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You searched for +publisher:"University of Michigan" +contributor:("Fingar, Diane C"). Showing records 1 – 19 of 19 total matches.

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University of Michigan

1. Li, Li. Upstream Components of mTORC1.

Degree: PhD, Biological Chemistry, 2011, University of Michigan

 The target of rapamycin (TOR) is an evolutionally conserved protein kinase that belongs to the phosphoinositide-3-kinase-related protein kinase family. It resides in two distinct protein… (more)

Subjects/Keywords: Mammalian Target of Rapamycin; Small GTPases; Protein Trafficking; MARK4; Biological Chemistry; Science

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APA (6th Edition):

Li, L. (2011). Upstream Components of mTORC1. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/84473

Chicago Manual of Style (16th Edition):

Li, Li. “Upstream Components of mTORC1.” 2011. Doctoral Dissertation, University of Michigan. Accessed January 25, 2020. http://hdl.handle.net/2027.42/84473.

MLA Handbook (7th Edition):

Li, Li. “Upstream Components of mTORC1.” 2011. Web. 25 Jan 2020.

Vancouver:

Li L. Upstream Components of mTORC1. [Internet] [Doctoral dissertation]. University of Michigan; 2011. [cited 2020 Jan 25]. Available from: http://hdl.handle.net/2027.42/84473.

Council of Science Editors:

Li L. Upstream Components of mTORC1. [Doctoral Dissertation]. University of Michigan; 2011. Available from: http://hdl.handle.net/2027.42/84473


University of Michigan

2. Dupzyk, Allison. Mechanism of Membrane Penetration by Nonenveloped Polyomavirus and Papillomavirus.

Degree: PhD, Microbiology & Immunology, 2019, University of Michigan

 Membrane penetration represents a critical step during virus infection. As nonenveloped viruses lack a surrounding lipid bilayer, they are unable to penetrate host membranes by… (more)

Subjects/Keywords: Nonenveloped virus membrane penetration; Microbiology and Immunology; Science

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APA (6th Edition):

Dupzyk, A. (2019). Mechanism of Membrane Penetration by Nonenveloped Polyomavirus and Papillomavirus. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/149876

Chicago Manual of Style (16th Edition):

Dupzyk, Allison. “Mechanism of Membrane Penetration by Nonenveloped Polyomavirus and Papillomavirus.” 2019. Doctoral Dissertation, University of Michigan. Accessed January 25, 2020. http://hdl.handle.net/2027.42/149876.

MLA Handbook (7th Edition):

Dupzyk, Allison. “Mechanism of Membrane Penetration by Nonenveloped Polyomavirus and Papillomavirus.” 2019. Web. 25 Jan 2020.

Vancouver:

Dupzyk A. Mechanism of Membrane Penetration by Nonenveloped Polyomavirus and Papillomavirus. [Internet] [Doctoral dissertation]. University of Michigan; 2019. [cited 2020 Jan 25]. Available from: http://hdl.handle.net/2027.42/149876.

Council of Science Editors:

Dupzyk A. Mechanism of Membrane Penetration by Nonenveloped Polyomavirus and Papillomavirus. [Doctoral Dissertation]. University of Michigan; 2019. Available from: http://hdl.handle.net/2027.42/149876


University of Michigan

3. Bohin, Natacha. Decrypting Intestinal Mucosal Repair.

Degree: PhD, Cellular & Molecular Biology, 2019, University of Michigan

 Regeneration is a word that has inspired the imagination of artists and scientists alike ever since the word’s inception in mid-14th century from Latin meaning… (more)

Subjects/Keywords: regeneration; stem cell; intestine; niche; mouse; organoid; Genetics; Medicine (General); Molecular, Cellular and Developmental Biology; Physiology; Radiology; Science (General); Health Sciences; Science

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APA (6th Edition):

Bohin, N. (2019). Decrypting Intestinal Mucosal Repair. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/150044

Chicago Manual of Style (16th Edition):

Bohin, Natacha. “Decrypting Intestinal Mucosal Repair.” 2019. Doctoral Dissertation, University of Michigan. Accessed January 25, 2020. http://hdl.handle.net/2027.42/150044.

MLA Handbook (7th Edition):

Bohin, Natacha. “Decrypting Intestinal Mucosal Repair.” 2019. Web. 25 Jan 2020.

