORECTAL CANCER.” 2015. Web. 30 Oct 2020.

Vancouver:

Santoro M. OBESITY, HYPERINSULINEMIA, THE INSULIN AND INSULIN-LIKE GROWTH FACTOR 1 RECEPTORS, AND RISK OF COLORECTAL CANCER. [Internet] [Thesis]. University of North Carolina; 2015. [cited 2020 Oct 30]. Available from: https://cdr.lib.unc.edu/record/uuid:94254ea7-0b9e-46e1-befd-07c9936b703a.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Santoro M. OBESITY, HYPERINSULINEMIA, THE INSULIN AND INSULIN-LIKE GROWTH FACTOR 1 RECEPTORS, AND RISK OF COLORECTAL CANCER. [Thesis]. University of North Carolina; 2015. Available from: https://cdr.lib.unc.edu/record/uuid:94254ea7-0b9e-46e1-befd-07c9936b703a

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

8. Mah, Amanda. Functional Impact of Obesity or Intermittent Feeding on Intestinal Stem Cells.

Degree: Nutrition, 2015, University of North Carolina

 Intestinal stem cells (ISCs) and progenitors constantly renew the intestinal epithelium. Effects of obesity or intermittent feeding specifically on ISCs versus progenitors are not defined.… (more)

Subjects/Keywords: Nutrition; Gillings School of Global Public Health; Department of Nutrition

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APA (6th Edition):

Mah, A. (2015). Functional Impact of Obesity or Intermittent Feeding on Intestinal Stem Cells. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:5700022a-2ec5-45e5-93aa-ed0300a0342f

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Mah, Amanda. “Functional Impact of Obesity or Intermittent Feeding on Intestinal Stem Cells.” 2015. Thesis, University of North Carolina. Accessed October 30, 2020. https://cdr.lib.unc.edu/record/uuid:5700022a-2ec5-45e5-93aa-ed0300a0342f.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Mah, Amanda. “Functional Impact of Obesity or Intermittent Feeding on Intestinal Stem Cells.” 2015. Web. 30 Oct 2020.

Vancouver:

Mah A. Functional Impact of Obesity or Intermittent Feeding on Intestinal Stem Cells. [Internet] [Thesis]. University of North Carolina; 2015. [cited 2020 Oct 30]. Available from: https://cdr.lib.unc.edu/record/uuid:5700022a-2ec5-45e5-93aa-ed0300a0342f.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Mah A. Functional Impact of Obesity or Intermittent Feeding on Intestinal Stem Cells. [Thesis]. University of North Carolina; 2015. Available from: https://cdr.lib.unc.edu/record/uuid:5700022a-2ec5-45e5-93aa-ed0300a0342f

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

9. Cooper, Daniel. Physiological Consequences of Compartmentalized Glycerolipid Synthesis.

Degree: Nutrition, 2015, University of North Carolina

 The metabolic derangements present in type 2 diabetes are associated with the accumulation of triacylglycerol (TAG) in adipose and non-adipose tissues. However, the mechanisms whereby… (more)

Subjects/Keywords: Biochemistry; Nutrition; Gillings School of Global Public Health; Department of Nutrition

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APA (6th Edition):

Cooper, D. (2015). Physiological Consequences of Compartmentalized Glycerolipid Synthesis. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:cdb988ba-d01d-4e5c-8693-fedec95fa5c1

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Cooper, Daniel. “Physiological Consequences of Compartmentalized Glycerolipid Synthesis.” 2015. Thesis, University of North Carolina. Accessed October 30, 2020. https://cdr.lib.unc.edu/record/uuid:cdb988ba-d01d-4e5c-8693-fedec95fa5c1.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Cooper, Daniel. “Physiological Consequences of Compartmentalized Glycerolipid Synthesis.” 2015. Web. 30 Oct 2020.

Vancouver:

Cooper D. Physiological Consequences of Compartmentalized Glycerolipid Synthesis. [Internet] [Thesis]. University of North Carolina; 2015. [cited 2020 Oct 30]. Available from: https://cdr.lib.unc.edu/record/uuid:cdb988ba-d01d-4e5c-8693-fedec95fa5c1.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Cooper D. Physiological Consequences of Compartmentalized Glycerolipid Synthesis. [Thesis]. University of North Carolina; 2015. Available from: https://cdr.lib.unc.edu/record/uuid:cdb988ba-d01d-4e5c-8693-fedec95fa5c1

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

10. Rollins, Brett. Regulation of the Epithelial Sodium Channel (ENaC) by the Short Palate, Lung, and Nasal Epithelial Clone (SPLUNC1).

