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You searched for +publisher:"University of New Mexico" +contributor:("Hathaway, Helen"). Showing records 1 – 20 of 20 total matches.

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University of New Mexico

1. Scaling, Allison. Characterization of G protein-coupled estrogen receptor (GPER) in breast epithelial proliferation and morphogenesis.

Degree: Biomedical Sciences Graduate Program, 2012, University of New Mexico

  Estrogen (17β-estradiol, E2) plays an important role in regulating an array of functions in both male and female reproductive physiology. In the mammary gland,… (more)

Subjects/Keywords: "GPER; GPR30; Estrogen; Estrogen Receptor; Breast; Proliferation"; Medicine and Health Sciences

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APA (6th Edition):

Scaling, A. (2012). Characterization of G protein-coupled estrogen receptor (GPER) in breast epithelial proliferation and morphogenesis. (Doctoral Dissertation). University of New Mexico. Retrieved from https://digitalrepository.unm.edu/biom_etds/57

Chicago Manual of Style (16th Edition):

Scaling, Allison. “Characterization of G protein-coupled estrogen receptor (GPER) in breast epithelial proliferation and morphogenesis.” 2012. Doctoral Dissertation, University of New Mexico. Accessed February 25, 2020. https://digitalrepository.unm.edu/biom_etds/57.

MLA Handbook (7th Edition):

Scaling, Allison. “Characterization of G protein-coupled estrogen receptor (GPER) in breast epithelial proliferation and morphogenesis.” 2012. Web. 25 Feb 2020.

Vancouver:

Scaling A. Characterization of G protein-coupled estrogen receptor (GPER) in breast epithelial proliferation and morphogenesis. [Internet] [Doctoral dissertation]. University of New Mexico; 2012. [cited 2020 Feb 25]. Available from: https://digitalrepository.unm.edu/biom_etds/57.

Council of Science Editors:

Scaling A. Characterization of G protein-coupled estrogen receptor (GPER) in breast epithelial proliferation and morphogenesis. [Doctoral Dissertation]. University of New Mexico; 2012. Available from: https://digitalrepository.unm.edu/biom_etds/57


University of New Mexico

2. Peretti, Amanda. The effects of ketorolac and its enantiomers on breast cancer proliferation and metastasis.

Degree: Biomedical Sciences Graduate Program, 2015, University of New Mexico

  Breast cancer is the second leading cause of cancer related deaths in women. Advanced breast cancer can metastasize to the lungs, liver, bones and… (more)

Subjects/Keywords: breast cancer; cancer; ketorolac; metastasis; NSAID; Medicine and Health Sciences

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APA (6th Edition):

Peretti, A. (2015). The effects of ketorolac and its enantiomers on breast cancer proliferation and metastasis. (Masters Thesis). University of New Mexico. Retrieved from https://digitalrepository.unm.edu/biom_etds/100

Chicago Manual of Style (16th Edition):

Peretti, Amanda. “The effects of ketorolac and its enantiomers on breast cancer proliferation and metastasis.” 2015. Masters Thesis, University of New Mexico. Accessed February 25, 2020. https://digitalrepository.unm.edu/biom_etds/100.

MLA Handbook (7th Edition):

Peretti, Amanda. “The effects of ketorolac and its enantiomers on breast cancer proliferation and metastasis.” 2015. Web. 25 Feb 2020.

Vancouver:

Peretti A. The effects of ketorolac and its enantiomers on breast cancer proliferation and metastasis. [Internet] [Masters thesis]. University of New Mexico; 2015. [cited 2020 Feb 25]. Available from: https://digitalrepository.unm.edu/biom_etds/100.

Council of Science Editors:

Peretti A. The effects of ketorolac and its enantiomers on breast cancer proliferation and metastasis. [Masters Thesis]. University of New Mexico; 2015. Available from: https://digitalrepository.unm.edu/biom_etds/100


University of New Mexico

3. Dennis, Megan. Identification and characterization of steroid receptor ligands.

Degree: Biomedical Sciences Graduate Program, 2010, University of New Mexico

 This work focuses on steroid receptors, including the androgen receptor (AR), the estrogen receptors (ERs) ERα and ERβ, known as the classical ERs, and GPR30,… (more)

Subjects/Keywords: Estrogen; Androgen; GPR30; GPER; Small molecule discovery; High throughput screening

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APA (6th Edition):

Dennis, M. (2010). Identification and characterization of steroid receptor ligands. (Doctoral Dissertation). University of New Mexico. Retrieved from https://digitalrepository.unm.edu/biom_etds/11

Chicago Manual of Style (16th Edition):

Dennis, Megan. “Identification and characterization of steroid receptor ligands.” 2010. Doctoral Dissertation, University of New Mexico. Accessed February 25, 2020. https://digitalrepository.unm.edu/biom_etds/11.

