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You searched for +publisher:"University of Kansas" +contributor:("Wood, John"). Showing records 1 – 17 of 17 total matches.

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University of Kansas

1. Allen, Katryn Miller. Mechanisms of Proinflammatory Signaling during Cholestasis.

Degree: PhD, Pharmacology, Toxicology & Therapeutics, 2011, University of Kansas

 Cholestasis is a condition in which bile flow from the liver to the intestines is inhibited. Loss of bile flow leads to increased concentrations of… (more)

Subjects/Keywords: Toxicology; Bile acids; Cholestasis; Early growth response factor-1; Inflammation

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APA (6th Edition):

Allen, K. M. (2011). Mechanisms of Proinflammatory Signaling during Cholestasis. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/7918

Chicago Manual of Style (16th Edition):

Allen, Katryn Miller. “Mechanisms of Proinflammatory Signaling during Cholestasis.” 2011. Doctoral Dissertation, University of Kansas. Accessed April 19, 2019. http://hdl.handle.net/1808/7918.

MLA Handbook (7th Edition):

Allen, Katryn Miller. “Mechanisms of Proinflammatory Signaling during Cholestasis.” 2011. Web. 19 Apr 2019.

Vancouver:

Allen KM. Mechanisms of Proinflammatory Signaling during Cholestasis. [Internet] [Doctoral dissertation]. University of Kansas; 2011. [cited 2019 Apr 19]. Available from: http://hdl.handle.net/1808/7918.

Council of Science Editors:

Allen KM. Mechanisms of Proinflammatory Signaling during Cholestasis. [Doctoral Dissertation]. University of Kansas; 2011. Available from: http://hdl.handle.net/1808/7918


University of Kansas

2. Woolbright, Ben. The role of bile acids during cholestasis in mice and man.

Degree: PhD, Pharmacology, Toxicology & Therapeutics, 2015, University of Kansas

 Cholestasis is a reduction in bile flow that occurs during numerous pathologies. Cholestasis leads to significant liver toxicity, biliary hyperplasia, and liver cirrhosis. The molecular… (more)

Subjects/Keywords: Toxicology; Pathology; Pharmacology; apoptosis; bile acid; cholestasis; hepatocytes; inflammation; neutrophil

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APA (6th Edition):

Woolbright, B. (2015). The role of bile acids during cholestasis in mice and man. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/19455

Chicago Manual of Style (16th Edition):

Woolbright, Ben. “The role of bile acids during cholestasis in mice and man.” 2015. Doctoral Dissertation, University of Kansas. Accessed April 19, 2019. http://hdl.handle.net/1808/19455.

MLA Handbook (7th Edition):

Woolbright, Ben. “The role of bile acids during cholestasis in mice and man.” 2015. Web. 19 Apr 2019.

Vancouver:

Woolbright B. The role of bile acids during cholestasis in mice and man. [Internet] [Doctoral dissertation]. University of Kansas; 2015. [cited 2019 Apr 19]. Available from: http://hdl.handle.net/1808/19455.

Council of Science Editors:

Woolbright B. The role of bile acids during cholestasis in mice and man. [Doctoral Dissertation]. University of Kansas; 2015. Available from: http://hdl.handle.net/1808/19455


University of Kansas

3. Xie, Yuchao. ACETAMINOPHEN HEPATOTOXICITY IN PRIMARY HUMAN HEPATOCYTES AND MICE.

Degree: PhD, Pharmacology, Toxicology & Therapeutics, 2016, University of Kansas

 Acetaminophen is the most prevalent cause of acute liver failure (ALF) and drug-induced liver injury (DILI) in western countries. Extensive studies have revealed important intracellular… (more)

Subjects/Keywords: Toxicology

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APA (6th Edition):

Xie, Y. (2016). ACETAMINOPHEN HEPATOTOXICITY IN PRIMARY HUMAN HEPATOCYTES AND MICE. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/22502

Chicago Manual of Style (16th Edition):

Xie, Yuchao. “ACETAMINOPHEN HEPATOTOXICITY IN PRIMARY HUMAN HEPATOCYTES AND MICE.” 2016. Doctoral Dissertation, University of Kansas. Accessed April 19, 2019. http://hdl.handle.net/1808/22502.

