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You searched for +publisher:"University of North Carolina" +contributor:("Deshmukh, Mohanish"). Showing records 1 – 22 of 22 total matches.

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University of North Carolina

1. Cusack, Corey Leigh. The molecular intersection between axon-specific pruning and neuronal apoptosis.

Degree: 2014, University of North Carolina

 Neurons can activate pathways to either destroy the whole cell via apoptosis or specifically degenerate only the axon. Axon-specific degeneration, also known as pruning, is… (more)

Subjects/Keywords: School of Medicine; UNC Neuroscience Center; Neuroscience Curriculum

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APA (6th Edition):

Cusack, C. L. (2014). The molecular intersection between axon-specific pruning and neuronal apoptosis. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:4f960235-bca7-4837-95aa-f4287e7f45a9

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Cusack, Corey Leigh. “The molecular intersection between axon-specific pruning and neuronal apoptosis.” 2014. Thesis, University of North Carolina. Accessed September 19, 2020. https://cdr.lib.unc.edu/record/uuid:4f960235-bca7-4837-95aa-f4287e7f45a9.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Cusack, Corey Leigh. “The molecular intersection between axon-specific pruning and neuronal apoptosis.” 2014. Web. 19 Sep 2020.

Vancouver:

Cusack CL. The molecular intersection between axon-specific pruning and neuronal apoptosis. [Internet] [Thesis]. University of North Carolina; 2014. [cited 2020 Sep 19]. Available from: https://cdr.lib.unc.edu/record/uuid:4f960235-bca7-4837-95aa-f4287e7f45a9.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Cusack CL. The molecular intersection between axon-specific pruning and neuronal apoptosis. [Thesis]. University of North Carolina; 2014. Available from: https://cdr.lib.unc.edu/record/uuid:4f960235-bca7-4837-95aa-f4287e7f45a9

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

2. Knight, Elizabeth R. Immune Molecules Regulate Medulloblastoma and Neuronal Apoptosis.

Degree: 2013, University of North Carolina

 While the brain has long been considered an immunoprivileged region, recent research reveals that immune genes play important roles in neurons and the nervous system.… (more)

Subjects/Keywords: School of Medicine; UNC Neuroscience Center; Neuroscience Curriculum

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APA (6th Edition):

Knight, E. R. (2013). Immune Molecules Regulate Medulloblastoma and Neuronal Apoptosis. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:03d3964d-b3da-4170-b7b2-0dbafbaf6f90

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Knight, Elizabeth R. “Immune Molecules Regulate Medulloblastoma and Neuronal Apoptosis.” 2013. Thesis, University of North Carolina. Accessed September 19, 2020. https://cdr.lib.unc.edu/record/uuid:03d3964d-b3da-4170-b7b2-0dbafbaf6f90.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Knight, Elizabeth R. “Immune Molecules Regulate Medulloblastoma and Neuronal Apoptosis.” 2013. Web. 19 Sep 2020.

Vancouver:

Knight ER. Immune Molecules Regulate Medulloblastoma and Neuronal Apoptosis. [Internet] [Thesis]. University of North Carolina; 2013. [cited 2020 Sep 19]. Available from: https://cdr.lib.unc.edu/record/uuid:03d3964d-b3da-4170-b7b2-0dbafbaf6f90.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Knight ER. Immune Molecules Regulate Medulloblastoma and Neuronal Apoptosis. [Thesis]. University of North Carolina; 2013. Available from: https://cdr.lib.unc.edu/record/uuid:03d3964d-b3da-4170-b7b2-0dbafbaf6f90

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

3. Kole, Adam Julian. Regulation of Neuronal Maturation and Apoptosis by Micrornas.

Degree: Cell Biology and Physiology, 2011, University of North Carolina

 Apoptosis, or programmed cell death, is a conserved and highly regulated pathway by which cells die. This form of cell death is important for normal… (more)

Subjects/Keywords: School of Medicine; Department of Cell Biology and Physiology

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APA (6th Edition):

Kole, A. J. (2011). Regulation of Neuronal Maturation and Apoptosis by Micrornas. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:cdb02870-d3aa-49af-a4a1-660b5cf76a73

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kole, Adam Julian. “Regulation of Neuronal Maturation and Apoptosis by Micrornas.” 2011. Thesis, University of North Carolina. Accessed September 19, 2020. https://cdr.lib.unc.edu/record/uuid:cdb02870-d3aa-49af-a4a1-660b5cf76a73.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kole, Adam Julian. “Regulation of Neuronal Maturation and Apoptosis by Micrornas.” 2011. Web. 19 Sep 2020.