MLA Handbook (7th Edition):

Dennis, Megan. “Identification and characterization of steroid receptor ligands.” 2010. Web. 25 Feb 2020.

Vancouver:

Dennis M. Identification and characterization of steroid receptor ligands. [Internet] [Doctoral dissertation]. University of New Mexico; 2010. [cited 2020 Feb 25]. Available from: https://digitalrepository.unm.edu/biom_etds/11.

Council of Science Editors:

Dennis M. Identification and characterization of steroid receptor ligands. [Doctoral Dissertation]. University of New Mexico; 2010. Available from: https://digitalrepository.unm.edu/biom_etds/11


University of New Mexico

4. Wu, Yuehan. Regulation of cyclin E1 by the breast cancer microenvironment.

Degree: Biomedical Sciences Graduate Program, 2010, University of New Mexico

 The behavior of breast epithelial cells is influenced by their microenvironment, which includes stromal cells and extracellular matrix. During breast cancer progression, the tissue microenvironment… (more)

Subjects/Keywords: cyclin E1; breast cancer; cell cycle; microenvironment

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APA (6th Edition):

Wu, Y. (2010). Regulation of cyclin E1 by the breast cancer microenvironment. (Doctoral Dissertation). University of New Mexico. Retrieved from https://digitalrepository.unm.edu/biom_etds/19

Chicago Manual of Style (16th Edition):

Wu, Yuehan. “Regulation of cyclin E1 by the breast cancer microenvironment.” 2010. Doctoral Dissertation, University of New Mexico. Accessed February 25, 2020. https://digitalrepository.unm.edu/biom_etds/19.

MLA Handbook (7th Edition):

Wu, Yuehan. “Regulation of cyclin E1 by the breast cancer microenvironment.” 2010. Web. 25 Feb 2020.

Vancouver:

Wu Y. Regulation of cyclin E1 by the breast cancer microenvironment. [Internet] [Doctoral dissertation]. University of New Mexico; 2010. [cited 2020 Feb 25]. Available from: https://digitalrepository.unm.edu/biom_etds/19.

Council of Science Editors:

Wu Y. Regulation of cyclin E1 by the breast cancer microenvironment. [Doctoral Dissertation]. University of New Mexico; 2010. Available from: https://digitalrepository.unm.edu/biom_etds/19


University of New Mexico

5. Rogers, Jason Hugh. Development of CD19 binding reagents for targeted nanoparticles.

Degree: Biomedical Sciences Graduate Program, 2013, University of New Mexico

 B-cell malignancies like Acute Lymphoblastic Leukemia (B-ALL), which often have high numbers of malignant cells in circulation in the blood would be an excellent model… (more)

Subjects/Keywords: leukemia; CD19; nanoparticle; phage display; ScFv antibody

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APA (6th Edition):

Rogers, J. H. (2013). Development of CD19 binding reagents for targeted nanoparticles. (Masters Thesis). University of New Mexico. Retrieved from https://digitalrepository.unm.edu/biom_etds/73

Chicago Manual of Style (16th Edition):

Rogers, Jason Hugh. “Development of CD19 binding reagents for targeted nanoparticles.” 2013. Masters Thesis, University of New Mexico. Accessed February 25, 2020. https://digitalrepository.unm.edu/biom_etds/73.

MLA Handbook (7th Edition):

Rogers, Jason Hugh. “Development of CD19 binding reagents for targeted nanoparticles.” 2013. Web. 25 Feb 2020.

Vancouver:

Rogers JH. Development of CD19 binding reagents for targeted nanoparticles. [Internet] [Masters thesis]. University of New Mexico; 2013. [cited 2020 Feb 25]. Available from: https://digitalrepository.unm.edu/biom_etds/73.

Council of Science Editors:

Rogers JH. Development of CD19 binding reagents for targeted nanoparticles. [Masters Thesis]. University of New Mexico; 2013. Available from: https://digitalrepository.unm.edu/biom_etds/73


University of New Mexico

6. Murton, Jaclyn. Targeted Cancer Nanotherapeutics by Adoptive Transfer of Mononuclear Splenocytes.

Degree: Biomedical Sciences Graduate Program, 2014, University of New Mexico

 The most common cause of cancer deaths among women is breast cancer. Current breast cancer chemotherapies rely on passive diffusion of the drug into the… (more)

Subjects/Keywords: "Nanotherapeutics; Protocells; Monocytes; Macrophages"

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APA (6th Edition):

Murton, J. (2014). Targeted Cancer Nanotherapeutics by Adoptive Transfer of Mononuclear Splenocytes. (Masters Thesis). University of New Mexico. Retrieved from https://digitalrepository.unm.edu/biom_etds/81

Chicago Manual of Style (16th Edition):

Murton, Jaclyn. “Targeted Cancer Nanotherapeutics by Adoptive Transfer of Mononuclear Splenocytes.” 2014. Masters Thesis, University of New Mexico. Accessed February 25, 2020. https://digitalrepository.unm.edu/biom_etds/81.