MLA Handbook (7th Edition):

Xie, Yuchao. “ACETAMINOPHEN HEPATOTOXICITY IN PRIMARY HUMAN HEPATOCYTES AND MICE.” 2016. Web. 19 Apr 2019.

Vancouver:

Xie Y. ACETAMINOPHEN HEPATOTOXICITY IN PRIMARY HUMAN HEPATOCYTES AND MICE. [Internet] [Doctoral dissertation]. University of Kansas; 2016. [cited 2019 Apr 19]. Available from: http://hdl.handle.net/1808/22502.

Council of Science Editors:

Xie Y. ACETAMINOPHEN HEPATOTOXICITY IN PRIMARY HUMAN HEPATOCYTES AND MICE. [Doctoral Dissertation]. University of Kansas; 2016. Available from: http://hdl.handle.net/1808/22502


University of Kansas

4. Venugopal, Anand. IDENTIFICATION OF THE RNA BINDING PROTEIN RBM3 AS A NOVEL EFFECTOR OF β-CATENIN SIGNALING AND COLON CANCER STEM CELLS.

Degree: PhD, Molecular & Integrative Physiology, 2014, University of Kansas

 The intestinal epithelium is one of the fastest renewing tissues within the adult. This renewal is primarily driven by the intestinal epithelial stem cell compartment… (more)

Subjects/Keywords: Molecular biology; Oncology; DCLK1; GSK3β

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APA (6th Edition):

Venugopal, A. (2014). IDENTIFICATION OF THE RNA BINDING PROTEIN RBM3 AS A NOVEL EFFECTOR OF β-CATENIN SIGNALING AND COLON CANCER STEM CELLS. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/22535

Chicago Manual of Style (16th Edition):

Venugopal, Anand. “IDENTIFICATION OF THE RNA BINDING PROTEIN RBM3 AS A NOVEL EFFECTOR OF β-CATENIN SIGNALING AND COLON CANCER STEM CELLS.” 2014. Doctoral Dissertation, University of Kansas. Accessed April 19, 2019. http://hdl.handle.net/1808/22535.

MLA Handbook (7th Edition):

Venugopal, Anand. “IDENTIFICATION OF THE RNA BINDING PROTEIN RBM3 AS A NOVEL EFFECTOR OF β-CATENIN SIGNALING AND COLON CANCER STEM CELLS.” 2014. Web. 19 Apr 2019.

Vancouver:

Venugopal A. IDENTIFICATION OF THE RNA BINDING PROTEIN RBM3 AS A NOVEL EFFECTOR OF β-CATENIN SIGNALING AND COLON CANCER STEM CELLS. [Internet] [Doctoral dissertation]. University of Kansas; 2014. [cited 2019 Apr 19]. Available from: http://hdl.handle.net/1808/22535.

Council of Science Editors:

Venugopal A. IDENTIFICATION OF THE RNA BINDING PROTEIN RBM3 AS A NOVEL EFFECTOR OF β-CATENIN SIGNALING AND COLON CANCER STEM CELLS. [Doctoral Dissertation]. University of Kansas; 2014. Available from: http://hdl.handle.net/1808/22535


University of Kansas

5. Weemhoff, James Lawrence. Mechanistic Biomarkers in Acute Liver Injury.

Degree: PhD, Pharmacology, Toxicology & Therapeutics, 2017, University of Kansas

 Acute liver failure continues to be a major medical problem. There are many underlying causes of acute liver failure, but drug induced liver injury is… (more)

Subjects/Keywords: Toxicology; Acetaminophen; Biomarkers; HepaRG; Hypoxic Hepatitis; Liver Injury; Liver Transplantation

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APA (6th Edition):

Weemhoff, J. L. (2017). Mechanistic Biomarkers in Acute Liver Injury. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/27044

Chicago Manual of Style (16th Edition):

Weemhoff, James Lawrence. “Mechanistic Biomarkers in Acute Liver Injury.” 2017. Doctoral Dissertation, University of Kansas. Accessed April 19, 2019. http://hdl.handle.net/1808/27044.