Vancouver:

Kole AJ. Regulation of Neuronal Maturation and Apoptosis by Micrornas. [Internet] [Thesis]. University of North Carolina; 2011. [cited 2020 Sep 19]. Available from: https://cdr.lib.unc.edu/record/uuid:cdb02870-d3aa-49af-a4a1-660b5cf76a73.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kole AJ. Regulation of Neuronal Maturation and Apoptosis by Micrornas. [Thesis]. University of North Carolina; 2011. Available from: https://cdr.lib.unc.edu/record/uuid:cdb02870-d3aa-49af-a4a1-660b5cf76a73

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

4. Harrell, Jessica R. How diverse cells position themselves in an embryo: variations on a common cytoskeletal theme.

Degree: Cell Biology and Physiology, 2010, University of North Carolina

 Understanding morphogenesis, the spatial and temporal distribution of cells during development of an organism, is a key goal in studies in developmental biology. Throughout diverse… (more)

Subjects/Keywords: School of Medicine; Department of Cell Biology and Physiology

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APA (6th Edition):

Harrell, J. R. (2010). How diverse cells position themselves in an embryo: variations on a common cytoskeletal theme. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:e5f2ee99-86c7-47b1-ac7f-60201771c102

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Harrell, Jessica R. “How diverse cells position themselves in an embryo: variations on a common cytoskeletal theme.” 2010. Thesis, University of North Carolina. Accessed September 19, 2020. https://cdr.lib.unc.edu/record/uuid:e5f2ee99-86c7-47b1-ac7f-60201771c102.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Harrell, Jessica R. “How diverse cells position themselves in an embryo: variations on a common cytoskeletal theme.” 2010. Web. 19 Sep 2020.

Vancouver:

Harrell JR. How diverse cells position themselves in an embryo: variations on a common cytoskeletal theme. [Internet] [Thesis]. University of North Carolina; 2010. [cited 2020 Sep 19]. Available from: https://cdr.lib.unc.edu/record/uuid:e5f2ee99-86c7-47b1-ac7f-60201771c102.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Harrell JR. How diverse cells position themselves in an embryo: variations on a common cytoskeletal theme. [Thesis]. University of North Carolina; 2010. Available from: https://cdr.lib.unc.edu/record/uuid:e5f2ee99-86c7-47b1-ac7f-60201771c102

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

5. Williams, Kimberly. NEUROTROPHIN-CHEMOKINE RECEPTOR INTERACTIONS ON MACROPHAGES REGULATE HIV NEUROPATHOGENESIS.

Degree: 2015, University of North Carolina

 Invading perivascular macrophages and microglial cells play a pivotal role in the neuropathogenesis of Human Immunodeficiency Virus (HIV) associated neurological disorders. As with many neurodegenerative… (more)

Subjects/Keywords: Neurosciences; Immunology; School of Medicine; UNC Neuroscience Center; Neuroscience Curriculum

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APA (6th Edition):

Williams, K. (2015). NEUROTROPHIN-CHEMOKINE RECEPTOR INTERACTIONS ON MACROPHAGES REGULATE HIV NEUROPATHOGENESIS. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:79d4ec87-ae10-4766-b524-40c7700a3f7e

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Williams, Kimberly. “NEUROTROPHIN-CHEMOKINE RECEPTOR INTERACTIONS ON MACROPHAGES REGULATE HIV NEUROPATHOGENESIS.” 2015. Thesis, University of North Carolina. Accessed September 19, 2020. https://cdr.lib.unc.edu/record/uuid:79d4ec87-ae10-4766-b524-40c7700a3f7e.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Williams, Kimberly. “NEUROTROPHIN-CHEMOKINE RECEPTOR INTERACTIONS ON MACROPHAGES REGULATE HIV NEUROPATHOGENESIS.” 2015. Web. 19 Sep 2020.

Vancouver:

Williams K. NEUROTROPHIN-CHEMOKINE RECEPTOR INTERACTIONS ON MACROPHAGES REGULATE HIV NEUROPATHOGENESIS. [Internet] [Thesis]. University of North Carolina; 2015. [cited 2020 Sep 19]. Available from: https://cdr.lib.unc.edu/record/uuid:79d4ec87-ae10-4766-b524-40c7700a3f7e.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Williams K. NEUROTROPHIN-CHEMOKINE RECEPTOR INTERACTIONS ON MACROPHAGES REGULATE HIV NEUROPATHOGENESIS. [Thesis]. University of North Carolina; 2015. Available from: https://cdr.lib.unc.edu/record/uuid:79d4ec87-ae10-4766-b524-40c7700a3f7e

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

6. Jones, Stephen. Nanoparticle Clearance is Governed by Th1/Th2 Immunity and Strain Background.

Degree: Cell Biology and Physiology, 2015, University of North Carolina

 Extended circulation of nanoparticles in blood is essential for most clinical applications. Nanoparticles are rapidly cleared by cells of the mononuclear phagocyte system (MPS). Approaches… (more)

Subjects/Keywords: Cytology; School of Medicine; Department of Cell Biology and Physiology

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APA (6th Edition):

Jones, S. (2015). Nanoparticle Clearance is Governed by Th1/Th2 Immunity and Strain Background. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:5180f990-8a54-4ac9-aec1-e00cd03b9ab3

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Jones, Stephen. “Nanoparticle Clearance is Governed by Th1/Th2 Immunity and Strain Background.” 2015. Thesis, University of North Carolina. Accessed September 19, 2020. https://cdr.lib.unc.edu/record/uuid:5180f990-8a54-4ac9-aec1-e00cd03b9ab3.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Jones, Stephen. “Nanoparticle Clearance is Governed by Th1/Th2 Immunity and Strain Background.” 2015. Web. 19 Sep 2020.

