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You searched for +publisher:"University of Michigan" +contributor:("Dressler, Gregory R."). Showing records 1 – 22 of 22 total matches.

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University of Michigan

1. Larsen, Brian Matthew. Hox6 function is Necessary for Endocrine Pancreas Development in vivo and in vitro.

Degree: PhD, Cellular and Molecular Biology, 2015, University of Michigan

 Diabetes affects millions of Americans and is caused by a disruption in β-cell abundance or function. Beta cells are one of the five types of… (more)

Subjects/Keywords: Hox6 function in pancreatic endocrine cell development; Molecular, Cellular and Developmental Biology; Science

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APA (6th Edition):

Larsen, B. M. (2015). Hox6 function is Necessary for Endocrine Pancreas Development in vivo and in vitro. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/116790

Chicago Manual of Style (16th Edition):

Larsen, Brian Matthew. “Hox6 function is Necessary for Endocrine Pancreas Development in vivo and in vitro.” 2015. Doctoral Dissertation, University of Michigan. Accessed January 23, 2020. http://hdl.handle.net/2027.42/116790.

MLA Handbook (7th Edition):

Larsen, Brian Matthew. “Hox6 function is Necessary for Endocrine Pancreas Development in vivo and in vitro.” 2015. Web. 23 Jan 2020.

Vancouver:

Larsen BM. Hox6 function is Necessary for Endocrine Pancreas Development in vivo and in vitro. [Internet] [Doctoral dissertation]. University of Michigan; 2015. [cited 2020 Jan 23]. Available from: http://hdl.handle.net/2027.42/116790.

Council of Science Editors:

Larsen BM. Hox6 function is Necessary for Endocrine Pancreas Development in vivo and in vitro. [Doctoral Dissertation]. University of Michigan; 2015. Available from: http://hdl.handle.net/2027.42/116790


University of Michigan

2. Townsend, Elizabeth Christine. Development of a Novel Inhibitor to the Conserved, Developmental Regulator, WDR5, for Treatment of Acute Leukemia.

Degree: PhD, Molecular & Cellular Pathology, 2012, University of Michigan

 A majority of cases of acute leukemia in infants, as well as a subset of secondary acute leukemia in adults is characterized by translocation of… (more)

Subjects/Keywords: Inhibition of MLL1 Complex Activity for Leukemia Therapy; Science

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APA (6th Edition):

Townsend, E. C. (2012). Development of a Novel Inhibitor to the Conserved, Developmental Regulator, WDR5, for Treatment of Acute Leukemia. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/96011

Chicago Manual of Style (16th Edition):

Townsend, Elizabeth Christine. “Development of a Novel Inhibitor to the Conserved, Developmental Regulator, WDR5, for Treatment of Acute Leukemia.” 2012. Doctoral Dissertation, University of Michigan. Accessed January 23, 2020. http://hdl.handle.net/2027.42/96011.

MLA Handbook (7th Edition):

Townsend, Elizabeth Christine. “Development of a Novel Inhibitor to the Conserved, Developmental Regulator, WDR5, for Treatment of Acute Leukemia.” 2012. Web. 23 Jan 2020.

Vancouver:

Townsend EC. Development of a Novel Inhibitor to the Conserved, Developmental Regulator, WDR5, for Treatment of Acute Leukemia. [Internet] [Doctoral dissertation]. University of Michigan; 2012. [cited 2020 Jan 23]. Available from: http://hdl.handle.net/2027.42/96011.

Council of Science Editors:

Townsend EC. Development of a Novel Inhibitor to the Conserved, Developmental Regulator, WDR5, for Treatment of Acute Leukemia. [Doctoral Dissertation]. University of Michigan; 2012. Available from: http://hdl.handle.net/2027.42/96011


University of Michigan

3. Serio, Justin. The Polymerase Associated Factor Complex-Protein Arginine Methyltransferase Axis: A Mechanism of Acute Leukemia.

Degree: PhD, Molecular & Cellular Pathology, 2017, University of Michigan

 Transcriptional and epigenetic mechanisms are pivotal to the maintenance of gene programs responsible for cellular homeostasis. Like many cancers, acute myeloid leukemia (AML) is a… (more)

Subjects/Keywords: Transcriptional and Epigenetic Regulation in Leukemia; Polymerase Associated Factor Complex; Protein Arginine Methyltransferases; Molecular, Cellular and Developmental Biology; Oncology and Hematology; Pathology; Health Sciences; Science

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APA (6th Edition):

Serio, J. (2017). The Polymerase Associated Factor Complex-Protein Arginine Methyltransferase Axis: A Mechanism of Acute Leukemia. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/140915

Chicago Manual of Style (16th Edition):

Serio, Justin. “The Polymerase Associated Factor Complex-Protein Arginine Methyltransferase Axis: A Mechanism of Acute Leukemia.” 2017. Doctoral Dissertation, University of Michigan. Accessed January 23, 2020. http://hdl.handle.net/2027.42/140915.