MLA Handbook (7th Edition):

Weemhoff, James Lawrence. “Mechanistic Biomarkers in Acute Liver Injury.” 2017. Web. 19 Apr 2019.

Vancouver:

Weemhoff JL. Mechanistic Biomarkers in Acute Liver Injury. [Internet] [Doctoral dissertation]. University of Kansas; 2017. [cited 2019 Apr 19]. Available from: http://hdl.handle.net/1808/27044.

Council of Science Editors:

Weemhoff JL. Mechanistic Biomarkers in Acute Liver Injury. [Doctoral Dissertation]. University of Kansas; 2017. Available from: http://hdl.handle.net/1808/27044


University of Kansas

6. Neradugomma, Naveen Kumar. Role of Prolactin and Prolactin Receptor Signaling in Colorectal Tumorigenesis.

Degree: PhD, Molecular & Integrative Physiology, 2014, University of Kansas

 Hormones are critical regulatory factors produced by the body to regulate diverse physiological activities such as energy homeostasis, growth and differentiation of a diverse array… (more)

Subjects/Keywords: Physiology; Molecular biology; Biology; Cancer Stem Cells; Colorectal Cancer; Copynumber Variation; JAK-STAT/ERK signaling; Prolactin; Prolactin Receptor

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APA (6th Edition):

Neradugomma, N. K. (2014). Role of Prolactin and Prolactin Receptor Signaling in Colorectal Tumorigenesis. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/19628

Chicago Manual of Style (16th Edition):

Neradugomma, Naveen Kumar. “Role of Prolactin and Prolactin Receptor Signaling in Colorectal Tumorigenesis.” 2014. Doctoral Dissertation, University of Kansas. Accessed April 19, 2019. http://hdl.handle.net/1808/19628.

MLA Handbook (7th Edition):

Neradugomma, Naveen Kumar. “Role of Prolactin and Prolactin Receptor Signaling in Colorectal Tumorigenesis.” 2014. Web. 19 Apr 2019.

Vancouver:

Neradugomma NK. Role of Prolactin and Prolactin Receptor Signaling in Colorectal Tumorigenesis. [Internet] [Doctoral dissertation]. University of Kansas; 2014. [cited 2019 Apr 19]. Available from: http://hdl.handle.net/1808/19628.

Council of Science Editors:

Neradugomma NK. Role of Prolactin and Prolactin Receptor Signaling in Colorectal Tumorigenesis. [Doctoral Dissertation]. University of Kansas; 2014. Available from: http://hdl.handle.net/1808/19628


University of Kansas

7. Clarke, Gwenaelle Laetitia. Regulation of sympathetic plasticity in the heart.

Degree: PhD, Molecular & Integrative Physiology, 2009, University of Kansas

 Following myocardial infarction, elevated sympathetic neuronal sprouting at the infarct border region leads to hyperexcitability which increases the risk of arrhythmias and sudden cardiac death.… (more)

Subjects/Keywords: Biology; Neurosciences; Animal physiology; Molecular biology; Adrenergic receptors; Myocardial infarction; Outgrowth; Plasticity; Sympathetic innervation

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APA (6th Edition):

Clarke, G. L. (2009). Regulation of sympathetic plasticity in the heart. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/6629

Chicago Manual of Style (16th Edition):

Clarke, Gwenaelle Laetitia. “Regulation of sympathetic plasticity in the heart.” 2009. Doctoral Dissertation, University of Kansas. Accessed April 19, 2019. http://hdl.handle.net/1808/6629.

MLA Handbook (7th Edition):

Clarke, Gwenaelle Laetitia. “Regulation of sympathetic plasticity in the heart.” 2009. Web. 19 Apr 2019.

Vancouver:

Clarke GL. Regulation of sympathetic plasticity in the heart. [Internet] [Doctoral dissertation]. University of Kansas; 2009. [cited 2019 Apr 19]. Available from: http://hdl.handle.net/1808/6629.