MLA Handbook (7th Edition):

Murton, Jaclyn. “Targeted Cancer Nanotherapeutics by Adoptive Transfer of Mononuclear Splenocytes.” 2014. Web. 25 Feb 2020.

Vancouver:

Murton J. Targeted Cancer Nanotherapeutics by Adoptive Transfer of Mononuclear Splenocytes. [Internet] [Masters thesis]. University of New Mexico; 2014. [cited 2020 Feb 25]. Available from: https://digitalrepository.unm.edu/biom_etds/81.

Council of Science Editors:

Murton J. Targeted Cancer Nanotherapeutics by Adoptive Transfer of Mononuclear Splenocytes. [Masters Thesis]. University of New Mexico; 2014. Available from: https://digitalrepository.unm.edu/biom_etds/81


University of New Mexico

7. Wagener, Brant. Regulation of N-formyl peptide receptor trafficking and signaling by arrestins.

Degree: Biomedical Sciences Graduate Program, 2009, University of New Mexico

 This work focuses on how arrestin regulates trafficking and signaling of the N-formyl peptide receptor (FPR), a G protein-coupled receptor (GPCR). GPCRs are involved in… (more)

Subjects/Keywords: arrestin; FPR; apoptosis; signaling; trafficking; recycling

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APA (6th Edition):

Wagener, B. (2009). Regulation of N-formyl peptide receptor trafficking and signaling by arrestins. (Doctoral Dissertation). University of New Mexico. Retrieved from https://digitalrepository.unm.edu/biom_etds/105

Chicago Manual of Style (16th Edition):

Wagener, Brant. “Regulation of N-formyl peptide receptor trafficking and signaling by arrestins.” 2009. Doctoral Dissertation, University of New Mexico. Accessed February 25, 2020. https://digitalrepository.unm.edu/biom_etds/105.

MLA Handbook (7th Edition):

Wagener, Brant. “Regulation of N-formyl peptide receptor trafficking and signaling by arrestins.” 2009. Web. 25 Feb 2020.

Vancouver:

Wagener B. Regulation of N-formyl peptide receptor trafficking and signaling by arrestins. [Internet] [Doctoral dissertation]. University of New Mexico; 2009. [cited 2020 Feb 25]. Available from: https://digitalrepository.unm.edu/biom_etds/105.

Council of Science Editors:

Wagener B. Regulation of N-formyl peptide receptor trafficking and signaling by arrestins. [Doctoral Dissertation]. University of New Mexico; 2009. Available from: https://digitalrepository.unm.edu/biom_etds/105


University of New Mexico

8. Alcon, Sara. G-Protein Coupled Estrogen Receptor Regulation of Migration and Metastasis in the Breast.

Degree: Biomedical Sciences Graduate Program, 2014, University of New Mexico

 Proliferation and migration are critical steps within normal mammary development and breast cancer progression. While 17β-estradiol (E2) stimulates proliferation in normal and breast cancer cells… (more)

Subjects/Keywords: Breast Cancer; Fibroblasts; GPER

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APA (6th Edition):

Alcon, S. (2014). G-Protein Coupled Estrogen Receptor Regulation of Migration and Metastasis in the Breast. (Doctoral Dissertation). University of New Mexico. Retrieved from https://digitalrepository.unm.edu/biom_etds/109

Chicago Manual of Style (16th Edition):

Alcon, Sara. “G-Protein Coupled Estrogen Receptor Regulation of Migration and Metastasis in the Breast.” 2014. Doctoral Dissertation, University of New Mexico. Accessed February 25, 2020. https://digitalrepository.unm.edu/biom_etds/109.

MLA Handbook (7th Edition):

Alcon, Sara. “G-Protein Coupled Estrogen Receptor Regulation of Migration and Metastasis in the Breast.” 2014. Web. 25 Feb 2020.

Vancouver:

Alcon S. G-Protein Coupled Estrogen Receptor Regulation of Migration and Metastasis in the Breast. [Internet] [Doctoral dissertation]. University of New Mexico; 2014. [cited 2020 Feb 25]. Available from: https://digitalrepository.unm.edu/biom_etds/109.

