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You searched for +publisher:"University of Michigan" +contributor:("Fearon, Eric R"). Showing records 1 – 30 of 40 total matches.

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1. Billi, Allison Chelsa. Biogenesis and Stability of Germline Small RNAs in C. elegans.

Degree: PhD, Human Genetics, 2015, University of Michigan

 Across the animal kingdom, small, noncoding RNAs preserve and promote fertility by engaging Argonaute effector proteins to silence deleterious genetic elements. Generated in germline and… (more)

Subjects/Keywords: Small RNAs; piRNAs; Endo-siRNAs; Caenorhabditis elegans; Germline; Epigenetics; Genetics; Science

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APA (6th Edition):

Billi, A. C. (2015). Biogenesis and Stability of Germline Small RNAs in C. elegans. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/111471

Chicago Manual of Style (16th Edition):

Billi, Allison Chelsa. “Biogenesis and Stability of Germline Small RNAs in C. elegans.” 2015. Doctoral Dissertation, University of Michigan. Accessed January 29, 2020. http://hdl.handle.net/2027.42/111471.

MLA Handbook (7th Edition):

Billi, Allison Chelsa. “Biogenesis and Stability of Germline Small RNAs in C. elegans.” 2015. Web. 29 Jan 2020.

Vancouver:

Billi AC. Biogenesis and Stability of Germline Small RNAs in C. elegans. [Internet] [Doctoral dissertation]. University of Michigan; 2015. [cited 2020 Jan 29]. Available from: http://hdl.handle.net/2027.42/111471.

Council of Science Editors:

Billi AC. Biogenesis and Stability of Germline Small RNAs in C. elegans. [Doctoral Dissertation]. University of Michigan; 2015. Available from: http://hdl.handle.net/2027.42/111471


University of Michigan

2. Gornick, Michele Caroline. Quantitative Approaches to Understanding Cancer Genomes.

Degree: PhD, Human Genetics, 2011, University of Michigan

 Recent advances in technology have enabled the systematic, genome-wide analysis of cancer genomes, providing greater insight into the genetic basis of cancer development and a… (more)

Subjects/Keywords: Cancer Genetics; Next-generation Sequencing; Colorectal Cancer; Breast Cancer; Genome; Genetics; Health Sciences

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APA (6th Edition):

Gornick, M. C. (2011). Quantitative Approaches to Understanding Cancer Genomes. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/89716

Chicago Manual of Style (16th Edition):

Gornick, Michele Caroline. “Quantitative Approaches to Understanding Cancer Genomes.” 2011. Doctoral Dissertation, University of Michigan. Accessed January 29, 2020. http://hdl.handle.net/2027.42/89716.

MLA Handbook (7th Edition):

Gornick, Michele Caroline. “Quantitative Approaches to Understanding Cancer Genomes.” 2011. Web. 29 Jan 2020.

Vancouver:

Gornick MC. Quantitative Approaches to Understanding Cancer Genomes. [Internet] [Doctoral dissertation]. University of Michigan; 2011. [cited 2020 Jan 29]. Available from: http://hdl.handle.net/2027.42/89716.

Council of Science Editors:

Gornick MC. Quantitative Approaches to Understanding Cancer Genomes. [Doctoral Dissertation]. University of Michigan; 2011. Available from: http://hdl.handle.net/2027.42/89716

3. Chin, Alana. Elucidating a novel WNT/?-CATENIN Signaling-Independent Environment that Precedes Villus Morphogenesis in the Embryonic Intestine.

Degree: PhD, Cell and Developmental Biology, 2017, University of Michigan

 The intestine is a vital organ responsible for several functions, including excretion of waste, acting as a major site of host immunity, and most importantly,… (more)

Subjects/Keywords: intestinal development; villus morphogenesis; cell signaling; Wnt/beta-catenin signaling pathway; Molecular, Cellular and Developmental Biology; Health Sciences

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APA (6th Edition):

Chin, A. (2017). Elucidating a novel WNT/?-CATENIN Signaling-Independent Environment that Precedes Villus Morphogenesis in the Embryonic Intestine. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/136965

Chicago Manual of Style (16th Edition):

Chin, Alana. “Elucidating a novel WNT/?-CATENIN Signaling-Independent Environment that Precedes Villus Morphogenesis in the Embryonic Intestine.” 2017. Doctoral Dissertation, University of Michigan. Accessed January 29, 2020. http://hdl.handle.net/2027.42/136965.

MLA Handbook (7th Edition):

Chin, Alana. “Elucidating a novel WNT/?-CATENIN Signaling-Independent Environment that Precedes Villus Morphogenesis in the Embryonic Intestine.” 2017. Web. 29 Jan 2020.

Vancouver:

Chin A. Elucidating a novel WNT/?-CATENIN Signaling-Independent Environment that Precedes Villus Morphogenesis in the Embryonic Intestine. [Internet] [Doctoral dissertation]. University of Michigan; 2017. [cited 2020 Jan 29]. Available from: http://hdl.handle.net/2027.42/136965.

Council of Science Editors:

Chin A. Elucidating a novel WNT/?-CATENIN Signaling-Independent Environment that Precedes Villus Morphogenesis in the Embryonic Intestine. [Doctoral Dissertation]. University of Michigan; 2017. Available from: http://hdl.handle.net/2027.42/136965


University of Michigan

4. Kennaley, Kelly. Identification and Characterization of TPRKB Dependency in TP53 Deficient Cancers.

Degree: PhD, Molecular & Cellular Pathology, 2019, University of Michigan

 Tumor protein 53 (TP53) is a transcription factor involved in regulating various facets of cellular functionality from its canonical functions in DNA damage response, cell… (more)

Subjects/Keywords: TPRKB; TP53; EKC/KEOPS; Targeted Cancer Therapy; Genetics; Molecular, Cellular and Developmental Biology; Science (General); Science

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APA (6th Edition):

Kennaley, K. (2019). Identification and Characterization of TPRKB Dependency in TP53 Deficient Cancers. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/151505

Chicago Manual of Style (16th Edition):

Kennaley, Kelly. “Identification and Characterization of TPRKB Dependency in TP53 Deficient Cancers.” 2019. Doctoral Dissertation, University of Michigan. Accessed January 29, 2020. http://hdl.handle.net/2027.42/151505.

MLA Handbook (7th Edition):

Kennaley, Kelly. “Identification and Characterization of TPRKB Dependency in TP53 Deficient Cancers.” 2019. Web. 29 Jan 2020.

Vancouver:

Kennaley K. Identification and Characterization of TPRKB Dependency in TP53 Deficient Cancers. [Internet] [Doctoral dissertation]. University of Michigan; 2019. [cited 2020 Jan 29]. Available from: http://hdl.handle.net/2027.42/151505.

