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You searched for +publisher:"University of New Mexico" +contributor:("Allan, Andrea"). Showing records 1 – 7 of 7 total matches.

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University of New Mexico

1. Kulkarny, Vibhati. Genetic contributions to brain fiber architecture and neuroanatomical alterations in alcohol dependent individuals : a correlation study.

Degree: Biomedical Sciences Graduate Program, 2010, University of New Mexico

 Alcohol dependence (AD) is a complex addictive disorder, affecting 5.4% of the general population during a lifetime (Kessler, 2005) and has a complex heterogenous phenotype,… (more)

Subjects/Keywords: Alcohol Dependence; Neuroimaging; Magnetic Resonance Imaging; Diffusion Tensor Imaging; Genetics; Single Nucleotide Polymorphsims

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APA (6th Edition):

Kulkarny, V. (2010). Genetic contributions to brain fiber architecture and neuroanatomical alterations in alcohol dependent individuals : a correlation study. (Doctoral Dissertation). University of New Mexico. Retrieved from https://digitalrepository.unm.edu/biom_etds/14

Chicago Manual of Style (16th Edition):

Kulkarny, Vibhati. “Genetic contributions to brain fiber architecture and neuroanatomical alterations in alcohol dependent individuals : a correlation study.” 2010. Doctoral Dissertation, University of New Mexico. Accessed July 20, 2019. https://digitalrepository.unm.edu/biom_etds/14.

MLA Handbook (7th Edition):

Kulkarny, Vibhati. “Genetic contributions to brain fiber architecture and neuroanatomical alterations in alcohol dependent individuals : a correlation study.” 2010. Web. 20 Jul 2019.

Vancouver:

Kulkarny V. Genetic contributions to brain fiber architecture and neuroanatomical alterations in alcohol dependent individuals : a correlation study. [Internet] [Doctoral dissertation]. University of New Mexico; 2010. [cited 2019 Jul 20]. Available from: https://digitalrepository.unm.edu/biom_etds/14.

Council of Science Editors:

Kulkarny V. Genetic contributions to brain fiber architecture and neuroanatomical alterations in alcohol dependent individuals : a correlation study. [Doctoral Dissertation]. University of New Mexico; 2010. Available from: https://digitalrepository.unm.edu/biom_etds/14


University of New Mexico

2. Baca, Michael. Gene-environment interactions affect long-term depression (LTD) through changes in dopamine D2 receptor affinity in Snap25 haplodeficient mice.

Degree: Biomedical Sciences Graduate Program, 2013, University of New Mexico

 Genes and environmental conditions interact in the development of cognitive capacities and each plays an important role in neuropsychiatric disorders such as attention deficit/hyperactivity disorder… (more)

Subjects/Keywords: Neuroscience; Gene-environment interactions; Electrophysiology

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APA (6th Edition):

Baca, M. (2013). Gene-environment interactions affect long-term depression (LTD) through changes in dopamine D2 receptor affinity in Snap25 haplodeficient mice. (Doctoral Dissertation). University of New Mexico. Retrieved from https://digitalrepository.unm.edu/biom_etds/111

Chicago Manual of Style (16th Edition):

Baca, Michael. “Gene-environment interactions affect long-term depression (LTD) through changes in dopamine D2 receptor affinity in Snap25 haplodeficient mice.” 2013. Doctoral Dissertation, University of New Mexico. Accessed July 20, 2019. https://digitalrepository.unm.edu/biom_etds/111.

MLA Handbook (7th Edition):

Baca, Michael. “Gene-environment interactions affect long-term depression (LTD) through changes in dopamine D2 receptor affinity in Snap25 haplodeficient mice.” 2013. Web. 20 Jul 2019.

Vancouver:

Baca M. Gene-environment interactions affect long-term depression (LTD) through changes in dopamine D2 receptor affinity in Snap25 haplodeficient mice. [Internet] [Doctoral dissertation]. University of New Mexico; 2013. [cited 2019 Jul 20]. Available from: https://digitalrepository.unm.edu/biom_etds/111.

Council of Science Editors:

Baca M. Gene-environment interactions affect long-term depression (LTD) through changes in dopamine D2 receptor affinity in Snap25 haplodeficient mice. [Doctoral Dissertation]. University of New Mexico; 2013. Available from: https://digitalrepository.unm.edu/biom_etds/111


University of New Mexico

3. Staples, Miranda. Consequences of Prenatal Ethanol Exposure and Maternal Stress on Offspring.

Degree: Biomedical Sciences Graduate Program, 2013, University of New Mexico

 Extensive evidence in humans suggests that exposure to insults during gestation, such as ethanol or maternal stress, can negatively impact the developing fetus in manners… (more)

Subjects/Keywords: Prenatal Ethanol; Prenatal Stress; rat; learning; anxiety

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APA (6th Edition):

Staples, M. (2013). Consequences of Prenatal Ethanol Exposure and Maternal Stress on Offspring. (Doctoral Dissertation). University of New Mexico. Retrieved from https://digitalrepository.unm.edu/biom_etds/144

Chicago Manual of Style (16th Edition):

Staples, Miranda. “Consequences of Prenatal Ethanol Exposure and Maternal Stress on Offspring.” 2013. Doctoral Dissertation, University of New Mexico. Accessed July 20, 2019. https://digitalrepository.unm.edu/biom_etds/144.