MLA Handbook (7th Edition):

Serio, Justin. “The Polymerase Associated Factor Complex-Protein Arginine Methyltransferase Axis: A Mechanism of Acute Leukemia.” 2017. Web. 23 Jan 2020.

Vancouver:

Serio J. The Polymerase Associated Factor Complex-Protein Arginine Methyltransferase Axis: A Mechanism of Acute Leukemia. [Internet] [Doctoral dissertation]. University of Michigan; 2017. [cited 2020 Jan 23]. Available from: http://hdl.handle.net/2027.42/140915.

Council of Science Editors:

Serio J. The Polymerase Associated Factor Complex-Protein Arginine Methyltransferase Axis: A Mechanism of Acute Leukemia. [Doctoral Dissertation]. University of Michigan; 2017. Available from: http://hdl.handle.net/2027.42/140915


University of Michigan

4. Bradford, Shayna. Developing Novel Therapeutics for Chronic Kidney Disease.

Degree: PhD, Molecular & Cellular Pathology, 2019, University of Michigan

 Chronic Kidney Disease (CKD) is a major global health burden. In the United States alone, roughly 30 million Americans have CKD. Each year in the… (more)

Subjects/Keywords: Drug Discovery; HTS (High-Throughput Screening); Kidney Fibrosis; BMP (Bone Morphogenetic Protein); PKD (Polycystic Kidney Disease); Kidney Cancer; Pax2 (Paired-homeobox 2); Biological Chemistry; Genetics; Molecular, Cellular and Developmental Biology; Science (General); Science

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APA (6th Edition):

Bradford, S. (2019). Developing Novel Therapeutics for Chronic Kidney Disease. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/150009

Chicago Manual of Style (16th Edition):

Bradford, Shayna. “Developing Novel Therapeutics for Chronic Kidney Disease.” 2019. Doctoral Dissertation, University of Michigan. Accessed January 23, 2020. http://hdl.handle.net/2027.42/150009.

MLA Handbook (7th Edition):

Bradford, Shayna. “Developing Novel Therapeutics for Chronic Kidney Disease.” 2019. Web. 23 Jan 2020.

Vancouver:

Bradford S. Developing Novel Therapeutics for Chronic Kidney Disease. [Internet] [Doctoral dissertation]. University of Michigan; 2019. [cited 2020 Jan 23]. Available from: http://hdl.handle.net/2027.42/150009.

Council of Science Editors:

Bradford S. Developing Novel Therapeutics for Chronic Kidney Disease. [Doctoral Dissertation]. University of Michigan; 2019. Available from: http://hdl.handle.net/2027.42/150009


University of Michigan

5. Zhang, Yajia. Discovery and Characterization of Non-Coding RNAs with Therapeutic and Diagnostic Potential in Prostate Cancer.

Degree: PhD, Molecular & Cellular Pathology, 2019, University of Michigan

 Prostate cancer is the most common malignancy and the second leading cause of death in American men. Primary prostate cancer is often hormone-dependent and relies… (more)

Subjects/Keywords: non-coding RNA; prostate cancer; Molecular, Cellular and Developmental Biology; Pathology; Health Sciences

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APA (6th Edition):

Zhang, Y. (2019). Discovery and Characterization of Non-Coding RNAs with Therapeutic and Diagnostic Potential in Prostate Cancer. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/151655

Chicago Manual of Style (16th Edition):

Zhang, Yajia. “Discovery and Characterization of Non-Coding RNAs with Therapeutic and Diagnostic Potential in Prostate Cancer.” 2019. Doctoral Dissertation, University of Michigan. Accessed January 23, 2020. http://hdl.handle.net/2027.42/151655.

MLA Handbook (7th Edition):

Zhang, Yajia. “Discovery and Characterization of Non-Coding RNAs with Therapeutic and Diagnostic Potential in Prostate Cancer.” 2019. Web. 23 Jan 2020.

Vancouver:

Zhang Y. Discovery and Characterization of Non-Coding RNAs with Therapeutic and Diagnostic Potential in Prostate Cancer. [Internet] [Doctoral dissertation]. University of Michigan; 2019. [cited 2020 Jan 23]. Available from: http://hdl.handle.net/2027.42/151655.

