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You searched for +publisher:"University of Kansas" +contributor:("Jaeschke, Hartmut"). Showing records 1 – 19 of 19 total matches.

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University of Kansas

1. Poudel, Samikshya. Role of Yes-Associated Protein (YAP) in Liver Injury and Regeneration following Acetaminophen Overdose.

Degree: MS, Pharmacology, Toxicology & Therapeutics, 2016, University of Kansas

 Acetaminophen (APAP) overdose is the major cause of Acute Liver Failure (ALF) in the Western world. Treatment options for APAP-induced ALF are limited. Studies have… (more)

Subjects/Keywords: Toxicology

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APA (6th Edition):

Poudel, S. (2016). Role of Yes-Associated Protein (YAP) in Liver Injury and Regeneration following Acetaminophen Overdose. (Masters Thesis). University of Kansas. Retrieved from http://hdl.handle.net/1808/24805

Chicago Manual of Style (16th Edition):

Poudel, Samikshya. “Role of Yes-Associated Protein (YAP) in Liver Injury and Regeneration following Acetaminophen Overdose.” 2016. Masters Thesis, University of Kansas. Accessed April 24, 2019. http://hdl.handle.net/1808/24805.

MLA Handbook (7th Edition):

Poudel, Samikshya. “Role of Yes-Associated Protein (YAP) in Liver Injury and Regeneration following Acetaminophen Overdose.” 2016. Web. 24 Apr 2019.

Vancouver:

Poudel S. Role of Yes-Associated Protein (YAP) in Liver Injury and Regeneration following Acetaminophen Overdose. [Internet] [Masters thesis]. University of Kansas; 2016. [cited 2019 Apr 24]. Available from: http://hdl.handle.net/1808/24805.

Council of Science Editors:

Poudel S. Role of Yes-Associated Protein (YAP) in Liver Injury and Regeneration following Acetaminophen Overdose. [Masters Thesis]. University of Kansas; 2016. Available from: http://hdl.handle.net/1808/24805


University of Kansas

2. McGreal, Steven. The Role of O-GlcNAc in Liver Injury and Regeneration.

Degree: PhD, Pharmacology, Toxicology & Therapeutics, 2017, University of Kansas

 O-GlcNAcylation is a covalent attachment of a single N-acetyl glucosamine to a serine or threonine residue of a protein. Unlike other forms of protein glycosylation,… (more)

Subjects/Keywords: Toxicology; Acetaminophen; Liver; O-GlcNAc; regeneration

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APA (6th Edition):

McGreal, S. (2017). The Role of O-GlcNAc in Liver Injury and Regeneration. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/27029

Chicago Manual of Style (16th Edition):

McGreal, Steven. “The Role of O-GlcNAc in Liver Injury and Regeneration.” 2017. Doctoral Dissertation, University of Kansas. Accessed April 24, 2019. http://hdl.handle.net/1808/27029.

MLA Handbook (7th Edition):

McGreal, Steven. “The Role of O-GlcNAc in Liver Injury and Regeneration.” 2017. Web. 24 Apr 2019.

Vancouver:

McGreal S. The Role of O-GlcNAc in Liver Injury and Regeneration. [Internet] [Doctoral dissertation]. University of Kansas; 2017. [cited 2019 Apr 24]. Available from: http://hdl.handle.net/1808/27029.

Council of Science Editors:

McGreal S. The Role of O-GlcNAc in Liver Injury and Regeneration. [Doctoral Dissertation]. University of Kansas; 2017. Available from: http://hdl.handle.net/1808/27029


University of Kansas

3. Allen, Katryn Miller. Mechanisms of Proinflammatory Signaling during Cholestasis.

Degree: PhD, Pharmacology, Toxicology & Therapeutics, 2011, University of Kansas

 Cholestasis is a condition in which bile flow from the liver to the intestines is inhibited. Loss of bile flow leads to increased concentrations of… (more)

Subjects/Keywords: Toxicology; Bile acids; Cholestasis; Early growth response factor-1; Inflammation

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APA (6th Edition):

Allen, K. M. (2011). Mechanisms of Proinflammatory Signaling during Cholestasis. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/7918

Chicago Manual of Style (16th Edition):

Allen, Katryn Miller. “Mechanisms of Proinflammatory Signaling during Cholestasis.” 2011. Doctoral Dissertation, University of Kansas. Accessed April 24, 2019. http://hdl.handle.net/1808/7918.

