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You searched for +publisher:"University of New Mexico" +contributor:("Wandinger-Ness, Angela"). Showing records 1 – 10 of 10 total matches.

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University of New Mexico

1. Guo, Yuna. R-ketorolac targets Cdc42 and Rac1 and alters ovarian cancer behaviors critical for invasion and metastasis.

Degree: Biomedical Sciences Graduate Program, 2015, University of New Mexico

  Cdc42 (cell division control protein 42) and Rac1 (Ras-related C3 botulinum) are attractive therapeutic targets in ovarian cancer based on their established importance in… (more)

Subjects/Keywords: Rho GTPases; ketorolac; ovarian cancer; NSAIDs; Cdc42; Rac1; Medicine and Health Sciences

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APA (6th Edition):

Guo, Y. (2015). R-ketorolac targets Cdc42 and Rac1 and alters ovarian cancer behaviors critical for invasion and metastasis. (Doctoral Dissertation). University of New Mexico. Retrieved from https://digitalrepository.unm.edu/biom_etds/127

Chicago Manual of Style (16th Edition):

Guo, Yuna. “R-ketorolac targets Cdc42 and Rac1 and alters ovarian cancer behaviors critical for invasion and metastasis.” 2015. Doctoral Dissertation, University of New Mexico. Accessed October 23, 2019. https://digitalrepository.unm.edu/biom_etds/127.

MLA Handbook (7th Edition):

Guo, Yuna. “R-ketorolac targets Cdc42 and Rac1 and alters ovarian cancer behaviors critical for invasion and metastasis.” 2015. Web. 23 Oct 2019.

Vancouver:

Guo Y. R-ketorolac targets Cdc42 and Rac1 and alters ovarian cancer behaviors critical for invasion and metastasis. [Internet] [Doctoral dissertation]. University of New Mexico; 2015. [cited 2019 Oct 23]. Available from: https://digitalrepository.unm.edu/biom_etds/127.

Council of Science Editors:

Guo Y. R-ketorolac targets Cdc42 and Rac1 and alters ovarian cancer behaviors critical for invasion and metastasis. [Doctoral Dissertation]. University of New Mexico; 2015. Available from: https://digitalrepository.unm.edu/biom_etds/127


University of New Mexico

2. Peretti, Amanda. The effects of ketorolac and its enantiomers on breast cancer proliferation and metastasis.

Degree: Biomedical Sciences Graduate Program, 2015, University of New Mexico

  Breast cancer is the second leading cause of cancer related deaths in women. Advanced breast cancer can metastasize to the lungs, liver, bones and… (more)

Subjects/Keywords: breast cancer; cancer; ketorolac; metastasis; NSAID; Medicine and Health Sciences

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APA (6th Edition):

Peretti, A. (2015). The effects of ketorolac and its enantiomers on breast cancer proliferation and metastasis. (Masters Thesis). University of New Mexico. Retrieved from https://digitalrepository.unm.edu/biom_etds/100

Chicago Manual of Style (16th Edition):

Peretti, Amanda. “The effects of ketorolac and its enantiomers on breast cancer proliferation and metastasis.” 2015. Masters Thesis, University of New Mexico. Accessed October 23, 2019. https://digitalrepository.unm.edu/biom_etds/100.

MLA Handbook (7th Edition):

Peretti, Amanda. “The effects of ketorolac and its enantiomers on breast cancer proliferation and metastasis.” 2015. Web. 23 Oct 2019.

Vancouver:

Peretti A. The effects of ketorolac and its enantiomers on breast cancer proliferation and metastasis. [Internet] [Masters thesis]. University of New Mexico; 2015. [cited 2019 Oct 23]. Available from: https://digitalrepository.unm.edu/biom_etds/100.

