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You searched for +publisher:"University of Kansas" +contributor:("Hagenbuch, Bruno"). Showing records 1 – 29 of 29 total matches.

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University of Kansas

1. Mickey, Kristen Elizabeth. Arsenic Toxicity and Altered Mitochondrial Bioenergetics in Response to Oxidative Stress.

Degree: MS, Pharmacology, Toxicology & Therapeutics, 2015, University of Kansas

 Environmental exposure to arsenic is a worldwide health concern which is linked to a number of diseases. Areas with arsenic levels above the current safe… (more)

Subjects/Keywords: Toxicology; Cellular biology; Molecular biology; Arsenic; Mitochondria; Oxidative Stress

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APA (6th Edition):

Mickey, K. E. (2015). Arsenic Toxicity and Altered Mitochondrial Bioenergetics in Response to Oxidative Stress. (Masters Thesis). University of Kansas. Retrieved from http://hdl.handle.net/1808/19447

Chicago Manual of Style (16th Edition):

Mickey, Kristen Elizabeth. “Arsenic Toxicity and Altered Mitochondrial Bioenergetics in Response to Oxidative Stress.” 2015. Masters Thesis, University of Kansas. Accessed January 19, 2021. http://hdl.handle.net/1808/19447.

MLA Handbook (7th Edition):

Mickey, Kristen Elizabeth. “Arsenic Toxicity and Altered Mitochondrial Bioenergetics in Response to Oxidative Stress.” 2015. Web. 19 Jan 2021.

Vancouver:

Mickey KE. Arsenic Toxicity and Altered Mitochondrial Bioenergetics in Response to Oxidative Stress. [Internet] [Masters thesis]. University of Kansas; 2015. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/1808/19447.

Council of Science Editors:

Mickey KE. Arsenic Toxicity and Altered Mitochondrial Bioenergetics in Response to Oxidative Stress. [Masters Thesis]. University of Kansas; 2015. Available from: http://hdl.handle.net/1808/19447


University of Kansas

2. Polireddy, Kishore. Identifying Novel Inhibitors of Epithelial to Mesenchymal Transition (EMT) for Targeting Pancreatic Cancer Metastasis and Cancer Stem Cells.

Degree: PhD, Pharmacology, Toxicology & Therapeutics, 2015, University of Kansas

 Pancreatic cancer is the fourth leading cause of cancer-related death in the United States, and it is expected to become the second-leading cause of cancer-related… (more)

Subjects/Keywords: Oncology; Biology; Cancer stem cells; EMT; Pancreatic cancer

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APA (6th Edition):

Polireddy, K. (2015). Identifying Novel Inhibitors of Epithelial to Mesenchymal Transition (EMT) for Targeting Pancreatic Cancer Metastasis and Cancer Stem Cells. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/21926

Chicago Manual of Style (16th Edition):

Polireddy, Kishore. “Identifying Novel Inhibitors of Epithelial to Mesenchymal Transition (EMT) for Targeting Pancreatic Cancer Metastasis and Cancer Stem Cells.” 2015. Doctoral Dissertation, University of Kansas. Accessed January 19, 2021. http://hdl.handle.net/1808/21926.

MLA Handbook (7th Edition):

Polireddy, Kishore. “Identifying Novel Inhibitors of Epithelial to Mesenchymal Transition (EMT) for Targeting Pancreatic Cancer Metastasis and Cancer Stem Cells.” 2015. Web. 19 Jan 2021.

Vancouver:

Polireddy K. Identifying Novel Inhibitors of Epithelial to Mesenchymal Transition (EMT) for Targeting Pancreatic Cancer Metastasis and Cancer Stem Cells. [Internet] [Doctoral dissertation]. University of Kansas; 2015. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/1808/21926.

Council of Science Editors:

Polireddy K. Identifying Novel Inhibitors of Epithelial to Mesenchymal Transition (EMT) for Targeting Pancreatic Cancer Metastasis and Cancer Stem Cells. [Doctoral Dissertation]. University of Kansas; 2015. Available from: http://hdl.handle.net/1808/21926


University of Kansas

3. Zhao, Wen. Identification and characterization of the transporters involved in the disposition of perfluoroalkyl substances.

Degree: PhD, Pharmacology, Toxicology & Therapeutics, 2016, University of Kansas

 Perfluoroalkyl substances (PFASs), including perfluoroalkyl carboxylates (PFCAs) and perfluoroalkyl sulfonates (PFSAs), are persistent amphiphilic chemicals many of which are distributed ubiquitously in the environment and… (more)

Subjects/Keywords: Pharmacology; Toxicology; Drug transporters; Enterohepatic circulation; Perfluoroalkyl substances; PFBS; PFHxS; PFOS

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APA (6th Edition):

Zhao, W. (2016). Identification and characterization of the transporters involved in the disposition of perfluoroalkyl substances. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/24802

Chicago Manual of Style (16th Edition):

Zhao, Wen. “Identification and characterization of the transporters involved in the disposition of perfluoroalkyl substances.” 2016. Doctoral Dissertation, University of Kansas. Accessed January 19, 2021. http://hdl.handle.net/1808/24802.

MLA Handbook (7th Edition):

Zhao, Wen. “Identification and characterization of the transporters involved in the disposition of perfluoroalkyl substances.” 2016. Web. 19 Jan 2021.

Vancouver:

Zhao W. Identification and characterization of the transporters involved in the disposition of perfluoroalkyl substances. [Internet] [Doctoral dissertation]. University of Kansas; 2016. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/1808/24802.

Council of Science Editors:

Zhao W. Identification and characterization of the transporters involved in the disposition of perfluoroalkyl substances. [Doctoral Dissertation]. University of Kansas; 2016. Available from: http://hdl.handle.net/1808/24802


University of Kansas

4. Bishop, Stephanie Cara. Development of Novel Fluorescence and NMR Reagents for Monitoring Protein-Protein Interactions Between Survivin and Its Protein Binding Partners.

