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You searched for +publisher:"University of Michigan" +contributor:("Lin, Jiandie"). Showing records 1 – 21 of 21 total matches.

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1. Chen, Tzong-Yang. Critical Genes Regulated by FoxO Transcription Factors in Life Span Control.

Degree: PhD, Cellular & Molec Biology PhD, 2017, University of Michigan

 FoxO transcription factors (TFs) control metabolism, development, and aging in diverse species. Mouse models implicate FoxO dysregulation in the pathogenesis of age-related disease, including type… (more)

Subjects/Keywords: aging; genetics; longevity; Molecular, Cellular and Developmental Biology; Science

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APA (6th Edition):

Chen, T. (2017). Critical Genes Regulated by FoxO Transcription Factors in Life Span Control. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/137072

Chicago Manual of Style (16th Edition):

Chen, Tzong-Yang. “Critical Genes Regulated by FoxO Transcription Factors in Life Span Control.” 2017. Doctoral Dissertation, University of Michigan. Accessed January 19, 2020. http://hdl.handle.net/2027.42/137072.

MLA Handbook (7th Edition):

Chen, Tzong-Yang. “Critical Genes Regulated by FoxO Transcription Factors in Life Span Control.” 2017. Web. 19 Jan 2020.

Vancouver:

Chen T. Critical Genes Regulated by FoxO Transcription Factors in Life Span Control. [Internet] [Doctoral dissertation]. University of Michigan; 2017. [cited 2020 Jan 19]. Available from: http://hdl.handle.net/2027.42/137072.

Council of Science Editors:

Chen T. Critical Genes Regulated by FoxO Transcription Factors in Life Span Control. [Doctoral Dissertation]. University of Michigan; 2017. Available from: http://hdl.handle.net/2027.42/137072


University of Michigan

2. Uhm, Maeran. Role of the Protein Kinase TBK1 in Insulin-Stimulated Glucose Transport.

Degree: PhD, Molecular and Integrative Physiology, 2015, University of Michigan

 Insulin stimulates glucose uptake in muscle and fat by promoting translocation of the facilitative transporter GLUT4 from intracellular compartments to the plasma membrane. While the… (more)

Subjects/Keywords: Insulin-stimulated glucose transport; Physiology; Health Sciences

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APA (6th Edition):

Uhm, M. (2015). Role of the Protein Kinase TBK1 in Insulin-Stimulated Glucose Transport. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/113634

Chicago Manual of Style (16th Edition):

Uhm, Maeran. “Role of the Protein Kinase TBK1 in Insulin-Stimulated Glucose Transport.” 2015. Doctoral Dissertation, University of Michigan. Accessed January 19, 2020. http://hdl.handle.net/2027.42/113634.

MLA Handbook (7th Edition):

Uhm, Maeran. “Role of the Protein Kinase TBK1 in Insulin-Stimulated Glucose Transport.” 2015. Web. 19 Jan 2020.

Vancouver:

Uhm M. Role of the Protein Kinase TBK1 in Insulin-Stimulated Glucose Transport. [Internet] [Doctoral dissertation]. University of Michigan; 2015. [cited 2020 Jan 19]. Available from: http://hdl.handle.net/2027.42/113634.

Council of Science Editors:

Uhm M. Role of the Protein Kinase TBK1 in Insulin-Stimulated Glucose Transport. [Doctoral Dissertation]. University of Michigan; 2015. Available from: http://hdl.handle.net/2027.42/113634


University of Michigan

3. Doche, Michael Edgar. Effects of Human Obesity-associated Mutations on Cellular Actions of the Adapter Protein SH2B1.

Degree: PhD, Molecular and Integrative Physiology, 2013, University of Michigan

 Src homology 2 B adapter protein 1 (SH2B1) modulates signaling by a variety of ligands that bind to receptor tyrosine kinases or JAK-associated cytokine receptors,… (more)

Subjects/Keywords: Genetics of Obesity; Leptin-resistance, Insulin-resistance and Hyperphagia; SH2B1 Regulation of Whole-body Energy Homeostasis; Genetics; Medicine (General); Neurosciences; Physiology; Health Sciences; Science

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APA (6th Edition):

Doche, M. E. (2013). Effects of Human Obesity-associated Mutations on Cellular Actions of the Adapter Protein SH2B1. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/98053

Chicago Manual of Style (16th Edition):

Doche, Michael Edgar. “Effects of Human Obesity-associated Mutations on Cellular Actions of the Adapter Protein SH2B1.” 2013. Doctoral Dissertation, University of Michigan. Accessed January 19, 2020. http://hdl.handle.net/2027.42/98053.

MLA Handbook (7th Edition):

Doche, Michael Edgar. “Effects of Human Obesity-associated Mutations on Cellular Actions of the Adapter Protein SH2B1.” 2013. Web. 19 Jan 2020.

