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You searched for +publisher:"University of North Carolina" +contributor:("Duncan, Joseph"). Showing records 1 – 5 of 5 total matches.

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University of North Carolina

1. Ezekwe, Ejiofofr. Cellular Mechanisms of Staphylococcus aureus α-hemolysinmediated Activation of the NLRP3 Inflammasome.

Degree: Pharmacology, 2016, University of North Carolina

 Background: Staphylococcus aureus toxin, α-hemolysin, is secreted as a soluble monomer that forms a heptameric pore in the membranes of a range of host cell… (more)

Subjects/Keywords: School of Medicine; Department of Pharmacology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Ezekwe, E. (2016). Cellular Mechanisms of Staphylococcus aureus α-hemolysinmediated Activation of the NLRP3 Inflammasome. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:b2765954-f2a2-473f-8501-6a8bd05a8935

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ezekwe, Ejiofofr. “Cellular Mechanisms of Staphylococcus aureus α-hemolysinmediated Activation of the NLRP3 Inflammasome.” 2016. Thesis, University of North Carolina. Accessed October 29, 2020. https://cdr.lib.unc.edu/record/uuid:b2765954-f2a2-473f-8501-6a8bd05a8935.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ezekwe, Ejiofofr. “Cellular Mechanisms of Staphylococcus aureus α-hemolysinmediated Activation of the NLRP3 Inflammasome.” 2016. Web. 29 Oct 2020.

Vancouver:

Ezekwe E. Cellular Mechanisms of Staphylococcus aureus α-hemolysinmediated Activation of the NLRP3 Inflammasome. [Internet] [Thesis]. University of North Carolina; 2016. [cited 2020 Oct 29]. Available from: https://cdr.lib.unc.edu/record/uuid:b2765954-f2a2-473f-8501-6a8bd05a8935.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ezekwe E. Cellular Mechanisms of Staphylococcus aureus α-hemolysinmediated Activation of the NLRP3 Inflammasome. [Thesis]. University of North Carolina; 2016. Available from: https://cdr.lib.unc.edu/record/uuid:b2765954-f2a2-473f-8501-6a8bd05a8935

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

2. Maltez, Vivien. Inflammasomes and Cytotoxic Lymphocytes Clear Bacteria From Different Intracellular Niches.

Degree: Microbiology and Immunology, 2017, University of North Carolina

 Programmed cell death is a powerful anti-microbial defense. The innate immune system is our first line of defense against infection, and the induction of cell… (more)

Subjects/Keywords: School of Medicine; Department of Microbiology and Immunology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Maltez, V. (2017). Inflammasomes and Cytotoxic Lymphocytes Clear Bacteria From Different Intracellular Niches. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:0eb5d888-178a-4a7c-b6af-78c92db7e502

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Maltez, Vivien. “Inflammasomes and Cytotoxic Lymphocytes Clear Bacteria From Different Intracellular Niches.” 2017. Thesis, University of North Carolina. Accessed October 29, 2020. https://cdr.lib.unc.edu/record/uuid:0eb5d888-178a-4a7c-b6af-78c92db7e502.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Maltez, Vivien. “Inflammasomes and Cytotoxic Lymphocytes Clear Bacteria From Different Intracellular Niches.” 2017. Web. 29 Oct 2020.

Vancouver:

Maltez V. Inflammasomes and Cytotoxic Lymphocytes Clear Bacteria From Different Intracellular Niches. [Internet] [Thesis]. University of North Carolina; 2017. [cited 2020 Oct 29]. Available from: https://cdr.lib.unc.edu/record/uuid:0eb5d888-178a-4a7c-b6af-78c92db7e502.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Maltez V. Inflammasomes and Cytotoxic Lymphocytes Clear Bacteria From Different Intracellular Niches. [Thesis]. University of North Carolina; 2017. Available from: https://cdr.lib.unc.edu/record/uuid:0eb5d888-178a-4a7c-b6af-78c92db7e502

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

3. Melehani, Jason. Mechanisms and consequences of Staphylococcus aureus Leukocidin AB-mediated activation of the host NLRP3 inflammasome.

Degree: Pharmacology, 2016, University of North Carolina

 The NLRP3 inflammasome is a critical innate immune sensor implicated in the pathogenesis of dozens of infectious and non-infectious diseases. Activation of the NLRP3 inflammasome… (more)

Subjects/Keywords: School of Medicine; Department of Pharmacology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Melehani, J. (2016). Mechanisms and consequences of Staphylococcus aureus Leukocidin AB-mediated activation of the host NLRP3 inflammasome. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:f98e7df6-408e-40db-8202-8ab65dd52e01

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Melehani, Jason. “Mechanisms and consequences of Staphylococcus aureus Leukocidin AB-mediated activation of the host NLRP3 inflammasome.” 2016. Thesis, University of North Carolina. Accessed October 29, 2020. https://cdr.lib.unc.edu/record/uuid:f98e7df6-408e-40db-8202-8ab65dd52e01.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Melehani, Jason. “Mechanisms and consequences of Staphylococcus aureus Leukocidin AB-mediated activation of the host NLRP3 inflammasome.” 2016. Web. 29 Oct 2020.