Council of Science Editors:

Clarke GL. Regulation of sympathetic plasticity in the heart. [Doctoral Dissertation]. University of Kansas; 2009. Available from: http://hdl.handle.net/1808/6629


University of Kansas

8. Thomas, George Patrick, Jr. Neural Substrates of Insulin-Mediated Memory Facilitation in Early Alzheimer's Disease; The Impact of the Apolipoprotein E-Epsilon-4 Allele on Hippocampal Insulin Responses.

Degree: PhD, Molecular & Integrative Physiology, 2009, University of Kansas

 Background: Several studies have demonstrated that insulin delivered by nasal spray acutely improves cognitive performance in early Alzheimer's disease. Furthermore, the apolipoprotein E-episilon-4 allele, a… (more)

Subjects/Keywords: Biology; Neurosciences; Physiology; Health sciences; Medicine and surgery; Alzheimer's disease; Fmri; Insulin; Memory; Mri

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APA (6th Edition):

Thomas, George Patrick, J. (2009). Neural Substrates of Insulin-Mediated Memory Facilitation in Early Alzheimer's Disease; The Impact of the Apolipoprotein E-Epsilon-4 Allele on Hippocampal Insulin Responses. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/6020

Chicago Manual of Style (16th Edition):

Thomas, George Patrick, Jr. “Neural Substrates of Insulin-Mediated Memory Facilitation in Early Alzheimer's Disease; The Impact of the Apolipoprotein E-Epsilon-4 Allele on Hippocampal Insulin Responses.” 2009. Doctoral Dissertation, University of Kansas. Accessed April 19, 2019. http://hdl.handle.net/1808/6020.

MLA Handbook (7th Edition):

Thomas, George Patrick, Jr. “Neural Substrates of Insulin-Mediated Memory Facilitation in Early Alzheimer's Disease; The Impact of the Apolipoprotein E-Epsilon-4 Allele on Hippocampal Insulin Responses.” 2009. Web. 19 Apr 2019.

Vancouver:

Thomas, George Patrick J. Neural Substrates of Insulin-Mediated Memory Facilitation in Early Alzheimer's Disease; The Impact of the Apolipoprotein E-Epsilon-4 Allele on Hippocampal Insulin Responses. [Internet] [Doctoral dissertation]. University of Kansas; 2009. [cited 2019 Apr 19]. Available from: http://hdl.handle.net/1808/6020.

Council of Science Editors:

Thomas, George Patrick J. Neural Substrates of Insulin-Mediated Memory Facilitation in Early Alzheimer's Disease; The Impact of the Apolipoprotein E-Epsilon-4 Allele on Hippocampal Insulin Responses. [Doctoral Dissertation]. University of Kansas; 2009. Available from: http://hdl.handle.net/1808/6020


University of Kansas

9. Saito, Chieko. PROTECTIVE STRATEGIES AGAINST ACETAMINOPHEN INDUCED HEPATOTOXICITY.

Degree: PhD, Pharmacology, Toxicology & Therapeutics, 2010, University of Kansas

 Acetaminophen (APAP) is a widely used analgesic, which is safe at therapeutic levels. APAP is mainly conjugated with glucuronic acid and sulfate to form water-soluble,… (more)

Subjects/Keywords: Health sciences; Toxicology; Acetaminophen; C-jun n-terminal kinase; Glutathione; Metallothionein; N-acetylcysteine

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APA (6th Edition):

Saito, C. (2010). PROTECTIVE STRATEGIES AGAINST ACETAMINOPHEN INDUCED HEPATOTOXICITY. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/6369

Chicago Manual of Style (16th Edition):

Saito, Chieko. “PROTECTIVE STRATEGIES AGAINST ACETAMINOPHEN INDUCED HEPATOTOXICITY.” 2010. Doctoral Dissertation, University of Kansas. Accessed April 19, 2019. http://hdl.handle.net/1808/6369.

MLA Handbook (7th Edition):

Saito, Chieko. “PROTECTIVE STRATEGIES AGAINST ACETAMINOPHEN INDUCED HEPATOTOXICITY.” 2010. Web. 19 Apr 2019.

