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You searched for +publisher:"University of Michigan" +contributor:("Margolis, Benjamin L"). Showing records 1 – 28 of 28 total matches.

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University of Michigan

1. Freddo, Andrew. New Perspectives on Intestinal Morphogenesis: The Role of Cell Division and Intraepithelial Forces in Villus Formation.

Degree: PhD, Cell & Developmental Biol PhD, 2018, University of Michigan

 Finger-like projections called villi convolute the intestinal surface, maximizing the area for nutrient absorption. Villi are rapidly formed and patterned during embryonic development; in the… (more)

Subjects/Keywords: small intestine; cell division; intestinal development; Molecular, Cellular and Developmental Biology; Health Sciences; Science

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APA (6th Edition):

Freddo, A. (2018). New Perspectives on Intestinal Morphogenesis: The Role of Cell Division and Intraepithelial Forces in Villus Formation. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/144092

Chicago Manual of Style (16th Edition):

Freddo, Andrew. “New Perspectives on Intestinal Morphogenesis: The Role of Cell Division and Intraepithelial Forces in Villus Formation.” 2018. Doctoral Dissertation, University of Michigan. Accessed January 17, 2020. http://hdl.handle.net/2027.42/144092.

MLA Handbook (7th Edition):

Freddo, Andrew. “New Perspectives on Intestinal Morphogenesis: The Role of Cell Division and Intraepithelial Forces in Villus Formation.” 2018. Web. 17 Jan 2020.

Vancouver:

Freddo A. New Perspectives on Intestinal Morphogenesis: The Role of Cell Division and Intraepithelial Forces in Villus Formation. [Internet] [Doctoral dissertation]. University of Michigan; 2018. [cited 2020 Jan 17]. Available from: http://hdl.handle.net/2027.42/144092.

Council of Science Editors:

Freddo A. New Perspectives on Intestinal Morphogenesis: The Role of Cell Division and Intraepithelial Forces in Villus Formation. [Doctoral Dissertation]. University of Michigan; 2018. Available from: http://hdl.handle.net/2027.42/144092


University of Michigan

2. Reyes, Ciara. Characterizing a Novel Role for the Cytokinesis Scaffolding Protein Anillin in Regulating Cell-cell Junctions.

Degree: PhD, Cellular and Molecular Biology, 2016, University of Michigan

 Anillin is a highly conserved scaffolding protein required for successful cytokinesis in multiple biological organisms, from flies to human. During cytokinesis, Anillin helps to crosslink… (more)

Subjects/Keywords: Anillin and cell-cell junctions; Molecular, Cellular and Developmental Biology; Science

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APA (6th Edition):

Reyes, C. (2016). Characterizing a Novel Role for the Cytokinesis Scaffolding Protein Anillin in Regulating Cell-cell Junctions. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/133500

Chicago Manual of Style (16th Edition):

Reyes, Ciara. “Characterizing a Novel Role for the Cytokinesis Scaffolding Protein Anillin in Regulating Cell-cell Junctions.” 2016. Doctoral Dissertation, University of Michigan. Accessed January 17, 2020. http://hdl.handle.net/2027.42/133500.

MLA Handbook (7th Edition):

Reyes, Ciara. “Characterizing a Novel Role for the Cytokinesis Scaffolding Protein Anillin in Regulating Cell-cell Junctions.” 2016. Web. 17 Jan 2020.

Vancouver:

Reyes C. Characterizing a Novel Role for the Cytokinesis Scaffolding Protein Anillin in Regulating Cell-cell Junctions. [Internet] [Doctoral dissertation]. University of Michigan; 2016. [cited 2020 Jan 17]. Available from: http://hdl.handle.net/2027.42/133500.

Council of Science Editors:

Reyes C. Characterizing a Novel Role for the Cytokinesis Scaffolding Protein Anillin in Regulating Cell-cell Junctions. [Doctoral Dissertation]. University of Michigan; 2016. Available from: http://hdl.handle.net/2027.42/133500


University of Michigan

3. Bahr, Julian. Macrophage-Dependent Trafficking and Remodeling of Extracellular Matrix Barriers.

Degree: PhD, Cancer Biology, 2018, University of Michigan

 Macrophages are evolutionarily-conserved immune cells distributed throughout all tissues in the body, which rapidly mobilize to defend against a range of insults. In executing events… (more)

Subjects/Keywords: Macrophage remodeling of extracellular matrix barriers; Microbiology and Immunology; Oncology and Hematology; Health Sciences; Science

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APA (6th Edition):

Bahr, J. (2018). Macrophage-Dependent Trafficking and Remodeling of Extracellular Matrix Barriers. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/147550

Chicago Manual of Style (16th Edition):

Bahr, Julian. “Macrophage-Dependent Trafficking and Remodeling of Extracellular Matrix Barriers.” 2018. Doctoral Dissertation, University of Michigan. Accessed January 17, 2020. http://hdl.handle.net/2027.42/147550.

MLA Handbook (7th Edition):

Bahr, Julian. “Macrophage-Dependent Trafficking and Remodeling of Extracellular Matrix Barriers.” 2018. Web. 17 Jan 2020.

Vancouver:

Bahr J. Macrophage-Dependent Trafficking and Remodeling of Extracellular Matrix Barriers. [Internet] [Doctoral dissertation]. University of Michigan; 2018. [cited 2020 Jan 17]. Available from: http://hdl.handle.net/2027.42/147550.

Council of Science Editors:

Bahr J. Macrophage-Dependent Trafficking and Remodeling of Extracellular Matrix Barriers. [Doctoral Dissertation]. University of Michigan; 2018. Available from: http://hdl.handle.net/2027.42/147550


University of Michigan

4. Koch, Aaron. Investigations into the Stucture and Function of Type I Polyketide Synthases.

Degree: PhD, Cancer Biology, 2017, University of Michigan

 The polyketide (PK) class of natural products constitutes an abundant array of secondary metabolites produced in microorganisms, many of which possess potential medicinal value, especially… (more)

Subjects/Keywords: natural products biosynthesis; Biological Chemistry; Science

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APA (6th Edition):

Koch, A. (2017). Investigations into the Stucture and Function of Type I Polyketide Synthases. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/140831

Chicago Manual of Style (16th Edition):

Koch, Aaron. “Investigations into the Stucture and Function of Type I Polyketide Synthases.” 2017. Doctoral Dissertation, University of Michigan. Accessed January 17, 2020. http://hdl.handle.net/2027.42/140831.