Council of Science Editors:

Kennaley K. Identification and Characterization of TPRKB Dependency in TP53 Deficient Cancers. [Doctoral Dissertation]. University of Michigan; 2019. Available from: http://hdl.handle.net/2027.42/151505


University of Michigan

5. Duan, Xiyu. MEMS-Based Endomicroscopes for High Resolution in vivo Imaging.

Degree: PhD, Biomedical Engineering, 2017, University of Michigan

 Intravital microscopy is an emerging methodology for performing real time imaging in live animals. This technology is playing a greater role in the study of… (more)

Subjects/Keywords: endomicroscope; endoscopy; MEMS; in vivo; colorectal cancer; Biomedical Engineering; Engineering

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APA (6th Edition):

Duan, X. (2017). MEMS-Based Endomicroscopes for High Resolution in vivo Imaging. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/136954

Chicago Manual of Style (16th Edition):

Duan, Xiyu. “MEMS-Based Endomicroscopes for High Resolution in vivo Imaging.” 2017. Doctoral Dissertation, University of Michigan. Accessed January 29, 2020. http://hdl.handle.net/2027.42/136954.

MLA Handbook (7th Edition):

Duan, Xiyu. “MEMS-Based Endomicroscopes for High Resolution in vivo Imaging.” 2017. Web. 29 Jan 2020.

Vancouver:

Duan X. MEMS-Based Endomicroscopes for High Resolution in vivo Imaging. [Internet] [Doctoral dissertation]. University of Michigan; 2017. [cited 2020 Jan 29]. Available from: http://hdl.handle.net/2027.42/136954.

Council of Science Editors:

Duan X. MEMS-Based Endomicroscopes for High Resolution in vivo Imaging. [Doctoral Dissertation]. University of Michigan; 2017. Available from: http://hdl.handle.net/2027.42/136954

6. Daigneault, Jillian. The Role of NELF in Mediating Human Gene Expression.

Degree: PhD, Cancer Biology, 2017, University of Michigan

 Transcription is the synthesis of RNA from a DNA template. Transcription has been shown to occur in three main steps, initiation, elongation and termination. Additional… (more)

Subjects/Keywords: RNA Pol II Pausing; NELF; Genetics; Health Sciences

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APA (6th Edition):

Daigneault, J. (2017). The Role of NELF in Mediating Human Gene Expression. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/138582

Chicago Manual of Style (16th Edition):

Daigneault, Jillian. “The Role of NELF in Mediating Human Gene Expression.” 2017. Doctoral Dissertation, University of Michigan. Accessed January 29, 2020. http://hdl.handle.net/2027.42/138582.

MLA Handbook (7th Edition):

Daigneault, Jillian. “The Role of NELF in Mediating Human Gene Expression.” 2017. Web. 29 Jan 2020.

Vancouver:

Daigneault J. The Role of NELF in Mediating Human Gene Expression. [Internet] [Doctoral dissertation]. University of Michigan; 2017. [cited 2020 Jan 29]. Available from: http://hdl.handle.net/2027.42/138582.

Council of Science Editors:

Daigneault J. The Role of NELF in Mediating Human Gene Expression. [Doctoral Dissertation]. University of Michigan; 2017. Available from: http://hdl.handle.net/2027.42/138582


University of Michigan

7. Pedersen Schuler, Elisabeth. The Function of Wnt/beta-catenin Signaling in Ewing Sarcoma and its Contribution to Pathogenesis.

Degree: PhD, Molecular & Cellular Path PhD, 2018, University of Michigan

 Ewing sarcoma is an aggressive bone and soft tissue tumor with a high propensity for metastasis; however, the mechanisms that contribute to this process are… (more)

Subjects/Keywords: The role of Wnt/beta-catenin signaling in Ewing sarcoma; Pathology; Health Sciences

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APA (6th Edition):

Pedersen Schuler, E. (2018). The Function of Wnt/beta-catenin Signaling in Ewing Sarcoma and its Contribution to Pathogenesis. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/144020

Chicago Manual of Style (16th Edition):

Pedersen Schuler, Elisabeth. “The Function of Wnt/beta-catenin Signaling in Ewing Sarcoma and its Contribution to Pathogenesis.” 2018. Doctoral Dissertation, University of Michigan. Accessed January 29, 2020. http://hdl.handle.net/2027.42/144020.

MLA Handbook (7th Edition):

Pedersen Schuler, Elisabeth. “The Function of Wnt/beta-catenin Signaling in Ewing Sarcoma and its Contribution to Pathogenesis.” 2018. Web. 29 Jan 2020.

Vancouver:

Pedersen Schuler E. The Function of Wnt/beta-catenin Signaling in Ewing Sarcoma and its Contribution to Pathogenesis. [Internet] [Doctoral dissertation]. University of Michigan; 2018. [cited 2020 Jan 29]. Available from: http://hdl.handle.net/2027.42/144020.

Council of Science Editors:

Pedersen Schuler E. The Function of Wnt/beta-catenin Signaling in Ewing Sarcoma and its Contribution to Pathogenesis. [Doctoral Dissertation]. University of Michigan; 2018. Available from: http://hdl.handle.net/2027.42/144020


University of Michigan

8. Shankar, Sunita. Insights into KRAS Biology through its Novel Interactions.

Degree: PhD, Molecular and Cellular Pathology, 2015, University of Michigan

 A third of all human cancers harbor mutations in the RAS family of genes, which encode members of small GTPases. RAS proteins transduce extracellular growth… (more)

Subjects/Keywords: KRAS rearrangements in metastatic prostate cancer; Outlier kinases in pancreatic cancer as targets for precision therapy; KRAS engages Argonaute 2 to promote cellular transformation; RAS and AGO2 interaction; Molecular, Cellular and Developmental Biology; Health Sciences

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APA (6th Edition):

Shankar, S. (2015). Insights into KRAS Biology through its Novel Interactions. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/113415

Chicago Manual of Style (16th Edition):

Shankar, Sunita. “Insights into KRAS Biology through its Novel Interactions.” 2015. Doctoral Dissertation, University of Michigan. Accessed January 29, 2020. http://hdl.handle.net/2027.42/113415.

MLA Handbook (7th Edition):

Shankar, Sunita. “Insights into KRAS Biology through its Novel Interactions.” 2015. Web. 29 Jan 2020.

Vancouver:

Shankar S. Insights into KRAS Biology through its Novel Interactions. [Internet] [Doctoral dissertation]. University of Michigan; 2015. [cited 2020 Jan 29]. Available from: http://hdl.handle.net/2027.42/113415.

