Advanced search options

Advanced Search Options 🞨

Browse by author name (“Author name starts with…”).

Find ETDs with:

in
/  
in
/  
in
/  
in

Written in Published in Earliest date Latest date

Sorted by

Results per page:

Sorted by: relevance · author · university · dateNew search

You searched for +publisher:"University of North Carolina" +contributor:("Johnson, Gary"). Showing records 1 – 22 of 22 total matches.

Search Limiters

Last 2 Years | English Only

No search limiters apply to these results.

▼ Search Limiters


University of North Carolina

1. Cronan, Mark Robert. MAP3K regulation of MAPK activation in vitro and tumor growth and metastasis in vivo.

Degree: Biochemistry and Biophysics, 2010, University of North Carolina

 Mitogen activated protein kinase (MAPK) signaling is frequently dysregulated in cancer and contributes to both tumor growth and metastasis. These signaling networks consist of a… (more)

Subjects/Keywords: School of Medicine; Department of Biochemistry and Biophysics

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Cronan, M. R. (2010). MAP3K regulation of MAPK activation in vitro and tumor growth and metastasis in vivo. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:a4c22497-e881-4c88-b485-6436bd7ee938

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Cronan, Mark Robert. “MAP3K regulation of MAPK activation in vitro and tumor growth and metastasis in vivo.” 2010. Thesis, University of North Carolina. Accessed September 22, 2020. https://cdr.lib.unc.edu/record/uuid:a4c22497-e881-4c88-b485-6436bd7ee938.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Cronan, Mark Robert. “MAP3K regulation of MAPK activation in vitro and tumor growth and metastasis in vivo.” 2010. Web. 22 Sep 2020.

Vancouver:

Cronan MR. MAP3K regulation of MAPK activation in vitro and tumor growth and metastasis in vivo. [Internet] [Thesis]. University of North Carolina; 2010. [cited 2020 Sep 22]. Available from: https://cdr.lib.unc.edu/record/uuid:a4c22497-e881-4c88-b485-6436bd7ee938.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Cronan MR. MAP3K regulation of MAPK activation in vitro and tumor growth and metastasis in vivo. [Thesis]. University of North Carolina; 2010. Available from: https://cdr.lib.unc.edu/record/uuid:a4c22497-e881-4c88-b485-6436bd7ee938

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

2. Richardson, Bryan Timothy. Defining the Molecular Mechanisms of the Cerebral Cavernous Malformation Proteins.

Degree: Pharmacology, 2013, University of North Carolina

 Cerebral cavernous malformations (CCM) are the second most common class of cerebrovascular brain malformations affecting .1-.5% of the population. The disease is manifested in endothelial… (more)

Subjects/Keywords: School of Medicine; Department of Pharmacology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Richardson, B. T. (2013). Defining the Molecular Mechanisms of the Cerebral Cavernous Malformation Proteins. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:6478fcfa-19a8-41df-9bea-f7ca335f9d7c

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Richardson, Bryan Timothy. “Defining the Molecular Mechanisms of the Cerebral Cavernous Malformation Proteins.” 2013. Thesis, University of North Carolina. Accessed September 22, 2020. https://cdr.lib.unc.edu/record/uuid:6478fcfa-19a8-41df-9bea-f7ca335f9d7c.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Richardson, Bryan Timothy. “Defining the Molecular Mechanisms of the Cerebral Cavernous Malformation Proteins.” 2013. Web. 22 Sep 2020.

Vancouver:

Richardson BT. Defining the Molecular Mechanisms of the Cerebral Cavernous Malformation Proteins. [Internet] [Thesis]. University of North Carolina; 2013. [cited 2020 Sep 22]. Available from: https://cdr.lib.unc.edu/record/uuid:6478fcfa-19a8-41df-9bea-f7ca335f9d7c.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Richardson BT. Defining the Molecular Mechanisms of the Cerebral Cavernous Malformation Proteins. [Thesis]. University of North Carolina; 2013. Available from: https://cdr.lib.unc.edu/record/uuid:6478fcfa-19a8-41df-9bea-f7ca335f9d7c

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

3. Whittle, Martin C. Targeting the Triple-Negative Breast Cancer Kinome with Chemical Proteomics.

Degree: Pharmacology, 2013, University of North Carolina

 Kinases are members of a large dynamic and cooperative signaling network, which senses inhibition of key nodal kinases and induces compensatory responses that offset pharmacological… (more)

Subjects/Keywords: School of Medicine; Department of Pharmacology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Whittle, M. C. (2013). Targeting the Triple-Negative Breast Cancer Kinome with Chemical Proteomics. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:9ae50bab-4c5d-47ec-a991-f6785a6e367b

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Whittle, Martin C. “Targeting the Triple-Negative Breast Cancer Kinome with Chemical Proteomics.” 2013. Thesis, University of North Carolina. Accessed September 22, 2020. https://cdr.lib.unc.edu/record/uuid:9ae50bab-4c5d-47ec-a991-f6785a6e367b.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Whittle, Martin C. “Targeting the Triple-Negative Breast Cancer Kinome with Chemical Proteomics.” 2013. Web. 22 Sep 2020.

