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You searched for +publisher:"University of North Carolina" +contributor:("Burridge, Keith"). Showing records 1 – 25 of 25 total matches.

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University of North Carolina

1. Aghajanian, Amir. A Novel Role for Reactive Oxygen Species in the Regulation of RhoA: Implications for Endothelial Permeability and Leukocyte Transmigration.

Degree: Cell Biology and Physiology, 2011, University of North Carolina

 The endothelial lining of the vasculature plays a critical role in regulating the passage of fluid, macromolecules, and cells between the blood and surrounding tissues.… (more)

Subjects/Keywords: School of Medicine; Department of Cell Biology and Physiology

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APA (6th Edition):

Aghajanian, A. (2011). A Novel Role for Reactive Oxygen Species in the Regulation of RhoA: Implications for Endothelial Permeability and Leukocyte Transmigration. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:e4986982-9dec-4e6f-99f4-fc1035e50693

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Aghajanian, Amir. “A Novel Role for Reactive Oxygen Species in the Regulation of RhoA: Implications for Endothelial Permeability and Leukocyte Transmigration.” 2011. Thesis, University of North Carolina. Accessed September 27, 2020. https://cdr.lib.unc.edu/record/uuid:e4986982-9dec-4e6f-99f4-fc1035e50693.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Aghajanian, Amir. “A Novel Role for Reactive Oxygen Species in the Regulation of RhoA: Implications for Endothelial Permeability and Leukocyte Transmigration.” 2011. Web. 27 Sep 2020.

Vancouver:

Aghajanian A. A Novel Role for Reactive Oxygen Species in the Regulation of RhoA: Implications for Endothelial Permeability and Leukocyte Transmigration. [Internet] [Thesis]. University of North Carolina; 2011. [cited 2020 Sep 27]. Available from: https://cdr.lib.unc.edu/record/uuid:e4986982-9dec-4e6f-99f4-fc1035e50693.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Aghajanian A. A Novel Role for Reactive Oxygen Species in the Regulation of RhoA: Implications for Endothelial Permeability and Leukocyte Transmigration. [Thesis]. University of North Carolina; 2011. Available from: https://cdr.lib.unc.edu/record/uuid:e4986982-9dec-4e6f-99f4-fc1035e50693

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

2. Tolbert, Caitlin Elizabeth. Characterizing the Role of Actin Binding, Bundling, and Tyrosine Phosphorylation in Modulating Vinculin Function.

Degree: Cell Biology and Physiology, 2013, University of North Carolina

 Vinculin is an essential adhesion protein involved in controlling motility and force transduction, in part by coupling the actin cytoskeleton to the extracellular matrix. Vinculin… (more)

Subjects/Keywords: School of Medicine; Department of Cell Biology and Physiology

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APA (6th Edition):

Tolbert, C. E. (2013). Characterizing the Role of Actin Binding, Bundling, and Tyrosine Phosphorylation in Modulating Vinculin Function. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:6e6836c7-afdf-4aa9-9fe0-d93e30cbe2b5

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Tolbert, Caitlin Elizabeth. “Characterizing the Role of Actin Binding, Bundling, and Tyrosine Phosphorylation in Modulating Vinculin Function.” 2013. Thesis, University of North Carolina. Accessed September 27, 2020. https://cdr.lib.unc.edu/record/uuid:6e6836c7-afdf-4aa9-9fe0-d93e30cbe2b5.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Tolbert, Caitlin Elizabeth. “Characterizing the Role of Actin Binding, Bundling, and Tyrosine Phosphorylation in Modulating Vinculin Function.” 2013. Web. 27 Sep 2020.

Vancouver:

Tolbert CE. Characterizing the Role of Actin Binding, Bundling, and Tyrosine Phosphorylation in Modulating Vinculin Function. [Internet] [Thesis]. University of North Carolina; 2013. [cited 2020 Sep 27]. Available from: https://cdr.lib.unc.edu/record/uuid:6e6836c7-afdf-4aa9-9fe0-d93e30cbe2b5.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Tolbert CE. Characterizing the Role of Actin Binding, Bundling, and Tyrosine Phosphorylation in Modulating Vinculin Function. [Thesis]. University of North Carolina; 2013. Available from: https://cdr.lib.unc.edu/record/uuid:6e6836c7-afdf-4aa9-9fe0-d93e30cbe2b5

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

3. Osborne, Lukas. Biophysics of Cancer Progression and High-Throughput Mechanical Characterization of Biomaterials.

Degree: Physics and Astronomy, 2015, University of North Carolina

 Cancer metastasis involves a series of events known as the metastatic cascade. In this complex progression, cancer cells detach from the primary tumor, invade the… (more)

Subjects/Keywords: Physics; Biophysics; Biomechanics; College of Arts and Sciences; Department of Physics and Astronomy

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APA (6th Edition):

Osborne, L. (2015). Biophysics of Cancer Progression and High-Throughput Mechanical Characterization of Biomaterials. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:ecab069a-3543-41d9-8a8b-14364ef9b9d3

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Osborne, Lukas. “Biophysics of Cancer Progression and High-Throughput Mechanical Characterization of Biomaterials.” 2015. Thesis, University of North Carolina. Accessed September 27, 2020. https://cdr.lib.unc.edu/record/uuid:ecab069a-3543-41d9-8a8b-14364ef9b9d3.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Osborne, Lukas. “Biophysics of Cancer Progression and High-Throughput Mechanical Characterization of Biomaterials.” 2015. Web. 27 Sep 2020.