MLA Handbook (7th Edition):

Allen, Katryn Miller. “Mechanisms of Proinflammatory Signaling during Cholestasis.” 2011. Web. 24 Apr 2019.

Vancouver:

Allen KM. Mechanisms of Proinflammatory Signaling during Cholestasis. [Internet] [Doctoral dissertation]. University of Kansas; 2011. [cited 2019 Apr 24]. Available from: http://hdl.handle.net/1808/7918.

Council of Science Editors:

Allen KM. Mechanisms of Proinflammatory Signaling during Cholestasis. [Doctoral Dissertation]. University of Kansas; 2011. Available from: http://hdl.handle.net/1808/7918


University of Kansas

4. Woolbright, Ben. The role of bile acids during cholestasis in mice and man.

Degree: PhD, Pharmacology, Toxicology & Therapeutics, 2015, University of Kansas

 Cholestasis is a reduction in bile flow that occurs during numerous pathologies. Cholestasis leads to significant liver toxicity, biliary hyperplasia, and liver cirrhosis. The molecular… (more)

Subjects/Keywords: Toxicology; Pathology; Pharmacology; apoptosis; bile acid; cholestasis; hepatocytes; inflammation; neutrophil

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APA (6th Edition):

Woolbright, B. (2015). The role of bile acids during cholestasis in mice and man. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/19455

Chicago Manual of Style (16th Edition):

Woolbright, Ben. “The role of bile acids during cholestasis in mice and man.” 2015. Doctoral Dissertation, University of Kansas. Accessed April 24, 2019. http://hdl.handle.net/1808/19455.

MLA Handbook (7th Edition):

Woolbright, Ben. “The role of bile acids during cholestasis in mice and man.” 2015. Web. 24 Apr 2019.

Vancouver:

Woolbright B. The role of bile acids during cholestasis in mice and man. [Internet] [Doctoral dissertation]. University of Kansas; 2015. [cited 2019 Apr 24]. Available from: http://hdl.handle.net/1808/19455.

Council of Science Editors:

Woolbright B. The role of bile acids during cholestasis in mice and man. [Doctoral Dissertation]. University of Kansas; 2015. Available from: http://hdl.handle.net/1808/19455


University of Kansas

5. Xie, Yuchao. ACETAMINOPHEN HEPATOTOXICITY IN PRIMARY HUMAN HEPATOCYTES AND MICE.

Degree: PhD, Pharmacology, Toxicology & Therapeutics, 2016, University of Kansas

 Acetaminophen is the most prevalent cause of acute liver failure (ALF) and drug-induced liver injury (DILI) in western countries. Extensive studies have revealed important intracellular… (more)

Subjects/Keywords: Toxicology

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APA (6th Edition):

Xie, Y. (2016). ACETAMINOPHEN HEPATOTOXICITY IN PRIMARY HUMAN HEPATOCYTES AND MICE. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/22502

Chicago Manual of Style (16th Edition):

Xie, Yuchao. “ACETAMINOPHEN HEPATOTOXICITY IN PRIMARY HUMAN HEPATOCYTES AND MICE.” 2016. Doctoral Dissertation, University of Kansas. Accessed April 24, 2019. http://hdl.handle.net/1808/22502.

MLA Handbook (7th Edition):

Xie, Yuchao. “ACETAMINOPHEN HEPATOTOXICITY IN PRIMARY HUMAN HEPATOCYTES AND MICE.” 2016. Web. 24 Apr 2019.

Vancouver:

Xie Y. ACETAMINOPHEN HEPATOTOXICITY IN PRIMARY HUMAN HEPATOCYTES AND MICE. [Internet] [Doctoral dissertation]. University of Kansas; 2016. [cited 2019 Apr 24]. Available from: http://hdl.handle.net/1808/22502.