MLA Handbook (7th Edition):

Koch, Aaron. “Investigations into the Stucture and Function of Type I Polyketide Synthases.” 2017. Web. 17 Jan 2020.

Vancouver:

Koch A. Investigations into the Stucture and Function of Type I Polyketide Synthases. [Internet] [Doctoral dissertation]. University of Michigan; 2017. [cited 2020 Jan 17]. Available from: http://hdl.handle.net/2027.42/140831.

Council of Science Editors:

Koch A. Investigations into the Stucture and Function of Type I Polyketide Synthases. [Doctoral Dissertation]. University of Michigan; 2017. Available from: http://hdl.handle.net/2027.42/140831


University of Michigan

5. Weng, Mo. Keeping Neural Progenitors on a Short Leash: Distinguishing Intermediate Progenitors from Stem Cells in Developing Drosophila Optic Lobe and Central Brain.

Degree: PhD, Cell and Developmental Biology, 2011, University of Michigan

 Producing intermediate progenitors is one of the critical strategies for stem cells to amplify their output and generate diversity, allowing stem cells to meet demanding… (more)

Subjects/Keywords: Intermediate Progenitor Cell; Molecular, Cellular and Developmental Biology; Science

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APA (6th Edition):

Weng, M. (2011). Keeping Neural Progenitors on a Short Leash: Distinguishing Intermediate Progenitors from Stem Cells in Developing Drosophila Optic Lobe and Central Brain. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/89726

Chicago Manual of Style (16th Edition):

Weng, Mo. “Keeping Neural Progenitors on a Short Leash: Distinguishing Intermediate Progenitors from Stem Cells in Developing Drosophila Optic Lobe and Central Brain.” 2011. Doctoral Dissertation, University of Michigan. Accessed January 17, 2020. http://hdl.handle.net/2027.42/89726.

MLA Handbook (7th Edition):

Weng, Mo. “Keeping Neural Progenitors on a Short Leash: Distinguishing Intermediate Progenitors from Stem Cells in Developing Drosophila Optic Lobe and Central Brain.” 2011. Web. 17 Jan 2020.

Vancouver:

Weng M. Keeping Neural Progenitors on a Short Leash: Distinguishing Intermediate Progenitors from Stem Cells in Developing Drosophila Optic Lobe and Central Brain. [Internet] [Doctoral dissertation]. University of Michigan; 2011. [cited 2020 Jan 17]. Available from: http://hdl.handle.net/2027.42/89726.

Council of Science Editors:

Weng M. Keeping Neural Progenitors on a Short Leash: Distinguishing Intermediate Progenitors from Stem Cells in Developing Drosophila Optic Lobe and Central Brain. [Doctoral Dissertation]. University of Michigan; 2011. Available from: http://hdl.handle.net/2027.42/89726


University of Michigan

6. Grabowska, Magdalena M. Characterizing the role of ADAM10 and 15 disintegrins in prostate biology and disease.

Degree: PhD, Cellular & Molecular Biology, 2011, University of Michigan

 During the progression of prostate cancer, the adhesion molecule epithelial (E)-cadherin can be lost from the cell surface by ADAM15 proteolytic processing, generating an extracellular… (more)

Subjects/Keywords: ADAM10; Soluble E-cadherin; EGFR; Prostate; Molecular, Cellular and Developmental Biology; Science

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APA (6th Edition):

Grabowska, M. M. (2011). Characterizing the role of ADAM10 and 15 disintegrins in prostate biology and disease. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/89823

Chicago Manual of Style (16th Edition):

Grabowska, Magdalena M. “Characterizing the role of ADAM10 and 15 disintegrins in prostate biology and disease.” 2011. Doctoral Dissertation, University of Michigan. Accessed January 17, 2020. http://hdl.handle.net/2027.42/89823.

MLA Handbook (7th Edition):

Grabowska, Magdalena M. “Characterizing the role of ADAM10 and 15 disintegrins in prostate biology and disease.” 2011. Web. 17 Jan 2020.

Vancouver:

Grabowska MM. Characterizing the role of ADAM10 and 15 disintegrins in prostate biology and disease. [Internet] [Doctoral dissertation]. University of Michigan; 2011. [cited 2020 Jan 17]. Available from: http://hdl.handle.net/2027.42/89823.

Council of Science Editors:

Grabowska MM. Characterizing the role of ADAM10 and 15 disintegrins in prostate biology and disease. [Doctoral Dissertation]. University of Michigan; 2011. Available from: http://hdl.handle.net/2027.42/89823

7. Lee, Chunghee. A WUSCHEL-Independent Stem Cell Specification Pathway is Repressed by PHB, PHV and CNA in Arabidopsis.

Degree: PhD, Molecular, Cellular and Developmental Biology, 2014, University of Michigan

 A tight balance between stem cell specification and differentiation is important to maintain a functional meristem and continuously initiate new organs throughout the lifespan of… (more)

Subjects/Keywords: A WUSCHEL (WUS) -Independent Stem Cell Specification Pathway Is Repressed by PHB, PHV, and CNA in Arabidopsis; HD-zip III Member; PHABULOSA (PHB), PHAVOLUTA (PHV), CORONA (CRN), REVOLUTA (REV); ARGONAUTE10 (AGO10); CLAVATA (CLV); Stem Cell Specification, Shoot Meristem; Molecular, Cellular and Developmental Biology; Science

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APA (6th Edition):

Lee, C. (2014). A WUSCHEL-Independent Stem Cell Specification Pathway is Repressed by PHB, PHV and CNA in Arabidopsis. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/107112

Chicago Manual of Style (16th Edition):

Lee, Chunghee. “A WUSCHEL-Independent Stem Cell Specification Pathway is Repressed by PHB, PHV and CNA in Arabidopsis.” 2014. Doctoral Dissertation, University of Michigan. Accessed January 17, 2020. http://hdl.handle.net/2027.42/107112.

MLA Handbook (7th Edition):

Lee, Chunghee. “A WUSCHEL-Independent Stem Cell Specification Pathway is Repressed by PHB, PHV and CNA in Arabidopsis.” 2014. Web. 17 Jan 2020.

Vancouver:

Lee C. A WUSCHEL-Independent Stem Cell Specification Pathway is Repressed by PHB, PHV and CNA in Arabidopsis. [Internet] [Doctoral dissertation]. University of Michigan; 2014. [cited 2020 Jan 17]. Available from: http://hdl.handle.net/2027.42/107112.