Vancouver:

Bohin N. Decrypting Intestinal Mucosal Repair. [Internet] [Doctoral dissertation]. University of Michigan; 2019. [cited 2020 Jan 25]. Available from: http://hdl.handle.net/2027.42/150044.

Council of Science Editors:

Bohin N. Decrypting Intestinal Mucosal Repair. [Doctoral Dissertation]. University of Michigan; 2019. Available from: http://hdl.handle.net/2027.42/150044


University of Michigan

4. Chung, Jooho. Cellular and Molecular Analysis of Notch Signaling in T Cells after Allogeneic Bone Marrow Transplantation.

Degree: PhD, Cellular & Molec Biology PhD, 2018, University of Michigan

 Allogeneic bone marrow transplantation (allo-BMT) is a potentially curative therapy for patients with cancer and hematological disorders. However, its success is limited by graft-versus-host disease… (more)

Subjects/Keywords: Notch signaling after allogeneic bone marrow transplantation; Molecular, Cellular and Developmental Biology; Science

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APA (6th Edition):

Chung, J. (2018). Cellular and Molecular Analysis of Notch Signaling in T Cells after Allogeneic Bone Marrow Transplantation. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/144004

Chicago Manual of Style (16th Edition):

Chung, Jooho. “Cellular and Molecular Analysis of Notch Signaling in T Cells after Allogeneic Bone Marrow Transplantation.” 2018. Doctoral Dissertation, University of Michigan. Accessed January 25, 2020. http://hdl.handle.net/2027.42/144004.

MLA Handbook (7th Edition):

Chung, Jooho. “Cellular and Molecular Analysis of Notch Signaling in T Cells after Allogeneic Bone Marrow Transplantation.” 2018. Web. 25 Jan 2020.

Vancouver:

Chung J. Cellular and Molecular Analysis of Notch Signaling in T Cells after Allogeneic Bone Marrow Transplantation. [Internet] [Doctoral dissertation]. University of Michigan; 2018. [cited 2020 Jan 25]. Available from: http://hdl.handle.net/2027.42/144004.

Council of Science Editors:

Chung J. Cellular and Molecular Analysis of Notch Signaling in T Cells after Allogeneic Bone Marrow Transplantation. [Doctoral Dissertation]. University of Michigan; 2018. Available from: http://hdl.handle.net/2027.42/144004


University of Michigan

5. Baldwin, Katherine Therese. Molecular Mechanisms of Immune-Mediated Axon Regeneration in the Injured Central Nervous System.

Degree: PhD, Cellular and Molecular Biology, 2015, University of Michigan

 In the injured adult mammalian central nervous system (CNS), severed axons fail to undergo spontaneous regeneration, leading to permanent neurological deficits, such as paralysis following… (more)

Subjects/Keywords: central nervous system regeneration; neuroinflammation; Neurosciences; Health Sciences

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APA (6th Edition):

Baldwin, K. T. (2015). Molecular Mechanisms of Immune-Mediated Axon Regeneration in the Injured Central Nervous System. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/113473

Chicago Manual of Style (16th Edition):

Baldwin, Katherine Therese. “Molecular Mechanisms of Immune-Mediated Axon Regeneration in the Injured Central Nervous System.” 2015. Doctoral Dissertation, University of Michigan. Accessed January 25, 2020. http://hdl.handle.net/2027.42/113473.

MLA Handbook (7th Edition):

Baldwin, Katherine Therese. “Molecular Mechanisms of Immune-Mediated Axon Regeneration in the Injured Central Nervous System.” 2015. Web. 25 Jan 2020.

Vancouver:

Baldwin KT. Molecular Mechanisms of Immune-Mediated Axon Regeneration in the Injured Central Nervous System. [Internet] [Doctoral dissertation]. University of Michigan; 2015. [cited 2020 Jan 25]. Available from: http://hdl.handle.net/2027.42/113473.

Council of Science Editors:

Baldwin KT. Molecular Mechanisms of Immune-Mediated Axon Regeneration in the Injured Central Nervous System. [Doctoral Dissertation]. University of Michigan; 2015. Available from: http://hdl.handle.net/2027.42/113473


University of Michigan

6. Chang, Lou. Insights from Murine Models of Neurofibromatosis Type I: The Etiology and Appropriate Therapeutic Windows for Peripheral Nerve Sheath Tumors.