Council of Science Editors:

Alcon S. G-Protein Coupled Estrogen Receptor Regulation of Migration and Metastasis in the Breast. [Doctoral Dissertation]. University of New Mexico; 2014. Available from: https://digitalrepository.unm.edu/biom_etds/109


University of New Mexico

9. Flook, Adam. Novel α-MSH Peptide Analogs for Melanoma Targeting.

Degree: Biomedical Sciences Graduate Program, 2014, University of New Mexico

 Skin cancer is the one of the most diagnosed cancers in the United States with increasing incidence over the past two decades. There are three… (more)

Subjects/Keywords: cancer biology; Melanocortin-1 receptor; alpha-melanocyte stimulating hormone; melanoma imaging; melanoma targeting; melanoma

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APA (6th Edition):

Flook, A. (2014). Novel α-MSH Peptide Analogs for Melanoma Targeting. (Doctoral Dissertation). University of New Mexico. Retrieved from https://digitalrepository.unm.edu/biom_etds/124

Chicago Manual of Style (16th Edition):

Flook, Adam. “Novel α-MSH Peptide Analogs for Melanoma Targeting.” 2014. Doctoral Dissertation, University of New Mexico. Accessed February 25, 2020. https://digitalrepository.unm.edu/biom_etds/124.

MLA Handbook (7th Edition):

Flook, Adam. “Novel α-MSH Peptide Analogs for Melanoma Targeting.” 2014. Web. 25 Feb 2020.

Vancouver:

Flook A. Novel α-MSH Peptide Analogs for Melanoma Targeting. [Internet] [Doctoral dissertation]. University of New Mexico; 2014. [cited 2020 Feb 25]. Available from: https://digitalrepository.unm.edu/biom_etds/124.

Council of Science Editors:

Flook A. Novel α-MSH Peptide Analogs for Melanoma Targeting. [Doctoral Dissertation]. University of New Mexico; 2014. Available from: https://digitalrepository.unm.edu/biom_etds/124


University of New Mexico

10. Staples, Miranda. Consequences of Prenatal Ethanol Exposure and Maternal Stress on Offspring.

Degree: Biomedical Sciences Graduate Program, 2013, University of New Mexico

 Extensive evidence in humans suggests that exposure to insults during gestation, such as ethanol or maternal stress, can negatively impact the developing fetus in manners… (more)

Subjects/Keywords: Prenatal Ethanol; Prenatal Stress; rat; learning; anxiety

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APA (6th Edition):

Staples, M. (2013). Consequences of Prenatal Ethanol Exposure and Maternal Stress on Offspring. (Doctoral Dissertation). University of New Mexico. Retrieved from https://digitalrepository.unm.edu/biom_etds/144

Chicago Manual of Style (16th Edition):

Staples, Miranda. “Consequences of Prenatal Ethanol Exposure and Maternal Stress on Offspring.” 2013. Doctoral Dissertation, University of New Mexico. Accessed February 25, 2020. https://digitalrepository.unm.edu/biom_etds/144.

MLA Handbook (7th Edition):

Staples, Miranda. “Consequences of Prenatal Ethanol Exposure and Maternal Stress on Offspring.” 2013. Web. 25 Feb 2020.

Vancouver:

Staples M. Consequences of Prenatal Ethanol Exposure and Maternal Stress on Offspring. [Internet] [Doctoral dissertation]. University of New Mexico; 2013. [cited 2020 Feb 25]. Available from: https://digitalrepository.unm.edu/biom_etds/144.

Council of Science Editors:

Staples M. Consequences of Prenatal Ethanol Exposure and Maternal Stress on Offspring. [Doctoral Dissertation]. University of New Mexico; 2013. Available from: https://digitalrepository.unm.edu/biom_etds/144


University of New Mexico

11. Zekas, Erin. GPER-MEDIATED REGULATION OF NUCLEAR AKT/FOXO3A SIGNALING.

Degree: Biomedical Sciences Graduate Program, 2014, University of New Mexico

 17β-estradiol (estrogen) has been demonstrated to regulate survival in breast cancer cells, which is partially mediated by its nuclear receptors ERα and ERβ and the… (more)

Subjects/Keywords: Breast Cancer Cell Signaling

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APA (6th Edition):

Zekas, E. (2014). GPER-MEDIATED REGULATION OF NUCLEAR AKT/FOXO3A SIGNALING. (Doctoral Dissertation). University of New Mexico. Retrieved from https://digitalrepository.unm.edu/biom_etds/153

Chicago Manual of Style (16th Edition):

Zekas, Erin. “GPER-MEDIATED REGULATION OF NUCLEAR AKT/FOXO3A SIGNALING.” 2014. Doctoral Dissertation, University of New Mexico. Accessed February 25, 2020. https://digitalrepository.unm.edu/biom_etds/153.