Degree: PhD, Pharmacology, Toxicology & Therapeutics, 2015, University of Kansas

 Inhibition of protein-protein interactions is a promising therapeutic strategy for targeting non-enzymatic proteins. One strategy to inhibit protein-protein interactions is to design inhibitors specifically toward… (more)

Subjects/Keywords: Pharmacology; Biochemistry; Organic chemistry; Copper-free click chemistry; Fluorescence; Lanthanide chelation; Nuclear Magnetic Resonance; Paramagnetism; Unnatural Amino Acid Incorporation

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APA (6th Edition):

Bishop, S. C. (2015). Development of Novel Fluorescence and NMR Reagents for Monitoring Protein-Protein Interactions Between Survivin and Its Protein Binding Partners. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/19468

Chicago Manual of Style (16th Edition):

Bishop, Stephanie Cara. “Development of Novel Fluorescence and NMR Reagents for Monitoring Protein-Protein Interactions Between Survivin and Its Protein Binding Partners.” 2015. Doctoral Dissertation, University of Kansas. Accessed January 19, 2021. http://hdl.handle.net/1808/19468.

MLA Handbook (7th Edition):

Bishop, Stephanie Cara. “Development of Novel Fluorescence and NMR Reagents for Monitoring Protein-Protein Interactions Between Survivin and Its Protein Binding Partners.” 2015. Web. 19 Jan 2021.

Vancouver:

Bishop SC. Development of Novel Fluorescence and NMR Reagents for Monitoring Protein-Protein Interactions Between Survivin and Its Protein Binding Partners. [Internet] [Doctoral dissertation]. University of Kansas; 2015. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/1808/19468.

Council of Science Editors:

Bishop SC. Development of Novel Fluorescence and NMR Reagents for Monitoring Protein-Protein Interactions Between Survivin and Its Protein Binding Partners. [Doctoral Dissertation]. University of Kansas; 2015. Available from: http://hdl.handle.net/1808/19468


University of Kansas

5. Zhang, Youcai. CHARACTERIZATION OF ORGANIC ANION TRANSPORTING POLYPEPTIDE 1A1 (OATP1A1) IN THE BILE ACID HOMEOSTASIS OF MICE.

Degree: PhD, Pharmacology, Toxicology & Therapeutics, 2011, University of Kansas

 Organic anion transporting polypeptides (human: OATPs; all other species: Oatps; gene symbol: SLCO/Slco) are sodium-independent transport systems that mediate the transmembrane transport of a wide… (more)

Subjects/Keywords: Toxicology; Bile acids; Cholestasis; Intestinal bacteria; Organic anion transporting polypeptide

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APA (6th Edition):

Zhang, Y. (2011). CHARACTERIZATION OF ORGANIC ANION TRANSPORTING POLYPEPTIDE 1A1 (OATP1A1) IN THE BILE ACID HOMEOSTASIS OF MICE. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/7930

Chicago Manual of Style (16th Edition):

Zhang, Youcai. “CHARACTERIZATION OF ORGANIC ANION TRANSPORTING POLYPEPTIDE 1A1 (OATP1A1) IN THE BILE ACID HOMEOSTASIS OF MICE.” 2011. Doctoral Dissertation, University of Kansas. Accessed January 19, 2021. http://hdl.handle.net/1808/7930.

MLA Handbook (7th Edition):

Zhang, Youcai. “CHARACTERIZATION OF ORGANIC ANION TRANSPORTING POLYPEPTIDE 1A1 (OATP1A1) IN THE BILE ACID HOMEOSTASIS OF MICE.” 2011. Web. 19 Jan 2021.

Vancouver:

Zhang Y. CHARACTERIZATION OF ORGANIC ANION TRANSPORTING POLYPEPTIDE 1A1 (OATP1A1) IN THE BILE ACID HOMEOSTASIS OF MICE. [Internet] [Doctoral dissertation]. University of Kansas; 2011. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/1808/7930.

Council of Science Editors:

Zhang Y. CHARACTERIZATION OF ORGANIC ANION TRANSPORTING POLYPEPTIDE 1A1 (OATP1A1) IN THE BILE ACID HOMEOSTASIS OF MICE. [Doctoral Dissertation]. University of Kansas; 2011. Available from: http://hdl.handle.net/1808/7930


University of Kansas

6. Selwyn Samraj, Felcy Pavithra. Alterations in bile acid homeostasis and drug metabolism in germ-free mice.

Degree: PhD, Pharmacology, Toxicology & Therapeutics, 2014, University of Kansas

 "We may be born 100% human but will die 90% bacterial – a truly complex organism!" (Goodacre, 2007). This statement reflects the fact that there are… (more)

Subjects/Keywords: Toxicology; Physiology; Bile acid; Drug metabolism; Germ-free mice

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APA (6th Edition):

Selwyn Samraj, F. P. (2014). Alterations in bile acid homeostasis and drug metabolism in germ-free mice. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/23957

Chicago Manual of Style (16th Edition):

Selwyn Samraj, Felcy Pavithra. “Alterations in bile acid homeostasis and drug metabolism in germ-free mice.” 2014. Doctoral Dissertation, University of Kansas. Accessed January 19, 2021. http://hdl.handle.net/1808/23957.

MLA Handbook (7th Edition):

Selwyn Samraj, Felcy Pavithra. “Alterations in bile acid homeostasis and drug metabolism in germ-free mice.” 2014. Web. 19 Jan 2021.

Vancouver:

Selwyn Samraj FP. Alterations in bile acid homeostasis and drug metabolism in germ-free mice. [Internet] [Doctoral dissertation]. University of Kansas; 2014. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/1808/23957.

Council of Science Editors:

Selwyn Samraj FP. Alterations in bile acid homeostasis and drug metabolism in germ-free mice. [Doctoral Dissertation]. University of Kansas; 2014. Available from: http://hdl.handle.net/1808/23957


University of Kansas

7. Tessman, Robert Thomas. The Role of Mitochondrial ATP-Binding Cassette Transporter ABCB6 in Metabolism and Energy Balance.