MLA Handbook (7th Edition):

Staples, Miranda. “Consequences of Prenatal Ethanol Exposure and Maternal Stress on Offspring.” 2013. Web. 20 Jul 2019.

Vancouver:

Staples M. Consequences of Prenatal Ethanol Exposure and Maternal Stress on Offspring. [Internet] [Doctoral dissertation]. University of New Mexico; 2013. [cited 2019 Jul 20]. Available from: https://digitalrepository.unm.edu/biom_etds/144.

Council of Science Editors:

Staples M. Consequences of Prenatal Ethanol Exposure and Maternal Stress on Offspring. [Doctoral Dissertation]. University of New Mexico; 2013. Available from: https://digitalrepository.unm.edu/biom_etds/144


University of New Mexico

4. Martinez-Finley, Ebany. Perinatal arsenic exposure inhibits binding ability of glucocorticoid receptors to nuclear response elements altering gene expression and affecting learning behavior.

Degree: Biomedical Sciences Graduate Program, 2010, University of New Mexico

 Learning deficits in children following arsenic (As) exposure via drinking water have been epidemiologically described in the last decade. Arsenic is a persistent environmental toxin… (more)

Subjects/Keywords: arsenic; glucocorticoid receptors; MAPK pathway; learning and memory

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APA (6th Edition):

Martinez-Finley, E. (2010). Perinatal arsenic exposure inhibits binding ability of glucocorticoid receptors to nuclear response elements altering gene expression and affecting learning behavior. (Doctoral Dissertation). University of New Mexico. Retrieved from https://digitalrepository.unm.edu/biom_etds/15

Chicago Manual of Style (16th Edition):

Martinez-Finley, Ebany. “Perinatal arsenic exposure inhibits binding ability of glucocorticoid receptors to nuclear response elements altering gene expression and affecting learning behavior.” 2010. Doctoral Dissertation, University of New Mexico. Accessed July 20, 2019. https://digitalrepository.unm.edu/biom_etds/15.

MLA Handbook (7th Edition):

Martinez-Finley, Ebany. “Perinatal arsenic exposure inhibits binding ability of glucocorticoid receptors to nuclear response elements altering gene expression and affecting learning behavior.” 2010. Web. 20 Jul 2019.

Vancouver:

Martinez-Finley E. Perinatal arsenic exposure inhibits binding ability of glucocorticoid receptors to nuclear response elements altering gene expression and affecting learning behavior. [Internet] [Doctoral dissertation]. University of New Mexico; 2010. [cited 2019 Jul 20]. Available from: https://digitalrepository.unm.edu/biom_etds/15.

Council of Science Editors:

Martinez-Finley E. Perinatal arsenic exposure inhibits binding ability of glucocorticoid receptors to nuclear response elements altering gene expression and affecting learning behavior. [Doctoral Dissertation]. University of New Mexico; 2010. Available from: https://digitalrepository.unm.edu/biom_etds/15


University of New Mexico

5. Brady, Megan. Prenatal Alcohol Exposure Effects on NMDA Receptor Subunit Composition and Function in the Mouse Dentate Gyrus.

Degree: Biomedical Sciences Graduate Program, 2012, University of New Mexico

 Moderate prenatal alcohol exposure (PAE) in humans leads to learning and memory impairments later in life. The range of physical, behavioral, emotional, and social dysfunctions… (more)

Subjects/Keywords: prenatal alcohol exposure; NMDA receptor; dentate gyrus; LTP; learning and memory

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APA (6th Edition):

Brady, M. (2012). Prenatal Alcohol Exposure Effects on NMDA Receptor Subunit Composition and Function in the Mouse Dentate Gyrus. (Doctoral Dissertation). University of New Mexico. Retrieved from https://digitalrepository.unm.edu/biom_etds/48

Chicago Manual of Style (16th Edition):

Brady, Megan. “Prenatal Alcohol Exposure Effects on NMDA Receptor Subunit Composition and Function in the Mouse Dentate Gyrus.” 2012. Doctoral Dissertation, University of New Mexico. Accessed July 20, 2019. https://digitalrepository.unm.edu/biom_etds/48.

