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You searched for +publisher:"University of North Carolina" +contributor:("Baldwin, Albert"). Showing records 1 – 19 of 19 total matches.

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University of North Carolina

1. Bang, Deepali. Oncogenic KRAS, GSK-3, NF-κB and TBK1: the interplay and consequences in promoting pancreatic and lung cancer.

Degree: Cell Biology and Physiology, 2014, University of North Carolina

 The development of KRAS targeted therapy has evolved into targeting the complex signaling pathways activated downstream of oncogenic KRAS and associated with disease progression. Recent… (more)

Subjects/Keywords: School of Medicine; Department of Cell Biology and Physiology

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APA (6th Edition):

Bang, D. (2014). Oncogenic KRAS, GSK-3, NF-κB and TBK1: the interplay and consequences in promoting pancreatic and lung cancer. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:44cd018a-4603-47ca-9d75-5478a07d041d

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Bang, Deepali. “Oncogenic KRAS, GSK-3, NF-κB and TBK1: the interplay and consequences in promoting pancreatic and lung cancer.” 2014. Thesis, University of North Carolina. Accessed August 05, 2020. https://cdr.lib.unc.edu/record/uuid:44cd018a-4603-47ca-9d75-5478a07d041d.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Bang, Deepali. “Oncogenic KRAS, GSK-3, NF-κB and TBK1: the interplay and consequences in promoting pancreatic and lung cancer.” 2014. Web. 05 Aug 2020.

Vancouver:

Bang D. Oncogenic KRAS, GSK-3, NF-κB and TBK1: the interplay and consequences in promoting pancreatic and lung cancer. [Internet] [Thesis]. University of North Carolina; 2014. [cited 2020 Aug 05]. Available from: https://cdr.lib.unc.edu/record/uuid:44cd018a-4603-47ca-9d75-5478a07d041d.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Bang D. Oncogenic KRAS, GSK-3, NF-κB and TBK1: the interplay and consequences in promoting pancreatic and lung cancer. [Thesis]. University of North Carolina; 2014. Available from: https://cdr.lib.unc.edu/record/uuid:44cd018a-4603-47ca-9d75-5478a07d041d

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

2. Stein, Sarah. The role of NF-κB in BCR-ABL-driven oncogenic transformation and in hematopoiesis.

Degree: Biology, 2012, University of North Carolina

 Chronic myeloid leukemia is a malignant clonal disorder of hematopoietic stem cells that results in increased and deregulated growth of myeloid cells. About 95% of… (more)

Subjects/Keywords: College of Arts and Sciences; Department of Biology

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APA (6th Edition):

Stein, S. (2012). The role of NF-κB in BCR-ABL-driven oncogenic transformation and in hematopoiesis. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:6c9905ba-038a-41d6-9398-8faaa489e59a

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Stein, Sarah. “The role of NF-κB in BCR-ABL-driven oncogenic transformation and in hematopoiesis.” 2012. Thesis, University of North Carolina. Accessed August 05, 2020. https://cdr.lib.unc.edu/record/uuid:6c9905ba-038a-41d6-9398-8faaa489e59a.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Stein, Sarah. “The role of NF-κB in BCR-ABL-driven oncogenic transformation and in hematopoiesis.” 2012. Web. 05 Aug 2020.

Vancouver:

Stein S. The role of NF-κB in BCR-ABL-driven oncogenic transformation and in hematopoiesis. [Internet] [Thesis]. University of North Carolina; 2012. [cited 2020 Aug 05]. Available from: https://cdr.lib.unc.edu/record/uuid:6c9905ba-038a-41d6-9398-8faaa489e59a.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Stein S. The role of NF-κB in BCR-ABL-driven oncogenic transformation and in hematopoiesis. [Thesis]. University of North Carolina; 2012. Available from: https://cdr.lib.unc.edu/record/uuid:6c9905ba-038a-41d6-9398-8faaa489e59a

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

3. Merkhofer, Evan Carroll. The role of the NF-κB pathway in Her2-overexpressing breast cancer.

Degree: 2010, University of North Carolina

 Overexpression of the membrane-bound receptor tyrosine kinase Her2 (ErbB-2, EGFR2) occurs in approximately 30% of all breast cancers and typically correlates with poor prognosis. Overexpression… (more)

Subjects/Keywords: School of Medicine; Curriculum in Genetics and Molecular Biology

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APA (6th Edition):

Merkhofer, E. C. (2010). The role of the NF-κB pathway in Her2-overexpressing breast cancer. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:5ceae1cf-5b44-495b-9d3f-b073044a35aa

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Merkhofer, Evan Carroll. “The role of the NF-κB pathway in Her2-overexpressing breast cancer.” 2010. Thesis, University of North Carolina. Accessed August 05, 2020. https://cdr.lib.unc.edu/record/uuid:5ceae1cf-5b44-495b-9d3f-b073044a35aa.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Merkhofer, Evan Carroll. “The role of the NF-κB pathway in Her2-overexpressing breast cancer.” 2010. Web. 05 Aug 2020.

