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You searched for +publisher:"University of Kansas" +contributor:("Staudinger, Jeff L."). Showing records 1 – 12 of 12 total matches.

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University of Kansas

1. Chen, Yao. Mass Spectrometry-Based Quantitative Proteomics for Drug-Metabolizing Enzymes.

Degree: PhD, Pharmaceutical Chemistry, 2016, University of Kansas

 Quantification of highly homologous human liver drug-metabolizing enzymes (DMEs) has been a challenging task in drug metabolism and disposition research, due to a lack of… (more)

Subjects/Keywords: Pharmaceutical sciences; Analytical chemistry; Drug-Metabolizing Enzymes; Liquid Chromatography; Mass Spectrometry Quantification; nanoUPLC-Q-TOF Data Independent Acquisition; Quantitative FASP; UPLC-MRM Targeted Proteomics

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APA (6th Edition):

Chen, Y. (2016). Mass Spectrometry-Based Quantitative Proteomics for Drug-Metabolizing Enzymes. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/25430

Chicago Manual of Style (16th Edition):

Chen, Yao. “Mass Spectrometry-Based Quantitative Proteomics for Drug-Metabolizing Enzymes.” 2016. Doctoral Dissertation, University of Kansas. Accessed January 26, 2021. http://hdl.handle.net/1808/25430.

MLA Handbook (7th Edition):

Chen, Yao. “Mass Spectrometry-Based Quantitative Proteomics for Drug-Metabolizing Enzymes.” 2016. Web. 26 Jan 2021.

Vancouver:

Chen Y. Mass Spectrometry-Based Quantitative Proteomics for Drug-Metabolizing Enzymes. [Internet] [Doctoral dissertation]. University of Kansas; 2016. [cited 2021 Jan 26]. Available from: http://hdl.handle.net/1808/25430.

Council of Science Editors:

Chen Y. Mass Spectrometry-Based Quantitative Proteomics for Drug-Metabolizing Enzymes. [Doctoral Dissertation]. University of Kansas; 2016. Available from: http://hdl.handle.net/1808/25430

2. Xu, Chenshu. REGULATION OF DRUG METABOLISM AND INFLAMMATION BY PREGNANE X RECEPTOR.

Degree: PhD, Pharmacology & Toxicology, 2011, University of Kansas

 Liver-enriched nuclear receptor (NR) proteins regulate the expression and activity of several pivotal hepatic biochemical pathways including the uptake, metabolism and excretion of cholesterol, bile… (more)

Subjects/Keywords: Pharmacology; Toxicology; Carboxylesterase; Constitutive androstane receptor; Inflammation; Pregnane x receptor; Sumoylation

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APA (6th Edition):

Xu, C. (2011). REGULATION OF DRUG METABOLISM AND INFLAMMATION BY PREGNANE X RECEPTOR. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/9708

Chicago Manual of Style (16th Edition):

Xu, Chenshu. “REGULATION OF DRUG METABOLISM AND INFLAMMATION BY PREGNANE X RECEPTOR.” 2011. Doctoral Dissertation, University of Kansas. Accessed January 26, 2021. http://hdl.handle.net/1808/9708.

MLA Handbook (7th Edition):

Xu, Chenshu. “REGULATION OF DRUG METABOLISM AND INFLAMMATION BY PREGNANE X RECEPTOR.” 2011. Web. 26 Jan 2021.

Vancouver:

Xu C. REGULATION OF DRUG METABOLISM AND INFLAMMATION BY PREGNANE X RECEPTOR. [Internet] [Doctoral dissertation]. University of Kansas; 2011. [cited 2021 Jan 26]. Available from: http://hdl.handle.net/1808/9708.

Council of Science Editors:

Xu C. REGULATION OF DRUG METABOLISM AND INFLAMMATION BY PREGNANE X RECEPTOR. [Doctoral Dissertation]. University of Kansas; 2011. Available from: http://hdl.handle.net/1808/9708


University of Kansas

3. Pan, Pan. Differential expression of neuregulin-1 isoforms and downregulation of erbin are associated with Erb B2 receptor activation in diabetic peripheral neuropathy.

Degree: PhD, Pharmacology & Toxicology, 2014, University of Kansas

 Aberrant neuron-glia interactions can contribute to a variety of neurodegenerative diseases. We have previously demonstrated that enhanced activation of Erb B2, which is a member… (more)

Subjects/Keywords: Pharmacology; Erlotinib; Intra epidermal nerve fibers; Nerve conduction velocity; Sensory hypoalgesia

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APA (6th Edition):

Pan, P. (2014). Differential expression of neuregulin-1 isoforms and downregulation of erbin are associated with Erb B2 receptor activation in diabetic peripheral neuropathy. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/21637

Chicago Manual of Style (16th Edition):

Pan, Pan. “Differential expression of neuregulin-1 isoforms and downregulation of erbin are associated with Erb B2 receptor activation in diabetic peripheral neuropathy.” 2014. Doctoral Dissertation, University of Kansas. Accessed January 26, 2021. http://hdl.handle.net/1808/21637.