Council of Science Editors:

Peretti A. The effects of ketorolac and its enantiomers on breast cancer proliferation and metastasis. [Masters Thesis]. University of New Mexico; 2015. Available from: https://digitalrepository.unm.edu/biom_etds/100


University of New Mexico

3. Schwartz, Samantha. Visualizing Mast Cell Activation: Single Molecule Dynamics of Early Events in FceRI Signaling.

Degree: Nanoscience and Microsystems, 2016, University of New Mexico

  Healthy immune cell behavior requires sensitive and robust control over the processes that regulate signal transduction. In this work we employ single molecule fluorescence… (more)

Subjects/Keywords: IgE Signaling; Single Molecule Flourescence Microscopy; Super-resolution Fluorescence Microscopy; Mast cell Signaling; Nanoscience and Nanotechnology

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APA (6th Edition):

Schwartz, S. (2016). Visualizing Mast Cell Activation: Single Molecule Dynamics of Early Events in FceRI Signaling. (Doctoral Dissertation). University of New Mexico. Retrieved from http://hdl.handle.net/1928/32334

Chicago Manual of Style (16th Edition):

Schwartz, Samantha. “Visualizing Mast Cell Activation: Single Molecule Dynamics of Early Events in FceRI Signaling.” 2016. Doctoral Dissertation, University of New Mexico. Accessed October 23, 2019. http://hdl.handle.net/1928/32334.

MLA Handbook (7th Edition):

Schwartz, Samantha. “Visualizing Mast Cell Activation: Single Molecule Dynamics of Early Events in FceRI Signaling.” 2016. Web. 23 Oct 2019.

Vancouver:

Schwartz S. Visualizing Mast Cell Activation: Single Molecule Dynamics of Early Events in FceRI Signaling. [Internet] [Doctoral dissertation]. University of New Mexico; 2016. [cited 2019 Oct 23]. Available from: http://hdl.handle.net/1928/32334.

Council of Science Editors:

Schwartz S. Visualizing Mast Cell Activation: Single Molecule Dynamics of Early Events in FceRI Signaling. [Doctoral Dissertation]. University of New Mexico; 2016. Available from: http://hdl.handle.net/1928/32334


University of New Mexico

4. Chavez, Miquella. Dynamics of cell-cell junctions in keratinocytes.

Degree: Biomedical Sciences Graduate Program, 2009, University of New Mexico

 Poor wound healing is a serious medical issue of particular concern in the elderly and people with diabetes. One major obstacle for these patients to… (more)

Subjects/Keywords: keratinocytes; wound healing

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APA (6th Edition):

Chavez, M. (2009). Dynamics of cell-cell junctions in keratinocytes. (Doctoral Dissertation). University of New Mexico. Retrieved from https://digitalrepository.unm.edu/biom_etds/28

Chicago Manual of Style (16th Edition):

Chavez, Miquella. “Dynamics of cell-cell junctions in keratinocytes.” 2009. Doctoral Dissertation, University of New Mexico. Accessed October 23, 2019. https://digitalrepository.unm.edu/biom_etds/28.

MLA Handbook (7th Edition):

Chavez, Miquella. “Dynamics of cell-cell junctions in keratinocytes.” 2009. Web. 23 Oct 2019.

Vancouver:

Chavez M. Dynamics of cell-cell junctions in keratinocytes. [Internet] [Doctoral dissertation]. University of New Mexico; 2009. [cited 2019 Oct 23]. Available from: https://digitalrepository.unm.edu/biom_etds/28.