Council of Science Editors:

Zhang Y. Discovery and Characterization of Non-Coding RNAs with Therapeutic and Diagnostic Potential in Prostate Cancer. [Doctoral Dissertation]. University of Michigan; 2019. Available from: http://hdl.handle.net/2027.42/151655

6. Sahu, Anirban. The Role of Long Noncoding RNA SChLAP1 in Prostate Cancer.

Degree: PhD, Molecular & Cellular Path PhD, 2017, University of Michigan

 Prostate cancer is the most common malignancy in U.S. men, accounting for nearly 30,000 deaths annually. While the majority of prostate cancers are indolent, a… (more)

Subjects/Keywords: prostate cancer; long noncoding RNA (lncRNA); SChLAP1; SWI/SNF nucleosome-remodeling complex; Polycomb Repressive Complex 2 (PRC2); Genetics; Molecular, Cellular and Developmental Biology; Oncology and Hematology; Pathology; Health Sciences; Science

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APA (6th Edition):

Sahu, A. (2017). The Role of Long Noncoding RNA SChLAP1 in Prostate Cancer. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/137075

Chicago Manual of Style (16th Edition):

Sahu, Anirban. “The Role of Long Noncoding RNA SChLAP1 in Prostate Cancer.” 2017. Doctoral Dissertation, University of Michigan. Accessed January 23, 2020. http://hdl.handle.net/2027.42/137075.

MLA Handbook (7th Edition):

Sahu, Anirban. “The Role of Long Noncoding RNA SChLAP1 in Prostate Cancer.” 2017. Web. 23 Jan 2020.

Vancouver:

Sahu A. The Role of Long Noncoding RNA SChLAP1 in Prostate Cancer. [Internet] [Doctoral dissertation]. University of Michigan; 2017. [cited 2020 Jan 23]. Available from: http://hdl.handle.net/2027.42/137075.

Council of Science Editors:

Sahu A. The Role of Long Noncoding RNA SChLAP1 in Prostate Cancer. [Doctoral Dissertation]. University of Michigan; 2017. Available from: http://hdl.handle.net/2027.42/137075


University of Michigan

7. Yu, Ting. Niemann-Pick Type C Disease: Molecular Mechanisms of Neurodegeneration and Targets for Therapeutic Intervention.

Degree: PhD, Molecular & Cellular Pathology, 2013, University of Michigan

 Niemann-Pick Type C disease (NPC) is a childhood-onset neurodegenerative disorder characterized by the accumulation of unesterified cholesterol and glycosphingolipids in late endosomes and lysosomes. Most… (more)

Subjects/Keywords: Niemann-Pick Type C; Neurodegeneration; Lysosomal Storage Disease; Cell Autonomous; Myelin; Proteostasis; Genetics; Neurosciences; Pathology; Health Sciences

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APA (6th Edition):

Yu, T. (2013). Niemann-Pick Type C Disease: Molecular Mechanisms of Neurodegeneration and Targets for Therapeutic Intervention. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/98048

Chicago Manual of Style (16th Edition):

Yu, Ting. “Niemann-Pick Type C Disease: Molecular Mechanisms of Neurodegeneration and Targets for Therapeutic Intervention.” 2013. Doctoral Dissertation, University of Michigan. Accessed January 23, 2020. http://hdl.handle.net/2027.42/98048.

MLA Handbook (7th Edition):

Yu, Ting. “Niemann-Pick Type C Disease: Molecular Mechanisms of Neurodegeneration and Targets for Therapeutic Intervention.” 2013. Web. 23 Jan 2020.

Vancouver:

Yu T. Niemann-Pick Type C Disease: Molecular Mechanisms of Neurodegeneration and Targets for Therapeutic Intervention. [Internet] [Doctoral dissertation]. University of Michigan; 2013. [cited 2020 Jan 23]. Available from: http://hdl.handle.net/2027.42/98048.

Council of Science Editors:

Yu T. Niemann-Pick Type C Disease: Molecular Mechanisms of Neurodegeneration and Targets for Therapeutic Intervention. [Doctoral Dissertation]. University of Michigan; 2013. Available from: http://hdl.handle.net/2027.42/98048


University of Michigan

8. Moore, Heather Marie. The Role of EZH2 in Breast Cancer Progression and Metastasis.

Degree: PhD, Cellular and Molecular Biology, 2013, University of Michigan

 Understanding how breast cancer cells disseminate and metastasize is essential to develop better treatments and to improve survival. Enhancer of Zeste Homolog 2 (EZH2) is… (more)

Subjects/Keywords: Breast Cancer Progression and Metastasis; EZH2; Polycomb; Pathology; Health Sciences

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APA (6th Edition):

Moore, H. M. (2013). The Role of EZH2 in Breast Cancer Progression and Metastasis. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/100098

Chicago Manual of Style (16th Edition):

Moore, Heather Marie. “The Role of EZH2 in Breast Cancer Progression and Metastasis.” 2013. Doctoral Dissertation, University of Michigan. Accessed January 23, 2020. http://hdl.handle.net/2027.42/100098.

MLA Handbook (7th Edition):

Moore, Heather Marie. “The Role of EZH2 in Breast Cancer Progression and Metastasis.” 2013. Web. 23 Jan 2020.

Vancouver:

Moore HM. The Role of EZH2 in Breast Cancer Progression and Metastasis. [Internet] [Doctoral dissertation]. University of Michigan; 2013. [cited 2020 Jan 23]. Available from: http://hdl.handle.net/2027.42/100098.