Degree: PhD, Pharmacology, Toxicology & Therapeutics, 2019, University of Kansas

 Abstract Obesity and the associated health risks represent a world-wide health and financial crisis. Lack of physical activity combined with excessive caloric intake are the… (more)

Subjects/Keywords: Toxicology; Dynamics; Metabolism; Mitochondria; Transporter

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APA (6th Edition):

Tessman, R. T. (2019). The Role of Mitochondrial ATP-Binding Cassette Transporter ABCB6 in Metabolism and Energy Balance. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/30121

Chicago Manual of Style (16th Edition):

Tessman, Robert Thomas. “The Role of Mitochondrial ATP-Binding Cassette Transporter ABCB6 in Metabolism and Energy Balance.” 2019. Doctoral Dissertation, University of Kansas. Accessed January 19, 2021. http://hdl.handle.net/1808/30121.

MLA Handbook (7th Edition):

Tessman, Robert Thomas. “The Role of Mitochondrial ATP-Binding Cassette Transporter ABCB6 in Metabolism and Energy Balance.” 2019. Web. 19 Jan 2021.

Vancouver:

Tessman RT. The Role of Mitochondrial ATP-Binding Cassette Transporter ABCB6 in Metabolism and Energy Balance. [Internet] [Doctoral dissertation]. University of Kansas; 2019. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/1808/30121.

Council of Science Editors:

Tessman RT. The Role of Mitochondrial ATP-Binding Cassette Transporter ABCB6 in Metabolism and Energy Balance. [Doctoral Dissertation]. University of Kansas; 2019. Available from: http://hdl.handle.net/1808/30121


University of Kansas

8. Zhang, Yuchen. IDENTIFICATION AND CHARACTERIZATION OF THE OLIGOMERIZATION AND STRUCTURAL FUNCTIONAL RELATIONSHIP OF ORGANIC ANION TRANSPORTING POLYPEPTIDE 1B3.

Degree: PhD, Pharmacology, Toxicology & Therapeutics, 2017, University of Kansas

 Many transporters are expressed at the basolateral membrane of human hepatocytes, including Organic Anion Transporting Polypeptide 1B1 (OATP1B1), OATP1B3, Organic Cation Transporter 1 (OCT1), Na+/Taurocholate… (more)

Subjects/Keywords: Pharmacology

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APA (6th Edition):

Zhang, Y. (2017). IDENTIFICATION AND CHARACTERIZATION OF THE OLIGOMERIZATION AND STRUCTURAL FUNCTIONAL RELATIONSHIP OF ORGANIC ANION TRANSPORTING POLYPEPTIDE 1B3. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/27333

Chicago Manual of Style (16th Edition):

Zhang, Yuchen. “IDENTIFICATION AND CHARACTERIZATION OF THE OLIGOMERIZATION AND STRUCTURAL FUNCTIONAL RELATIONSHIP OF ORGANIC ANION TRANSPORTING POLYPEPTIDE 1B3.” 2017. Doctoral Dissertation, University of Kansas. Accessed January 19, 2021. http://hdl.handle.net/1808/27333.

MLA Handbook (7th Edition):

Zhang, Yuchen. “IDENTIFICATION AND CHARACTERIZATION OF THE OLIGOMERIZATION AND STRUCTURAL FUNCTIONAL RELATIONSHIP OF ORGANIC ANION TRANSPORTING POLYPEPTIDE 1B3.” 2017. Web. 19 Jan 2021.

Vancouver:

Zhang Y. IDENTIFICATION AND CHARACTERIZATION OF THE OLIGOMERIZATION AND STRUCTURAL FUNCTIONAL RELATIONSHIP OF ORGANIC ANION TRANSPORTING POLYPEPTIDE 1B3. [Internet] [Doctoral dissertation]. University of Kansas; 2017. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/1808/27333.

Council of Science Editors:

Zhang Y. IDENTIFICATION AND CHARACTERIZATION OF THE OLIGOMERIZATION AND STRUCTURAL FUNCTIONAL RELATIONSHIP OF ORGANIC ANION TRANSPORTING POLYPEPTIDE 1B3. [Doctoral Dissertation]. University of Kansas; 2017. Available from: http://hdl.handle.net/1808/27333


University of Kansas

9. Bastola, Prabhakar. Targeting vulnerabilities through inhibiting the valosin-containing protein (VCP/p97) in ovarian cancer.

Degree: PhD, Pharmacology, Toxicology & Therapeutics, 2018, University of Kansas

 Ovarian cancer is the fifth leading cause of cancer-related death among women and the deadliest of all gynecological cancers. Treatment failure is a major contributing… (more)

Subjects/Keywords: Pharmacology; endoplasmic reticulum stress; ovarian cancer; protein quality control; unfolded protein response; Valosin-containing protein; VCP/p97 inhibitors

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APA (6th Edition):

Bastola, P. (2018). Targeting vulnerabilities through inhibiting the valosin-containing protein (VCP/p97) in ovarian cancer. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/27334

Chicago Manual of Style (16th Edition):

Bastola, Prabhakar. “Targeting vulnerabilities through inhibiting the valosin-containing protein (VCP/p97) in ovarian cancer.” 2018. Doctoral Dissertation, University of Kansas. Accessed January 19, 2021. http://hdl.handle.net/1808/27334.

MLA Handbook (7th Edition):

Bastola, Prabhakar. “Targeting vulnerabilities through inhibiting the valosin-containing protein (VCP/p97) in ovarian cancer.” 2018. Web. 19 Jan 2021.

Vancouver:

Bastola P. Targeting vulnerabilities through inhibiting the valosin-containing protein (VCP/p97) in ovarian cancer. [Internet] [Doctoral dissertation]. University of Kansas; 2018. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/1808/27334.

Council of Science Editors:

Bastola P. Targeting vulnerabilities through inhibiting the valosin-containing protein (VCP/p97) in ovarian cancer. [Doctoral Dissertation]. University of Kansas; 2018. Available from: http://hdl.handle.net/1808/27334


University of Kansas

10. Ogilvie, Brian Wayne. AN IN VITRO INVESTIGATION INTO THE MECHANISM OF THE CLINICALLY RELEVANT DRUG-DRUG INTERACTION BETWEEN OMEPRAZOLE OR ESOMEPRAZOLE AND CLOPIDOGREL.