Council of Science Editors:

Chavez M. Dynamics of cell-cell junctions in keratinocytes. [Doctoral Dissertation]. University of New Mexico; 2009. Available from: https://digitalrepository.unm.edu/biom_etds/28


University of New Mexico

5. Zekas, Erin. GPER-MEDIATED REGULATION OF NUCLEAR AKT/FOXO3A SIGNALING.

Degree: Biomedical Sciences Graduate Program, 2014, University of New Mexico

 17β-estradiol (estrogen) has been demonstrated to regulate survival in breast cancer cells, which is partially mediated by its nuclear receptors ERα and ERβ and the… (more)

Subjects/Keywords: Breast Cancer Cell Signaling

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APA (6th Edition):

Zekas, E. (2014). GPER-MEDIATED REGULATION OF NUCLEAR AKT/FOXO3A SIGNALING. (Doctoral Dissertation). University of New Mexico. Retrieved from https://digitalrepository.unm.edu/biom_etds/153

Chicago Manual of Style (16th Edition):

Zekas, Erin. “GPER-MEDIATED REGULATION OF NUCLEAR AKT/FOXO3A SIGNALING.” 2014. Doctoral Dissertation, University of New Mexico. Accessed October 23, 2019. https://digitalrepository.unm.edu/biom_etds/153.

MLA Handbook (7th Edition):

Zekas, Erin. “GPER-MEDIATED REGULATION OF NUCLEAR AKT/FOXO3A SIGNALING.” 2014. Web. 23 Oct 2019.

Vancouver:

Zekas E. GPER-MEDIATED REGULATION OF NUCLEAR AKT/FOXO3A SIGNALING. [Internet] [Doctoral dissertation]. University of New Mexico; 2014. [cited 2019 Oct 23]. Available from: https://digitalrepository.unm.edu/biom_etds/153.

Council of Science Editors:

Zekas E. GPER-MEDIATED REGULATION OF NUCLEAR AKT/FOXO3A SIGNALING. [Doctoral Dissertation]. University of New Mexico; 2014. Available from: https://digitalrepository.unm.edu/biom_etds/153


University of New Mexico

6. Agola, Jacob. Assessment of functional characteristics of small GTPases using small molecules.

Degree: Biomedical Sciences Graduate Program, 2011, University of New Mexico

 Rab and Rho subfamilies of GTPases are functionally linked to intracellular trafficking and organization of the cytoskeleton respectively. Despite their roles, use of small molecule… (more)

Subjects/Keywords: Rab; Rho; Rac; Cdc42 and Ras GTPases; drug discovery; fluorescent GTP and GDP; enzyme kinetics; GEF; flow cytometry; ovarian cancer; competitive and allosteric inhibitors

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APA (6th Edition):

Agola, J. (2011). Assessment of functional characteristics of small GTPases using small molecules. (Doctoral Dissertation). University of New Mexico. Retrieved from https://digitalrepository.unm.edu/biom_etds/108

Chicago Manual of Style (16th Edition):

Agola, Jacob. “Assessment of functional characteristics of small GTPases using small molecules.” 2011. Doctoral Dissertation, University of New Mexico. Accessed October 23, 2019. https://digitalrepository.unm.edu/biom_etds/108.

MLA Handbook (7th Edition):

Agola, Jacob. “Assessment of functional characteristics of small GTPases using small molecules.” 2011. Web. 23 Oct 2019.

Vancouver:

Agola J. Assessment of functional characteristics of small GTPases using small molecules. [Internet] [Doctoral dissertation]. University of New Mexico; 2011. [cited 2019 Oct 23]. Available from: https://digitalrepository.unm.edu/biom_etds/108.

Council of Science Editors:

Agola J. Assessment of functional characteristics of small GTPases using small molecules. [Doctoral Dissertation]. University of New Mexico; 2011. Available from: https://digitalrepository.unm.edu/biom_etds/108


University of New Mexico

7. Kenney, Shelby. Characterization of R-ketorolac as a single enantiomer selective inhibitor of Cdc42 and Rac1 in ovarian cancer.