Council of Science Editors:

Shankar S. Insights into KRAS Biology through its Novel Interactions. [Doctoral Dissertation]. University of Michigan; 2015. Available from: http://hdl.handle.net/2027.42/113415


University of Michigan

9. Wang, Jingya. The Role of the MLL-HOXA9 Axis in Normal and Malignant Hematopoiesis.

Degree: PhD, Molecular & Cellular Pathology, 2013, University of Michigan

 The MLL-HOXA9 axis plays a critical role in the regulation of development and hematopoiesis. Chromosome translocations of MLL are closely associated with human leukemia and… (more)

Subjects/Keywords: MLL; ASB2; Ubiquitination; Bromodomain; PHD Finger; HOXA9; Pathology; Health Sciences

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APA (6th Edition):

Wang, J. (2013). The Role of the MLL-HOXA9 Axis in Normal and Malignant Hematopoiesis. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/100097

Chicago Manual of Style (16th Edition):

Wang, Jingya. “The Role of the MLL-HOXA9 Axis in Normal and Malignant Hematopoiesis.” 2013. Doctoral Dissertation, University of Michigan. Accessed January 29, 2020. http://hdl.handle.net/2027.42/100097.

MLA Handbook (7th Edition):

Wang, Jingya. “The Role of the MLL-HOXA9 Axis in Normal and Malignant Hematopoiesis.” 2013. Web. 29 Jan 2020.

Vancouver:

Wang J. The Role of the MLL-HOXA9 Axis in Normal and Malignant Hematopoiesis. [Internet] [Doctoral dissertation]. University of Michigan; 2013. [cited 2020 Jan 29]. Available from: http://hdl.handle.net/2027.42/100097.

Council of Science Editors:

Wang J. The Role of the MLL-HOXA9 Axis in Normal and Malignant Hematopoiesis. [Doctoral Dissertation]. University of Michigan; 2013. Available from: http://hdl.handle.net/2027.42/100097


University of Michigan

10. Chang, Lou. Insights from Murine Models of Neurofibromatosis Type I: The Etiology and Appropriate Therapeutic Windows for Peripheral Nerve Sheath Tumors.

Degree: PhD, Cell and Developmental Biology, 2011, University of Michigan

 Neurofibromatosis Type I (NF1) is a common autosomal dominant disorder that afflicts approximately one in 3,500 live births. Individuals afflicted with NF1 are susceptible to… (more)

Subjects/Keywords: Cancer; Schwann Cell; Cell-of-Origin; Intervention; Pre-clinical; NF1; Molecular, Cellular and Developmental Biology; Health Sciences

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APA (6th Edition):

Chang, L. (2011). Insights from Murine Models of Neurofibromatosis Type I: The Etiology and Appropriate Therapeutic Windows for Peripheral Nerve Sheath Tumors. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/84543

Chicago Manual of Style (16th Edition):

Chang, Lou. “Insights from Murine Models of Neurofibromatosis Type I: The Etiology and Appropriate Therapeutic Windows for Peripheral Nerve Sheath Tumors.” 2011. Doctoral Dissertation, University of Michigan. Accessed January 29, 2020. http://hdl.handle.net/2027.42/84543.

MLA Handbook (7th Edition):

Chang, Lou. “Insights from Murine Models of Neurofibromatosis Type I: The Etiology and Appropriate Therapeutic Windows for Peripheral Nerve Sheath Tumors.” 2011. Web. 29 Jan 2020.

Vancouver:

Chang L. Insights from Murine Models of Neurofibromatosis Type I: The Etiology and Appropriate Therapeutic Windows for Peripheral Nerve Sheath Tumors. [Internet] [Doctoral dissertation]. University of Michigan; 2011. [cited 2020 Jan 29]. Available from: http://hdl.handle.net/2027.42/84543.

Council of Science Editors:

Chang L. Insights from Murine Models of Neurofibromatosis Type I: The Etiology and Appropriate Therapeutic Windows for Peripheral Nerve Sheath Tumors. [Doctoral Dissertation]. University of Michigan; 2011. Available from: http://hdl.handle.net/2027.42/84543


University of Michigan

11. Rowe, Robert Grant. Differential Regulation of Two- and Three-Dimensional Cell Function.

Degree: PhD, Cellular & Molecular Biology, 2011, University of Michigan

 The extracellular matrix (ECM) is a heterogeneous network of proteins, glycoproteins, and proteoglycans that not only scaffolds tissues but also acts as a central modulator… (more)

Subjects/Keywords: Extracellular Matrix; Molecular, Cellular and Developmental Biology; Science

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APA (6th Edition):

Rowe, R. G. (2011). Differential Regulation of Two- and Three-Dimensional Cell Function. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/84611

Chicago Manual of Style (16th Edition):

Rowe, Robert Grant. “Differential Regulation of Two- and Three-Dimensional Cell Function.” 2011. Doctoral Dissertation, University of Michigan. Accessed January 29, 2020. http://hdl.handle.net/2027.42/84611.

MLA Handbook (7th Edition):

Rowe, Robert Grant. “Differential Regulation of Two- and Three-Dimensional Cell Function.” 2011. Web. 29 Jan 2020.

Vancouver:

Rowe RG. Differential Regulation of Two- and Three-Dimensional Cell Function. [Internet] [Doctoral dissertation]. University of Michigan; 2011. [cited 2020 Jan 29]. Available from: http://hdl.handle.net/2027.42/84611.

Council of Science Editors:

Rowe RG. Differential Regulation of Two- and Three-Dimensional Cell Function. [Doctoral Dissertation]. University of Michigan; 2011. Available from: http://hdl.handle.net/2027.42/84611


University of Michigan

12. Dosch, Joseph Scott. Examining the Role of Hedgehog Signaling in the Pancreatic Tumor Microenvironment.

Degree: PhD, Cellular & Molecular Biology, 2011, University of Michigan

 The Hedgehog (Hh) pathway is a conserved signaling network that plays a critical role during embryonic development as well as in the maintenance of adult… (more)

Subjects/Keywords: Pancreatic Cancer; Hedgehog Pathway; Health Sciences

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APA (6th Edition):

Dosch, J. S. (2011). Examining the Role of Hedgehog Signaling in the Pancreatic Tumor Microenvironment. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/86276

Chicago Manual of Style (16th Edition):

Dosch, Joseph Scott. “Examining the Role of Hedgehog Signaling in the Pancreatic Tumor Microenvironment.” 2011. Doctoral Dissertation, University of Michigan. Accessed January 29, 2020. http://hdl.handle.net/2027.42/86276.

MLA Handbook (7th Edition):

Dosch, Joseph Scott. “Examining the Role of Hedgehog Signaling in the Pancreatic Tumor Microenvironment.” 2011. Web. 29 Jan 2020.

Vancouver:

Dosch JS. Examining the Role of Hedgehog Signaling in the Pancreatic Tumor Microenvironment. [Internet] [Doctoral dissertation]. University of Michigan; 2011. [cited 2020 Jan 29]. Available from: http://hdl.handle.net/2027.42/86276.