Vancouver:

Doche ME. Effects of Human Obesity-associated Mutations on Cellular Actions of the Adapter Protein SH2B1. [Internet] [Doctoral dissertation]. University of Michigan; 2013. [cited 2020 Jan 19]. Available from: http://hdl.handle.net/2027.42/98053.

Council of Science Editors:

Doche ME. Effects of Human Obesity-associated Mutations on Cellular Actions of the Adapter Protein SH2B1. [Doctoral Dissertation]. University of Michigan; 2013. Available from: http://hdl.handle.net/2027.42/98053


University of Michigan

4. Dai, Wei. New Insights from Studying Insulin-like Growth Factor (IGF)-Binding Protein-5 in Zebrafish : Role of IGF Signaling in Epithelial Calcium Absorption.

Degree: PhD, Molecular, Cellular, and Developmental Biology, 2011, University of Michigan

 Insulin-like growth factors (IGFs) are potent regulators of development and homeostasis. IGF-binding proteins (IGFBPs) are important modulators of IGF signaling. In this study, I took… (more)

Subjects/Keywords: Acclimation; Calcium Homeostasis; Molecular, Cellular and Developmental Biology; Science

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APA (6th Edition):

Dai, W. (2011). New Insights from Studying Insulin-like Growth Factor (IGF)-Binding Protein-5 in Zebrafish : Role of IGF Signaling in Epithelial Calcium Absorption. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/84651

Chicago Manual of Style (16th Edition):

Dai, Wei. “New Insights from Studying Insulin-like Growth Factor (IGF)-Binding Protein-5 in Zebrafish : Role of IGF Signaling in Epithelial Calcium Absorption.” 2011. Doctoral Dissertation, University of Michigan. Accessed January 19, 2020. http://hdl.handle.net/2027.42/84651.

MLA Handbook (7th Edition):

Dai, Wei. “New Insights from Studying Insulin-like Growth Factor (IGF)-Binding Protein-5 in Zebrafish : Role of IGF Signaling in Epithelial Calcium Absorption.” 2011. Web. 19 Jan 2020.

Vancouver:

Dai W. New Insights from Studying Insulin-like Growth Factor (IGF)-Binding Protein-5 in Zebrafish : Role of IGF Signaling in Epithelial Calcium Absorption. [Internet] [Doctoral dissertation]. University of Michigan; 2011. [cited 2020 Jan 19]. Available from: http://hdl.handle.net/2027.42/84651.

Council of Science Editors:

Dai W. New Insights from Studying Insulin-like Growth Factor (IGF)-Binding Protein-5 in Zebrafish : Role of IGF Signaling in Epithelial Calcium Absorption. [Doctoral Dissertation]. University of Michigan; 2011. Available from: http://hdl.handle.net/2027.42/84651


University of Michigan

5. Zhang, Liang Samantha. Photoperiodic Properties of Circadian Rhythm in Rat.

Degree: PhD, Neuroscience, 2011, University of Michigan

 Animals have intrinsic circadian periods of around 24 hours. In order to synchronize to the 24-hour day, circadian rhythms entrain to photic time cues and… (more)

Subjects/Keywords: Circadian Rhythms; Melatonin; Photoperiod; Free-running Period; Phase Angle of Entrainment; Jetlag; Neurosciences; Health Sciences

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APA (6th Edition):

Zhang, L. S. (2011). Photoperiodic Properties of Circadian Rhythm in Rat. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/89623

Chicago Manual of Style (16th Edition):

Zhang, Liang Samantha. “Photoperiodic Properties of Circadian Rhythm in Rat.” 2011. Doctoral Dissertation, University of Michigan. Accessed January 19, 2020. http://hdl.handle.net/2027.42/89623.

MLA Handbook (7th Edition):

Zhang, Liang Samantha. “Photoperiodic Properties of Circadian Rhythm in Rat.” 2011. Web. 19 Jan 2020.

Vancouver:

Zhang LS. Photoperiodic Properties of Circadian Rhythm in Rat. [Internet] [Doctoral dissertation]. University of Michigan; 2011. [cited 2020 Jan 19]. Available from: http://hdl.handle.net/2027.42/89623.

