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You searched for +publisher:"University of Louisville" +contributor:("Bodduluri, Haribabu"). Showing records 1 – 10 of 10 total matches.

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University of Louisville

1. Roberson, Carolyn Denise. Modulation of the ovarian cancer humoral response by tumor-derived exosomes.

Degree: PhD, 2014, University of Louisville

  The pathogenesis of epithelial ovarian cancer is complicated by its diagnosis during the latter stages of the disease resulting from lack of symptoms or… (more)

Subjects/Keywords: Microbiology; Oncology

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APA (6th Edition):

Roberson, C. D. (2014). Modulation of the ovarian cancer humoral response by tumor-derived exosomes. (Doctoral Dissertation). University of Louisville. Retrieved from 10.18297/etd/1771 ; https://ir.library.louisville.edu/etd/1771

Chicago Manual of Style (16th Edition):

Roberson, Carolyn Denise. “Modulation of the ovarian cancer humoral response by tumor-derived exosomes.” 2014. Doctoral Dissertation, University of Louisville. Accessed July 16, 2019. 10.18297/etd/1771 ; https://ir.library.louisville.edu/etd/1771.

MLA Handbook (7th Edition):

Roberson, Carolyn Denise. “Modulation of the ovarian cancer humoral response by tumor-derived exosomes.” 2014. Web. 16 Jul 2019.

Vancouver:

Roberson CD. Modulation of the ovarian cancer humoral response by tumor-derived exosomes. [Internet] [Doctoral dissertation]. University of Louisville; 2014. [cited 2019 Jul 16]. Available from: 10.18297/etd/1771 ; https://ir.library.louisville.edu/etd/1771.

Council of Science Editors:

Roberson CD. Modulation of the ovarian cancer humoral response by tumor-derived exosomes. [Doctoral Dissertation]. University of Louisville; 2014. Available from: 10.18297/etd/1771 ; https://ir.library.louisville.edu/etd/1771


University of Louisville

2. Bhutiani, Neal. Combination oral immunotherapy with IL-10 and IL-12 effectively treats colon tumors via synergistic effects on IL-17 producing T cells and colon epithelial barrier integrity.

Degree: PhD, 2018, University of Louisville

  In this dissertation, the relationship between colon cancer and inflammation, the utility of novel imaging modalities for diagnosis of colitis and cancer, and the… (more)

Subjects/Keywords: colon cancer; immunotherapy; T cells; nanoparticles; epithelial barrier integrity; Medical Immunology

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APA (6th Edition):

Bhutiani, N. (2018). Combination oral immunotherapy with IL-10 and IL-12 effectively treats colon tumors via synergistic effects on IL-17 producing T cells and colon epithelial barrier integrity. (Doctoral Dissertation). University of Louisville. Retrieved from 10.18297/etd/3013 ; https://ir.library.louisville.edu/etd/3013

Chicago Manual of Style (16th Edition):

Bhutiani, Neal. “Combination oral immunotherapy with IL-10 and IL-12 effectively treats colon tumors via synergistic effects on IL-17 producing T cells and colon epithelial barrier integrity.” 2018. Doctoral Dissertation, University of Louisville. Accessed July 16, 2019. 10.18297/etd/3013 ; https://ir.library.louisville.edu/etd/3013.

MLA Handbook (7th Edition):

Bhutiani, Neal. “Combination oral immunotherapy with IL-10 and IL-12 effectively treats colon tumors via synergistic effects on IL-17 producing T cells and colon epithelial barrier integrity.” 2018. Web. 16 Jul 2019.

Vancouver:

Bhutiani N. Combination oral immunotherapy with IL-10 and IL-12 effectively treats colon tumors via synergistic effects on IL-17 producing T cells and colon epithelial barrier integrity. [Internet] [Doctoral dissertation]. University of Louisville; 2018. [cited 2019 Jul 16]. Available from: 10.18297/etd/3013 ; https://ir.library.louisville.edu/etd/3013.