Vancouver:

Osborne L. Biophysics of Cancer Progression and High-Throughput Mechanical Characterization of Biomaterials. [Internet] [Thesis]. University of North Carolina; 2015. [cited 2020 Sep 27]. Available from: https://cdr.lib.unc.edu/record/uuid:ecab069a-3543-41d9-8a8b-14364ef9b9d3.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Osborne L. Biophysics of Cancer Progression and High-Throughput Mechanical Characterization of Biomaterials. [Thesis]. University of North Carolina; 2015. Available from: https://cdr.lib.unc.edu/record/uuid:ecab069a-3543-41d9-8a8b-14364ef9b9d3

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

4. Black, Justin. Exploration of the role of CIB1 in cell survival and tumor growth.

Degree: Biochemistry and Biophysics, 2015, University of North Carolina

 CIB1 is an intracellular protein with diverse functions in cancer cell biology. Here I explore two important functions of CIB1: 1) The role of CIB1… (more)

Subjects/Keywords: Biochemistry; Cytology; Genetics; School of Medicine; Department of Biochemistry and Biophysics

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APA (6th Edition):

Black, J. (2015). Exploration of the role of CIB1 in cell survival and tumor growth. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:b58ffcb6-4707-43a6-b86e-4644085f46e1

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Black, Justin. “Exploration of the role of CIB1 in cell survival and tumor growth.” 2015. Thesis, University of North Carolina. Accessed September 27, 2020. https://cdr.lib.unc.edu/record/uuid:b58ffcb6-4707-43a6-b86e-4644085f46e1.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Black, Justin. “Exploration of the role of CIB1 in cell survival and tumor growth.” 2015. Web. 27 Sep 2020.

Vancouver:

Black J. Exploration of the role of CIB1 in cell survival and tumor growth. [Internet] [Thesis]. University of North Carolina; 2015. [cited 2020 Sep 27]. Available from: https://cdr.lib.unc.edu/record/uuid:b58ffcb6-4707-43a6-b86e-4644085f46e1.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Black J. Exploration of the role of CIB1 in cell survival and tumor growth. [Thesis]. University of North Carolina; 2015. Available from: https://cdr.lib.unc.edu/record/uuid:b58ffcb6-4707-43a6-b86e-4644085f46e1

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

5. Weise Cross, Laura. Regulation of Smooth Muscle Cell Phenotype by the RhoA Effectors mDia1 and mDia2.

Degree: Pathology and Laboratory Medicine, 2015, University of North Carolina

 Smooth muscle cells (SMCs) play an important role in vascular development and disease. Vascular SMCs undergo profound changes in phenotype in response to environmental cues… (more)

Subjects/Keywords: Pathology; Cytology; Molecular biology; School of Medicine; Department of Pathology and Laboratory Medicine

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APA (6th Edition):

Weise Cross, L. (2015). Regulation of Smooth Muscle Cell Phenotype by the RhoA Effectors mDia1 and mDia2. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:b656650d-84e2-48f7-ac8b-4135ba45a063

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Weise Cross, Laura. “Regulation of Smooth Muscle Cell Phenotype by the RhoA Effectors mDia1 and mDia2.” 2015. Thesis, University of North Carolina. Accessed September 27, 2020. https://cdr.lib.unc.edu/record/uuid:b656650d-84e2-48f7-ac8b-4135ba45a063.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Weise Cross, Laura. “Regulation of Smooth Muscle Cell Phenotype by the RhoA Effectors mDia1 and mDia2.” 2015. Web. 27 Sep 2020.

Vancouver:

Weise Cross L. Regulation of Smooth Muscle Cell Phenotype by the RhoA Effectors mDia1 and mDia2. [Internet] [Thesis]. University of North Carolina; 2015. [cited 2020 Sep 27]. Available from: https://cdr.lib.unc.edu/record/uuid:b656650d-84e2-48f7-ac8b-4135ba45a063.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Weise Cross L. Regulation of Smooth Muscle Cell Phenotype by the RhoA Effectors mDia1 and mDia2. [Thesis]. University of North Carolina; 2015. Available from: https://cdr.lib.unc.edu/record/uuid:b656650d-84e2-48f7-ac8b-4135ba45a063

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

6. Jones, Stephen. Nanoparticle Clearance is Governed by Th1/Th2 Immunity and Strain Background.

Degree: Cell Biology and Physiology, 2015, University of North Carolina

 Extended circulation of nanoparticles in blood is essential for most clinical applications. Nanoparticles are rapidly cleared by cells of the mononuclear phagocyte system (MPS). Approaches… (more)

Subjects/Keywords: Cytology; School of Medicine; Department of Cell Biology and Physiology

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APA (6th Edition):

Jones, S. (2015). Nanoparticle Clearance is Governed by Th1/Th2 Immunity and Strain Background. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:5180f990-8a54-4ac9-aec1-e00cd03b9ab3

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Jones, Stephen. “Nanoparticle Clearance is Governed by Th1/Th2 Immunity and Strain Background.” 2015. Thesis, University of North Carolina. Accessed September 27, 2020. https://cdr.lib.unc.edu/record/uuid:5180f990-8a54-4ac9-aec1-e00cd03b9ab3.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Jones, Stephen. “Nanoparticle Clearance is Governed by Th1/Th2 Immunity and Strain Background.” 2015. Web. 27 Sep 2020.

Vancouver:

Jones S. Nanoparticle Clearance is Governed by Th1/Th2 Immunity and Strain Background. [Internet] [Thesis]. University of North Carolina; 2015. [cited 2020 Sep 27]. Available from: https://cdr.lib.unc.edu/record/uuid:5180f990-8a54-4ac9-aec1-e00cd03b9ab3.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Jones S. Nanoparticle Clearance is Governed by Th1/Th2 Immunity and Strain Background. [Thesis]. University of North Carolina; 2015. Available from: https://cdr.lib.unc.edu/record/uuid:5180f990-8a54-4ac9-aec1-e00cd03b9ab3

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

7. Graham, David. THE NUCLEUS IN CELL POLARIZATION, MIGRATION, AND MECHANOTRANSDUCTION.

Degree: Cell Biology and Physiology, 2018, University of North Carolina

 Many signaling pathways converge on the nucleus to regulate critical nuclear events such as transcription, DNA replication and cell cycle progression. While the vast majority… (more)

Subjects/Keywords: School of Medicine; Department of Cell Biology and Physiology

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APA (6th Edition):

Graham, D. (2018). THE NUCLEUS IN CELL POLARIZATION, MIGRATION, AND MECHANOTRANSDUCTION. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:70d2088e-2976-40ab-af42-90274aad35ca

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Graham, David. “THE NUCLEUS IN CELL POLARIZATION, MIGRATION, AND MECHANOTRANSDUCTION.” 2018. Thesis, University of North Carolina. Accessed September 27, 2020. https://cdr.lib.unc.edu/record/uuid:70d2088e-2976-40ab-af42-90274aad35ca.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Graham, David. “THE NUCLEUS IN CELL POLARIZATION, MIGRATION, AND MECHANOTRANSDUCTION.” 2018. Web. 27 Sep 2020.