Council of Science Editors:

Xie Y. ACETAMINOPHEN HEPATOTOXICITY IN PRIMARY HUMAN HEPATOCYTES AND MICE. [Doctoral Dissertation]. University of Kansas; 2016. Available from: http://hdl.handle.net/1808/22502


University of Kansas

6. Du, Kuo. MITOCHONDRIAL OXIDATIVE STRESS AND BIOGENESIS DURING ACETAMINOPHEN HEPATOTOXICITY.

Degree: PhD, Pharmacology, Toxicology & Therapeutics, 2017, University of Kansas

 Acetaminophen (APAP) overdose causes severe hepatotoxicity in animals and humans. Although numerous studies have established the existence of an extensive oxidative stress during APAP hepatotoxicity,… (more)

Subjects/Keywords: Toxicology; Pharmacology; Molecular biology; Acetaminophen; liver toxicity; metformin; mitochondria; mito-tempo; oxidative stress

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APA (6th Edition):

Du, K. (2017). MITOCHONDRIAL OXIDATIVE STRESS AND BIOGENESIS DURING ACETAMINOPHEN HEPATOTOXICITY. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/26928

Chicago Manual of Style (16th Edition):

Du, Kuo. “MITOCHONDRIAL OXIDATIVE STRESS AND BIOGENESIS DURING ACETAMINOPHEN HEPATOTOXICITY.” 2017. Doctoral Dissertation, University of Kansas. Accessed April 24, 2019. http://hdl.handle.net/1808/26928.

MLA Handbook (7th Edition):

Du, Kuo. “MITOCHONDRIAL OXIDATIVE STRESS AND BIOGENESIS DURING ACETAMINOPHEN HEPATOTOXICITY.” 2017. Web. 24 Apr 2019.

Vancouver:

Du K. MITOCHONDRIAL OXIDATIVE STRESS AND BIOGENESIS DURING ACETAMINOPHEN HEPATOTOXICITY. [Internet] [Doctoral dissertation]. University of Kansas; 2017. [cited 2019 Apr 24]. Available from: http://hdl.handle.net/1808/26928.

Council of Science Editors:

Du K. MITOCHONDRIAL OXIDATIVE STRESS AND BIOGENESIS DURING ACETAMINOPHEN HEPATOTOXICITY. [Doctoral Dissertation]. University of Kansas; 2017. Available from: http://hdl.handle.net/1808/26928


University of Kansas

7. Weemhoff, James Lawrence. Mechanistic Biomarkers in Acute Liver Injury.

Degree: PhD, Pharmacology, Toxicology & Therapeutics, 2017, University of Kansas

 Acute liver failure continues to be a major medical problem. There are many underlying causes of acute liver failure, but drug induced liver injury is… (more)

Subjects/Keywords: Toxicology; Acetaminophen; Biomarkers; HepaRG; Hypoxic Hepatitis; Liver Injury; Liver Transplantation

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APA (6th Edition):

Weemhoff, J. L. (2017). Mechanistic Biomarkers in Acute Liver Injury. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/27044

Chicago Manual of Style (16th Edition):

Weemhoff, James Lawrence. “Mechanistic Biomarkers in Acute Liver Injury.” 2017. Doctoral Dissertation, University of Kansas. Accessed April 24, 2019. http://hdl.handle.net/1808/27044.

MLA Handbook (7th Edition):

Weemhoff, James Lawrence. “Mechanistic Biomarkers in Acute Liver Injury.” 2017. Web. 24 Apr 2019.

Vancouver:

Weemhoff JL. Mechanistic Biomarkers in Acute Liver Injury. [Internet] [Doctoral dissertation]. University of Kansas; 2017. [cited 2019 Apr 24]. Available from: http://hdl.handle.net/1808/27044.

Council of Science Editors:

Weemhoff JL. Mechanistic Biomarkers in Acute Liver Injury. [Doctoral Dissertation]. University of Kansas; 2017. Available from: http://hdl.handle.net/1808/27044

8. Williams, Jessica A. R. The Role of Parkin and Mitophagy in Acetaminophen and Alcohol-induced Liver Injuries.

Degree: PhD, Pharmacology, Toxicology & Therapeutics, 2015, University of Kansas

 Acetaminophen (APAP) is the leading cause of acute liver failure in the United States, and alcoholic liver disease (ALD) is a worldwide health problem that… (more)

Subjects/Keywords: Toxicology; Acetaminophen; Alcohol; Autophagy; Liver Injury; Mitophagy; Parkin

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APA (6th Edition):

Williams, J. A. R. (2015). The Role of Parkin and Mitophagy in Acetaminophen and Alcohol-induced Liver Injuries. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/19470

Chicago Manual of Style (16th Edition):

Williams, Jessica A R. “The Role of Parkin and Mitophagy in Acetaminophen and Alcohol-induced Liver Injuries.” 2015. Doctoral Dissertation, University of Kansas. Accessed April 24, 2019. http://hdl.handle.net/1808/19470.