Vancouver:

Merkhofer EC. The role of the NF-κB pathway in Her2-overexpressing breast cancer. [Internet] [Thesis]. University of North Carolina; 2010. [cited 2020 Aug 05]. Available from: https://cdr.lib.unc.edu/record/uuid:5ceae1cf-5b44-495b-9d3f-b073044a35aa.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Merkhofer EC. The role of the NF-κB pathway in Her2-overexpressing breast cancer. [Thesis]. University of North Carolina; 2010. Available from: https://cdr.lib.unc.edu/record/uuid:5ceae1cf-5b44-495b-9d3f-b073044a35aa

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

4. Comb, William C. Examining novel roles for the IκB kinase in coordinating the cellular response to metabolic stress.

Degree: 2011, University of North Carolina

 The induction of mammalian autophagy, a conserved cellular bulk-degradation process, was recently shown to require Inhibitor of κB (IκB) Kinase (IKK), the upstream regulator of… (more)

Subjects/Keywords: School of Medicine; Curriculum in Genetics and Molecular Biology

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APA (6th Edition):

Comb, W. C. (2011). Examining novel roles for the IκB kinase in coordinating the cellular response to metabolic stress. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:fb51c2e1-7886-4b7d-9a15-9d5e24ea0230

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Comb, William C. “Examining novel roles for the IκB kinase in coordinating the cellular response to metabolic stress.” 2011. Thesis, University of North Carolina. Accessed August 05, 2020. https://cdr.lib.unc.edu/record/uuid:fb51c2e1-7886-4b7d-9a15-9d5e24ea0230.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Comb, William C. “Examining novel roles for the IκB kinase in coordinating the cellular response to metabolic stress.” 2011. Web. 05 Aug 2020.

Vancouver:

Comb WC. Examining novel roles for the IκB kinase in coordinating the cellular response to metabolic stress. [Internet] [Thesis]. University of North Carolina; 2011. [cited 2020 Aug 05]. Available from: https://cdr.lib.unc.edu/record/uuid:fb51c2e1-7886-4b7d-9a15-9d5e24ea0230.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Comb WC. Examining novel roles for the IκB kinase in coordinating the cellular response to metabolic stress. [Thesis]. University of North Carolina; 2011. Available from: https://cdr.lib.unc.edu/record/uuid:fb51c2e1-7886-4b7d-9a15-9d5e24ea0230

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

5. Rizzardi, Lindsay Faircloth. Epigenetic Factors Are Dynamically Regulated Throughout The Cell Cycle And Are Required For Efficient DNA Replication.

Degree: 2013, University of North Carolina

 Chromatin consists of DNA wrapped around a core of histone proteins that can be modified to elicit distinct cellular responses. All DNA-templated processes including DNA… (more)

Subjects/Keywords: School of Medicine; Curriculum in Genetics and Molecular Biology

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APA (6th Edition):

Rizzardi, L. F. (2013). Epigenetic Factors Are Dynamically Regulated Throughout The Cell Cycle And Are Required For Efficient DNA Replication. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:08380d60-c1fb-48fe-aa17-ba65b9fffae9

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Rizzardi, Lindsay Faircloth. “Epigenetic Factors Are Dynamically Regulated Throughout The Cell Cycle And Are Required For Efficient DNA Replication.” 2013. Thesis, University of North Carolina. Accessed August 05, 2020. https://cdr.lib.unc.edu/record/uuid:08380d60-c1fb-48fe-aa17-ba65b9fffae9.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Rizzardi, Lindsay Faircloth. “Epigenetic Factors Are Dynamically Regulated Throughout The Cell Cycle And Are Required For Efficient DNA Replication.” 2013. Web. 05 Aug 2020.