Council of Science Editors:

Moore HM. The Role of EZH2 in Breast Cancer Progression and Metastasis. [Doctoral Dissertation]. University of Michigan; 2013. Available from: http://hdl.handle.net/2027.42/100098


University of Michigan

9. Geister, Krista Anne. The Genetic and Molecular Etiologies of Two Spontaneous Mouse Models of Skeletal Dysplasia and Infertility.

Degree: PhD, Cellular & Molecular Biology, 2013, University of Michigan

 High-throughput genotyping and sequencing technologies have stimulated an accelerated pace of Mendelian gene discovery. Forty-one novel genetic causes of skeletal dysplasia have been uncovered in… (more)

Subjects/Keywords: Skeletal Dysplasia; Infertility; Mouse Models of Genetic Disorders; Genetics; Health Sciences

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APA (6th Edition):

Geister, K. A. (2013). The Genetic and Molecular Etiologies of Two Spontaneous Mouse Models of Skeletal Dysplasia and Infertility. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/100101

Chicago Manual of Style (16th Edition):

Geister, Krista Anne. “The Genetic and Molecular Etiologies of Two Spontaneous Mouse Models of Skeletal Dysplasia and Infertility.” 2013. Doctoral Dissertation, University of Michigan. Accessed January 23, 2020. http://hdl.handle.net/2027.42/100101.

MLA Handbook (7th Edition):

Geister, Krista Anne. “The Genetic and Molecular Etiologies of Two Spontaneous Mouse Models of Skeletal Dysplasia and Infertility.” 2013. Web. 23 Jan 2020.

Vancouver:

Geister KA. The Genetic and Molecular Etiologies of Two Spontaneous Mouse Models of Skeletal Dysplasia and Infertility. [Internet] [Doctoral dissertation]. University of Michigan; 2013. [cited 2020 Jan 23]. Available from: http://hdl.handle.net/2027.42/100101.

Council of Science Editors:

Geister KA. The Genetic and Molecular Etiologies of Two Spontaneous Mouse Models of Skeletal Dysplasia and Infertility. [Doctoral Dissertation]. University of Michigan; 2013. Available from: http://hdl.handle.net/2027.42/100101


University of Michigan

10. Menon, Tushar. Regulation of Androgen-Responsive Transcription by the Chromatin Remodeling Enzyme CHD8.

Degree: PhD, Biological Chemistry, 2010, University of Michigan

 Eukaryotic DNA is packaged into a highly condensed chromatin state, which inherently serves as a barrier to critical cellular processes such as DNA replication, repair… (more)

Subjects/Keywords: CHD8; Androgen Receptor; Biological Chemistry; Science

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APA (6th Edition):

Menon, T. (2010). Regulation of Androgen-Responsive Transcription by the Chromatin Remodeling Enzyme CHD8. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/75964

Chicago Manual of Style (16th Edition):

Menon, Tushar. “Regulation of Androgen-Responsive Transcription by the Chromatin Remodeling Enzyme CHD8.” 2010. Doctoral Dissertation, University of Michigan. Accessed January 23, 2020. http://hdl.handle.net/2027.42/75964.

MLA Handbook (7th Edition):

Menon, Tushar. “Regulation of Androgen-Responsive Transcription by the Chromatin Remodeling Enzyme CHD8.” 2010. Web. 23 Jan 2020.

Vancouver:

Menon T. Regulation of Androgen-Responsive Transcription by the Chromatin Remodeling Enzyme CHD8. [Internet] [Doctoral dissertation]. University of Michigan; 2010. [cited 2020 Jan 23]. Available from: http://hdl.handle.net/2027.42/75964.

Council of Science Editors:

Menon T. Regulation of Androgen-Responsive Transcription by the Chromatin Remodeling Enzyme CHD8. [Doctoral Dissertation]. University of Michigan; 2010. Available from: http://hdl.handle.net/2027.42/75964


University of Michigan

11. Schaefer, Stacy. Otic Regeneration and Development: Advancement of Stem Cell-Based Methodology for In Vitro Modeling of Mammalian Inner Ear Sensory Epithelia.

Degree: PhD, Neuroscience, 2018, University of Michigan

 Hearing loss treatments have improved significantly with the advent of cochlear implants and advancement of hearing aids. Still, they fall short of full restoration of… (more)

Subjects/Keywords: Developmental biology; Stem cells; Inner ear; Organoids; Regeneration; Neuroscience; Molecular, Cellular and Developmental Biology; Science (General); Science

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APA (6th Edition):

Schaefer, S. (2018). Otic Regeneration and Development: Advancement of Stem Cell-Based Methodology for In Vitro Modeling of Mammalian Inner Ear Sensory Epithelia. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/144011

Chicago Manual of Style (16th Edition):

Schaefer, Stacy. “Otic Regeneration and Development: Advancement of Stem Cell-Based Methodology for In Vitro Modeling of Mammalian Inner Ear Sensory Epithelia.” 2018. Doctoral Dissertation, University of Michigan. Accessed January 23, 2020. http://hdl.handle.net/2027.42/144011.