MLA Handbook (7th Edition):

Williams, Jessica A R. “The Role of Parkin and Mitophagy in Acetaminophen and Alcohol-induced Liver Injuries.” 2015. Web. 24 Apr 2019.

Vancouver:

Williams JAR. The Role of Parkin and Mitophagy in Acetaminophen and Alcohol-induced Liver Injuries. [Internet] [Doctoral dissertation]. University of Kansas; 2015. [cited 2019 Apr 24]. Available from: http://hdl.handle.net/1808/19470.

Council of Science Editors:

Williams JAR. The Role of Parkin and Mitophagy in Acetaminophen and Alcohol-induced Liver Injuries. [Doctoral Dissertation]. University of Kansas; 2015. Available from: http://hdl.handle.net/1808/19470


University of Kansas

9. McCracken, Jennifer M. HEPATIC WOUND HEALING FOLLOWING ACUTE AND CHRONIC LIVER INJURY: A POTENTIAL ROLE FOR THE HYALURONAN NETWORK.

Degree: PhD, Pharmacology, Toxicology & Therapeutics, 2017, University of Kansas

 Chronic liver disease is the 12th leading cause of death in the United States and consists of a continuum of pathologies. Following initial injury, a… (more)

Subjects/Keywords: Toxicology; hyaluronan; hyaluronan mediated motility receptor; liver injury; liver wound healing

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APA (6th Edition):

McCracken, J. M. (2017). HEPATIC WOUND HEALING FOLLOWING ACUTE AND CHRONIC LIVER INJURY: A POTENTIAL ROLE FOR THE HYALURONAN NETWORK. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/26930

Chicago Manual of Style (16th Edition):

McCracken, Jennifer M. “HEPATIC WOUND HEALING FOLLOWING ACUTE AND CHRONIC LIVER INJURY: A POTENTIAL ROLE FOR THE HYALURONAN NETWORK.” 2017. Doctoral Dissertation, University of Kansas. Accessed April 24, 2019. http://hdl.handle.net/1808/26930.

MLA Handbook (7th Edition):

McCracken, Jennifer M. “HEPATIC WOUND HEALING FOLLOWING ACUTE AND CHRONIC LIVER INJURY: A POTENTIAL ROLE FOR THE HYALURONAN NETWORK.” 2017. Web. 24 Apr 2019.

Vancouver:

McCracken JM. HEPATIC WOUND HEALING FOLLOWING ACUTE AND CHRONIC LIVER INJURY: A POTENTIAL ROLE FOR THE HYALURONAN NETWORK. [Internet] [Doctoral dissertation]. University of Kansas; 2017. [cited 2019 Apr 24]. Available from: http://hdl.handle.net/1808/26930.

Council of Science Editors:

McCracken JM. HEPATIC WOUND HEALING FOLLOWING ACUTE AND CHRONIC LIVER INJURY: A POTENTIAL ROLE FOR THE HYALURONAN NETWORK. [Doctoral Dissertation]. University of Kansas; 2017. Available from: http://hdl.handle.net/1808/26930


University of Kansas

10. Shawgo, Mary E. NEW INSIGHTS INTO THE REGULATION OF MITOCHONDRIAL OUTER MEMBRANE PERMEABILIZATION DURING APOPTOSIS.

Degree: PhD, Pharmacology, Toxicology & Therapeutics, 2009, University of Kansas

 Disruption of normal apoptosis can contribute to the onset of cancer. Additionally, many cancer drugs are effective for their ability to initiate the apoptotic process.… (more)

Subjects/Keywords: Health sciences; Pharmacology; Cell biology; Apoptosis; Cancer; Chemotherapy

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APA (6th Edition):

Shawgo, M. E. (2009). NEW INSIGHTS INTO THE REGULATION OF MITOCHONDRIAL OUTER MEMBRANE PERMEABILIZATION DURING APOPTOSIS. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/5943

Chicago Manual of Style (16th Edition):

Shawgo, Mary E. “NEW INSIGHTS INTO THE REGULATION OF MITOCHONDRIAL OUTER MEMBRANE PERMEABILIZATION DURING APOPTOSIS.” 2009. Doctoral Dissertation, University of Kansas. Accessed April 24, 2019. http://hdl.handle.net/1808/5943.