Council of Science Editors:

Lee C. A WUSCHEL-Independent Stem Cell Specification Pathway is Repressed by PHB, PHV and CNA in Arabidopsis. [Doctoral Dissertation]. University of Michigan; 2014. Available from: http://hdl.handle.net/2027.42/107112

8. Jenkins, Paul Michael. Mechanisms of Ciliary Targeting of the Olfactory Cyclic Nucleotide-Gated Channel.

Degree: PhD, Pharmacology, 2010, University of Michigan

 The olfactory system gives us an awareness of our environment by allowing us to detect volatile airborne stimuli. The components necessary for this odorant detection… (more)

Subjects/Keywords: Cilia Trafficking; Pharmacy and Pharmacology; Health Sciences

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APA (6th Edition):

Jenkins, P. M. (2010). Mechanisms of Ciliary Targeting of the Olfactory Cyclic Nucleotide-Gated Channel. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/75938

Chicago Manual of Style (16th Edition):

Jenkins, Paul Michael. “Mechanisms of Ciliary Targeting of the Olfactory Cyclic Nucleotide-Gated Channel.” 2010. Doctoral Dissertation, University of Michigan. Accessed January 17, 2020. http://hdl.handle.net/2027.42/75938.

MLA Handbook (7th Edition):

Jenkins, Paul Michael. “Mechanisms of Ciliary Targeting of the Olfactory Cyclic Nucleotide-Gated Channel.” 2010. Web. 17 Jan 2020.

Vancouver:

Jenkins PM. Mechanisms of Ciliary Targeting of the Olfactory Cyclic Nucleotide-Gated Channel. [Internet] [Doctoral dissertation]. University of Michigan; 2010. [cited 2020 Jan 17]. Available from: http://hdl.handle.net/2027.42/75938.

Council of Science Editors:

Jenkins PM. Mechanisms of Ciliary Targeting of the Olfactory Cyclic Nucleotide-Gated Channel. [Doctoral Dissertation]. University of Michigan; 2010. Available from: http://hdl.handle.net/2027.42/75938

9. Kee, Hooi Lynn. Mechanisms of Ciliary Entry.

Degree: PhD, Cell and Developmental Biology, 2012, University of Michigan

 Cilia are microtubule-based projections that extend from the surface of all mammalian cells. These specialized organelles function in motility and in sensing extracellular signals during… (more)

Subjects/Keywords: Ciliary Trafficking; Molecular, Cellular and Developmental Biology; Science

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APA (6th Edition):

Kee, H. L. (2012). Mechanisms of Ciliary Entry. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/96014

Chicago Manual of Style (16th Edition):

Kee, Hooi Lynn. “Mechanisms of Ciliary Entry.” 2012. Doctoral Dissertation, University of Michigan. Accessed January 17, 2020. http://hdl.handle.net/2027.42/96014.

MLA Handbook (7th Edition):

Kee, Hooi Lynn. “Mechanisms of Ciliary Entry.” 2012. Web. 17 Jan 2020.

Vancouver:

Kee HL. Mechanisms of Ciliary Entry. [Internet] [Doctoral dissertation]. University of Michigan; 2012. [cited 2020 Jan 17]. Available from: http://hdl.handle.net/2027.42/96014.

Council of Science Editors:

Kee HL. Mechanisms of Ciliary Entry. [Doctoral Dissertation]. University of Michigan; 2012. Available from: http://hdl.handle.net/2027.42/96014

10. Zhang, Peng. Mechanisms and Regulation of Transforming Growth Factor Superfamily Mediated Gene Expression.

Degree: PhD, Molecular & Cellular Pathology, 2012, University of Michigan

 The TGF-beta superfamily, including TGF-betas and BMPs, is critical for normal embryonic development, as well as disease progression, and is tightly regulated both within and… (more)

Subjects/Keywords: TGF-beta; Epithelial Mesenchymal Transition; Gene Regulation; Wnt Signaling; Groucho Proteins; Renal Fibrosis; Pathology; Health Sciences

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APA (6th Edition):

Zhang, P. (2012). Mechanisms and Regulation of Transforming Growth Factor Superfamily Mediated Gene Expression. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/96131

Chicago Manual of Style (16th Edition):

Zhang, Peng. “Mechanisms and Regulation of Transforming Growth Factor Superfamily Mediated Gene Expression.” 2012. Doctoral Dissertation, University of Michigan. Accessed January 17, 2020. http://hdl.handle.net/2027.42/96131.

MLA Handbook (7th Edition):

Zhang, Peng. “Mechanisms and Regulation of Transforming Growth Factor Superfamily Mediated Gene Expression.” 2012. Web. 17 Jan 2020.

Vancouver:

Zhang P. Mechanisms and Regulation of Transforming Growth Factor Superfamily Mediated Gene Expression. [Internet] [Doctoral dissertation]. University of Michigan; 2012. [cited 2020 Jan 17]. Available from: http://hdl.handle.net/2027.42/96131.

Council of Science Editors:

Zhang P. Mechanisms and Regulation of Transforming Growth Factor Superfamily Mediated Gene Expression. [Doctoral Dissertation]. University of Michigan; 2012. Available from: http://hdl.handle.net/2027.42/96131

11. Cao, Siyan. Endoplasmic Reticulum Stress in Intestinal Epithelial Cells and Inflammatory Bowel Disease.

Degree: PhD, Biological Chemistry, 2013, University of Michigan

 Recent studies linked endoplasmic reticulum stress and the unfolded protein response (UPR) to inflammatory bowel disease (IBD), which includes Crohn’s disease and ulcerative colitis. My… (more)

Subjects/Keywords: Endoplasmic Reticulum Stress; Inflammatory Bowel Disease; Intestinal Epithelial Cells; Biological Chemistry; Health Sciences

…x28;ULAM) at the University of Michigan Medical Center with free access to water and… …guidelines approved by the University of Michigan Committee on the Use and Care of Animals (… 

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APA (6th Edition):

Cao, S. (2013). Endoplasmic Reticulum Stress in Intestinal Epithelial Cells and Inflammatory Bowel Disease. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/99965

Chicago Manual of Style (16th Edition):

Cao, Siyan. “Endoplasmic Reticulum Stress in Intestinal Epithelial Cells and Inflammatory Bowel Disease.” 2013. Doctoral Dissertation, University of Michigan. Accessed January 17, 2020. http://hdl.handle.net/2027.42/99965.