Degree: PhD, Pharmacology, Toxicology & Therapeutics, 2015, University of Kansas

 Clopidogrel is a thienopyridine antiplatelet prodrug that was approved by the US FDA in 1997 and quickly supplanted ticlopidine as the primary drug therapy for… (more)

Subjects/Keywords: Toxicology; Clopidogrel; Cytochrome P450; Drug-drug interactions; Esomeprazole; Omeprazole

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APA (6th Edition):

Ogilvie, B. W. (2015). AN IN VITRO INVESTIGATION INTO THE MECHANISM OF THE CLINICALLY RELEVANT DRUG-DRUG INTERACTION BETWEEN OMEPRAZOLE OR ESOMEPRAZOLE AND CLOPIDOGREL. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/19459

Chicago Manual of Style (16th Edition):

Ogilvie, Brian Wayne. “AN IN VITRO INVESTIGATION INTO THE MECHANISM OF THE CLINICALLY RELEVANT DRUG-DRUG INTERACTION BETWEEN OMEPRAZOLE OR ESOMEPRAZOLE AND CLOPIDOGREL.” 2015. Doctoral Dissertation, University of Kansas. Accessed January 19, 2021. http://hdl.handle.net/1808/19459.

MLA Handbook (7th Edition):

Ogilvie, Brian Wayne. “AN IN VITRO INVESTIGATION INTO THE MECHANISM OF THE CLINICALLY RELEVANT DRUG-DRUG INTERACTION BETWEEN OMEPRAZOLE OR ESOMEPRAZOLE AND CLOPIDOGREL.” 2015. Web. 19 Jan 2021.

Vancouver:

Ogilvie BW. AN IN VITRO INVESTIGATION INTO THE MECHANISM OF THE CLINICALLY RELEVANT DRUG-DRUG INTERACTION BETWEEN OMEPRAZOLE OR ESOMEPRAZOLE AND CLOPIDOGREL. [Internet] [Doctoral dissertation]. University of Kansas; 2015. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/1808/19459.

Council of Science Editors:

Ogilvie BW. AN IN VITRO INVESTIGATION INTO THE MECHANISM OF THE CLINICALLY RELEVANT DRUG-DRUG INTERACTION BETWEEN OMEPRAZOLE OR ESOMEPRAZOLE AND CLOPIDOGREL. [Doctoral Dissertation]. University of Kansas; 2015. Available from: http://hdl.handle.net/1808/19459

11. Roth, Megan Elizabeth. Substrate Dependent Alterations of Organic Anion Transporting Polypeptide 1B3 (OATP1B3).

Degree: PhD, Pharmacology, Toxicology & Therapeutics, 2011, University of Kansas

 Organic anion transporting polypeptides (OATPs) are multispecific transporters that mediate the uptake of numerous drugs and xenobiotics into cells. Alterations in the function of the… (more)

Subjects/Keywords: Pharmacology; Physiology; Drug-drug interactions; Oatp; Structure-function; Transporters

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APA (6th Edition):

Roth, M. E. (2011). Substrate Dependent Alterations of Organic Anion Transporting Polypeptide 1B3 (OATP1B3). (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/9715

Chicago Manual of Style (16th Edition):

Roth, Megan Elizabeth. “Substrate Dependent Alterations of Organic Anion Transporting Polypeptide 1B3 (OATP1B3).” 2011. Doctoral Dissertation, University of Kansas. Accessed January 19, 2021. http://hdl.handle.net/1808/9715.

MLA Handbook (7th Edition):

Roth, Megan Elizabeth. “Substrate Dependent Alterations of Organic Anion Transporting Polypeptide 1B3 (OATP1B3).” 2011. Web. 19 Jan 2021.

Vancouver:

Roth ME. Substrate Dependent Alterations of Organic Anion Transporting Polypeptide 1B3 (OATP1B3). [Internet] [Doctoral dissertation]. University of Kansas; 2011. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/1808/9715.

Council of Science Editors:

Roth ME. Substrate Dependent Alterations of Organic Anion Transporting Polypeptide 1B3 (OATP1B3). [Doctoral Dissertation]. University of Kansas; 2011. Available from: http://hdl.handle.net/1808/9715


University of Kansas

12. Flynn, Colleen A. FEXOFENADINE AND ORGANIC ANION TRANSPORTING POLYPEPTIDES (OATPs): TRANSPORT AND DRUG-DRUG INTERACTIONS.

Degree: PhD, Pharmacology, Toxicology & Therapeutics, 2011, University of Kansas

 Transporters play a major role in the absorption and disposition of fexofenadine, suggesting this drug could be used as a probe of transporter activity. When… (more)

Subjects/Keywords: Toxicology; Drug-drug interactions; Fexofenadine; Oatp; Probe cocktail; Single nucleotide polymorphism

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APA (6th Edition):

Flynn, C. A. (2011). FEXOFENADINE AND ORGANIC ANION TRANSPORTING POLYPEPTIDES (OATPs): TRANSPORT AND DRUG-DRUG INTERACTIONS. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/9723

Chicago Manual of Style (16th Edition):

Flynn, Colleen A. “FEXOFENADINE AND ORGANIC ANION TRANSPORTING POLYPEPTIDES (OATPs): TRANSPORT AND DRUG-DRUG INTERACTIONS.” 2011. Doctoral Dissertation, University of Kansas. Accessed January 19, 2021. http://hdl.handle.net/1808/9723.

MLA Handbook (7th Edition):

Flynn, Colleen A. “FEXOFENADINE AND ORGANIC ANION TRANSPORTING POLYPEPTIDES (OATPs): TRANSPORT AND DRUG-DRUG INTERACTIONS.” 2011. Web. 19 Jan 2021.

Vancouver:

Flynn CA. FEXOFENADINE AND ORGANIC ANION TRANSPORTING POLYPEPTIDES (OATPs): TRANSPORT AND DRUG-DRUG INTERACTIONS. [Internet] [Doctoral dissertation]. University of Kansas; 2011. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/1808/9723.