MLA Handbook (7th Edition):

Schaefer, Stacy. “Otic Regeneration and Development: Advancement of Stem Cell-Based Methodology for In Vitro Modeling of Mammalian Inner Ear Sensory Epithelia.” 2018. Web. 23 Jan 2020.

Vancouver:

Schaefer S. Otic Regeneration and Development: Advancement of Stem Cell-Based Methodology for In Vitro Modeling of Mammalian Inner Ear Sensory Epithelia. [Internet] [Doctoral dissertation]. University of Michigan; 2018. [cited 2020 Jan 23]. Available from: http://hdl.handle.net/2027.42/144011.

Council of Science Editors:

Schaefer S. Otic Regeneration and Development: Advancement of Stem Cell-Based Methodology for In Vitro Modeling of Mammalian Inner Ear Sensory Epithelia. [Doctoral Dissertation]. University of Michigan; 2018. Available from: http://hdl.handle.net/2027.42/144011

12. Broekema, Nicole Marie. Determinants of Archetype BK Polyomavirus Replication.

Degree: PhD, Microbiology & Immunology, 2013, University of Michigan

 BK polyomavirus (BKPyV) is a widespread, small double-stranded DNA virus that is an emerging pathogen in immunocompromised individuals. Following an initial asymptomatic infection, it establishes… (more)

Subjects/Keywords: BK Polyomavirus; Archetype Virus; Microbiology and Immunology; Science

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APA (6th Edition):

Broekema, N. M. (2013). Determinants of Archetype BK Polyomavirus Replication. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/97966

Chicago Manual of Style (16th Edition):

Broekema, Nicole Marie. “Determinants of Archetype BK Polyomavirus Replication.” 2013. Doctoral Dissertation, University of Michigan. Accessed January 23, 2020. http://hdl.handle.net/2027.42/97966.

MLA Handbook (7th Edition):

Broekema, Nicole Marie. “Determinants of Archetype BK Polyomavirus Replication.” 2013. Web. 23 Jan 2020.

Vancouver:

Broekema NM. Determinants of Archetype BK Polyomavirus Replication. [Internet] [Doctoral dissertation]. University of Michigan; 2013. [cited 2020 Jan 23]. Available from: http://hdl.handle.net/2027.42/97966.

Council of Science Editors:

Broekema NM. Determinants of Archetype BK Polyomavirus Replication. [Doctoral Dissertation]. University of Michigan; 2013. Available from: http://hdl.handle.net/2027.42/97966

13. Ozkurede, Varol Ulas. Improved Mitochondrial Stress Response in Long-lived Snell Dwarf Mice.

Degree: PhD, Molecular & Cellular Pathology, 2018, University of Michigan

 Upregulation of the mitochondrial unfolded protein response (mtUPR) as a result of alterations in mitochondrial protein stoichiometry has been proposed as a common pathway in… (more)

Subjects/Keywords: Aging; Mitochondrial Stress Response; mtUPR; Longevity; Lifespan Extension; Snell Dwarf Mice; Geriatrics; Molecular, Cellular and Developmental Biology; Pathology; Health Sciences; Science

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APA (6th Edition):

Ozkurede, V. U. (2018). Improved Mitochondrial Stress Response in Long-lived Snell Dwarf Mice. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/147485

Chicago Manual of Style (16th Edition):

Ozkurede, Varol Ulas. “Improved Mitochondrial Stress Response in Long-lived Snell Dwarf Mice.” 2018. Doctoral Dissertation, University of Michigan. Accessed January 23, 2020. http://hdl.handle.net/2027.42/147485.

MLA Handbook (7th Edition):

Ozkurede, Varol Ulas. “Improved Mitochondrial Stress Response in Long-lived Snell Dwarf Mice.” 2018. Web. 23 Jan 2020.

Vancouver:

Ozkurede VU. Improved Mitochondrial Stress Response in Long-lived Snell Dwarf Mice. [Internet] [Doctoral dissertation]. University of Michigan; 2018. [cited 2020 Jan 23]. Available from: http://hdl.handle.net/2027.42/147485.

Council of Science Editors:

Ozkurede VU. Improved Mitochondrial Stress Response in Long-lived Snell Dwarf Mice. [Doctoral Dissertation]. University of Michigan; 2018. Available from: http://hdl.handle.net/2027.42/147485

14. Zhang, Peng. Mechanisms and Regulation of Transforming Growth Factor Superfamily Mediated Gene Expression.

Degree: PhD, Molecular & Cellular Pathology, 2012, University of Michigan

 The TGF-beta superfamily, including TGF-betas and BMPs, is critical for normal embryonic development, as well as disease progression, and is tightly regulated both within and… (more)

Subjects/Keywords: TGF-beta; Epithelial Mesenchymal Transition; Gene Regulation; Wnt Signaling; Groucho Proteins; Renal Fibrosis; Pathology; Health Sciences

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APA (6th Edition):

Zhang, P. (2012). Mechanisms and Regulation of Transforming Growth Factor Superfamily Mediated Gene Expression. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/96131

Chicago Manual of Style (16th Edition):

Zhang, Peng. “Mechanisms and Regulation of Transforming Growth Factor Superfamily Mediated Gene Expression.” 2012. Doctoral Dissertation, University of Michigan. Accessed January 23, 2020. http://hdl.handle.net/2027.42/96131.