Council of Science Editors:

Zhang LS. Photoperiodic Properties of Circadian Rhythm in Rat. [Doctoral Dissertation]. University of Michigan; 2011. Available from: http://hdl.handle.net/2027.42/89623


University of Michigan

6. Molusky, Matthew M. Control of Glucose Homeostatis through Ubiquitin-Specific Protease 2.

Degree: PhD, Cellular & Molecular Biology, 2011, University of Michigan

 Hepatic gluconeogenesis is important for maintaining steady blood glucose levels during starvation and through light/dark cycles. The regulatory network that transduces hormonal and circadian signals… (more)

Subjects/Keywords: Glucose Homeostasis; Gluconeogenesis; Glucocorticoids; Molecular, Cellular and Developmental Biology; Science

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APA (6th Edition):

Molusky, M. M. (2011). Control of Glucose Homeostatis through Ubiquitin-Specific Protease 2. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/89630

Chicago Manual of Style (16th Edition):

Molusky, Matthew M. “Control of Glucose Homeostatis through Ubiquitin-Specific Protease 2.” 2011. Doctoral Dissertation, University of Michigan. Accessed January 19, 2020. http://hdl.handle.net/2027.42/89630.

MLA Handbook (7th Edition):

Molusky, Matthew M. “Control of Glucose Homeostatis through Ubiquitin-Specific Protease 2.” 2011. Web. 19 Jan 2020.

Vancouver:

Molusky MM. Control of Glucose Homeostatis through Ubiquitin-Specific Protease 2. [Internet] [Doctoral dissertation]. University of Michigan; 2011. [cited 2020 Jan 19]. Available from: http://hdl.handle.net/2027.42/89630.

Council of Science Editors:

Molusky MM. Control of Glucose Homeostatis through Ubiquitin-Specific Protease 2. [Doctoral Dissertation]. University of Michigan; 2011. Available from: http://hdl.handle.net/2027.42/89630


University of Michigan

7. Funai, Katsuhiko. Effects of In Vivo Exercise and In Vitro Contractile Activity on the Regulation of AS160, TBC1D1 and Glucose Transport in Rat Skeletal Muscle.

Degree: PhD, Kinesiology, 2010, University of Michigan

 A single bout of exercise leads to an increase in insulin-independent and insulin-dependent increase in glucose transport (GT). Phosphorylation of two members of the TBC1… (more)

Subjects/Keywords: Insulin Sensitivity; Glucose Uptake; GLUT4 Translocation; Glycogen; AMPK; CaMK; Kinesiology and Sports; Molecular, Cellular and Developmental Biology; Physiology; Health Sciences

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APA (6th Edition):

Funai, K. (2010). Effects of In Vivo Exercise and In Vitro Contractile Activity on the Regulation of AS160, TBC1D1 and Glucose Transport in Rat Skeletal Muscle. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/75927

Chicago Manual of Style (16th Edition):

Funai, Katsuhiko. “Effects of In Vivo Exercise and In Vitro Contractile Activity on the Regulation of AS160, TBC1D1 and Glucose Transport in Rat Skeletal Muscle.” 2010. Doctoral Dissertation, University of Michigan. Accessed January 19, 2020. http://hdl.handle.net/2027.42/75927.

MLA Handbook (7th Edition):

Funai, Katsuhiko. “Effects of In Vivo Exercise and In Vitro Contractile Activity on the Regulation of AS160, TBC1D1 and Glucose Transport in Rat Skeletal Muscle.” 2010. Web. 19 Jan 2020.

Vancouver:

Funai K. Effects of In Vivo Exercise and In Vitro Contractile Activity on the Regulation of AS160, TBC1D1 and Glucose Transport in Rat Skeletal Muscle. [Internet] [Doctoral dissertation]. University of Michigan; 2010. [cited 2020 Jan 19]. Available from: http://hdl.handle.net/2027.42/75927.

Council of Science Editors:

Funai K. Effects of In Vivo Exercise and In Vitro Contractile Activity on the Regulation of AS160, TBC1D1 and Glucose Transport in Rat Skeletal Muscle. [Doctoral Dissertation]. University of Michigan; 2010. Available from: http://hdl.handle.net/2027.42/75927


University of Michigan

8. Pineault, Kyriel. Hox11-Expression Defines a Skeletal Mesenchymal Stem Cell that Contributes to Skeleton Development, Growth, and Repair.

Degree: PhD, Cell and Developmental Biology, 2018, University of Michigan

 The skeleton is one of the most widely explored organs for defining mesenchymal stem/progenitor cell (MSC) populations, and there is significant interest in MSCs for… (more)

Subjects/Keywords: Hox genes; skeleton stem cell; mesenchymal stem/stromal cell (MSC); skeletal patterning; lineage-trace; developmental biology; Molecular, Cellular and Developmental Biology; Science

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APA (6th Edition):

Pineault, K. (2018). Hox11-Expression Defines a Skeletal Mesenchymal Stem Cell that Contributes to Skeleton Development, Growth, and Repair. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/147658

Chicago Manual of Style (16th Edition):

Pineault, Kyriel. “Hox11-Expression Defines a Skeletal Mesenchymal Stem Cell that Contributes to Skeleton Development, Growth, and Repair.” 2018. Doctoral Dissertation, University of Michigan. Accessed January 19, 2020. http://hdl.handle.net/2027.42/147658.