Council of Science Editors:

Flynn CA. FEXOFENADINE AND ORGANIC ANION TRANSPORTING POLYPEPTIDES (OATPs): TRANSPORT AND DRUG-DRUG INTERACTIONS. [Doctoral Dissertation]. University of Kansas; 2011. Available from: http://hdl.handle.net/1808/9723


University of Kansas

13. Boxberger, Kelli Harmon. IDENTIFICATION OF ENDOGENOUS FUNCTION AND SUBSTRATE-DEPENDENT INTERACTIONS OF ORGANIC CATION TRANSPORTER 1.

Degree: PhD, Pharmacology, Toxicology & Therapeutics, 2018, University of Kansas

 The human organic cation transporter 1 (hOCT1) is a polyspecific transporter, primarily expressed in the liver, which is known to interact with a large number… (more)

Subjects/Keywords: Pharmacology; computational modeling; drug disposition; drug-drug interactions; organic cation transporter; substrate-dependent interactions; transporters

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APA (6th Edition):

Boxberger, K. H. (2018). IDENTIFICATION OF ENDOGENOUS FUNCTION AND SUBSTRATE-DEPENDENT INTERACTIONS OF ORGANIC CATION TRANSPORTER 1. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/27078

Chicago Manual of Style (16th Edition):

Boxberger, Kelli Harmon. “IDENTIFICATION OF ENDOGENOUS FUNCTION AND SUBSTRATE-DEPENDENT INTERACTIONS OF ORGANIC CATION TRANSPORTER 1.” 2018. Doctoral Dissertation, University of Kansas. Accessed January 19, 2021. http://hdl.handle.net/1808/27078.

MLA Handbook (7th Edition):

Boxberger, Kelli Harmon. “IDENTIFICATION OF ENDOGENOUS FUNCTION AND SUBSTRATE-DEPENDENT INTERACTIONS OF ORGANIC CATION TRANSPORTER 1.” 2018. Web. 19 Jan 2021.

Vancouver:

Boxberger KH. IDENTIFICATION OF ENDOGENOUS FUNCTION AND SUBSTRATE-DEPENDENT INTERACTIONS OF ORGANIC CATION TRANSPORTER 1. [Internet] [Doctoral dissertation]. University of Kansas; 2018. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/1808/27078.

Council of Science Editors:

Boxberger KH. IDENTIFICATION OF ENDOGENOUS FUNCTION AND SUBSTRATE-DEPENDENT INTERACTIONS OF ORGANIC CATION TRANSPORTER 1. [Doctoral Dissertation]. University of Kansas; 2018. Available from: http://hdl.handle.net/1808/27078


University of Kansas

14. Curry, Joshua Nicholas. The role of claudin-2 in the proximal tubule and kidney stone disease.

Degree: PhD, Molecular & Integrative Physiology, 2018, University of Kansas

 The concentration of circulating blood calcium is vital to the function of many cellular processes. As such, it is maintained within a narrow range through… (more)

Subjects/Keywords: Physiology; Genetics; Medicine; calcium; claudins; ion transport; kidney stones; nephrocalcinosis; tight junctions

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APA (6th Edition):

Curry, J. N. (2018). The role of claudin-2 in the proximal tubule and kidney stone disease. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/30132

Chicago Manual of Style (16th Edition):

Curry, Joshua Nicholas. “The role of claudin-2 in the proximal tubule and kidney stone disease.” 2018. Doctoral Dissertation, University of Kansas. Accessed January 19, 2021. http://hdl.handle.net/1808/30132.

MLA Handbook (7th Edition):

Curry, Joshua Nicholas. “The role of claudin-2 in the proximal tubule and kidney stone disease.” 2018. Web. 19 Jan 2021.

Vancouver:

Curry JN. The role of claudin-2 in the proximal tubule and kidney stone disease. [Internet] [Doctoral dissertation]. University of Kansas; 2018. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/1808/30132.

Council of Science Editors:

Curry JN. The role of claudin-2 in the proximal tubule and kidney stone disease. [Doctoral Dissertation]. University of Kansas; 2018. Available from: http://hdl.handle.net/1808/30132

15. Holt, Briana. Early growth response (Egr)-1: A novel anti-fibrotic mediator in liver.

Degree: MS, Pharmacology, Toxicology & Therapeutics, 2014, University of Kansas

 Chronic alcohol consumption can lead to an aberrant wound healing response of the liver, or hepatic fibrosis. Egr-1 is an essential regulator of many genes… (more)

Subjects/Keywords: Toxicology

…Use Committee at the University of Kansas Medical Center. Mice deficient in Egr-1 were bred… 

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APA (6th Edition):

Holt, B. (2014). Early growth response (Egr)-1: A novel anti-fibrotic mediator in liver. (Masters Thesis). University of Kansas. Retrieved from http://hdl.handle.net/1808/18422

Chicago Manual of Style (16th Edition):

Holt, Briana. “Early growth response (Egr)-1: A novel anti-fibrotic mediator in liver.” 2014. Masters Thesis, University of Kansas. Accessed January 19, 2021. http://hdl.handle.net/1808/18422.

MLA Handbook (7th Edition):

Holt, Briana. “Early growth response (Egr)-1: A novel anti-fibrotic mediator in liver.” 2014. Web. 19 Jan 2021.

Vancouver:

Holt B. Early growth response (Egr)-1: A novel anti-fibrotic mediator in liver. [Internet] [Masters thesis]. University of Kansas; 2014. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/1808/18422.

Council of Science Editors:

Holt B. Early growth response (Egr)-1: A novel anti-fibrotic mediator in liver. [Masters Thesis]. University of Kansas; 2014. Available from: http://hdl.handle.net/1808/18422

16. Pei, Lei. PARACELLULAR EPITHELIAL TRANSPORT MAXIMIZES ENERGY EFFICIENCY IN THE KIDNEY.

Degree: PhD, Molecular & Integrative Physiology, 2016, University of Kansas

 Claudins are tight junction transmembrane proteins that act as paracellular ion channels. The proximal renal tubule reabsorbs 70% of glomerulus-filtered Na+. Of this Na+, up… (more)

Subjects/Keywords: Physiology; acute kidney injury; claudin-2; Hypoxia; oxygen utilization efficiency

…Care and Use Committee at the University of Kansas Medical Center. Data analysis Data are… 

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APA (6th Edition):

Pei, L. (2016). PARACELLULAR EPITHELIAL TRANSPORT MAXIMIZES ENERGY EFFICIENCY IN THE KIDNEY. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/22506

Chicago Manual of Style (16th Edition):

Pei, Lei. “PARACELLULAR EPITHELIAL TRANSPORT MAXIMIZES ENERGY EFFICIENCY IN THE KIDNEY.” 2016. Doctoral Dissertation, University of Kansas. Accessed January 19, 2021. http://hdl.handle.net/1808/22506.