Vancouver:

Whittle MC. Targeting the Triple-Negative Breast Cancer Kinome with Chemical Proteomics. [Internet] [Thesis]. University of North Carolina; 2013. [cited 2020 Sep 22]. Available from: https://cdr.lib.unc.edu/record/uuid:9ae50bab-4c5d-47ec-a991-f6785a6e367b.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Whittle MC. Targeting the Triple-Negative Breast Cancer Kinome with Chemical Proteomics. [Thesis]. University of North Carolina; 2013. Available from: https://cdr.lib.unc.edu/record/uuid:9ae50bab-4c5d-47ec-a991-f6785a6e367b

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

4. Borikova, Asya Lazarova. Regulation and Function of Cerebral Cavernous Malformation Proteins.

Degree: Pharmacology, 2012, University of North Carolina

 Cerebral Cavernous Malformations (CCM) is a genetic disease that causes dilated, leaky blood vessels in the brain and manifests with neurological deficits, seizures and hemorrhagic… (more)

Subjects/Keywords: School of Medicine; Department of Pharmacology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Borikova, A. L. (2012). Regulation and Function of Cerebral Cavernous Malformation Proteins. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:cda18322-bbb6-4353-a015-77693c45aa2b

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Borikova, Asya Lazarova. “Regulation and Function of Cerebral Cavernous Malformation Proteins.” 2012. Thesis, University of North Carolina. Accessed September 22, 2020. https://cdr.lib.unc.edu/record/uuid:cda18322-bbb6-4353-a015-77693c45aa2b.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Borikova, Asya Lazarova. “Regulation and Function of Cerebral Cavernous Malformation Proteins.” 2012. Web. 22 Sep 2020.

Vancouver:

Borikova AL. Regulation and Function of Cerebral Cavernous Malformation Proteins. [Internet] [Thesis]. University of North Carolina; 2012. [cited 2020 Sep 22]. Available from: https://cdr.lib.unc.edu/record/uuid:cda18322-bbb6-4353-a015-77693c45aa2b.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Borikova AL. Regulation and Function of Cerebral Cavernous Malformation Proteins. [Thesis]. University of North Carolina; 2012. Available from: https://cdr.lib.unc.edu/record/uuid:cda18322-bbb6-4353-a015-77693c45aa2b

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

5. Dibble, Christopher F. Defining the Cerebral Cavernous Malformation Protein Signaling Network in Endothelial Cells.

Degree: Pharmacology, 2013, University of North Carolina

 Cerebral cavernous malformations (CCM) are cerebrovascular lesions occurring with homozygous loss of function mutations of CCM1, -2, and -3. CCMs can cause neurological deficits, seizures,… (more)

Subjects/Keywords: School of Medicine; Department of Pharmacology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Dibble, C. F. (2013). Defining the Cerebral Cavernous Malformation Protein Signaling Network in Endothelial Cells. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:27ad7963-2d52-4383-ac66-787358469055

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Dibble, Christopher F. “Defining the Cerebral Cavernous Malformation Protein Signaling Network in Endothelial Cells.” 2013. Thesis, University of North Carolina. Accessed September 22, 2020. https://cdr.lib.unc.edu/record/uuid:27ad7963-2d52-4383-ac66-787358469055.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Dibble, Christopher F. “Defining the Cerebral Cavernous Malformation Protein Signaling Network in Endothelial Cells.” 2013. Web. 22 Sep 2020.

Vancouver:

Dibble CF. Defining the Cerebral Cavernous Malformation Protein Signaling Network in Endothelial Cells. [Internet] [Thesis]. University of North Carolina; 2013. [cited 2020 Sep 22]. Available from: https://cdr.lib.unc.edu/record/uuid:27ad7963-2d52-4383-ac66-787358469055.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Dibble CF. Defining the Cerebral Cavernous Malformation Protein Signaling Network in Endothelial Cells. [Thesis]. University of North Carolina; 2013. Available from: https://cdr.lib.unc.edu/record/uuid:27ad7963-2d52-4383-ac66-787358469055

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

6. Rubel, Carrie. Defining the Molecular Mechanisms of Ubiquitin Proteasome System Dysfunction as a Driver of Disease: CHIP mutation in SCAR16.

Degree: Pharmacology, 2015, University of North Carolina

 All cells must respond to changes in their environment including a plethora of physiologic and pathologic stresses in order to maintain homeostasis and survive. Protein… (more)

Subjects/Keywords: Pharmacology; Cytology; Biochemistry; School of Medicine; Department of Pharmacology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Rubel, C. (2015). Defining the Molecular Mechanisms of Ubiquitin Proteasome System Dysfunction as a Driver of Disease: CHIP mutation in SCAR16. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:ef6ac26c-167b-4433-aa30-f24b3bace803

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Rubel, Carrie. “Defining the Molecular Mechanisms of Ubiquitin Proteasome System Dysfunction as a Driver of Disease: CHIP mutation in SCAR16.” 2015. Thesis, University of North Carolina. Accessed September 22, 2020. https://cdr.lib.unc.edu/record/uuid:ef6ac26c-167b-4433-aa30-f24b3bace803.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Rubel, Carrie. “Defining the Molecular Mechanisms of Ubiquitin Proteasome System Dysfunction as a Driver of Disease: CHIP mutation in SCAR16.” 2015. Web. 22 Sep 2020.