Vancouver:

Saito C. PROTECTIVE STRATEGIES AGAINST ACETAMINOPHEN INDUCED HEPATOTOXICITY. [Internet] [Doctoral dissertation]. University of Kansas; 2010. [cited 2019 Apr 19]. Available from: http://hdl.handle.net/1808/6369.

Council of Science Editors:

Saito C. PROTECTIVE STRATEGIES AGAINST ACETAMINOPHEN INDUCED HEPATOTOXICITY. [Doctoral Dissertation]. University of Kansas; 2010. Available from: http://hdl.handle.net/1808/6369


University of Kansas

10. O'Bryhim, Bliss. Genetic Susceptibility to the Murine Model of Retinopathy of Prematurity: Identification of a Novel Role of Tyrosinase in Retinal Angiogenic Regulation.

Degree: PhD, Molecular & Integrative Physiology, 2013, University of Kansas

 Retinopathy of Prematurity (ROP) is a major cause of vision loss in the pediatric population. A growing body of evidence suggests that a heritable component… (more)

Subjects/Keywords: Ophthalmology; angiogenesis; endothelial progenitor cells; genetics; oxygen induced retinopathy; retinopathy of prematurity

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APA (6th Edition):

O'Bryhim, B. (2013). Genetic Susceptibility to the Murine Model of Retinopathy of Prematurity: Identification of a Novel Role of Tyrosinase in Retinal Angiogenic Regulation. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/19626

Chicago Manual of Style (16th Edition):

O'Bryhim, Bliss. “Genetic Susceptibility to the Murine Model of Retinopathy of Prematurity: Identification of a Novel Role of Tyrosinase in Retinal Angiogenic Regulation.” 2013. Doctoral Dissertation, University of Kansas. Accessed April 19, 2019. http://hdl.handle.net/1808/19626.

MLA Handbook (7th Edition):

O'Bryhim, Bliss. “Genetic Susceptibility to the Murine Model of Retinopathy of Prematurity: Identification of a Novel Role of Tyrosinase in Retinal Angiogenic Regulation.” 2013. Web. 19 Apr 2019.

Vancouver:

O'Bryhim B. Genetic Susceptibility to the Murine Model of Retinopathy of Prematurity: Identification of a Novel Role of Tyrosinase in Retinal Angiogenic Regulation. [Internet] [Doctoral dissertation]. University of Kansas; 2013. [cited 2019 Apr 19]. Available from: http://hdl.handle.net/1808/19626.

Council of Science Editors:

O'Bryhim B. Genetic Susceptibility to the Murine Model of Retinopathy of Prematurity: Identification of a Novel Role of Tyrosinase in Retinal Angiogenic Regulation. [Doctoral Dissertation]. University of Kansas; 2013. Available from: http://hdl.handle.net/1808/19626


University of Kansas

11. Jansson, Kyle. Novel effects of ouabain in autosomal dominant polycystic kidney disease cystogenesis.

Degree: PhD, Molecular & Integrative Physiology, 2013, University of Kansas

 Autosomal dominant polycystic kidney disease (ADPKD) is caused by mutations in Pkd1 or Pkd2, genes encoding for polycystin-1 (PC-1) and polycystin-2 (PC-2), respectively. ADPKD is… (more)

Subjects/Keywords: Physiology; Medicine; ADPKD; Na; K-ATPase; ouabain

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APA (6th Edition):

Jansson, K. (2013). Novel effects of ouabain in autosomal dominant polycystic kidney disease cystogenesis. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/19625

Chicago Manual of Style (16th Edition):

Jansson, Kyle. “Novel effects of ouabain in autosomal dominant polycystic kidney disease cystogenesis.” 2013. Doctoral Dissertation, University of Kansas. Accessed April 19, 2019. http://hdl.handle.net/1808/19625.

MLA Handbook (7th Edition):

Jansson, Kyle. “Novel effects of ouabain in autosomal dominant polycystic kidney disease cystogenesis.” 2013. Web. 19 Apr 2019.

