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You searched for +publisher:"University of North Carolina" +contributor:("Der, Channing"). Showing records 1 – 19 of 19 total matches.

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University of North Carolina

1. Cook, Danielle. Mechanisms of Ect2 Regulation in Cytokinesis and Oncogenesis.

Degree: 2013, University of North Carolina

 Ect2 is a member of the human Dbl family of guanine nucleotide exchange factors and are activators of Rho family small GTPases (RhoA, Rac1 and… (more)

Subjects/Keywords: Eshelman School of Pharmacy; Division of Chemical Biology and Medicinal Chemistry

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APA (6th Edition):

Cook, D. (2013). Mechanisms of Ect2 Regulation in Cytokinesis and Oncogenesis. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:d4687ab7-79a9-4fc6-b29f-165a8bf45208

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Cook, Danielle. “Mechanisms of Ect2 Regulation in Cytokinesis and Oncogenesis.” 2013. Thesis, University of North Carolina. Accessed September 23, 2020. https://cdr.lib.unc.edu/record/uuid:d4687ab7-79a9-4fc6-b29f-165a8bf45208.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Cook, Danielle. “Mechanisms of Ect2 Regulation in Cytokinesis and Oncogenesis.” 2013. Web. 23 Sep 2020.

Vancouver:

Cook D. Mechanisms of Ect2 Regulation in Cytokinesis and Oncogenesis. [Internet] [Thesis]. University of North Carolina; 2013. [cited 2020 Sep 23]. Available from: https://cdr.lib.unc.edu/record/uuid:d4687ab7-79a9-4fc6-b29f-165a8bf45208.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Cook D. Mechanisms of Ect2 Regulation in Cytokinesis and Oncogenesis. [Thesis]. University of North Carolina; 2013. Available from: https://cdr.lib.unc.edu/record/uuid:d4687ab7-79a9-4fc6-b29f-165a8bf45208

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

2. Dardy, Jasmyn. Validation of PAK Kinases for the Development of Novel Therapeutics for Pancreatic Cancer Treatment.

Degree: Pharmacology, 2012, University of North Carolina

 Pancreatic cancer is the 4th leading cause of cancer deaths in the US and essentially all who are diagnosed with this disease will die. Poor… (more)

Subjects/Keywords: School of Medicine; Department of Pharmacology

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APA (6th Edition):

Dardy, J. (2012). Validation of PAK Kinases for the Development of Novel Therapeutics for Pancreatic Cancer Treatment. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:d38f8022-7dbe-4973-8425-326645671831

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Dardy, Jasmyn. “Validation of PAK Kinases for the Development of Novel Therapeutics for Pancreatic Cancer Treatment.” 2012. Thesis, University of North Carolina. Accessed September 23, 2020. https://cdr.lib.unc.edu/record/uuid:d38f8022-7dbe-4973-8425-326645671831.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Dardy, Jasmyn. “Validation of PAK Kinases for the Development of Novel Therapeutics for Pancreatic Cancer Treatment.” 2012. Web. 23 Sep 2020.

Vancouver:

Dardy J. Validation of PAK Kinases for the Development of Novel Therapeutics for Pancreatic Cancer Treatment. [Internet] [Thesis]. University of North Carolina; 2012. [cited 2020 Sep 23]. Available from: https://cdr.lib.unc.edu/record/uuid:d38f8022-7dbe-4973-8425-326645671831.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Dardy J. Validation of PAK Kinases for the Development of Novel Therapeutics for Pancreatic Cancer Treatment. [Thesis]. University of North Carolina; 2012. Available from: https://cdr.lib.unc.edu/record/uuid:d38f8022-7dbe-4973-8425-326645671831

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

3. Martin, Timothy Dean. Regulation and Roles of Ral GTPase Signaling Components in Oncogenesis.

Degree: Pharmacology, 2013, University of North Carolina

 Since their discovery in 1986, Ral (Ras-like) GTPases have emerged as critical regulators of diverse cellular functions. Like Ras, the Ral proteins cycle between an… (more)

Subjects/Keywords: School of Medicine; Department of Pharmacology

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APA (6th Edition):

Martin, T. D. (2013). Regulation and Roles of Ral GTPase Signaling Components in Oncogenesis. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:635f1a26-171a-4346-9bc1-3facec547daa

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Martin, Timothy Dean. “Regulation and Roles of Ral GTPase Signaling Components in Oncogenesis.” 2013. Thesis, University of North Carolina. Accessed September 23, 2020. https://cdr.lib.unc.edu/record/uuid:635f1a26-171a-4346-9bc1-3facec547daa.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Martin, Timothy Dean. “Regulation and Roles of Ral GTPase Signaling Components in Oncogenesis.” 2013. Web. 23 Sep 2020.