Vancouver:

Rubel C. Defining the Molecular Mechanisms of Ubiquitin Proteasome System Dysfunction as a Driver of Disease: CHIP mutation in SCAR16. [Internet] [Thesis]. University of North Carolina; 2015. [cited 2020 Sep 22]. Available from: https://cdr.lib.unc.edu/record/uuid:ef6ac26c-167b-4433-aa30-f24b3bace803.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Rubel C. Defining the Molecular Mechanisms of Ubiquitin Proteasome System Dysfunction as a Driver of Disease: CHIP mutation in SCAR16. [Thesis]. University of North Carolina; 2015. Available from: https://cdr.lib.unc.edu/record/uuid:ef6ac26c-167b-4433-aa30-f24b3bace803

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

7. Vincent Jordan, Nicole M. Epigenetic Regulation of Epithelial-Mesenchymal Transition in Epithelial Stem Cells and Triple Negative Breast Cancer.

Degree: Pharmacology, 2012, University of North Carolina

 Epithelial-mesenchymal transition (EMT) is a cellular program responsible for the conversion of stationary epithelial cells characterized by apical-basolateral polarity to motile mesenchymal cells with front-back… (more)

Subjects/Keywords: School of Medicine; Department of Pharmacology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Vincent Jordan, N. M. (2012). Epigenetic Regulation of Epithelial-Mesenchymal Transition in Epithelial Stem Cells and Triple Negative Breast Cancer. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:de2f88af-a114-41ec-90e7-8eec07b91fbc

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Vincent Jordan, Nicole M. “Epigenetic Regulation of Epithelial-Mesenchymal Transition in Epithelial Stem Cells and Triple Negative Breast Cancer.” 2012. Thesis, University of North Carolina. Accessed September 22, 2020. https://cdr.lib.unc.edu/record/uuid:de2f88af-a114-41ec-90e7-8eec07b91fbc.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Vincent Jordan, Nicole M. “Epigenetic Regulation of Epithelial-Mesenchymal Transition in Epithelial Stem Cells and Triple Negative Breast Cancer.” 2012. Web. 22 Sep 2020.

Vancouver:

Vincent Jordan NM. Epigenetic Regulation of Epithelial-Mesenchymal Transition in Epithelial Stem Cells and Triple Negative Breast Cancer. [Internet] [Thesis]. University of North Carolina; 2012. [cited 2020 Sep 22]. Available from: https://cdr.lib.unc.edu/record/uuid:de2f88af-a114-41ec-90e7-8eec07b91fbc.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Vincent Jordan NM. Epigenetic Regulation of Epithelial-Mesenchymal Transition in Epithelial Stem Cells and Triple Negative Breast Cancer. [Thesis]. University of North Carolina; 2012. Available from: https://cdr.lib.unc.edu/record/uuid:de2f88af-a114-41ec-90e7-8eec07b91fbc

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

8. Gentry, Leanna. Targeting the Ras-Ral effector pathway for cancer treatment.

Degree: Pharmacology, 2015, University of North Carolina

 The RAS oncogene is the most frequently mutated gene in human cancers, and this activated Ras oncoprotein has been shown to be required for both… (more)

Subjects/Keywords: Pharmacology; School of Medicine; Department of Pharmacology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Gentry, L. (2015). Targeting the Ras-Ral effector pathway for cancer treatment. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:c75ba6bc-3f00-4527-8f20-029bf699aad2

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Gentry, Leanna. “Targeting the Ras-Ral effector pathway for cancer treatment.” 2015. Thesis, University of North Carolina. Accessed September 22, 2020. https://cdr.lib.unc.edu/record/uuid:c75ba6bc-3f00-4527-8f20-029bf699aad2.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Gentry, Leanna. “Targeting the Ras-Ral effector pathway for cancer treatment.” 2015. Web. 22 Sep 2020.