MLA Handbook (7th Edition):

Pan, Pan. “Differential expression of neuregulin-1 isoforms and downregulation of erbin are associated with Erb B2 receptor activation in diabetic peripheral neuropathy.” 2014. Web. 26 Jan 2021.

Vancouver:

Pan P. Differential expression of neuregulin-1 isoforms and downregulation of erbin are associated with Erb B2 receptor activation in diabetic peripheral neuropathy. [Internet] [Doctoral dissertation]. University of Kansas; 2014. [cited 2021 Jan 26]. Available from: http://hdl.handle.net/1808/21637.

Council of Science Editors:

Pan P. Differential expression of neuregulin-1 isoforms and downregulation of erbin are associated with Erb B2 receptor activation in diabetic peripheral neuropathy. [Doctoral Dissertation]. University of Kansas; 2014. Available from: http://hdl.handle.net/1808/21637

4. Lichti-Kaiser, Kristin Nicole. Regulation of the Pregnane X Receptor Signaling Pathway.

Degree: PhD, Pharmacology & Toxicology, 2009, University of Kansas

 Liver-enriched nuclear receptors (NRs) collectively function as metabolic and toxicological `sensors' that mediate liver-specific gene-activation in mammals. NR-mediated gene-environment interaction regulates important steps in the… (more)

Subjects/Keywords: Health sciences; Pharmacology; Cell signaling; Gene expression; Pregnane x receptor

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APA (6th Edition):

Lichti-Kaiser, K. N. (2009). Regulation of the Pregnane X Receptor Signaling Pathway. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/6188

Chicago Manual of Style (16th Edition):

Lichti-Kaiser, Kristin Nicole. “Regulation of the Pregnane X Receptor Signaling Pathway.” 2009. Doctoral Dissertation, University of Kansas. Accessed January 26, 2021. http://hdl.handle.net/1808/6188.

MLA Handbook (7th Edition):

Lichti-Kaiser, Kristin Nicole. “Regulation of the Pregnane X Receptor Signaling Pathway.” 2009. Web. 26 Jan 2021.

Vancouver:

Lichti-Kaiser KN. Regulation of the Pregnane X Receptor Signaling Pathway. [Internet] [Doctoral dissertation]. University of Kansas; 2009. [cited 2021 Jan 26]. Available from: http://hdl.handle.net/1808/6188.

Council of Science Editors:

Lichti-Kaiser KN. Regulation of the Pregnane X Receptor Signaling Pathway. [Doctoral Dissertation]. University of Kansas; 2009. Available from: http://hdl.handle.net/1808/6188

5. Sun, Mengxi. UBIQUITINATION AND SUMOYLATION OF PREGNANE X RECEPTOR.

Degree: PhD, Pharmacology & Toxicology, 2015, University of Kansas

 Abstract Pregnane X receptor (PXR, NR1I2) is a ligand-activated nuclear receptor (NR) superfamily member expressed at high levels in the liver and intestine of mammals.… (more)

Subjects/Keywords: Pharmacology; Toxicology; PXR; SUMOylation; ubiquitination

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APA (6th Edition):

Sun, M. (2015). UBIQUITINATION AND SUMOYLATION OF PREGNANE X RECEPTOR. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/21668

Chicago Manual of Style (16th Edition):

Sun, Mengxi. “UBIQUITINATION AND SUMOYLATION OF PREGNANE X RECEPTOR.” 2015. Doctoral Dissertation, University of Kansas. Accessed January 26, 2021. http://hdl.handle.net/1808/21668.

MLA Handbook (7th Edition):

Sun, Mengxi. “UBIQUITINATION AND SUMOYLATION OF PREGNANE X RECEPTOR.” 2015. Web. 26 Jan 2021.

Vancouver:

Sun M. UBIQUITINATION AND SUMOYLATION OF PREGNANE X RECEPTOR. [Internet] [Doctoral dissertation]. University of Kansas; 2015. [cited 2021 Jan 26]. Available from: http://hdl.handle.net/1808/21668.