MLA Handbook (7th Edition):

Cao, Siyan. “Endoplasmic Reticulum Stress in Intestinal Epithelial Cells and Inflammatory Bowel Disease.” 2013. Web. 17 Jan 2020.

Vancouver:

Cao S. Endoplasmic Reticulum Stress in Intestinal Epithelial Cells and Inflammatory Bowel Disease. [Internet] [Doctoral dissertation]. University of Michigan; 2013. [cited 2020 Jan 17]. Available from: http://hdl.handle.net/2027.42/99965.

Council of Science Editors:

Cao S. Endoplasmic Reticulum Stress in Intestinal Epithelial Cells and Inflammatory Bowel Disease. [Doctoral Dissertation]. University of Michigan; 2013. Available from: http://hdl.handle.net/2027.42/99965

12. Rosenthal, Devin Thomas. Interrogating the Molecular Mechanisms of Breast Cancer Metastasis: The Contributions of RhoC GTPase and p38y MAPK.

Degree: PhD, Cellular & Molecular Biology, 2011, University of Michigan

 Breast cancer lethality is primarily due to cancer cell metastasis away from the primary tumor to vital organs. Understanding and targeting the molecular mechanisms contributing… (more)

Subjects/Keywords: Breast Cancer; P38 MAPK; Cell Motility; Cancer Stem Cell; Metastasis; Molecular, Cellular and Developmental Biology; Science (General); Science

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APA (6th Edition):

Rosenthal, D. T. (2011). Interrogating the Molecular Mechanisms of Breast Cancer Metastasis: The Contributions of RhoC GTPase and p38y MAPK. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/86269

Chicago Manual of Style (16th Edition):

Rosenthal, Devin Thomas. “Interrogating the Molecular Mechanisms of Breast Cancer Metastasis: The Contributions of RhoC GTPase and p38y MAPK.” 2011. Doctoral Dissertation, University of Michigan. Accessed January 17, 2020. http://hdl.handle.net/2027.42/86269.

MLA Handbook (7th Edition):

Rosenthal, Devin Thomas. “Interrogating the Molecular Mechanisms of Breast Cancer Metastasis: The Contributions of RhoC GTPase and p38y MAPK.” 2011. Web. 17 Jan 2020.

Vancouver:

Rosenthal DT. Interrogating the Molecular Mechanisms of Breast Cancer Metastasis: The Contributions of RhoC GTPase and p38y MAPK. [Internet] [Doctoral dissertation]. University of Michigan; 2011. [cited 2020 Jan 17]. Available from: http://hdl.handle.net/2027.42/86269.

Council of Science Editors:

Rosenthal DT. Interrogating the Molecular Mechanisms of Breast Cancer Metastasis: The Contributions of RhoC GTPase and p38y MAPK. [Doctoral Dissertation]. University of Michigan; 2011. Available from: http://hdl.handle.net/2027.42/86269

13. Pieczynski, Jay Nicholas. Regulation of Apical Polarity Complexes.

Degree: PhD, Biological Chemistry, 2010, University of Michigan

 Epithelial tissue lines exterior surfaces of the body and organ compartments, providing a multitude of functions including filtration, barrier establishment, and secretion. Epithelial cells are… (more)

Subjects/Keywords: Epithelial Polarity; Biological Chemistry; Molecular, Cellular and Developmental Biology; Health Sciences

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APA (6th Edition):

Pieczynski, J. N. (2010). Regulation of Apical Polarity Complexes. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/78816

Chicago Manual of Style (16th Edition):

Pieczynski, Jay Nicholas. “Regulation of Apical Polarity Complexes.” 2010. Doctoral Dissertation, University of Michigan. Accessed January 17, 2020. http://hdl.handle.net/2027.42/78816.

MLA Handbook (7th Edition):

Pieczynski, Jay Nicholas. “Regulation of Apical Polarity Complexes.” 2010. Web. 17 Jan 2020.

Vancouver:

Pieczynski JN. Regulation of Apical Polarity Complexes. [Internet] [Doctoral dissertation]. University of Michigan; 2010. [cited 2020 Jan 17]. Available from: http://hdl.handle.net/2027.42/78816.

Council of Science Editors:

Pieczynski JN. Regulation of Apical Polarity Complexes. [Doctoral Dissertation]. University of Michigan; 2010. Available from: http://hdl.handle.net/2027.42/78816

14. Lanning, Nathan James. Molecular Mechanisms of Growth Hormone-Induced Signal Transduction and SH2B1.

Degree: PhD, Cellular & Molecular Biology, 2010, University of Michigan

 Growth hormone (GH) regulates overall body growth and metabolism and is used therapeutically for a variety of clinical applications. GH binding to its receptor activates… (more)

Subjects/Keywords: Growth Hormone; JAK2; SH2B1; Focal Adhesion; Spectrin; Phosphorylation; Molecular, Cellular and Developmental Biology; Science

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APA (6th Edition):

Lanning, N. J. (2010). Molecular Mechanisms of Growth Hormone-Induced Signal Transduction and SH2B1. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/75851

Chicago Manual of Style (16th Edition):

Lanning, Nathan James. “Molecular Mechanisms of Growth Hormone-Induced Signal Transduction and SH2B1.” 2010. Doctoral Dissertation, University of Michigan. Accessed January 17, 2020. http://hdl.handle.net/2027.42/75851.

MLA Handbook (7th Edition):

Lanning, Nathan James. “Molecular Mechanisms of Growth Hormone-Induced Signal Transduction and SH2B1.” 2010. Web. 17 Jan 2020.

Vancouver:

Lanning NJ. Molecular Mechanisms of Growth Hormone-Induced Signal Transduction and SH2B1. [Internet] [Doctoral dissertation]. University of Michigan; 2010. [cited 2020 Jan 17]. Available from: http://hdl.handle.net/2027.42/75851.

Council of Science Editors:

Lanning NJ. Molecular Mechanisms of Growth Hormone-Induced Signal Transduction and SH2B1. [Doctoral Dissertation]. University of Michigan; 2010. Available from: http://hdl.handle.net/2027.42/75851

15. Shelby, Shameka J. MERTK-mediated Signaling in the Retinal Pigment Epithelium: Insights into the Mechanism of RPE Phagocytosis.