Council of Science Editors:

Dosch JS. Examining the Role of Hedgehog Signaling in the Pancreatic Tumor Microenvironment. [Doctoral Dissertation]. University of Michigan; 2011. Available from: http://hdl.handle.net/2027.42/86276


University of Michigan

13. Weng, Mo. Keeping Neural Progenitors on a Short Leash: Distinguishing Intermediate Progenitors from Stem Cells in Developing Drosophila Optic Lobe and Central Brain.

Degree: PhD, Cell and Developmental Biology, 2011, University of Michigan

 Producing intermediate progenitors is one of the critical strategies for stem cells to amplify their output and generate diversity, allowing stem cells to meet demanding… (more)

Subjects/Keywords: Intermediate Progenitor Cell; Molecular, Cellular and Developmental Biology; Science

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APA (6th Edition):

Weng, M. (2011). Keeping Neural Progenitors on a Short Leash: Distinguishing Intermediate Progenitors from Stem Cells in Developing Drosophila Optic Lobe and Central Brain. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/89726

Chicago Manual of Style (16th Edition):

Weng, Mo. “Keeping Neural Progenitors on a Short Leash: Distinguishing Intermediate Progenitors from Stem Cells in Developing Drosophila Optic Lobe and Central Brain.” 2011. Doctoral Dissertation, University of Michigan. Accessed January 29, 2020. http://hdl.handle.net/2027.42/89726.

MLA Handbook (7th Edition):

Weng, Mo. “Keeping Neural Progenitors on a Short Leash: Distinguishing Intermediate Progenitors from Stem Cells in Developing Drosophila Optic Lobe and Central Brain.” 2011. Web. 29 Jan 2020.

Vancouver:

Weng M. Keeping Neural Progenitors on a Short Leash: Distinguishing Intermediate Progenitors from Stem Cells in Developing Drosophila Optic Lobe and Central Brain. [Internet] [Doctoral dissertation]. University of Michigan; 2011. [cited 2020 Jan 29]. Available from: http://hdl.handle.net/2027.42/89726.

Council of Science Editors:

Weng M. Keeping Neural Progenitors on a Short Leash: Distinguishing Intermediate Progenitors from Stem Cells in Developing Drosophila Optic Lobe and Central Brain. [Doctoral Dissertation]. University of Michigan; 2011. Available from: http://hdl.handle.net/2027.42/89726

14. Ropa, James. Epigenetic and Transcriptional Regulation of Self Renewal in Acute Myeloid Leukemia.

Degree: PhD, Molecular & Cellular Pathology, 2019, University of Michigan

 Acute myeloid leukemia (AML) is diagnosed in >20,000 people/year in the United States alone and is associated with a poor prognosis. AML arises due to… (more)

Subjects/Keywords: Acute myeloid leukemia; Epigenetics and epigenomics; Histone H3K9 methylation; Self-renewal; Transcriptional repression; Proteomics and protein-protein interactions; Biological Chemistry; Molecular, Cellular and Developmental Biology; Oncology and Hematology; Pathology; Health Sciences; Science

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APA (6th Edition):

Ropa, J. (2019). Epigenetic and Transcriptional Regulation of Self Renewal in Acute Myeloid Leukemia. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/149783

Chicago Manual of Style (16th Edition):

Ropa, James. “Epigenetic and Transcriptional Regulation of Self Renewal in Acute Myeloid Leukemia.” 2019. Doctoral Dissertation, University of Michigan. Accessed January 29, 2020. http://hdl.handle.net/2027.42/149783.

MLA Handbook (7th Edition):

Ropa, James. “Epigenetic and Transcriptional Regulation of Self Renewal in Acute Myeloid Leukemia.” 2019. Web. 29 Jan 2020.

Vancouver:

Ropa J. Epigenetic and Transcriptional Regulation of Self Renewal in Acute Myeloid Leukemia. [Internet] [Doctoral dissertation]. University of Michigan; 2019. [cited 2020 Jan 29]. Available from: http://hdl.handle.net/2027.42/149783.

Council of Science Editors:

Ropa J. Epigenetic and Transcriptional Regulation of Self Renewal in Acute Myeloid Leukemia. [Doctoral Dissertation]. University of Michigan; 2019. Available from: http://hdl.handle.net/2027.42/149783


University of Michigan

15. Altemus, Megan. Metabolic and Microenvironmental Determinants of Breast Cancer Metastasis: Effects of Glycogen Utilization on Metastatic Phenotypes and a Predictive Brain Metastasis Microfluidic Device.

Degree: PhD, Cancer Biology, 2019, University of Michigan

 Breast cancer has the highest incidence rates of all cancer types among women in the United States, and the second highest mortality. While survival is… (more)

Subjects/Keywords: breast cancer; glycogen metabolism; brain metastasis; microfluidic devices; Oncology and Hematology; Health Sciences

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APA (6th Edition):

Altemus, M. (2019). Metabolic and Microenvironmental Determinants of Breast Cancer Metastasis: Effects of Glycogen Utilization on Metastatic Phenotypes and a Predictive Brain Metastasis Microfluidic Device. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/151668

Chicago Manual of Style (16th Edition):

Altemus, Megan. “Metabolic and Microenvironmental Determinants of Breast Cancer Metastasis: Effects of Glycogen Utilization on Metastatic Phenotypes and a Predictive Brain Metastasis Microfluidic Device.” 2019. Doctoral Dissertation, University of Michigan. Accessed January 29, 2020. http://hdl.handle.net/2027.42/151668.

MLA Handbook (7th Edition):

Altemus, Megan. “Metabolic and Microenvironmental Determinants of Breast Cancer Metastasis: Effects of Glycogen Utilization on Metastatic Phenotypes and a Predictive Brain Metastasis Microfluidic Device.” 2019. Web. 29 Jan 2020.

Vancouver:

Altemus M. Metabolic and Microenvironmental Determinants of Breast Cancer Metastasis: Effects of Glycogen Utilization on Metastatic Phenotypes and a Predictive Brain Metastasis Microfluidic Device. [Internet] [Doctoral dissertation]. University of Michigan; 2019. [cited 2020 Jan 29]. Available from: http://hdl.handle.net/2027.42/151668.

Council of Science Editors:

Altemus M. Metabolic and Microenvironmental Determinants of Breast Cancer Metastasis: Effects of Glycogen Utilization on Metastatic Phenotypes and a Predictive Brain Metastasis Microfluidic Device. [Doctoral Dissertation]. University of Michigan; 2019. Available from: http://hdl.handle.net/2027.42/151668

16. Regal, Joshua A. Characterization of MRE11 Mutants Provides Insight into Human Genetic Disease, Cancer, and Clinical Intervention.

