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You searched for +publisher:"University of Missouri – Columbia" +contributor:("Sun, Grace Y."). Showing records 1 – 8 of 8 total matches.

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University of Missouri – Columbia

1. He, Yan, 1979-. Amyloid-beta toxicity in neurons and potential botanical compounds for prevention of Alzheimer's disease.

Degree: 2011, University of Missouri – Columbia

 [ACCESS RESTRICTED TO THE UNIVERSITY OF MISSOURI AT REQUEST OF AUTHOR.] Alzheimer's disease (AD) is characterized by a progressive decline in memory and cognitive function… (more)

Subjects/Keywords: NMDA receptor; oligomeric amyloid-beta peptides; oxidative damage; mitochondrial dysfunction; botanical antioxidants; Amyloid beta-peptides  – pharmacology; Catechin  – analogs & derivatives; Catechin  – pharmacology; N-Methylaspartate  – pharmacology|Reactive Oxygen Species  – metabolism; NADPH Oxidase  – metabolism; Arachidonic Acid  – metabolism; Neurons  – drug effects; Neuroprotective Agents  – pharmacology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

He, Yan, 1. (2011). Amyloid-beta toxicity in neurons and potential botanical compounds for prevention of Alzheimer's disease. (Thesis). University of Missouri – Columbia. Retrieved from http://hdl.handle.net/10355/15839

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

He, Yan, 1979-. “Amyloid-beta toxicity in neurons and potential botanical compounds for prevention of Alzheimer's disease.” 2011. Thesis, University of Missouri – Columbia. Accessed March 06, 2021. http://hdl.handle.net/10355/15839.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

He, Yan, 1979-. “Amyloid-beta toxicity in neurons and potential botanical compounds for prevention of Alzheimer's disease.” 2011. Web. 06 Mar 2021.

Vancouver:

He, Yan 1. Amyloid-beta toxicity in neurons and potential botanical compounds for prevention of Alzheimer's disease. [Internet] [Thesis]. University of Missouri – Columbia; 2011. [cited 2021 Mar 06]. Available from: http://hdl.handle.net/10355/15839.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

He, Yan 1. Amyloid-beta toxicity in neurons and potential botanical compounds for prevention of Alzheimer's disease. [Thesis]. University of Missouri – Columbia; 2011. Available from: http://hdl.handle.net/10355/15839

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Missouri – Columbia

2. Ni, Yawen. Tnf[alpha] alters occludin and endothelial cell barrier function : role of p38MAPK.

Degree: 2016, University of Missouri – Columbia

 Occludin is one of the key tight junction (TJ) proteins in endothelial cells and it plays an important role in modulating blood brain barrier (BBB)… (more)

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Ni, Y. (2016). Tnf[alpha] alters occludin and endothelial cell barrier function : role of p38MAPK. (Thesis). University of Missouri – Columbia. Retrieved from http://hdl.handle.net/10355/65977

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ni, Yawen. “Tnf[alpha] alters occludin and endothelial cell barrier function : role of p38MAPK.” 2016. Thesis, University of Missouri – Columbia. Accessed March 06, 2021. http://hdl.handle.net/10355/65977.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ni, Yawen. “Tnf[alpha] alters occludin and endothelial cell barrier function : role of p38MAPK.” 2016. Web. 06 Mar 2021.

Vancouver:

Ni Y. Tnf[alpha] alters occludin and endothelial cell barrier function : role of p38MAPK. [Internet] [Thesis]. University of Missouri – Columbia; 2016. [cited 2021 Mar 06]. Available from: http://hdl.handle.net/10355/65977.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ni Y. Tnf[alpha] alters occludin and endothelial cell barrier function : role of p38MAPK. [Thesis]. University of Missouri – Columbia; 2016. Available from: http://hdl.handle.net/10355/65977

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Missouri – Columbia

3. Chuang, Dennis Yi-Lun, 1985-. The role of cytosolic phospholipase A2 in microglial signaling pathways during neuroinflammation.