MLA Handbook (7th Edition):

Zekas, Erin. “GPER-MEDIATED REGULATION OF NUCLEAR AKT/FOXO3A SIGNALING.” 2014. Web. 25 Feb 2020.

Vancouver:

Zekas E. GPER-MEDIATED REGULATION OF NUCLEAR AKT/FOXO3A SIGNALING. [Internet] [Doctoral dissertation]. University of New Mexico; 2014. [cited 2020 Feb 25]. Available from: https://digitalrepository.unm.edu/biom_etds/153.

Council of Science Editors:

Zekas E. GPER-MEDIATED REGULATION OF NUCLEAR AKT/FOXO3A SIGNALING. [Doctoral Dissertation]. University of New Mexico; 2014. Available from: https://digitalrepository.unm.edu/biom_etds/153


University of New Mexico

12. Griego, Anastacia. Evaluation of the Epidermal Growth Factor Receptor Signaling Pathway as a Therapeutic Target in Human Papillomavirus-Associated Disease.

Degree: Biomedical Sciences Graduate Program, 2016, University of New Mexico

  Human papillomaviruses (HPVs) are the most common sexually transmitted infectious agents. They are responsible for >99% of all cervical cancers as well as subsets… (more)

Subjects/Keywords: HPV; EGFR; growth factor receptor; human papillomavirus; cetuximab; cancer; Medicine and Health Sciences

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APA (6th Edition):

Griego, A. (2016). Evaluation of the Epidermal Growth Factor Receptor Signaling Pathway as a Therapeutic Target in Human Papillomavirus-Associated Disease. (Doctoral Dissertation). University of New Mexico. Retrieved from https://digitalrepository.unm.edu/biom_etds/155

Chicago Manual of Style (16th Edition):

Griego, Anastacia. “Evaluation of the Epidermal Growth Factor Receptor Signaling Pathway as a Therapeutic Target in Human Papillomavirus-Associated Disease.” 2016. Doctoral Dissertation, University of New Mexico. Accessed February 25, 2020. https://digitalrepository.unm.edu/biom_etds/155.

MLA Handbook (7th Edition):

Griego, Anastacia. “Evaluation of the Epidermal Growth Factor Receptor Signaling Pathway as a Therapeutic Target in Human Papillomavirus-Associated Disease.” 2016. Web. 25 Feb 2020.

Vancouver:

Griego A. Evaluation of the Epidermal Growth Factor Receptor Signaling Pathway as a Therapeutic Target in Human Papillomavirus-Associated Disease. [Internet] [Doctoral dissertation]. University of New Mexico; 2016. [cited 2020 Feb 25]. Available from: https://digitalrepository.unm.edu/biom_etds/155.

Council of Science Editors:

Griego A. Evaluation of the Epidermal Growth Factor Receptor Signaling Pathway as a Therapeutic Target in Human Papillomavirus-Associated Disease. [Doctoral Dissertation]. University of New Mexico; 2016. Available from: https://digitalrepository.unm.edu/biom_etds/155


University of New Mexico

13. Marjon, Nicole. In Vivo Characterization of G Protein-Coupled Estrogen Receptor in Mammary Tumorigenesis.

Degree: Biomedical Sciences Graduate Program, 2015, University of New Mexico

  The steroid hormone, estrogen (17β-estradiol or E2), is involved in numerous and varied physiological processes. Until recently, all E2-dependent effects were thought to be… (more)

Subjects/Keywords: Breast Cancer; G protein-coupled estrogen receptor; metastasis; microenvironment; small molecule therapy; Medicine and Health Sciences

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APA (6th Edition):

Marjon, N. (2015). In Vivo Characterization of G Protein-Coupled Estrogen Receptor in Mammary Tumorigenesis. (Doctoral Dissertation). University of New Mexico. Retrieved from https://digitalrepository.unm.edu/biom_etds/137

Chicago Manual of Style (16th Edition):

Marjon, Nicole. “In Vivo Characterization of G Protein-Coupled Estrogen Receptor in Mammary Tumorigenesis.” 2015. Doctoral Dissertation, University of New Mexico. Accessed February 25, 2020. https://digitalrepository.unm.edu/biom_etds/137.