Degree: PhD, Biological Chemistry, 2012, University of Michigan

 A key function of the retinal pigment epithelium (RPE) is the phagocytic uptake of outer segment (OS) membranes shed from the distal tips of the… (more)

Subjects/Keywords: RPE Phagocytosis; MERTK; Regulation of RPE Phagocytosis; Biological Chemistry; Science

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APA (6th Edition):

Shelby, S. J. (2012). MERTK-mediated Signaling in the Retinal Pigment Epithelium: Insights into the Mechanism of RPE Phagocytosis. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/96052

Chicago Manual of Style (16th Edition):

Shelby, Shameka J. “MERTK-mediated Signaling in the Retinal Pigment Epithelium: Insights into the Mechanism of RPE Phagocytosis.” 2012. Doctoral Dissertation, University of Michigan. Accessed January 17, 2020. http://hdl.handle.net/2027.42/96052.

MLA Handbook (7th Edition):

Shelby, Shameka J. “MERTK-mediated Signaling in the Retinal Pigment Epithelium: Insights into the Mechanism of RPE Phagocytosis.” 2012. Web. 17 Jan 2020.

Vancouver:

Shelby SJ. MERTK-mediated Signaling in the Retinal Pigment Epithelium: Insights into the Mechanism of RPE Phagocytosis. [Internet] [Doctoral dissertation]. University of Michigan; 2012. [cited 2020 Jan 17]. Available from: http://hdl.handle.net/2027.42/96052.

Council of Science Editors:

Shelby SJ. MERTK-mediated Signaling in the Retinal Pigment Epithelium: Insights into the Mechanism of RPE Phagocytosis. [Doctoral Dissertation]. University of Michigan; 2012. Available from: http://hdl.handle.net/2027.42/96052

16. Giebink, Heather M. Insights into the Molecular Mechanism of Axon Outgrowth by Myelin Associated Inhibitors.

Degree: PhD, Biological Chemistry, 2012, University of Michigan

 Repair after injury to the adult mammalian central nervous system (CNS) is hindered by inhibitory proteins, including the myelin associated inhibitors (MAIs): NogoA, MAG, and… (more)

Subjects/Keywords: Myelin Associated Inhibitors; POSH; Biological Chemistry; Health Sciences

…at the University of Michigan has identified potential inhibitors of the Shroom3-ROCK… 

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APA (6th Edition):

Giebink, H. M. (2012). Insights into the Molecular Mechanism of Axon Outgrowth by Myelin Associated Inhibitors. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/93965

Chicago Manual of Style (16th Edition):

Giebink, Heather M. “Insights into the Molecular Mechanism of Axon Outgrowth by Myelin Associated Inhibitors.” 2012. Doctoral Dissertation, University of Michigan. Accessed January 17, 2020. http://hdl.handle.net/2027.42/93965.

MLA Handbook (7th Edition):

Giebink, Heather M. “Insights into the Molecular Mechanism of Axon Outgrowth by Myelin Associated Inhibitors.” 2012. Web. 17 Jan 2020.

Vancouver:

Giebink HM. Insights into the Molecular Mechanism of Axon Outgrowth by Myelin Associated Inhibitors. [Internet] [Doctoral dissertation]. University of Michigan; 2012. [cited 2020 Jan 17]. Available from: http://hdl.handle.net/2027.42/93965.

Council of Science Editors:

Giebink HM. Insights into the Molecular Mechanism of Axon Outgrowth by Myelin Associated Inhibitors. [Doctoral Dissertation]. University of Michigan; 2012. Available from: http://hdl.handle.net/2027.42/93965

17. Llewellyn, George Nicholas. Determinants of HIV-1 Gag Localization to Uropods in Polarized T Cells and the Role Uropods Play in Virus Spread.

Degree: PhD, Cellular & Molecular Biology, 2012, University of Michigan

 HIV-1 is a deadly virus that has killed millions of people around the world. One of the primary targets of HIV-1 in the human body… (more)

Subjects/Keywords: HIV-1 Localizes to Uropods; Microbiology and Immunology; Health Sciences

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APA (6th Edition):

Llewellyn, G. N. (2012). Determinants of HIV-1 Gag Localization to Uropods in Polarized T Cells and the Role Uropods Play in Virus Spread. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/91579

Chicago Manual of Style (16th Edition):

Llewellyn, George Nicholas. “Determinants of HIV-1 Gag Localization to Uropods in Polarized T Cells and the Role Uropods Play in Virus Spread.” 2012. Doctoral Dissertation, University of Michigan. Accessed January 17, 2020. http://hdl.handle.net/2027.42/91579.

MLA Handbook (7th Edition):

Llewellyn, George Nicholas. “Determinants of HIV-1 Gag Localization to Uropods in Polarized T Cells and the Role Uropods Play in Virus Spread.” 2012. Web. 17 Jan 2020.

Vancouver:

Llewellyn GN. Determinants of HIV-1 Gag Localization to Uropods in Polarized T Cells and the Role Uropods Play in Virus Spread. [Internet] [Doctoral dissertation]. University of Michigan; 2012. [cited 2020 Jan 17]. Available from: http://hdl.handle.net/2027.42/91579.

Council of Science Editors:

Llewellyn GN. Determinants of HIV-1 Gag Localization to Uropods in Polarized T Cells and the Role Uropods Play in Virus Spread. [Doctoral Dissertation]. University of Michigan; 2012. Available from: http://hdl.handle.net/2027.42/91579

18. Joe, Ray. Insight into the Brain-Specific Alpha Isoform of the Scaffold Protein SH2B1 and its Rare Obesity-Associated Variants.

Degree: PhD, Cellular & Molecular Biology, 2017, University of Michigan

 Obesity poses a major health problem since it increases the risk for type 2 diabetes, metabolic syndrome, heart disease, and cancer. Mutations in SH2B1 have… (more)

Subjects/Keywords: Cell Signaling; Neurotrophic Factors; Obesity; Scaffold Protein; Tyrosine Kinases; Tyrosine Phosphorylation; Molecular, Cellular and Developmental Biology; Health Sciences

…x28;L. Rui, University of Michigan; personal communication). Both male and female Sh2b1… 

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APA (6th Edition):

Joe, R. (2017). Insight into the Brain-Specific Alpha Isoform of the Scaffold Protein SH2B1 and its Rare Obesity-Associated Variants. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/137175

Chicago Manual of Style (16th Edition):

Joe, Ray. “Insight into the Brain-Specific Alpha Isoform of the Scaffold Protein SH2B1 and its Rare Obesity-Associated Variants.” 2017. Doctoral Dissertation, University of Michigan. Accessed January 17, 2020. http://hdl.handle.net/2027.42/137175.