Vancouver:

Rizzardi LF. Epigenetic Factors Are Dynamically Regulated Throughout The Cell Cycle And Are Required For Efficient DNA Replication. [Internet] [Thesis]. University of North Carolina; 2013. [cited 2020 Aug 05]. Available from: https://cdr.lib.unc.edu/record/uuid:08380d60-c1fb-48fe-aa17-ba65b9fffae9.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Rizzardi LF. Epigenetic Factors Are Dynamically Regulated Throughout The Cell Cycle And Are Required For Efficient DNA Replication. [Thesis]. University of North Carolina; 2013. Available from: https://cdr.lib.unc.edu/record/uuid:08380d60-c1fb-48fe-aa17-ba65b9fffae9

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

6. Maxfield, Kimberly. Genetic Dissection of Therapeutic Intervention Targets in Triple Negative Breast Cancer.

Degree: Pharmacology, 2015, University of North Carolina

 Triple negative breast cancer (TNBC) is the most aggressive and metastatic type of breast cancer, accounting for 20% of all breast cancer diagnoses. Currently, there… (more)

Subjects/Keywords: Pharmacology; Biology; School of Medicine; Department of Pharmacology

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APA (6th Edition):

Maxfield, K. (2015). Genetic Dissection of Therapeutic Intervention Targets in Triple Negative Breast Cancer. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:318b1399-81a7-4737-b0c7-96f242284420

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Maxfield, Kimberly. “Genetic Dissection of Therapeutic Intervention Targets in Triple Negative Breast Cancer.” 2015. Thesis, University of North Carolina. Accessed August 05, 2020. https://cdr.lib.unc.edu/record/uuid:318b1399-81a7-4737-b0c7-96f242284420.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Maxfield, Kimberly. “Genetic Dissection of Therapeutic Intervention Targets in Triple Negative Breast Cancer.” 2015. Web. 05 Aug 2020.

Vancouver:

Maxfield K. Genetic Dissection of Therapeutic Intervention Targets in Triple Negative Breast Cancer. [Internet] [Thesis]. University of North Carolina; 2015. [cited 2020 Aug 05]. Available from: https://cdr.lib.unc.edu/record/uuid:318b1399-81a7-4737-b0c7-96f242284420.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Maxfield K. Genetic Dissection of Therapeutic Intervention Targets in Triple Negative Breast Cancer. [Thesis]. University of North Carolina; 2015. Available from: https://cdr.lib.unc.edu/record/uuid:318b1399-81a7-4737-b0c7-96f242284420

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

7. Fahey, Catherine. The Effect of Cancer-Associated SETD2 Mutations on Transcription and Chromatin Organization.

Degree: 2017, University of North Carolina

 Clear cell renal cell carcinoma is characterized by mutations in chromatin modifying enzymes. Among these is SETD2, a non-redundant histone H3 lysine 36 methyltransferase. Mutations… (more)

Subjects/Keywords: School of Medicine; Curriculum in Genetics and Molecular Biology

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APA (6th Edition):

Fahey, C. (2017). The Effect of Cancer-Associated SETD2 Mutations on Transcription and Chromatin Organization. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:0fa1e41e-c273-499c-8cfd-f30baee6360f

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Fahey, Catherine. “The Effect of Cancer-Associated SETD2 Mutations on Transcription and Chromatin Organization.” 2017. Thesis, University of North Carolina. Accessed August 05, 2020. https://cdr.lib.unc.edu/record/uuid:0fa1e41e-c273-499c-8cfd-f30baee6360f.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Fahey, Catherine. “The Effect of Cancer-Associated SETD2 Mutations on Transcription and Chromatin Organization.” 2017. Web. 05 Aug 2020.

Vancouver:

Fahey C. The Effect of Cancer-Associated SETD2 Mutations on Transcription and Chromatin Organization. [Internet] [Thesis]. University of North Carolina; 2017. [cited 2020 Aug 05]. Available from: https://cdr.lib.unc.edu/record/uuid:0fa1e41e-c273-499c-8cfd-f30baee6360f.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Fahey C. The Effect of Cancer-Associated SETD2 Mutations on Transcription and Chromatin Organization. [Thesis]. University of North Carolina; 2017. Available from: https://cdr.lib.unc.edu/record/uuid:0fa1e41e-c273-499c-8cfd-f30baee6360f