Vancouver:

Martin TD. Regulation and Roles of Ral GTPase Signaling Components in Oncogenesis. [Internet] [Thesis]. University of North Carolina; 2013. [cited 2020 Sep 23]. Available from: https://cdr.lib.unc.edu/record/uuid:635f1a26-171a-4346-9bc1-3facec547daa.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Martin TD. Regulation and Roles of Ral GTPase Signaling Components in Oncogenesis. [Thesis]. University of North Carolina; 2013. Available from: https://cdr.lib.unc.edu/record/uuid:635f1a26-171a-4346-9bc1-3facec547daa

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

4. Stevens, Ellen V. Identification of Novel Therapeutic Approaches and Targets for the Treatment of Ovarian Carcinoma.

Degree: Pharmacology, 2011, University of North Carolina

 The most imperative issues for ovarian carcinoma patients, due to high relapse rates against mainstay therapies, is to identify new molecular targets and develop novel… (more)

Subjects/Keywords: School of Medicine; Department of Pharmacology

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APA (6th Edition):

Stevens, E. V. (2011). Identification of Novel Therapeutic Approaches and Targets for the Treatment of Ovarian Carcinoma. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:2a81a7e4-092c-46a3-b558-f87642e52ed8

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Stevens, Ellen V. “Identification of Novel Therapeutic Approaches and Targets for the Treatment of Ovarian Carcinoma.” 2011. Thesis, University of North Carolina. Accessed September 23, 2020. https://cdr.lib.unc.edu/record/uuid:2a81a7e4-092c-46a3-b558-f87642e52ed8.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Stevens, Ellen V. “Identification of Novel Therapeutic Approaches and Targets for the Treatment of Ovarian Carcinoma.” 2011. Web. 23 Sep 2020.

Vancouver:

Stevens EV. Identification of Novel Therapeutic Approaches and Targets for the Treatment of Ovarian Carcinoma. [Internet] [Thesis]. University of North Carolina; 2011. [cited 2020 Sep 23]. Available from: https://cdr.lib.unc.edu/record/uuid:2a81a7e4-092c-46a3-b558-f87642e52ed8.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Stevens EV. Identification of Novel Therapeutic Approaches and Targets for the Treatment of Ovarian Carcinoma. [Thesis]. University of North Carolina; 2011. Available from: https://cdr.lib.unc.edu/record/uuid:2a81a7e4-092c-46a3-b558-f87642e52ed8

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

5. Zand, Tanya Parisaw. Ras signaling through the RalGEF-Ral pathway in C. elegans.

Degree: Pharmacology, 2010, University of North Carolina

 The classical Ras effector pathway involves activation of the Raf-MEK-ERK mitogen-activated protein kinase cascade. Recent studies show that a second Ras effector cascade, Ral guanine… (more)

Subjects/Keywords: School of Medicine; Department of Pharmacology

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APA (6th Edition):

Zand, T. P. (2010). Ras signaling through the RalGEF-Ral pathway in C. elegans. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:752d8586-c911-4e85-8cec-3671c6564a4d

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Zand, Tanya Parisaw. “Ras signaling through the RalGEF-Ral pathway in C. elegans.” 2010. Thesis, University of North Carolina. Accessed September 23, 2020. https://cdr.lib.unc.edu/record/uuid:752d8586-c911-4e85-8cec-3671c6564a4d.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Zand, Tanya Parisaw. “Ras signaling through the RalGEF-Ral pathway in C. elegans.” 2010. Web. 23 Sep 2020.