Degree: PhD, Cell and Developmental Biology, 2011, University of Michigan

 Neurofibromatosis Type I (NF1) is a common autosomal dominant disorder that afflicts approximately one in 3,500 live births. Individuals afflicted with NF1 are susceptible to… (more)

Subjects/Keywords: Cancer; Schwann Cell; Cell-of-Origin; Intervention; Pre-clinical; NF1; Molecular, Cellular and Developmental Biology; Health Sciences

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APA (6th Edition):

Chang, L. (2011). Insights from Murine Models of Neurofibromatosis Type I: The Etiology and Appropriate Therapeutic Windows for Peripheral Nerve Sheath Tumors. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/84543

Chicago Manual of Style (16th Edition):

Chang, Lou. “Insights from Murine Models of Neurofibromatosis Type I: The Etiology and Appropriate Therapeutic Windows for Peripheral Nerve Sheath Tumors.” 2011. Doctoral Dissertation, University of Michigan. Accessed January 25, 2020. http://hdl.handle.net/2027.42/84543.

MLA Handbook (7th Edition):

Chang, Lou. “Insights from Murine Models of Neurofibromatosis Type I: The Etiology and Appropriate Therapeutic Windows for Peripheral Nerve Sheath Tumors.” 2011. Web. 25 Jan 2020.

Vancouver:

Chang L. Insights from Murine Models of Neurofibromatosis Type I: The Etiology and Appropriate Therapeutic Windows for Peripheral Nerve Sheath Tumors. [Internet] [Doctoral dissertation]. University of Michigan; 2011. [cited 2020 Jan 25]. Available from: http://hdl.handle.net/2027.42/84543.

Council of Science Editors:

Chang L. Insights from Murine Models of Neurofibromatosis Type I: The Etiology and Appropriate Therapeutic Windows for Peripheral Nerve Sheath Tumors. [Doctoral Dissertation]. University of Michigan; 2011. Available from: http://hdl.handle.net/2027.42/84543


University of Michigan

7. Villanueva, Eneida Cristina. Unraveling Novel Leptin-Dependent Signaling Pathways in the Basomedial Hypothalamus.

Degree: PhD, Molecular and Integrative Physiology, 2010, University of Michigan

 Leptin is secreted by the adipose tissue in proportion to fat stores and acts on the hypothalamus to inhibit appetite and promote energy expenditure. Leptin… (more)

Subjects/Keywords: Leptin Signaling Pathways in the Hypothalamus; Physiology; Health Sciences; Science

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APA (6th Edition):

Villanueva, E. C. (2010). Unraveling Novel Leptin-Dependent Signaling Pathways in the Basomedial Hypothalamus. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/75887

Chicago Manual of Style (16th Edition):

Villanueva, Eneida Cristina. “Unraveling Novel Leptin-Dependent Signaling Pathways in the Basomedial Hypothalamus.” 2010. Doctoral Dissertation, University of Michigan. Accessed January 25, 2020. http://hdl.handle.net/2027.42/75887.

MLA Handbook (7th Edition):

Villanueva, Eneida Cristina. “Unraveling Novel Leptin-Dependent Signaling Pathways in the Basomedial Hypothalamus.” 2010. Web. 25 Jan 2020.

Vancouver:

Villanueva EC. Unraveling Novel Leptin-Dependent Signaling Pathways in the Basomedial Hypothalamus. [Internet] [Doctoral dissertation]. University of Michigan; 2010. [cited 2020 Jan 25]. Available from: http://hdl.handle.net/2027.42/75887.

Council of Science Editors:

Villanueva EC. Unraveling Novel Leptin-Dependent Signaling Pathways in the Basomedial Hypothalamus. [Doctoral Dissertation]. University of Michigan; 2010. Available from: http://hdl.handle.net/2027.42/75887

8. Magnuson, Brian Richard. Regulation of the Mechanistic Target of Rapamycin by Cellular Stress.

Degree: PhD, Cell and Developmental Biology, 2013, University of Michigan

 In complex eukaryotes, cell, tissue, and organismal homeostasis requires proper sensing of growth factors and nutrients. The mechanistic target of rapamycin (mTOR) functions as a… (more)

Subjects/Keywords: Signal Transduction; Kinase; Molecular, Cellular and Developmental Biology; Health Sciences

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APA (6th Edition):

Magnuson, B. R. (2013). Regulation of the Mechanistic Target of Rapamycin by Cellular Stress. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/97910

Chicago Manual of Style (16th Edition):

Magnuson, Brian Richard. “Regulation of the Mechanistic Target of Rapamycin by Cellular Stress.” 2013. Doctoral Dissertation, University of Michigan. Accessed January 25, 2020. http://hdl.handle.net/2027.42/97910.