MLA Handbook (7th Edition):

Pei, Lei. “PARACELLULAR EPITHELIAL TRANSPORT MAXIMIZES ENERGY EFFICIENCY IN THE KIDNEY.” 2016. Web. 19 Jan 2021.

Vancouver:

Pei L. PARACELLULAR EPITHELIAL TRANSPORT MAXIMIZES ENERGY EFFICIENCY IN THE KIDNEY. [Internet] [Doctoral dissertation]. University of Kansas; 2016. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/1808/22506.

Council of Science Editors:

Pei L. PARACELLULAR EPITHELIAL TRANSPORT MAXIMIZES ENERGY EFFICIENCY IN THE KIDNEY. [Doctoral Dissertation]. University of Kansas; 2016. Available from: http://hdl.handle.net/1808/22506

17. Walesky, Chad Michael. Role of Hepatocyte Nuclear Factor 4 alpha in Hepatocyte Proliferation.

Degree: PhD, Pharmacology, Toxicology & Therapeutics, 2014, University of Kansas

 Hepatocyte Nuclear Factor 4 alpha (HNF4α) is the master regulator of hepatocyte differentiation. It is involved in the up-regulation of genes involved in many classic… (more)

Subjects/Keywords: Toxicology; Cancer; Gene expression; Hepatocyte; Hepatocyte nuclear factor 4; Liver; Proliferation

University of Kansas Medical Center under a standard 12-h light/dark cycle with access to chow and… 

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APA (6th Edition):

Walesky, C. M. (2014). Role of Hepatocyte Nuclear Factor 4 alpha in Hepatocyte Proliferation. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/14505

Chicago Manual of Style (16th Edition):

Walesky, Chad Michael. “Role of Hepatocyte Nuclear Factor 4 alpha in Hepatocyte Proliferation.” 2014. Doctoral Dissertation, University of Kansas. Accessed January 19, 2021. http://hdl.handle.net/1808/14505.

MLA Handbook (7th Edition):

Walesky, Chad Michael. “Role of Hepatocyte Nuclear Factor 4 alpha in Hepatocyte Proliferation.” 2014. Web. 19 Jan 2021.

Vancouver:

Walesky CM. Role of Hepatocyte Nuclear Factor 4 alpha in Hepatocyte Proliferation. [Internet] [Doctoral dissertation]. University of Kansas; 2014. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/1808/14505.

Council of Science Editors:

Walesky CM. Role of Hepatocyte Nuclear Factor 4 alpha in Hepatocyte Proliferation. [Doctoral Dissertation]. University of Kansas; 2014. Available from: http://hdl.handle.net/1808/14505

18. Sullivan, Bradley P. Role of Coagulation in Xenobiotic-Induced Liver Injury.

Degree: PhD, Pharmacology, Toxicology & Therapeutics, 2012, University of Kansas

 The liver is a common target for xenobiotic-induced toxicity. Of importance, synthesis of soluble coagulation factors by the liver plays an essential role in hemostasis.… (more)

Subjects/Keywords: Toxicology; Acetaminophen; Alpha-naphthylisothiocyanate; Blood coagulation; Fibrosis; Liver injury; Platelet

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APA (6th Edition):

Sullivan, B. P. (2012). Role of Coagulation in Xenobiotic-Induced Liver Injury. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/10291

Chicago Manual of Style (16th Edition):

Sullivan, Bradley P. “Role of Coagulation in Xenobiotic-Induced Liver Injury.” 2012. Doctoral Dissertation, University of Kansas. Accessed January 19, 2021. http://hdl.handle.net/1808/10291.

MLA Handbook (7th Edition):

Sullivan, Bradley P. “Role of Coagulation in Xenobiotic-Induced Liver Injury.” 2012. Web. 19 Jan 2021.

Vancouver:

Sullivan BP. Role of Coagulation in Xenobiotic-Induced Liver Injury. [Internet] [Doctoral dissertation]. University of Kansas; 2012. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/1808/10291.

Council of Science Editors:

Sullivan BP. Role of Coagulation in Xenobiotic-Induced Liver Injury. [Doctoral Dissertation]. University of Kansas; 2012. Available from: http://hdl.handle.net/1808/10291

19. Thomas, Ann M. THE ROLE OF EPIGENETICS IN TRANSCRIPTIONAL REGULATION OF FXR AND SILENCING FXR EXPRESSION IN HUMAN COLON CANCER.

Degree: PhD, Pharmacology, Toxicology & Therapeutics, 2011, University of Kansas

 Farnesoid X receptor (FXR) is a ligand activated transcription factor belonging to the nuclear receptor superfamily and bile acids are its endogenous ligands. FXR is… (more)

Subjects/Keywords: Cellular biology; Colon cancer; DNA methylation; Epigenetics; Farnesoid x receptor; Nuclear receptors

…Toxicology & Therapeutics at the University of Kansas Medical Center, particularly members of the… 

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APA (6th Edition):

Thomas, A. M. (2011). THE ROLE OF EPIGENETICS IN TRANSCRIPTIONAL REGULATION OF FXR AND SILENCING FXR EXPRESSION IN HUMAN COLON CANCER. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/8386

Chicago Manual of Style (16th Edition):

Thomas, Ann M. “THE ROLE OF EPIGENETICS IN TRANSCRIPTIONAL REGULATION OF FXR AND SILENCING FXR EXPRESSION IN HUMAN COLON CANCER.” 2011. Doctoral Dissertation, University of Kansas. Accessed January 19, 2021. http://hdl.handle.net/1808/8386.

MLA Handbook (7th Edition):

Thomas, Ann M. “THE ROLE OF EPIGENETICS IN TRANSCRIPTIONAL REGULATION OF FXR AND SILENCING FXR EXPRESSION IN HUMAN COLON CANCER.” 2011. Web. 19 Jan 2021.