MLA Handbook (7th Edition):

Marjon, Nicole. “In Vivo Characterization of G Protein-Coupled Estrogen Receptor in Mammary Tumorigenesis.” 2015. Web. 25 Feb 2020.

Vancouver:

Marjon N. In Vivo Characterization of G Protein-Coupled Estrogen Receptor in Mammary Tumorigenesis. [Internet] [Doctoral dissertation]. University of New Mexico; 2015. [cited 2020 Feb 25]. Available from: https://digitalrepository.unm.edu/biom_etds/137.

Council of Science Editors:

Marjon N. In Vivo Characterization of G Protein-Coupled Estrogen Receptor in Mammary Tumorigenesis. [Doctoral Dissertation]. University of New Mexico; 2015. Available from: https://digitalrepository.unm.edu/biom_etds/137


University of New Mexico

14. White, Kirsten Anne Meyer. Oncogene-dependent regulation of autophagic flux in melanoma.

Degree: Biomedical Sciences Graduate Program, 2015, University of New Mexico

  Skin cancer is the one of the most diagnosed cancers in the United States with increasing incidence of 6% every year. In 2015, it… (more)

Subjects/Keywords: Autophagy; melanoma; oncogenes; Medicine and Health Sciences

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APA (6th Edition):

White, K. A. M. (2015). Oncogene-dependent regulation of autophagic flux in melanoma. (Doctoral Dissertation). University of New Mexico. Retrieved from https://digitalrepository.unm.edu/biom_etds/150

Chicago Manual of Style (16th Edition):

White, Kirsten Anne Meyer. “Oncogene-dependent regulation of autophagic flux in melanoma.” 2015. Doctoral Dissertation, University of New Mexico. Accessed February 25, 2020. https://digitalrepository.unm.edu/biom_etds/150.

MLA Handbook (7th Edition):

White, Kirsten Anne Meyer. “Oncogene-dependent regulation of autophagic flux in melanoma.” 2015. Web. 25 Feb 2020.

Vancouver:

White KAM. Oncogene-dependent regulation of autophagic flux in melanoma. [Internet] [Doctoral dissertation]. University of New Mexico; 2015. [cited 2020 Feb 25]. Available from: https://digitalrepository.unm.edu/biom_etds/150.

Council of Science Editors:

White KAM. Oncogene-dependent regulation of autophagic flux in melanoma. [Doctoral Dissertation]. University of New Mexico; 2015. Available from: https://digitalrepository.unm.edu/biom_etds/150


University of New Mexico

15. Fredette, Natalie C. The Role of G protein-coupled estrogen receptor in vascular function and hypertension.

Degree: Biomedical Sciences Graduate Program, 2015, University of New Mexico

  Regulation of arterial tone relies heavily on molecular crosstalk between endothelial and smooth muscle cells. Although it has been demonstrated that estrogen (E2) exerts… (more)

Subjects/Keywords: superoxide; G protein coupled estrogen receptor; nitric oxide; hypertension; Medicine and Health Sciences

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APA (6th Edition):

Fredette, N. C. (2015). The Role of G protein-coupled estrogen receptor in vascular function and hypertension. (Doctoral Dissertation). University of New Mexico. Retrieved from https://digitalrepository.unm.edu/biom_etds/126

Chicago Manual of Style (16th Edition):

Fredette, Natalie C. “The Role of G protein-coupled estrogen receptor in vascular function and hypertension.” 2015. Doctoral Dissertation, University of New Mexico. Accessed February 25, 2020. https://digitalrepository.unm.edu/biom_etds/126.

MLA Handbook (7th Edition):

Fredette, Natalie C. “The Role of G protein-coupled estrogen receptor in vascular function and hypertension.” 2015. Web. 25 Feb 2020.

Vancouver:

Fredette NC. The Role of G protein-coupled estrogen receptor in vascular function and hypertension. [Internet] [Doctoral dissertation]. University of New Mexico; 2015. [cited 2020 Feb 25]. Available from: https://digitalrepository.unm.edu/biom_etds/126.