Council of Science Editors:

Bhutiani N. Combination oral immunotherapy with IL-10 and IL-12 effectively treats colon tumors via synergistic effects on IL-17 producing T cells and colon epithelial barrier integrity. [Doctoral Dissertation]. University of Louisville; 2018. Available from: 10.18297/etd/3013 ; https://ir.library.louisville.edu/etd/3013


University of Louisville

3. Gerlach, Rachael Lask. Early host response and immune signaling to 2009 pandemic influenza A (H1N1) viruses in primary cell culture models.

Degree: PhD, 2015, University of Louisville

  Influenza A virus (IAV) subtypes and even genotypes within subtypes can show differences in tropism (host, cell type), magnitude of infection, immune response and… (more)

Subjects/Keywords: Immunology and Infectious Disease; Microbiology

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APA (6th Edition):

Gerlach, R. L. (2015). Early host response and immune signaling to 2009 pandemic influenza A (H1N1) viruses in primary cell culture models. (Doctoral Dissertation). University of Louisville. Retrieved from 10.18297/etd/2078 ; https://ir.library.louisville.edu/etd/2078

Chicago Manual of Style (16th Edition):

Gerlach, Rachael Lask. “Early host response and immune signaling to 2009 pandemic influenza A (H1N1) viruses in primary cell culture models.” 2015. Doctoral Dissertation, University of Louisville. Accessed July 16, 2019. 10.18297/etd/2078 ; https://ir.library.louisville.edu/etd/2078.

MLA Handbook (7th Edition):

Gerlach, Rachael Lask. “Early host response and immune signaling to 2009 pandemic influenza A (H1N1) viruses in primary cell culture models.” 2015. Web. 16 Jul 2019.

Vancouver:

Gerlach RL. Early host response and immune signaling to 2009 pandemic influenza A (H1N1) viruses in primary cell culture models. [Internet] [Doctoral dissertation]. University of Louisville; 2015. [cited 2019 Jul 16]. Available from: 10.18297/etd/2078 ; https://ir.library.louisville.edu/etd/2078.

Council of Science Editors:

Gerlach RL. Early host response and immune signaling to 2009 pandemic influenza A (H1N1) viruses in primary cell culture models. [Doctoral Dissertation]. University of Louisville; 2015. Available from: 10.18297/etd/2078 ; https://ir.library.louisville.edu/etd/2078


University of Louisville

4. Kolb, Joseph Peterson. A mechanism for trif adaptor-biased signaling by toll-like receptor 4.

Degree: PhD, 2014, University of Louisville

  Host cells respond to bacterial lipid A through Toll-like receptor 4 (TLR4). Activation of TLR4 by lipid A triggers a response that involves two… (more)

Subjects/Keywords: Microbiology

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APA (6th Edition):

Kolb, J. P. (2014). A mechanism for trif adaptor-biased signaling by toll-like receptor 4. (Doctoral Dissertation). University of Louisville. Retrieved from 10.18297/etd/1755 ; https://ir.library.louisville.edu/etd/1755

Chicago Manual of Style (16th Edition):

Kolb, Joseph Peterson. “A mechanism for trif adaptor-biased signaling by toll-like receptor 4.” 2014. Doctoral Dissertation, University of Louisville. Accessed July 16, 2019. 10.18297/etd/1755 ; https://ir.library.louisville.edu/etd/1755.

MLA Handbook (7th Edition):

Kolb, Joseph Peterson. “A mechanism for trif adaptor-biased signaling by toll-like receptor 4.” 2014. Web. 16 Jul 2019.

Vancouver:

Kolb JP. A mechanism for trif adaptor-biased signaling by toll-like receptor 4. [Internet] [Doctoral dissertation]. University of Louisville; 2014. [cited 2019 Jul 16]. Available from: 10.18297/etd/1755 ; https://ir.library.louisville.edu/etd/1755.