Degree: PhD, Molecular and Cellular Pathology, 2015, University of Michigan

 DNA double strand breaks (DSBs) pose a serious threat to cellular and organism well-being. In response to DSBs, MRE11/RAD50/NBS1 (MRN) initiates DNA repair and facilitates… (more)

Subjects/Keywords: DNA damage response; cancer; human genetic disease; Molecular, Cellular and Developmental Biology; Science

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APA (6th Edition):

Regal, J. A. (2015). Characterization of MRE11 Mutants Provides Insight into Human Genetic Disease, Cancer, and Clinical Intervention. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/111392

Chicago Manual of Style (16th Edition):

Regal, Joshua A. “Characterization of MRE11 Mutants Provides Insight into Human Genetic Disease, Cancer, and Clinical Intervention.” 2015. Doctoral Dissertation, University of Michigan. Accessed January 29, 2020. http://hdl.handle.net/2027.42/111392.

MLA Handbook (7th Edition):

Regal, Joshua A. “Characterization of MRE11 Mutants Provides Insight into Human Genetic Disease, Cancer, and Clinical Intervention.” 2015. Web. 29 Jan 2020.

Vancouver:

Regal JA. Characterization of MRE11 Mutants Provides Insight into Human Genetic Disease, Cancer, and Clinical Intervention. [Internet] [Doctoral dissertation]. University of Michigan; 2015. [cited 2020 Jan 29]. Available from: http://hdl.handle.net/2027.42/111392.

Council of Science Editors:

Regal JA. Characterization of MRE11 Mutants Provides Insight into Human Genetic Disease, Cancer, and Clinical Intervention. [Doctoral Dissertation]. University of Michigan; 2015. Available from: http://hdl.handle.net/2027.42/111392

17. Jones, Morgan. New Insights into the Regulation of Hematopoietic Stem Cell Self-Renewal.

Degree: PhD, Cellular and Molecular Biology, 2015, University of Michigan

 Self-renewal is essential for stem cell maintenance. In the blood system, rare hematopoietic stem cells (HSCs) must maintain their self-renewal potential to sustain long-term homeostasis.… (more)

Subjects/Keywords: hematopoietic stem cells; telomere; shelterin; histone methyltransferase; quiescence; trithorax; Molecular, Cellular and Developmental Biology; Oncology and Hematology; Health Sciences

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APA (6th Edition):

Jones, M. (2015). New Insights into the Regulation of Hematopoietic Stem Cell Self-Renewal. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/111406

Chicago Manual of Style (16th Edition):

Jones, Morgan. “New Insights into the Regulation of Hematopoietic Stem Cell Self-Renewal.” 2015. Doctoral Dissertation, University of Michigan. Accessed January 29, 2020. http://hdl.handle.net/2027.42/111406.

MLA Handbook (7th Edition):

Jones, Morgan. “New Insights into the Regulation of Hematopoietic Stem Cell Self-Renewal.” 2015. Web. 29 Jan 2020.

Vancouver:

Jones M. New Insights into the Regulation of Hematopoietic Stem Cell Self-Renewal. [Internet] [Doctoral dissertation]. University of Michigan; 2015. [cited 2020 Jan 29]. Available from: http://hdl.handle.net/2027.42/111406.

Council of Science Editors:

Jones M. New Insights into the Regulation of Hematopoietic Stem Cell Self-Renewal. [Doctoral Dissertation]. University of Michigan; 2015. Available from: http://hdl.handle.net/2027.42/111406

18. Gibbons, Garrett S. The Histone Methyltransferase DOT1L: Discovery of Small-Molecule Inhibitors and its Role in Wnt Signaling.

Degree: PhD, Molecular and Cellular Pathology, 2014, University of Michigan

 Covalent post translational modifications of histone proteins are an important mechanism of epigenetic gene regulation that modulate chromatin structure. Methylation of histone lysine residues is… (more)

Subjects/Keywords: DOT1L; Epigenetics; Leukemia; Histone methyltransferase; Cancer; Chromatin; Biological Chemistry; Chemistry; Science

…trifluoromethansulfonate TRRAP - Transformation/transcription domain-associated protein UMD – University of… …Michigan Disruptor of H3K79 methylation UV-vis – ultra violet- visible light spectrum WDR5 – WD… 

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APA (6th Edition):

Gibbons, G. S. (2014). The Histone Methyltransferase DOT1L: Discovery of Small-Molecule Inhibitors and its Role in Wnt Signaling. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/110377

Chicago Manual of Style (16th Edition):

Gibbons, Garrett S. “The Histone Methyltransferase DOT1L: Discovery of Small-Molecule Inhibitors and its Role in Wnt Signaling.” 2014. Doctoral Dissertation, University of Michigan. Accessed January 29, 2020. http://hdl.handle.net/2027.42/110377.

MLA Handbook (7th Edition):

Gibbons, Garrett S. “The Histone Methyltransferase DOT1L: Discovery of Small-Molecule Inhibitors and its Role in Wnt Signaling.” 2014. Web. 29 Jan 2020.

Vancouver:

Gibbons GS. The Histone Methyltransferase DOT1L: Discovery of Small-Molecule Inhibitors and its Role in Wnt Signaling. [Internet] [Doctoral dissertation]. University of Michigan; 2014. [cited 2020 Jan 29]. Available from: http://hdl.handle.net/2027.42/110377.

Council of Science Editors:

Gibbons GS. The Histone Methyltransferase DOT1L: Discovery of Small-Molecule Inhibitors and its Role in Wnt Signaling. [Doctoral Dissertation]. University of Michigan; 2014. Available from: http://hdl.handle.net/2027.42/110377

19. Chang, Jin-Hee. General and Tissue Specific Gene Regulation by the WNT Signaling Pathway in Drosophila.

Degree: PhD, Molecular, Cellular, and Developmental Biology, 2008, University of Michigan

 Secreted proteins of the Wnt family act through a conserved signaling cascade to regulate gene expression. While many genes are regulated by this pathway in… (more)

Subjects/Keywords: Wnt; Wg; Spenito; Naked Cuticle; Molecular, Cellular and Developmental Biology; Science

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APA (6th Edition):

Chang, J. (2008). General and Tissue Specific Gene Regulation by the WNT Signaling Pathway in Drosophila. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/60792

Chicago Manual of Style (16th Edition):

Chang, Jin-Hee. “General and Tissue Specific Gene Regulation by the WNT Signaling Pathway in Drosophila.” 2008. Doctoral Dissertation, University of Michigan. Accessed January 29, 2020. http://hdl.handle.net/2027.42/60792.

MLA Handbook (7th Edition):

Chang, Jin-Hee. “General and Tissue Specific Gene Regulation by the WNT Signaling Pathway in Drosophila.” 2008. Web. 29 Jan 2020.