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

8. Hayes, Tikvah. Targeting mutant KRAS in pancreatic cancer.

Degree: 2016, University of North Carolina

 The development of pharmacologic inhibitors of the KRAS oncoprotein, which is mutated in ~30% of all human cancers, has been at the forefront of drug… (more)

Subjects/Keywords: School of Medicine; Curriculum in Genetics and Molecular Biology

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APA (6th Edition):

Hayes, T. (2016). Targeting mutant KRAS in pancreatic cancer. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:f816a0b0-3d43-4afb-9623-dcbfb1d7bd39

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hayes, Tikvah. “Targeting mutant KRAS in pancreatic cancer.” 2016. Thesis, University of North Carolina. Accessed August 05, 2020. https://cdr.lib.unc.edu/record/uuid:f816a0b0-3d43-4afb-9623-dcbfb1d7bd39.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hayes, Tikvah. “Targeting mutant KRAS in pancreatic cancer.” 2016. Web. 05 Aug 2020.

Vancouver:

Hayes T. Targeting mutant KRAS in pancreatic cancer. [Internet] [Thesis]. University of North Carolina; 2016. [cited 2020 Aug 05]. Available from: https://cdr.lib.unc.edu/record/uuid:f816a0b0-3d43-4afb-9623-dcbfb1d7bd39.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hayes T. Targeting mutant KRAS in pancreatic cancer. [Thesis]. University of North Carolina; 2016. Available from: https://cdr.lib.unc.edu/record/uuid:f816a0b0-3d43-4afb-9623-dcbfb1d7bd39

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

9. Rinkenbaugh, Amanda. Roles of the NF-kappaB Pathway in Glioblastoma Stem Cells and Chordoma.

Degree: Pathology and Laboratory Medicine, 2016, University of North Carolina

 The NF-κB pathway consists of a family of five transcription factors: RelA/p65, RelB, c-Rel, p100/p52, and p105/p50. Originally discovered for its involvement in inflammation and… (more)

Subjects/Keywords: School of Medicine; Department of Pathology and Laboratory Medicine

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APA (6th Edition):

Rinkenbaugh, A. (2016). Roles of the NF-kappaB Pathway in Glioblastoma Stem Cells and Chordoma. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:4046c94e-b8aa-4bba-93e0-ac8d1490b55d

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Rinkenbaugh, Amanda. “Roles of the NF-kappaB Pathway in Glioblastoma Stem Cells and Chordoma.” 2016. Thesis, University of North Carolina. Accessed August 05, 2020. https://cdr.lib.unc.edu/record/uuid:4046c94e-b8aa-4bba-93e0-ac8d1490b55d.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Rinkenbaugh, Amanda. “Roles of the NF-kappaB Pathway in Glioblastoma Stem Cells and Chordoma.” 2016. Web. 05 Aug 2020.

Vancouver:

Rinkenbaugh A. Roles of the NF-kappaB Pathway in Glioblastoma Stem Cells and Chordoma. [Internet] [Thesis]. University of North Carolina; 2016. [cited 2020 Aug 05]. Available from: https://cdr.lib.unc.edu/record/uuid:4046c94e-b8aa-4bba-93e0-ac8d1490b55d.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Rinkenbaugh A. Roles of the NF-kappaB Pathway in Glioblastoma Stem Cells and Chordoma. [Thesis]. University of North Carolina; 2016. Available from: https://cdr.lib.unc.edu/record/uuid:4046c94e-b8aa-4bba-93e0-ac8d1490b55d

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

10. Hsia, Hung-Ching. ROLES AND REGULATION OF STAT3 IN INNATE IMMUNITY.

Degree: Cell Biology and Physiology, 2017, University of North Carolina

 Innate immunity is the first line of host defense to microbial infections. The rapid induction of the innate immune response is achieved through recognition of… (more)

Subjects/Keywords: School of Medicine; Department of Cell Biology and Physiology

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APA (6th Edition):

Hsia, H. (2017). ROLES AND REGULATION OF STAT3 IN INNATE IMMUNITY. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:757e9627-ce60-4c50-9567-df37da02c123

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hsia, Hung-Ching. “ROLES AND REGULATION OF STAT3 IN INNATE IMMUNITY.” 2017. Thesis, University of North Carolina. Accessed August 05, 2020. https://cdr.lib.unc.edu/record/uuid:757e9627-ce60-4c50-9567-df37da02c123.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hsia, Hung-Ching. “ROLES AND REGULATION OF STAT3 IN INNATE IMMUNITY.” 2017. Web. 05 Aug 2020.