MLA Handbook (7th Edition):

Zhang, Peng. “Mechanisms and Regulation of Transforming Growth Factor Superfamily Mediated Gene Expression.” 2012. Web. 23 Jan 2020.

Vancouver:

Zhang P. Mechanisms and Regulation of Transforming Growth Factor Superfamily Mediated Gene Expression. [Internet] [Doctoral dissertation]. University of Michigan; 2012. [cited 2020 Jan 23]. Available from: http://hdl.handle.net/2027.42/96131.

Council of Science Editors:

Zhang P. Mechanisms and Regulation of Transforming Growth Factor Superfamily Mediated Gene Expression. [Doctoral Dissertation]. University of Michigan; 2012. Available from: http://hdl.handle.net/2027.42/96131

15. Lobo, Neethan A. Analysis of Murine Mammary Epithelial Cellular Hierarchy.

Degree: PhD, Cellular & Molecular Biology, 2009, University of Michigan

 The murine mammary system is a complex milieu of epithelial cell types that function together to support lactogenesis. The breast tissue is spatially and temporally… (more)

Subjects/Keywords: Stem Cell; Self-renewal; TGF Beta; Microarray; Differentiation; Mammary; Molecular, Cellular and Developmental Biology; Science

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APA (6th Edition):

Lobo, N. A. (2009). Analysis of Murine Mammary Epithelial Cellular Hierarchy. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/63787

Chicago Manual of Style (16th Edition):

Lobo, Neethan A. “Analysis of Murine Mammary Epithelial Cellular Hierarchy.” 2009. Doctoral Dissertation, University of Michigan. Accessed January 23, 2020. http://hdl.handle.net/2027.42/63787.

MLA Handbook (7th Edition):

Lobo, Neethan A. “Analysis of Murine Mammary Epithelial Cellular Hierarchy.” 2009. Web. 23 Jan 2020.

Vancouver:

Lobo NA. Analysis of Murine Mammary Epithelial Cellular Hierarchy. [Internet] [Doctoral dissertation]. University of Michigan; 2009. [cited 2020 Jan 23]. Available from: http://hdl.handle.net/2027.42/63787.

Council of Science Editors:

Lobo NA. Analysis of Murine Mammary Epithelial Cellular Hierarchy. [Doctoral Dissertation]. University of Michigan; 2009. Available from: http://hdl.handle.net/2027.42/63787

16. Tan, Jiaying. Leukemogenic Mechanisms of MLL Fusion Proteins.

Degree: PhD, Molecular & Cellular Pathology, 2012, University of Michigan

 Translocations that generate MLL fusion proteins are common causes of human acute leukemias. Aberrant target gene activation is the primary driver of MLL-rearranged leukemogenesis, but… (more)

Subjects/Keywords: Mixed Lineage Leukemia; CBX8; Polycomb-group Proteins; Science (General); Science

…after the final replating. All animal studies were approved by the University of Michigan… …University of Michigan High-Throughput Protein Lab. Bacteria was grown at 37 degrees Celsius in TB… 

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APA (6th Edition):

Tan, J. (2012). Leukemogenic Mechanisms of MLL Fusion Proteins. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/91545

Chicago Manual of Style (16th Edition):

Tan, Jiaying. “Leukemogenic Mechanisms of MLL Fusion Proteins.” 2012. Doctoral Dissertation, University of Michigan. Accessed January 23, 2020. http://hdl.handle.net/2027.42/91545.

MLA Handbook (7th Edition):

Tan, Jiaying. “Leukemogenic Mechanisms of MLL Fusion Proteins.” 2012. Web. 23 Jan 2020.

Vancouver:

Tan J. Leukemogenic Mechanisms of MLL Fusion Proteins. [Internet] [Doctoral dissertation]. University of Michigan; 2012. [cited 2020 Jan 23]. Available from: http://hdl.handle.net/2027.42/91545.