Vancouver:

Jones S. Nanoparticle Clearance is Governed by Th1/Th2 Immunity and Strain Background. [Internet] [Thesis]. University of North Carolina; 2015. [cited 2020 Sep 19]. Available from: https://cdr.lib.unc.edu/record/uuid:5180f990-8a54-4ac9-aec1-e00cd03b9ab3.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Jones S. Nanoparticle Clearance is Governed by Th1/Th2 Immunity and Strain Background. [Thesis]. University of North Carolina; 2015. Available from: https://cdr.lib.unc.edu/record/uuid:5180f990-8a54-4ac9-aec1-e00cd03b9ab3

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

7. Tech, Katherine. The intersection between aerobic glycolysis, cerebellar neurogenesis, and medulloblastoma.

Degree: Biomedical Engineering, 2016, University of North Carolina

 Aerobic glycolysis supports proliferation in development and cancer through unresolved mechanisms. Aerobic glycolysis, which supports the generation of biomass, is integral to cerebellar granule neuron… (more)

Subjects/Keywords: School of Medicine; UNC/NCSU Joint Department of Biomedical Engineering

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APA (6th Edition):

Tech, K. (2016). The intersection between aerobic glycolysis, cerebellar neurogenesis, and medulloblastoma. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:30193238-2506-4637-a00b-79c1249138b1

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Tech, Katherine. “The intersection between aerobic glycolysis, cerebellar neurogenesis, and medulloblastoma.” 2016. Thesis, University of North Carolina. Accessed September 19, 2020. https://cdr.lib.unc.edu/record/uuid:30193238-2506-4637-a00b-79c1249138b1.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Tech, Katherine. “The intersection between aerobic glycolysis, cerebellar neurogenesis, and medulloblastoma.” 2016. Web. 19 Sep 2020.

Vancouver:

Tech K. The intersection between aerobic glycolysis, cerebellar neurogenesis, and medulloblastoma. [Internet] [Thesis]. University of North Carolina; 2016. [cited 2020 Sep 19]. Available from: https://cdr.lib.unc.edu/record/uuid:30193238-2506-4637-a00b-79c1249138b1.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Tech K. The intersection between aerobic glycolysis, cerebellar neurogenesis, and medulloblastoma. [Thesis]. University of North Carolina; 2016. Available from: https://cdr.lib.unc.edu/record/uuid:30193238-2506-4637-a00b-79c1249138b1

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

8. Lang, Patrick. ATR is a Novel Therapeutic Target for Medulloblastoma Identified by its Role in Cerebellar Development.

Degree: Cell Biology and Physiology, 2017, University of North Carolina

 Microcephaly and brain tumors can both arise as a consequence of dysregulated expansion of neural progenitor cells. Inadequate progenitor proliferation, premature cell cycle exit, and… (more)

Subjects/Keywords: School of Medicine; Department of Cell Biology and Physiology

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APA (6th Edition):

Lang, P. (2017). ATR is a Novel Therapeutic Target for Medulloblastoma Identified by its Role in Cerebellar Development. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:08fa1633-1467-4166-81f0-2aabbd239f0f

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lang, Patrick. “ATR is a Novel Therapeutic Target for Medulloblastoma Identified by its Role in Cerebellar Development.” 2017. Thesis, University of North Carolina. Accessed September 19, 2020. https://cdr.lib.unc.edu/record/uuid:08fa1633-1467-4166-81f0-2aabbd239f0f.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lang, Patrick. “ATR is a Novel Therapeutic Target for Medulloblastoma Identified by its Role in Cerebellar Development.” 2017. Web. 19 Sep 2020.

Vancouver:

Lang P. ATR is a Novel Therapeutic Target for Medulloblastoma Identified by its Role in Cerebellar Development. [Internet] [Thesis]. University of North Carolina; 2017. [cited 2020 Sep 19]. Available from: https://cdr.lib.unc.edu/record/uuid:08fa1633-1467-4166-81f0-2aabbd239f0f.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lang P. ATR is a Novel Therapeutic Target for Medulloblastoma Identified by its Role in Cerebellar Development. [Thesis]. University of North Carolina; 2017. Available from: https://cdr.lib.unc.edu/record/uuid:08fa1633-1467-4166-81f0-2aabbd239f0f

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

9. Nakamura, Ayumi. MOLECULAR MECHANISMS IN THE DEVELOPING AND MATURING BRAIN.

Degree: 2016, University of North Carolina

 Neurons, unlike other cell types, persist throughout the entire lifespan of an organism. Additionally, neurons use multiple anti-apoptotic brakes during different stages of their life… (more)

Subjects/Keywords: School of Medicine; UNC Neuroscience Center; Neuroscience Curriculum

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APA (6th Edition):

Nakamura, A. (2016). MOLECULAR MECHANISMS IN THE DEVELOPING AND MATURING BRAIN. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:fbd36d57-8943-476a-a98e-5dbd6094ba66

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Nakamura, Ayumi. “MOLECULAR MECHANISMS IN THE DEVELOPING AND MATURING BRAIN.” 2016. Thesis, University of North Carolina. Accessed September 19, 2020. https://cdr.lib.unc.edu/record/uuid:fbd36d57-8943-476a-a98e-5dbd6094ba66.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Nakamura, Ayumi. “MOLECULAR MECHANISMS IN THE DEVELOPING AND MATURING BRAIN.” 2016. Web. 19 Sep 2020.