Vancouver:

Hsia H. ROLES AND REGULATION OF STAT3 IN INNATE IMMUNITY. [Internet] [Thesis]. University of North Carolina; 2017. [cited 2020 Aug 05]. Available from: https://cdr.lib.unc.edu/record/uuid:757e9627-ce60-4c50-9567-df37da02c123.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hsia H. ROLES AND REGULATION OF STAT3 IN INNATE IMMUNITY. [Thesis]. University of North Carolina; 2017. Available from: https://cdr.lib.unc.edu/record/uuid:757e9627-ce60-4c50-9567-df37da02c123

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

11. Cooper, Matthew James. Kinome Profiling of Drug-Resistant Myeloid Leukemia to Guide the Use of Targeted Therapy.

Degree: 2013, University of North Carolina

 Protein kinases are key mediators of cellular signaling. Consequently, dysregulated kinases are central to cancer initiation, progression and acquired drug resistance, and are a major… (more)

Subjects/Keywords: School of Medicine; Curriculum in Genetics and Molecular Biology

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APA (6th Edition):

Cooper, M. J. (2013). Kinome Profiling of Drug-Resistant Myeloid Leukemia to Guide the Use of Targeted Therapy. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:39c80d6c-0cfd-494d-96b1-aea19c48ea22

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Cooper, Matthew James. “Kinome Profiling of Drug-Resistant Myeloid Leukemia to Guide the Use of Targeted Therapy.” 2013. Thesis, University of North Carolina. Accessed August 05, 2020. https://cdr.lib.unc.edu/record/uuid:39c80d6c-0cfd-494d-96b1-aea19c48ea22.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Cooper, Matthew James. “Kinome Profiling of Drug-Resistant Myeloid Leukemia to Guide the Use of Targeted Therapy.” 2013. Web. 05 Aug 2020.

Vancouver:

Cooper MJ. Kinome Profiling of Drug-Resistant Myeloid Leukemia to Guide the Use of Targeted Therapy. [Internet] [Thesis]. University of North Carolina; 2013. [cited 2020 Aug 05]. Available from: https://cdr.lib.unc.edu/record/uuid:39c80d6c-0cfd-494d-96b1-aea19c48ea22.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Cooper MJ. Kinome Profiling of Drug-Resistant Myeloid Leukemia to Guide the Use of Targeted Therapy. [Thesis]. University of North Carolina; 2013. Available from: https://cdr.lib.unc.edu/record/uuid:39c80d6c-0cfd-494d-96b1-aea19c48ea22

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

12. Tollini, Laura. Exploring the In Vivo Role of the Mdm2 Ring Finger Domain in p53 Regulation.

Degree: 2014, University of North Carolina

 Classically characterized as an “overarching” tumor suppressor, p53 is frequently found mutated, deleted, or misregulated in human cancers. Through its role as a transcription factor,… (more)

Subjects/Keywords: Molecular biology; School of Medicine; Curriculum in Genetics and Molecular Biology

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APA (6th Edition):

Tollini, L. (2014). Exploring the In Vivo Role of the Mdm2 Ring Finger Domain in p53 Regulation. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:088fdc11-df44-4c72-958f-f21e90a102f0

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Tollini, Laura. “Exploring the In Vivo Role of the Mdm2 Ring Finger Domain in p53 Regulation.” 2014. Thesis, University of North Carolina. Accessed August 05, 2020. https://cdr.lib.unc.edu/record/uuid:088fdc11-df44-4c72-958f-f21e90a102f0.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Tollini, Laura. “Exploring the In Vivo Role of the Mdm2 Ring Finger Domain in p53 Regulation.” 2014. Web. 05 Aug 2020.

Vancouver:

Tollini L. Exploring the In Vivo Role of the Mdm2 Ring Finger Domain in p53 Regulation. [Internet] [Thesis]. University of North Carolina; 2014. [cited 2020 Aug 05]. Available from: https://cdr.lib.unc.edu/record/uuid:088fdc11-df44-4c72-958f-f21e90a102f0.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Tollini L. Exploring the In Vivo Role of the Mdm2 Ring Finger Domain in p53 Regulation. [Thesis]. University of North Carolina; 2014. Available from: https://cdr.lib.unc.edu/record/uuid:088fdc11-df44-4c72-958f-f21e90a102f0