Vancouver:

Zand TP. Ras signaling through the RalGEF-Ral pathway in C. elegans. [Internet] [Thesis]. University of North Carolina; 2010. [cited 2020 Sep 23]. Available from: https://cdr.lib.unc.edu/record/uuid:752d8586-c911-4e85-8cec-3671c6564a4d.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Zand TP. Ras signaling through the RalGEF-Ral pathway in C. elegans. [Thesis]. University of North Carolina; 2010. Available from: https://cdr.lib.unc.edu/record/uuid:752d8586-c911-4e85-8cec-3671c6564a4d

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

6. Turner, Abigail. Epidermal Growth Factor Receptor Tyrosine Kinase Assays in Single Intact Cells Using Capillary Electrophoresis.

Degree: Chemistry, 2015, University of North Carolina

 Quantification of abnormal Epidermal Growth Factor Receptor (EGFR) tyrosine kinase activity is critical to the clinical success of targeted inhibitors used to treat EGFR-dependent cancers.… (more)

Subjects/Keywords: Chemistry; College of Arts and Sciences; Department of Chemistry

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APA (6th Edition):

Turner, A. (2015). Epidermal Growth Factor Receptor Tyrosine Kinase Assays in Single Intact Cells Using Capillary Electrophoresis. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:524c1556-dc54-49ee-9403-39d0fbb45663

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Turner, Abigail. “Epidermal Growth Factor Receptor Tyrosine Kinase Assays in Single Intact Cells Using Capillary Electrophoresis.” 2015. Thesis, University of North Carolina. Accessed September 23, 2020. https://cdr.lib.unc.edu/record/uuid:524c1556-dc54-49ee-9403-39d0fbb45663.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Turner, Abigail. “Epidermal Growth Factor Receptor Tyrosine Kinase Assays in Single Intact Cells Using Capillary Electrophoresis.” 2015. Web. 23 Sep 2020.

Vancouver:

Turner A. Epidermal Growth Factor Receptor Tyrosine Kinase Assays in Single Intact Cells Using Capillary Electrophoresis. [Internet] [Thesis]. University of North Carolina; 2015. [cited 2020 Sep 23]. Available from: https://cdr.lib.unc.edu/record/uuid:524c1556-dc54-49ee-9403-39d0fbb45663.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Turner A. Epidermal Growth Factor Receptor Tyrosine Kinase Assays in Single Intact Cells Using Capillary Electrophoresis. [Thesis]. University of North Carolina; 2015. Available from: https://cdr.lib.unc.edu/record/uuid:524c1556-dc54-49ee-9403-39d0fbb45663

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

7. Gentry, Leanna. Targeting the Ras-Ral effector pathway for cancer treatment.

Degree: Pharmacology, 2015, University of North Carolina

 The RAS oncogene is the most frequently mutated gene in human cancers, and this activated Ras oncoprotein has been shown to be required for both… (more)

Subjects/Keywords: Pharmacology; School of Medicine; Department of Pharmacology

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APA (6th Edition):

Gentry, L. (2015). Targeting the Ras-Ral effector pathway for cancer treatment. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:c75ba6bc-3f00-4527-8f20-029bf699aad2

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Gentry, Leanna. “Targeting the Ras-Ral effector pathway for cancer treatment.” 2015. Thesis, University of North Carolina. Accessed September 23, 2020. https://cdr.lib.unc.edu/record/uuid:c75ba6bc-3f00-4527-8f20-029bf699aad2.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Gentry, Leanna. “Targeting the Ras-Ral effector pathway for cancer treatment.” 2015. Web. 23 Sep 2020.

Vancouver:

Gentry L. Targeting the Ras-Ral effector pathway for cancer treatment. [Internet] [Thesis]. University of North Carolina; 2015. [cited 2020 Sep 23]. Available from: https://cdr.lib.unc.edu/record/uuid:c75ba6bc-3f00-4527-8f20-029bf699aad2.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Gentry L. Targeting the Ras-Ral effector pathway for cancer treatment. [Thesis]. University of North Carolina; 2015. Available from: https://cdr.lib.unc.edu/record/uuid:c75ba6bc-3f00-4527-8f20-029bf699aad2

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

8. Jacak, Ronald. Explicit Consideration of Solubility and Interaction Specificity in Computational Protein Design.

Degree: Biochemistry and Biophysics, 2011, University of North Carolina

 Most successes to date in computational protein design have relied on optimizing sequences to fit well for a single structure. Multistate design represents a new… (more)

Subjects/Keywords: School of Medicine; Department of Biochemistry and Biophysics

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APA (6th Edition):