Vancouver:

Nakamura A. MOLECULAR MECHANISMS IN THE DEVELOPING AND MATURING BRAIN. [Internet] [Thesis]. University of North Carolina; 2016. [cited 2020 Sep 19]. Available from: https://cdr.lib.unc.edu/record/uuid:fbd36d57-8943-476a-a98e-5dbd6094ba66.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Nakamura A. MOLECULAR MECHANISMS IN THE DEVELOPING AND MATURING BRAIN. [Thesis]. University of North Carolina; 2016. Available from: https://cdr.lib.unc.edu/record/uuid:fbd36d57-8943-476a-a98e-5dbd6094ba66

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

10. Rinkenbaugh, Amanda. Roles of the NF-kappaB Pathway in Glioblastoma Stem Cells and Chordoma.

Degree: Pathology and Laboratory Medicine, 2016, University of North Carolina

 The NF-κB pathway consists of a family of five transcription factors: RelA/p65, RelB, c-Rel, p100/p52, and p105/p50. Originally discovered for its involvement in inflammation and… (more)

Subjects/Keywords: School of Medicine; Department of Pathology and Laboratory Medicine

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APA (6th Edition):

Rinkenbaugh, A. (2016). Roles of the NF-kappaB Pathway in Glioblastoma Stem Cells and Chordoma. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:4046c94e-b8aa-4bba-93e0-ac8d1490b55d

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Rinkenbaugh, Amanda. “Roles of the NF-kappaB Pathway in Glioblastoma Stem Cells and Chordoma.” 2016. Thesis, University of North Carolina. Accessed September 19, 2020. https://cdr.lib.unc.edu/record/uuid:4046c94e-b8aa-4bba-93e0-ac8d1490b55d.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Rinkenbaugh, Amanda. “Roles of the NF-kappaB Pathway in Glioblastoma Stem Cells and Chordoma.” 2016. Web. 19 Sep 2020.

Vancouver:

Rinkenbaugh A. Roles of the NF-kappaB Pathway in Glioblastoma Stem Cells and Chordoma. [Internet] [Thesis]. University of North Carolina; 2016. [cited 2020 Sep 19]. Available from: https://cdr.lib.unc.edu/record/uuid:4046c94e-b8aa-4bba-93e0-ac8d1490b55d.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Rinkenbaugh A. Roles of the NF-kappaB Pathway in Glioblastoma Stem Cells and Chordoma. [Thesis]. University of North Carolina; 2016. Available from: https://cdr.lib.unc.edu/record/uuid:4046c94e-b8aa-4bba-93e0-ac8d1490b55d

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

11. Orgel, Kelly. Advances in Treatments and Animal Models of Peanut Allergy.

Degree: Cell Biology and Physiology, 2018, University of North Carolina

 Food allergies are a growing health concern affecting approximately 6-8% of the US population. In particular, peanut allergy has an estimated prevalence of greater than… (more)

Subjects/Keywords: School of Medicine; Department of Cell Biology and Physiology

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APA (6th Edition):

Orgel, K. (2018). Advances in Treatments and Animal Models of Peanut Allergy. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:d7fad2d6-7f70-4e8a-9a06-f50aa2902145

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Orgel, Kelly. “Advances in Treatments and Animal Models of Peanut Allergy.” 2018. Thesis, University of North Carolina. Accessed September 19, 2020. https://cdr.lib.unc.edu/record/uuid:d7fad2d6-7f70-4e8a-9a06-f50aa2902145.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Orgel, Kelly. “Advances in Treatments and Animal Models of Peanut Allergy.” 2018. Web. 19 Sep 2020.