Council of Science Editors:

Sun M. UBIQUITINATION AND SUMOYLATION OF PREGNANE X RECEPTOR. [Doctoral Dissertation]. University of Kansas; 2015. Available from: http://hdl.handle.net/1808/21668

6. Zhang, Liang. Pharmacological Induction of Molecular Chaperones Restores Mitochondrial Function in Hyperglycemically Stressed Sensory Neurons.

Degree: PhD, Pharmacology & Toxicology, 2012, University of Kansas

 Distal diabetic peripheral polyneuropathy (DPN) is a prevalent complication resulting from chronic hyperglycemia in diabetic patients. It is associated with incapacitating pain, foot ulceration, and… (more)

Subjects/Keywords: Pharmacology; Chaperones; Diabetic neuropathy; Hyperglycemia; Mitochodrial dysfunction; Oxidative stress; Sensory neurons

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APA (6th Edition):

Zhang, L. (2012). Pharmacological Induction of Molecular Chaperones Restores Mitochondrial Function in Hyperglycemically Stressed Sensory Neurons. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/10622

Chicago Manual of Style (16th Edition):

Zhang, Liang. “Pharmacological Induction of Molecular Chaperones Restores Mitochondrial Function in Hyperglycemically Stressed Sensory Neurons.” 2012. Doctoral Dissertation, University of Kansas. Accessed January 26, 2021. http://hdl.handle.net/1808/10622.

MLA Handbook (7th Edition):

Zhang, Liang. “Pharmacological Induction of Molecular Chaperones Restores Mitochondrial Function in Hyperglycemically Stressed Sensory Neurons.” 2012. Web. 26 Jan 2021.

Vancouver:

Zhang L. Pharmacological Induction of Molecular Chaperones Restores Mitochondrial Function in Hyperglycemically Stressed Sensory Neurons. [Internet] [Doctoral dissertation]. University of Kansas; 2012. [cited 2021 Jan 26]. Available from: http://hdl.handle.net/1808/10622.

Council of Science Editors:

Zhang L. Pharmacological Induction of Molecular Chaperones Restores Mitochondrial Function in Hyperglycemically Stressed Sensory Neurons. [Doctoral Dissertation]. University of Kansas; 2012. Available from: http://hdl.handle.net/1808/10622


University of Kansas

7. Zhang, Hongsheng. Identification and Biochemical Characterization of PGC-1beta-Interacting Proteins.

Degree: MS, Pharmacology & Toxicology, 2008, University of Kansas

 Nuclear receptors (NRs) are ligand-activated transcription factors. They regulate key biological processes including homeostasis of lipophilic molecules, organogenesis, development, and reproduction. The ability of NRs… (more)

Subjects/Keywords: Health sciences; Pharmacology; Toxicology; Baf60a; Metabolic syndrome; Pgc-1b-interacting proteins; Pgc-1beta; Pp4r1; Yeast two-hybrid screen

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APA (6th Edition):

Zhang, H. (2008). Identification and Biochemical Characterization of PGC-1beta-Interacting Proteins. (Masters Thesis). University of Kansas. Retrieved from http://hdl.handle.net/1808/4341

Chicago Manual of Style (16th Edition):

Zhang, Hongsheng. “Identification and Biochemical Characterization of PGC-1beta-Interacting Proteins.” 2008. Masters Thesis, University of Kansas. Accessed January 26, 2021. http://hdl.handle.net/1808/4341.

MLA Handbook (7th Edition):

Zhang, Hongsheng. “Identification and Biochemical Characterization of PGC-1beta-Interacting Proteins.” 2008. Web. 26 Jan 2021.

Vancouver:

Zhang H. Identification and Biochemical Characterization of PGC-1beta-Interacting Proteins. [Internet] [Masters thesis]. University of Kansas; 2008. [cited 2021 Jan 26]. Available from: http://hdl.handle.net/1808/4341.

Council of Science Editors:

Zhang H. Identification and Biochemical Characterization of PGC-1beta-Interacting Proteins. [Masters Thesis]. University of Kansas; 2008. Available from: http://hdl.handle.net/1808/4341


University of Kansas

8. Vasquez, Francisco Edmundo. Hyperglycemia Increases Mitochondrial Protein Expression in the Absence of Oxidative Stress.

Degree: MS, Pharmacology & Toxicology, 2008, University of Kansas

 Long-term diabetes may cause diabetic peripheral neuropathy (DPN). Hyperglycemic-induced oxidative stress is widely accepted as the contributing factor in the development of diabetic complications. The… (more)

Subjects/Keywords: Health sciences; Pharmacology; Toxicology; Diabetes; Oxidative stress; Schwann cells; Silac; Superoxide

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APA (6th Edition):

Vasquez, F. E. (2008). Hyperglycemia Increases Mitochondrial Protein Expression in the Absence of Oxidative Stress. (Masters Thesis). University of Kansas. Retrieved from http://hdl.handle.net/1808/4323

Chicago Manual of Style (16th Edition):

Vasquez, Francisco Edmundo. “Hyperglycemia Increases Mitochondrial Protein Expression in the Absence of Oxidative Stress.” 2008. Masters Thesis, University of Kansas. Accessed January 26, 2021. http://hdl.handle.net/1808/4323.