Council of Science Editors:

Tan J. Leukemogenic Mechanisms of MLL Fusion Proteins. [Doctoral Dissertation]. University of Michigan; 2012. Available from: http://hdl.handle.net/2027.42/91545

17. Staubach Grosse, Ann Marie. Evidence for a New Model of Intestinal Morphogenesis.

Degree: PhD, Cellular & Molecular Biology, 2011, University of Michigan

 During vertebrate intestinal development, coordinated morphogenetic movements between E12.5 and E16.5 transform the epithelial layer from a flat surface into evaginating villi and simultaneously generate… (more)

Subjects/Keywords: Intestinal Development; Lumen Formation; Epithelial Remodeling; Mouse Morphogenesis; Pseudostratified Epithelium; Intestinal Lengthening; Molecular, Cellular and Developmental Biology; Science

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APA (6th Edition):

Staubach Grosse, A. M. (2011). Evidence for a New Model of Intestinal Morphogenesis. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/86275

Chicago Manual of Style (16th Edition):

Staubach Grosse, Ann Marie. “Evidence for a New Model of Intestinal Morphogenesis.” 2011. Doctoral Dissertation, University of Michigan. Accessed January 23, 2020. http://hdl.handle.net/2027.42/86275.

MLA Handbook (7th Edition):

Staubach Grosse, Ann Marie. “Evidence for a New Model of Intestinal Morphogenesis.” 2011. Web. 23 Jan 2020.

Vancouver:

Staubach Grosse AM. Evidence for a New Model of Intestinal Morphogenesis. [Internet] [Doctoral dissertation]. University of Michigan; 2011. [cited 2020 Jan 23]. Available from: http://hdl.handle.net/2027.42/86275.

Council of Science Editors:

Staubach Grosse AM. Evidence for a New Model of Intestinal Morphogenesis. [Doctoral Dissertation]. University of Michigan; 2011. Available from: http://hdl.handle.net/2027.42/86275

18. Monroe, Sara C. Mechanisms of MLL Fusion Protein-Mediated Leukemic Transformation.

Degree: PhD, Molecular & Cellular Pathology, 2010, University of Michigan

 Leukemogenic MLL fusion proteins, including MLL-AF4, MLL-AF9, and MLL-ENL, transform through up regulated expression of HOX genes and the HOX cofactor MEIS1. How they lead… (more)

Subjects/Keywords: MLL; Leukemia; HOX; AF9; ENL; Molecular, Cellular and Developmental Biology; Health Sciences

…strainer caps, and flow cytometry was performed at the University of Michigan Flow Cytometry Core… …sorted at the University of Michigan Flow Cytometry Core one week after transduction. Cell… 

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APA (6th Edition):

Monroe, S. C. (2010). Mechanisms of MLL Fusion Protein-Mediated Leukemic Transformation. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/78966

Chicago Manual of Style (16th Edition):

Monroe, Sara C. “Mechanisms of MLL Fusion Protein-Mediated Leukemic Transformation.” 2010. Doctoral Dissertation, University of Michigan. Accessed January 23, 2020. http://hdl.handle.net/2027.42/78966.

MLA Handbook (7th Edition):

Monroe, Sara C. “Mechanisms of MLL Fusion Protein-Mediated Leukemic Transformation.” 2010. Web. 23 Jan 2020.

Vancouver:

Monroe SC. Mechanisms of MLL Fusion Protein-Mediated Leukemic Transformation. [Internet] [Doctoral dissertation]. University of Michigan; 2010. [cited 2020 Jan 23]. Available from: http://hdl.handle.net/2027.42/78966.

Council of Science Editors:

Monroe SC. Mechanisms of MLL Fusion Protein-Mediated Leukemic Transformation. [Doctoral Dissertation]. University of Michigan; 2010. Available from: http://hdl.handle.net/2027.42/78966

19. Waite, Mindy Rachelle. Pleiotropic and Isoform-Specific Functions of PITX2 in Brain Development.

Degree: PhD, Cellular & Molecular Biology, 2012, University of Michigan

 Neuronal diversification in the developing brain is a coordinated process requiring complex genetic regulation. Transcriptional control of gene expression is known to regulate proliferation, migration,… (more)

Subjects/Keywords: Transcription Factor Control of Brain Development; Differentiation; Migration; Molecular, Cellular and Developmental Biology; Health Sciences

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APA (6th Edition):

Waite, M. R. (2012). Pleiotropic and Isoform-Specific Functions of PITX2 in Brain Development. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/94088

Chicago Manual of Style (16th Edition):

Waite, Mindy Rachelle. “Pleiotropic and Isoform-Specific Functions of PITX2 in Brain Development.” 2012. Doctoral Dissertation, University of Michigan. Accessed January 23, 2020. http://hdl.handle.net/2027.42/94088.

MLA Handbook (7th Edition):

Waite, Mindy Rachelle. “Pleiotropic and Isoform-Specific Functions of PITX2 in Brain Development.” 2012. Web. 23 Jan 2020.

Vancouver:

Waite MR. Pleiotropic and Isoform-Specific Functions of PITX2 in Brain Development. [Internet] [Doctoral dissertation]. University of Michigan; 2012. [cited 2020 Jan 23]. Available from: http://hdl.handle.net/2027.42/94088.

