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You searched for +publisher:"University of Kansas" +contributor:("Robertson, John D."). Showing records 1 – 6 of 6 total matches.

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University of Kansas

1. Karnes, Hope Elizabeth. Deafferentation-Induced Neuroplasticity in Chick Auditory Neurons: A Matter of Life or Death.

Degree: PhD, Anatomy & Cell Biology, 2009, University of Kansas

 Forty percent of elderly Americans suffer from hearing loss. Hearing loss is a significant public health concern, associated with social withdrawal, communication delays, and reduced… (more)

Subjects/Keywords: Biology; Neurosciences; Cell biology; Molecular biology; Apoptosis; Auditory system; Laser capture microdissection; Mitochondria; Sensory system

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Karnes, H. E. (2009). Deafferentation-Induced Neuroplasticity in Chick Auditory Neurons: A Matter of Life or Death. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/5574

Chicago Manual of Style (16th Edition):

Karnes, Hope Elizabeth. “Deafferentation-Induced Neuroplasticity in Chick Auditory Neurons: A Matter of Life or Death.” 2009. Doctoral Dissertation, University of Kansas. Accessed April 23, 2019. http://hdl.handle.net/1808/5574.

MLA Handbook (7th Edition):

Karnes, Hope Elizabeth. “Deafferentation-Induced Neuroplasticity in Chick Auditory Neurons: A Matter of Life or Death.” 2009. Web. 23 Apr 2019.

Vancouver:

Karnes HE. Deafferentation-Induced Neuroplasticity in Chick Auditory Neurons: A Matter of Life or Death. [Internet] [Doctoral dissertation]. University of Kansas; 2009. [cited 2019 Apr 23]. Available from: http://hdl.handle.net/1808/5574.

Council of Science Editors:

Karnes HE. Deafferentation-Induced Neuroplasticity in Chick Auditory Neurons: A Matter of Life or Death. [Doctoral Dissertation]. University of Kansas; 2009. Available from: http://hdl.handle.net/1808/5574


University of Kansas

2. Shawgo, Mary E. NEW INSIGHTS INTO THE REGULATION OF MITOCHONDRIAL OUTER MEMBRANE PERMEABILIZATION DURING APOPTOSIS.

Degree: PhD, Pharmacology, Toxicology & Therapeutics, 2009, University of Kansas

 Disruption of normal apoptosis can contribute to the onset of cancer. Additionally, many cancer drugs are effective for their ability to initiate the apoptotic process.… (more)

Subjects/Keywords: Health sciences; Pharmacology; Cell biology; Apoptosis; Cancer; Chemotherapy

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Shawgo, M. E. (2009). NEW INSIGHTS INTO THE REGULATION OF MITOCHONDRIAL OUTER MEMBRANE PERMEABILIZATION DURING APOPTOSIS. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/5943

Chicago Manual of Style (16th Edition):

Shawgo, Mary E. “NEW INSIGHTS INTO THE REGULATION OF MITOCHONDRIAL OUTER MEMBRANE PERMEABILIZATION DURING APOPTOSIS.” 2009. Doctoral Dissertation, University of Kansas. Accessed April 23, 2019. http://hdl.handle.net/1808/5943.

MLA Handbook (7th Edition):

Shawgo, Mary E. “NEW INSIGHTS INTO THE REGULATION OF MITOCHONDRIAL OUTER MEMBRANE PERMEABILIZATION DURING APOPTOSIS.” 2009. Web. 23 Apr 2019.

Vancouver:

Shawgo ME. NEW INSIGHTS INTO THE REGULATION OF MITOCHONDRIAL OUTER MEMBRANE PERMEABILIZATION DURING APOPTOSIS. [Internet] [Doctoral dissertation]. University of Kansas; 2009. [cited 2019 Apr 23]. Available from: http://hdl.handle.net/1808/5943.