MLA Handbook (7th Edition):

Pineault, Kyriel. “Hox11-Expression Defines a Skeletal Mesenchymal Stem Cell that Contributes to Skeleton Development, Growth, and Repair.” 2018. Web. 19 Jan 2020.

Vancouver:

Pineault K. Hox11-Expression Defines a Skeletal Mesenchymal Stem Cell that Contributes to Skeleton Development, Growth, and Repair. [Internet] [Doctoral dissertation]. University of Michigan; 2018. [cited 2020 Jan 19]. Available from: http://hdl.handle.net/2027.42/147658.

Council of Science Editors:

Pineault K. Hox11-Expression Defines a Skeletal Mesenchymal Stem Cell that Contributes to Skeleton Development, Growth, and Repair. [Doctoral Dissertation]. University of Michigan; 2018. Available from: http://hdl.handle.net/2027.42/147658


University of Michigan

9. Chung, Jooho. Cellular and Molecular Analysis of Notch Signaling in T Cells after Allogeneic Bone Marrow Transplantation.

Degree: PhD, Cellular & Molec Biology PhD, 2018, University of Michigan

 Allogeneic bone marrow transplantation (allo-BMT) is a potentially curative therapy for patients with cancer and hematological disorders. However, its success is limited by graft-versus-host disease… (more)

Subjects/Keywords: Notch signaling after allogeneic bone marrow transplantation; Molecular, Cellular and Developmental Biology; Science

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APA (6th Edition):

Chung, J. (2018). Cellular and Molecular Analysis of Notch Signaling in T Cells after Allogeneic Bone Marrow Transplantation. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/144004

Chicago Manual of Style (16th Edition):

Chung, Jooho. “Cellular and Molecular Analysis of Notch Signaling in T Cells after Allogeneic Bone Marrow Transplantation.” 2018. Doctoral Dissertation, University of Michigan. Accessed January 19, 2020. http://hdl.handle.net/2027.42/144004.

MLA Handbook (7th Edition):

Chung, Jooho. “Cellular and Molecular Analysis of Notch Signaling in T Cells after Allogeneic Bone Marrow Transplantation.” 2018. Web. 19 Jan 2020.

Vancouver:

Chung J. Cellular and Molecular Analysis of Notch Signaling in T Cells after Allogeneic Bone Marrow Transplantation. [Internet] [Doctoral dissertation]. University of Michigan; 2018. [cited 2020 Jan 19]. Available from: http://hdl.handle.net/2027.42/144004.

Council of Science Editors:

Chung J. Cellular and Molecular Analysis of Notch Signaling in T Cells after Allogeneic Bone Marrow Transplantation. [Doctoral Dissertation]. University of Michigan; 2018. Available from: http://hdl.handle.net/2027.42/144004

10. Harvey, Innocence. Adipose and Muscle Tissue in Glucocorticoid-Induced Metabolic Disease.

Degree: PhD, Nutritional Sciences, 2018, University of Michigan

 Introduction: Glucocorticoids are steroid hormones induced by stress that are necessaryfor proper glucose homeostasis and tissue development. Glucocorticoids also have potent immunosuppressant properties and are… (more)

Subjects/Keywords: Glucocorticoids; Lipolysis; Insulin resistance; Non-alcoholic fatty liver disease (NAFLD); Juvenile dexamethasone exposure; Muscle atrophy; Biological Chemistry; Genetics; Physiology; Science (General); Science

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APA (6th Edition):

Harvey, I. (2018). Adipose and Muscle Tissue in Glucocorticoid-Induced Metabolic Disease. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/147601

Chicago Manual of Style (16th Edition):

Harvey, Innocence. “Adipose and Muscle Tissue in Glucocorticoid-Induced Metabolic Disease.” 2018. Doctoral Dissertation, University of Michigan. Accessed January 19, 2020. http://hdl.handle.net/2027.42/147601.

MLA Handbook (7th Edition):

Harvey, Innocence. “Adipose and Muscle Tissue in Glucocorticoid-Induced Metabolic Disease.” 2018. Web. 19 Jan 2020.

Vancouver:

Harvey I. Adipose and Muscle Tissue in Glucocorticoid-Induced Metabolic Disease. [Internet] [Doctoral dissertation]. University of Michigan; 2018. [cited 2020 Jan 19]. Available from: http://hdl.handle.net/2027.42/147601.