MLA Handbook (7th Edition):

Magnuson, Brian Richard. “Regulation of the Mechanistic Target of Rapamycin by Cellular Stress.” 2013. Web. 25 Jan 2020.

Vancouver:

Magnuson BR. Regulation of the Mechanistic Target of Rapamycin by Cellular Stress. [Internet] [Doctoral dissertation]. University of Michigan; 2013. [cited 2020 Jan 25]. Available from: http://hdl.handle.net/2027.42/97910.

Council of Science Editors:

Magnuson BR. Regulation of the Mechanistic Target of Rapamycin by Cellular Stress. [Doctoral Dissertation]. University of Michigan; 2013. Available from: http://hdl.handle.net/2027.42/97910

9. Dezsi, Kaleena Bernardi. Derlin-1 and the E3 Ubiquitin Ligases Hrd1 and gp78 Facilitate Cholera Toxin Retro-translocation.

Degree: PhD, Cell and Developmental Biology, 2009, University of Michigan

 The endoplasmic reticulum (ER) is the site of folding and assembly for membrane and secretory proteins. When proteins fail to reach their proper conformations, the… (more)

Subjects/Keywords: Cholera Toxin; ERAD; ER; Derlin; Ubiquitin; Ligase; Molecular, Cellular and Developmental Biology; Science

…construct and the monoclonal antibody against HA were gifts from K. Verhey (University of… …Michigan). The CFTR construct was a gift from R. Frizzell (University of Pittsburgh… 

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APA (6th Edition):

Dezsi, K. B. (2009). Derlin-1 and the E3 Ubiquitin Ligases Hrd1 and gp78 Facilitate Cholera Toxin Retro-translocation. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/64597

Chicago Manual of Style (16th Edition):

Dezsi, Kaleena Bernardi. “Derlin-1 and the E3 Ubiquitin Ligases Hrd1 and gp78 Facilitate Cholera Toxin Retro-translocation.” 2009. Doctoral Dissertation, University of Michigan. Accessed January 25, 2020. http://hdl.handle.net/2027.42/64597.

MLA Handbook (7th Edition):

Dezsi, Kaleena Bernardi. “Derlin-1 and the E3 Ubiquitin Ligases Hrd1 and gp78 Facilitate Cholera Toxin Retro-translocation.” 2009. Web. 25 Jan 2020.

Vancouver:

Dezsi KB. Derlin-1 and the E3 Ubiquitin Ligases Hrd1 and gp78 Facilitate Cholera Toxin Retro-translocation. [Internet] [Doctoral dissertation]. University of Michigan; 2009. [cited 2020 Jan 25]. Available from: http://hdl.handle.net/2027.42/64597.

Council of Science Editors:

Dezsi KB. Derlin-1 and the E3 Ubiquitin Ligases Hrd1 and gp78 Facilitate Cholera Toxin Retro-translocation. [Doctoral Dissertation]. University of Michigan; 2009. Available from: http://hdl.handle.net/2027.42/64597

10. Lee, Jae Young. The Regulation of Signaling in Hematopietic Stem Cell Maintenance.

Degree: PhD, Cell and Developmental Biology, 2012, University of Michigan

 Hematopoietic stem cells (HSCs) maintain themselves throughout life by undergoing self-renewing divisions and by differentiating to generate all the blood and immune system cells in… (more)

Subjects/Keywords: Pten; HSC; Leukemia; Ink4a/Arf; P53; Reactive Oxygen Species; Molecular, Cellular and Developmental Biology; Health Sciences

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APA (6th Edition):

Lee, J. Y. (2012). The Regulation of Signaling in Hematopietic Stem Cell Maintenance. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/91538

Chicago Manual of Style (16th Edition):

Lee, Jae Young. “The Regulation of Signaling in Hematopietic Stem Cell Maintenance.” 2012. Doctoral Dissertation, University of Michigan. Accessed January 25, 2020. http://hdl.handle.net/2027.42/91538.

MLA Handbook (7th Edition):

Lee, Jae Young. “The Regulation of Signaling in Hematopietic Stem Cell Maintenance.” 2012. Web. 25 Jan 2020.