Council of Science Editors:

Shawgo ME. NEW INSIGHTS INTO THE REGULATION OF MITOCHONDRIAL OUTER MEMBRANE PERMEABILIZATION DURING APOPTOSIS. [Doctoral Dissertation]. University of Kansas; 2009. Available from: http://hdl.handle.net/1808/5943


University of Kansas

3. Saito, Chieko. PROTECTIVE STRATEGIES AGAINST ACETAMINOPHEN INDUCED HEPATOTOXICITY.

Degree: PhD, Pharmacology, Toxicology & Therapeutics, 2010, University of Kansas

 Acetaminophen (APAP) is a widely used analgesic, which is safe at therapeutic levels. APAP is mainly conjugated with glucuronic acid and sulfate to form water-soluble,… (more)

Subjects/Keywords: Health sciences; Toxicology; Acetaminophen; C-jun n-terminal kinase; Glutathione; Metallothionein; N-acetylcysteine

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Saito, C. (2010). PROTECTIVE STRATEGIES AGAINST ACETAMINOPHEN INDUCED HEPATOTOXICITY. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/6369

Chicago Manual of Style (16th Edition):

Saito, Chieko. “PROTECTIVE STRATEGIES AGAINST ACETAMINOPHEN INDUCED HEPATOTOXICITY.” 2010. Doctoral Dissertation, University of Kansas. Accessed April 23, 2019. http://hdl.handle.net/1808/6369.

MLA Handbook (7th Edition):

Saito, Chieko. “PROTECTIVE STRATEGIES AGAINST ACETAMINOPHEN INDUCED HEPATOTOXICITY.” 2010. Web. 23 Apr 2019.

Vancouver:

Saito C. PROTECTIVE STRATEGIES AGAINST ACETAMINOPHEN INDUCED HEPATOTOXICITY. [Internet] [Doctoral dissertation]. University of Kansas; 2010. [cited 2019 Apr 23]. Available from: http://hdl.handle.net/1808/6369.

Council of Science Editors:

Saito C. PROTECTIVE STRATEGIES AGAINST ACETAMINOPHEN INDUCED HEPATOTOXICITY. [Doctoral Dissertation]. University of Kansas; 2010. Available from: http://hdl.handle.net/1808/6369


University of Kansas

4. Wang, Yang. NUCLEAR FUNCTIONS OF ADENOMATOUS POLYPOSIS COLI: REGULATION OF THE G2-M CELL CYCLE TRANSITION & INTERMEDIATE FILAMENT INTERACTION.

Degree: PhD, Molecular Biosciences, 2009, University of Kansas

 Mutations in Adenomatous Polyposis Coli (APC) initiate most colorectal cancers. APC is implicated in regulating cell cycle and the cytoskeleton. I identified topoisomerase IIalpha (topo… (more)

Subjects/Keywords: Cell biology; Molecular biology; Adenomatous polyposis coli; Cell cycle; G2 decatenation checkpoint; Intermediate filament; Lamin b1; Topoisomerase iialpha

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Wang, Y. (2009). NUCLEAR FUNCTIONS OF ADENOMATOUS POLYPOSIS COLI: REGULATION OF THE G2-M CELL CYCLE TRANSITION & INTERMEDIATE FILAMENT INTERACTION. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/5459

Chicago Manual of Style (16th Edition):

Wang, Yang. “NUCLEAR FUNCTIONS OF ADENOMATOUS POLYPOSIS COLI: REGULATION OF THE G2-M CELL CYCLE TRANSITION & INTERMEDIATE FILAMENT INTERACTION.” 2009. Doctoral Dissertation, University of Kansas. Accessed April 23, 2019. http://hdl.handle.net/1808/5459.

MLA Handbook (7th Edition):

Wang, Yang. “NUCLEAR FUNCTIONS OF ADENOMATOUS POLYPOSIS COLI: REGULATION OF THE G2-M CELL CYCLE TRANSITION & INTERMEDIATE FILAMENT INTERACTION.” 2009. Web. 23 Apr 2019.

Vancouver:

Wang Y. NUCLEAR FUNCTIONS OF ADENOMATOUS POLYPOSIS COLI: REGULATION OF THE G2-M CELL CYCLE TRANSITION & INTERMEDIATE FILAMENT INTERACTION. [Internet] [Doctoral dissertation]. University of Kansas; 2009. [cited 2019 Apr 23]. Available from: http://hdl.handle.net/1808/5459.