Council of Science Editors:

Harvey I. Adipose and Muscle Tissue in Glucocorticoid-Induced Metabolic Disease. [Doctoral Dissertation]. University of Michigan; 2018. Available from: http://hdl.handle.net/2027.42/147601

11. Emont, Margo. Subcutaneous Fat: Thermogenesis and Metabolic Benefits.

Degree: PhD, Molecular and Integrative Physiology, 2017, University of Michigan

 Obesity and its associated metabolic diseases present a major public health problem around the world. The discovery that thermogenic fat is active in adult humans… (more)

Subjects/Keywords: beige fat; brown fat; obesity; thermogenesis; Physiology; Science

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APA (6th Edition):

Emont, M. (2017). Subcutaneous Fat: Thermogenesis and Metabolic Benefits. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/144148

Chicago Manual of Style (16th Edition):

Emont, Margo. “Subcutaneous Fat: Thermogenesis and Metabolic Benefits.” 2017. Doctoral Dissertation, University of Michigan. Accessed January 19, 2020. http://hdl.handle.net/2027.42/144148.

MLA Handbook (7th Edition):

Emont, Margo. “Subcutaneous Fat: Thermogenesis and Metabolic Benefits.” 2017. Web. 19 Jan 2020.

Vancouver:

Emont M. Subcutaneous Fat: Thermogenesis and Metabolic Benefits. [Internet] [Doctoral dissertation]. University of Michigan; 2017. [cited 2020 Jan 19]. Available from: http://hdl.handle.net/2027.42/144148.

Council of Science Editors:

Emont M. Subcutaneous Fat: Thermogenesis and Metabolic Benefits. [Doctoral Dissertation]. University of Michigan; 2017. Available from: http://hdl.handle.net/2027.42/144148

12. Cunningham, Corey. Cells Deploy a Two-Pronged Quality Control Strategy to Degrade Misfolded Proinsulin Mutants.

Degree: PhD, Cellular & Molecular Biology, 2019, University of Michigan

 There are over 30 missense mutations found in the human insulin gene responsible for a newly-characterized diabetic syndrome called Mutant INS-gene-induced Diabetes of Youth (MIDY).… (more)

Subjects/Keywords: Protein Quality Control; Endoplasmic Reticulum; Mutant INS-gene-induced Diabetes of Youth; ER-associated degradation; ER-autophagy; Biological Chemistry; Molecular, Cellular and Developmental Biology; Science (General); Science

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APA (6th Edition):

Cunningham, C. (2019). Cells Deploy a Two-Pronged Quality Control Strategy to Degrade Misfolded Proinsulin Mutants. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/149827

Chicago Manual of Style (16th Edition):

Cunningham, Corey. “Cells Deploy a Two-Pronged Quality Control Strategy to Degrade Misfolded Proinsulin Mutants.” 2019. Doctoral Dissertation, University of Michigan. Accessed January 19, 2020. http://hdl.handle.net/2027.42/149827.

MLA Handbook (7th Edition):

Cunningham, Corey. “Cells Deploy a Two-Pronged Quality Control Strategy to Degrade Misfolded Proinsulin Mutants.” 2019. Web. 19 Jan 2020.

Vancouver:

Cunningham C. Cells Deploy a Two-Pronged Quality Control Strategy to Degrade Misfolded Proinsulin Mutants. [Internet] [Doctoral dissertation]. University of Michigan; 2019. [cited 2020 Jan 19]. Available from: http://hdl.handle.net/2027.42/149827.

Council of Science Editors:

Cunningham C. Cells Deploy a Two-Pronged Quality Control Strategy to Degrade Misfolded Proinsulin Mutants. [Doctoral Dissertation]. University of Michigan; 2019. Available from: http://hdl.handle.net/2027.42/149827

13. Ozkurede, Varol Ulas. Improved Mitochondrial Stress Response in Long-lived Snell Dwarf Mice.

Degree: PhD, Molecular & Cellular Pathology, 2018, University of Michigan

 Upregulation of the mitochondrial unfolded protein response (mtUPR) as a result of alterations in mitochondrial protein stoichiometry has been proposed as a common pathway in… (more)

Subjects/Keywords: Aging; Mitochondrial Stress Response; mtUPR; Longevity; Lifespan Extension; Snell Dwarf Mice; Geriatrics; Molecular, Cellular and Developmental Biology; Pathology; Health Sciences; Science

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APA (6th Edition):

Ozkurede, V. U. (2018). Improved Mitochondrial Stress Response in Long-lived Snell Dwarf Mice. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/147485

Chicago Manual of Style (16th Edition):

Ozkurede, Varol Ulas. “Improved Mitochondrial Stress Response in Long-lived Snell Dwarf Mice.” 2018. Doctoral Dissertation, University of Michigan. Accessed January 19, 2020. http://hdl.handle.net/2027.42/147485.

MLA Handbook (7th Edition):

Ozkurede, Varol Ulas. “Improved Mitochondrial Stress Response in Long-lived Snell Dwarf Mice.” 2018. Web. 19 Jan 2020.