Council of Science Editors:

Waite MR. Pleiotropic and Isoform-Specific Functions of PITX2 in Brain Development. [Doctoral Dissertation]. University of Michigan; 2012. Available from: http://hdl.handle.net/2027.42/94088

20. Grimley, Edward. Toward Molecularly Targeted Therapies for Renal Disease.

Degree: PhD, Molecular & Cellular Pathology, 2017, University of Michigan

 Pax2 is a developmental control gene that is essential for organogenesis of the kidney and urogenital tract. Genes of this nature are generally suppressed in… (more)

Subjects/Keywords: Pax2; paired domain; drug discovery; high-throughput screen; virtual screen; kidney disease; Biological Chemistry; Genetics; Molecular, Cellular and Developmental Biology; Science (General); Science

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APA (6th Edition):

Grimley, E. (2017). Toward Molecularly Targeted Therapies for Renal Disease. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/138499

Chicago Manual of Style (16th Edition):

Grimley, Edward. “Toward Molecularly Targeted Therapies for Renal Disease.” 2017. Doctoral Dissertation, University of Michigan. Accessed January 23, 2020. http://hdl.handle.net/2027.42/138499.

MLA Handbook (7th Edition):

Grimley, Edward. “Toward Molecularly Targeted Therapies for Renal Disease.” 2017. Web. 23 Jan 2020.

Vancouver:

Grimley E. Toward Molecularly Targeted Therapies for Renal Disease. [Internet] [Doctoral dissertation]. University of Michigan; 2017. [cited 2020 Jan 23]. Available from: http://hdl.handle.net/2027.42/138499.

Council of Science Editors:

Grimley E. Toward Molecularly Targeted Therapies for Renal Disease. [Doctoral Dissertation]. University of Michigan; 2017. Available from: http://hdl.handle.net/2027.42/138499

21. Udager, Aaron Mark. Cell-specific Gene Expression: Pylorus Morphogenesis and Hedgehog-regulated Enhancers.

Degree: PhD, Cell and Developmental Biology, 2012, University of Michigan

 The precise spatiotemporal control of gene expression is integral to the survival of all organisms. Inappropriate gene expression can lead to developmental defects in newborns,… (more)

Subjects/Keywords: Computational Biology; Intestine Development; Hedgehog Signaling; Pyloric Sphincter; Gata3; Enhancers; Molecular, Cellular and Developmental Biology; Health Sciences

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APA (6th Edition):

Udager, A. M. (2012). Cell-specific Gene Expression: Pylorus Morphogenesis and Hedgehog-regulated Enhancers. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/91524

Chicago Manual of Style (16th Edition):

Udager, Aaron Mark. “Cell-specific Gene Expression: Pylorus Morphogenesis and Hedgehog-regulated Enhancers.” 2012. Doctoral Dissertation, University of Michigan. Accessed January 23, 2020. http://hdl.handle.net/2027.42/91524.

MLA Handbook (7th Edition):

Udager, Aaron Mark. “Cell-specific Gene Expression: Pylorus Morphogenesis and Hedgehog-regulated Enhancers.” 2012. Web. 23 Jan 2020.

Vancouver:

Udager AM. Cell-specific Gene Expression: Pylorus Morphogenesis and Hedgehog-regulated Enhancers. [Internet] [Doctoral dissertation]. University of Michigan; 2012. [cited 2020 Jan 23]. Available from: http://hdl.handle.net/2027.42/91524.

Council of Science Editors:

Udager AM. Cell-specific Gene Expression: Pylorus Morphogenesis and Hedgehog-regulated Enhancers. [Doctoral Dissertation]. University of Michigan; 2012. Available from: http://hdl.handle.net/2027.42/91524


University of Michigan

22. Yallowitz, Alisha Ruth. Hox Function in Mammalian Kidney Development.

Degree: PhD, Cell and Developmental Biology, 2009, University of Michigan

 The Hox gene complexes encode an evolutionarily conserved set of transcription factors important for the anteroposterior (AP) patterning of the body plan, and are essential… (more)

Subjects/Keywords: Hox; Kidney; Development; Genetics; Molecular, Cellular and Developmental Biology; Health Sciences

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Yallowitz, A. R. (2009). Hox Function in Mammalian Kidney Development. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/63845

Chicago Manual of Style (16th Edition):

Yallowitz, Alisha Ruth. “Hox Function in Mammalian Kidney Development.” 2009. Doctoral Dissertation, University of Michigan. Accessed January 23, 2020. http://hdl.handle.net/2027.42/63845.

MLA Handbook (7th Edition):

Yallowitz, Alisha Ruth. “Hox Function in Mammalian Kidney Development.” 2009. Web. 23 Jan 2020.

Vancouver:

Yallowitz AR. Hox Function in Mammalian Kidney Development. [Internet] [Doctoral dissertation]. University of Michigan; 2009. [cited 2020 Jan 23]. Available from: http://hdl.handle.net/2027.42/63845.

Council of Science Editors:

Yallowitz AR. Hox Function in Mammalian Kidney Development. [Doctoral Dissertation]. University of Michigan; 2009. Available from: http://hdl.handle.net/2027.42/63845

.