Council of Science Editors:

Wang Y. NUCLEAR FUNCTIONS OF ADENOMATOUS POLYPOSIS COLI: REGULATION OF THE G2-M CELL CYCLE TRANSITION & INTERMEDIATE FILAMENT INTERACTION. [Doctoral Dissertation]. University of Kansas; 2009. Available from: http://hdl.handle.net/1808/5459

5. Shelton, Shary Nicole. MECHANISMS OF MITOCHONDRIA-MEDIATED APOPTOSIS INDUCED BY CYTOTOXIC STRESS.

Degree: PhD, Pharmacology, Toxicology & Therapeutics, 2010, University of Kansas

 Defects within the apoptotic pathway are thought to contribute to tumorigenesis and therapeutic resistance. Although most cytotoxic anti-cancer drugs are thought to activate the mitochondria-mediated… (more)

Subjects/Keywords: Molecular biology; Apoptosis; DNA damage; Hsp90

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Shelton, S. N. (2010). MECHANISMS OF MITOCHONDRIA-MEDIATED APOPTOSIS INDUCED BY CYTOTOXIC STRESS. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/6761

Chicago Manual of Style (16th Edition):

Shelton, Shary Nicole. “MECHANISMS OF MITOCHONDRIA-MEDIATED APOPTOSIS INDUCED BY CYTOTOXIC STRESS.” 2010. Doctoral Dissertation, University of Kansas. Accessed April 23, 2019. http://hdl.handle.net/1808/6761.

MLA Handbook (7th Edition):

Shelton, Shary Nicole. “MECHANISMS OF MITOCHONDRIA-MEDIATED APOPTOSIS INDUCED BY CYTOTOXIC STRESS.” 2010. Web. 23 Apr 2019.

Vancouver:

Shelton SN. MECHANISMS OF MITOCHONDRIA-MEDIATED APOPTOSIS INDUCED BY CYTOTOXIC STRESS. [Internet] [Doctoral dissertation]. University of Kansas; 2010. [cited 2019 Apr 23]. Available from: http://hdl.handle.net/1808/6761.

Council of Science Editors:

Shelton SN. MECHANISMS OF MITOCHONDRIA-MEDIATED APOPTOSIS INDUCED BY CYTOTOXIC STRESS. [Doctoral Dissertation]. University of Kansas; 2010. Available from: http://hdl.handle.net/1808/6761


University of Kansas

6. Hontz, Adrianne Elizabeth. AURORA KINASES IN SOLELY ESTROGEN-INDUCED ONCOGENESIS: RELATION TO CENTROSOME AMPLIFICATION AND CHROMOSOMAL INSTABILITY.

Degree: PH.D., Pharmacology, Toxicology & Therapeutics, 2008, University of Kansas

 Estrogens play a crucial role in the causation and development of sporadic breast cancer, which accounts for ~ 90 - 95% of all breast cancer… (more)

Subjects/Keywords: Health sciences; Oncology; Pharmacology; Molecular biology; Breast cancer; Estrogen; Aurora kinases; Centrosome amplification; Chromosomal instability; Aneuploidy

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Hontz, A. E. (2008). AURORA KINASES IN SOLELY ESTROGEN-INDUCED ONCOGENESIS: RELATION TO CENTROSOME AMPLIFICATION AND CHROMOSOMAL INSTABILITY. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/4234

Chicago Manual of Style (16th Edition):

Hontz, Adrianne Elizabeth. “AURORA KINASES IN SOLELY ESTROGEN-INDUCED ONCOGENESIS: RELATION TO CENTROSOME AMPLIFICATION AND CHROMOSOMAL INSTABILITY.” 2008. Doctoral Dissertation, University of Kansas. Accessed April 23, 2019. http://hdl.handle.net/1808/4234.

MLA Handbook (7th Edition):

Hontz, Adrianne Elizabeth. “AURORA KINASES IN SOLELY ESTROGEN-INDUCED ONCOGENESIS: RELATION TO CENTROSOME AMPLIFICATION AND CHROMOSOMAL INSTABILITY.” 2008. Web. 23 Apr 2019.

Vancouver:

Hontz AE. AURORA KINASES IN SOLELY ESTROGEN-INDUCED ONCOGENESIS: RELATION TO CENTROSOME AMPLIFICATION AND CHROMOSOMAL INSTABILITY. [Internet] [Doctoral dissertation]. University of Kansas; 2008. [cited 2019 Apr 23]. Available from: http://hdl.handle.net/1808/4234.

Council of Science Editors:

Hontz AE. AURORA KINASES IN SOLELY ESTROGEN-INDUCED ONCOGENESIS: RELATION TO CENTROSOME AMPLIFICATION AND CHROMOSOMAL INSTABILITY. [Doctoral Dissertation]. University of Kansas; 2008. Available from: http://hdl.handle.net/1808/4234

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