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

13. Leslie, Patrick. Analysis of the p53 Regulator MDM2 and the Identification of the Novel p53 Target Gene LRP1.

Degree: 2016, University of North Carolina

 The transcription factor p53 responds to many stresses and regulates many different pathways. The earliest characterized functions of p53 include the induction of cell cycle… (more)

Subjects/Keywords: School of Medicine; Curriculum in Genetics and Molecular Biology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Leslie, P. (2016). Analysis of the p53 Regulator MDM2 and the Identification of the Novel p53 Target Gene LRP1. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:49a0f53d-536d-4d50-a5a2-22210a46f820

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Leslie, Patrick. “Analysis of the p53 Regulator MDM2 and the Identification of the Novel p53 Target Gene LRP1.” 2016. Thesis, University of North Carolina. Accessed August 05, 2020. https://cdr.lib.unc.edu/record/uuid:49a0f53d-536d-4d50-a5a2-22210a46f820.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Leslie, Patrick. “Analysis of the p53 Regulator MDM2 and the Identification of the Novel p53 Target Gene LRP1.” 2016. Web. 05 Aug 2020.

Vancouver:

Leslie P. Analysis of the p53 Regulator MDM2 and the Identification of the Novel p53 Target Gene LRP1. [Internet] [Thesis]. University of North Carolina; 2016. [cited 2020 Aug 05]. Available from: https://cdr.lib.unc.edu/record/uuid:49a0f53d-536d-4d50-a5a2-22210a46f820.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Leslie P. Analysis of the p53 Regulator MDM2 and the Identification of the Novel p53 Target Gene LRP1. [Thesis]. University of North Carolina; 2016. Available from: https://cdr.lib.unc.edu/record/uuid:49a0f53d-536d-4d50-a5a2-22210a46f820

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

14. Mulvaney, Kathleen. PROTEOMIC DISSECTION OF KEAP1/NRF2 SIGNALING TO DETERMINE NEW PATHWAY INTERACTORS IN CANCER.

Degree: Cell Biology and Physiology, 2016, University of North Carolina

 KEAP1/NRF2 signaling regulates intracellular reactive oxygen species and protects cells from reactive oxygen-induced damage. KEAP1 serves as the substrate adaptor for a CULLIN3-based E3 ubiquitin… (more)

Subjects/Keywords: School of Medicine; Department of Cell Biology and Physiology

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APA (6th Edition):

Mulvaney, K. (2016). PROTEOMIC DISSECTION OF KEAP1/NRF2 SIGNALING TO DETERMINE NEW PATHWAY INTERACTORS IN CANCER. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:6a38b6e9-c9a6-4280-9816-da904d71b6d0

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Mulvaney, Kathleen. “PROTEOMIC DISSECTION OF KEAP1/NRF2 SIGNALING TO DETERMINE NEW PATHWAY INTERACTORS IN CANCER.” 2016. Thesis, University of North Carolina. Accessed August 05, 2020. https://cdr.lib.unc.edu/record/uuid:6a38b6e9-c9a6-4280-9816-da904d71b6d0.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Mulvaney, Kathleen. “PROTEOMIC DISSECTION OF KEAP1/NRF2 SIGNALING TO DETERMINE NEW PATHWAY INTERACTORS IN CANCER.” 2016. Web. 05 Aug 2020.

Vancouver:

Mulvaney K. PROTEOMIC DISSECTION OF KEAP1/NRF2 SIGNALING TO DETERMINE NEW PATHWAY INTERACTORS IN CANCER. [Internet] [Thesis]. University of North Carolina; 2016. [cited 2020 Aug 05]. Available from: https://cdr.lib.unc.edu/record/uuid:6a38b6e9-c9a6-4280-9816-da904d71b6d0.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Mulvaney K. PROTEOMIC DISSECTION OF KEAP1/NRF2 SIGNALING TO DETERMINE NEW PATHWAY INTERACTORS IN CANCER. [Thesis]. University of North Carolina; 2016. Available from: https://cdr.lib.unc.edu/record/uuid:6a38b6e9-c9a6-4280-9816-da904d71b6d0

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

15. Bailey, Sean. Inhibition of compensatory survival and proliferative pathway activation induced by mTOR inhibition in renal cell carcinoma.