MLA Handbook (7th Edition):

Shawgo, Mary E. “NEW INSIGHTS INTO THE REGULATION OF MITOCHONDRIAL OUTER MEMBRANE PERMEABILIZATION DURING APOPTOSIS.” 2009. Web. 24 Apr 2019.

Vancouver:

Shawgo ME. NEW INSIGHTS INTO THE REGULATION OF MITOCHONDRIAL OUTER MEMBRANE PERMEABILIZATION DURING APOPTOSIS. [Internet] [Doctoral dissertation]. University of Kansas; 2009. [cited 2019 Apr 24]. Available from: http://hdl.handle.net/1808/5943.

Council of Science Editors:

Shawgo ME. NEW INSIGHTS INTO THE REGULATION OF MITOCHONDRIAL OUTER MEMBRANE PERMEABILIZATION DURING APOPTOSIS. [Doctoral Dissertation]. University of Kansas; 2009. Available from: http://hdl.handle.net/1808/5943


University of Kansas

11. Saito, Chieko. PROTECTIVE STRATEGIES AGAINST ACETAMINOPHEN INDUCED HEPATOTOXICITY.

Degree: PhD, Pharmacology, Toxicology & Therapeutics, 2010, University of Kansas

 Acetaminophen (APAP) is a widely used analgesic, which is safe at therapeutic levels. APAP is mainly conjugated with glucuronic acid and sulfate to form water-soluble,… (more)

Subjects/Keywords: Health sciences; Toxicology; Acetaminophen; C-jun n-terminal kinase; Glutathione; Metallothionein; N-acetylcysteine

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APA (6th Edition):

Saito, C. (2010). PROTECTIVE STRATEGIES AGAINST ACETAMINOPHEN INDUCED HEPATOTOXICITY. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/6369

Chicago Manual of Style (16th Edition):

Saito, Chieko. “PROTECTIVE STRATEGIES AGAINST ACETAMINOPHEN INDUCED HEPATOTOXICITY.” 2010. Doctoral Dissertation, University of Kansas. Accessed April 24, 2019. http://hdl.handle.net/1808/6369.

MLA Handbook (7th Edition):

Saito, Chieko. “PROTECTIVE STRATEGIES AGAINST ACETAMINOPHEN INDUCED HEPATOTOXICITY.” 2010. Web. 24 Apr 2019.

Vancouver:

Saito C. PROTECTIVE STRATEGIES AGAINST ACETAMINOPHEN INDUCED HEPATOTOXICITY. [Internet] [Doctoral dissertation]. University of Kansas; 2010. [cited 2019 Apr 24]. Available from: http://hdl.handle.net/1808/6369.

Council of Science Editors:

Saito C. PROTECTIVE STRATEGIES AGAINST ACETAMINOPHEN INDUCED HEPATOTOXICITY. [Doctoral Dissertation]. University of Kansas; 2010. Available from: http://hdl.handle.net/1808/6369


University of Kansas

12. Borude, Prachi C. ROLE OF DNA DAMAGE RESPONSE IN LIVER REGENERATION AFTER APAP OVERDOSE INDUCED ALF.

Degree: PhD, Pharmacology, Toxicology & Therapeutics, 2017, University of Kansas

 Acetaminophen (APAP) overdose is a leading cause of acute liver failure (ALF) with limited treatment options. The mechanisms of APAP-induced liver injury include formation of… (more)

Subjects/Keywords: Toxicology

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APA (6th Edition):

Borude, P. C. (2017). ROLE OF DNA DAMAGE RESPONSE IN LIVER REGENERATION AFTER APAP OVERDOSE INDUCED ALF. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/27046

Chicago Manual of Style (16th Edition):

Borude, Prachi C. “ROLE OF DNA DAMAGE RESPONSE IN LIVER REGENERATION AFTER APAP OVERDOSE INDUCED ALF.” 2017. Doctoral Dissertation, University of Kansas. Accessed April 24, 2019. http://hdl.handle.net/1808/27046.

MLA Handbook (7th Edition):

Borude, Prachi C. “ROLE OF DNA DAMAGE RESPONSE IN LIVER REGENERATION AFTER APAP OVERDOSE INDUCED ALF.” 2017. Web. 24 Apr 2019.