Vancouver:

Gentry L. Targeting the Ras-Ral effector pathway for cancer treatment. [Internet] [Thesis]. University of North Carolina; 2015. [cited 2020 Sep 22]. Available from: https://cdr.lib.unc.edu/record/uuid:c75ba6bc-3f00-4527-8f20-029bf699aad2.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Gentry L. Targeting the Ras-Ral effector pathway for cancer treatment. [Thesis]. University of North Carolina; 2015. Available from: https://cdr.lib.unc.edu/record/uuid:c75ba6bc-3f00-4527-8f20-029bf699aad2

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

9. Maxfield, Kimberly. Genetic Dissection of Therapeutic Intervention Targets in Triple Negative Breast Cancer.

Degree: Pharmacology, 2015, University of North Carolina

 Triple negative breast cancer (TNBC) is the most aggressive and metastatic type of breast cancer, accounting for 20% of all breast cancer diagnoses. Currently, there… (more)

Subjects/Keywords: Pharmacology; Biology; School of Medicine; Department of Pharmacology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Maxfield, K. (2015). Genetic Dissection of Therapeutic Intervention Targets in Triple Negative Breast Cancer. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:318b1399-81a7-4737-b0c7-96f242284420

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Maxfield, Kimberly. “Genetic Dissection of Therapeutic Intervention Targets in Triple Negative Breast Cancer.” 2015. Thesis, University of North Carolina. Accessed September 22, 2020. https://cdr.lib.unc.edu/record/uuid:318b1399-81a7-4737-b0c7-96f242284420.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Maxfield, Kimberly. “Genetic Dissection of Therapeutic Intervention Targets in Triple Negative Breast Cancer.” 2015. Web. 22 Sep 2020.

Vancouver:

Maxfield K. Genetic Dissection of Therapeutic Intervention Targets in Triple Negative Breast Cancer. [Internet] [Thesis]. University of North Carolina; 2015. [cited 2020 Sep 22]. Available from: https://cdr.lib.unc.edu/record/uuid:318b1399-81a7-4737-b0c7-96f242284420.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Maxfield K. Genetic Dissection of Therapeutic Intervention Targets in Triple Negative Breast Cancer. [Thesis]. University of North Carolina; 2015. Available from: https://cdr.lib.unc.edu/record/uuid:318b1399-81a7-4737-b0c7-96f242284420

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

10. Zimmerman, Eric. MOLECULAR ADAPTATION TO ANTI-CANCER CHEMOTHERAPY IN LEUKEMIA.

Degree: Pharmacology, 2011, University of North Carolina

 Drug resistance to anti-cancer chemotherapy is a significant barrier to the treatment of leukemia patients. Many times, resistance results from molecular adaptation to drug exposure,… (more)

Subjects/Keywords: School of Medicine; Department of Pharmacology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Zimmerman, E. (2011). MOLECULAR ADAPTATION TO ANTI-CANCER CHEMOTHERAPY IN LEUKEMIA. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:ff100956-ccba-4659-b349-99bda9a325d6

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Zimmerman, Eric. “MOLECULAR ADAPTATION TO ANTI-CANCER CHEMOTHERAPY IN LEUKEMIA.” 2011. Thesis, University of North Carolina. Accessed September 22, 2020. https://cdr.lib.unc.edu/record/uuid:ff100956-ccba-4659-b349-99bda9a325d6.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Zimmerman, Eric. “MOLECULAR ADAPTATION TO ANTI-CANCER CHEMOTHERAPY IN LEUKEMIA.” 2011. Web. 22 Sep 2020.

Vancouver:

Zimmerman E. MOLECULAR ADAPTATION TO ANTI-CANCER CHEMOTHERAPY IN LEUKEMIA. [Internet] [Thesis]. University of North Carolina; 2011. [cited 2020 Sep 22]. Available from: https://cdr.lib.unc.edu/record/uuid:ff100956-ccba-4659-b349-99bda9a325d6.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Zimmerman E. MOLECULAR ADAPTATION TO ANTI-CANCER CHEMOTHERAPY IN LEUKEMIA. [Thesis]. University of North Carolina; 2011. Available from: https://cdr.lib.unc.edu/record/uuid:ff100956-ccba-4659-b349-99bda9a325d6

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

11. Alan, Jamie. TYROSINE KINASE MODULATION OF TRAFFICKING AND BIOLOGICAL FUNCTIONS OF THE ATYPICAL RHO GTPASE, WRCH-1.

Degree: Pharmacology, 2010, University of North Carolina

 Wrch-1 is an atypical Rho family small GTPase with roles in oncogenic transformation, epithelial cell morphogenesis, osteoclastogenesis, and migration. We have shown previously that Wrch-1… (more)

Subjects/Keywords: School of Medicine; Department of Pharmacology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Alan, J. (2010). TYROSINE KINASE MODULATION OF TRAFFICKING AND BIOLOGICAL FUNCTIONS OF THE ATYPICAL RHO GTPASE, WRCH-1. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:afbc9ce5-f21d-4707-8744-357971e1eb3b

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Alan, Jamie. “TYROSINE KINASE MODULATION OF TRAFFICKING AND BIOLOGICAL FUNCTIONS OF THE ATYPICAL RHO GTPASE, WRCH-1.” 2010. Thesis, University of North Carolina. Accessed September 22, 2020. https://cdr.lib.unc.edu/record/uuid:afbc9ce5-f21d-4707-8744-357971e1eb3b.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Alan, Jamie. “TYROSINE KINASE MODULATION OF TRAFFICKING AND BIOLOGICAL FUNCTIONS OF THE ATYPICAL RHO GTPASE, WRCH-1.” 2010. Web. 22 Sep 2020.