Vancouver:

Ozkurede VU. Improved Mitochondrial Stress Response in Long-lived Snell Dwarf Mice. [Internet] [Doctoral dissertation]. University of Michigan; 2018. [cited 2020 Jan 19]. Available from: http://hdl.handle.net/2027.42/147485.

Council of Science Editors:

Ozkurede VU. Improved Mitochondrial Stress Response in Long-lived Snell Dwarf Mice. [Doctoral Dissertation]. University of Michigan; 2018. Available from: http://hdl.handle.net/2027.42/147485

14. Ma, Di. Autophagy: Circadian Regulation and Role in Non-Alcoholic Fatty Liver Disease.

Degree: PhD, Cell and Developmental Biology, 2013, University of Michigan

 Metabolic syndrome has become a global health care challenge characterized by increased risk for type 2 diabetes, cardiovascular disease, and non-alcoholic fatty liver disease. Recent… (more)

Subjects/Keywords: Autophagy; Circadian Clock; Nonalcoholic Steatohepatitis; Molecular, Cellular and Developmental Biology; Science

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APA (6th Edition):

Ma, D. (2013). Autophagy: Circadian Regulation and Role in Non-Alcoholic Fatty Liver Disease. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/98023

Chicago Manual of Style (16th Edition):

Ma, Di. “Autophagy: Circadian Regulation and Role in Non-Alcoholic Fatty Liver Disease.” 2013. Doctoral Dissertation, University of Michigan. Accessed January 19, 2020. http://hdl.handle.net/2027.42/98023.

MLA Handbook (7th Edition):

Ma, Di. “Autophagy: Circadian Regulation and Role in Non-Alcoholic Fatty Liver Disease.” 2013. Web. 19 Jan 2020.

Vancouver:

Ma D. Autophagy: Circadian Regulation and Role in Non-Alcoholic Fatty Liver Disease. [Internet] [Doctoral dissertation]. University of Michigan; 2013. [cited 2020 Jan 19]. Available from: http://hdl.handle.net/2027.42/98023.

Council of Science Editors:

Ma D. Autophagy: Circadian Regulation and Role in Non-Alcoholic Fatty Liver Disease. [Doctoral Dissertation]. University of Michigan; 2013. Available from: http://hdl.handle.net/2027.42/98023

15. Simon, Becky R. Regulation of Adipocyte Differentiation and Metabolism by Artificial Sweeteners and Sweet Taste Receptors.

Degree: PhD, Cellular & Molecular Biology, 2013, University of Michigan

 To maintain energetic homeostasis, adipose tissue must be uniquely sensitive to nutritional status. One mechanism for nutrient sensing in metabolic systems is through sweet taste… (more)

Subjects/Keywords: Adipogenesis; Nutrient Sensing; Taste Receptors; Molecular, Cellular and Developmental Biology; Health Sciences; Science

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APA (6th Edition):

Simon, B. R. (2013). Regulation of Adipocyte Differentiation and Metabolism by Artificial Sweeteners and Sweet Taste Receptors. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/99775

Chicago Manual of Style (16th Edition):

Simon, Becky R. “Regulation of Adipocyte Differentiation and Metabolism by Artificial Sweeteners and Sweet Taste Receptors.” 2013. Doctoral Dissertation, University of Michigan. Accessed January 19, 2020. http://hdl.handle.net/2027.42/99775.

MLA Handbook (7th Edition):

Simon, Becky R. “Regulation of Adipocyte Differentiation and Metabolism by Artificial Sweeteners and Sweet Taste Receptors.” 2013. Web. 19 Jan 2020.

Vancouver:

Simon BR. Regulation of Adipocyte Differentiation and Metabolism by Artificial Sweeteners and Sweet Taste Receptors. [Internet] [Doctoral dissertation]. University of Michigan; 2013. [cited 2020 Jan 19]. Available from: http://hdl.handle.net/2027.42/99775.

Council of Science Editors:

Simon BR. Regulation of Adipocyte Differentiation and Metabolism by Artificial Sweeteners and Sweet Taste Receptors. [Doctoral Dissertation]. University of Michigan; 2013. Available from: http://hdl.handle.net/2027.42/99775

16. Lin, Grace. A Novel Transcriptional Repressor-activator Relationship in Growth Hormone-regulated Gene Expression.

Degree: PhD, Cellular and Molecular Biology, 2012, University of Michigan

 Growth Hormone (GH), a major regulator of normal growth and metabolism, regulates diverse physiological processes through regulation of specific target genes. A key activator of… (more)

Subjects/Keywords: Regulation of Gene Transcription; Transcriptional Repressor; Bcl6; Stat5; Growth Hormone; Molecular, Cellular and Developmental Biology; Science

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APA (6th Edition):

Lin, G. (2012). A Novel Transcriptional Repressor-activator Relationship in Growth Hormone-regulated Gene Expression. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/95945

Chicago Manual of Style (16th Edition):

Lin, Grace. “A Novel Transcriptional Repressor-activator Relationship in Growth Hormone-regulated Gene Expression.” 2012. Doctoral Dissertation, University of Michigan. Accessed January 19, 2020. http://hdl.handle.net/2027.42/95945.