Vancouver:

Thomas AM. THE ROLE OF EPIGENETICS IN TRANSCRIPTIONAL REGULATION OF FXR AND SILENCING FXR EXPRESSION IN HUMAN COLON CANCER. [Internet] [Doctoral dissertation]. University of Kansas; 2011. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/1808/8386.

Council of Science Editors:

Thomas AM. THE ROLE OF EPIGENETICS IN TRANSCRIPTIONAL REGULATION OF FXR AND SILENCING FXR EXPRESSION IN HUMAN COLON CANCER. [Doctoral Dissertation]. University of Kansas; 2011. Available from: http://hdl.handle.net/1808/8386

20. Shelton, Shary Nicole. MECHANISMS OF MITOCHONDRIA-MEDIATED APOPTOSIS INDUCED BY CYTOTOXIC STRESS.

Degree: PhD, Pharmacology, Toxicology & Therapeutics, 2010, University of Kansas

 Defects within the apoptotic pathway are thought to contribute to tumorigenesis and therapeutic resistance. Although most cytotoxic anti-cancer drugs are thought to activate the mitochondria-mediated… (more)

Subjects/Keywords: Molecular biology; Apoptosis; DNA damage; Hsp90

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APA (6th Edition):

Shelton, S. N. (2010). MECHANISMS OF MITOCHONDRIA-MEDIATED APOPTOSIS INDUCED BY CYTOTOXIC STRESS. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/6761

Chicago Manual of Style (16th Edition):

Shelton, Shary Nicole. “MECHANISMS OF MITOCHONDRIA-MEDIATED APOPTOSIS INDUCED BY CYTOTOXIC STRESS.” 2010. Doctoral Dissertation, University of Kansas. Accessed January 19, 2021. http://hdl.handle.net/1808/6761.

MLA Handbook (7th Edition):

Shelton, Shary Nicole. “MECHANISMS OF MITOCHONDRIA-MEDIATED APOPTOSIS INDUCED BY CYTOTOXIC STRESS.” 2010. Web. 19 Jan 2021.

Vancouver:

Shelton SN. MECHANISMS OF MITOCHONDRIA-MEDIATED APOPTOSIS INDUCED BY CYTOTOXIC STRESS. [Internet] [Doctoral dissertation]. University of Kansas; 2010. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/1808/6761.

Council of Science Editors:

Shelton SN. MECHANISMS OF MITOCHONDRIA-MEDIATED APOPTOSIS INDUCED BY CYTOTOXIC STRESS. [Doctoral Dissertation]. University of Kansas; 2010. Available from: http://hdl.handle.net/1808/6761

21. Araya, Juan Jose. Phase-Trafficking Methods in Natural Products, Modulators of Organic Anion Transporting Polypeptides from Rollinia emarginata, and Pregnane and Cardiac Glycosides from Asclepias spp.

Degree: PhD, Medicinal Chemistry, 2012, University of Kansas

 For decades, chemists and medicinal chemists have found in nature the source of inspiration for drug discovery and development. This work describes several aspects of… (more)

Subjects/Keywords: Organic chemistry; Asclepias; Cardenolides; Natural products; Nmr; Oatp; Phase-trafficking

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APA (6th Edition):

Araya, J. J. (2012). Phase-Trafficking Methods in Natural Products, Modulators of Organic Anion Transporting Polypeptides from Rollinia emarginata, and Pregnane and Cardiac Glycosides from Asclepias spp. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/9993

Chicago Manual of Style (16th Edition):

Araya, Juan Jose. “Phase-Trafficking Methods in Natural Products, Modulators of Organic Anion Transporting Polypeptides from Rollinia emarginata, and Pregnane and Cardiac Glycosides from Asclepias spp.” 2012. Doctoral Dissertation, University of Kansas. Accessed January 19, 2021. http://hdl.handle.net/1808/9993.

MLA Handbook (7th Edition):

Araya, Juan Jose. “Phase-Trafficking Methods in Natural Products, Modulators of Organic Anion Transporting Polypeptides from Rollinia emarginata, and Pregnane and Cardiac Glycosides from Asclepias spp.” 2012. Web. 19 Jan 2021.

Vancouver:

Araya JJ. Phase-Trafficking Methods in Natural Products, Modulators of Organic Anion Transporting Polypeptides from Rollinia emarginata, and Pregnane and Cardiac Glycosides from Asclepias spp. [Internet] [Doctoral dissertation]. University of Kansas; 2012. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/1808/9993.

Council of Science Editors:

Araya JJ. Phase-Trafficking Methods in Natural Products, Modulators of Organic Anion Transporting Polypeptides from Rollinia emarginata, and Pregnane and Cardiac Glycosides from Asclepias spp. [Doctoral Dissertation]. University of Kansas; 2012. Available from: http://hdl.handle.net/1808/9993

22. Wu, Kai Connie. THE ROLE OF NRF2 IN PREVENTING OXIDATIVE/ELECTROPHILIC STRESS-INDUCED LIVER INJURY.

Degree: PhD, Pharmacology, Toxicology & Therapeutics, 2012, University of Kansas

 Redox maintenance is critical for all biological species. Amplification of mechanisms that reduces oxidative/electrophilic stress promotes health and extends life. Nuclear factor, erythroid derived 2,… (more)

Subjects/Keywords: Toxicology; High throughput screening; Liver; Microarray; Nrf2; Oxidative stress

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APA (6th Edition):

Wu, K. C. (2012). THE ROLE OF NRF2 IN PREVENTING OXIDATIVE/ELECTROPHILIC STRESS-INDUCED LIVER INJURY. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/10289

Chicago Manual of Style (16th Edition):

Wu, Kai Connie. “THE ROLE OF NRF2 IN PREVENTING OXIDATIVE/ELECTROPHILIC STRESS-INDUCED LIVER INJURY.” 2012. Doctoral Dissertation, University of Kansas. Accessed January 19, 2021. http://hdl.handle.net/1808/10289.