Vancouver:

Borude PC. ROLE OF DNA DAMAGE RESPONSE IN LIVER REGENERATION AFTER APAP OVERDOSE INDUCED ALF. [Internet] [Doctoral dissertation]. University of Kansas; 2017. [cited 2019 Apr 24]. Available from: http://hdl.handle.net/1808/27046.

Council of Science Editors:

Borude PC. ROLE OF DNA DAMAGE RESPONSE IN LIVER REGENERATION AFTER APAP OVERDOSE INDUCED ALF. [Doctoral Dissertation]. University of Kansas; 2017. Available from: http://hdl.handle.net/1808/27046


University of Kansas

13. Bhushan, Bharat. MECHANISMS OF LIVER REGENERATION AFTER ACETAMINOPHEN-INDUCED HEPATOTOXICITY IN MICE.

Degree: PhD, Pharmacology, Toxicology & Therapeutics, 2016, University of Kansas

 Overdose of acetaminophen (APAP) is the major cause of acute liver failure (ALF) in the western world with very limited treatment options. Recent studies suggest… (more)

Subjects/Keywords: Toxicology; Pharmacology; Acetaminophen; EGF receptor; Liver; mice model; Regeneration; Wnt-Beta Catenin-GSK3 Beta pathway

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APA (6th Edition):

Bhushan, B. (2016). MECHANISMS OF LIVER REGENERATION AFTER ACETAMINOPHEN-INDUCED HEPATOTOXICITY IN MICE. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/25366

Chicago Manual of Style (16th Edition):

Bhushan, Bharat. “MECHANISMS OF LIVER REGENERATION AFTER ACETAMINOPHEN-INDUCED HEPATOTOXICITY IN MICE.” 2016. Doctoral Dissertation, University of Kansas. Accessed April 24, 2019. http://hdl.handle.net/1808/25366.

MLA Handbook (7th Edition):

Bhushan, Bharat. “MECHANISMS OF LIVER REGENERATION AFTER ACETAMINOPHEN-INDUCED HEPATOTOXICITY IN MICE.” 2016. Web. 24 Apr 2019.

Vancouver:

Bhushan B. MECHANISMS OF LIVER REGENERATION AFTER ACETAMINOPHEN-INDUCED HEPATOTOXICITY IN MICE. [Internet] [Doctoral dissertation]. University of Kansas; 2016. [cited 2019 Apr 24]. Available from: http://hdl.handle.net/1808/25366.

Council of Science Editors:

Bhushan B. MECHANISMS OF LIVER REGENERATION AFTER ACETAMINOPHEN-INDUCED HEPATOTOXICITY IN MICE. [Doctoral Dissertation]. University of Kansas; 2016. Available from: http://hdl.handle.net/1808/25366

14. Xu, Ming. The role of Ncb5or in fatty acid metabolism and redox homeostasis.

Degree: PhD, Physical Therapy & Rehabilitation Sciences, 2011, University of Kansas

 The endoplasmic reticulum (ER)-associated NADH cytochrome b5 oxidoreductase (Ncb5or) is widely distributed in animal tissues. It contains two redox domains that are homologous to microsomal… (more)

Subjects/Keywords: Biochemistry; Cellular biology; Fatty acid desaturation; Ncb5or; Oxidative stress; Saturated fatty acid metabolism

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APA (6th Edition):

Xu, M. (2011). The role of Ncb5or in fatty acid metabolism and redox homeostasis. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/9711

Chicago Manual of Style (16th Edition):

Xu, Ming. “The role of Ncb5or in fatty acid metabolism and redox homeostasis.” 2011. Doctoral Dissertation, University of Kansas. Accessed April 24, 2019. http://hdl.handle.net/1808/9711.

MLA Handbook (7th Edition):

Xu, Ming. “The role of Ncb5or in fatty acid metabolism and redox homeostasis.” 2011. Web. 24 Apr 2019.

Vancouver:

Xu M. The role of Ncb5or in fatty acid metabolism and redox homeostasis. [Internet] [Doctoral dissertation]. University of Kansas; 2011. [cited 2019 Apr 24]. Available from: http://hdl.handle.net/1808/9711.