Vancouver:

Orgel K. Advances in Treatments and Animal Models of Peanut Allergy. [Internet] [Thesis]. University of North Carolina; 2018. [cited 2020 Sep 19]. Available from: https://cdr.lib.unc.edu/record/uuid:d7fad2d6-7f70-4e8a-9a06-f50aa2902145.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Orgel K. Advances in Treatments and Animal Models of Peanut Allergy. [Thesis]. University of North Carolina; 2018. Available from: https://cdr.lib.unc.edu/record/uuid:d7fad2d6-7f70-4e8a-9a06-f50aa2902145

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

12. Annis, Ryan. RESTRICTION OF APOPTOSIS IN MATURE NEURONS BY MULTIPLE REDUNDANT BRAKES.

Degree: 2016, University of North Carolina

 Apoptotic cell death is a key part of normal nervous system development, as a process that allows for the controlled removal of developing neurons that… (more)

Subjects/Keywords: School of Medicine; UNC Neuroscience Center; Neuroscience Curriculum

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APA (6th Edition):

Annis, R. (2016). RESTRICTION OF APOPTOSIS IN MATURE NEURONS BY MULTIPLE REDUNDANT BRAKES. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:c8a1b623-2537-40da-8f74-110f1874d7ed

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Annis, Ryan. “RESTRICTION OF APOPTOSIS IN MATURE NEURONS BY MULTIPLE REDUNDANT BRAKES.” 2016. Thesis, University of North Carolina. Accessed September 19, 2020. https://cdr.lib.unc.edu/record/uuid:c8a1b623-2537-40da-8f74-110f1874d7ed.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Annis, Ryan. “RESTRICTION OF APOPTOSIS IN MATURE NEURONS BY MULTIPLE REDUNDANT BRAKES.” 2016. Web. 19 Sep 2020.

Vancouver:

Annis R. RESTRICTION OF APOPTOSIS IN MATURE NEURONS BY MULTIPLE REDUNDANT BRAKES. [Internet] [Thesis]. University of North Carolina; 2016. [cited 2020 Sep 19]. Available from: https://cdr.lib.unc.edu/record/uuid:c8a1b623-2537-40da-8f74-110f1874d7ed.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Annis R. RESTRICTION OF APOPTOSIS IN MATURE NEURONS BY MULTIPLE REDUNDANT BRAKES. [Thesis]. University of North Carolina; 2016. Available from: https://cdr.lib.unc.edu/record/uuid:c8a1b623-2537-40da-8f74-110f1874d7ed

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

13. Taus, Patrick. FATE1: Contributor to Tumor Cell Fitness and Example of an Oncogene-Activated Cancer Testis Antigen.

Degree: Pharmacology, 2016, University of North Carolina

 Cancer Testis Antigens (CTAs) are a group of genes defined by their unique expression pattern, normally expressed nearly exclusively in gametogenic tissues they are also… (more)

Subjects/Keywords: School of Medicine; Department of Pharmacology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Taus, P. (2016). FATE1: Contributor to Tumor Cell Fitness and Example of an Oncogene-Activated Cancer Testis Antigen. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:a51f678f-bfa2-487e-b60e-d2e64c321185

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Taus, Patrick. “FATE1: Contributor to Tumor Cell Fitness and Example of an Oncogene-Activated Cancer Testis Antigen.” 2016. Thesis, University of North Carolina. Accessed September 19, 2020. https://cdr.lib.unc.edu/record/uuid:a51f678f-bfa2-487e-b60e-d2e64c321185.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Taus, Patrick. “FATE1: Contributor to Tumor Cell Fitness and Example of an Oncogene-Activated Cancer Testis Antigen.” 2016. Web. 19 Sep 2020.

Vancouver:

Taus P. FATE1: Contributor to Tumor Cell Fitness and Example of an Oncogene-Activated Cancer Testis Antigen. [Internet] [Thesis]. University of North Carolina; 2016. [cited 2020 Sep 19]. Available from: https://cdr.lib.unc.edu/record/uuid:a51f678f-bfa2-487e-b60e-d2e64c321185.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Taus P. FATE1: Contributor to Tumor Cell Fitness and Example of an Oncogene-Activated Cancer Testis Antigen. [Thesis]. University of North Carolina; 2016. Available from: https://cdr.lib.unc.edu/record/uuid:a51f678f-bfa2-487e-b60e-d2e64c321185

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

14. Winkle, Cortney. STAYING TRIM: HOW TRIM9 CONSTRAINS NEURONAL CELL MORPHOLOGY TO REGULATE CONNECTIVITY.

Degree: 2016, University of North Carolina

 The connectivity of the nervous system is established during development as axons branch and project to synaptic targets. The extracellular axon guidance cue Netrin-1 and… (more)

Subjects/Keywords: School of Medicine; UNC Neuroscience Center; Neuroscience Curriculum

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Winkle, C. (2016). STAYING TRIM: HOW TRIM9 CONSTRAINS NEURONAL CELL MORPHOLOGY TO REGULATE CONNECTIVITY. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:14b7ba01-bca3-486e-833a-9b4ba0ef004b

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Winkle, Cortney. “STAYING TRIM: HOW TRIM9 CONSTRAINS NEURONAL CELL MORPHOLOGY TO REGULATE CONNECTIVITY.” 2016. Thesis, University of North Carolina. Accessed September 19, 2020. https://cdr.lib.unc.edu/record/uuid:14b7ba01-bca3-486e-833a-9b4ba0ef004b.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Winkle, Cortney. “STAYING TRIM: HOW TRIM9 CONSTRAINS NEURONAL CELL MORPHOLOGY TO REGULATE CONNECTIVITY.” 2016. Web. 19 Sep 2020.