Degree: 2014, University of North Carolina

 The mammalian target of rapamycin (mTOR) is a key regulator of tumor progression in a variety of cancers and has been shown to be dysregulated… (more)

Subjects/Keywords: Molecular biology; Genetics; School of Medicine; Curriculum in Genetics and Molecular Biology

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APA (6th Edition):

Bailey, S. (2014). Inhibition of compensatory survival and proliferative pathway activation induced by mTOR inhibition in renal cell carcinoma. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:3ecdd41c-8862-4504-a14a-8ead22d81d74

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Bailey, Sean. “Inhibition of compensatory survival and proliferative pathway activation induced by mTOR inhibition in renal cell carcinoma.” 2014. Thesis, University of North Carolina. Accessed August 05, 2020. https://cdr.lib.unc.edu/record/uuid:3ecdd41c-8862-4504-a14a-8ead22d81d74.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Bailey, Sean. “Inhibition of compensatory survival and proliferative pathway activation induced by mTOR inhibition in renal cell carcinoma.” 2014. Web. 05 Aug 2020.

Vancouver:

Bailey S. Inhibition of compensatory survival and proliferative pathway activation induced by mTOR inhibition in renal cell carcinoma. [Internet] [Thesis]. University of North Carolina; 2014. [cited 2020 Aug 05]. Available from: https://cdr.lib.unc.edu/record/uuid:3ecdd41c-8862-4504-a14a-8ead22d81d74.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Bailey S. Inhibition of compensatory survival and proliferative pathway activation induced by mTOR inhibition in renal cell carcinoma. [Thesis]. University of North Carolina; 2014. Available from: https://cdr.lib.unc.edu/record/uuid:3ecdd41c-8862-4504-a14a-8ead22d81d74

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

16. Adli, Mezher. Investigating the role of IKKε (epsilon) in cancer-associated NF-κB activity.

Degree: Biology, 2007, University of North Carolina

 Nuclear Factor κB (NF-κB) has been studied extensively as an inducible transcriptional regulator of the immune and inflammatory responses. NF-κB activation downstream of LPS or… (more)

Subjects/Keywords: College of Arts and Sciences; Department of Biology

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APA (6th Edition):

Adli, M. (2007). Investigating the role of IKKε (epsilon) in cancer-associated NF-κB activity. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:1ba6ce64-e440-4012-8144-4559ef68836f

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Adli, Mezher. “Investigating the role of IKKε (epsilon) in cancer-associated NF-κB activity.” 2007. Thesis, University of North Carolina. Accessed August 05, 2020. https://cdr.lib.unc.edu/record/uuid:1ba6ce64-e440-4012-8144-4559ef68836f.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Adli, Mezher. “Investigating the role of IKKε (epsilon) in cancer-associated NF-κB activity.” 2007. Web. 05 Aug 2020.

Vancouver:

Adli M. Investigating the role of IKKε (epsilon) in cancer-associated NF-κB activity. [Internet] [Thesis]. University of North Carolina; 2007. [cited 2020 Aug 05]. Available from: https://cdr.lib.unc.edu/record/uuid:1ba6ce64-e440-4012-8144-4559ef68836f.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Adli M. Investigating the role of IKKε (epsilon) in cancer-associated NF-κB activity. [Thesis]. University of North Carolina; 2007. Available from: https://cdr.lib.unc.edu/record/uuid:1ba6ce64-e440-4012-8144-4559ef68836f

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

17. Kashatus, David. An Investigation of the Function of IκB Family Member Bcl-3 and its Role in Oncogenesis.

Degree: 2006, University of North Carolina

 Bcl-3 was originally identified as a protein that is highly expressed in certain B-cell lymphomas harboring a 14;19 translocation, and more recent evidence indicates that… (more)

Subjects/Keywords: School of Medicine; Curriculum in Genetics and Molecular Biology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Kashatus, D. (2006). An Investigation of the Function of IκB Family Member Bcl-3 and its Role in Oncogenesis. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:336bcb0e-ed02-4745-a284-9110fa5adcc6

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kashatus, David. “An Investigation of the Function of IκB Family Member Bcl-3 and its Role in Oncogenesis.” 2006. Thesis, University of North Carolina. Accessed August 05, 2020. https://cdr.lib.unc.edu/record/uuid:336bcb0e-ed02-4745-a284-9110fa5adcc6.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kashatus, David. “An Investigation of the Function of IκB Family Member Bcl-3 and its Role in Oncogenesis.” 2006. Web. 05 Aug 2020.