MLA Handbook (7th Edition):

Joe, Ray. “Insight into the Brain-Specific Alpha Isoform of the Scaffold Protein SH2B1 and its Rare Obesity-Associated Variants.” 2017. Web. 17 Jan 2020.

Vancouver:

Joe R. Insight into the Brain-Specific Alpha Isoform of the Scaffold Protein SH2B1 and its Rare Obesity-Associated Variants. [Internet] [Doctoral dissertation]. University of Michigan; 2017. [cited 2020 Jan 17]. Available from: http://hdl.handle.net/2027.42/137175.

Council of Science Editors:

Joe R. Insight into the Brain-Specific Alpha Isoform of the Scaffold Protein SH2B1 and its Rare Obesity-Associated Variants. [Doctoral Dissertation]. University of Michigan; 2017. Available from: http://hdl.handle.net/2027.42/137175

19. Lam, Alice D. Investigations into the Dynamic Molecular Mechanisms that Govern the Final Stages of Neurotransmitter Release.

Degree: PhD, Neuroscience, 2010, University of Michigan

 The release of neurotransmitter from synaptic vesicles is a process that underlies almost all information transfer within the nervous system. This process is exquisitely regulated,… (more)

Subjects/Keywords: Neurotransmitter Release; Membrane Fusion; SNARE Protein; TIRF; FRET; Neurosciences; Health Sciences

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APA (6th Edition):

Lam, A. D. (2010). Investigations into the Dynamic Molecular Mechanisms that Govern the Final Stages of Neurotransmitter Release. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/75929

Chicago Manual of Style (16th Edition):

Lam, Alice D. “Investigations into the Dynamic Molecular Mechanisms that Govern the Final Stages of Neurotransmitter Release.” 2010. Doctoral Dissertation, University of Michigan. Accessed January 17, 2020. http://hdl.handle.net/2027.42/75929.

MLA Handbook (7th Edition):

Lam, Alice D. “Investigations into the Dynamic Molecular Mechanisms that Govern the Final Stages of Neurotransmitter Release.” 2010. Web. 17 Jan 2020.

Vancouver:

Lam AD. Investigations into the Dynamic Molecular Mechanisms that Govern the Final Stages of Neurotransmitter Release. [Internet] [Doctoral dissertation]. University of Michigan; 2010. [cited 2020 Jan 17]. Available from: http://hdl.handle.net/2027.42/75929.

Council of Science Editors:

Lam AD. Investigations into the Dynamic Molecular Mechanisms that Govern the Final Stages of Neurotransmitter Release. [Doctoral Dissertation]. University of Michigan; 2010. Available from: http://hdl.handle.net/2027.42/75929

20. Philips, Steven T. Investigating the Role of HIP1/PDGFβR in Leukemogenesis and Imatinib Sensitivity.

Degree: PhD, Cellular & Molecular Biology, 2012, University of Michigan

 The Huntingtin interacting protein 1 (HIP1)-platelet-derived growth factor beta receptor (PDGFβR) fusion oncogene (H/P), resulting from a somatic t(5;7) translocation, causes expression of a constitutively… (more)

Subjects/Keywords: HIP1/PDGFbetaR; Chronic Myeloid Leukemia; Imatinib; Oncology and Hematology; Health Sciences

…per UCUCA guidelines at the University of Michigan. The TRAMP and MMTV-myc allele-containing… 

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APA (6th Edition):

Philips, S. T. (2012). Investigating the Role of HIP1/PDGFβR in Leukemogenesis and Imatinib Sensitivity. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/91549

Chicago Manual of Style (16th Edition):

Philips, Steven T. “Investigating the Role of HIP1/PDGFβR in Leukemogenesis and Imatinib Sensitivity.” 2012. Doctoral Dissertation, University of Michigan. Accessed January 17, 2020. http://hdl.handle.net/2027.42/91549.

MLA Handbook (7th Edition):

Philips, Steven T. “Investigating the Role of HIP1/PDGFβR in Leukemogenesis and Imatinib Sensitivity.” 2012. Web. 17 Jan 2020.

Vancouver:

Philips ST. Investigating the Role of HIP1/PDGFβR in Leukemogenesis and Imatinib Sensitivity. [Internet] [Doctoral dissertation]. University of Michigan; 2012. [cited 2020 Jan 17]. Available from: http://hdl.handle.net/2027.42/91549.

Council of Science Editors:

Philips ST. Investigating the Role of HIP1/PDGFβR in Leukemogenesis and Imatinib Sensitivity. [Doctoral Dissertation]. University of Michigan; 2012. Available from: http://hdl.handle.net/2027.42/91549

21. Haenfler, Jill Marie. The role of Lgl, aPKC, and Numb in Distinguishing Neural Stem Cells from progenitor cells in Drosophila.

Degree: PhD, Cellular & Molecular Biology, 2012, University of Michigan

 Asymmetric cell division is a conserved mechanism for distinguishing cells following mitosis in order to produce two daughter cells with unique functions and characteristics. Significant… (more)

Subjects/Keywords: Asymmetric Division; Cell Polarity; Intermediate Neural Progenitor; Neural Stem Cell; Lethal Giant Larvae; Numb; Molecular, Cellular and Developmental Biology; Science

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APA (6th Edition):

Haenfler, J. M. (2012). The role of Lgl, aPKC, and Numb in Distinguishing Neural Stem Cells from progenitor cells in Drosophila. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/94007

Chicago Manual of Style (16th Edition):

Haenfler, Jill Marie. “The role of Lgl, aPKC, and Numb in Distinguishing Neural Stem Cells from progenitor cells in Drosophila.” 2012. Doctoral Dissertation, University of Michigan. Accessed January 17, 2020. http://hdl.handle.net/2027.42/94007.

MLA Handbook (7th Edition):

Haenfler, Jill Marie. “The role of Lgl, aPKC, and Numb in Distinguishing Neural Stem Cells from progenitor cells in Drosophila.” 2012. Web. 17 Jan 2020.

Vancouver:

Haenfler JM. The role of Lgl, aPKC, and Numb in Distinguishing Neural Stem Cells from progenitor cells in Drosophila. [Internet] [Doctoral dissertation]. University of Michigan; 2012. [cited 2020 Jan 17]. Available from: http://hdl.handle.net/2027.42/94007.

Council of Science Editors:

Haenfler JM. The role of Lgl, aPKC, and Numb in Distinguishing Neural Stem Cells from progenitor cells in Drosophila. [Doctoral Dissertation]. University of Michigan; 2012. Available from: http://hdl.handle.net/2027.42/94007


University of Michigan

22. O'Leary, Erin Elizabeth. Identification of Steroid-Sensitive Gene-1 as a Novel JAK2 Binding Protein and the Physiological Consequences of this Interaction.