MLA Handbook (7th Edition):

Vasquez, Francisco Edmundo. “Hyperglycemia Increases Mitochondrial Protein Expression in the Absence of Oxidative Stress.” 2008. Web. 26 Jan 2021.

Vancouver:

Vasquez FE. Hyperglycemia Increases Mitochondrial Protein Expression in the Absence of Oxidative Stress. [Internet] [Masters thesis]. University of Kansas; 2008. [cited 2021 Jan 26]. Available from: http://hdl.handle.net/1808/4323.

Council of Science Editors:

Vasquez FE. Hyperglycemia Increases Mitochondrial Protein Expression in the Absence of Oxidative Stress. [Masters Thesis]. University of Kansas; 2008. Available from: http://hdl.handle.net/1808/4323


University of Kansas

9. Hughes, John Clayton. Disruption of Orolingual Behavior in Rats Treated with Atypical Antipsychotic Drugs.

Degree: MS, Pharmacology & Toxicology, 2008, University of Kansas

 The atypical antipsychotics are a group of second generation drugs used for the treatment of schizophrenia, schizoaffective disorder, as well as some forms of bipolar… (more)

Subjects/Keywords: Biology; Neurosciences; Antipsychotics; Clozapine; Extrapyramidal side effects; Hypoglossal; Orolingual; Power spectrum

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APA (6th Edition):

Hughes, J. C. (2008). Disruption of Orolingual Behavior in Rats Treated with Atypical Antipsychotic Drugs. (Masters Thesis). University of Kansas. Retrieved from http://hdl.handle.net/1808/4340

Chicago Manual of Style (16th Edition):

Hughes, John Clayton. “Disruption of Orolingual Behavior in Rats Treated with Atypical Antipsychotic Drugs.” 2008. Masters Thesis, University of Kansas. Accessed January 26, 2021. http://hdl.handle.net/1808/4340.

MLA Handbook (7th Edition):

Hughes, John Clayton. “Disruption of Orolingual Behavior in Rats Treated with Atypical Antipsychotic Drugs.” 2008. Web. 26 Jan 2021.

Vancouver:

Hughes JC. Disruption of Orolingual Behavior in Rats Treated with Atypical Antipsychotic Drugs. [Internet] [Masters thesis]. University of Kansas; 2008. [cited 2021 Jan 26]. Available from: http://hdl.handle.net/1808/4340.

Council of Science Editors:

Hughes JC. Disruption of Orolingual Behavior in Rats Treated with Atypical Antipsychotic Drugs. [Masters Thesis]. University of Kansas; 2008. Available from: http://hdl.handle.net/1808/4340


University of Kansas

10. Nair, Lakshmy. Development and validation of transgene constructs in order to generate bi-transgenic mice models for Diabetic Peripheral Neuropathy research.

Degree: MS, Pharmacology & Toxicology, 2010, University of Kansas

 Diabetic peripheral neuropathy (DPN), a common complication of diabetes, involves nerve damage in the arms and legs. Segmental demyelination is one of the basic patterns… (more)

Subjects/Keywords: Molecular biology; Biology; Neurosciences; Cell biology; Diabetic peripheral neuropathy; Schwann cells; Tet-on gene regulation system; Transgene construction; Transgene expression; Transgenic mouse model

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APA (6th Edition):

Nair, L. (2010). Development and validation of transgene constructs in order to generate bi-transgenic mice models for Diabetic Peripheral Neuropathy research. (Masters Thesis). University of Kansas. Retrieved from http://hdl.handle.net/1808/6300

Chicago Manual of Style (16th Edition):

Nair, Lakshmy. “Development and validation of transgene constructs in order to generate bi-transgenic mice models for Diabetic Peripheral Neuropathy research.” 2010. Masters Thesis, University of Kansas. Accessed January 26, 2021. http://hdl.handle.net/1808/6300.

MLA Handbook (7th Edition):

Nair, Lakshmy. “Development and validation of transgene constructs in order to generate bi-transgenic mice models for Diabetic Peripheral Neuropathy research.” 2010. Web. 26 Jan 2021.