Council of Science Editors:

Kolb JP. A mechanism for trif adaptor-biased signaling by toll-like receptor 4. [Doctoral Dissertation]. University of Louisville; 2014. Available from: 10.18297/etd/1755 ; https://ir.library.louisville.edu/etd/1755


University of Louisville

5. Pandit, Harshulkumar M. Wnt/β-Catenin mediated cancer stem cell activation in hepatocellular carcinoma.

Degree: PhD, 2018, University of Louisville

  Hepatocellular carcinoma (HCC) is one of the deadliest cancers, with variable presentation, high chemotherapy resistance and early tumor recurrence. In this dissertation, we have… (more)

Subjects/Keywords: hepatocellular carcinoma (HCC); cancer stem cells (CSCs); Wnt/β-catenin signaling; epithelial cell adhesion molecule (EpCAM); orthotopic HCC mouse models; Neoplasms; Oncology; Pharmacology

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APA (6th Edition):

Pandit, H. M. (2018). Wnt/β-Catenin mediated cancer stem cell activation in hepatocellular carcinoma. (Doctoral Dissertation). University of Louisville. Retrieved from 10.18297/etd/3010 ; https://ir.library.louisville.edu/etd/3010

Chicago Manual of Style (16th Edition):

Pandit, Harshulkumar M. “Wnt/β-Catenin mediated cancer stem cell activation in hepatocellular carcinoma.” 2018. Doctoral Dissertation, University of Louisville. Accessed July 16, 2019. 10.18297/etd/3010 ; https://ir.library.louisville.edu/etd/3010.

MLA Handbook (7th Edition):

Pandit, Harshulkumar M. “Wnt/β-Catenin mediated cancer stem cell activation in hepatocellular carcinoma.” 2018. Web. 16 Jul 2019.

Vancouver:

Pandit HM. Wnt/β-Catenin mediated cancer stem cell activation in hepatocellular carcinoma. [Internet] [Doctoral dissertation]. University of Louisville; 2018. [cited 2019 Jul 16]. Available from: 10.18297/etd/3010 ; https://ir.library.louisville.edu/etd/3010.

Council of Science Editors:

Pandit HM. Wnt/β-Catenin mediated cancer stem cell activation in hepatocellular carcinoma. [Doctoral Dissertation]. University of Louisville; 2018. Available from: 10.18297/etd/3010 ; https://ir.library.louisville.edu/etd/3010


University of Louisville

6. Hegde, Bindu. Inflammasome independent leukotriene-b4 production drives crystalline silica induced sterile inflammation.

Degree: PhD, 2018, University of Louisville

  Silicosis is an irreversible lung inflammatory disease caused by chronic exposure to crystalline silica (CS) and is one of the most prevalent occupational diseases… (more)

Subjects/Keywords: crystalline silica; LTB4; inflammasome; lipidosome; sterile inflammation; Medical Immunology; Medical Microbiology

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APA (6th Edition):

Hegde, B. (2018). Inflammasome independent leukotriene-b4 production drives crystalline silica induced sterile inflammation. (Doctoral Dissertation). University of Louisville. Retrieved from 10.18297/etd/2946 ; https://ir.library.louisville.edu/etd/2946

Chicago Manual of Style (16th Edition):

Hegde, Bindu. “Inflammasome independent leukotriene-b4 production drives crystalline silica induced sterile inflammation.” 2018. Doctoral Dissertation, University of Louisville. Accessed July 16, 2019. 10.18297/etd/2946 ; https://ir.library.louisville.edu/etd/2946.

MLA Handbook (7th Edition):

Hegde, Bindu. “Inflammasome independent leukotriene-b4 production drives crystalline silica induced sterile inflammation.” 2018. Web. 16 Jul 2019.

Vancouver:

Hegde B. Inflammasome independent leukotriene-b4 production drives crystalline silica induced sterile inflammation. [Internet] [Doctoral dissertation]. University of Louisville; 2018. [cited 2019 Jul 16]. Available from: 10.18297/etd/2946 ; https://ir.library.louisville.edu/etd/2946.

Council of Science Editors:

Hegde B. Inflammasome independent leukotriene-b4 production drives crystalline silica induced sterile inflammation. [Doctoral Dissertation]. University of Louisville; 2018. Available from: 10.18297/etd/2946 ; https://ir.library.louisville.edu/etd/2946


University of Louisville

7. Baldauf, Keegan J. A plant-made cholera toxin B subunit enhances mucosal wound healing and protects against ulcerative colitis and colon cancer.