Degree: 2016, University of Missouri – Columbia

 Oxidative and nitrosative stress is known to play an important role in neurodegenerative and neuroinflammatory diseases, such as Alzheimer's disease, Parkinson's disease, ischemic[slash]hemorrhagic stroke, and… (more)

Subjects/Keywords: Microglia; Nervous system  – Degeneration; Phospholipase A2

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APA (6th Edition):

Chuang, Dennis Yi-Lun, 1. (2016). The role of cytosolic phospholipase A2 in microglial signaling pathways during neuroinflammation. (Thesis). University of Missouri – Columbia. Retrieved from https://doi.org/10.32469/10355/56462

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Chuang, Dennis Yi-Lun, 1985-. “The role of cytosolic phospholipase A2 in microglial signaling pathways during neuroinflammation.” 2016. Thesis, University of Missouri – Columbia. Accessed March 06, 2021. https://doi.org/10.32469/10355/56462.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Chuang, Dennis Yi-Lun, 1985-. “The role of cytosolic phospholipase A2 in microglial signaling pathways during neuroinflammation.” 2016. Web. 06 Mar 2021.

Vancouver:

Chuang, Dennis Yi-Lun 1. The role of cytosolic phospholipase A2 in microglial signaling pathways during neuroinflammation. [Internet] [Thesis]. University of Missouri – Columbia; 2016. [cited 2021 Mar 06]. Available from: https://doi.org/10.32469/10355/56462.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Chuang, Dennis Yi-Lun 1. The role of cytosolic phospholipase A2 in microglial signaling pathways during neuroinflammation. [Thesis]. University of Missouri – Columbia; 2016. Available from: https://doi.org/10.32469/10355/56462

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Missouri – Columbia

4. Jensen, Michael D., 1979-. Secretory phospholipase A2-IIA in Alzheimer's disease and inflammatory responses in astrocytes.

Degree: 2009, University of Missouri – Columbia

 Alzheimer's disease (AD) is a progressive, fatal neurodegenerative disease marked with dementia. Secretory phospholipase A2-IIA (sPLA2-IIA) is an inflammatory protein known to have a role… (more)

Subjects/Keywords: Alzheimer disease  – physiopathology; Group II Phospholipases A2  – metabolism; Alzheimer disease  – enzymology; Astrocytes  – metabolism; NADPH Oxidase  – metabolism; Inflammation  – physiopathology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Jensen, Michael D., 1. (2009). Secretory phospholipase A2-IIA in Alzheimer's disease and inflammatory responses in astrocytes. (Thesis). University of Missouri – Columbia. Retrieved from https://doi.org/10.32469/10355/6738

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Jensen, Michael D., 1979-. “Secretory phospholipase A2-IIA in Alzheimer's disease and inflammatory responses in astrocytes.” 2009. Thesis, University of Missouri – Columbia. Accessed March 06, 2021. https://doi.org/10.32469/10355/6738.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Jensen, Michael D., 1979-. “Secretory phospholipase A2-IIA in Alzheimer's disease and inflammatory responses in astrocytes.” 2009. Web. 06 Mar 2021.

Vancouver:

Jensen, Michael D. 1. Secretory phospholipase A2-IIA in Alzheimer's disease and inflammatory responses in astrocytes. [Internet] [Thesis]. University of Missouri – Columbia; 2009. [cited 2021 Mar 06]. Available from: https://doi.org/10.32469/10355/6738.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Jensen, Michael D. 1. Secretory phospholipase A2-IIA in Alzheimer's disease and inflammatory responses in astrocytes. [Thesis]. University of Missouri – Columbia; 2009. Available from: https://doi.org/10.32469/10355/6738

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Missouri – Columbia

5. Shelat, Phullara B., 1978-. Amyloid beta induces cPLA2 activation by an NADPH oxidase-dependent mechanism in neurons.

Degree: 2008, University of Missouri – Columbia

 [ACCESS RESTRICTED TO THE UNIVERSITY OF MISSOURI AT REQUEST OF AUTHOR.] Alzheimer's disease (AD) is a neurodegenerative disorder affecting more than 20 million people worldwide.… (more)