Vancouver:

Graham D. THE NUCLEUS IN CELL POLARIZATION, MIGRATION, AND MECHANOTRANSDUCTION. [Internet] [Thesis]. University of North Carolina; 2018. [cited 2020 Sep 27]. Available from: https://cdr.lib.unc.edu/record/uuid:70d2088e-2976-40ab-af42-90274aad35ca.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Graham D. THE NUCLEUS IN CELL POLARIZATION, MIGRATION, AND MECHANOTRANSDUCTION. [Thesis]. University of North Carolina; 2018. Available from: https://cdr.lib.unc.edu/record/uuid:70d2088e-2976-40ab-af42-90274aad35ca

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

8. Kutys, Matthew. A Novel, Matrix-Specific GEF/GAP Interaction Regulates Rho GTPase Crosstalk Critical for 3D Collagen Migration.

Degree: Cell Biology and Physiology, 2014, University of North Carolina

 Differential activation of the Rho family GTPases, Cdc42, Rac1, and RhoA, helps to govern the distinct morphological and migratory phenotypes downstream of adhesion to different… (more)

Subjects/Keywords: Cytology; Biochemistry; School of Medicine; Department of Cell Biology and Physiology

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APA (6th Edition):

Kutys, M. (2014). A Novel, Matrix-Specific GEF/GAP Interaction Regulates Rho GTPase Crosstalk Critical for 3D Collagen Migration. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:bf6d58e1-869c-49e5-baff-5df0a348eaec

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kutys, Matthew. “A Novel, Matrix-Specific GEF/GAP Interaction Regulates Rho GTPase Crosstalk Critical for 3D Collagen Migration.” 2014. Thesis, University of North Carolina. Accessed September 27, 2020. https://cdr.lib.unc.edu/record/uuid:bf6d58e1-869c-49e5-baff-5df0a348eaec.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kutys, Matthew. “A Novel, Matrix-Specific GEF/GAP Interaction Regulates Rho GTPase Crosstalk Critical for 3D Collagen Migration.” 2014. Web. 27 Sep 2020.

Vancouver:

Kutys M. A Novel, Matrix-Specific GEF/GAP Interaction Regulates Rho GTPase Crosstalk Critical for 3D Collagen Migration. [Internet] [Thesis]. University of North Carolina; 2014. [cited 2020 Sep 27]. Available from: https://cdr.lib.unc.edu/record/uuid:bf6d58e1-869c-49e5-baff-5df0a348eaec.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kutys M. A Novel, Matrix-Specific GEF/GAP Interaction Regulates Rho GTPase Crosstalk Critical for 3D Collagen Migration. [Thesis]. University of North Carolina; 2014. Available from: https://cdr.lib.unc.edu/record/uuid:bf6d58e1-869c-49e5-baff-5df0a348eaec

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

9. Lessey-Morillon, Elizabeth. The RhoA Guanine Nucleotide Exchange Factor, LARG, Mediates ICAM-1-Dependent Mechanotransduction In Endothelial Cells To Stimulate Transendothelial Migration.

Degree: Cell Biology and Physiology, 2014, University of North Carolina

 RhoA-mediated cytoskeletal rearrangements in endothelial cells (ECs) play an active role in leukocyte transendothelial cell migration (TEM), a normal physiological process in which leukocytes cross… (more)

Subjects/Keywords: Cytology; School of Medicine; Department of Cell Biology and Physiology

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APA (6th Edition):

Lessey-Morillon, E. (2014). The RhoA Guanine Nucleotide Exchange Factor, LARG, Mediates ICAM-1-Dependent Mechanotransduction In Endothelial Cells To Stimulate Transendothelial Migration. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:e0425b15-91e2-46a9-aead-d82aee48dc8b

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lessey-Morillon, Elizabeth. “The RhoA Guanine Nucleotide Exchange Factor, LARG, Mediates ICAM-1-Dependent Mechanotransduction In Endothelial Cells To Stimulate Transendothelial Migration.” 2014. Thesis, University of North Carolina. Accessed September 27, 2020. https://cdr.lib.unc.edu/record/uuid:e0425b15-91e2-46a9-aead-d82aee48dc8b.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lessey-Morillon, Elizabeth. “The RhoA Guanine Nucleotide Exchange Factor, LARG, Mediates ICAM-1-Dependent Mechanotransduction In Endothelial Cells To Stimulate Transendothelial Migration.” 2014. Web. 27 Sep 2020.

Vancouver:

Lessey-Morillon E. The RhoA Guanine Nucleotide Exchange Factor, LARG, Mediates ICAM-1-Dependent Mechanotransduction In Endothelial Cells To Stimulate Transendothelial Migration. [Internet] [Thesis]. University of North Carolina; 2014. [cited 2020 Sep 27]. Available from: https://cdr.lib.unc.edu/record/uuid:e0425b15-91e2-46a9-aead-d82aee48dc8b.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lessey-Morillon E. The RhoA Guanine Nucleotide Exchange Factor, LARG, Mediates ICAM-1-Dependent Mechanotransduction In Endothelial Cells To Stimulate Transendothelial Migration. [Thesis]. University of North Carolina; 2014. Available from: https://cdr.lib.unc.edu/record/uuid:e0425b15-91e2-46a9-aead-d82aee48dc8b

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

10. Case, Lindsay. SPATIOTEMPORAL COORDINATION OF THE ACTIN CYTOSKELETON AND INTEGRIN ADHESION.

Degree: Cell Biology and Physiology, 2014, University of North Carolina

 Integrin-based adhesions mediate critical interactions between the cell and its external environment. Integrins assemble into macromolecular "focal adhesions" (FAs) that contain hundreds of proteins and… (more)

Subjects/Keywords: Cytology; School of Medicine; Department of Cell Biology and Physiology

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APA (6th Edition):

Case, L. (2014). SPATIOTEMPORAL COORDINATION OF THE ACTIN CYTOSKELETON AND INTEGRIN ADHESION. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:7cda075f-4cd5-4f13-b243-6ce9d9cd0d48

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Case, Lindsay. “SPATIOTEMPORAL COORDINATION OF THE ACTIN CYTOSKELETON AND INTEGRIN ADHESION.” 2014. Thesis, University of North Carolina. Accessed September 27, 2020. https://cdr.lib.unc.edu/record/uuid:7cda075f-4cd5-4f13-b243-6ce9d9cd0d48.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Case, Lindsay. “SPATIOTEMPORAL COORDINATION OF THE ACTIN CYTOSKELETON AND INTEGRIN ADHESION.” 2014. Web. 27 Sep 2020.

