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You searched for +publisher:"University of Manitoba" +contributor:("Nachtigal, Mark"). Showing records 1 – 18 of 18 total matches.

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University of Manitoba

1. Ganguly, Esha. Effect of 5-Aza-2´-Deoxycytidine and Trichostatin A on Endogenous Versus Ectopic Expression of Placental Members of the Human Growth Hormone Gene Family.

Degree: Physiology and Pathophysiology, 2016, University of Manitoba

 Background: The genes coding for human (h) chorionic somatomammotropin (CS), hCS-A and hCS-B, and placental growth hormone (GH-V), hGH-V are located at a single locus… (more)

Subjects/Keywords: 5-Aza-2´-Deoxycytidine; Trichostatin A; Acetylation; DNA Methylation; Placental; GH-CS

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APA (6th Edition):

Ganguly, E. (2016). Effect of 5-Aza-2´-Deoxycytidine and Trichostatin A on Endogenous Versus Ectopic Expression of Placental Members of the Human Growth Hormone Gene Family. (Masters Thesis). University of Manitoba. Retrieved from http://hdl.handle.net/1993/31147

Chicago Manual of Style (16th Edition):

Ganguly, Esha. “Effect of 5-Aza-2´-Deoxycytidine and Trichostatin A on Endogenous Versus Ectopic Expression of Placental Members of the Human Growth Hormone Gene Family.” 2016. Masters Thesis, University of Manitoba. Accessed January 24, 2020. http://hdl.handle.net/1993/31147.

MLA Handbook (7th Edition):

Ganguly, Esha. “Effect of 5-Aza-2´-Deoxycytidine and Trichostatin A on Endogenous Versus Ectopic Expression of Placental Members of the Human Growth Hormone Gene Family.” 2016. Web. 24 Jan 2020.

Vancouver:

Ganguly E. Effect of 5-Aza-2´-Deoxycytidine and Trichostatin A on Endogenous Versus Ectopic Expression of Placental Members of the Human Growth Hormone Gene Family. [Internet] [Masters thesis]. University of Manitoba; 2016. [cited 2020 Jan 24]. Available from: http://hdl.handle.net/1993/31147.

Council of Science Editors:

Ganguly E. Effect of 5-Aza-2´-Deoxycytidine and Trichostatin A on Endogenous Versus Ectopic Expression of Placental Members of the Human Growth Hormone Gene Family. [Masters Thesis]. University of Manitoba; 2016. Available from: http://hdl.handle.net/1993/31147


University of Manitoba

2. McAndrew, Erin N. Exploiting RAD54B-deficiency in colorectal cancer cells through synthetic lethal targeting of PARP1.

Degree: Biochemistry and Medical Genetics, 2016, University of Manitoba

 Colorectal cancer (CRC) is the second leading cause of cancer-related deaths in Canada each year. Currently, most therapeutic approaches target rapidly dividing cancer cells by… (more)

Subjects/Keywords: colorectal cancer; Synthetic lethal; RAD54B; Precision medicine; PARP1

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APA (6th Edition):

McAndrew, E. N. (2016). Exploiting RAD54B-deficiency in colorectal cancer cells through synthetic lethal targeting of PARP1. (Masters Thesis). University of Manitoba. Retrieved from http://hdl.handle.net/1993/31779

Chicago Manual of Style (16th Edition):

McAndrew, Erin N. “Exploiting RAD54B-deficiency in colorectal cancer cells through synthetic lethal targeting of PARP1.” 2016. Masters Thesis, University of Manitoba. Accessed January 24, 2020. http://hdl.handle.net/1993/31779.

MLA Handbook (7th Edition):

McAndrew, Erin N. “Exploiting RAD54B-deficiency in colorectal cancer cells through synthetic lethal targeting of PARP1.” 2016. Web. 24 Jan 2020.

Vancouver:

McAndrew EN. Exploiting RAD54B-deficiency in colorectal cancer cells through synthetic lethal targeting of PARP1. [Internet] [Masters thesis]. University of Manitoba; 2016. [cited 2020 Jan 24]. Available from: http://hdl.handle.net/1993/31779.