Council of Science Editors:

Xu M. The role of Ncb5or in fatty acid metabolism and redox homeostasis. [Doctoral Dissertation]. University of Kansas; 2011. Available from: http://hdl.handle.net/1808/9711

15. Sullivan, Bradley P. Role of Coagulation in Xenobiotic-Induced Liver Injury.

Degree: PhD, Pharmacology, Toxicology & Therapeutics, 2012, University of Kansas

 The liver is a common target for xenobiotic-induced toxicity. Of importance, synthesis of soluble coagulation factors by the liver plays an essential role in hemostasis.… (more)

Subjects/Keywords: Toxicology; Acetaminophen; Alpha-naphthylisothiocyanate; Blood coagulation; Fibrosis; Liver injury; Platelet

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APA (6th Edition):

Sullivan, B. P. (2012). Role of Coagulation in Xenobiotic-Induced Liver Injury. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/10291

Chicago Manual of Style (16th Edition):

Sullivan, Bradley P. “Role of Coagulation in Xenobiotic-Induced Liver Injury.” 2012. Doctoral Dissertation, University of Kansas. Accessed April 24, 2019. http://hdl.handle.net/1808/10291.

MLA Handbook (7th Edition):

Sullivan, Bradley P. “Role of Coagulation in Xenobiotic-Induced Liver Injury.” 2012. Web. 24 Apr 2019.

Vancouver:

Sullivan BP. Role of Coagulation in Xenobiotic-Induced Liver Injury. [Internet] [Doctoral dissertation]. University of Kansas; 2012. [cited 2019 Apr 24]. Available from: http://hdl.handle.net/1808/10291.

Council of Science Editors:

Sullivan BP. Role of Coagulation in Xenobiotic-Induced Liver Injury. [Doctoral Dissertation]. University of Kansas; 2012. Available from: http://hdl.handle.net/1808/10291

16. Walesky, Chad Michael. Role of Hepatocyte Nuclear Factor 4 alpha in Hepatocyte Proliferation.

Degree: PhD, Pharmacology, Toxicology & Therapeutics, 2014, University of Kansas

 Hepatocyte Nuclear Factor 4 alpha (HNF4α) is the master regulator of hepatocyte differentiation. It is involved in the up-regulation of genes involved in many classic… (more)

Subjects/Keywords: Toxicology; Cancer; Gene expression; Hepatocyte; Hepatocyte nuclear factor 4; Liver; Proliferation

University of Kansas Medical Center under a standard 12-h light/dark cycle with access to chow and… 

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APA (6th Edition):

Walesky, C. M. (2014). Role of Hepatocyte Nuclear Factor 4 alpha in Hepatocyte Proliferation. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/14505

Chicago Manual of Style (16th Edition):

Walesky, Chad Michael. “Role of Hepatocyte Nuclear Factor 4 alpha in Hepatocyte Proliferation.” 2014. Doctoral Dissertation, University of Kansas. Accessed April 24, 2019. http://hdl.handle.net/1808/14505.

MLA Handbook (7th Edition):

Walesky, Chad Michael. “Role of Hepatocyte Nuclear Factor 4 alpha in Hepatocyte Proliferation.” 2014. Web. 24 Apr 2019.

Vancouver:

Walesky CM. Role of Hepatocyte Nuclear Factor 4 alpha in Hepatocyte Proliferation. [Internet] [Doctoral dissertation]. University of Kansas; 2014. [cited 2019 Apr 24]. Available from: http://hdl.handle.net/1808/14505.

Council of Science Editors:

Walesky CM. Role of Hepatocyte Nuclear Factor 4 alpha in Hepatocyte Proliferation. [Doctoral Dissertation]. University of Kansas; 2014. Available from: http://hdl.handle.net/1808/14505

17. Shelton, Shary Nicole. MECHANISMS OF MITOCHONDRIA-MEDIATED APOPTOSIS INDUCED BY CYTOTOXIC STRESS.

Degree: PhD, Pharmacology, Toxicology & Therapeutics, 2010, University of Kansas

 Defects within the apoptotic pathway are thought to contribute to tumorigenesis and therapeutic resistance. Although most cytotoxic anti-cancer drugs are thought to activate the mitochondria-mediated… (more)

Subjects/Keywords: Molecular biology; Apoptosis; DNA damage; Hsp90

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Shelton, S. N. (2010). MECHANISMS OF MITOCHONDRIA-MEDIATED APOPTOSIS INDUCED BY CYTOTOXIC STRESS. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/6761

Chicago Manual of Style (16th Edition):

Shelton, Shary Nicole. “MECHANISMS OF MITOCHONDRIA-MEDIATED APOPTOSIS INDUCED BY CYTOTOXIC STRESS.” 2010. Doctoral Dissertation, University of Kansas. Accessed April 24, 2019. http://hdl.handle.net/1808/6761.