Vancouver:

Case L. SPATIOTEMPORAL COORDINATION OF THE ACTIN CYTOSKELETON AND INTEGRIN ADHESION. [Internet] [Thesis]. University of North Carolina; 2014. [cited 2020 Sep 27]. Available from: https://cdr.lib.unc.edu/record/uuid:7cda075f-4cd5-4f13-b243-6ce9d9cd0d48.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Case L. SPATIOTEMPORAL COORDINATION OF THE ACTIN CYTOSKELETON AND INTEGRIN ADHESION. [Thesis]. University of North Carolina; 2014. Available from: https://cdr.lib.unc.edu/record/uuid:7cda075f-4cd5-4f13-b243-6ce9d9cd0d48

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

11. Kechele, Daniel. Novel Functions of Adrenomedullin, Receptor Activity-Modifying Protein 2, and Gpr182 in Cardiovascular Disease and Cancer.

Degree: Cell Biology and Physiology, 2016, University of North Carolina

 The multifunctional circulating peptide, adrenomedullin (AM) signaling through its G protein-coupled receptor (GPCR) complex, comprised of the calcitonin receptor-like receptor (CLR) and receptor activity-modifying protein… (more)

Subjects/Keywords: School of Medicine; Department of Cell Biology and Physiology

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APA (6th Edition):

Kechele, D. (2016). Novel Functions of Adrenomedullin, Receptor Activity-Modifying Protein 2, and Gpr182 in Cardiovascular Disease and Cancer. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:3ea1610a-b414-45d7-86ec-dc7e8f817969

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kechele, Daniel. “Novel Functions of Adrenomedullin, Receptor Activity-Modifying Protein 2, and Gpr182 in Cardiovascular Disease and Cancer.” 2016. Thesis, University of North Carolina. Accessed September 27, 2020. https://cdr.lib.unc.edu/record/uuid:3ea1610a-b414-45d7-86ec-dc7e8f817969.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kechele, Daniel. “Novel Functions of Adrenomedullin, Receptor Activity-Modifying Protein 2, and Gpr182 in Cardiovascular Disease and Cancer.” 2016. Web. 27 Sep 2020.

Vancouver:

Kechele D. Novel Functions of Adrenomedullin, Receptor Activity-Modifying Protein 2, and Gpr182 in Cardiovascular Disease and Cancer. [Internet] [Thesis]. University of North Carolina; 2016. [cited 2020 Sep 27]. Available from: https://cdr.lib.unc.edu/record/uuid:3ea1610a-b414-45d7-86ec-dc7e8f817969.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kechele D. Novel Functions of Adrenomedullin, Receptor Activity-Modifying Protein 2, and Gpr182 in Cardiovascular Disease and Cancer. [Thesis]. University of North Carolina; 2016. Available from: https://cdr.lib.unc.edu/record/uuid:3ea1610a-b414-45d7-86ec-dc7e8f817969

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

12. Grabon, Agnieszka. Atomistic and Cellular Analysis of Start Phosphatidylinositol Transfer Proteins in the Context of the Phospholipid Extraction Mechanism and Phosphoinositide Signaling in the Parasite Toxoplasma.

Degree: Cell Biology and Physiology, 2016, University of North Carolina

 Phosphatidylinositol transfer proteins (PITPs) are essential regulators of the interface between phosphoinositide (PIP) signaling and membrane trafficking in eukaryotic cells. Genetic derangement of PITPs in… (more)

Subjects/Keywords: School of Medicine; Department of Cell Biology and Physiology

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APA (6th Edition):

Grabon, A. (2016). Atomistic and Cellular Analysis of Start Phosphatidylinositol Transfer Proteins in the Context of the Phospholipid Extraction Mechanism and Phosphoinositide Signaling in the Parasite Toxoplasma. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:686007f0-702c-405a-9545-7dafd11bd8fa

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Grabon, Agnieszka. “Atomistic and Cellular Analysis of Start Phosphatidylinositol Transfer Proteins in the Context of the Phospholipid Extraction Mechanism and Phosphoinositide Signaling in the Parasite Toxoplasma.” 2016. Thesis, University of North Carolina. Accessed September 27, 2020. https://cdr.lib.unc.edu/record/uuid:686007f0-702c-405a-9545-7dafd11bd8fa.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Grabon, Agnieszka. “Atomistic and Cellular Analysis of Start Phosphatidylinositol Transfer Proteins in the Context of the Phospholipid Extraction Mechanism and Phosphoinositide Signaling in the Parasite Toxoplasma.” 2016. Web. 27 Sep 2020.

Vancouver:

Grabon A. Atomistic and Cellular Analysis of Start Phosphatidylinositol Transfer Proteins in the Context of the Phospholipid Extraction Mechanism and Phosphoinositide Signaling in the Parasite Toxoplasma. [Internet] [Thesis]. University of North Carolina; 2016. [cited 2020 Sep 27]. Available from: https://cdr.lib.unc.edu/record/uuid:686007f0-702c-405a-9545-7dafd11bd8fa.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Grabon A. Atomistic and Cellular Analysis of Start Phosphatidylinositol Transfer Proteins in the Context of the Phospholipid Extraction Mechanism and Phosphoinositide Signaling in the Parasite Toxoplasma. [Thesis]. University of North Carolina; 2016. Available from: https://cdr.lib.unc.edu/record/uuid:686007f0-702c-405a-9545-7dafd11bd8fa

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

13. Cupp, Timothy. In Vivo Examination of the Molecular Mechanics Underlying Apical Constriction's Initiation in C. elegans Gastrulation.