Vancouver:

Alan J. TYROSINE KINASE MODULATION OF TRAFFICKING AND BIOLOGICAL FUNCTIONS OF THE ATYPICAL RHO GTPASE, WRCH-1. [Internet] [Thesis]. University of North Carolina; 2010. [cited 2020 Sep 22]. Available from: https://cdr.lib.unc.edu/record/uuid:afbc9ce5-f21d-4707-8744-357971e1eb3b.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Alan J. TYROSINE KINASE MODULATION OF TRAFFICKING AND BIOLOGICAL FUNCTIONS OF THE ATYPICAL RHO GTPASE, WRCH-1. [Thesis]. University of North Carolina; 2010. Available from: https://cdr.lib.unc.edu/record/uuid:afbc9ce5-f21d-4707-8744-357971e1eb3b

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

12. Hayes, Tikvah. Targeting mutant KRAS in pancreatic cancer.

Degree: 2016, University of North Carolina

 The development of pharmacologic inhibitors of the KRAS oncoprotein, which is mutated in ~30% of all human cancers, has been at the forefront of drug… (more)

Subjects/Keywords: School of Medicine; Curriculum in Genetics and Molecular Biology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Hayes, T. (2016). Targeting mutant KRAS in pancreatic cancer. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:f816a0b0-3d43-4afb-9623-dcbfb1d7bd39

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hayes, Tikvah. “Targeting mutant KRAS in pancreatic cancer.” 2016. Thesis, University of North Carolina. Accessed September 22, 2020. https://cdr.lib.unc.edu/record/uuid:f816a0b0-3d43-4afb-9623-dcbfb1d7bd39.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hayes, Tikvah. “Targeting mutant KRAS in pancreatic cancer.” 2016. Web. 22 Sep 2020.

Vancouver:

Hayes T. Targeting mutant KRAS in pancreatic cancer. [Internet] [Thesis]. University of North Carolina; 2016. [cited 2020 Sep 22]. Available from: https://cdr.lib.unc.edu/record/uuid:f816a0b0-3d43-4afb-9623-dcbfb1d7bd39.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hayes T. Targeting mutant KRAS in pancreatic cancer. [Thesis]. University of North Carolina; 2016. Available from: https://cdr.lib.unc.edu/record/uuid:f816a0b0-3d43-4afb-9623-dcbfb1d7bd39

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

13. Zhou, Bingying. RAS signaling and therapeutic resistance in melanoma.

Degree: Pharmacology, 2016, University of North Carolina

 Increasing appreciation of the complexity of RAS signaling in cancer has led to a renewed wave of RAS research. I have focused on two key… (more)

Subjects/Keywords: School of Medicine; Department of Pharmacology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Zhou, B. (2016). RAS signaling and therapeutic resistance in melanoma. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:26347cfa-34a3-4849-b350-f66b6a70334a

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Zhou, Bingying. “RAS signaling and therapeutic resistance in melanoma.” 2016. Thesis, University of North Carolina. Accessed September 22, 2020. https://cdr.lib.unc.edu/record/uuid:26347cfa-34a3-4849-b350-f66b6a70334a.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Zhou, Bingying. “RAS signaling and therapeutic resistance in melanoma.” 2016. Web. 22 Sep 2020.

Vancouver:

Zhou B. RAS signaling and therapeutic resistance in melanoma. [Internet] [Thesis]. University of North Carolina; 2016. [cited 2020 Sep 22]. Available from: https://cdr.lib.unc.edu/record/uuid:26347cfa-34a3-4849-b350-f66b6a70334a.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Zhou B. RAS signaling and therapeutic resistance in melanoma. [Thesis]. University of North Carolina; 2016. Available from: https://cdr.lib.unc.edu/record/uuid:26347cfa-34a3-4849-b350-f66b6a70334a

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

14. Anders, Penny. Modulation of Signal Transduction and Transformation by Kaposi’s Sarcoma-Associated Herpesvirus.

Degree: Microbiology and Immunology, 2016, University of North Carolina

 Kaposi’s sarcoma associated-herpesvirus (KSHV) is a double-strand DNA gamma herpesvirus that establishes lifelong latent infection in the human host. KSHV infection is associated with several… (more)

Subjects/Keywords: School of Medicine; Department of Microbiology and Immunology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Anders, P. (2016). Modulation of Signal Transduction and Transformation by Kaposi’s Sarcoma-Associated Herpesvirus. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:d18be56b-2603-463f-8fc4-b155ab1e432a

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Anders, Penny. “Modulation of Signal Transduction and Transformation by Kaposi’s Sarcoma-Associated Herpesvirus.” 2016. Thesis, University of North Carolina. Accessed September 22, 2020. https://cdr.lib.unc.edu/record/uuid:d18be56b-2603-463f-8fc4-b155ab1e432a.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Anders, Penny. “Modulation of Signal Transduction and Transformation by Kaposi’s Sarcoma-Associated Herpesvirus.” 2016. Web. 22 Sep 2020.