Vancouver:

Chang J. General and Tissue Specific Gene Regulation by the WNT Signaling Pathway in Drosophila. [Internet] [Doctoral dissertation]. University of Michigan; 2008. [cited 2020 Jan 29]. Available from: http://hdl.handle.net/2027.42/60792.

Council of Science Editors:

Chang J. General and Tissue Specific Gene Regulation by the WNT Signaling Pathway in Drosophila. [Doctoral Dissertation]. University of Michigan; 2008. Available from: http://hdl.handle.net/2027.42/60792

20. Lee, Jae Young. The Regulation of Signaling in Hematopietic Stem Cell Maintenance.

Degree: PhD, Cell and Developmental Biology, 2012, University of Michigan

 Hematopoietic stem cells (HSCs) maintain themselves throughout life by undergoing self-renewing divisions and by differentiating to generate all the blood and immune system cells in… (more)

Subjects/Keywords: Pten; HSC; Leukemia; Ink4a/Arf; P53; Reactive Oxygen Species; Molecular, Cellular and Developmental Biology; Health Sciences

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APA (6th Edition):

Lee, J. Y. (2012). The Regulation of Signaling in Hematopietic Stem Cell Maintenance. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/91538

Chicago Manual of Style (16th Edition):

Lee, Jae Young. “The Regulation of Signaling in Hematopietic Stem Cell Maintenance.” 2012. Doctoral Dissertation, University of Michigan. Accessed January 29, 2020. http://hdl.handle.net/2027.42/91538.

MLA Handbook (7th Edition):

Lee, Jae Young. “The Regulation of Signaling in Hematopietic Stem Cell Maintenance.” 2012. Web. 29 Jan 2020.

Vancouver:

Lee JY. The Regulation of Signaling in Hematopietic Stem Cell Maintenance. [Internet] [Doctoral dissertation]. University of Michigan; 2012. [cited 2020 Jan 29]. Available from: http://hdl.handle.net/2027.42/91538.

Council of Science Editors:

Lee JY. The Regulation of Signaling in Hematopietic Stem Cell Maintenance. [Doctoral Dissertation]. University of Michigan; 2012. Available from: http://hdl.handle.net/2027.42/91538

21. Grimley, Edward. Toward Molecularly Targeted Therapies for Renal Disease.

Degree: PhD, Molecular & Cellular Pathology, 2017, University of Michigan

 Pax2 is a developmental control gene that is essential for organogenesis of the kidney and urogenital tract. Genes of this nature are generally suppressed in… (more)

Subjects/Keywords: Pax2; paired domain; drug discovery; high-throughput screen; virtual screen; kidney disease; Biological Chemistry; Genetics; Molecular, Cellular and Developmental Biology; Science (General); Science

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APA (6th Edition):

Grimley, E. (2017). Toward Molecularly Targeted Therapies for Renal Disease. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/138499

Chicago Manual of Style (16th Edition):

Grimley, Edward. “Toward Molecularly Targeted Therapies for Renal Disease.” 2017. Doctoral Dissertation, University of Michigan. Accessed January 29, 2020. http://hdl.handle.net/2027.42/138499.

MLA Handbook (7th Edition):

Grimley, Edward. “Toward Molecularly Targeted Therapies for Renal Disease.” 2017. Web. 29 Jan 2020.

Vancouver:

Grimley E. Toward Molecularly Targeted Therapies for Renal Disease. [Internet] [Doctoral dissertation]. University of Michigan; 2017. [cited 2020 Jan 29]. Available from: http://hdl.handle.net/2027.42/138499.

Council of Science Editors:

Grimley E. Toward Molecularly Targeted Therapies for Renal Disease. [Doctoral Dissertation]. University of Michigan; 2017. Available from: http://hdl.handle.net/2027.42/138499

22. Zhang, Peng. Mechanisms and Regulation of Transforming Growth Factor Superfamily Mediated Gene Expression.

Degree: PhD, Molecular & Cellular Pathology, 2012, University of Michigan

 The TGF-beta superfamily, including TGF-betas and BMPs, is critical for normal embryonic development, as well as disease progression, and is tightly regulated both within and… (more)

Subjects/Keywords: TGF-beta; Epithelial Mesenchymal Transition; Gene Regulation; Wnt Signaling; Groucho Proteins; Renal Fibrosis; Pathology; Health Sciences

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APA (6th Edition):

Zhang, P. (2012). Mechanisms and Regulation of Transforming Growth Factor Superfamily Mediated Gene Expression. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/96131

Chicago Manual of Style (16th Edition):

Zhang, Peng. “Mechanisms and Regulation of Transforming Growth Factor Superfamily Mediated Gene Expression.” 2012. Doctoral Dissertation, University of Michigan. Accessed January 29, 2020. http://hdl.handle.net/2027.42/96131.

MLA Handbook (7th Edition):

Zhang, Peng. “Mechanisms and Regulation of Transforming Growth Factor Superfamily Mediated Gene Expression.” 2012. Web. 29 Jan 2020.

Vancouver:

Zhang P. Mechanisms and Regulation of Transforming Growth Factor Superfamily Mediated Gene Expression. [Internet] [Doctoral dissertation]. University of Michigan; 2012. [cited 2020 Jan 29]. Available from: http://hdl.handle.net/2027.42/96131.

Council of Science Editors:

Zhang P. Mechanisms and Regulation of Transforming Growth Factor Superfamily Mediated Gene Expression. [Doctoral Dissertation]. University of Michigan; 2012. Available from: http://hdl.handle.net/2027.42/96131

23. Feinberg, Tamar Yael. Extracellular Matrix Remodeling and the Control of Branching Morphogenetic Programs.

Degree: PhD, Cellular and Molecular Biology, 2016, University of Michigan

 Epithelial cells and endothelial cells initiate distinct branching morphogenetic programs during their coordinated invasion and proliferation into the interstitial compartment, a tissue comprised of mesenchymal… (more)

Subjects/Keywords: Biological Sciences; Developmental Biology; Branching Morphogenesis; Cell Migration; Molecular, Cellular and Developmental Biology; Science

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APA (6th Edition):

Feinberg, T. Y. (2016). Extracellular Matrix Remodeling and the Control of Branching Morphogenetic Programs. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/120758

Chicago Manual of Style (16th Edition):

Feinberg, Tamar Yael. “Extracellular Matrix Remodeling and the Control of Branching Morphogenetic Programs.” 2016. Doctoral Dissertation, University of Michigan. Accessed January 29, 2020. http://hdl.handle.net/2027.42/120758.

MLA Handbook (7th Edition):

Feinberg, Tamar Yael. “Extracellular Matrix Remodeling and the Control of Branching Morphogenetic Programs.” 2016. Web. 29 Jan 2020.