Council of Science Editors:

McAndrew EN. Exploiting RAD54B-deficiency in colorectal cancer cells through synthetic lethal targeting of PARP1. [Masters Thesis]. University of Manitoba; 2016. Available from: http://hdl.handle.net/1993/31779


University of Manitoba

3. Blankstein, Anna R. Siramesine and lapatinib induce synergic cell death via a ferroptotic mechanism in lung adenocarcinoma and glioblastoma cells.

Degree: Biochemistry and Medical Genetics, 2017, University of Manitoba

 In cancer cells, the most common forms of cell death are often actively inhibited, contributing to the development of drug resistance. Identifying and exploiting alternative… (more)

Subjects/Keywords: Cell death; Ferroptosis; Glioblastoma; Lung adenocarcinoma; Drug synergy; Drug repurposing

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APA (6th Edition):

Blankstein, A. R. (2017). Siramesine and lapatinib induce synergic cell death via a ferroptotic mechanism in lung adenocarcinoma and glioblastoma cells. (Masters Thesis). University of Manitoba. Retrieved from http://hdl.handle.net/1993/32404

Chicago Manual of Style (16th Edition):

Blankstein, Anna R. “Siramesine and lapatinib induce synergic cell death via a ferroptotic mechanism in lung adenocarcinoma and glioblastoma cells.” 2017. Masters Thesis, University of Manitoba. Accessed January 24, 2020. http://hdl.handle.net/1993/32404.

MLA Handbook (7th Edition):

Blankstein, Anna R. “Siramesine and lapatinib induce synergic cell death via a ferroptotic mechanism in lung adenocarcinoma and glioblastoma cells.” 2017. Web. 24 Jan 2020.

Vancouver:

Blankstein AR. Siramesine and lapatinib induce synergic cell death via a ferroptotic mechanism in lung adenocarcinoma and glioblastoma cells. [Internet] [Masters thesis]. University of Manitoba; 2017. [cited 2020 Jan 24]. Available from: http://hdl.handle.net/1993/32404.

Council of Science Editors:

Blankstein AR. Siramesine and lapatinib induce synergic cell death via a ferroptotic mechanism in lung adenocarcinoma and glioblastoma cells. [Masters Thesis]. University of Manitoba; 2017. Available from: http://hdl.handle.net/1993/32404


University of Manitoba

4. Jahan, Fahmida. Zeb2: A novel regulator of cardiac fibroblast to myofibroblast transition.

Degree: Biochemistry and Medical Genetics, 2014, University of Manitoba

 Cardiac fibroblast to myofibroblast phenoconversion is a critical step during the development of cardiac fibrosis. Myofibroblasts chronically remodel extracellular matrix that results in myocardial stiffening,… (more)

Subjects/Keywords: Zeb2; Meox2; Cardiac fibroblast; Myofibroblast; Phenoconversion; Cardiac fibrosis

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APA (6th Edition):

Jahan, F. (2014). Zeb2: A novel regulator of cardiac fibroblast to myofibroblast transition. (Masters Thesis). University of Manitoba. Retrieved from http://hdl.handle.net/1993/31090

Chicago Manual of Style (16th Edition):

Jahan, Fahmida. “Zeb2: A novel regulator of cardiac fibroblast to myofibroblast transition.” 2014. Masters Thesis, University of Manitoba. Accessed January 24, 2020. http://hdl.handle.net/1993/31090.

MLA Handbook (7th Edition):

Jahan, Fahmida. “Zeb2: A novel regulator of cardiac fibroblast to myofibroblast transition.” 2014. Web. 24 Jan 2020.

Vancouver:

Jahan F. Zeb2: A novel regulator of cardiac fibroblast to myofibroblast transition. [Internet] [Masters thesis]. University of Manitoba; 2014. [cited 2020 Jan 24]. Available from: http://hdl.handle.net/1993/31090.