Council of Science Editors:

Fredette NC. The Role of G protein-coupled estrogen receptor in vascular function and hypertension. [Doctoral Dissertation]. University of New Mexico; 2015. Available from: https://digitalrepository.unm.edu/biom_etds/126


University of New Mexico

16. Lovato, TyAnna L. DEFINING THE COMPLEXITIES OF MEF2 FUNCTION AND REGULATION IN DROSOPHILA.

Degree: UNM Biology Department, 2014, University of New Mexico

 The development of cardiac and somatic muscle is coordinated by many factors that are highly conserved across species. Mutations in the coding or regulatory sequences… (more)

Subjects/Keywords: heart development; RNAseq; MEF2; Tinman; Pannier; genetic regulation; temperature sensitive mutations

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APA (6th Edition):

Lovato, T. L. (2014). DEFINING THE COMPLEXITIES OF MEF2 FUNCTION AND REGULATION IN DROSOPHILA. (Doctoral Dissertation). University of New Mexico. Retrieved from https://digitalrepository.unm.edu/biol_etds/72

Chicago Manual of Style (16th Edition):

Lovato, TyAnna L. “DEFINING THE COMPLEXITIES OF MEF2 FUNCTION AND REGULATION IN DROSOPHILA.” 2014. Doctoral Dissertation, University of New Mexico. Accessed February 25, 2020. https://digitalrepository.unm.edu/biol_etds/72.

MLA Handbook (7th Edition):

Lovato, TyAnna L. “DEFINING THE COMPLEXITIES OF MEF2 FUNCTION AND REGULATION IN DROSOPHILA.” 2014. Web. 25 Feb 2020.

Vancouver:

Lovato TL. DEFINING THE COMPLEXITIES OF MEF2 FUNCTION AND REGULATION IN DROSOPHILA. [Internet] [Doctoral dissertation]. University of New Mexico; 2014. [cited 2020 Feb 25]. Available from: https://digitalrepository.unm.edu/biol_etds/72.

Council of Science Editors:

Lovato TL. DEFINING THE COMPLEXITIES OF MEF2 FUNCTION AND REGULATION IN DROSOPHILA. [Doctoral Dissertation]. University of New Mexico; 2014. Available from: https://digitalrepository.unm.edu/biol_etds/72


University of New Mexico

17. Unione, Amelia H.T. The role of Bcl-2 modifying factor (Bmf) in airway epithelial cell death.

Degree: Biomedical Sciences Graduate Program, 2010, University of New Mexico

 One of the components that obstruct the airway in asthma is the sudden and increased secretion of mucus from metaplastic mucous cells in small airways.… (more)

Subjects/Keywords: airway epithelial cells; Bmf

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APA (6th Edition):

Unione, A. H. T. (2010). The role of Bcl-2 modifying factor (Bmf) in airway epithelial cell death. (Doctoral Dissertation). University of New Mexico. Retrieved from https://digitalrepository.unm.edu/biom_etds/22

Chicago Manual of Style (16th Edition):

Unione, Amelia H T. “The role of Bcl-2 modifying factor (Bmf) in airway epithelial cell death.” 2010. Doctoral Dissertation, University of New Mexico. Accessed February 25, 2020. https://digitalrepository.unm.edu/biom_etds/22.

MLA Handbook (7th Edition):

Unione, Amelia H T. “The role of Bcl-2 modifying factor (Bmf) in airway epithelial cell death.” 2010. Web. 25 Feb 2020.

Vancouver:

Unione AHT. The role of Bcl-2 modifying factor (Bmf) in airway epithelial cell death. [Internet] [Doctoral dissertation]. University of New Mexico; 2010. [cited 2020 Feb 25]. Available from: https://digitalrepository.unm.edu/biom_etds/22.

Council of Science Editors:

Unione AHT. The role of Bcl-2 modifying factor (Bmf) in airway epithelial cell death. [Doctoral Dissertation]. University of New Mexico; 2010. Available from: https://digitalrepository.unm.edu/biom_etds/22


University of New Mexico

18. Zhou, Ye. c-myb alternative splicing: a novel biomarker in leukemia.

Degree: Biomedical Sciences Graduate Program, 2011, University of New Mexico

 c-myb encodes a transcription factor that is essential for hematopoiesis and for normal development of other tissues. There is ample evidence showing the activated alleles… (more)

Subjects/Keywords: c-myb; alternative splicing; biomarker; next-generation sequencing; single molecule assay

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Zhou, Y. (2011). c-myb alternative splicing: a novel biomarker in leukemia. (Doctoral Dissertation). University of New Mexico. Retrieved from https://digitalrepository.unm.edu/biom_etds/41

Chicago Manual of Style (16th Edition):

Zhou, Ye. “c-myb alternative splicing: a novel biomarker in leukemia.” 2011. Doctoral Dissertation, University of New Mexico. Accessed February 25, 2020. https://digitalrepository.unm.edu/biom_etds/41.

MLA Handbook (7th Edition):

Zhou, Ye. “c-myb alternative splicing: a novel biomarker in leukemia.” 2011. Web. 25 Feb 2020.

Vancouver:

Zhou Y. c-myb alternative splicing: a novel biomarker in leukemia. [Internet] [Doctoral dissertation]. University of New Mexico; 2011. [cited 2020 Feb 25]. Available from: https://digitalrepository.unm.edu/biom_etds/41.