Vancouver:

Winkle C. STAYING TRIM: HOW TRIM9 CONSTRAINS NEURONAL CELL MORPHOLOGY TO REGULATE CONNECTIVITY. [Internet] [Thesis]. University of North Carolina; 2016. [cited 2020 Sep 19]. Available from: https://cdr.lib.unc.edu/record/uuid:14b7ba01-bca3-486e-833a-9b4ba0ef004b.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Winkle C. STAYING TRIM: HOW TRIM9 CONSTRAINS NEURONAL CELL MORPHOLOGY TO REGULATE CONNECTIVITY. [Thesis]. University of North Carolina; 2016. Available from: https://cdr.lib.unc.edu/record/uuid:14b7ba01-bca3-486e-833a-9b4ba0ef004b

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

15. Annis, Ryan. RESTRICTION OF APOPTOSIS IN MATURE NEURONS BY MULTIPLE REDUNDANT BRAKES.

Degree: 2016, University of North Carolina

 Apoptotic cell death is a key part of normal nervous system development, as a process that allows for the controlled removal of developing neurons that… (more)

Subjects/Keywords: School of Medicine; UNC Neuroscience Center; Neuroscience Curriculum

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Annis, R. (2016). RESTRICTION OF APOPTOSIS IN MATURE NEURONS BY MULTIPLE REDUNDANT BRAKES. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:be6c26e5-f1ce-42bd-b753-4d52ccddd099

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Annis, Ryan. “RESTRICTION OF APOPTOSIS IN MATURE NEURONS BY MULTIPLE REDUNDANT BRAKES.” 2016. Thesis, University of North Carolina. Accessed September 19, 2020. https://cdr.lib.unc.edu/record/uuid:be6c26e5-f1ce-42bd-b753-4d52ccddd099.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Annis, Ryan. “RESTRICTION OF APOPTOSIS IN MATURE NEURONS BY MULTIPLE REDUNDANT BRAKES.” 2016. Web. 19 Sep 2020.

Vancouver:

Annis R. RESTRICTION OF APOPTOSIS IN MATURE NEURONS BY MULTIPLE REDUNDANT BRAKES. [Internet] [Thesis]. University of North Carolina; 2016. [cited 2020 Sep 19]. Available from: https://cdr.lib.unc.edu/record/uuid:be6c26e5-f1ce-42bd-b753-4d52ccddd099.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Annis R. RESTRICTION OF APOPTOSIS IN MATURE NEURONS BY MULTIPLE REDUNDANT BRAKES. [Thesis]. University of North Carolina; 2016. Available from: https://cdr.lib.unc.edu/record/uuid:be6c26e5-f1ce-42bd-b753-4d52ccddd099

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

16. Bonder, Daniel. Astrocytic Gq-GPCR-Linked IP3R-Dependent Ca2+ Signaling Does Not Mediate Neurovascular Coupling in Mouse Visual Cortex in vivo.

Degree: 2014, University of North Carolina

 Local blood flow is modulated in response to changing patterns of neuronal activity (Roy and Sherrington, 1890), a process termed neurovascular coupling. It has been… (more)

Subjects/Keywords: Neurosciences; Cytology; School of Medicine; UNC Neuroscience Center; Neuroscience Curriculum

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APA (6th Edition):

Bonder, D. (2014). Astrocytic Gq-GPCR-Linked IP3R-Dependent Ca2+ Signaling Does Not Mediate Neurovascular Coupling in Mouse Visual Cortex in vivo. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:780de0e7-bf00-411b-88b7-4ca854b38cca

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Bonder, Daniel. “Astrocytic Gq-GPCR-Linked IP3R-Dependent Ca2+ Signaling Does Not Mediate Neurovascular Coupling in Mouse Visual Cortex in vivo.” 2014. Thesis, University of North Carolina. Accessed September 19, 2020. https://cdr.lib.unc.edu/record/uuid:780de0e7-bf00-411b-88b7-4ca854b38cca.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Bonder, Daniel. “Astrocytic Gq-GPCR-Linked IP3R-Dependent Ca2+ Signaling Does Not Mediate Neurovascular Coupling in Mouse Visual Cortex in vivo.” 2014. Web. 19 Sep 2020.

Vancouver:

Bonder D. Astrocytic Gq-GPCR-Linked IP3R-Dependent Ca2+ Signaling Does Not Mediate Neurovascular Coupling in Mouse Visual Cortex in vivo. [Internet] [Thesis]. University of North Carolina; 2014. [cited 2020 Sep 19]. Available from: https://cdr.lib.unc.edu/record/uuid:780de0e7-bf00-411b-88b7-4ca854b38cca.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Bonder D. Astrocytic Gq-GPCR-Linked IP3R-Dependent Ca2+ Signaling Does Not Mediate Neurovascular Coupling in Mouse Visual Cortex in vivo. [Thesis]. University of North Carolina; 2014. Available from: https://cdr.lib.unc.edu/record/uuid:780de0e7-bf00-411b-88b7-4ca854b38cca