Council of Science Editors:

Jahan F. Zeb2: A novel regulator of cardiac fibroblast to myofibroblast transition. [Masters Thesis]. University of Manitoba; 2014. Available from: http://hdl.handle.net/1993/31090


University of Manitoba

5. Hasan, S M Naimul. Characterization of mutations and phenotypes associated with HYAL2 deficiency.

Degree: Biochemistry and Medical Genetics, 2017, University of Manitoba

 Hyaluronidase 2 (HYAL2) is an endoglycosidase involved in the cleavage of hyaluronic acid (HA). HA is abundantly present in the extracellular matrix (ECM) and reported… (more)

Subjects/Keywords: Hyaluronidase 2; Hyaluronic acid; Mutation; Extracellular matrix; Cleft palate

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APA (6th Edition):

Hasan, S. M. N. (2017). Characterization of mutations and phenotypes associated with HYAL2 deficiency. (Masters Thesis). University of Manitoba. Retrieved from http://hdl.handle.net/1993/32693

Chicago Manual of Style (16th Edition):

Hasan, S M Naimul. “Characterization of mutations and phenotypes associated with HYAL2 deficiency.” 2017. Masters Thesis, University of Manitoba. Accessed January 24, 2020. http://hdl.handle.net/1993/32693.

MLA Handbook (7th Edition):

Hasan, S M Naimul. “Characterization of mutations and phenotypes associated with HYAL2 deficiency.” 2017. Web. 24 Jan 2020.

Vancouver:

Hasan SMN. Characterization of mutations and phenotypes associated with HYAL2 deficiency. [Internet] [Masters thesis]. University of Manitoba; 2017. [cited 2020 Jan 24]. Available from: http://hdl.handle.net/1993/32693.

Council of Science Editors:

Hasan SMN. Characterization of mutations and phenotypes associated with HYAL2 deficiency. [Masters Thesis]. University of Manitoba; 2017. Available from: http://hdl.handle.net/1993/32693


University of Manitoba

6. Vinith, Ramya. Micro-computed tomography assessment of skeletal structure in a mucopolysaccharidosis IX mouse model.

Degree: Biochemistry and Medical Genetics, 2014, University of Manitoba

 Mucopolysaccharidosis (MPS) IX is a lysosomal storage disorder caused by a deficiency of hyaluronidase 1 (HYAL1). With few patients described, to extend our understanding of… (more)

Subjects/Keywords: Genetics

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APA (6th Edition):

Vinith, R. (2014). Micro-computed tomography assessment of skeletal structure in a mucopolysaccharidosis IX mouse model. (Masters Thesis). University of Manitoba. Retrieved from http://hdl.handle.net/1993/30122

Chicago Manual of Style (16th Edition):

Vinith, Ramya. “Micro-computed tomography assessment of skeletal structure in a mucopolysaccharidosis IX mouse model.” 2014. Masters Thesis, University of Manitoba. Accessed January 24, 2020. http://hdl.handle.net/1993/30122.

MLA Handbook (7th Edition):

Vinith, Ramya. “Micro-computed tomography assessment of skeletal structure in a mucopolysaccharidosis IX mouse model.” 2014. Web. 24 Jan 2020.

Vancouver:

Vinith R. Micro-computed tomography assessment of skeletal structure in a mucopolysaccharidosis IX mouse model. [Internet] [Masters thesis]. University of Manitoba; 2014. [cited 2020 Jan 24]. Available from: http://hdl.handle.net/1993/30122.

Council of Science Editors:

Vinith R. Micro-computed tomography assessment of skeletal structure in a mucopolysaccharidosis IX mouse model. [Masters Thesis]. University of Manitoba; 2014. Available from: http://hdl.handle.net/1993/30122


University of Manitoba

7. Bungsy, Manisha. Determining the impact diminished RBX1 expression has on chromosome stability in cancer.

Degree: Biochemistry and Medical Genetics, 2019, University of Manitoba

 Chromosome instability (CIN) is an aberrant phenotype observed in nearly all cancer types including colorectal cancer and potentially, high-grade serous ovarian cancer (HGSOC). CIN is… (more)

Subjects/Keywords: Biochemistry; Medical Genetics; Ovarian Cancer

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APA (6th Edition):

Bungsy, M. (2019). Determining the impact diminished RBX1 expression has on chromosome stability in cancer. (Masters Thesis). University of Manitoba. Retrieved from http://hdl.handle.net/1993/34482

Chicago Manual of Style (16th Edition):

Bungsy, Manisha. “Determining the impact diminished RBX1 expression has on chromosome stability in cancer.” 2019. Masters Thesis, University of Manitoba. Accessed January 24, 2020. http://hdl.handle.net/1993/34482.