Jacak, R. (2011). Explicit Consideration of Solubility and Interaction Specificity in Computational Protein Design. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:eee31c27-82d0-4e6b-bb39-6456860e0255

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Jacak, Ronald. “Explicit Consideration of Solubility and Interaction Specificity in Computational Protein Design.” 2011. Thesis, University of North Carolina. Accessed September 23, 2020. https://cdr.lib.unc.edu/record/uuid:eee31c27-82d0-4e6b-bb39-6456860e0255.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Jacak, Ronald. “Explicit Consideration of Solubility and Interaction Specificity in Computational Protein Design.” 2011. Web. 23 Sep 2020.

Vancouver:

Jacak R. Explicit Consideration of Solubility and Interaction Specificity in Computational Protein Design. [Internet] [Thesis]. University of North Carolina; 2011. [cited 2020 Sep 23]. Available from: https://cdr.lib.unc.edu/record/uuid:eee31c27-82d0-4e6b-bb39-6456860e0255.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Jacak R. Explicit Consideration of Solubility and Interaction Specificity in Computational Protein Design. [Thesis]. University of North Carolina; 2011. Available from: https://cdr.lib.unc.edu/record/uuid:eee31c27-82d0-4e6b-bb39-6456860e0255

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

9. Alan, Jamie. TYROSINE KINASE MODULATION OF TRAFFICKING AND BIOLOGICAL FUNCTIONS OF THE ATYPICAL RHO GTPASE, WRCH-1.

Degree: Pharmacology, 2010, University of North Carolina

 Wrch-1 is an atypical Rho family small GTPase with roles in oncogenic transformation, epithelial cell morphogenesis, osteoclastogenesis, and migration. We have shown previously that Wrch-1… (more)

Subjects/Keywords: School of Medicine; Department of Pharmacology

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APA (6th Edition):

Alan, J. (2010). TYROSINE KINASE MODULATION OF TRAFFICKING AND BIOLOGICAL FUNCTIONS OF THE ATYPICAL RHO GTPASE, WRCH-1. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:afbc9ce5-f21d-4707-8744-357971e1eb3b

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Alan, Jamie. “TYROSINE KINASE MODULATION OF TRAFFICKING AND BIOLOGICAL FUNCTIONS OF THE ATYPICAL RHO GTPASE, WRCH-1.” 2010. Thesis, University of North Carolina. Accessed September 23, 2020. https://cdr.lib.unc.edu/record/uuid:afbc9ce5-f21d-4707-8744-357971e1eb3b.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Alan, Jamie. “TYROSINE KINASE MODULATION OF TRAFFICKING AND BIOLOGICAL FUNCTIONS OF THE ATYPICAL RHO GTPASE, WRCH-1.” 2010. Web. 23 Sep 2020.

Vancouver:

Alan J. TYROSINE KINASE MODULATION OF TRAFFICKING AND BIOLOGICAL FUNCTIONS OF THE ATYPICAL RHO GTPASE, WRCH-1. [Internet] [Thesis]. University of North Carolina; 2010. [cited 2020 Sep 23]. Available from: https://cdr.lib.unc.edu/record/uuid:afbc9ce5-f21d-4707-8744-357971e1eb3b.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Alan J. TYROSINE KINASE MODULATION OF TRAFFICKING AND BIOLOGICAL FUNCTIONS OF THE ATYPICAL RHO GTPASE, WRCH-1. [Thesis]. University of North Carolina; 2010. Available from: https://cdr.lib.unc.edu/record/uuid:afbc9ce5-f21d-4707-8744-357971e1eb3b

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

10. Hayes, Tikvah. Targeting mutant KRAS in pancreatic cancer.

Degree: 2016, University of North Carolina

 The development of pharmacologic inhibitors of the KRAS oncoprotein, which is mutated in ~30% of all human cancers, has been at the forefront of drug… (more)

Subjects/Keywords: School of Medicine; Curriculum in Genetics and Molecular Biology

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APA (6th Edition):

Hayes, T. (2016). Targeting mutant KRAS in pancreatic cancer. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:f816a0b0-3d43-4afb-9623-dcbfb1d7bd39

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hayes, Tikvah. “Targeting mutant KRAS in pancreatic cancer.” 2016. Thesis, University of North Carolina. Accessed September 23, 2020. https://cdr.lib.unc.edu/record/uuid:f816a0b0-3d43-4afb-9623-dcbfb1d7bd39.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hayes, Tikvah. “Targeting mutant KRAS in pancreatic cancer.” 2016. Web. 23 Sep 2020.