Degree: PhD, Cellular & Molecular Biology, 2008, University of Michigan

 Growth hormone (GH) is an important regulator of body growth and metabolism. GH binding to its receptor activates the receptor-associated tyrosine kinase, JAK2, which in… (more)

Subjects/Keywords: Growth Hormone; JAK2; Signal Transducers and Activators of Transcription (Stat); Signal Transduction; Steroid-sensitive Gene-1; Biological Chemistry; Science (General); Science

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APA (6th Edition):

O'Leary, E. E. (2008). Identification of Steroid-Sensitive Gene-1 as a Novel JAK2 Binding Protein and the Physiological Consequences of this Interaction. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/61754

Chicago Manual of Style (16th Edition):

O'Leary, Erin Elizabeth. “Identification of Steroid-Sensitive Gene-1 as a Novel JAK2 Binding Protein and the Physiological Consequences of this Interaction.” 2008. Doctoral Dissertation, University of Michigan. Accessed January 17, 2020. http://hdl.handle.net/2027.42/61754.

MLA Handbook (7th Edition):

O'Leary, Erin Elizabeth. “Identification of Steroid-Sensitive Gene-1 as a Novel JAK2 Binding Protein and the Physiological Consequences of this Interaction.” 2008. Web. 17 Jan 2020.

Vancouver:

O'Leary EE. Identification of Steroid-Sensitive Gene-1 as a Novel JAK2 Binding Protein and the Physiological Consequences of this Interaction. [Internet] [Doctoral dissertation]. University of Michigan; 2008. [cited 2020 Jan 17]. Available from: http://hdl.handle.net/2027.42/61754.

Council of Science Editors:

O'Leary EE. Identification of Steroid-Sensitive Gene-1 as a Novel JAK2 Binding Protein and the Physiological Consequences of this Interaction. [Doctoral Dissertation]. University of Michigan; 2008. Available from: http://hdl.handle.net/2027.42/61754


University of Michigan

23. Wang, Qian. The Role of PALS1 in Mammalian Epithelial Polarity.

Degree: PhD, Biological Chemistry, 2007, University of Michigan

 Polarity is an intrinsic feature of epithelial cells, and it is reflected by the differential distribution of proteins and lipids in the apical and basolateral… (more)

Subjects/Keywords: Cell Polarity; PALS1; Biological Chemistry; Health Sciences; Science

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APA (6th Edition):

Wang, Q. (2007). The Role of PALS1 in Mammalian Epithelial Polarity. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/57624

Chicago Manual of Style (16th Edition):

Wang, Qian. “The Role of PALS1 in Mammalian Epithelial Polarity.” 2007. Doctoral Dissertation, University of Michigan. Accessed January 17, 2020. http://hdl.handle.net/2027.42/57624.

MLA Handbook (7th Edition):

Wang, Qian. “The Role of PALS1 in Mammalian Epithelial Polarity.” 2007. Web. 17 Jan 2020.

Vancouver:

Wang Q. The Role of PALS1 in Mammalian Epithelial Polarity. [Internet] [Doctoral dissertation]. University of Michigan; 2007. [cited 2020 Jan 17]. Available from: http://hdl.handle.net/2027.42/57624.

Council of Science Editors:

Wang Q. The Role of PALS1 in Mammalian Epithelial Polarity. [Doctoral Dissertation]. University of Michigan; 2007. Available from: http://hdl.handle.net/2027.42/57624


University of Michigan

24. Moore, Brian A. Structures of Exocyst Subunit Exo70 from Yeast and Mouse.

Degree: PhD, Biological Chemistry, 2007, University of Michigan

 Exocytosis is a eukaryotic process in which vesicles deliver membrane and other cargoes to and across the plasma membrane. The exocyst is a tethering complex… (more)

Subjects/Keywords: Exocyst; Exocytosis; Vesicle Tethering; Vesicle Transport; X-ray Crystallography; Biological Chemistry; Health Sciences; Science

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APA (6th Edition):

Moore, B. A. (2007). Structures of Exocyst Subunit Exo70 from Yeast and Mouse. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/57641

Chicago Manual of Style (16th Edition):

Moore, Brian A. “Structures of Exocyst Subunit Exo70 from Yeast and Mouse.” 2007. Doctoral Dissertation, University of Michigan. Accessed January 17, 2020. http://hdl.handle.net/2027.42/57641.

MLA Handbook (7th Edition):

Moore, Brian A. “Structures of Exocyst Subunit Exo70 from Yeast and Mouse.” 2007. Web. 17 Jan 2020.

Vancouver:

Moore BA. Structures of Exocyst Subunit Exo70 from Yeast and Mouse. [Internet] [Doctoral dissertation]. University of Michigan; 2007. [cited 2020 Jan 17]. Available from: http://hdl.handle.net/2027.42/57641.

Council of Science Editors:

Moore BA. Structures of Exocyst Subunit Exo70 from Yeast and Mouse. [Doctoral Dissertation]. University of Michigan; 2007. Available from: http://hdl.handle.net/2027.42/57641


University of Michigan

25. Chen, Xiao-wei. Coordination of Cell Signaling and Transport Machineries in Insulin-Stimulated Glucose Transport.

Degree: PhD, Molecular and Integrative Physiology, 2008, University of Michigan

 Insulin-stimulated glucose transport is the rate-limiting step in glucose disposal and utilization. Insulin increases glucose uptake in fat and muscle through the translocation of the… (more)

Subjects/Keywords: Insulin; Glut4; Trafficking; Physiology; Science

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APA (6th Edition):

Chen, X. (2008). Coordination of Cell Signaling and Transport Machineries in Insulin-Stimulated Glucose Transport. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/58527

Chicago Manual of Style (16th Edition):

Chen, Xiao-wei. “Coordination of Cell Signaling and Transport Machineries in Insulin-Stimulated Glucose Transport.” 2008. Doctoral Dissertation, University of Michigan. Accessed January 17, 2020. http://hdl.handle.net/2027.42/58527.

MLA Handbook (7th Edition):

Chen, Xiao-wei. “Coordination of Cell Signaling and Transport Machineries in Insulin-Stimulated Glucose Transport.” 2008. Web. 17 Jan 2020.