MLA Handbook (7th Edition):

Lin, Grace. “A Novel Transcriptional Repressor-activator Relationship in Growth Hormone-regulated Gene Expression.” 2012. Web. 19 Jan 2020.

Vancouver:

Lin G. A Novel Transcriptional Repressor-activator Relationship in Growth Hormone-regulated Gene Expression. [Internet] [Doctoral dissertation]. University of Michigan; 2012. [cited 2020 Jan 19]. Available from: http://hdl.handle.net/2027.42/95945.

Council of Science Editors:

Lin G. A Novel Transcriptional Repressor-activator Relationship in Growth Hormone-regulated Gene Expression. [Doctoral Dissertation]. University of Michigan; 2012. Available from: http://hdl.handle.net/2027.42/95945

17. Mowers, Jonathan. Mechanisms by which the IKK-related Kinases Affect Energy Expenditure.

Degree: PhD, Molecular & Integrative Physiology, 2014, University of Michigan

 Numerous studies have implicated an inflammatory link between obesity and type 2 diabetes. We applied a multidisciplinary approach spanning from in vivo animal physiology to… (more)

Subjects/Keywords: Obesity; 3T3-L1 Adipocytes; Mouse Studies; Physiology; Science

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Mowers, J. (2014). Mechanisms by which the IKK-related Kinases Affect Energy Expenditure. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/107243

Chicago Manual of Style (16th Edition):

Mowers, Jonathan. “Mechanisms by which the IKK-related Kinases Affect Energy Expenditure.” 2014. Doctoral Dissertation, University of Michigan. Accessed January 19, 2020. http://hdl.handle.net/2027.42/107243.

MLA Handbook (7th Edition):

Mowers, Jonathan. “Mechanisms by which the IKK-related Kinases Affect Energy Expenditure.” 2014. Web. 19 Jan 2020.

Vancouver:

Mowers J. Mechanisms by which the IKK-related Kinases Affect Energy Expenditure. [Internet] [Doctoral dissertation]. University of Michigan; 2014. [cited 2020 Jan 19]. Available from: http://hdl.handle.net/2027.42/107243.

Council of Science Editors:

Mowers J. Mechanisms by which the IKK-related Kinases Affect Energy Expenditure. [Doctoral Dissertation]. University of Michigan; 2014. Available from: http://hdl.handle.net/2027.42/107243

18. Wang, Guoxiao. Regulation of Adipose Tissue Function and Metabolic Homeostasis.

Degree: PhD, Cellular & Molecular Biology, 2014, University of Michigan

 Metabolic syndrome is emerging as a global epidemic that increases the risk for type 2 diabetes, cardiovascular disease, and fatty liver disease. Brown and white… (more)

Subjects/Keywords: Adipose Tissue Function and Metabolic Homeostasis; Molecular, Cellular and Developmental Biology; Physiology; Health Sciences

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APA (6th Edition):

Wang, G. (2014). Regulation of Adipose Tissue Function and Metabolic Homeostasis. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/107321

Chicago Manual of Style (16th Edition):

Wang, Guoxiao. “Regulation of Adipose Tissue Function and Metabolic Homeostasis.” 2014. Doctoral Dissertation, University of Michigan. Accessed January 19, 2020. http://hdl.handle.net/2027.42/107321.

MLA Handbook (7th Edition):

Wang, Guoxiao. “Regulation of Adipose Tissue Function and Metabolic Homeostasis.” 2014. Web. 19 Jan 2020.

Vancouver:

Wang G. Regulation of Adipose Tissue Function and Metabolic Homeostasis. [Internet] [Doctoral dissertation]. University of Michigan; 2014. [cited 2020 Jan 19]. Available from: http://hdl.handle.net/2027.42/107321.

Council of Science Editors:

Wang G. Regulation of Adipose Tissue Function and Metabolic Homeostasis. [Doctoral Dissertation]. University of Michigan; 2014. Available from: http://hdl.handle.net/2027.42/107321

19. Hu, Ming. Bayesian Modeling for High Throughput Genomic Data.

Degree: PhD, Biostatistics, 2010, University of Michigan

 The explosion of high throughput genomic data in recent years has already altered our view of the extent and complexity of biology. Technologically specific features,… (more)

Subjects/Keywords: Bayesian Modeling; High Throughput Genomic Data; MCMC; ChIP-Seq; RNA-Seq; Microarray; Genetics; Public Health; Statistics and Numeric Data; Health Sciences; Science

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APA (6th Edition):

Hu, M. (2010). Bayesian Modeling for High Throughput Genomic Data. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/78939

Chicago Manual of Style (16th Edition):

Hu, Ming. “Bayesian Modeling for High Throughput Genomic Data.” 2010. Doctoral Dissertation, University of Michigan. Accessed January 19, 2020. http://hdl.handle.net/2027.42/78939.