Vancouver:

Jansson K. Novel effects of ouabain in autosomal dominant polycystic kidney disease cystogenesis. [Internet] [Doctoral dissertation]. University of Kansas; 2013. [cited 2019 Apr 19]. Available from: http://hdl.handle.net/1808/19625.

Council of Science Editors:

Jansson K. Novel effects of ouabain in autosomal dominant polycystic kidney disease cystogenesis. [Doctoral Dissertation]. University of Kansas; 2013. Available from: http://hdl.handle.net/1808/19625


University of Kansas

12. Cantilena, Amy Rose. The Role of Endoglin in the Immunomodulatory Capacities of Mesenchymal Stem Cells and the Relationship to Hyperbaric Oxygen Therapy.

Degree: PhD, Molecular & Integrative Physiology, 2017, University of Kansas

 Mesenchymal stem cells (MSCs), a type of “adult” stem cell found in most organs, represent an emerging tool in the field of regenerative medicine. In… (more)

Subjects/Keywords: Physiology; Immunology; Cell Therapy; Differentiation; Hyperbaric Oxygen Therapy; Immunomodulation; Mesenchymal Stem Cells; Transplant

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APA (6th Edition):

Cantilena, A. R. (2017). The Role of Endoglin in the Immunomodulatory Capacities of Mesenchymal Stem Cells and the Relationship to Hyperbaric Oxygen Therapy. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/25872

Chicago Manual of Style (16th Edition):

Cantilena, Amy Rose. “The Role of Endoglin in the Immunomodulatory Capacities of Mesenchymal Stem Cells and the Relationship to Hyperbaric Oxygen Therapy.” 2017. Doctoral Dissertation, University of Kansas. Accessed April 19, 2019. http://hdl.handle.net/1808/25872.

MLA Handbook (7th Edition):

Cantilena, Amy Rose. “The Role of Endoglin in the Immunomodulatory Capacities of Mesenchymal Stem Cells and the Relationship to Hyperbaric Oxygen Therapy.” 2017. Web. 19 Apr 2019.

Vancouver:

Cantilena AR. The Role of Endoglin in the Immunomodulatory Capacities of Mesenchymal Stem Cells and the Relationship to Hyperbaric Oxygen Therapy. [Internet] [Doctoral dissertation]. University of Kansas; 2017. [cited 2019 Apr 19]. Available from: http://hdl.handle.net/1808/25872.

Council of Science Editors:

Cantilena AR. The Role of Endoglin in the Immunomodulatory Capacities of Mesenchymal Stem Cells and the Relationship to Hyperbaric Oxygen Therapy. [Doctoral Dissertation]. University of Kansas; 2017. Available from: http://hdl.handle.net/1808/25872


University of Kansas

13. Venugopal, Jessica Dawn. Contributions of Ouabain to the Autosomal Dominant Polycystic Kidney Disease Phenotype.

Degree: PhD, Molecular & Integrative Physiology, 2016, University of Kansas

 Autosomal dominant polycystic kidney disease (ADPKD) is a genetic disorder, caused by mutations in either of the PKD1 or PKD2 genes, which leads to the… (more)

Subjects/Keywords: Physiology; Apoptosis; Calcium; EMT; L-type calcium channels; Na; K-ATPase; Polycystins

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APA (6th Edition):

Venugopal, J. D. (2016). Contributions of Ouabain to the Autosomal Dominant Polycystic Kidney Disease Phenotype. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/24813

Chicago Manual of Style (16th Edition):

Venugopal, Jessica Dawn. “Contributions of Ouabain to the Autosomal Dominant Polycystic Kidney Disease Phenotype.” 2016. Doctoral Dissertation, University of Kansas. Accessed April 19, 2019. http://hdl.handle.net/1808/24813.

MLA Handbook (7th Edition):

Venugopal, Jessica Dawn. “Contributions of Ouabain to the Autosomal Dominant Polycystic Kidney Disease Phenotype.” 2016. Web. 19 Apr 2019.

Vancouver:

Venugopal JD. Contributions of Ouabain to the Autosomal Dominant Polycystic Kidney Disease Phenotype. [Internet] [Doctoral dissertation]. University of Kansas; 2016. [cited 2019 Apr 19]. Available from: http://hdl.handle.net/1808/24813.