Vancouver:

Anders P. Modulation of Signal Transduction and Transformation by Kaposi’s Sarcoma-Associated Herpesvirus. [Internet] [Thesis]. University of North Carolina; 2016. [cited 2020 Sep 22]. Available from: https://cdr.lib.unc.edu/record/uuid:d18be56b-2603-463f-8fc4-b155ab1e432a.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Anders P. Modulation of Signal Transduction and Transformation by Kaposi’s Sarcoma-Associated Herpesvirus. [Thesis]. University of North Carolina; 2016. Available from: https://cdr.lib.unc.edu/record/uuid:d18be56b-2603-463f-8fc4-b155ab1e432a

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

15. Mills, Christine. Ubiquitin and Ubiquitin-Like Proteins in Cell Cycle Regulation.

Degree: Pharmacology, 2018, University of North Carolina

 Cell cycle is a tightly regulated process; however, it is mis-regulated in many cancers, leading to increased proliferation. Our lab is interested in better understanding… (more)

Subjects/Keywords: School of Medicine; Department of Pharmacology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Mills, C. (2018). Ubiquitin and Ubiquitin-Like Proteins in Cell Cycle Regulation. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:4ee84949-170b-485b-b110-7e036074c852

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Mills, Christine. “Ubiquitin and Ubiquitin-Like Proteins in Cell Cycle Regulation.” 2018. Thesis, University of North Carolina. Accessed September 22, 2020. https://cdr.lib.unc.edu/record/uuid:4ee84949-170b-485b-b110-7e036074c852.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Mills, Christine. “Ubiquitin and Ubiquitin-Like Proteins in Cell Cycle Regulation.” 2018. Web. 22 Sep 2020.

Vancouver:

Mills C. Ubiquitin and Ubiquitin-Like Proteins in Cell Cycle Regulation. [Internet] [Thesis]. University of North Carolina; 2018. [cited 2020 Sep 22]. Available from: https://cdr.lib.unc.edu/record/uuid:4ee84949-170b-485b-b110-7e036074c852.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Mills C. Ubiquitin and Ubiquitin-Like Proteins in Cell Cycle Regulation. [Thesis]. University of North Carolina; 2018. Available from: https://cdr.lib.unc.edu/record/uuid:4ee84949-170b-485b-b110-7e036074c852

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

16. Melehani, Jason. Mechanisms and consequences of Staphylococcus aureus Leukocidin AB-mediated activation of the host NLRP3 inflammasome.

Degree: Pharmacology, 2016, University of North Carolina

 The NLRP3 inflammasome is a critical innate immune sensor implicated in the pathogenesis of dozens of infectious and non-infectious diseases. Activation of the NLRP3 inflammasome… (more)

Subjects/Keywords: School of Medicine; Department of Pharmacology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Melehani, J. (2016). Mechanisms and consequences of Staphylococcus aureus Leukocidin AB-mediated activation of the host NLRP3 inflammasome. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:f98e7df6-408e-40db-8202-8ab65dd52e01

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Melehani, Jason. “Mechanisms and consequences of Staphylococcus aureus Leukocidin AB-mediated activation of the host NLRP3 inflammasome.” 2016. Thesis, University of North Carolina. Accessed September 22, 2020. https://cdr.lib.unc.edu/record/uuid:f98e7df6-408e-40db-8202-8ab65dd52e01.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Melehani, Jason. “Mechanisms and consequences of Staphylococcus aureus Leukocidin AB-mediated activation of the host NLRP3 inflammasome.” 2016. Web. 22 Sep 2020.

Vancouver:

Melehani J. Mechanisms and consequences of Staphylococcus aureus Leukocidin AB-mediated activation of the host NLRP3 inflammasome. [Internet] [Thesis]. University of North Carolina; 2016. [cited 2020 Sep 22]. Available from: https://cdr.lib.unc.edu/record/uuid:f98e7df6-408e-40db-8202-8ab65dd52e01.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Melehani J. Mechanisms and consequences of Staphylococcus aureus Leukocidin AB-mediated activation of the host NLRP3 inflammasome. [Thesis]. University of North Carolina; 2016. Available from: https://cdr.lib.unc.edu/record/uuid:f98e7df6-408e-40db-8202-8ab65dd52e01

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

17. Baker, Nicole. Evaluating the therapeutic potential of the PAK1 and TBK1 kinases in pancreatic ductal adenocarcinoma.

Degree: Pharmacology, 2016, University of North Carolina

 Pancreatic ductal adenocarcinoma (PDAC) is an extremely lethal cancer characterized by a high frequency (>95%) of activating mutations in the KRAS oncogene, which is a… (more)

Subjects/Keywords: School of Medicine; Department of Pharmacology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Baker, N. (2016). Evaluating the therapeutic potential of the PAK1 and TBK1 kinases in pancreatic ductal adenocarcinoma. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:6dc9a875-b142-43f8-b49e-11482c6de322

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Baker, Nicole. “Evaluating the therapeutic potential of the PAK1 and TBK1 kinases in pancreatic ductal adenocarcinoma.” 2016. Thesis, University of North Carolina. Accessed September 22, 2020. https://cdr.lib.unc.edu/record/uuid:6dc9a875-b142-43f8-b49e-11482c6de322.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Baker, Nicole. “Evaluating the therapeutic potential of the PAK1 and TBK1 kinases in pancreatic ductal adenocarcinoma.” 2016. Web. 22 Sep 2020.

