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You searched for +publisher:"University of North Carolina" +contributor:("Anderson, James M."). Showing records 1 – 4 of 4 total matches.

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University of North Carolina

1. Rollins, Brett. Regulation of the Epithelial Sodium Channel (ENaC) by the Short Palate, Lung, and Nasal Epithelial Clone (SPLUNC1).

Degree: Cell Biology and Physiology, 2010, University of North Carolina

 The airways rely on mucociliary clearance (MCC) to remove inhaled particulates. Initiating events in chronic airway diseases, such as cystic fibrosis (CF), have been traced… (more)

Subjects/Keywords: School of Medicine; Department of Cell Biology and Physiology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Rollins, B. (2010). Regulation of the Epithelial Sodium Channel (ENaC) by the Short Palate, Lung, and Nasal Epithelial Clone (SPLUNC1). (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:23a7af78-feaa-4a84-8f8f-be2a38b9c449

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Rollins, Brett. “Regulation of the Epithelial Sodium Channel (ENaC) by the Short Palate, Lung, and Nasal Epithelial Clone (SPLUNC1).” 2010. Thesis, University of North Carolina. Accessed September 24, 2020. https://cdr.lib.unc.edu/record/uuid:23a7af78-feaa-4a84-8f8f-be2a38b9c449.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Rollins, Brett. “Regulation of the Epithelial Sodium Channel (ENaC) by the Short Palate, Lung, and Nasal Epithelial Clone (SPLUNC1).” 2010. Web. 24 Sep 2020.

Vancouver:

Rollins B. Regulation of the Epithelial Sodium Channel (ENaC) by the Short Palate, Lung, and Nasal Epithelial Clone (SPLUNC1). [Internet] [Thesis]. University of North Carolina; 2010. [cited 2020 Sep 24]. Available from: https://cdr.lib.unc.edu/record/uuid:23a7af78-feaa-4a84-8f8f-be2a38b9c449.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Rollins B. Regulation of the Epithelial Sodium Channel (ENaC) by the Short Palate, Lung, and Nasal Epithelial Clone (SPLUNC1). [Thesis]. University of North Carolina; 2010. Available from: https://cdr.lib.unc.edu/record/uuid:23a7af78-feaa-4a84-8f8f-be2a38b9c449

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

2. Rasmussen, Julia E. Claudins and Regulation of the Paracellular Transport System.

Degree: Cell Biology and Physiology, 2006, University of North Carolina

 Epithelia create barriers and regulate solute and water transport through transcellular and paracellular pathways. The paracellular pathways of different tissues vary remarkably in size selectivity,… (more)

Subjects/Keywords: School of Medicine; Department of Cell Biology and Physiology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Rasmussen, J. E. (2006). Claudins and Regulation of the Paracellular Transport System. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:c5fa670d-53c1-49cd-8372-ff508f85019c

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Rasmussen, Julia E. “Claudins and Regulation of the Paracellular Transport System.” 2006. Thesis, University of North Carolina. Accessed September 24, 2020. https://cdr.lib.unc.edu/record/uuid:c5fa670d-53c1-49cd-8372-ff508f85019c.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Rasmussen, Julia E. “Claudins and Regulation of the Paracellular Transport System.” 2006. Web. 24 Sep 2020.

Vancouver:

Rasmussen JE. Claudins and Regulation of the Paracellular Transport System. [Internet] [Thesis]. University of North Carolina; 2006. [cited 2020 Sep 24]. Available from: https://cdr.lib.unc.edu/record/uuid:c5fa670d-53c1-49cd-8372-ff508f85019c.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Rasmussen JE. Claudins and Regulation of the Paracellular Transport System. [Thesis]. University of North Carolina; 2006. Available from: https://cdr.lib.unc.edu/record/uuid:c5fa670d-53c1-49cd-8372-ff508f85019c

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

3. Jacobs, Damon. Myosin 5c is a class V myosin that functions in secretory granule trafficking.

Degree: Cell Biology and Physiology, 2008, University of North Carolina

 Myosin motor proteins are a super family of mechanoenzymes that carry out diverse functions in plants and animals. Class V myosins power the movement of… (more)

Subjects/Keywords: School of Medicine; Department of Cell Biology and Physiology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Jacobs, D. (2008). Myosin 5c is a class V myosin that functions in secretory granule trafficking. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:3539c3d8-3029-4bbc-a9f3-fe836240ee48

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Jacobs, Damon. “Myosin 5c is a class V myosin that functions in secretory granule trafficking.” 2008. Thesis, University of North Carolina. Accessed September 24, 2020. https://cdr.lib.unc.edu/record/uuid:3539c3d8-3029-4bbc-a9f3-fe836240ee48.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Jacobs, Damon. “Myosin 5c is a class V myosin that functions in secretory granule trafficking.” 2008. Web. 24 Sep 2020.

Vancouver:

Jacobs D. Myosin 5c is a class V myosin that functions in secretory granule trafficking. [Internet] [Thesis]. University of North Carolina; 2008. [cited 2020 Sep 24]. Available from: https://cdr.lib.unc.edu/record/uuid:3539c3d8-3029-4bbc-a9f3-fe836240ee48.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Jacobs D. Myosin 5c is a class V myosin that functions in secretory granule trafficking. [Thesis]. University of North Carolina; 2008. Available from: https://cdr.lib.unc.edu/record/uuid:3539c3d8-3029-4bbc-a9f3-fe836240ee48

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

4. Brady, Donita. The transforming Rho family GTPase, Wrch-1, regulates epithelial cell morphogenesis through modulating cell junctions and actin cytoskeletal dynamics.

Degree: Pharmacology, 2008, University of North Carolina

 Rho GTPases are members of the Ras superfamily of GTP binding proteins that function as molecular switches. In their GTP-bound, active state, Rho proteins are… (more)

Subjects/Keywords: School of Medicine; Department of Pharmacology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Brady, D. (2008). The transforming Rho family GTPase, Wrch-1, regulates epithelial cell morphogenesis through modulating cell junctions and actin cytoskeletal dynamics. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:0e2e6907-aef0-4d56-8361-dc656a966d8a

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Brady, Donita. “The transforming Rho family GTPase, Wrch-1, regulates epithelial cell morphogenesis through modulating cell junctions and actin cytoskeletal dynamics.” 2008. Thesis, University of North Carolina. Accessed September 24, 2020. https://cdr.lib.unc.edu/record/uuid:0e2e6907-aef0-4d56-8361-dc656a966d8a.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Brady, Donita. “The transforming Rho family GTPase, Wrch-1, regulates epithelial cell morphogenesis through modulating cell junctions and actin cytoskeletal dynamics.” 2008. Web. 24 Sep 2020.

Vancouver:

Brady D. The transforming Rho family GTPase, Wrch-1, regulates epithelial cell morphogenesis through modulating cell junctions and actin cytoskeletal dynamics. [Internet] [Thesis]. University of North Carolina; 2008. [cited 2020 Sep 24]. Available from: https://cdr.lib.unc.edu/record/uuid:0e2e6907-aef0-4d56-8361-dc656a966d8a.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Brady D. The transforming Rho family GTPase, Wrch-1, regulates epithelial cell morphogenesis through modulating cell junctions and actin cytoskeletal dynamics. [Thesis]. University of North Carolina; 2008. Available from: https://cdr.lib.unc.edu/record/uuid:0e2e6907-aef0-4d56-8361-dc656a966d8a

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

.