MLA Handbook (7th Edition):

Bungsy, Manisha. “Determining the impact diminished RBX1 expression has on chromosome stability in cancer.” 2019. Web. 24 Jan 2020.

Vancouver:

Bungsy M. Determining the impact diminished RBX1 expression has on chromosome stability in cancer. [Internet] [Masters thesis]. University of Manitoba; 2019. [cited 2020 Jan 24]. Available from: http://hdl.handle.net/1993/34482.

Council of Science Editors:

Bungsy M. Determining the impact diminished RBX1 expression has on chromosome stability in cancer. [Masters Thesis]. University of Manitoba; 2019. Available from: http://hdl.handle.net/1993/34482


University of Manitoba

8. Vakili-Tajareh, Hana. Regulation of human pituitary growth hormone gene (hGH1) expression by energy homeostasis.

Degree: Physiology and Pathophysiology, 2011, University of Manitoba

 Human (h) growth hormone (GH) levels decline rapidly in response to excess caloric intake before there is any evidence of obesity. In this thesis, the… (more)

Subjects/Keywords: Growth Hormone; Gene regulation; Excess caloric intake; Physical activity

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APA (6th Edition):

Vakili-Tajareh, H. (2011). Regulation of human pituitary growth hormone gene (hGH1) expression by energy homeostasis. (Thesis). University of Manitoba. Retrieved from http://hdl.handle.net/1993/30395

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Vakili-Tajareh, Hana. “Regulation of human pituitary growth hormone gene (hGH1) expression by energy homeostasis.” 2011. Thesis, University of Manitoba. Accessed January 24, 2020. http://hdl.handle.net/1993/30395.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Vakili-Tajareh, Hana. “Regulation of human pituitary growth hormone gene (hGH1) expression by energy homeostasis.” 2011. Web. 24 Jan 2020.

Vancouver:

Vakili-Tajareh H. Regulation of human pituitary growth hormone gene (hGH1) expression by energy homeostasis. [Internet] [Thesis]. University of Manitoba; 2011. [cited 2020 Jan 24]. Available from: http://hdl.handle.net/1993/30395.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Vakili-Tajareh H. Regulation of human pituitary growth hormone gene (hGH1) expression by energy homeostasis. [Thesis]. University of Manitoba; 2011. Available from: http://hdl.handle.net/1993/30395

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Manitoba

9. Murali, Megha. Elucidating the effect of sex on the metabolic and vascular perturbations induced by the absence of adiponectin.

Degree: Biochemistry and Medical Genetics, 2016, University of Manitoba

 Adiponectin is an abundant hormone secreted by adipocytes that exhibits anti-diabetic, anti-inflammatory and anti-atherogenic properties. However, in obesity, as adipocytes enlarge, adiponectin secretion declines. A… (more)

Subjects/Keywords: Adiponectin

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APA (6th Edition):

Murali, M. (2016). Elucidating the effect of sex on the metabolic and vascular perturbations induced by the absence of adiponectin. (Masters Thesis). University of Manitoba. Retrieved from http://hdl.handle.net/1993/31788

Chicago Manual of Style (16th Edition):

Murali, Megha. “Elucidating the effect of sex on the metabolic and vascular perturbations induced by the absence of adiponectin.” 2016. Masters Thesis, University of Manitoba. Accessed January 24, 2020. http://hdl.handle.net/1993/31788.

MLA Handbook (7th Edition):

Murali, Megha. “Elucidating the effect of sex on the metabolic and vascular perturbations induced by the absence of adiponectin.” 2016. Web. 24 Jan 2020.

Vancouver:

Murali M. Elucidating the effect of sex on the metabolic and vascular perturbations induced by the absence of adiponectin. [Internet] [Masters thesis]. University of Manitoba; 2016. [cited 2020 Jan 24]. Available from: http://hdl.handle.net/1993/31788.