Vancouver:

Baker N. Evaluating the therapeutic potential of the PAK1 and TBK1 kinases in pancreatic ductal adenocarcinoma. [Internet] [Thesis]. University of North Carolina; 2016. [cited 2020 Sep 22]. Available from: https://cdr.lib.unc.edu/record/uuid:6dc9a875-b142-43f8-b49e-11482c6de322.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Baker N. Evaluating the therapeutic potential of the PAK1 and TBK1 kinases in pancreatic ductal adenocarcinoma. [Thesis]. University of North Carolina; 2016. Available from: https://cdr.lib.unc.edu/record/uuid:6dc9a875-b142-43f8-b49e-11482c6de322

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

18. Ryan, Meagan. TARGETING THE ERK MAPK PATHWAY IN RAS-DRIVEN CANCERS.

Degree: Pharmacology, 2016, University of North Carolina

 RAS mutations are frequently found in the deadliest cancers in the United States, and there is a renewed interest in identifying therapeutic strategies to target… (more)

Subjects/Keywords: School of Medicine; Department of Pharmacology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Ryan, M. (2016). TARGETING THE ERK MAPK PATHWAY IN RAS-DRIVEN CANCERS. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:c9d9e3f8-a30a-4eb5-ba62-c021deb34805

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ryan, Meagan. “TARGETING THE ERK MAPK PATHWAY IN RAS-DRIVEN CANCERS.” 2016. Thesis, University of North Carolina. Accessed September 22, 2020. https://cdr.lib.unc.edu/record/uuid:c9d9e3f8-a30a-4eb5-ba62-c021deb34805.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ryan, Meagan. “TARGETING THE ERK MAPK PATHWAY IN RAS-DRIVEN CANCERS.” 2016. Web. 22 Sep 2020.

Vancouver:

Ryan M. TARGETING THE ERK MAPK PATHWAY IN RAS-DRIVEN CANCERS. [Internet] [Thesis]. University of North Carolina; 2016. [cited 2020 Sep 22]. Available from: https://cdr.lib.unc.edu/record/uuid:c9d9e3f8-a30a-4eb5-ba62-c021deb34805.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ryan M. TARGETING THE ERK MAPK PATHWAY IN RAS-DRIVEN CANCERS. [Thesis]. University of North Carolina; 2016. Available from: https://cdr.lib.unc.edu/record/uuid:c9d9e3f8-a30a-4eb5-ba62-c021deb34805

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

19. Stratford, Jeran. Aberrant Gene Expression: Diagnostic Markers and Therapeutic Targets for Pancreatic Cancer.

Degree: Pharmacology, 2014, University of North Carolina

 Pancreatic ductal adenocarcinoma (PDAC) is an aggressive cancer and the fourth leading cause of cancer-related death in the United States. The overall median survival for… (more)

Subjects/Keywords: Pharmacology; School of Medicine; Department of Pharmacology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Stratford, J. (2014). Aberrant Gene Expression: Diagnostic Markers and Therapeutic Targets for Pancreatic Cancer. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:84f3613f-5709-4ae6-8407-999e9eb3a6eb

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Stratford, Jeran. “Aberrant Gene Expression: Diagnostic Markers and Therapeutic Targets for Pancreatic Cancer.” 2014. Thesis, University of North Carolina. Accessed September 22, 2020. https://cdr.lib.unc.edu/record/uuid:84f3613f-5709-4ae6-8407-999e9eb3a6eb.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Stratford, Jeran. “Aberrant Gene Expression: Diagnostic Markers and Therapeutic Targets for Pancreatic Cancer.” 2014. Web. 22 Sep 2020.

Vancouver:

Stratford J. Aberrant Gene Expression: Diagnostic Markers and Therapeutic Targets for Pancreatic Cancer. [Internet] [Thesis]. University of North Carolina; 2014. [cited 2020 Sep 22]. Available from: https://cdr.lib.unc.edu/record/uuid:84f3613f-5709-4ae6-8407-999e9eb3a6eb.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Stratford J. Aberrant Gene Expression: Diagnostic Markers and Therapeutic Targets for Pancreatic Cancer. [Thesis]. University of North Carolina; 2014. Available from: https://cdr.lib.unc.edu/record/uuid:84f3613f-5709-4ae6-8407-999e9eb3a6eb

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

20. Okumu, Denis. Lyn regulates drug resistance mechanisms in chronic myelogenous leukemia (CML).

Degree: Pharmacology, 2018, University of North Carolina

 Acquired resistance to anti-cancer therapy presents a critical challenge to effective clinical management of chronic myelogenous leukemia (CML). Drug-resistant CML cells devise diverse molecular adaptations… (more)