MLA Handbook (7th Edition):

Hu, Ming. “Bayesian Modeling for High Throughput Genomic Data.” 2010. Web. 19 Jan 2020.

Vancouver:

Hu M. Bayesian Modeling for High Throughput Genomic Data. [Internet] [Doctoral dissertation]. University of Michigan; 2010. [cited 2020 Jan 19]. Available from: http://hdl.handle.net/2027.42/78939.

Council of Science Editors:

Hu M. Bayesian Modeling for High Throughput Genomic Data. [Doctoral Dissertation]. University of Michigan; 2010. Available from: http://hdl.handle.net/2027.42/78939

20. Xiong, Tingting. Cascading Samll GTPases in Insulin Action.

Degree: PhD, Molecular and Integrative Physiology, 2012, University of Michigan

 Insulin stimulates glucose uptake into adipocytes and muscle cells by stimulating the translocation of the glucose transporter 4 Glut4 from intracellular storage vesicles to the… (more)

Subjects/Keywords: Small GTPase; Akt Signaling; Glut4 Translocation; Insulin Action; Physiology; Health Sciences

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APA (6th Edition):

Xiong, T. (2012). Cascading Samll GTPases in Insulin Action. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/93937

Chicago Manual of Style (16th Edition):

Xiong, Tingting. “Cascading Samll GTPases in Insulin Action.” 2012. Doctoral Dissertation, University of Michigan. Accessed January 19, 2020. http://hdl.handle.net/2027.42/93937.

MLA Handbook (7th Edition):

Xiong, Tingting. “Cascading Samll GTPases in Insulin Action.” 2012. Web. 19 Jan 2020.

Vancouver:

Xiong T. Cascading Samll GTPases in Insulin Action. [Internet] [Doctoral dissertation]. University of Michigan; 2012. [cited 2020 Jan 19]. Available from: http://hdl.handle.net/2027.42/93937.

Council of Science Editors:

Xiong T. Cascading Samll GTPases in Insulin Action. [Doctoral Dissertation]. University of Michigan; 2012. Available from: http://hdl.handle.net/2027.42/93937


University of Michigan

21. Zhao, Bin. Inactivation of YAP-TEAD by the Hippo Pathway is Involved in Growth Control Inactivation of YAP-TEAD by the Hippo Pathway is Involved in Growth Control and Cancer.

Degree: PhD, Biological Chemistry, 2009, University of Michigan

 The mechanism of body and organ size control is an unsolved puzzle. Recent Drosophila genetics studies established the key role of the Hippo pathway and… (more)

Subjects/Keywords: YAP; TEAD; Cancer; Biological Chemistry; Science

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Zhao, B. (2009). Inactivation of YAP-TEAD by the Hippo Pathway is Involved in Growth Control Inactivation of YAP-TEAD by the Hippo Pathway is Involved in Growth Control and Cancer. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/62289

Chicago Manual of Style (16th Edition):

Zhao, Bin. “Inactivation of YAP-TEAD by the Hippo Pathway is Involved in Growth Control Inactivation of YAP-TEAD by the Hippo Pathway is Involved in Growth Control and Cancer.” 2009. Doctoral Dissertation, University of Michigan. Accessed January 19, 2020. http://hdl.handle.net/2027.42/62289.

MLA Handbook (7th Edition):

Zhao, Bin. “Inactivation of YAP-TEAD by the Hippo Pathway is Involved in Growth Control Inactivation of YAP-TEAD by the Hippo Pathway is Involved in Growth Control and Cancer.” 2009. Web. 19 Jan 2020.

Vancouver:

Zhao B. Inactivation of YAP-TEAD by the Hippo Pathway is Involved in Growth Control Inactivation of YAP-TEAD by the Hippo Pathway is Involved in Growth Control and Cancer. [Internet] [Doctoral dissertation]. University of Michigan; 2009. [cited 2020 Jan 19]. Available from: http://hdl.handle.net/2027.42/62289.

Council of Science Editors:

Zhao B. Inactivation of YAP-TEAD by the Hippo Pathway is Involved in Growth Control Inactivation of YAP-TEAD by the Hippo Pathway is Involved in Growth Control and Cancer. [Doctoral Dissertation]. University of Michigan; 2009. Available from: http://hdl.handle.net/2027.42/62289

.