Council of Science Editors:

Murali M. Elucidating the effect of sex on the metabolic and vascular perturbations induced by the absence of adiponectin. [Masters Thesis]. University of Manitoba; 2016. Available from: http://hdl.handle.net/1993/31788


University of Manitoba

10. Liang, Lisa. Exploring Novel Drug Treatments for Chemotherapy Resistance In Human Epithelial Ovarian Cancer (EOC).

Degree: Biochemistry and Medical Genetics, 2016, University of Manitoba

 Chemotherapy resistance in human epithelial ovarian cancer (EOC) is a significant reason for the high rate of death among patients. We hypothesized that chemotherapy- resistant… (more)

Subjects/Keywords: Exploring; Novel; Drug; Treatments; Chemotherapy; Resistance; Human; Epithelial; Ovarian; Cancer

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APA (6th Edition):

Liang, L. (2016). Exploring Novel Drug Treatments for Chemotherapy Resistance In Human Epithelial Ovarian Cancer (EOC). (Masters Thesis). University of Manitoba. Retrieved from http://hdl.handle.net/1993/31623

Chicago Manual of Style (16th Edition):

Liang, Lisa. “Exploring Novel Drug Treatments for Chemotherapy Resistance In Human Epithelial Ovarian Cancer (EOC).” 2016. Masters Thesis, University of Manitoba. Accessed January 24, 2020. http://hdl.handle.net/1993/31623.

MLA Handbook (7th Edition):

Liang, Lisa. “Exploring Novel Drug Treatments for Chemotherapy Resistance In Human Epithelial Ovarian Cancer (EOC).” 2016. Web. 24 Jan 2020.

Vancouver:

Liang L. Exploring Novel Drug Treatments for Chemotherapy Resistance In Human Epithelial Ovarian Cancer (EOC). [Internet] [Masters thesis]. University of Manitoba; 2016. [cited 2020 Jan 24]. Available from: http://hdl.handle.net/1993/31623.

Council of Science Editors:

Liang L. Exploring Novel Drug Treatments for Chemotherapy Resistance In Human Epithelial Ovarian Cancer (EOC). [Masters Thesis]. University of Manitoba; 2016. Available from: http://hdl.handle.net/1993/31623


University of Manitoba

11. Thompson, Laura. Microscopy-based high-content analyses identify novel chromosome instability genes including SKP1.

Degree: Biochemistry and Medical Genetics, 2018, University of Manitoba

 Cancer is a devastating disease responsible for ~80,000 deaths in Canada each year. Chromosome instability (CIN) is an abnormal phenotype frequently observed in cancer, characterized… (more)

Subjects/Keywords: Chromosome instability; Cancer; Fluorescence microscopy; Genetics; SKP1

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APA (6th Edition):

Thompson, L. (2018). Microscopy-based high-content analyses identify novel chromosome instability genes including SKP1. (Thesis). University of Manitoba. Retrieved from http://hdl.handle.net/1993/33282

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Thompson, Laura. “Microscopy-based high-content analyses identify novel chromosome instability genes including SKP1.” 2018. Thesis, University of Manitoba. Accessed January 24, 2020. http://hdl.handle.net/1993/33282.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Thompson, Laura. “Microscopy-based high-content analyses identify novel chromosome instability genes including SKP1.” 2018. Web. 24 Jan 2020.

Vancouver:

Thompson L. Microscopy-based high-content analyses identify novel chromosome instability genes including SKP1. [Internet] [Thesis]. University of Manitoba; 2018. [cited 2020 Jan 24]. Available from: http://hdl.handle.net/1993/33282.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Thompson L. Microscopy-based high-content analyses identify novel chromosome instability genes including SKP1. [Thesis]. University of Manitoba; 2018. Available from: http://hdl.handle.net/1993/33282

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Manitoba

12. Kaur, Ravinder. Neural derivatives from human embryonic stem cells: a cellular and molecular model for studying the role of orthodenticle homeobox2 in medulloblastoma progression.