Degree: Cell Biology and Physiology, 2016, University of North Carolina

 One remarkable finding from research in developmental biology is that surprisingly few cellular behaviors are responsible for the wide variety of morphogenetic events common among… (more)

Subjects/Keywords: School of Medicine; Department of Cell Biology and Physiology

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APA (6th Edition):

Cupp, T. (2016). In Vivo Examination of the Molecular Mechanics Underlying Apical Constriction's Initiation in C. elegans Gastrulation. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:6922a566-d772-4c1d-b88e-fc149f19a83e

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Cupp, Timothy. “In Vivo Examination of the Molecular Mechanics Underlying Apical Constriction's Initiation in C. elegans Gastrulation.” 2016. Thesis, University of North Carolina. Accessed September 27, 2020. https://cdr.lib.unc.edu/record/uuid:6922a566-d772-4c1d-b88e-fc149f19a83e.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Cupp, Timothy. “In Vivo Examination of the Molecular Mechanics Underlying Apical Constriction's Initiation in C. elegans Gastrulation.” 2016. Web. 27 Sep 2020.

Vancouver:

Cupp T. In Vivo Examination of the Molecular Mechanics Underlying Apical Constriction's Initiation in C. elegans Gastrulation. [Internet] [Thesis]. University of North Carolina; 2016. [cited 2020 Sep 27]. Available from: https://cdr.lib.unc.edu/record/uuid:6922a566-d772-4c1d-b88e-fc149f19a83e.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Cupp T. In Vivo Examination of the Molecular Mechanics Underlying Apical Constriction's Initiation in C. elegans Gastrulation. [Thesis]. University of North Carolina; 2016. Available from: https://cdr.lib.unc.edu/record/uuid:6922a566-d772-4c1d-b88e-fc149f19a83e

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

14. Givens, Chris. Hemodynamic Mechanisms Regulating Inflammatory Vascular Remodeling.

Degree: Cell Biology and Physiology, 2017, University of North Carolina

 Hemodynamic forces are critical regulators of vascular health and disease. Shear stress, the frictional force of blood flowing over the endothelium, is a major hemodynamic… (more)

Subjects/Keywords: School of Medicine; Department of Cell Biology and Physiology

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APA (6th Edition):

Givens, C. (2017). Hemodynamic Mechanisms Regulating Inflammatory Vascular Remodeling. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:b23e2bf4-3d53-4768-a9b5-8967ea17d7a9

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Givens, Chris. “Hemodynamic Mechanisms Regulating Inflammatory Vascular Remodeling.” 2017. Thesis, University of North Carolina. Accessed September 27, 2020. https://cdr.lib.unc.edu/record/uuid:b23e2bf4-3d53-4768-a9b5-8967ea17d7a9.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Givens, Chris. “Hemodynamic Mechanisms Regulating Inflammatory Vascular Remodeling.” 2017. Web. 27 Sep 2020.

Vancouver:

Givens C. Hemodynamic Mechanisms Regulating Inflammatory Vascular Remodeling. [Internet] [Thesis]. University of North Carolina; 2017. [cited 2020 Sep 27]. Available from: https://cdr.lib.unc.edu/record/uuid:b23e2bf4-3d53-4768-a9b5-8967ea17d7a9.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Givens C. Hemodynamic Mechanisms Regulating Inflammatory Vascular Remodeling. [Thesis]. University of North Carolina; 2017. Available from: https://cdr.lib.unc.edu/record/uuid:b23e2bf4-3d53-4768-a9b5-8967ea17d7a9

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

15. Watson, Leah. Endo-Exocytic Trafficking in Regulation of Cdc42 Polarity.

Degree: Cell Biology and Physiology, 2014, University of North Carolina

 The precise subcellular localization of the Rho GTPase Cdc42 is essential for its spatial and temporal control of polarized growth and division. In budding yeast,… (more)

Subjects/Keywords: Cytology; Molecular biology; Genetics; School of Medicine; Department of Cell Biology and Physiology

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APA (6th Edition):

Watson, L. (2014). Endo-Exocytic Trafficking in Regulation of Cdc42 Polarity. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:4c013b02-b8cd-42ea-a474-8994b6ddacbc

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Watson, Leah. “Endo-Exocytic Trafficking in Regulation of Cdc42 Polarity.” 2014. Thesis, University of North Carolina. Accessed September 27, 2020. https://cdr.lib.unc.edu/record/uuid:4c013b02-b8cd-42ea-a474-8994b6ddacbc.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Watson, Leah. “Endo-Exocytic Trafficking in Regulation of Cdc42 Polarity.” 2014. Web. 27 Sep 2020.

Vancouver:

Watson L. Endo-Exocytic Trafficking in Regulation of Cdc42 Polarity. [Internet] [Thesis]. University of North Carolina; 2014. [cited 2020 Sep 27]. Available from: https://cdr.lib.unc.edu/record/uuid:4c013b02-b8cd-42ea-a474-8994b6ddacbc.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Watson L. Endo-Exocytic Trafficking in Regulation of Cdc42 Polarity. [Thesis]. University of North Carolina; 2014. Available from: https://cdr.lib.unc.edu/record/uuid:4c013b02-b8cd-42ea-a474-8994b6ddacbc

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

16. Cook, Aaron. Calcium-induced structural rearrangements release autoinhibition in the Rap-GEF, CalDAG-GEFI.

Degree: Biochemistry and Biophysics, 2018, University of North Carolina

 When blood vessels are injured, extracellular matrix is exposed leading to the recruitment and activation of platelets where they aggregate to staunch bleeding. To aggregate,… (more)

Subjects/Keywords: School of Medicine; Department of Biochemistry and Biophysics

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APA (6th Edition):

Cook, A. (2018). Calcium-induced structural rearrangements release autoinhibition in the Rap-GEF, CalDAG-GEFI. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:63251c98-a8a2-4560-8ddb-0842b5b02f18

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Cook, Aaron. “Calcium-induced structural rearrangements release autoinhibition in the Rap-GEF, CalDAG-GEFI.” 2018. Thesis, University of North Carolina. Accessed September 27, 2020. https://cdr.lib.unc.edu/record/uuid:63251c98-a8a2-4560-8ddb-0842b5b02f18.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Cook, Aaron. “Calcium-induced structural rearrangements release autoinhibition in the Rap-GEF, CalDAG-GEFI.” 2018. Web. 27 Sep 2020.