Subjects/Keywords: Alzheimer's disease  – Pathophysiology; Amyloid beta-protein  – Toxicology; Amyloid beta-protein  – Effect of drugs on; NAD (Coenzyme); Active oxygen; Phospholipase A2  – Effect of drugs on; Alzheimer disease  – physiopathology; Amyloid beta-protein  – Toxicity; NADPH Oxidase  – metabolism; Reactive Oxygen Species  – metabolism; Amyloid beta-Protein  – pharmacology; NADPH Oxidase  – drug effects; Phospholipases A2, Cytosolic  – metabolism; Phospholipases A2, Cytosolic  – drug effects; Extracellular Signal-Related MAP Kinases  – metabolism; Extracellular Signal-Related MAP Kinases  – drug effects; Anachidonic Acid  – metabolism

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Shelat, Phullara B., 1. (2008). Amyloid beta induces cPLA2 activation by an NADPH oxidase-dependent mechanism in neurons. (Thesis). University of Missouri – Columbia. Retrieved from https://doi.org/10.32469/10355/7205

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Shelat, Phullara B., 1978-. “Amyloid beta induces cPLA2 activation by an NADPH oxidase-dependent mechanism in neurons.” 2008. Thesis, University of Missouri – Columbia. Accessed March 06, 2021. https://doi.org/10.32469/10355/7205.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Shelat, Phullara B., 1978-. “Amyloid beta induces cPLA2 activation by an NADPH oxidase-dependent mechanism in neurons.” 2008. Web. 06 Mar 2021.

Vancouver:

Shelat, Phullara B. 1. Amyloid beta induces cPLA2 activation by an NADPH oxidase-dependent mechanism in neurons. [Internet] [Thesis]. University of Missouri – Columbia; 2008. [cited 2021 Mar 06]. Available from: https://doi.org/10.32469/10355/7205.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Shelat, Phullara B. 1. Amyloid beta induces cPLA2 activation by an NADPH oxidase-dependent mechanism in neurons. [Thesis]. University of Missouri – Columbia; 2008. Available from: https://doi.org/10.32469/10355/7205

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Missouri – Columbia

6. Hu, Chunhua. Activation of astrocytes: involvement of NADPH oxidase and cytosolic phospholipase A2.

Degree: 2007, University of Missouri – Columbia

 [ACCESS RESTRICTED TO THE UNIVERSITY OF MISSOURI AT AUTHOR'S REQUEST.] Reactive oxygen species (ROS) are produced in cells by enzymic and non-enzymic mechanisms and play… (more)

Subjects/Keywords: Astrocytes; NAD (Coenzyme); Apoptosis; Cell membranes; Phospholipase A2  – Inhibitors; Cytosol; Oxidative stress; Active oxygen; Astrocytes  – physiology; NADPH Oxidase  – physiology; Apoptosis  – physiology; Cell Membrane; Central Nervous System  – physiology; NADP  – antagonists & inhibitors; Oxidative Stress  – physiology; Phospholipases A  – metabolism; Reactive Oxygen Species  – metabolism; Vitamin K3

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Hu, C. (2007). Activation of astrocytes: involvement of NADPH oxidase and cytosolic phospholipase A2. (Thesis). University of Missouri – Columbia. Retrieved from https://doi.org/10.32469/10355/6270

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hu, Chunhua. “Activation of astrocytes: involvement of NADPH oxidase and cytosolic phospholipase A2.” 2007. Thesis, University of Missouri – Columbia. Accessed March 06, 2021. https://doi.org/10.32469/10355/6270.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hu, Chunhua. “Activation of astrocytes: involvement of NADPH oxidase and cytosolic phospholipase A2.” 2007. Web. 06 Mar 2021.

Vancouver:

Hu C. Activation of astrocytes: involvement of NADPH oxidase and cytosolic phospholipase A2. [Internet] [Thesis]. University of Missouri – Columbia; 2007. [cited 2021 Mar 06]. Available from: https://doi.org/10.32469/10355/6270.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hu C. Activation of astrocytes: involvement of NADPH oxidase and cytosolic phospholipase A2. [Thesis]. University of Missouri – Columbia; 2007. Available from: https://doi.org/10.32469/10355/6270

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Missouri – Columbia

7. Jensen, Michael D., 1979-. Secretory phospholipase A2-IIA in Alzheimer's disease and inflammatory responses in astrocytes.