Degree: Biochemistry and Medical Genetics, 2015, University of Manitoba

 Medulloblastoma (MB) is the most common malignant primary pediatric brain tumor and is divided into 4 subtypes based on different genomic alterations and gene expression… (more)

Subjects/Keywords: Human embryonic stem cells; Neural precursors; Medulloblastoma; OTX2

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APA (6th Edition):

Kaur, R. (2015). Neural derivatives from human embryonic stem cells: a cellular and molecular model for studying the role of orthodenticle homeobox2 in medulloblastoma progression. (Thesis). University of Manitoba. Retrieved from http://hdl.handle.net/1993/31914

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kaur, Ravinder. “Neural derivatives from human embryonic stem cells: a cellular and molecular model for studying the role of orthodenticle homeobox2 in medulloblastoma progression.” 2015. Thesis, University of Manitoba. Accessed January 24, 2020. http://hdl.handle.net/1993/31914.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kaur, Ravinder. “Neural derivatives from human embryonic stem cells: a cellular and molecular model for studying the role of orthodenticle homeobox2 in medulloblastoma progression.” 2015. Web. 24 Jan 2020.

Vancouver:

Kaur R. Neural derivatives from human embryonic stem cells: a cellular and molecular model for studying the role of orthodenticle homeobox2 in medulloblastoma progression. [Internet] [Thesis]. University of Manitoba; 2015. [cited 2020 Jan 24]. Available from: http://hdl.handle.net/1993/31914.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kaur R. Neural derivatives from human embryonic stem cells: a cellular and molecular model for studying the role of orthodenticle homeobox2 in medulloblastoma progression. [Thesis]. University of Manitoba; 2015. Available from: http://hdl.handle.net/1993/31914

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Manitoba

13. Wang, Jie. Fibroblast growth factor-16 and acute doxorubicin cardiotoxicity: a target for early protection.

Degree: Physiology and Pathophysiology, 2018, University of Manitoba

 Background: Doxorubicin is an anti-cancer drug that is widely used in chemotherapy. However, doxorubicin-induced cardiotoxicity is a major risk factor for cancer patients and survivors,… (more)

Subjects/Keywords: Fibroblast growth factor 16; doxorubicin; multidrug resistance protein 1; cardioprotection

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APA (6th Edition):

Wang, J. (2018). Fibroblast growth factor-16 and acute doxorubicin cardiotoxicity: a target for early protection. (Thesis). University of Manitoba. Retrieved from http://hdl.handle.net/1993/33048

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Wang, Jie. “Fibroblast growth factor-16 and acute doxorubicin cardiotoxicity: a target for early protection.” 2018. Thesis, University of Manitoba. Accessed January 24, 2020. http://hdl.handle.net/1993/33048.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Wang, Jie. “Fibroblast growth factor-16 and acute doxorubicin cardiotoxicity: a target for early protection.” 2018. Web. 24 Jan 2020.

Vancouver:

Wang J. Fibroblast growth factor-16 and acute doxorubicin cardiotoxicity: a target for early protection. [Internet] [Thesis]. University of Manitoba; 2018. [cited 2020 Jan 24]. Available from: http://hdl.handle.net/1993/33048.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Wang J. Fibroblast growth factor-16 and acute doxorubicin cardiotoxicity: a target for early protection. [Thesis]. University of Manitoba; 2018. Available from: http://hdl.handle.net/1993/33048

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Manitoba

14. Lepage, Chloe C. Evaluating the impact diminished SKP1 and CUL1 expression have on chromosome instability and high-grade serous ovarian cancer pathogenesis.

Degree: Biochemistry and Medical Genetics, 2019, University of Manitoba

 High-grade serous ovarian cancer (HGSOC) is the most common and lethal ovarian cancer subtype. Chromosome instability (CIN; an increased rate of chromosome gains and losses)… (more)

Subjects/Keywords: High-grade serous ovarian cancer; Chromosome instability; Biochemistry; Genetics; Cancer; SCF complex; Cellular transformation

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APA (6th Edition):

Lepage, C. C. (2019). Evaluating the impact diminished SKP1 and CUL1 expression have on chromosome instability and high-grade serous ovarian cancer pathogenesis. (Masters Thesis). University of Manitoba. Retrieved from http://hdl.handle.net/1993/34461

Chicago Manual of Style (16th Edition):

Lepage, Chloe C. “Evaluating the impact diminished SKP1 and CUL1 expression have on chromosome instability and high-grade serous ovarian cancer pathogenesis.” 2019. Masters Thesis, University of Manitoba. Accessed January 24, 2020. http://hdl.handle.net/1993/34461.