Vancouver:

Cook A. Calcium-induced structural rearrangements release autoinhibition in the Rap-GEF, CalDAG-GEFI. [Internet] [Thesis]. University of North Carolina; 2018. [cited 2020 Sep 27]. Available from: https://cdr.lib.unc.edu/record/uuid:63251c98-a8a2-4560-8ddb-0842b5b02f18.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Cook A. Calcium-induced structural rearrangements release autoinhibition in the Rap-GEF, CalDAG-GEFI. [Thesis]. University of North Carolina; 2018. Available from: https://cdr.lib.unc.edu/record/uuid:63251c98-a8a2-4560-8ddb-0842b5b02f18

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

17. Zwarycz, Bailey. EXTRINSIC REGULATION OF INTESTINAL STEM CELL PROLIFERATION AND DIFFERENTIATION BY NICHE COMPONENTS.

Degree: Cell Biology and Physiology, 2018, University of North Carolina

 The small intestinal epithelium facilitates the absorption of nutrients and provides a barrier against damaging toxins, indigestible contents, and microbes in the intestinal lumen. The… (more)

Subjects/Keywords: School of Medicine; Department of Cell Biology and Physiology

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APA (6th Edition):

Zwarycz, B. (2018). EXTRINSIC REGULATION OF INTESTINAL STEM CELL PROLIFERATION AND DIFFERENTIATION BY NICHE COMPONENTS. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:60ffa5d1-d9d6-4d7a-bf12-d68d7c878011

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Zwarycz, Bailey. “EXTRINSIC REGULATION OF INTESTINAL STEM CELL PROLIFERATION AND DIFFERENTIATION BY NICHE COMPONENTS.” 2018. Thesis, University of North Carolina. Accessed September 27, 2020. https://cdr.lib.unc.edu/record/uuid:60ffa5d1-d9d6-4d7a-bf12-d68d7c878011.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Zwarycz, Bailey. “EXTRINSIC REGULATION OF INTESTINAL STEM CELL PROLIFERATION AND DIFFERENTIATION BY NICHE COMPONENTS.” 2018. Web. 27 Sep 2020.

Vancouver:

Zwarycz B. EXTRINSIC REGULATION OF INTESTINAL STEM CELL PROLIFERATION AND DIFFERENTIATION BY NICHE COMPONENTS. [Internet] [Thesis]. University of North Carolina; 2018. [cited 2020 Sep 27]. Available from: https://cdr.lib.unc.edu/record/uuid:60ffa5d1-d9d6-4d7a-bf12-d68d7c878011.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Zwarycz B. EXTRINSIC REGULATION OF INTESTINAL STEM CELL PROLIFERATION AND DIFFERENTIATION BY NICHE COMPONENTS. [Thesis]. University of North Carolina; 2018. Available from: https://cdr.lib.unc.edu/record/uuid:60ffa5d1-d9d6-4d7a-bf12-d68d7c878011

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

18. Rowell, Temperance. Flavor-Dependent Effects of E-cigarette Liquids and their Chemical Constituents on Lung Epithelial Toxicity and Cell Ca2+ Signaling.

Degree: Cell Biology and Physiology, 2018, University of North Carolina

 E-cigarettes (e-cigs) are a cigarette alternative that do not contain tobacco or tar, and whose e-liquids are available in over 7,000 flavors. While marketed as… (more)

Subjects/Keywords: School of Medicine; Department of Cell Biology and Physiology

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APA (6th Edition):

Rowell, T. (2018). Flavor-Dependent Effects of E-cigarette Liquids and their Chemical Constituents on Lung Epithelial Toxicity and Cell Ca2+ Signaling. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:c954e24e-9b3f-45df-9d7a-e3c0dfad0e30

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Rowell, Temperance. “Flavor-Dependent Effects of E-cigarette Liquids and their Chemical Constituents on Lung Epithelial Toxicity and Cell Ca2+ Signaling.” 2018. Thesis, University of North Carolina. Accessed September 27, 2020. https://cdr.lib.unc.edu/record/uuid:c954e24e-9b3f-45df-9d7a-e3c0dfad0e30.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Rowell, Temperance. “Flavor-Dependent Effects of E-cigarette Liquids and their Chemical Constituents on Lung Epithelial Toxicity and Cell Ca2+ Signaling.” 2018. Web. 27 Sep 2020.

Vancouver:

Rowell T. Flavor-Dependent Effects of E-cigarette Liquids and their Chemical Constituents on Lung Epithelial Toxicity and Cell Ca2+ Signaling. [Internet] [Thesis]. University of North Carolina; 2018. [cited 2020 Sep 27]. Available from: https://cdr.lib.unc.edu/record/uuid:c954e24e-9b3f-45df-9d7a-e3c0dfad0e30.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Rowell T. Flavor-Dependent Effects of E-cigarette Liquids and their Chemical Constituents on Lung Epithelial Toxicity and Cell Ca2+ Signaling. [Thesis]. University of North Carolina; 2018. Available from: https://cdr.lib.unc.edu/record/uuid:c954e24e-9b3f-45df-9d7a-e3c0dfad0e30

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

19. Sallee, Jennifer L. Density-Enhanced Protein Tyrosine Phosphatase 1 (DEP-1) Regulation of Epithelial Cell Adhesions.

Degree: Cell Biology and Physiology, 2008, University of North Carolina

 Cell-cell adhesions are critical to the development and maintenance of multicellular organisms. Increased tyrosine phosphorylation of junctional proteins has been associated with promoting disassembly of… (more)

Subjects/Keywords: School of Medicine; Department of Cell Biology and Physiology

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APA (6th Edition):

Sallee, J. L. (2008). Density-Enhanced Protein Tyrosine Phosphatase 1 (DEP-1) Regulation of Epithelial Cell Adhesions. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:f747be53-261a-4e2c-80cc-a597cabc167b

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Sallee, Jennifer L. “Density-Enhanced Protein Tyrosine Phosphatase 1 (DEP-1) Regulation of Epithelial Cell Adhesions.” 2008. Thesis, University of North Carolina. Accessed September 27, 2020. https://cdr.lib.unc.edu/record/uuid:f747be53-261a-4e2c-80cc-a597cabc167b.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Sallee, Jennifer L. “Density-Enhanced Protein Tyrosine Phosphatase 1 (DEP-1) Regulation of Epithelial Cell Adhesions.” 2008. Web. 27 Sep 2020.