Vancouver:

Hayes T. Targeting mutant KRAS in pancreatic cancer. [Internet] [Thesis]. University of North Carolina; 2016. [cited 2020 Sep 23]. Available from: https://cdr.lib.unc.edu/record/uuid:f816a0b0-3d43-4afb-9623-dcbfb1d7bd39.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hayes T. Targeting mutant KRAS in pancreatic cancer. [Thesis]. University of North Carolina; 2016. Available from: https://cdr.lib.unc.edu/record/uuid:f816a0b0-3d43-4afb-9623-dcbfb1d7bd39

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

11. Zhou, Bingying. RAS signaling and therapeutic resistance in melanoma.

Degree: Pharmacology, 2016, University of North Carolina

 Increasing appreciation of the complexity of RAS signaling in cancer has led to a renewed wave of RAS research. I have focused on two key… (more)

Subjects/Keywords: School of Medicine; Department of Pharmacology

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APA (6th Edition):

Zhou, B. (2016). RAS signaling and therapeutic resistance in melanoma. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:26347cfa-34a3-4849-b350-f66b6a70334a

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Zhou, Bingying. “RAS signaling and therapeutic resistance in melanoma.” 2016. Thesis, University of North Carolina. Accessed September 23, 2020. https://cdr.lib.unc.edu/record/uuid:26347cfa-34a3-4849-b350-f66b6a70334a.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Zhou, Bingying. “RAS signaling and therapeutic resistance in melanoma.” 2016. Web. 23 Sep 2020.

Vancouver:

Zhou B. RAS signaling and therapeutic resistance in melanoma. [Internet] [Thesis]. University of North Carolina; 2016. [cited 2020 Sep 23]. Available from: https://cdr.lib.unc.edu/record/uuid:26347cfa-34a3-4849-b350-f66b6a70334a.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Zhou B. RAS signaling and therapeutic resistance in melanoma. [Thesis]. University of North Carolina; 2016. Available from: https://cdr.lib.unc.edu/record/uuid:26347cfa-34a3-4849-b350-f66b6a70334a

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

12. Baker, Nicole. Evaluating the therapeutic potential of the PAK1 and TBK1 kinases in pancreatic ductal adenocarcinoma.

Degree: Pharmacology, 2016, University of North Carolina

 Pancreatic ductal adenocarcinoma (PDAC) is an extremely lethal cancer characterized by a high frequency (>95%) of activating mutations in the KRAS oncogene, which is a… (more)

Subjects/Keywords: School of Medicine; Department of Pharmacology

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APA (6th Edition):

Baker, N. (2016). Evaluating the therapeutic potential of the PAK1 and TBK1 kinases in pancreatic ductal adenocarcinoma. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:6dc9a875-b142-43f8-b49e-11482c6de322

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Baker, Nicole. “Evaluating the therapeutic potential of the PAK1 and TBK1 kinases in pancreatic ductal adenocarcinoma.” 2016. Thesis, University of North Carolina. Accessed September 23, 2020. https://cdr.lib.unc.edu/record/uuid:6dc9a875-b142-43f8-b49e-11482c6de322.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Baker, Nicole. “Evaluating the therapeutic potential of the PAK1 and TBK1 kinases in pancreatic ductal adenocarcinoma.” 2016. Web. 23 Sep 2020.

Vancouver:

Baker N. Evaluating the therapeutic potential of the PAK1 and TBK1 kinases in pancreatic ductal adenocarcinoma. [Internet] [Thesis]. University of North Carolina; 2016. [cited 2020 Sep 23]. Available from: https://cdr.lib.unc.edu/record/uuid:6dc9a875-b142-43f8-b49e-11482c6de322.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Baker N. Evaluating the therapeutic potential of the PAK1 and TBK1 kinases in pancreatic ductal adenocarcinoma. [Thesis]. University of North Carolina; 2016. Available from: https://cdr.lib.unc.edu/record/uuid:6dc9a875-b142-43f8-b49e-11482c6de322