Council of Science Editors:

Venugopal JD. Contributions of Ouabain to the Autosomal Dominant Polycystic Kidney Disease Phenotype. [Doctoral Dissertation]. University of Kansas; 2016. Available from: http://hdl.handle.net/1808/24813


University of Kansas

14. Chao, Jie. MECHANISMS OF MICROVASCULAR INFLAMMATION INDUCED BY ALVEOLAR HYPOXIA.

Degree: PhD, Molecular & Integrative Physiology, 2010, University of Kansas

 Alveolar hypoxia is observed in a number of clinical settings, and is frequently associated with systemic effects, many of which present an inflammatory component. Reduction… (more)

Subjects/Keywords: Biology; Physiology; Alveolar macrophage; Hypoxia; Inflammation; Mast cell; Mcp-1; Microcirculation

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APA (6th Edition):

Chao, J. (2010). MECHANISMS OF MICROVASCULAR INFLAMMATION INDUCED BY ALVEOLAR HYPOXIA. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/7425

Chicago Manual of Style (16th Edition):

Chao, Jie. “MECHANISMS OF MICROVASCULAR INFLAMMATION INDUCED BY ALVEOLAR HYPOXIA.” 2010. Doctoral Dissertation, University of Kansas. Accessed April 19, 2019. http://hdl.handle.net/1808/7425.

MLA Handbook (7th Edition):

Chao, Jie. “MECHANISMS OF MICROVASCULAR INFLAMMATION INDUCED BY ALVEOLAR HYPOXIA.” 2010. Web. 19 Apr 2019.

Vancouver:

Chao J. MECHANISMS OF MICROVASCULAR INFLAMMATION INDUCED BY ALVEOLAR HYPOXIA. [Internet] [Doctoral dissertation]. University of Kansas; 2010. [cited 2019 Apr 19]. Available from: http://hdl.handle.net/1808/7425.

Council of Science Editors:

Chao J. MECHANISMS OF MICROVASCULAR INFLAMMATION INDUCED BY ALVEOLAR HYPOXIA. [Doctoral Dissertation]. University of Kansas; 2010. Available from: http://hdl.handle.net/1808/7425

15. Sullivan, Bradley P. Role of Coagulation in Xenobiotic-Induced Liver Injury.

Degree: PhD, Pharmacology, Toxicology & Therapeutics, 2012, University of Kansas

 The liver is a common target for xenobiotic-induced toxicity. Of importance, synthesis of soluble coagulation factors by the liver plays an essential role in hemostasis.… (more)

Subjects/Keywords: Toxicology; Acetaminophen; Alpha-naphthylisothiocyanate; Blood coagulation; Fibrosis; Liver injury; Platelet

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APA (6th Edition):

Sullivan, B. P. (2012). Role of Coagulation in Xenobiotic-Induced Liver Injury. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/10291

Chicago Manual of Style (16th Edition):

Sullivan, Bradley P. “Role of Coagulation in Xenobiotic-Induced Liver Injury.” 2012. Doctoral Dissertation, University of Kansas. Accessed April 19, 2019. http://hdl.handle.net/1808/10291.

MLA Handbook (7th Edition):

Sullivan, Bradley P. “Role of Coagulation in Xenobiotic-Induced Liver Injury.” 2012. Web. 19 Apr 2019.

Vancouver:

Sullivan BP. Role of Coagulation in Xenobiotic-Induced Liver Injury. [Internet] [Doctoral dissertation]. University of Kansas; 2012. [cited 2019 Apr 19]. Available from: http://hdl.handle.net/1808/10291.

Council of Science Editors:

Sullivan BP. Role of Coagulation in Xenobiotic-Induced Liver Injury. [Doctoral Dissertation]. University of Kansas; 2012. Available from: http://hdl.handle.net/1808/10291

16. Coyan, Garrett Nicholas. Induced Hypothermia During Resuscitation from Hemorrhagic Shock Attenuates Microvascular Inflammation in the Rat Mesenteric Microcirculation.