Vancouver:

Sallee JL. Density-Enhanced Protein Tyrosine Phosphatase 1 (DEP-1) Regulation of Epithelial Cell Adhesions. [Internet] [Thesis]. University of North Carolina; 2008. [cited 2020 Sep 27]. Available from: https://cdr.lib.unc.edu/record/uuid:f747be53-261a-4e2c-80cc-a597cabc167b.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Sallee JL. Density-Enhanced Protein Tyrosine Phosphatase 1 (DEP-1) Regulation of Epithelial Cell Adhesions. [Thesis]. University of North Carolina; 2008. Available from: https://cdr.lib.unc.edu/record/uuid:f747be53-261a-4e2c-80cc-a597cabc167b

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

20. Dubash, Adi Dara. Regulation of RhoA GTPase signaling by guanine nucleotide exchange factors in response to extracellular matrix adhesion and DNA damage.

Degree: Cell Biology and Physiology, 2009, University of North Carolina

 The Rho family of GTPases are proteins which regulate a large number of biological functions, including cell proliferation, survival, adhesion, and migration. Rho proteins cycle… (more)

Subjects/Keywords: School of Medicine; Department of Cell Biology and Physiology

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APA (6th Edition):

Dubash, A. D. (2009). Regulation of RhoA GTPase signaling by guanine nucleotide exchange factors in response to extracellular matrix adhesion and DNA damage. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:67b0bbd9-46d8-42a7-adae-fc36997d2e3e

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Dubash, Adi Dara. “Regulation of RhoA GTPase signaling by guanine nucleotide exchange factors in response to extracellular matrix adhesion and DNA damage.” 2009. Thesis, University of North Carolina. Accessed September 27, 2020. https://cdr.lib.unc.edu/record/uuid:67b0bbd9-46d8-42a7-adae-fc36997d2e3e.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Dubash, Adi Dara. “Regulation of RhoA GTPase signaling by guanine nucleotide exchange factors in response to extracellular matrix adhesion and DNA damage.” 2009. Web. 27 Sep 2020.

Vancouver:

Dubash AD. Regulation of RhoA GTPase signaling by guanine nucleotide exchange factors in response to extracellular matrix adhesion and DNA damage. [Internet] [Thesis]. University of North Carolina; 2009. [cited 2020 Sep 27]. Available from: https://cdr.lib.unc.edu/record/uuid:67b0bbd9-46d8-42a7-adae-fc36997d2e3e.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Dubash AD. Regulation of RhoA GTPase signaling by guanine nucleotide exchange factors in response to extracellular matrix adhesion and DNA damage. [Thesis]. University of North Carolina; 2009. Available from: https://cdr.lib.unc.edu/record/uuid:67b0bbd9-46d8-42a7-adae-fc36997d2e3e

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

21. Hart, Stephanie Hall. Vascular endothelial cadherin phosphorylation modulates endothelial cell permeability and leukocyte transendothelial migration.

Degree: Pharmacology, 2008, University of North Carolina

 Leukocyte transendothelial migration (TEM) is a key step in many functions of the immune system such as immune surveillance, inflammation and wound repair. The controlled… (more)

Subjects/Keywords: School of Medicine; Department of Pharmacology

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APA (6th Edition):

Hart, S. H. (2008). Vascular endothelial cadherin phosphorylation modulates endothelial cell permeability and leukocyte transendothelial migration. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:15275d40-be69-4da7-8a73-435e0e9cf49a

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hart, Stephanie Hall. “Vascular endothelial cadherin phosphorylation modulates endothelial cell permeability and leukocyte transendothelial migration.” 2008. Thesis, University of North Carolina. Accessed September 27, 2020. https://cdr.lib.unc.edu/record/uuid:15275d40-be69-4da7-8a73-435e0e9cf49a.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hart, Stephanie Hall. “Vascular endothelial cadherin phosphorylation modulates endothelial cell permeability and leukocyte transendothelial migration.” 2008. Web. 27 Sep 2020.

Vancouver:

Hart SH. Vascular endothelial cadherin phosphorylation modulates endothelial cell permeability and leukocyte transendothelial migration. [Internet] [Thesis]. University of North Carolina; 2008. [cited 2020 Sep 27]. Available from: https://cdr.lib.unc.edu/record/uuid:15275d40-be69-4da7-8a73-435e0e9cf49a.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hart SH. Vascular endothelial cadherin phosphorylation modulates endothelial cell permeability and leukocyte transendothelial migration. [Thesis]. University of North Carolina; 2008. Available from: https://cdr.lib.unc.edu/record/uuid:15275d40-be69-4da7-8a73-435e0e9cf49a

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

22. Garrett, Tiana Aisha. Vascular endothelial growth factor (VEGF) regulation of endothelial cell behavior through the Rho family of small GTPases.

Degree: Cell Biology and Physiology, 2007, University of North Carolina

 Vascular endothelial growth factor (VEGF) is an important signaling molecule in endothelial cell behavior and function during embryogenesis and the maintenance of adult tissues. VEGF… (more)

Subjects/Keywords: School of Medicine; Department of Cell Biology and Physiology

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APA (6th Edition):

Garrett, T. A. (2007). Vascular endothelial growth factor (VEGF) regulation of endothelial cell behavior through the Rho family of small GTPases. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:fc41c80b-4acc-4d93-8c29-a411559da48b

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Garrett, Tiana Aisha. “Vascular endothelial growth factor (VEGF) regulation of endothelial cell behavior through the Rho family of small GTPases.” 2007. Thesis, University of North Carolina. Accessed September 27, 2020. https://cdr.lib.unc.edu/record/uuid:fc41c80b-4acc-4d93-8c29-a411559da48b.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Garrett, Tiana Aisha. “Vascular endothelial growth factor (VEGF) regulation of endothelial cell behavior through the Rho family of small GTPases.” 2007. Web. 27 Sep 2020.