MLA Handbook (7th Edition):

Lepage, Chloe C. “Evaluating the impact diminished SKP1 and CUL1 expression have on chromosome instability and high-grade serous ovarian cancer pathogenesis.” 2019. Web. 24 Jan 2020.

Vancouver:

Lepage CC. Evaluating the impact diminished SKP1 and CUL1 expression have on chromosome instability and high-grade serous ovarian cancer pathogenesis. [Internet] [Masters thesis]. University of Manitoba; 2019. [cited 2020 Jan 24]. Available from: http://hdl.handle.net/1993/34461.

Council of Science Editors:

Lepage CC. Evaluating the impact diminished SKP1 and CUL1 expression have on chromosome instability and high-grade serous ovarian cancer pathogenesis. [Masters Thesis]. University of Manitoba; 2019. Available from: http://hdl.handle.net/1993/34461

15. Wang, Jie. Fibroblast growth factor-2 protects neonatal rat cardiac myocytes from doxorubicin-induced damage via protein kinase C- dependent effects on efflux drug transporters.

Degree: Physiology, 2013, University of Manitoba

 Introduction: Therapeutic agents like doxorubicin, an anthracycline antibiotic drug, are widely used in cancer chemotherapy. The use of doxorubicin is limited however by an increased… (more)

Subjects/Keywords: FGF-2; Doxorubicin; efflux drug transporters; PKC; neonatal rat cardiomyocytes

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APA (6th Edition):

Wang, J. (2013). Fibroblast growth factor-2 protects neonatal rat cardiac myocytes from doxorubicin-induced damage via protein kinase C- dependent effects on efflux drug transporters. (Masters Thesis). University of Manitoba. Retrieved from http://hdl.handle.net/1993/18318

Chicago Manual of Style (16th Edition):

Wang, Jie. “Fibroblast growth factor-2 protects neonatal rat cardiac myocytes from doxorubicin-induced damage via protein kinase C- dependent effects on efflux drug transporters.” 2013. Masters Thesis, University of Manitoba. Accessed January 24, 2020. http://hdl.handle.net/1993/18318.

MLA Handbook (7th Edition):

Wang, Jie. “Fibroblast growth factor-2 protects neonatal rat cardiac myocytes from doxorubicin-induced damage via protein kinase C- dependent effects on efflux drug transporters.” 2013. Web. 24 Jan 2020.

Vancouver:

Wang J. Fibroblast growth factor-2 protects neonatal rat cardiac myocytes from doxorubicin-induced damage via protein kinase C- dependent effects on efflux drug transporters. [Internet] [Masters thesis]. University of Manitoba; 2013. [cited 2020 Jan 24]. Available from: http://hdl.handle.net/1993/18318.

Council of Science Editors:

Wang J. Fibroblast growth factor-2 protects neonatal rat cardiac myocytes from doxorubicin-induced damage via protein kinase C- dependent effects on efflux drug transporters. [Masters Thesis]. University of Manitoba; 2013. Available from: http://hdl.handle.net/1993/18318

16. Adewumi, Ifeoluwa. TNF-alpha gene signature in triple-negative breast cancer cells.

Degree: Biochemistry and Medical Genetics, 2017, University of Manitoba

 Triple-negative breast cancer patients have an increased likelihood of distant recurrence and death when compared with patients with other subtypes of breast cancer. High persistence… (more)

Subjects/Keywords: Cancer Epigenetics

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APA (6th Edition):

Adewumi, I. (2017). TNF-alpha gene signature in triple-negative breast cancer cells. (Masters Thesis). University of Manitoba. Retrieved from http://hdl.handle.net/1993/32363

Chicago Manual of Style (16th Edition):

Adewumi, Ifeoluwa. “TNF-alpha gene signature in triple-negative breast cancer cells.” 2017. Masters Thesis, University of Manitoba. Accessed January 24, 2020. http://hdl.handle.net/1993/32363.