Degree: Biomedical Sciences Graduate Program, 2015, University of New Mexico

 Ovarian cancer is the 5th leading cause of cancer death for women in the United States and is frequently diagnosed at an advanced stage with… (more)

Subjects/Keywords: Cdc42; Rac1; ovarian cancer; ketorolac; migration; adhesion; Rho GTPases

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APA (6th Edition):

Kenney, S. (2015). Characterization of R-ketorolac as a single enantiomer selective inhibitor of Cdc42 and Rac1 in ovarian cancer. (Doctoral Dissertation). University of New Mexico. Retrieved from https://digitalrepository.unm.edu/biom_etds/132

Chicago Manual of Style (16th Edition):

Kenney, Shelby. “Characterization of R-ketorolac as a single enantiomer selective inhibitor of Cdc42 and Rac1 in ovarian cancer.” 2015. Doctoral Dissertation, University of New Mexico. Accessed October 23, 2019. https://digitalrepository.unm.edu/biom_etds/132.

MLA Handbook (7th Edition):

Kenney, Shelby. “Characterization of R-ketorolac as a single enantiomer selective inhibitor of Cdc42 and Rac1 in ovarian cancer.” 2015. Web. 23 Oct 2019.

Vancouver:

Kenney S. Characterization of R-ketorolac as a single enantiomer selective inhibitor of Cdc42 and Rac1 in ovarian cancer. [Internet] [Doctoral dissertation]. University of New Mexico; 2015. [cited 2019 Oct 23]. Available from: https://digitalrepository.unm.edu/biom_etds/132.

Council of Science Editors:

Kenney S. Characterization of R-ketorolac as a single enantiomer selective inhibitor of Cdc42 and Rac1 in ovarian cancer. [Doctoral Dissertation]. University of New Mexico; 2015. Available from: https://digitalrepository.unm.edu/biom_etds/132


University of New Mexico

8. Franco Nitta, Carolina. Crosstalk between adipocytes and immune cells in adipose tissue in an obese inflammatory state: role of contact-mediated signaling.

Degree: Biomedical Sciences Graduate Program, 2013, University of New Mexico

  Obesity is defined as heightened fat accumulation leading to health impairments. It has been directly correlated to cardiovascular disease, type II diabetes mellitus, and… (more)

Subjects/Keywords: obesity; inflammation; cytokine; adipose tissue; immune cell; signaling; cell adhesion molecule; Medicine and Health Sciences

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APA (6th Edition):

Franco Nitta, C. (2013). Crosstalk between adipocytes and immune cells in adipose tissue in an obese inflammatory state: role of contact-mediated signaling. (Doctoral Dissertation). University of New Mexico. Retrieved from https://digitalrepository.unm.edu/biom_etds/125

Chicago Manual of Style (16th Edition):

Franco Nitta, Carolina. “Crosstalk between adipocytes and immune cells in adipose tissue in an obese inflammatory state: role of contact-mediated signaling.” 2013. Doctoral Dissertation, University of New Mexico. Accessed October 23, 2019. https://digitalrepository.unm.edu/biom_etds/125.

MLA Handbook (7th Edition):

Franco Nitta, Carolina. “Crosstalk between adipocytes and immune cells in adipose tissue in an obese inflammatory state: role of contact-mediated signaling.” 2013. Web. 23 Oct 2019.

Vancouver:

Franco Nitta C. Crosstalk between adipocytes and immune cells in adipose tissue in an obese inflammatory state: role of contact-mediated signaling. [Internet] [Doctoral dissertation]. University of New Mexico; 2013. [cited 2019 Oct 23]. Available from: https://digitalrepository.unm.edu/biom_etds/125.

Council of Science Editors:

Franco Nitta C. Crosstalk between adipocytes and immune cells in adipose tissue in an obese inflammatory state: role of contact-mediated signaling. [Doctoral Dissertation]. University of New Mexico; 2013. Available from: https://digitalrepository.unm.edu/biom_etds/125


University of New Mexico

9. Reed, Jamie. Assessment of the biocompatibility, stability, and suitability of novel thermoresponsive films for the rapid generation of cellular constructs.