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

13. Ryan, Meagan. TARGETING THE ERK MAPK PATHWAY IN RAS-DRIVEN CANCERS.

Degree: Pharmacology, 2016, University of North Carolina

 RAS mutations are frequently found in the deadliest cancers in the United States, and there is a renewed interest in identifying therapeutic strategies to target… (more)

Subjects/Keywords: School of Medicine; Department of Pharmacology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Ryan, M. (2016). TARGETING THE ERK MAPK PATHWAY IN RAS-DRIVEN CANCERS. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:c9d9e3f8-a30a-4eb5-ba62-c021deb34805

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ryan, Meagan. “TARGETING THE ERK MAPK PATHWAY IN RAS-DRIVEN CANCERS.” 2016. Thesis, University of North Carolina. Accessed September 23, 2020. https://cdr.lib.unc.edu/record/uuid:c9d9e3f8-a30a-4eb5-ba62-c021deb34805.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ryan, Meagan. “TARGETING THE ERK MAPK PATHWAY IN RAS-DRIVEN CANCERS.” 2016. Web. 23 Sep 2020.

Vancouver:

Ryan M. TARGETING THE ERK MAPK PATHWAY IN RAS-DRIVEN CANCERS. [Internet] [Thesis]. University of North Carolina; 2016. [cited 2020 Sep 23]. Available from: https://cdr.lib.unc.edu/record/uuid:c9d9e3f8-a30a-4eb5-ba62-c021deb34805.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ryan M. TARGETING THE ERK MAPK PATHWAY IN RAS-DRIVEN CANCERS. [Thesis]. University of North Carolina; 2016. Available from: https://cdr.lib.unc.edu/record/uuid:c9d9e3f8-a30a-4eb5-ba62-c021deb34805

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

14. Stratford, Jeran. Aberrant Gene Expression: Diagnostic Markers and Therapeutic Targets for Pancreatic Cancer.

Degree: Pharmacology, 2014, University of North Carolina

 Pancreatic ductal adenocarcinoma (PDAC) is an aggressive cancer and the fourth leading cause of cancer-related death in the United States. The overall median survival for… (more)

Subjects/Keywords: Pharmacology; School of Medicine; Department of Pharmacology

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APA (6th Edition):

Stratford, J. (2014). Aberrant Gene Expression: Diagnostic Markers and Therapeutic Targets for Pancreatic Cancer. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:84f3613f-5709-4ae6-8407-999e9eb3a6eb

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Stratford, Jeran. “Aberrant Gene Expression: Diagnostic Markers and Therapeutic Targets for Pancreatic Cancer.” 2014. Thesis, University of North Carolina. Accessed September 23, 2020. https://cdr.lib.unc.edu/record/uuid:84f3613f-5709-4ae6-8407-999e9eb3a6eb.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Stratford, Jeran. “Aberrant Gene Expression: Diagnostic Markers and Therapeutic Targets for Pancreatic Cancer.” 2014. Web. 23 Sep 2020.

Vancouver:

Stratford J. Aberrant Gene Expression: Diagnostic Markers and Therapeutic Targets for Pancreatic Cancer. [Internet] [Thesis]. University of North Carolina; 2014. [cited 2020 Sep 23]. Available from: https://cdr.lib.unc.edu/record/uuid:84f3613f-5709-4ae6-8407-999e9eb3a6eb.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Stratford J. Aberrant Gene Expression: Diagnostic Markers and Therapeutic Targets for Pancreatic Cancer. [Thesis]. University of North Carolina; 2014. Available from: https://cdr.lib.unc.edu/record/uuid:84f3613f-5709-4ae6-8407-999e9eb3a6eb

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

15. Cha, Jiyoung. The role and mechanism of fibroblast growth factor receptor 2 in cellular transformation.

Degree: Pharmacology, 2008, University of North Carolina

 Our laboratory recently identified fibroblast growth factor receptor 2 (FGFR2) as a transforming gene using a cDNA expression library screen for novel oncogenes expressed in… (more)

Subjects/Keywords: School of Medicine; Department of Pharmacology

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APA (6th Edition):

Cha, J. (2008). The role and mechanism of fibroblast growth factor receptor 2 in cellular transformation. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:d19076c8-bba0-4958-85d3-0fb113d870aa

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Cha, Jiyoung. “The role and mechanism of fibroblast growth factor receptor 2 in cellular transformation.” 2008. Thesis, University of North Carolina. Accessed September 23, 2020. https://cdr.lib.unc.edu/record/uuid:d19076c8-bba0-4958-85d3-0fb113d870aa.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Cha, Jiyoung. “The role and mechanism of fibroblast growth factor receptor 2 in cellular transformation.” 2008. Web. 23 Sep 2020.