Vancouver:

Nair L. Development and validation of transgene constructs in order to generate bi-transgenic mice models for Diabetic Peripheral Neuropathy research. [Internet] [Masters thesis]. University of Kansas; 2010. [cited 2021 Jan 26]. Available from: http://hdl.handle.net/1808/6300.

Council of Science Editors:

Nair L. Development and validation of transgene constructs in order to generate bi-transgenic mice models for Diabetic Peripheral Neuropathy research. [Masters Thesis]. University of Kansas; 2010. Available from: http://hdl.handle.net/1808/6300


University of Kansas

11. Song, Peizhen. EFFECT OF BILE ACID FEEDING AND SEQUESTRATION ON LIVER BILE ACID COMPOSITION AND GENE REGULATION IN MICE.

Degree: PhD, Pharmacology, Toxicology & Therapeutics, 2010, University of Kansas

 The dissertation investigates the non-hepatotoxic doses of five bile acids (BAs) in the feed of mice, as well as adaptations in the expression of genes… (more)

Subjects/Keywords: Toxicology; Pathology; Bile acids; Cholestyramine resin; Cyp7a1; Cyp8b1; Fgf15; Fxr

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APA (6th Edition):

Song, P. (2010). EFFECT OF BILE ACID FEEDING AND SEQUESTRATION ON LIVER BILE ACID COMPOSITION AND GENE REGULATION IN MICE. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/7756

Chicago Manual of Style (16th Edition):

Song, Peizhen. “EFFECT OF BILE ACID FEEDING AND SEQUESTRATION ON LIVER BILE ACID COMPOSITION AND GENE REGULATION IN MICE.” 2010. Doctoral Dissertation, University of Kansas. Accessed January 26, 2021. http://hdl.handle.net/1808/7756.

MLA Handbook (7th Edition):

Song, Peizhen. “EFFECT OF BILE ACID FEEDING AND SEQUESTRATION ON LIVER BILE ACID COMPOSITION AND GENE REGULATION IN MICE.” 2010. Web. 26 Jan 2021.

Vancouver:

Song P. EFFECT OF BILE ACID FEEDING AND SEQUESTRATION ON LIVER BILE ACID COMPOSITION AND GENE REGULATION IN MICE. [Internet] [Doctoral dissertation]. University of Kansas; 2010. [cited 2021 Jan 26]. Available from: http://hdl.handle.net/1808/7756.

Council of Science Editors:

Song P. EFFECT OF BILE ACID FEEDING AND SEQUESTRATION ON LIVER BILE ACID COMPOSITION AND GENE REGULATION IN MICE. [Doctoral Dissertation]. University of Kansas; 2010. Available from: http://hdl.handle.net/1808/7756


University of Kansas

12. Singh, Rakesh K. Novel Role of the JAK-STAT Pathway in Mediating the Effects of Atypical Antipsychotics on 5-HT2A Receptor Signaling.

Degree: PH.D., Pharmacology & Toxicology, 2008, University of Kansas

 The therapeutic benefits of atypical antipsychotics are proposed to be mediated by antagonism and subsequent desensitization of 5-HT2A receptor signaling; however, the mechanisms underlying this… (more)

Subjects/Keywords: Health sciences; Pharmacology

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APA (6th Edition):

Singh, R. K. (2008). Novel Role of the JAK-STAT Pathway in Mediating the Effects of Atypical Antipsychotics on 5-HT2A Receptor Signaling. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/5237

Chicago Manual of Style (16th Edition):

Singh, Rakesh K. “Novel Role of the JAK-STAT Pathway in Mediating the Effects of Atypical Antipsychotics on 5-HT2A Receptor Signaling.” 2008. Doctoral Dissertation, University of Kansas. Accessed January 26, 2021. http://hdl.handle.net/1808/5237.

MLA Handbook (7th Edition):

Singh, Rakesh K. “Novel Role of the JAK-STAT Pathway in Mediating the Effects of Atypical Antipsychotics on 5-HT2A Receptor Signaling.” 2008. Web. 26 Jan 2021.

Vancouver:

Singh RK. Novel Role of the JAK-STAT Pathway in Mediating the Effects of Atypical Antipsychotics on 5-HT2A Receptor Signaling. [Internet] [Doctoral dissertation]. University of Kansas; 2008. [cited 2021 Jan 26]. Available from: http://hdl.handle.net/1808/5237.

Council of Science Editors:

Singh RK. Novel Role of the JAK-STAT Pathway in Mediating the Effects of Atypical Antipsychotics on 5-HT2A Receptor Signaling. [Doctoral Dissertation]. University of Kansas; 2008. Available from: http://hdl.handle.net/1808/5237

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