Subjects/Keywords: School of Medicine; Department of Pharmacology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Okumu, D. (2018). Lyn regulates drug resistance mechanisms in chronic myelogenous leukemia (CML). (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:070f858a-ab62-46b2-bec6-bf6e771fc340

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Okumu, Denis. “Lyn regulates drug resistance mechanisms in chronic myelogenous leukemia (CML).” 2018. Thesis, University of North Carolina. Accessed September 22, 2020. https://cdr.lib.unc.edu/record/uuid:070f858a-ab62-46b2-bec6-bf6e771fc340.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Okumu, Denis. “Lyn regulates drug resistance mechanisms in chronic myelogenous leukemia (CML).” 2018. Web. 22 Sep 2020.

Vancouver:

Okumu D. Lyn regulates drug resistance mechanisms in chronic myelogenous leukemia (CML). [Internet] [Thesis]. University of North Carolina; 2018. [cited 2020 Sep 22]. Available from: https://cdr.lib.unc.edu/record/uuid:070f858a-ab62-46b2-bec6-bf6e771fc340.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Okumu D. Lyn regulates drug resistance mechanisms in chronic myelogenous leukemia (CML). [Thesis]. University of North Carolina; 2018. Available from: https://cdr.lib.unc.edu/record/uuid:070f858a-ab62-46b2-bec6-bf6e771fc340

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

21. Crose, Lisa Eileen Stalheim. Regulation and function of the cerebral cavernous malformation 2 protein.

Degree: Pharmacology, 2009, University of North Carolina

 Cerebral cavernous malformations (CCM) are vascular lesions of the central nervous system characterized as clusters of dilated, thin-walled blood vessels. CCM lesions are fragile and… (more)

Subjects/Keywords: School of Medicine; Department of Pharmacology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Crose, L. E. S. (2009). Regulation and function of the cerebral cavernous malformation 2 protein. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:7a7bbdba-6b98-4bfa-bf8f-cf6fd10f2eb9

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Crose, Lisa Eileen Stalheim. “Regulation and function of the cerebral cavernous malformation 2 protein.” 2009. Thesis, University of North Carolina. Accessed September 22, 2020. https://cdr.lib.unc.edu/record/uuid:7a7bbdba-6b98-4bfa-bf8f-cf6fd10f2eb9.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Crose, Lisa Eileen Stalheim. “Regulation and function of the cerebral cavernous malformation 2 protein.” 2009. Web. 22 Sep 2020.

Vancouver:

Crose LES. Regulation and function of the cerebral cavernous malformation 2 protein. [Internet] [Thesis]. University of North Carolina; 2009. [cited 2020 Sep 22]. Available from: https://cdr.lib.unc.edu/record/uuid:7a7bbdba-6b98-4bfa-bf8f-cf6fd10f2eb9.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Crose LES. Regulation and function of the cerebral cavernous malformation 2 protein. [Thesis]. University of North Carolina; 2009. Available from: https://cdr.lib.unc.edu/record/uuid:7a7bbdba-6b98-4bfa-bf8f-cf6fd10f2eb9

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

22. DeNofrio, Jan. Characterization of Platelet Function and Hemostasis in the Cib1-/- Mouse.

Degree: 2008, University of North Carolina

 Platelet activation initiates platelet-platelet aggregation, platelet-extracellular matrix interactions and thrombus formation. Platelet aggregation requires activation of the αIIbβ3 integrin, an event regulated by the integrin… (more)

Subjects/Keywords: School of Medicine; Curriculum in Genetics and Molecular Biology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

DeNofrio, J. (2008). Characterization of Platelet Function and Hemostasis in the Cib1-/- Mouse. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:654d2a52-5937-4ff8-905e-98b7c43d7438

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

DeNofrio, Jan. “Characterization of Platelet Function and Hemostasis in the Cib1-/- Mouse.” 2008. Thesis, University of North Carolina. Accessed September 22, 2020. https://cdr.lib.unc.edu/record/uuid:654d2a52-5937-4ff8-905e-98b7c43d7438.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

DeNofrio, Jan. “Characterization of Platelet Function and Hemostasis in the Cib1-/- Mouse.” 2008. Web. 22 Sep 2020.

Vancouver:

DeNofrio J. Characterization of Platelet Function and Hemostasis in the Cib1-/- Mouse. [Internet] [Thesis]. University of North Carolina; 2008. [cited 2020 Sep 22]. Available from: https://cdr.lib.unc.edu/record/uuid:654d2a52-5937-4ff8-905e-98b7c43d7438.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

DeNofrio J. Characterization of Platelet Function and Hemostasis in the Cib1-/- Mouse. [Thesis]. University of North Carolina; 2008. Available from: https://cdr.lib.unc.edu/record/uuid:654d2a52-5937-4ff8-905e-98b7c43d7438

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

.