Degree: Cell Biology and Physiology, 2010, University of North Carolina

 The airways rely on mucociliary clearance (MCC) to remove inhaled particulates. Initiating events in chronic airway diseases, such as cystic fibrosis (CF), have been traced… (more)

Subjects/Keywords: School of Medicine; Department of Cell Biology and Physiology

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APA (6th Edition):

Rollins, B. (2010). Regulation of the Epithelial Sodium Channel (ENaC) by the Short Palate, Lung, and Nasal Epithelial Clone (SPLUNC1). (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:23a7af78-feaa-4a84-8f8f-be2a38b9c449

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Rollins, Brett. “Regulation of the Epithelial Sodium Channel (ENaC) by the Short Palate, Lung, and Nasal Epithelial Clone (SPLUNC1).” 2010. Thesis, University of North Carolina. Accessed October 30, 2020. https://cdr.lib.unc.edu/record/uuid:23a7af78-feaa-4a84-8f8f-be2a38b9c449.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Rollins, Brett. “Regulation of the Epithelial Sodium Channel (ENaC) by the Short Palate, Lung, and Nasal Epithelial Clone (SPLUNC1).” 2010. Web. 30 Oct 2020.

Vancouver:

Rollins B. Regulation of the Epithelial Sodium Channel (ENaC) by the Short Palate, Lung, and Nasal Epithelial Clone (SPLUNC1). [Internet] [Thesis]. University of North Carolina; 2010. [cited 2020 Oct 30]. Available from: https://cdr.lib.unc.edu/record/uuid:23a7af78-feaa-4a84-8f8f-be2a38b9c449.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Rollins B. Regulation of the Epithelial Sodium Channel (ENaC) by the Short Palate, Lung, and Nasal Epithelial Clone (SPLUNC1). [Thesis]. University of North Carolina; 2010. Available from: https://cdr.lib.unc.edu/record/uuid:23a7af78-feaa-4a84-8f8f-be2a38b9c449

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

11. Foti, Stacey. Novel AAV-mediated Therapeutic Strategies for Epilepsy.

Degree: 2008, University of North Carolina

 Epilepsy afflicts 2.1 million people in the United States, and many patients develop drug resistant seizures. For these patients, gene therapy may be an attractive… (more)

Subjects/Keywords: School of Medicine; UNC Neuroscience Center; Neuroscience Curriculum

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APA (6th Edition):

Foti, S. (2008). Novel AAV-mediated Therapeutic Strategies for Epilepsy. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:a332c4e2-79fa-4b41-97b5-d1786e2656b8

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Foti, Stacey. “Novel AAV-mediated Therapeutic Strategies for Epilepsy.” 2008. Thesis, University of North Carolina. Accessed October 30, 2020. https://cdr.lib.unc.edu/record/uuid:a332c4e2-79fa-4b41-97b5-d1786e2656b8.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Foti, Stacey. “Novel AAV-mediated Therapeutic Strategies for Epilepsy.” 2008. Web. 30 Oct 2020.

Vancouver:

Foti S. Novel AAV-mediated Therapeutic Strategies for Epilepsy. [Internet] [Thesis]. University of North Carolina; 2008. [cited 2020 Oct 30]. Available from: https://cdr.lib.unc.edu/record/uuid:a332c4e2-79fa-4b41-97b5-d1786e2656b8.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Foti S. Novel AAV-mediated Therapeutic Strategies for Epilepsy. [Thesis]. University of North Carolina; 2008. Available from: https://cdr.lib.unc.edu/record/uuid:a332c4e2-79fa-4b41-97b5-d1786e2656b8

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

12. Peck, Bailey. Analysis of microRNA Function in the Gastrointestinal Tract.

Degree: 2016, University of North Carolina

 microRNAs (miRNAs) are a group of small non-coding RNAs that regulate gene expression through post-transcriptional targeting of messenger RNAs (mRNAs). Discovered in mammals in 2001,… (more)

Subjects/Keywords: School of Medicine; Curriculum in Genetics and Molecular Biology

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APA (6th Edition):

Peck, B. (2016). Analysis of microRNA Function in the Gastrointestinal Tract. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:3dff714d-1778-48fd-a3fd-0aa1a1b2a67c

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Peck, Bailey. “Analysis of microRNA Function in the Gastrointestinal Tract.” 2016. Thesis, University of North Carolina. Accessed October 30, 2020. https://cdr.lib.unc.edu/record/uuid:3dff714d-1778-48fd-a3fd-0aa1a1b2a67c.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Peck, Bailey. “Analysis of microRNA Function in the Gastrointestinal Tract.” 2016. Web. 30 Oct 2020.

Vancouver:

Peck B. Analysis of microRNA Function in the Gastrointestinal Tract. [Internet] [Thesis]. University of North Carolina; 2016. [cited 2020 Oct 30]. Available from: https://cdr.lib.unc.edu/record/uuid:3dff714d-1778-48fd-a3fd-0aa1a1b2a67c.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Peck B. Analysis of microRNA Function in the Gastrointestinal Tract. [Thesis]. University of North Carolina; 2016. Available from: https://cdr.lib.unc.edu/record/uuid:3dff714d-1778-48fd-a3fd-0aa1a1b2a67c

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

13. Andres, Sarah. Expression patterns and roles of the insulin receptor in the intestinal epithelium.

Degree: Cell Biology and Physiology, 2014, University of North Carolina

 Hyperinsulinemia predicts increased risk of precancerous colon adenoma. Obesity, hyperinsulinemia, and insulin resistance are also associated with aberrant lipid handling by the small intestine. Despite… (more)