MLA Handbook (7th Edition):

Shelton, Shary Nicole. “MECHANISMS OF MITOCHONDRIA-MEDIATED APOPTOSIS INDUCED BY CYTOTOXIC STRESS.” 2010. Web. 24 Apr 2019.

Vancouver:

Shelton SN. MECHANISMS OF MITOCHONDRIA-MEDIATED APOPTOSIS INDUCED BY CYTOTOXIC STRESS. [Internet] [Doctoral dissertation]. University of Kansas; 2010. [cited 2019 Apr 24]. Available from: http://hdl.handle.net/1808/6761.

Council of Science Editors:

Shelton SN. MECHANISMS OF MITOCHONDRIA-MEDIATED APOPTOSIS INDUCED BY CYTOTOXIC STRESS. [Doctoral Dissertation]. University of Kansas; 2010. Available from: http://hdl.handle.net/1808/6761

18. Williams, Clarence David. Role of Interleukin-1beta and neutrophil activation during acetaminophen overdose.

Degree: PhD, Pharmacology, Toxicology & Therapeutics, 2012, University of Kansas

 Acetaminophen (APAP) overdose is the leading cause of acute liver failure in the US. APAP is metabolized to a reactive metabolite that causes hepatotoxicity in… (more)

Subjects/Keywords: Toxicology; Acetaminophen; Il-1beta; Neutrophil

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APA (6th Edition):

Williams, C. D. (2012). Role of Interleukin-1beta and neutrophil activation during acetaminophen overdose. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/10290

Chicago Manual of Style (16th Edition):

Williams, Clarence David. “Role of Interleukin-1beta and neutrophil activation during acetaminophen overdose.” 2012. Doctoral Dissertation, University of Kansas. Accessed April 24, 2019. http://hdl.handle.net/1808/10290.

MLA Handbook (7th Edition):

Williams, Clarence David. “Role of Interleukin-1beta and neutrophil activation during acetaminophen overdose.” 2012. Web. 24 Apr 2019.

Vancouver:

Williams CD. Role of Interleukin-1beta and neutrophil activation during acetaminophen overdose. [Internet] [Doctoral dissertation]. University of Kansas; 2012. [cited 2019 Apr 24]. Available from: http://hdl.handle.net/1808/10290.

Council of Science Editors:

Williams CD. Role of Interleukin-1beta and neutrophil activation during acetaminophen overdose. [Doctoral Dissertation]. University of Kansas; 2012. Available from: http://hdl.handle.net/1808/10290

19. McGill, Mitchell Ryan. Acetaminophen Hepatotoxicity in Humans and Mice.

Degree: PhD, Pharmacology, Toxicology & Therapeutics, 2013, University of Kansas

 Acetaminophen (APAP) is a popular analgesic and antipyretic. Most of a therapeutic dose is glucuronidated or sulfated and excreted. A small amount is converted by… (more)

Subjects/Keywords: Toxicology; Physiology; Pathology; Acetaminophen; Human; Liver; Mitochondria; Translational research

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

McGill, M. R. (2013). Acetaminophen Hepatotoxicity in Humans and Mice. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/12178

Chicago Manual of Style (16th Edition):

McGill, Mitchell Ryan. “Acetaminophen Hepatotoxicity in Humans and Mice.” 2013. Doctoral Dissertation, University of Kansas. Accessed April 24, 2019. http://hdl.handle.net/1808/12178.

MLA Handbook (7th Edition):

McGill, Mitchell Ryan. “Acetaminophen Hepatotoxicity in Humans and Mice.” 2013. Web. 24 Apr 2019.

Vancouver:

McGill MR. Acetaminophen Hepatotoxicity in Humans and Mice. [Internet] [Doctoral dissertation]. University of Kansas; 2013. [cited 2019 Apr 24]. Available from: http://hdl.handle.net/1808/12178.

Council of Science Editors:

McGill MR. Acetaminophen Hepatotoxicity in Humans and Mice. [Doctoral Dissertation]. University of Kansas; 2013. Available from: http://hdl.handle.net/1808/12178

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