Vancouver:

Lee JY. The Regulation of Signaling in Hematopietic Stem Cell Maintenance. [Internet] [Doctoral dissertation]. University of Michigan; 2012. [cited 2020 Jan 25]. Available from: http://hdl.handle.net/2027.42/91538.

Council of Science Editors:

Lee JY. The Regulation of Signaling in Hematopietic Stem Cell Maintenance. [Doctoral Dissertation]. University of Michigan; 2012. Available from: http://hdl.handle.net/2027.42/91538

11. Cunningham, Corey. Cells Deploy a Two-Pronged Quality Control Strategy to Degrade Misfolded Proinsulin Mutants.

Degree: PhD, Cellular & Molecular Biology, 2019, University of Michigan

 There are over 30 missense mutations found in the human insulin gene responsible for a newly-characterized diabetic syndrome called Mutant INS-gene-induced Diabetes of Youth (MIDY).… (more)

Subjects/Keywords: Protein Quality Control; Endoplasmic Reticulum; Mutant INS-gene-induced Diabetes of Youth; ER-associated degradation; ER-autophagy; Biological Chemistry; Molecular, Cellular and Developmental Biology; Science (General); Science

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APA (6th Edition):

Cunningham, C. (2019). Cells Deploy a Two-Pronged Quality Control Strategy to Degrade Misfolded Proinsulin Mutants. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/149827

Chicago Manual of Style (16th Edition):

Cunningham, Corey. “Cells Deploy a Two-Pronged Quality Control Strategy to Degrade Misfolded Proinsulin Mutants.” 2019. Doctoral Dissertation, University of Michigan. Accessed January 25, 2020. http://hdl.handle.net/2027.42/149827.

MLA Handbook (7th Edition):

Cunningham, Corey. “Cells Deploy a Two-Pronged Quality Control Strategy to Degrade Misfolded Proinsulin Mutants.” 2019. Web. 25 Jan 2020.

Vancouver:

Cunningham C. Cells Deploy a Two-Pronged Quality Control Strategy to Degrade Misfolded Proinsulin Mutants. [Internet] [Doctoral dissertation]. University of Michigan; 2019. [cited 2020 Jan 25]. Available from: http://hdl.handle.net/2027.42/149827.

Council of Science Editors:

Cunningham C. Cells Deploy a Two-Pronged Quality Control Strategy to Degrade Misfolded Proinsulin Mutants. [Doctoral Dissertation]. University of Michigan; 2019. Available from: http://hdl.handle.net/2027.42/149827

12. Ma, Di. Autophagy: Circadian Regulation and Role in Non-Alcoholic Fatty Liver Disease.

Degree: PhD, Cell and Developmental Biology, 2013, University of Michigan

 Metabolic syndrome has become a global health care challenge characterized by increased risk for type 2 diabetes, cardiovascular disease, and non-alcoholic fatty liver disease. Recent… (more)

Subjects/Keywords: Autophagy; Circadian Clock; Nonalcoholic Steatohepatitis; Molecular, Cellular and Developmental Biology; Science

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APA (6th Edition):

Ma, D. (2013). Autophagy: Circadian Regulation and Role in Non-Alcoholic Fatty Liver Disease. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/98023

Chicago Manual of Style (16th Edition):

Ma, Di. “Autophagy: Circadian Regulation and Role in Non-Alcoholic Fatty Liver Disease.” 2013. Doctoral Dissertation, University of Michigan. Accessed January 25, 2020. http://hdl.handle.net/2027.42/98023.

MLA Handbook (7th Edition):

Ma, Di. “Autophagy: Circadian Regulation and Role in Non-Alcoholic Fatty Liver Disease.” 2013. Web. 25 Jan 2020.

Vancouver:

Ma D. Autophagy: Circadian Regulation and Role in Non-Alcoholic Fatty Liver Disease. [Internet] [Doctoral dissertation]. University of Michigan; 2013. [cited 2020 Jan 25]. Available from: http://hdl.handle.net/2027.42/98023.