Council of Science Editors:

Zhou Y. c-myb alternative splicing: a novel biomarker in leukemia. [Doctoral Dissertation]. University of New Mexico; 2011. Available from: https://digitalrepository.unm.edu/biom_etds/41


University of New Mexico

19. Bruce, Veronica. EFFECTS OF LOW-DOSE GAMMA RADIATION IN A MURINE MODEL OF BENZO[A]PYRENE-INDUCED LUNG CANCER.

Degree: Biomedical Sciences Graduate Program, 2015, University of New Mexico

 Lung cancer is the leading cause of cancer related mortality in the United States and in the world. One of the carcinogenic compounds found in… (more)

Subjects/Keywords: Low-dose radiation; Benzo[a]pyrene; lung cancer; gamma radiation; radiation; cytokine

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Bruce, V. (2015). EFFECTS OF LOW-DOSE GAMMA RADIATION IN A MURINE MODEL OF BENZO[A]PYRENE-INDUCED LUNG CANCER. (Doctoral Dissertation). University of New Mexico. Retrieved from https://digitalrepository.unm.edu/biom_etds/114

Chicago Manual of Style (16th Edition):

Bruce, Veronica. “EFFECTS OF LOW-DOSE GAMMA RADIATION IN A MURINE MODEL OF BENZO[A]PYRENE-INDUCED LUNG CANCER.” 2015. Doctoral Dissertation, University of New Mexico. Accessed February 25, 2020. https://digitalrepository.unm.edu/biom_etds/114.

MLA Handbook (7th Edition):

Bruce, Veronica. “EFFECTS OF LOW-DOSE GAMMA RADIATION IN A MURINE MODEL OF BENZO[A]PYRENE-INDUCED LUNG CANCER.” 2015. Web. 25 Feb 2020.

Vancouver:

Bruce V. EFFECTS OF LOW-DOSE GAMMA RADIATION IN A MURINE MODEL OF BENZO[A]PYRENE-INDUCED LUNG CANCER. [Internet] [Doctoral dissertation]. University of New Mexico; 2015. [cited 2020 Feb 25]. Available from: https://digitalrepository.unm.edu/biom_etds/114.

Council of Science Editors:

Bruce V. EFFECTS OF LOW-DOSE GAMMA RADIATION IN A MURINE MODEL OF BENZO[A]PYRENE-INDUCED LUNG CANCER. [Doctoral Dissertation]. University of New Mexico; 2015. Available from: https://digitalrepository.unm.edu/biom_etds/114


University of New Mexico

20. Kenney, Shelby. Characterization of R-ketorolac as a single enantiomer selective inhibitor of Cdc42 and Rac1 in ovarian cancer.

Degree: Biomedical Sciences Graduate Program, 2015, University of New Mexico

 Ovarian cancer is the 5th leading cause of cancer death for women in the United States and is frequently diagnosed at an advanced stage with… (more)

Subjects/Keywords: Cdc42; Rac1; ovarian cancer; ketorolac; migration; adhesion; Rho GTPases

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Kenney, S. (2015). Characterization of R-ketorolac as a single enantiomer selective inhibitor of Cdc42 and Rac1 in ovarian cancer. (Doctoral Dissertation). University of New Mexico. Retrieved from https://digitalrepository.unm.edu/biom_etds/132

Chicago Manual of Style (16th Edition):

Kenney, Shelby. “Characterization of R-ketorolac as a single enantiomer selective inhibitor of Cdc42 and Rac1 in ovarian cancer.” 2015. Doctoral Dissertation, University of New Mexico. Accessed February 25, 2020. https://digitalrepository.unm.edu/biom_etds/132.

MLA Handbook (7th Edition):

Kenney, Shelby. “Characterization of R-ketorolac as a single enantiomer selective inhibitor of Cdc42 and Rac1 in ovarian cancer.” 2015. Web. 25 Feb 2020.

Vancouver:

Kenney S. Characterization of R-ketorolac as a single enantiomer selective inhibitor of Cdc42 and Rac1 in ovarian cancer. [Internet] [Doctoral dissertation]. University of New Mexico; 2015. [cited 2020 Feb 25]. Available from: https://digitalrepository.unm.edu/biom_etds/132.

Council of Science Editors:

Kenney S. Characterization of R-ketorolac as a single enantiomer selective inhibitor of Cdc42 and Rac1 in ovarian cancer. [Doctoral Dissertation]. University of New Mexico; 2015. Available from: https://digitalrepository.unm.edu/biom_etds/132

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