Degree: Chemical and Biological Engineering, 2011, University of New Mexico

 Stimuli responsive polymers (SRP) are of great interest in the bioengineering community due to their use in applications such as drug delivery and tissue engineering.… (more)

Subjects/Keywords: Thermoresponsive; pNIPAM; Biocompatibility; XPS; Surface analysis; Spheroids; Acrylimide – Thermal properties.; Attachment mechanisms (Biology); Acrylamide – Biocompatibility.; Bioengineering – Materials.

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APA (6th Edition):

Reed, J. (2011). Assessment of the biocompatibility, stability, and suitability of novel thermoresponsive films for the rapid generation of cellular constructs. (Doctoral Dissertation). University of New Mexico. Retrieved from http://hdl.handle.net/1928/12865

Chicago Manual of Style (16th Edition):

Reed, Jamie. “Assessment of the biocompatibility, stability, and suitability of novel thermoresponsive films for the rapid generation of cellular constructs.” 2011. Doctoral Dissertation, University of New Mexico. Accessed October 23, 2019. http://hdl.handle.net/1928/12865.

MLA Handbook (7th Edition):

Reed, Jamie. “Assessment of the biocompatibility, stability, and suitability of novel thermoresponsive films for the rapid generation of cellular constructs.” 2011. Web. 23 Oct 2019.

Vancouver:

Reed J. Assessment of the biocompatibility, stability, and suitability of novel thermoresponsive films for the rapid generation of cellular constructs. [Internet] [Doctoral dissertation]. University of New Mexico; 2011. [cited 2019 Oct 23]. Available from: http://hdl.handle.net/1928/12865.

Council of Science Editors:

Reed J. Assessment of the biocompatibility, stability, and suitability of novel thermoresponsive films for the rapid generation of cellular constructs. [Doctoral Dissertation]. University of New Mexico; 2011. Available from: http://hdl.handle.net/1928/12865


University of New Mexico

10. BasuRay, Soumik. Role of Rab7 in the pathogenesis of Charcot-Marie-Tooth Type2B (CMT2B) Disease.

Degree: Biomedical Sciences Graduate Program, 2013, University of New Mexico

 Charcot-Marie-Tooth (CMT) Disease is a group of peripheral neuropathies affecting 1 in 2500 individuals. It is characterized by progressive sensory loss and distal muscle weakness… (more)

Subjects/Keywords: Rab7; CMT2B disease; peripheral neuropathy; endosomal trafficking; endosomal signaling; NGF; TrkA; EGF; EGFR; p38; ERK; c-fos; Egr-1

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APA (6th Edition):

BasuRay, S. (2013). Role of Rab7 in the pathogenesis of Charcot-Marie-Tooth Type2B (CMT2B) Disease. (Doctoral Dissertation). University of New Mexico. Retrieved from https://digitalrepository.unm.edu/biom_etds/112

Chicago Manual of Style (16th Edition):

BasuRay, Soumik. “Role of Rab7 in the pathogenesis of Charcot-Marie-Tooth Type2B (CMT2B) Disease.” 2013. Doctoral Dissertation, University of New Mexico. Accessed October 23, 2019. https://digitalrepository.unm.edu/biom_etds/112.

MLA Handbook (7th Edition):

BasuRay, Soumik. “Role of Rab7 in the pathogenesis of Charcot-Marie-Tooth Type2B (CMT2B) Disease.” 2013. Web. 23 Oct 2019.

Vancouver:

BasuRay S. Role of Rab7 in the pathogenesis of Charcot-Marie-Tooth Type2B (CMT2B) Disease. [Internet] [Doctoral dissertation]. University of New Mexico; 2013. [cited 2019 Oct 23]. Available from: https://digitalrepository.unm.edu/biom_etds/112.

Council of Science Editors:

BasuRay S. Role of Rab7 in the pathogenesis of Charcot-Marie-Tooth Type2B (CMT2B) Disease. [Doctoral Dissertation]. University of New Mexico; 2013. Available from: https://digitalrepository.unm.edu/biom_etds/112

.