Vancouver:

Cha J. The role and mechanism of fibroblast growth factor receptor 2 in cellular transformation. [Internet] [Thesis]. University of North Carolina; 2008. [cited 2020 Sep 23]. Available from: https://cdr.lib.unc.edu/record/uuid:d19076c8-bba0-4958-85d3-0fb113d870aa.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Cha J. The role and mechanism of fibroblast growth factor receptor 2 in cellular transformation. [Thesis]. University of North Carolina; 2008. Available from: https://cdr.lib.unc.edu/record/uuid:d19076c8-bba0-4958-85d3-0fb113d870aa

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

16. Hanker, Ariella Binah. Ras family GTPases involved in breast cancer.

Degree: 2009, University of North Carolina

 The Ras branch of the Ras superfamily of small GTPases consists of over thirty-six proteins that regulate a wide array of cellular processes. Mutations in… (more)

Subjects/Keywords: School of Medicine; Curriculum in Genetics and Molecular Biology

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APA (6th Edition):

Hanker, A. B. (2009). Ras family GTPases involved in breast cancer. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:371d70e6-4d53-4143-adc0-ab60d835474a

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hanker, Ariella Binah. “Ras family GTPases involved in breast cancer.” 2009. Thesis, University of North Carolina. Accessed September 23, 2020. https://cdr.lib.unc.edu/record/uuid:371d70e6-4d53-4143-adc0-ab60d835474a.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hanker, Ariella Binah. “Ras family GTPases involved in breast cancer.” 2009. Web. 23 Sep 2020.

Vancouver:

Hanker AB. Ras family GTPases involved in breast cancer. [Internet] [Thesis]. University of North Carolina; 2009. [cited 2020 Sep 23]. Available from: https://cdr.lib.unc.edu/record/uuid:371d70e6-4d53-4143-adc0-ab60d835474a.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hanker AB. Ras family GTPases involved in breast cancer. [Thesis]. University of North Carolina; 2009. Available from: https://cdr.lib.unc.edu/record/uuid:371d70e6-4d53-4143-adc0-ab60d835474a

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

17. DeNofrio, Jan. Characterization of Platelet Function and Hemostasis in the Cib1-/- Mouse.

Degree: 2008, University of North Carolina

 Platelet activation initiates platelet-platelet aggregation, platelet-extracellular matrix interactions and thrombus formation. Platelet aggregation requires activation of the αIIbβ3 integrin, an event regulated by the integrin… (more)

Subjects/Keywords: School of Medicine; Curriculum in Genetics and Molecular Biology

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APA (6th Edition):

DeNofrio, J. (2008). Characterization of Platelet Function and Hemostasis in the Cib1-/- Mouse. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:654d2a52-5937-4ff8-905e-98b7c43d7438

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

DeNofrio, Jan. “Characterization of Platelet Function and Hemostasis in the Cib1-/- Mouse.” 2008. Thesis, University of North Carolina. Accessed September 23, 2020. https://cdr.lib.unc.edu/record/uuid:654d2a52-5937-4ff8-905e-98b7c43d7438.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

DeNofrio, Jan. “Characterization of Platelet Function and Hemostasis in the Cib1-/- Mouse.” 2008. Web. 23 Sep 2020.

Vancouver:

DeNofrio J. Characterization of Platelet Function and Hemostasis in the Cib1-/- Mouse. [Internet] [Thesis]. University of North Carolina; 2008. [cited 2020 Sep 23]. Available from: https://cdr.lib.unc.edu/record/uuid:654d2a52-5937-4ff8-905e-98b7c43d7438.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

DeNofrio J. Characterization of Platelet Function and Hemostasis in the Cib1-/- Mouse. [Thesis]. University of North Carolina; 2008. Available from: https://cdr.lib.unc.edu/record/uuid:654d2a52-5937-4ff8-905e-98b7c43d7438

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

18. Brady, Donita. The transforming Rho family GTPase, Wrch-1, regulates epithelial cell morphogenesis through modulating cell junctions and actin cytoskeletal dynamics.