Vancouver:

Melehani J. Mechanisms and consequences of Staphylococcus aureus Leukocidin AB-mediated activation of the host NLRP3 inflammasome. [Internet] [Thesis]. University of North Carolina; 2016. [cited 2020 Oct 29]. Available from: https://cdr.lib.unc.edu/record/uuid:f98e7df6-408e-40db-8202-8ab65dd52e01.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Melehani J. Mechanisms and consequences of Staphylococcus aureus Leukocidin AB-mediated activation of the host NLRP3 inflammasome. [Thesis]. University of North Carolina; 2016. Available from: https://cdr.lib.unc.edu/record/uuid:f98e7df6-408e-40db-8202-8ab65dd52e01

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

4. Perez-Chanona, Ernesto. Microbial signaling in the intestinal ischemia/reperfusion injury response.

Degree: Pharmacology, 2014, University of North Carolina

 The intestine is home to one of the most diverse and populous ecosystems on earth. The intestinal epithelium the single layer sheet of columnar cells… (more)

Subjects/Keywords: Pharmacology; Microbiology; Immunology; School of Medicine; Department of Pharmacology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Perez-Chanona, E. (2014). Microbial signaling in the intestinal ischemia/reperfusion injury response. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:19a719f1-4373-4d1e-8380-3944bd7f33cd

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Perez-Chanona, Ernesto. “Microbial signaling in the intestinal ischemia/reperfusion injury response.” 2014. Thesis, University of North Carolina. Accessed October 29, 2020. https://cdr.lib.unc.edu/record/uuid:19a719f1-4373-4d1e-8380-3944bd7f33cd.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Perez-Chanona, Ernesto. “Microbial signaling in the intestinal ischemia/reperfusion injury response.” 2014. Web. 29 Oct 2020.

Vancouver:

Perez-Chanona E. Microbial signaling in the intestinal ischemia/reperfusion injury response. [Internet] [Thesis]. University of North Carolina; 2014. [cited 2020 Oct 29]. Available from: https://cdr.lib.unc.edu/record/uuid:19a719f1-4373-4d1e-8380-3944bd7f33cd.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Perez-Chanona E. Microbial signaling in the intestinal ischemia/reperfusion injury response. [Thesis]. University of North Carolina; 2014. Available from: https://cdr.lib.unc.edu/record/uuid:19a719f1-4373-4d1e-8380-3944bd7f33cd

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

5. Knilans, Kayla. The role of lytic transglycosylases LtgA and LtgD in innate immune recognition and pathogenesis of Neisseria gonorrhoeae.

Degree: Pharmacology, 2014, University of North Carolina

 During growth, Neisseria gonorrhoeae releases inflammatory 1,6-anhydro peptidoglycan monomers through the action of lytic transglycosylases LtgA and LtgD. N. gonorrhoeae lacking LtgA and LtgD release… (more)

Subjects/Keywords: Microbiology; Immunology; School of Medicine; Department of Pharmacology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Knilans, K. (2014). The role of lytic transglycosylases LtgA and LtgD in innate immune recognition and pathogenesis of Neisseria gonorrhoeae. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:3004d864-4c35-4ef7-b34e-6f9d3f637eda

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Knilans, Kayla. “The role of lytic transglycosylases LtgA and LtgD in innate immune recognition and pathogenesis of Neisseria gonorrhoeae.” 2014. Thesis, University of North Carolina. Accessed October 29, 2020. https://cdr.lib.unc.edu/record/uuid:3004d864-4c35-4ef7-b34e-6f9d3f637eda.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Knilans, Kayla. “The role of lytic transglycosylases LtgA and LtgD in innate immune recognition and pathogenesis of Neisseria gonorrhoeae.” 2014. Web. 29 Oct 2020.

Vancouver:

Knilans K. The role of lytic transglycosylases LtgA and LtgD in innate immune recognition and pathogenesis of Neisseria gonorrhoeae. [Internet] [Thesis]. University of North Carolina; 2014. [cited 2020 Oct 29]. Available from: https://cdr.lib.unc.edu/record/uuid:3004d864-4c35-4ef7-b34e-6f9d3f637eda.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Knilans K. The role of lytic transglycosylases LtgA and LtgD in innate immune recognition and pathogenesis of Neisseria gonorrhoeae. [Thesis]. University of North Carolina; 2014. Available from: https://cdr.lib.unc.edu/record/uuid:3004d864-4c35-4ef7-b34e-6f9d3f637eda

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

.