Degree: PhD, 2015, University of Louisville

  This dissertation describes the previously unidentified effects of a plant-produced recombinant cholera toxin B subunit (CTBp) on the gastrointestinal (GI) tract and its ability… (more)

Subjects/Keywords: ulcerative colitis; cholera toxin B subunit; inflammatory bowel disease; inflammation; Pharmacology

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APA (6th Edition):

Baldauf, K. J. (2015). A plant-made cholera toxin B subunit enhances mucosal wound healing and protects against ulcerative colitis and colon cancer. (Doctoral Dissertation). University of Louisville. Retrieved from 10.18297/etd/2328 ; https://ir.library.louisville.edu/etd/2328

Chicago Manual of Style (16th Edition):

Baldauf, Keegan J. “A plant-made cholera toxin B subunit enhances mucosal wound healing and protects against ulcerative colitis and colon cancer.” 2015. Doctoral Dissertation, University of Louisville. Accessed July 16, 2019. 10.18297/etd/2328 ; https://ir.library.louisville.edu/etd/2328.

MLA Handbook (7th Edition):

Baldauf, Keegan J. “A plant-made cholera toxin B subunit enhances mucosal wound healing and protects against ulcerative colitis and colon cancer.” 2015. Web. 16 Jul 2019.

Vancouver:

Baldauf KJ. A plant-made cholera toxin B subunit enhances mucosal wound healing and protects against ulcerative colitis and colon cancer. [Internet] [Doctoral dissertation]. University of Louisville; 2015. [cited 2019 Jul 16]. Available from: 10.18297/etd/2328 ; https://ir.library.louisville.edu/etd/2328.

Council of Science Editors:

Baldauf KJ. A plant-made cholera toxin B subunit enhances mucosal wound healing and protects against ulcerative colitis and colon cancer. [Doctoral Dissertation]. University of Louisville; 2015. Available from: 10.18297/etd/2328 ; https://ir.library.louisville.edu/etd/2328


University of Louisville

8. Chheda, Zinal S. Chemoattractant receptors BLT1 and CXCR3 regulate anti-tumor immunity by facilitating CD8+ T cell migration to tumors.

Degree: PhD, 2015, University of Louisville

  Presence of increased numbers of CD8+ T cells in the tumors correspond to better overall survival in the patients. Variety of immuno-therapies have shown… (more)

Subjects/Keywords: anti-tumor immunity; melanoma; CTL infiltration to tumors; PD-1 blockade; LTB4; CXCL9 and CXCL10; Immunity

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APA (6th Edition):

Chheda, Z. S. (2015). Chemoattractant receptors BLT1 and CXCR3 regulate anti-tumor immunity by facilitating CD8+ T cell migration to tumors. (Doctoral Dissertation). University of Louisville. Retrieved from 10.18297/etd/2294 ; https://ir.library.louisville.edu/etd/2294

Chicago Manual of Style (16th Edition):

Chheda, Zinal S. “Chemoattractant receptors BLT1 and CXCR3 regulate anti-tumor immunity by facilitating CD8+ T cell migration to tumors.” 2015. Doctoral Dissertation, University of Louisville. Accessed July 16, 2019. 10.18297/etd/2294 ; https://ir.library.louisville.edu/etd/2294.

MLA Handbook (7th Edition):

Chheda, Zinal S. “Chemoattractant receptors BLT1 and CXCR3 regulate anti-tumor immunity by facilitating CD8+ T cell migration to tumors.” 2015. Web. 16 Jul 2019.

Vancouver:

Chheda ZS. Chemoattractant receptors BLT1 and CXCR3 regulate anti-tumor immunity by facilitating CD8+ T cell migration to tumors. [Internet] [Doctoral dissertation]. University of Louisville; 2015. [cited 2019 Jul 16]. Available from: 10.18297/etd/2294 ; https://ir.library.louisville.edu/etd/2294.

Council of Science Editors:

Chheda ZS. Chemoattractant receptors BLT1 and CXCR3 regulate anti-tumor immunity by facilitating CD8+ T cell migration to tumors. [Doctoral Dissertation]. University of Louisville; 2015. Available from: 10.18297/etd/2294 ; https://ir.library.louisville.edu/etd/2294


University of Louisville

9. Mathis, Steven, 1979-. Non-redundant and critical roles for leukotriene B4 receptors BLT1 and BLT2 in mouse models of inflammatory arthritis.