Council of Science Editors:

Ma D. Autophagy: Circadian Regulation and Role in Non-Alcoholic Fatty Liver Disease. [Doctoral Dissertation]. University of Michigan; 2013. Available from: http://hdl.handle.net/2027.42/98023

13. Akgul, Seckin. The Functions of Rictor, Pten and p53 Signaling Pathways in Brain Tumors and Brain Development.

Degree: PhD, Cellular and Molecular Biology, 2015, University of Michigan

 Glioblastoma is the most common malignant neoplasm of the central nervous system. Despite intense treatment plans and supportive care, the median survival rate remains approximately… (more)

Subjects/Keywords: Adult and pediatric brain tumors; Glioblastoma; Medulloblastoma; Rictor/mTORC2; RTK/PI3K/AKT signaling pathway; TP53/p53 signaling pathway; Molecular, Cellular and Developmental Biology; Science

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APA (6th Edition):

Akgul, S. (2015). The Functions of Rictor, Pten and p53 Signaling Pathways in Brain Tumors and Brain Development. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/113298

Chicago Manual of Style (16th Edition):

Akgul, Seckin. “The Functions of Rictor, Pten and p53 Signaling Pathways in Brain Tumors and Brain Development.” 2015. Doctoral Dissertation, University of Michigan. Accessed January 25, 2020. http://hdl.handle.net/2027.42/113298.

MLA Handbook (7th Edition):

Akgul, Seckin. “The Functions of Rictor, Pten and p53 Signaling Pathways in Brain Tumors and Brain Development.” 2015. Web. 25 Jan 2020.

Vancouver:

Akgul S. The Functions of Rictor, Pten and p53 Signaling Pathways in Brain Tumors and Brain Development. [Internet] [Doctoral dissertation]. University of Michigan; 2015. [cited 2020 Jan 25]. Available from: http://hdl.handle.net/2027.42/113298.

Council of Science Editors:

Akgul S. The Functions of Rictor, Pten and p53 Signaling Pathways in Brain Tumors and Brain Development. [Doctoral Dissertation]. University of Michigan; 2015. Available from: http://hdl.handle.net/2027.42/113298

14. Dumas, Kathleen Johanna. Characterization of Novel Regulators of FoxO Transcription Factors.

Degree: PhD, Cellular & Molecular Biology, 2013, University of Michigan

 FoxO transcription factors were first implicated as regulators of longevity in C. elegans. Reducing insulin/insulin-like growth factor signaling (ILS) extends C. elegans life span in… (more)

Subjects/Keywords: FoxO; Biology of Aging; Caenorhabditis Elegans; Genetics; Molecular, Cellular and Developmental Biology; Science

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APA (6th Edition):

Dumas, K. J. (2013). Characterization of Novel Regulators of FoxO Transcription Factors. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/97969

Chicago Manual of Style (16th Edition):

Dumas, Kathleen Johanna. “Characterization of Novel Regulators of FoxO Transcription Factors.” 2013. Doctoral Dissertation, University of Michigan. Accessed January 25, 2020. http://hdl.handle.net/2027.42/97969.

MLA Handbook (7th Edition):

Dumas, Kathleen Johanna. “Characterization of Novel Regulators of FoxO Transcription Factors.” 2013. Web. 25 Jan 2020.

Vancouver:

Dumas KJ. Characterization of Novel Regulators of FoxO Transcription Factors. [Internet] [Doctoral dissertation]. University of Michigan; 2013. [cited 2020 Jan 25]. Available from: http://hdl.handle.net/2027.42/97969.

Council of Science Editors:

Dumas KJ. Characterization of Novel Regulators of FoxO Transcription Factors. [Doctoral Dissertation]. University of Michigan; 2013. Available from: http://hdl.handle.net/2027.42/97969

15. Henry, Fredrick E. Novel Roles for mTORC1-dependent Translational Control during Synaptic Homeostasis.

Degree: PhD, Neuroscience, 2014, University of Michigan

 The mechanistic target of rapamycin complex 1 (mTORC1), a kinase involved in regulating translation initiation, has recently emerged as a critical player responsible for orchestrating… (more)

Subjects/Keywords: Neuroscience; Plasticity; Synapse; MTOR; Autism; Physiology; Science

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APA (6th Edition):

Henry, F. E. (2014). Novel Roles for mTORC1-dependent Translational Control during Synaptic Homeostasis. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/107207

Chicago Manual of Style (16th Edition):

Henry, Fredrick E. “Novel Roles for mTORC1-dependent Translational Control during Synaptic Homeostasis.” 2014. Doctoral Dissertation, University of Michigan. Accessed January 25, 2020. http://hdl.handle.net/2027.42/107207.

MLA Handbook (7th Edition):

Henry, Fredrick E. “Novel Roles for mTORC1-dependent Translational Control during Synaptic Homeostasis.” 2014. Web. 25 Jan 2020.