Vancouver:

Kashatus D. An Investigation of the Function of IκB Family Member Bcl-3 and its Role in Oncogenesis. [Internet] [Thesis]. University of North Carolina; 2006. [cited 2020 Aug 05]. Available from: https://cdr.lib.unc.edu/record/uuid:336bcb0e-ed02-4745-a284-9110fa5adcc6.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kashatus D. An Investigation of the Function of IκB Family Member Bcl-3 and its Role in Oncogenesis. [Thesis]. University of North Carolina; 2006. Available from: https://cdr.lib.unc.edu/record/uuid:336bcb0e-ed02-4745-a284-9110fa5adcc6

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

18. Wilson, Willie. The regulation of constitutive NF-κB activity by glycogen synthase kinase-3 in pancreatic cancer.

Degree: 2009, University of North Carolina

 The development of pancreatic cancer chemotherapy has evolved into targeting the complex signaling pathways associated with disease progression. Recent focus has been made on targeting… (more)

Subjects/Keywords: School of Medicine; Curriculum in Genetics and Molecular Biology

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APA (6th Edition):

Wilson, W. (2009). The regulation of constitutive NF-κB activity by glycogen synthase kinase-3 in pancreatic cancer. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:73ea7216-e073-4451-931c-cdd3dfdda4d9

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Wilson, Willie. “The regulation of constitutive NF-κB activity by glycogen synthase kinase-3 in pancreatic cancer.” 2009. Thesis, University of North Carolina. Accessed August 05, 2020. https://cdr.lib.unc.edu/record/uuid:73ea7216-e073-4451-931c-cdd3dfdda4d9.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Wilson, Willie. “The regulation of constitutive NF-κB activity by glycogen synthase kinase-3 in pancreatic cancer.” 2009. Web. 05 Aug 2020.

Vancouver:

Wilson W. The regulation of constitutive NF-κB activity by glycogen synthase kinase-3 in pancreatic cancer. [Internet] [Thesis]. University of North Carolina; 2009. [cited 2020 Aug 05]. Available from: https://cdr.lib.unc.edu/record/uuid:73ea7216-e073-4451-931c-cdd3dfdda4d9.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Wilson W. The regulation of constitutive NF-κB activity by glycogen synthase kinase-3 in pancreatic cancer. [Thesis]. University of North Carolina; 2009. Available from: https://cdr.lib.unc.edu/record/uuid:73ea7216-e073-4451-931c-cdd3dfdda4d9

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

19. Cheely, Adam Webster. Regulation and functional impact of lipopolysaccharide induced Nod2 gene expression in the murine epididymal epithelial cell line PC1.

Degree: Biology, 2008, University of North Carolina

 In this study we investigated the regulation and function of Nod2 in the immortalized epididymal epithelial cell line PC1 (proximal caput 1) following lipopolysaccharide (LPS)… (more)

Subjects/Keywords: College of Arts and Sciences; Department of Biology

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APA (6th Edition):

Cheely, A. W. (2008). Regulation and functional impact of lipopolysaccharide induced Nod2 gene expression in the murine epididymal epithelial cell line PC1. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:38ac30d2-66ce-426f-b79a-7fde74fccf34

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Cheely, Adam Webster. “Regulation and functional impact of lipopolysaccharide induced Nod2 gene expression in the murine epididymal epithelial cell line PC1.” 2008. Thesis, University of North Carolina. Accessed August 05, 2020. https://cdr.lib.unc.edu/record/uuid:38ac30d2-66ce-426f-b79a-7fde74fccf34.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Cheely, Adam Webster. “Regulation and functional impact of lipopolysaccharide induced Nod2 gene expression in the murine epididymal epithelial cell line PC1.” 2008. Web. 05 Aug 2020.

Vancouver:

Cheely AW. Regulation and functional impact of lipopolysaccharide induced Nod2 gene expression in the murine epididymal epithelial cell line PC1. [Internet] [Thesis]. University of North Carolina; 2008. [cited 2020 Aug 05]. Available from: https://cdr.lib.unc.edu/record/uuid:38ac30d2-66ce-426f-b79a-7fde74fccf34.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Cheely AW. Regulation and functional impact of lipopolysaccharide induced Nod2 gene expression in the murine epididymal epithelial cell line PC1. [Thesis]. University of North Carolina; 2008. Available from: https://cdr.lib.unc.edu/record/uuid:38ac30d2-66ce-426f-b79a-7fde74fccf34

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

.