Degree: MS, Clinical Research, 2013, University of Kansas

 Introduction: Hemorrhagic shock is a major cause of morbidity and mortality in trauma patients. Microvascular inflammation occurs during resuscitation following hemorrhagic shock, and is a… (more)

Subjects/Keywords: Physiology; Surgery; Hemorrhagic shock; Hypothermia; Microvascular inflammation; Resuscitation

…and Use Committee of the University of Kansas Medical Center. The University of Kansas… 

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Coyan, G. N. (2013). Induced Hypothermia During Resuscitation from Hemorrhagic Shock Attenuates Microvascular Inflammation in the Rat Mesenteric Microcirculation. (Masters Thesis). University of Kansas. Retrieved from http://hdl.handle.net/1808/12165

Chicago Manual of Style (16th Edition):

Coyan, Garrett Nicholas. “Induced Hypothermia During Resuscitation from Hemorrhagic Shock Attenuates Microvascular Inflammation in the Rat Mesenteric Microcirculation.” 2013. Masters Thesis, University of Kansas. Accessed April 19, 2019. http://hdl.handle.net/1808/12165.

MLA Handbook (7th Edition):

Coyan, Garrett Nicholas. “Induced Hypothermia During Resuscitation from Hemorrhagic Shock Attenuates Microvascular Inflammation in the Rat Mesenteric Microcirculation.” 2013. Web. 19 Apr 2019.

Vancouver:

Coyan GN. Induced Hypothermia During Resuscitation from Hemorrhagic Shock Attenuates Microvascular Inflammation in the Rat Mesenteric Microcirculation. [Internet] [Masters thesis]. University of Kansas; 2013. [cited 2019 Apr 19]. Available from: http://hdl.handle.net/1808/12165.

Council of Science Editors:

Coyan GN. Induced Hypothermia During Resuscitation from Hemorrhagic Shock Attenuates Microvascular Inflammation in the Rat Mesenteric Microcirculation. [Masters Thesis]. University of Kansas; 2013. Available from: http://hdl.handle.net/1808/12165

17. Quinn, Carrie. EXERCISE-INDUCED ORGAN PROTECTION IN A MURINE MODEL OF CARDIAC ARREST AND RESUSCITATION.

Degree: PhD, Molecular & Integrative Physiology, 2012, University of Kansas

 Cardiac arrest causes whole-body ischemic injury and cell death. Successful cardiopulmonary resuscitation paradoxically confounds recovery by increasing the rate of cellular death and tissue damage… (more)

Subjects/Keywords: Physiology; Barnes maze; Cardiac arrest; Cpr; Exercise preconditioning; Neurologic function

…approved by the University of Kansas Medical Center Institutional Animal Care and Use Committee… 

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Quinn, C. (2012). EXERCISE-INDUCED ORGAN PROTECTION IN A MURINE MODEL OF CARDIAC ARREST AND RESUSCITATION. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/10247

Chicago Manual of Style (16th Edition):

Quinn, Carrie. “EXERCISE-INDUCED ORGAN PROTECTION IN A MURINE MODEL OF CARDIAC ARREST AND RESUSCITATION.” 2012. Doctoral Dissertation, University of Kansas. Accessed April 19, 2019. http://hdl.handle.net/1808/10247.

MLA Handbook (7th Edition):

Quinn, Carrie. “EXERCISE-INDUCED ORGAN PROTECTION IN A MURINE MODEL OF CARDIAC ARREST AND RESUSCITATION.” 2012. Web. 19 Apr 2019.

Vancouver:

Quinn C. EXERCISE-INDUCED ORGAN PROTECTION IN A MURINE MODEL OF CARDIAC ARREST AND RESUSCITATION. [Internet] [Doctoral dissertation]. University of Kansas; 2012. [cited 2019 Apr 19]. Available from: http://hdl.handle.net/1808/10247.

Council of Science Editors:

Quinn C. EXERCISE-INDUCED ORGAN PROTECTION IN A MURINE MODEL OF CARDIAC ARREST AND RESUSCITATION. [Doctoral Dissertation]. University of Kansas; 2012. Available from: http://hdl.handle.net/1808/10247

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