Degree: 2009, University of Missouri – Columbia

 Alzheimer's disease (AD) is a progressive, fatal neurodegenerative disease marked with dementia. Secretory phospholipase A2-IIA (sPLA2-IIA) is an inflammatory protein known to have a role… (more)

Subjects/Keywords: Alzheimer disease  – physiopathology; Group II Phospholipases A2  – metabolism; Alzheimer disease  – enzymology; Astrocytes  – metabolism; NADPH Oxidase  – metabolism; Inflammation  – physiopathology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Jensen, Michael D., 1. (2009). Secretory phospholipase A2-IIA in Alzheimer's disease and inflammatory responses in astrocytes. (Thesis). University of Missouri – Columbia. Retrieved from http://hdl.handle.net/10355/6738

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Jensen, Michael D., 1979-. “Secretory phospholipase A2-IIA in Alzheimer's disease and inflammatory responses in astrocytes.” 2009. Thesis, University of Missouri – Columbia. Accessed March 06, 2021. http://hdl.handle.net/10355/6738.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Jensen, Michael D., 1979-. “Secretory phospholipase A2-IIA in Alzheimer's disease and inflammatory responses in astrocytes.” 2009. Web. 06 Mar 2021.

Vancouver:

Jensen, Michael D. 1. Secretory phospholipase A2-IIA in Alzheimer's disease and inflammatory responses in astrocytes. [Internet] [Thesis]. University of Missouri – Columbia; 2009. [cited 2021 Mar 06]. Available from: http://hdl.handle.net/10355/6738.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Jensen, Michael D. 1. Secretory phospholipase A2-IIA in Alzheimer's disease and inflammatory responses in astrocytes. [Thesis]. University of Missouri – Columbia; 2009. Available from: http://hdl.handle.net/10355/6738

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Missouri – Columbia

8. Shelat, Phullara B., 1978-. Amyloid beta induces cPLA2 activation by an NADPH oxidase-dependent mechanism in neurons.

Degree: 2008, University of Missouri – Columbia

 [ACCESS RESTRICTED TO THE UNIVERSITY OF MISSOURI AT REQUEST OF AUTHOR.] Alzheimer's disease (AD) is a neurodegenerative disorder affecting more than 20 million people worldwide.… (more)

Subjects/Keywords: Alzheimer's disease  – Pathophysiology; Amyloid beta-protein  – Toxicology; Amyloid beta-protein  – Effect of drugs on; NAD (Coenzyme); Active oxygen; Phospholipase A2  – Effect of drugs on; Alzheimer disease  – physiopathology; Amyloid beta-protein  – Toxicity; NADPH Oxidase  – metabolism; Reactive Oxygen Species  – metabolism; Amyloid beta-Protein  – pharmacology; NADPH Oxidase  – drug effects; Phospholipases A2, Cytosolic  – metabolism; Phospholipases A2, Cytosolic  – drug effects; Extracellular Signal-Related MAP Kinases  – metabolism; Extracellular Signal-Related MAP Kinases  – drug effects; Anachidonic Acid  – metabolism

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Shelat, Phullara B., 1. (2008). Amyloid beta induces cPLA2 activation by an NADPH oxidase-dependent mechanism in neurons. (Thesis). University of Missouri – Columbia. Retrieved from http://hdl.handle.net/10355/7205

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Shelat, Phullara B., 1978-. “Amyloid beta induces cPLA2 activation by an NADPH oxidase-dependent mechanism in neurons.” 2008. Thesis, University of Missouri – Columbia. Accessed March 06, 2021. http://hdl.handle.net/10355/7205.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Shelat, Phullara B., 1978-. “Amyloid beta induces cPLA2 activation by an NADPH oxidase-dependent mechanism in neurons.” 2008. Web. 06 Mar 2021.

Vancouver:

Shelat, Phullara B. 1. Amyloid beta induces cPLA2 activation by an NADPH oxidase-dependent mechanism in neurons. [Internet] [Thesis]. University of Missouri – Columbia; 2008. [cited 2021 Mar 06]. Available from: http://hdl.handle.net/10355/7205.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Shelat, Phullara B. 1. Amyloid beta induces cPLA2 activation by an NADPH oxidase-dependent mechanism in neurons. [Thesis]. University of Missouri – Columbia; 2008. Available from: http://hdl.handle.net/10355/7205

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

.