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

17. Tsao, Li-Chung. Virus-Host Interactions in HIV-1 Pathogenesis and Viral Immune Evasion.

Degree: 2016, University of North Carolina

 Human viruses such as the Human Immunodeficiency virus (HIV-1) causes chronic diseases and is a major public health concern. My dissertation focuses on the molecular… (more)

Subjects/Keywords: School of Medicine; Curriculum in Genetics and Molecular Biology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Tsao, L. (2016). Virus-Host Interactions in HIV-1 Pathogenesis and Viral Immune Evasion. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:d21f72b1-b423-4f34-a6f3-a474cb2b261d

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Tsao, Li-Chung. “Virus-Host Interactions in HIV-1 Pathogenesis and Viral Immune Evasion.” 2016. Thesis, University of North Carolina. Accessed September 19, 2020. https://cdr.lib.unc.edu/record/uuid:d21f72b1-b423-4f34-a6f3-a474cb2b261d.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Tsao, Li-Chung. “Virus-Host Interactions in HIV-1 Pathogenesis and Viral Immune Evasion.” 2016. Web. 19 Sep 2020.

Vancouver:

Tsao L. Virus-Host Interactions in HIV-1 Pathogenesis and Viral Immune Evasion. [Internet] [Thesis]. University of North Carolina; 2016. [cited 2020 Sep 19]. Available from: https://cdr.lib.unc.edu/record/uuid:d21f72b1-b423-4f34-a6f3-a474cb2b261d.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Tsao L. Virus-Host Interactions in HIV-1 Pathogenesis and Viral Immune Evasion. [Thesis]. University of North Carolina; 2016. Available from: https://cdr.lib.unc.edu/record/uuid:d21f72b1-b423-4f34-a6f3-a474cb2b261d

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

18. Smith, Michelle I. Regulation of apoptosis in postmitotic cells.

Degree: Cell Biology and Physiology, 2008, University of North Carolina

 Apoptosis is a genetically regulated evolutionarily conserved form of cell death. During the intrinsic pathway of apoptosis, the Bcl-2 family of proteins regulates the release… (more)

Subjects/Keywords: School of Medicine; Department of Cell Biology and Physiology

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APA (6th Edition):

Smith, M. I. (2008). Regulation of apoptosis in postmitotic cells. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:8d747944-10e6-40dc-bdd4-0fe99d36a791

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Smith, Michelle I. “Regulation of apoptosis in postmitotic cells.” 2008. Thesis, University of North Carolina. Accessed September 19, 2020. https://cdr.lib.unc.edu/record/uuid:8d747944-10e6-40dc-bdd4-0fe99d36a791.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Smith, Michelle I. “Regulation of apoptosis in postmitotic cells.” 2008. Web. 19 Sep 2020.

Vancouver:

Smith MI. Regulation of apoptosis in postmitotic cells. [Internet] [Thesis]. University of North Carolina; 2008. [cited 2020 Sep 19]. Available from: https://cdr.lib.unc.edu/record/uuid:8d747944-10e6-40dc-bdd4-0fe99d36a791.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Smith MI. Regulation of apoptosis in postmitotic cells. [Thesis]. University of North Carolina; 2008. Available from: https://cdr.lib.unc.edu/record/uuid:8d747944-10e6-40dc-bdd4-0fe99d36a791

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

19. Huang, Yolanda Y. Regulation of apoptosis at the point of cytochrome c in disease models.

Degree: Cell Biology and Physiology, 2009, University of North Carolina

 Apoptosis is a tightly regulated genetic process which governs the ability of a cell to undergo death under various developmental and pathological stimuli. The apoptotic… (more)

Subjects/Keywords: School of Medicine; Department of Cell Biology and Physiology

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APA (6th Edition):

Huang, Y. Y. (2009). Regulation of apoptosis at the point of cytochrome c in disease models. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:4e2d95b9-08bd-4fa9-ad1e-abd0588a1850

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Huang, Yolanda Y. “Regulation of apoptosis at the point of cytochrome c in disease models.” 2009. Thesis, University of North Carolina. Accessed September 19, 2020. https://cdr.lib.unc.edu/record/uuid:4e2d95b9-08bd-4fa9-ad1e-abd0588a1850.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Huang, Yolanda Y. “Regulation of apoptosis at the point of cytochrome c in disease models.” 2009. Web. 19 Sep 2020.