Vancouver:

Henry FE. Novel Roles for mTORC1-dependent Translational Control during Synaptic Homeostasis. [Internet] [Doctoral dissertation]. University of Michigan; 2014. [cited 2020 Jan 25]. Available from: http://hdl.handle.net/2027.42/107207.

Council of Science Editors:

Henry FE. Novel Roles for mTORC1-dependent Translational Control during Synaptic Homeostasis. [Doctoral Dissertation]. University of Michigan; 2014. Available from: http://hdl.handle.net/2027.42/107207

16. Waghray, Meghna. Cell Specific Regulation of Sonic Hedgehog in Adult Stomach: Understanding Mechanisms Leading to Gastric Atrophy/Metaplasia. Mechanisms Leading to Gastric Atrophy/Metaplasia.

Degree: PhD, Cell and Developmental Biology, 2009, University of Michigan

 Helicobacter induced gastritis of the corpus results in loss of parietal cell function, loss of the oxyntic gland (atrophy), changes that predispose to gastric cancer.… (more)

Subjects/Keywords: Helicobacter Induced IL-1b Regulation of Sonic Hedgehog; Health Sciences; Science

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APA (6th Edition):

Waghray, M. (2009). Cell Specific Regulation of Sonic Hedgehog in Adult Stomach: Understanding Mechanisms Leading to Gastric Atrophy/Metaplasia. Mechanisms Leading to Gastric Atrophy/Metaplasia. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/62276

Chicago Manual of Style (16th Edition):

Waghray, Meghna. “Cell Specific Regulation of Sonic Hedgehog in Adult Stomach: Understanding Mechanisms Leading to Gastric Atrophy/Metaplasia. Mechanisms Leading to Gastric Atrophy/Metaplasia.” 2009. Doctoral Dissertation, University of Michigan. Accessed January 25, 2020. http://hdl.handle.net/2027.42/62276.

MLA Handbook (7th Edition):

Waghray, Meghna. “Cell Specific Regulation of Sonic Hedgehog in Adult Stomach: Understanding Mechanisms Leading to Gastric Atrophy/Metaplasia. Mechanisms Leading to Gastric Atrophy/Metaplasia.” 2009. Web. 25 Jan 2020.

Vancouver:

Waghray M. Cell Specific Regulation of Sonic Hedgehog in Adult Stomach: Understanding Mechanisms Leading to Gastric Atrophy/Metaplasia. Mechanisms Leading to Gastric Atrophy/Metaplasia. [Internet] [Doctoral dissertation]. University of Michigan; 2009. [cited 2020 Jan 25]. Available from: http://hdl.handle.net/2027.42/62276.

Council of Science Editors:

Waghray M. Cell Specific Regulation of Sonic Hedgehog in Adult Stomach: Understanding Mechanisms Leading to Gastric Atrophy/Metaplasia. Mechanisms Leading to Gastric Atrophy/Metaplasia. [Doctoral Dissertation]. University of Michigan; 2009. Available from: http://hdl.handle.net/2027.42/62276

17. Buller, Carolyn L. Role of GLUT1 in the Mammalian Target of Rapamycin Pathway: Mechanisms of Regulation.

Degree: PhD, Molecular and Integrative Physiology, 2010, University of Michigan

 GLUT1-mediated glucose transport is a highly regulated process and is dependent on a variety of signaling events. The glycogen synthase kinase (GSK)-3/tuberous sclerosis(TSC)/mammalian target of… (more)

Subjects/Keywords: GLUT1 and MTORC1; Molecular, Cellular and Developmental Biology; Physiology; Health Sciences

University of Michigan). The anti β-tubulin antibody was obtained from Upstate Biotechnology… 

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Buller, C. L. (2010). Role of GLUT1 in the Mammalian Target of Rapamycin Pathway: Mechanisms of Regulation. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/77734

Chicago Manual of Style (16th Edition):

Buller, Carolyn L. “Role of GLUT1 in the Mammalian Target of Rapamycin Pathway: Mechanisms of Regulation.” 2010. Doctoral Dissertation, University of Michigan. Accessed January 25, 2020. http://hdl.handle.net/2027.42/77734.

MLA Handbook (7th Edition):

Buller, Carolyn L. “Role of GLUT1 in the Mammalian Target of Rapamycin Pathway: Mechanisms of Regulation.” 2010. Web. 25 Jan 2020.