Vancouver:

Garrett TA. Vascular endothelial growth factor (VEGF) regulation of endothelial cell behavior through the Rho family of small GTPases. [Internet] [Thesis]. University of North Carolina; 2007. [cited 2020 Sep 27]. Available from: https://cdr.lib.unc.edu/record/uuid:fc41c80b-4acc-4d93-8c29-a411559da48b.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Garrett TA. Vascular endothelial growth factor (VEGF) regulation of endothelial cell behavior through the Rho family of small GTPases. [Thesis]. University of North Carolina; 2007. Available from: https://cdr.lib.unc.edu/record/uuid:fc41c80b-4acc-4d93-8c29-a411559da48b

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

23. Allingham, Michael John. Novel roles for ICAM1 in leukocyte transendothelial migration.

Degree: Cell Biology and Physiology, 2007, University of North Carolina

 Leukocyte transendothelial migration (TEM) is a critical regulated step in inflammation and is therefore a potential target in the treatment of inflammatory diseases. During TEM,… (more)

Subjects/Keywords: School of Medicine; Department of Cell Biology and Physiology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Allingham, M. J. (2007). Novel roles for ICAM1 in leukocyte transendothelial migration. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:fe05c3f6-c56f-4e69-9039-0f50a1458a5b

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Allingham, Michael John. “Novel roles for ICAM1 in leukocyte transendothelial migration.” 2007. Thesis, University of North Carolina. Accessed September 27, 2020. https://cdr.lib.unc.edu/record/uuid:fe05c3f6-c56f-4e69-9039-0f50a1458a5b.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Allingham, Michael John. “Novel roles for ICAM1 in leukocyte transendothelial migration.” 2007. Web. 27 Sep 2020.

Vancouver:

Allingham MJ. Novel roles for ICAM1 in leukocyte transendothelial migration. [Internet] [Thesis]. University of North Carolina; 2007. [cited 2020 Sep 27]. Available from: https://cdr.lib.unc.edu/record/uuid:fe05c3f6-c56f-4e69-9039-0f50a1458a5b.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Allingham MJ. Novel roles for ICAM1 in leukocyte transendothelial migration. [Thesis]. University of North Carolina; 2007. Available from: https://cdr.lib.unc.edu/record/uuid:fe05c3f6-c56f-4e69-9039-0f50a1458a5b

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

24. Yohe, Marielle E. Regulation of the transforming immortalized mammary protein and its homologs by auto-inhibition and tyrosine phosphorylation.

Degree: Pharmacology, 2007, University of North Carolina

 Dbl-related oncoproteins are guanine nucleotide exchange factors (GEFs) specific for Rho-family GTPases and typically possess tandem Dbl (DH) and pleckstrin homology (PH) domains that act… (more)

Subjects/Keywords: School of Medicine; Department of Pharmacology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Yohe, M. E. (2007). Regulation of the transforming immortalized mammary protein and its homologs by auto-inhibition and tyrosine phosphorylation. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:17c5a702-f51c-47d6-a9e6-9b476967a72e

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Yohe, Marielle E. “Regulation of the transforming immortalized mammary protein and its homologs by auto-inhibition and tyrosine phosphorylation.” 2007. Thesis, University of North Carolina. Accessed September 27, 2020. https://cdr.lib.unc.edu/record/uuid:17c5a702-f51c-47d6-a9e6-9b476967a72e.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Yohe, Marielle E. “Regulation of the transforming immortalized mammary protein and its homologs by auto-inhibition and tyrosine phosphorylation.” 2007. Web. 27 Sep 2020.

Vancouver:

Yohe ME. Regulation of the transforming immortalized mammary protein and its homologs by auto-inhibition and tyrosine phosphorylation. [Internet] [Thesis]. University of North Carolina; 2007. [cited 2020 Sep 27]. Available from: https://cdr.lib.unc.edu/record/uuid:17c5a702-f51c-47d6-a9e6-9b476967a72e.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Yohe ME. Regulation of the transforming immortalized mammary protein and its homologs by auto-inhibition and tyrosine phosphorylation. [Thesis]. University of North Carolina; 2007. Available from: https://cdr.lib.unc.edu/record/uuid:17c5a702-f51c-47d6-a9e6-9b476967a72e

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

25. Mukherjee, Malini. Development and Characterization of Mouse Models for Breast Cancer.

Degree: Biochemistry and Biophysics, 2008, University of North Carolina

 While previous studies using genetically engineered mice (GEM) have indicated potential effects of several aberrations observed in human breast cancer, the combined role of loss… (more)

Subjects/Keywords: School of Medicine; Department of Biochemistry and Biophysics

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Mukherjee, M. (2008). Development and Characterization of Mouse Models for Breast Cancer. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:ff875959-3e33-4c47-8d66-75c262ec4feb

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Mukherjee, Malini. “Development and Characterization of Mouse Models for Breast Cancer.” 2008. Thesis, University of North Carolina. Accessed September 27, 2020. https://cdr.lib.unc.edu/record/uuid:ff875959-3e33-4c47-8d66-75c262ec4feb.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Mukherjee, Malini. “Development and Characterization of Mouse Models for Breast Cancer.” 2008. Web. 27 Sep 2020.

Vancouver:

Mukherjee M. Development and Characterization of Mouse Models for Breast Cancer. [Internet] [Thesis]. University of North Carolina; 2008. [cited 2020 Sep 27]. Available from: https://cdr.lib.unc.edu/record/uuid:ff875959-3e33-4c47-8d66-75c262ec4feb.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Mukherjee M. Development and Characterization of Mouse Models for Breast Cancer. [Thesis]. University of North Carolina; 2008. Available from: https://cdr.lib.unc.edu/record/uuid:ff875959-3e33-4c47-8d66-75c262ec4feb

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

.