MLA Handbook (7th Edition):

Adewumi, Ifeoluwa. “TNF-alpha gene signature in triple-negative breast cancer cells.” 2017. Web. 24 Jan 2020.

Vancouver:

Adewumi I. TNF-alpha gene signature in triple-negative breast cancer cells. [Internet] [Masters thesis]. University of Manitoba; 2017. [cited 2020 Jan 24]. Available from: http://hdl.handle.net/1993/32363.

Council of Science Editors:

Adewumi I. TNF-alpha gene signature in triple-negative breast cancer cells. [Masters Thesis]. University of Manitoba; 2017. Available from: http://hdl.handle.net/1993/32363

17. Stromecki, Margaret. Characterization of a novel OTX2-driven stem cell program in group 3 and group 4 medulloblastoma.

Degree: Biochemistry and Medical Genetics, 2017, University of Manitoba

 Medulloblastoma (MB) is a primary pediatric brain cancer that is frequently accompanied by metastasis and poor long-term prognosis. Our goal is to identify new signaling… (more)

Subjects/Keywords: Medulloblastoma; cancer stem cells; OTX2; axon guidance genes; semaphorin; Rho

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Stromecki, M. (2017). Characterization of a novel OTX2-driven stem cell program in group 3 and group 4 medulloblastoma. (Masters Thesis). University of Manitoba. Retrieved from http://hdl.handle.net/1993/32396

Chicago Manual of Style (16th Edition):

Stromecki, Margaret. “Characterization of a novel OTX2-driven stem cell program in group 3 and group 4 medulloblastoma.” 2017. Masters Thesis, University of Manitoba. Accessed January 24, 2020. http://hdl.handle.net/1993/32396.

MLA Handbook (7th Edition):

Stromecki, Margaret. “Characterization of a novel OTX2-driven stem cell program in group 3 and group 4 medulloblastoma.” 2017. Web. 24 Jan 2020.

Vancouver:

Stromecki M. Characterization of a novel OTX2-driven stem cell program in group 3 and group 4 medulloblastoma. [Internet] [Masters thesis]. University of Manitoba; 2017. [cited 2020 Jan 24]. Available from: http://hdl.handle.net/1993/32396.

Council of Science Editors:

Stromecki M. Characterization of a novel OTX2-driven stem cell program in group 3 and group 4 medulloblastoma. [Masters Thesis]. University of Manitoba; 2017. Available from: http://hdl.handle.net/1993/32396

18. Aiken, Christopher. Characterization of cellular heterogeneity in the SHH subgroup of medulloblastoma.

Degree: Physiology and Pathophysiology, 2018, University of Manitoba

 Medulloblastoma (MB) is the most common form of primary malignant pediatric brain cancer. MB is divided into 5 molecular subgroups; Wnt, Sonic Hedgehog (SHH) p53… (more)

Subjects/Keywords: Stem Cell; Medulloblastoma; Brain Cancer

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APA (6th Edition):

Aiken, C. (2018). Characterization of cellular heterogeneity in the SHH subgroup of medulloblastoma. (Masters Thesis). University of Manitoba. Retrieved from http://hdl.handle.net/1993/33407

Chicago Manual of Style (16th Edition):

Aiken, Christopher. “Characterization of cellular heterogeneity in the SHH subgroup of medulloblastoma.” 2018. Masters Thesis, University of Manitoba. Accessed January 24, 2020. http://hdl.handle.net/1993/33407.

MLA Handbook (7th Edition):

Aiken, Christopher. “Characterization of cellular heterogeneity in the SHH subgroup of medulloblastoma.” 2018. Web. 24 Jan 2020.

Vancouver:

Aiken C. Characterization of cellular heterogeneity in the SHH subgroup of medulloblastoma. [Internet] [Masters thesis]. University of Manitoba; 2018. [cited 2020 Jan 24]. Available from: http://hdl.handle.net/1993/33407.

Council of Science Editors:

Aiken C. Characterization of cellular heterogeneity in the SHH subgroup of medulloblastoma. [Masters Thesis]. University of Manitoba; 2018. Available from: http://hdl.handle.net/1993/33407

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