Vancouver:

Buller CL. Role of GLUT1 in the Mammalian Target of Rapamycin Pathway: Mechanisms of Regulation. [Internet] [Doctoral dissertation]. University of Michigan; 2010. [cited 2020 Jan 25]. Available from: http://hdl.handle.net/2027.42/77734.

Council of Science Editors:

Buller CL. Role of GLUT1 in the Mammalian Target of Rapamycin Pathway: Mechanisms of Regulation. [Doctoral Dissertation]. University of Michigan; 2010. Available from: http://hdl.handle.net/2027.42/77734


University of Michigan

18. Hammond, Jennetta Watson. Kinesin Regulation: Discovering the Mechanisms that Mediate Autoinhibition, Cargo Complex Formation, and Selective Microtubule Use in Neurons.

Degree: PhD, Cell and Developmental Biology, 2008, University of Michigan

 Kinesin motors play an indispensable role in intracellular trafficking by driving the long-distance transport of protein and vesicular cargoes along microtubules. My thesis work has… (more)

Subjects/Keywords: Kinesin; Protein Trafficking; Molecular Motors; KIF1A; Microtubule Post-translational Modifications; KIF17; Molecular, Cellular and Developmental Biology; Science

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Hammond, J. W. (2008). Kinesin Regulation: Discovering the Mechanisms that Mediate Autoinhibition, Cargo Complex Formation, and Selective Microtubule Use in Neurons. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/61604

Chicago Manual of Style (16th Edition):

Hammond, Jennetta Watson. “Kinesin Regulation: Discovering the Mechanisms that Mediate Autoinhibition, Cargo Complex Formation, and Selective Microtubule Use in Neurons.” 2008. Doctoral Dissertation, University of Michigan. Accessed January 25, 2020. http://hdl.handle.net/2027.42/61604.

MLA Handbook (7th Edition):

Hammond, Jennetta Watson. “Kinesin Regulation: Discovering the Mechanisms that Mediate Autoinhibition, Cargo Complex Formation, and Selective Microtubule Use in Neurons.” 2008. Web. 25 Jan 2020.

Vancouver:

Hammond JW. Kinesin Regulation: Discovering the Mechanisms that Mediate Autoinhibition, Cargo Complex Formation, and Selective Microtubule Use in Neurons. [Internet] [Doctoral dissertation]. University of Michigan; 2008. [cited 2020 Jan 25]. Available from: http://hdl.handle.net/2027.42/61604.

Council of Science Editors:

Hammond JW. Kinesin Regulation: Discovering the Mechanisms that Mediate Autoinhibition, Cargo Complex Formation, and Selective Microtubule Use in Neurons. [Doctoral Dissertation]. University of Michigan; 2008. Available from: http://hdl.handle.net/2027.42/61604


University of Michigan

19. Ekim Ustunel, Bilgen. Novel Regulation of mTOR Complex 1 Signaling by Site-Specific mTOR Phosphorylation.

Degree: PhD, Cell and Developmental Biology, 2012, University of Michigan

Subjects/Keywords: MTOR; Molecular, Cellular and Developmental Biology; Science

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Ekim Ustunel, B. (2012). Novel Regulation of mTOR Complex 1 Signaling by Site-Specific mTOR Phosphorylation. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/94087

Chicago Manual of Style (16th Edition):

Ekim Ustunel, Bilgen. “Novel Regulation of mTOR Complex 1 Signaling by Site-Specific mTOR Phosphorylation.” 2012. Doctoral Dissertation, University of Michigan. Accessed January 25, 2020. http://hdl.handle.net/2027.42/94087.

MLA Handbook (7th Edition):

Ekim Ustunel, Bilgen. “Novel Regulation of mTOR Complex 1 Signaling by Site-Specific mTOR Phosphorylation.” 2012. Web. 25 Jan 2020.

Vancouver:

Ekim Ustunel B. Novel Regulation of mTOR Complex 1 Signaling by Site-Specific mTOR Phosphorylation. [Internet] [Doctoral dissertation]. University of Michigan; 2012. [cited 2020 Jan 25]. Available from: http://hdl.handle.net/2027.42/94087.

Council of Science Editors:

Ekim Ustunel B. Novel Regulation of mTOR Complex 1 Signaling by Site-Specific mTOR Phosphorylation. [Doctoral Dissertation]. University of Michigan; 2012. Available from: http://hdl.handle.net/2027.42/94087

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