Degree: PhD, 2007, University of Louisville

 Inflammation is now recognized as an important factor in several age-related diseases such as arthritis, atherosclerosis, multiple sclerosis and diabetes. In each case, sub-clinical chronic… (more)

Subjects/Keywords: Leukotriene; Inflammatory arthritis; Arthritis

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APA (6th Edition):

Mathis, Steven, 1. (2007). Non-redundant and critical roles for leukotriene B4 receptors BLT1 and BLT2 in mouse models of inflammatory arthritis. (Doctoral Dissertation). University of Louisville. Retrieved from 10.18297/etd/920 ; https://ir.library.louisville.edu/etd/920

Chicago Manual of Style (16th Edition):

Mathis, Steven, 1979-. “Non-redundant and critical roles for leukotriene B4 receptors BLT1 and BLT2 in mouse models of inflammatory arthritis.” 2007. Doctoral Dissertation, University of Louisville. Accessed July 16, 2019. 10.18297/etd/920 ; https://ir.library.louisville.edu/etd/920.

MLA Handbook (7th Edition):

Mathis, Steven, 1979-. “Non-redundant and critical roles for leukotriene B4 receptors BLT1 and BLT2 in mouse models of inflammatory arthritis.” 2007. Web. 16 Jul 2019.

Vancouver:

Mathis, Steven 1. Non-redundant and critical roles for leukotriene B4 receptors BLT1 and BLT2 in mouse models of inflammatory arthritis. [Internet] [Doctoral dissertation]. University of Louisville; 2007. [cited 2019 Jul 16]. Available from: 10.18297/etd/920 ; https://ir.library.louisville.edu/etd/920.

Council of Science Editors:

Mathis, Steven 1. Non-redundant and critical roles for leukotriene B4 receptors BLT1 and BLT2 in mouse models of inflammatory arthritis. [Doctoral Dissertation]. University of Louisville; 2007. Available from: 10.18297/etd/920 ; https://ir.library.louisville.edu/etd/920

10. Pandit, Harshulkumar M. Identifying Hepatocellular Carcinoma (HCC) cells with cancer stem cell-like properties.

Degree: MS, 2015, University of Louisville

  Hepatocellular Carcinoma (HCC) is a challenging condition to treat in the clinic due to its distinct tumor phenotype, aggressive growth, and chemotherapy resistance. Cancer… (more)

Subjects/Keywords: Pharmacy and Pharmaceutical Sciences

…Institutional Animal Care and Use Committee (IACUC) at University of Louisville (UofL… …Animal Care. All mice were housed in the University of Louisville Research Resources Center at… …Human Study at University of Louisville. The study samples were retrospectively collected from… 

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APA (6th Edition):

Pandit, H. M. (2015). Identifying Hepatocellular Carcinoma (HCC) cells with cancer stem cell-like properties. (Masters Thesis). University of Louisville. Retrieved from 10.18297/etd/2211 ; https://ir.library.louisville.edu/etd/2211

Chicago Manual of Style (16th Edition):

Pandit, Harshulkumar M. “Identifying Hepatocellular Carcinoma (HCC) cells with cancer stem cell-like properties.” 2015. Masters Thesis, University of Louisville. Accessed July 16, 2019. 10.18297/etd/2211 ; https://ir.library.louisville.edu/etd/2211.

MLA Handbook (7th Edition):

Pandit, Harshulkumar M. “Identifying Hepatocellular Carcinoma (HCC) cells with cancer stem cell-like properties.” 2015. Web. 16 Jul 2019.

Vancouver:

Pandit HM. Identifying Hepatocellular Carcinoma (HCC) cells with cancer stem cell-like properties. [Internet] [Masters thesis]. University of Louisville; 2015. [cited 2019 Jul 16]. Available from: 10.18297/etd/2211 ; https://ir.library.louisville.edu/etd/2211.

Council of Science Editors:

Pandit HM. Identifying Hepatocellular Carcinoma (HCC) cells with cancer stem cell-like properties. [Masters Thesis]. University of Louisville; 2015. Available from: 10.18297/etd/2211 ; https://ir.library.louisville.edu/etd/2211

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