Vancouver:

Feinberg TY. Extracellular Matrix Remodeling and the Control of Branching Morphogenetic Programs. [Internet] [Doctoral dissertation]. University of Michigan; 2016. [cited 2020 Jan 29]. Available from: http://hdl.handle.net/2027.42/120758.

Council of Science Editors:

Feinberg TY. Extracellular Matrix Remodeling and the Control of Branching Morphogenetic Programs. [Doctoral Dissertation]. University of Michigan; 2016. Available from: http://hdl.handle.net/2027.42/120758

24. Wong, Chun-Shu. The Role of Adenomatous Polyposis Coli in T cell biology: Development, Naïve T cell maintenance, and T cell Homeostatic Proliferation.

Degree: PhD, Immunology, 2015, University of Michigan

 T cell development begins with the migration of early progenitors from the bone marrow to the thymus. Once out of the thymus, naïve T cells… (more)

Subjects/Keywords: role Adenomatous Polyposis Coli in T cell biology; Microbiology and Immunology; Science

…the University of Michigan and the Children’s National Medical Center. Flow cytometric… 

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APA (6th Edition):

Wong, C. (2015). The Role of Adenomatous Polyposis Coli in T cell biology: Development, Naïve T cell maintenance, and T cell Homeostatic Proliferation. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/111478

Chicago Manual of Style (16th Edition):

Wong, Chun-Shu. “The Role of Adenomatous Polyposis Coli in T cell biology: Development, Naïve T cell maintenance, and T cell Homeostatic Proliferation.” 2015. Doctoral Dissertation, University of Michigan. Accessed January 29, 2020. http://hdl.handle.net/2027.42/111478.

MLA Handbook (7th Edition):

Wong, Chun-Shu. “The Role of Adenomatous Polyposis Coli in T cell biology: Development, Naïve T cell maintenance, and T cell Homeostatic Proliferation.” 2015. Web. 29 Jan 2020.

Vancouver:

Wong C. The Role of Adenomatous Polyposis Coli in T cell biology: Development, Naïve T cell maintenance, and T cell Homeostatic Proliferation. [Internet] [Doctoral dissertation]. University of Michigan; 2015. [cited 2020 Jan 29]. Available from: http://hdl.handle.net/2027.42/111478.

Council of Science Editors:

Wong C. The Role of Adenomatous Polyposis Coli in T cell biology: Development, Naïve T cell maintenance, and T cell Homeostatic Proliferation. [Doctoral Dissertation]. University of Michigan; 2015. Available from: http://hdl.handle.net/2027.42/111478

25. Kuang, Chaoyuan. Establishing the Drosophila Larval Neuroblast as a System to Study Regulated Cell Death in vivo.

Degree: PhD, Cellular and Molecular Biology, 2015, University of Michigan

 Programmed cell death is often thought of as a developmental process that is genetically hardwired to occur in the organism in a spatially and temporally… (more)

Subjects/Keywords: stem cell survival; necrotic cell death; cell survival checkpoint; genotoxic stress; Molecular, Cellular and Developmental Biology; Science

…Sciences Institute, University of Michigan, Ann Arbor, MI 48109 7. Graduate Program in Molecular… 

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APA (6th Edition):

Kuang, C. (2015). Establishing the Drosophila Larval Neuroblast as a System to Study Regulated Cell Death in vivo. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/111586

Chicago Manual of Style (16th Edition):

Kuang, Chaoyuan. “Establishing the Drosophila Larval Neuroblast as a System to Study Regulated Cell Death in vivo.” 2015. Doctoral Dissertation, University of Michigan. Accessed January 29, 2020. http://hdl.handle.net/2027.42/111586.

MLA Handbook (7th Edition):

Kuang, Chaoyuan. “Establishing the Drosophila Larval Neuroblast as a System to Study Regulated Cell Death in vivo.” 2015. Web. 29 Jan 2020.

Vancouver:

Kuang C. Establishing the Drosophila Larval Neuroblast as a System to Study Regulated Cell Death in vivo. [Internet] [Doctoral dissertation]. University of Michigan; 2015. [cited 2020 Jan 29]. Available from: http://hdl.handle.net/2027.42/111586.

Council of Science Editors:

Kuang C. Establishing the Drosophila Larval Neuroblast as a System to Study Regulated Cell Death in vivo. [Doctoral Dissertation]. University of Michigan; 2015. Available from: http://hdl.handle.net/2027.42/111586

26. Mazzoni, Serina Marie. A Role for AXIN2 in Oncogenesis.

Degree: PhD, Human Genetics, 2014, University of Michigan

 The axis inhibition proteins 1 and 2 (AXIN1, AXIN2) are negative regulators of Wnt/beta-catenin signaling. The Wnt pathway has a key role in cell fate… (more)

Subjects/Keywords: AXIN2 in colorectal cancer; Wnt signaling; AXIN; conductin; Genetics; Science

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APA (6th Edition):

Mazzoni, S. M. (2014). A Role for AXIN2 in Oncogenesis. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/110453

Chicago Manual of Style (16th Edition):

Mazzoni, Serina Marie. “A Role for AXIN2 in Oncogenesis.” 2014. Doctoral Dissertation, University of Michigan. Accessed January 29, 2020. http://hdl.handle.net/2027.42/110453.

MLA Handbook (7th Edition):

Mazzoni, Serina Marie. “A Role for AXIN2 in Oncogenesis.” 2014. Web. 29 Jan 2020.

Vancouver:

Mazzoni SM. A Role for AXIN2 in Oncogenesis. [Internet] [Doctoral dissertation]. University of Michigan; 2014. [cited 2020 Jan 29]. Available from: http://hdl.handle.net/2027.42/110453.

Council of Science Editors:

Mazzoni SM. A Role for AXIN2 in Oncogenesis. [Doctoral Dissertation]. University of Michigan; 2014. Available from: http://hdl.handle.net/2027.42/110453

27. Akgul, Seckin. The Functions of Rictor, Pten and p53 Signaling Pathways in Brain Tumors and Brain Development.

Degree: PhD, Cellular and Molecular Biology, 2015, University of Michigan

 Glioblastoma is the most common malignant neoplasm of the central nervous system. Despite intense treatment plans and supportive care, the median survival rate remains approximately… (more)

Subjects/Keywords: Adult and pediatric brain tumors; Glioblastoma; Medulloblastoma; Rictor/mTORC2; RTK/PI3K/AKT signaling pathway; TP53/p53 signaling pathway; Molecular, Cellular and Developmental Biology; Science

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APA (6th Edition):

Akgul, S. (2015). The Functions of Rictor, Pten and p53 Signaling Pathways in Brain Tumors and Brain Development. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/113298

Chicago Manual of Style (16th Edition):

Akgul, Seckin. “The Functions of Rictor, Pten and p53 Signaling Pathways in Brain Tumors and Brain Development.” 2015. Doctoral Dissertation, University of Michigan. Accessed January 29, 2020. http://hdl.handle.net/2027.42/113298.