Vancouver:

Huang YY. Regulation of apoptosis at the point of cytochrome c in disease models. [Internet] [Thesis]. University of North Carolina; 2009. [cited 2020 Sep 19]. Available from: https://cdr.lib.unc.edu/record/uuid:4e2d95b9-08bd-4fa9-ad1e-abd0588a1850.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Huang YY. Regulation of apoptosis at the point of cytochrome c in disease models. [Thesis]. University of North Carolina; 2009. Available from: https://cdr.lib.unc.edu/record/uuid:4e2d95b9-08bd-4fa9-ad1e-abd0588a1850

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

20. Vaughn, Allyson Evans. Postmitochondrial regulation of apoptosis in neurons and cancer cells.

Degree: Cell Biology and Physiology, 2007, University of North Carolina

 The apoptotic pathway is a genetically conserved, energetic process that is essential for the development and homeostasis of organisms. Aberrant apoptosis, however, can result in… (more)

Subjects/Keywords: School of Medicine; Department of Cell Biology and Physiology

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APA (6th Edition):

Vaughn, A. E. (2007). Postmitochondrial regulation of apoptosis in neurons and cancer cells. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:6e4b5a6d-1395-481c-a82d-23d947e69b67

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Vaughn, Allyson Evans. “Postmitochondrial regulation of apoptosis in neurons and cancer cells.” 2007. Thesis, University of North Carolina. Accessed September 19, 2020. https://cdr.lib.unc.edu/record/uuid:6e4b5a6d-1395-481c-a82d-23d947e69b67.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Vaughn, Allyson Evans. “Postmitochondrial regulation of apoptosis in neurons and cancer cells.” 2007. Web. 19 Sep 2020.

Vancouver:

Vaughn AE. Postmitochondrial regulation of apoptosis in neurons and cancer cells. [Internet] [Thesis]. University of North Carolina; 2007. [cited 2020 Sep 19]. Available from: https://cdr.lib.unc.edu/record/uuid:6e4b5a6d-1395-481c-a82d-23d947e69b67.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Vaughn AE. Postmitochondrial regulation of apoptosis in neurons and cancer cells. [Thesis]. University of North Carolina; 2007. Available from: https://cdr.lib.unc.edu/record/uuid:6e4b5a6d-1395-481c-a82d-23d947e69b67

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

21. Powell, Ashton. Molecular Mechanisms Specifying the Topography of Thalamocortical Projectionsin the Ventral Telencephalon.

Degree: 2008, University of North Carolina

 The topographic projection of each thalamic nucleus to a unique set of cortical areas underlies the input specificity characterizing each sensory modality. Although the importance… (more)

Subjects/Keywords: School of Medicine; UNC Neuroscience Center; Neuroscience Curriculum

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Powell, A. (2008). Molecular Mechanisms Specifying the Topography of Thalamocortical Projectionsin the Ventral Telencephalon. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:c6d7a3a6-9267-4049-a631-1027e437b6d3

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Powell, Ashton. “Molecular Mechanisms Specifying the Topography of Thalamocortical Projectionsin the Ventral Telencephalon.” 2008. Thesis, University of North Carolina. Accessed September 19, 2020. https://cdr.lib.unc.edu/record/uuid:c6d7a3a6-9267-4049-a631-1027e437b6d3.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Powell, Ashton. “Molecular Mechanisms Specifying the Topography of Thalamocortical Projectionsin the Ventral Telencephalon.” 2008. Web. 19 Sep 2020.

Vancouver:

Powell A. Molecular Mechanisms Specifying the Topography of Thalamocortical Projectionsin the Ventral Telencephalon. [Internet] [Thesis]. University of North Carolina; 2008. [cited 2020 Sep 19]. Available from: https://cdr.lib.unc.edu/record/uuid:c6d7a3a6-9267-4049-a631-1027e437b6d3.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Powell A. Molecular Mechanisms Specifying the Topography of Thalamocortical Projectionsin the Ventral Telencephalon. [Thesis]. University of North Carolina; 2008. Available from: https://cdr.lib.unc.edu/record/uuid:c6d7a3a6-9267-4049-a631-1027e437b6d3

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

22. Foti, Stacey. Novel AAV-mediated Therapeutic Strategies for Epilepsy.

Degree: 2008, University of North Carolina

 Epilepsy afflicts 2.1 million people in the United States, and many patients develop drug resistant seizures. For these patients, gene therapy may be an attractive… (more)

Subjects/Keywords: School of Medicine; UNC Neuroscience Center; Neuroscience Curriculum

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Foti, S. (2008). Novel AAV-mediated Therapeutic Strategies for Epilepsy. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:a332c4e2-79fa-4b41-97b5-d1786e2656b8

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Foti, Stacey. “Novel AAV-mediated Therapeutic Strategies for Epilepsy.” 2008. Thesis, University of North Carolina. Accessed September 19, 2020. https://cdr.lib.unc.edu/record/uuid:a332c4e2-79fa-4b41-97b5-d1786e2656b8.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Foti, Stacey. “Novel AAV-mediated Therapeutic Strategies for Epilepsy.” 2008. Web. 19 Sep 2020.

Vancouver:

Foti S. Novel AAV-mediated Therapeutic Strategies for Epilepsy. [Internet] [Thesis]. University of North Carolina; 2008. [cited 2020 Sep 19]. Available from: https://cdr.lib.unc.edu/record/uuid:a332c4e2-79fa-4b41-97b5-d1786e2656b8.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Foti S. Novel AAV-mediated Therapeutic Strategies for Epilepsy. [Thesis]. University of North Carolina; 2008. Available from: https://cdr.lib.unc.edu/record/uuid:a332c4e2-79fa-4b41-97b5-d1786e2656b8

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

.