Degree: Pharmacology, 2008, University of North Carolina

 Rho GTPases are members of the Ras superfamily of GTP binding proteins that function as molecular switches. In their GTP-bound, active state, Rho proteins are… (more)

Subjects/Keywords: School of Medicine; Department of Pharmacology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Brady, D. (2008). The transforming Rho family GTPase, Wrch-1, regulates epithelial cell morphogenesis through modulating cell junctions and actin cytoskeletal dynamics. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:0e2e6907-aef0-4d56-8361-dc656a966d8a

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Brady, Donita. “The transforming Rho family GTPase, Wrch-1, regulates epithelial cell morphogenesis through modulating cell junctions and actin cytoskeletal dynamics.” 2008. Thesis, University of North Carolina. Accessed September 23, 2020. https://cdr.lib.unc.edu/record/uuid:0e2e6907-aef0-4d56-8361-dc656a966d8a.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Brady, Donita. “The transforming Rho family GTPase, Wrch-1, regulates epithelial cell morphogenesis through modulating cell junctions and actin cytoskeletal dynamics.” 2008. Web. 23 Sep 2020.

Vancouver:

Brady D. The transforming Rho family GTPase, Wrch-1, regulates epithelial cell morphogenesis through modulating cell junctions and actin cytoskeletal dynamics. [Internet] [Thesis]. University of North Carolina; 2008. [cited 2020 Sep 23]. Available from: https://cdr.lib.unc.edu/record/uuid:0e2e6907-aef0-4d56-8361-dc656a966d8a.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Brady D. The transforming Rho family GTPase, Wrch-1, regulates epithelial cell morphogenesis through modulating cell junctions and actin cytoskeletal dynamics. [Thesis]. University of North Carolina; 2008. Available from: https://cdr.lib.unc.edu/record/uuid:0e2e6907-aef0-4d56-8361-dc656a966d8a

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

19. Roberts, Patrick. Targeting Ras and Rho Family GTPases for the Treatment of Cancer Through Inhibition of CAAX-Signaled Modifications and the ERK MAPK Pathway.

Degree: 2008, University of North Carolina

 Ras and Rho family GTPases share many structural and biochemical similarities while regulating distinct cellular functions. One such similarity is the carboxyl-terminal CAAX motif (C… (more)

Subjects/Keywords: Eshelman School of Pharmacy; Division of Pharmacotherapy and Experimental Therapeutics

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Roberts, P. (2008). Targeting Ras and Rho Family GTPases for the Treatment of Cancer Through Inhibition of CAAX-Signaled Modifications and the ERK MAPK Pathway. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:3f483630-1fb8-4ca3-97e6-5a9984477b03

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Roberts, Patrick. “Targeting Ras and Rho Family GTPases for the Treatment of Cancer Through Inhibition of CAAX-Signaled Modifications and the ERK MAPK Pathway.” 2008. Thesis, University of North Carolina. Accessed September 23, 2020. https://cdr.lib.unc.edu/record/uuid:3f483630-1fb8-4ca3-97e6-5a9984477b03.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Roberts, Patrick. “Targeting Ras and Rho Family GTPases for the Treatment of Cancer Through Inhibition of CAAX-Signaled Modifications and the ERK MAPK Pathway.” 2008. Web. 23 Sep 2020.

Vancouver:

Roberts P. Targeting Ras and Rho Family GTPases for the Treatment of Cancer Through Inhibition of CAAX-Signaled Modifications and the ERK MAPK Pathway. [Internet] [Thesis]. University of North Carolina; 2008. [cited 2020 Sep 23]. Available from: https://cdr.lib.unc.edu/record/uuid:3f483630-1fb8-4ca3-97e6-5a9984477b03.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Roberts P. Targeting Ras and Rho Family GTPases for the Treatment of Cancer Through Inhibition of CAAX-Signaled Modifications and the ERK MAPK Pathway. [Thesis]. University of North Carolina; 2008. Available from: https://cdr.lib.unc.edu/record/uuid:3f483630-1fb8-4ca3-97e6-5a9984477b03

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

.