MLA Handbook (7th Edition):

Akgul, Seckin. “The Functions of Rictor, Pten and p53 Signaling Pathways in Brain Tumors and Brain Development.” 2015. Web. 29 Jan 2020.

Vancouver:

Akgul S. The Functions of Rictor, Pten and p53 Signaling Pathways in Brain Tumors and Brain Development. [Internet] [Doctoral dissertation]. University of Michigan; 2015. [cited 2020 Jan 29]. Available from: http://hdl.handle.net/2027.42/113298.

Council of Science Editors:

Akgul S. The Functions of Rictor, Pten and p53 Signaling Pathways in Brain Tumors and Brain Development. [Doctoral Dissertation]. University of Michigan; 2015. Available from: http://hdl.handle.net/2027.42/113298

28. Zwaans, Bernadette Margaretha Maria. Exploring the Roles of the Histone Deacetylase and Longevity Factor SIRT6 in Cancer.

Degree: PhD, Molecular and Cellular Pathology, 2014, University of Michigan

 Identification of pathways that regulate longevity in an evolutionarily conserved manner is a major focus of modern biogerontology. Interest in the sirtuin family of deacetylases/ADP-ribosyltransferases/deacylases… (more)

Subjects/Keywords: Sirtuin; Cancer; histone deacetylation; cancer metabolism; Molecular, Cellular and Developmental Biology; Science

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APA (6th Edition):

Zwaans, B. M. M. (2014). Exploring the Roles of the Histone Deacetylase and Longevity Factor SIRT6 in Cancer. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/110378

Chicago Manual of Style (16th Edition):

Zwaans, Bernadette Margaretha Maria. “Exploring the Roles of the Histone Deacetylase and Longevity Factor SIRT6 in Cancer.” 2014. Doctoral Dissertation, University of Michigan. Accessed January 29, 2020. http://hdl.handle.net/2027.42/110378.

MLA Handbook (7th Edition):

Zwaans, Bernadette Margaretha Maria. “Exploring the Roles of the Histone Deacetylase and Longevity Factor SIRT6 in Cancer.” 2014. Web. 29 Jan 2020.

Vancouver:

Zwaans BMM. Exploring the Roles of the Histone Deacetylase and Longevity Factor SIRT6 in Cancer. [Internet] [Doctoral dissertation]. University of Michigan; 2014. [cited 2020 Jan 29]. Available from: http://hdl.handle.net/2027.42/110378.

Council of Science Editors:

Zwaans BMM. Exploring the Roles of the Histone Deacetylase and Longevity Factor SIRT6 in Cancer. [Doctoral Dissertation]. University of Michigan; 2014. Available from: http://hdl.handle.net/2027.42/110378

29. Li, Bo. Development and Application of Novel Methods to Study Tumor Heterogeneity and Cancer Genome Evolution.

Degree: PhD, Bioinformatics, 2014, University of Michigan

 Cancer is one of the leading causes of death worldwide. In recent years, with the aid of high-throughput genomic technologies, large cohorts of tumor samples… (more)

Subjects/Keywords: tumor subtype; intra-tumor heterogeneity; short tandem repeat expansion; Genetics; Health Sciences

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APA (6th Edition):

Li, B. (2014). Development and Application of Novel Methods to Study Tumor Heterogeneity and Cancer Genome Evolution. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/110386

Chicago Manual of Style (16th Edition):

Li, Bo. “Development and Application of Novel Methods to Study Tumor Heterogeneity and Cancer Genome Evolution.” 2014. Doctoral Dissertation, University of Michigan. Accessed January 29, 2020. http://hdl.handle.net/2027.42/110386.

MLA Handbook (7th Edition):

Li, Bo. “Development and Application of Novel Methods to Study Tumor Heterogeneity and Cancer Genome Evolution.” 2014. Web. 29 Jan 2020.

Vancouver:

Li B. Development and Application of Novel Methods to Study Tumor Heterogeneity and Cancer Genome Evolution. [Internet] [Doctoral dissertation]. University of Michigan; 2014. [cited 2020 Jan 29]. Available from: http://hdl.handle.net/2027.42/110386.

Council of Science Editors:

Li B. Development and Application of Novel Methods to Study Tumor Heterogeneity and Cancer Genome Evolution. [Doctoral Dissertation]. University of Michigan; 2014. Available from: http://hdl.handle.net/2027.42/110386

30. Krill, Kenneth Thomas. Effects of Dicer Inactivation in the Developing Mouse Adrenal Cortex and Micro-RNAs in Adrenocortical Carcinoma.

Degree: PhD, Cellular and Molecular Biology, 2014, University of Michigan

 Adrenocortical carcinoma (ACC) is a rare yet highly aggressive form of cancer with limited treatment options and poor prognosis. Insulin-like growth factor 2 (IGF2) is… (more)

Subjects/Keywords: MiRNA; MicroRNA; Adrenal Gland; Adrenal Cancer; Adrenal Development; Nr6a1; Molecular, Cellular and Developmental Biology; Science

…and characterized. Jerome W. Conn, a University of Michigan Medical School alumnus and… 

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APA (6th Edition):

Krill, K. T. (2014). Effects of Dicer Inactivation in the Developing Mouse Adrenal Cortex and Micro-RNAs in Adrenocortical Carcinoma. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/107297

Chicago Manual of Style (16th Edition):

Krill, Kenneth Thomas. “Effects of Dicer Inactivation in the Developing Mouse Adrenal Cortex and Micro-RNAs in Adrenocortical Carcinoma.” 2014. Doctoral Dissertation, University of Michigan. Accessed January 29, 2020. http://hdl.handle.net/2027.42/107297.

MLA Handbook (7th Edition):

Krill, Kenneth Thomas. “Effects of Dicer Inactivation in the Developing Mouse Adrenal Cortex and Micro-RNAs in Adrenocortical Carcinoma.” 2014. Web. 29 Jan 2020.

Vancouver:

Krill KT. Effects of Dicer Inactivation in the Developing Mouse Adrenal Cortex and Micro-RNAs in Adrenocortical Carcinoma. [Internet] [Doctoral dissertation]. University of Michigan; 2014. [cited 2020 Jan 29]. Available from: http://hdl.handle.net/2027.42/107297.

Council of Science Editors:

Krill KT. Effects of Dicer Inactivation in the Developing Mouse Adrenal Cortex and Micro-RNAs in Adrenocortical Carcinoma. [Doctoral Dissertation]. University of Michigan; 2014. Available from: http://hdl.handle.net/2027.42/107297

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