MLA Handbook (7th Edition):

Brady, Megan. “Prenatal Alcohol Exposure Effects on NMDA Receptor Subunit Composition and Function in the Mouse Dentate Gyrus.” 2012. Web. 20 Jul 2019.

Vancouver:

Brady M. Prenatal Alcohol Exposure Effects on NMDA Receptor Subunit Composition and Function in the Mouse Dentate Gyrus. [Internet] [Doctoral dissertation]. University of New Mexico; 2012. [cited 2019 Jul 20]. Available from: https://digitalrepository.unm.edu/biom_etds/48.

Council of Science Editors:

Brady M. Prenatal Alcohol Exposure Effects on NMDA Receptor Subunit Composition and Function in the Mouse Dentate Gyrus. [Doctoral Dissertation]. University of New Mexico; 2012. Available from: https://digitalrepository.unm.edu/biom_etds/48


University of New Mexico

6. King, Brenee S. The Contribution of Poly(ADP-ribose)polymerase-1 Activity in the Nucleotide Excision Repair Pathway.

Degree: Biomedical Sciences Graduate Program, 2012, University of New Mexico

  Exposure to ultraviolet radiation (UVR) promotes the formation of UVR-induced, DNA helix distorting photolesions such as (6-4) pyrimidine-pyrimidone photoproducts (6-4 PPs) and cyclobutane pyrimidine… (more)

Subjects/Keywords: "NER; PARP; DNA repair; ultraviolet radiation; XPA; XPC"; Medicine and Health Sciences

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

King, B. S. (2012). The Contribution of Poly(ADP-ribose)polymerase-1 Activity in the Nucleotide Excision Repair Pathway. (Doctoral Dissertation). University of New Mexico. Retrieved from https://digitalrepository.unm.edu/biom_etds/66

Chicago Manual of Style (16th Edition):

King, Brenee S. “The Contribution of Poly(ADP-ribose)polymerase-1 Activity in the Nucleotide Excision Repair Pathway.” 2012. Doctoral Dissertation, University of New Mexico. Accessed July 20, 2019. https://digitalrepository.unm.edu/biom_etds/66.

MLA Handbook (7th Edition):

King, Brenee S. “The Contribution of Poly(ADP-ribose)polymerase-1 Activity in the Nucleotide Excision Repair Pathway.” 2012. Web. 20 Jul 2019.

Vancouver:

King BS. The Contribution of Poly(ADP-ribose)polymerase-1 Activity in the Nucleotide Excision Repair Pathway. [Internet] [Doctoral dissertation]. University of New Mexico; 2012. [cited 2019 Jul 20]. Available from: https://digitalrepository.unm.edu/biom_etds/66.

Council of Science Editors:

King BS. The Contribution of Poly(ADP-ribose)polymerase-1 Activity in the Nucleotide Excision Repair Pathway. [Doctoral Dissertation]. University of New Mexico; 2012. Available from: https://digitalrepository.unm.edu/biom_etds/66


University of New Mexico

7. Tyler, Christina Rene. Aberrant neurogenic and epigenetic processes are associated with depression induced by developmental arsenic exposure.

Degree: Biomedical Sciences Graduate Program, 2014, University of New Mexico

  Arsenic is a pervasive environmental contaminant derived from both natural and anthropogenic sources. Epidemiological evidence supports a correlation between arsenic exposure in drinking water… (more)

Subjects/Keywords: neurobiology; arsenic; depression; epigenetics; neurogenesis; Medicine and Health Sciences

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Tyler, C. R. (2014). Aberrant neurogenic and epigenetic processes are associated with depression induced by developmental arsenic exposure. (Doctoral Dissertation). University of New Mexico. Retrieved from https://digitalrepository.unm.edu/biom_etds/146

Chicago Manual of Style (16th Edition):

Tyler, Christina Rene. “Aberrant neurogenic and epigenetic processes are associated with depression induced by developmental arsenic exposure.” 2014. Doctoral Dissertation, University of New Mexico. Accessed July 20, 2019. https://digitalrepository.unm.edu/biom_etds/146.

MLA Handbook (7th Edition):

Tyler, Christina Rene. “Aberrant neurogenic and epigenetic processes are associated with depression induced by developmental arsenic exposure.” 2014. Web. 20 Jul 2019.

Vancouver:

Tyler CR. Aberrant neurogenic and epigenetic processes are associated with depression induced by developmental arsenic exposure. [Internet] [Doctoral dissertation]. University of New Mexico; 2014. [cited 2019 Jul 20]. Available from: https://digitalrepository.unm.edu/biom_etds/146.

Council of Science Editors:

Tyler CR. Aberrant neurogenic and epigenetic processes are associated with depression induced by developmental arsenic exposure. [Doctoral Dissertation]. University of New Mexico; 2014. Available from: https://digitalrepository.unm.edu/biom_etds/146

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