Subjects/Keywords: School of Medicine; Department of Cell Biology and Physiology

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APA (6th Edition):

Andres, S. (2014). Expression patterns and roles of the insulin receptor in the intestinal epithelium. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:a6501346-6c7d-4b2d-b0f9-5c7753098c7e

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Andres, Sarah. “Expression patterns and roles of the insulin receptor in the intestinal epithelium.” 2014. Thesis, University of North Carolina. Accessed October 30, 2020. https://cdr.lib.unc.edu/record/uuid:a6501346-6c7d-4b2d-b0f9-5c7753098c7e.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Andres, Sarah. “Expression patterns and roles of the insulin receptor in the intestinal epithelium.” 2014. Web. 30 Oct 2020.

Vancouver:

Andres S. Expression patterns and roles of the insulin receptor in the intestinal epithelium. [Internet] [Thesis]. University of North Carolina; 2014. [cited 2020 Oct 30]. Available from: https://cdr.lib.unc.edu/record/uuid:a6501346-6c7d-4b2d-b0f9-5c7753098c7e.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Andres S. Expression patterns and roles of the insulin receptor in the intestinal epithelium. [Thesis]. University of North Carolina; 2014. Available from: https://cdr.lib.unc.edu/record/uuid:a6501346-6c7d-4b2d-b0f9-5c7753098c7e

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

14. Freeman, Leslie. A Role for the NLR Family Members NLRC4 And NLRP3 in Astrocytic Inflammasome Activation and Astrogliosis.

Degree: 2016, University of North Carolina

 The inflammasome is implicated in many inflammatory diseases but has been primarily studied in the macrophage-myeloid lineage. Here we demonstrate a physiologic role for nucleotide-binding… (more)

Subjects/Keywords: School of Medicine; Curriculum in Genetics and Molecular Biology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Freeman, L. (2016). A Role for the NLR Family Members NLRC4 And NLRP3 in Astrocytic Inflammasome Activation and Astrogliosis. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:ec028a13-7c5a-41ed-af8d-3b5a5865278d

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Freeman, Leslie. “A Role for the NLR Family Members NLRC4 And NLRP3 in Astrocytic Inflammasome Activation and Astrogliosis.” 2016. Thesis, University of North Carolina. Accessed October 30, 2020. https://cdr.lib.unc.edu/record/uuid:ec028a13-7c5a-41ed-af8d-3b5a5865278d.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Freeman, Leslie. “A Role for the NLR Family Members NLRC4 And NLRP3 in Astrocytic Inflammasome Activation and Astrogliosis.” 2016. Web. 30 Oct 2020.

Vancouver:

Freeman L. A Role for the NLR Family Members NLRC4 And NLRP3 in Astrocytic Inflammasome Activation and Astrogliosis. [Internet] [Thesis]. University of North Carolina; 2016. [cited 2020 Oct 30]. Available from: https://cdr.lib.unc.edu/record/uuid:ec028a13-7c5a-41ed-af8d-3b5a5865278d.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Freeman L. A Role for the NLR Family Members NLRC4 And NLRP3 in Astrocytic Inflammasome Activation and Astrogliosis. [Thesis]. University of North Carolina; 2016. Available from: https://cdr.lib.unc.edu/record/uuid:ec028a13-7c5a-41ed-af8d-3b5a5865278d

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

15. Perez-Chanona, Ernesto. Microbial signaling in the intestinal ischemia/reperfusion injury response.

Degree: Pharmacology, 2014, University of North Carolina

 The intestine is home to one of the most diverse and populous ecosystems on earth. The intestinal epithelium the single layer sheet of columnar cells… (more)

Subjects/Keywords: Pharmacology; Microbiology; Immunology; School of Medicine; Department of Pharmacology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Perez-Chanona, E. (2014). Microbial signaling in the intestinal ischemia/reperfusion injury response. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:19a719f1-4373-4d1e-8380-3944bd7f33cd

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Perez-Chanona, Ernesto. “Microbial signaling in the intestinal ischemia/reperfusion injury response.” 2014. Thesis, University of North Carolina. Accessed October 30, 2020. https://cdr.lib.unc.edu/record/uuid:19a719f1-4373-4d1e-8380-3944bd7f33cd.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Perez-Chanona, Ernesto. “Microbial signaling in the intestinal ischemia/reperfusion injury response.” 2014. Web. 30 Oct 2020.

Vancouver:

Perez-Chanona E. Microbial signaling in the intestinal ischemia/reperfusion injury response. [Internet] [Thesis]. University of North Carolina; 2014. [cited 2020 Oct 30]. Available from: https://cdr.lib.unc.edu/record/uuid:19a719f1-4373-4d1e-8380-3944bd7f33cd.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Perez-Chanona E. Microbial signaling in the intestinal ischemia/reperfusion injury response. [Thesis]. University of North Carolina; 2014. Available from: https://cdr.lib.unc.edu/record/uuid:19a719f1-4373-4d1e-8380-3944bd7f33cd

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

.