Vancouver:

Chen X. Coordination of Cell Signaling and Transport Machineries in Insulin-Stimulated Glucose Transport. [Internet] [Doctoral dissertation]. University of Michigan; 2008. [cited 2020 Jan 17]. Available from: http://hdl.handle.net/2027.42/58527.

Council of Science Editors:

Chen X. Coordination of Cell Signaling and Transport Machineries in Insulin-Stimulated Glucose Transport. [Doctoral Dissertation]. University of Michigan; 2008. Available from: http://hdl.handle.net/2027.42/58527


University of Michigan

26. Lee, Chung-Han. Glucose Starvation Induces Apoptosis of TSC-/- cells in a p53-dependent Glucose Starvation Induces Apoptosis of TSC-/- cells in a p53-dependent Manner.

Degree: PhD, Biological Chemistry, 2009, University of Michigan

 Tuberous Sclerosis Complex (TSC) is associated with uncontrolled mTOR activation and hamartoma formation. We show that glucose starvation induces apoptosis in TSC cells in a… (more)

Subjects/Keywords: MTOR; Apoptosis; P53; Rapamycin

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APA (6th Edition):

Lee, C. (2009). Glucose Starvation Induces Apoptosis of TSC-/- cells in a p53-dependent Glucose Starvation Induces Apoptosis of TSC-/- cells in a p53-dependent Manner. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/62191

Chicago Manual of Style (16th Edition):

Lee, Chung-Han. “Glucose Starvation Induces Apoptosis of TSC-/- cells in a p53-dependent Glucose Starvation Induces Apoptosis of TSC-/- cells in a p53-dependent Manner.” 2009. Doctoral Dissertation, University of Michigan. Accessed January 17, 2020. http://hdl.handle.net/2027.42/62191.

MLA Handbook (7th Edition):

Lee, Chung-Han. “Glucose Starvation Induces Apoptosis of TSC-/- cells in a p53-dependent Glucose Starvation Induces Apoptosis of TSC-/- cells in a p53-dependent Manner.” 2009. Web. 17 Jan 2020.

Vancouver:

Lee C. Glucose Starvation Induces Apoptosis of TSC-/- cells in a p53-dependent Glucose Starvation Induces Apoptosis of TSC-/- cells in a p53-dependent Manner. [Internet] [Doctoral dissertation]. University of Michigan; 2009. [cited 2020 Jan 17]. Available from: http://hdl.handle.net/2027.42/62191.

Council of Science Editors:

Lee C. Glucose Starvation Induces Apoptosis of TSC-/- cells in a p53-dependent Glucose Starvation Induces Apoptosis of TSC-/- cells in a p53-dependent Manner. [Doctoral Dissertation]. University of Michigan; 2009. Available from: http://hdl.handle.net/2027.42/62191


University of Michigan

27. Zhao, Bin. Inactivation of YAP-TEAD by the Hippo Pathway is Involved in Growth Control Inactivation of YAP-TEAD by the Hippo Pathway is Involved in Growth Control and Cancer.

Degree: PhD, Biological Chemistry, 2009, University of Michigan

 The mechanism of body and organ size control is an unsolved puzzle. Recent Drosophila genetics studies established the key role of the Hippo pathway and… (more)

Subjects/Keywords: YAP; TEAD; Cancer; Biological Chemistry; Science

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APA (6th Edition):

Zhao, B. (2009). Inactivation of YAP-TEAD by the Hippo Pathway is Involved in Growth Control Inactivation of YAP-TEAD by the Hippo Pathway is Involved in Growth Control and Cancer. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/62289

Chicago Manual of Style (16th Edition):

Zhao, Bin. “Inactivation of YAP-TEAD by the Hippo Pathway is Involved in Growth Control Inactivation of YAP-TEAD by the Hippo Pathway is Involved in Growth Control and Cancer.” 2009. Doctoral Dissertation, University of Michigan. Accessed January 17, 2020. http://hdl.handle.net/2027.42/62289.

MLA Handbook (7th Edition):

Zhao, Bin. “Inactivation of YAP-TEAD by the Hippo Pathway is Involved in Growth Control Inactivation of YAP-TEAD by the Hippo Pathway is Involved in Growth Control and Cancer.” 2009. Web. 17 Jan 2020.

Vancouver:

Zhao B. Inactivation of YAP-TEAD by the Hippo Pathway is Involved in Growth Control Inactivation of YAP-TEAD by the Hippo Pathway is Involved in Growth Control and Cancer. [Internet] [Doctoral dissertation]. University of Michigan; 2009. [cited 2020 Jan 17]. Available from: http://hdl.handle.net/2027.42/62289.

Council of Science Editors:

Zhao B. Inactivation of YAP-TEAD by the Hippo Pathway is Involved in Growth Control Inactivation of YAP-TEAD by the Hippo Pathway is Involved in Growth Control and Cancer. [Doctoral Dissertation]. University of Michigan; 2009. Available from: http://hdl.handle.net/2027.42/62289


University of Michigan

28. Lapinski, Philip E. The Role of p120 RasGAP in T Cells and Lymphatics.

Degree: PhD, Immunology, 2009, University of Michigan

 ABSTRACT THE ROLE OF p120 RasGAP IN T CELLS AND LYMPHATICS by Philip E. Lapinski Chair: Philip D. King Ras is an intracellular signaling molecule… (more)

Subjects/Keywords: RASA1; T Cells; Lymphatics; Gene Targeting; Microbiology and Immunology; Science

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APA (6th Edition):

Lapinski, P. E. (2009). The Role of p120 RasGAP in T Cells and Lymphatics. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/63814

Chicago Manual of Style (16th Edition):

Lapinski, Philip E. “The Role of p120 RasGAP in T Cells and Lymphatics.” 2009. Doctoral Dissertation, University of Michigan. Accessed January 17, 2020. http://hdl.handle.net/2027.42/63814.

MLA Handbook (7th Edition):

Lapinski, Philip E. “The Role of p120 RasGAP in T Cells and Lymphatics.” 2009. Web. 17 Jan 2020.

Vancouver:

Lapinski PE. The Role of p120 RasGAP in T Cells and Lymphatics. [Internet] [Doctoral dissertation]. University of Michigan; 2009. [cited 2020 Jan 17]. Available from: http://hdl.handle.net/2027.42/63814.

Council of Science Editors:

Lapinski PE. The Role of p120 RasGAP in T Cells and Lymphatics. [Doctoral Dissertation]. University of Michigan; 2009. Available from: http://hdl.handle.net/2027.42/63814

.