MLA Handbook (7th Edition):

Wu, Kai Connie. “THE ROLE OF NRF2 IN PREVENTING OXIDATIVE/ELECTROPHILIC STRESS-INDUCED LIVER INJURY.” 2012. Web. 19 Jan 2021.

Vancouver:

Wu KC. THE ROLE OF NRF2 IN PREVENTING OXIDATIVE/ELECTROPHILIC STRESS-INDUCED LIVER INJURY. [Internet] [Doctoral dissertation]. University of Kansas; 2012. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/1808/10289.

Council of Science Editors:

Wu KC. THE ROLE OF NRF2 IN PREVENTING OXIDATIVE/ELECTROPHILIC STRESS-INDUCED LIVER INJURY. [Doctoral Dissertation]. University of Kansas; 2012. Available from: http://hdl.handle.net/1808/10289

23. Hays, Amanda Lynne. Targeting Organic Anion Transporting Polypeptides in Cancer to Improve Diagnostics and Therapy.

Degree: PhD, Pharmacology, Toxicology & Therapeutics, 2012, University of Kansas

 Organic Anion Transporting Polypeptides (OATPs) are multispecific transport proteins that mediate the uptake of numerous endogenous and exogenous compounds into cells. Recently, OATPs have been… (more)

Subjects/Keywords: Pharmacology; Cancer; Oatp; Transporters

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APA (6th Edition):

Hays, A. L. (2012). Targeting Organic Anion Transporting Polypeptides in Cancer to Improve Diagnostics and Therapy. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/11446

Chicago Manual of Style (16th Edition):

Hays, Amanda Lynne. “Targeting Organic Anion Transporting Polypeptides in Cancer to Improve Diagnostics and Therapy.” 2012. Doctoral Dissertation, University of Kansas. Accessed January 19, 2021. http://hdl.handle.net/1808/11446.

MLA Handbook (7th Edition):

Hays, Amanda Lynne. “Targeting Organic Anion Transporting Polypeptides in Cancer to Improve Diagnostics and Therapy.” 2012. Web. 19 Jan 2021.

Vancouver:

Hays AL. Targeting Organic Anion Transporting Polypeptides in Cancer to Improve Diagnostics and Therapy. [Internet] [Doctoral dissertation]. University of Kansas; 2012. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/1808/11446.

Council of Science Editors:

Hays AL. Targeting Organic Anion Transporting Polypeptides in Cancer to Improve Diagnostics and Therapy. [Doctoral Dissertation]. University of Kansas; 2012. Available from: http://hdl.handle.net/1808/11446


University of Kansas

24. Cui, Yue. DEVELOPMENTAL REGULATION OF THE DRUG-PROCESSING GENOME IN MOUSE LIVER.

Degree: PhD, Pharmacology, Toxicology & Therapeutics, 2010, University of Kansas

 Despite the recent progress in understanding the expression patterns and regulatory mechanisms of drug-processing genes, namely phase-I and -II drug metabolizing enzymes and transporters in… (more)

Subjects/Keywords: Health sciences; Toxicology; Pharmacology; Molecular biology; Bile acids; Drug metabolism; Epigenetics; Liver development; Nuclear receptors; Transporters

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APA (6th Edition):

Cui, Y. (2010). DEVELOPMENTAL REGULATION OF THE DRUG-PROCESSING GENOME IN MOUSE LIVER. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/6777

Chicago Manual of Style (16th Edition):

Cui, Yue. “DEVELOPMENTAL REGULATION OF THE DRUG-PROCESSING GENOME IN MOUSE LIVER.” 2010. Doctoral Dissertation, University of Kansas. Accessed January 19, 2021. http://hdl.handle.net/1808/6777.

MLA Handbook (7th Edition):

Cui, Yue. “DEVELOPMENTAL REGULATION OF THE DRUG-PROCESSING GENOME IN MOUSE LIVER.” 2010. Web. 19 Jan 2021.

Vancouver:

Cui Y. DEVELOPMENTAL REGULATION OF THE DRUG-PROCESSING GENOME IN MOUSE LIVER. [Internet] [Doctoral dissertation]. University of Kansas; 2010. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/1808/6777.

Council of Science Editors:

Cui Y. DEVELOPMENTAL REGULATION OF THE DRUG-PROCESSING GENOME IN MOUSE LIVER. [Doctoral Dissertation]. University of Kansas; 2010. Available from: http://hdl.handle.net/1808/6777


University of Kansas

25. Pacyniak, Erik Kristofer. Molecular Mechanism of Polybrominated Diphenyl Ether Disposition in the Liver.

Degree: PhD, Pharmacology, Toxicology & Therapeutics, 2010, University of Kansas

 Polybrominated diphenyl ethers (PBDEs) were introduced in the late 1970's as additive flame retardants incorporated into textiles, electronics, plastics and furniture. Although 2,2',3,3',4,4',5,5',6,6'-decabromodiphenyl ether (BDE209)… (more)

Subjects/Keywords: Toxicology; Constitutive androstane receptor; Nuclear receptors; Organic anion transporting polypeptide; Polybrominated diphenyl ethers; Pregnane x receptor

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APA (6th Edition):

Pacyniak, E. K. (2010). Molecular Mechanism of Polybrominated Diphenyl Ether Disposition in the Liver. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/7717

Chicago Manual of Style (16th Edition):

Pacyniak, Erik Kristofer. “Molecular Mechanism of Polybrominated Diphenyl Ether Disposition in the Liver.” 2010. Doctoral Dissertation, University of Kansas. Accessed January 19, 2021. http://hdl.handle.net/1808/7717.

MLA Handbook (7th Edition):

Pacyniak, Erik Kristofer. “Molecular Mechanism of Polybrominated Diphenyl Ether Disposition in the Liver.” 2010. Web. 19 Jan 2021.

Vancouver:

Pacyniak EK. Molecular Mechanism of Polybrominated Diphenyl Ether Disposition in the Liver. [Internet] [Doctoral dissertation]. University of Kansas; 2010. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/1808/7717.

Council of Science Editors:

Pacyniak EK. Molecular Mechanism of Polybrominated Diphenyl Ether Disposition in the Liver. [Doctoral Dissertation]. University of Kansas; 2010. Available from: http://hdl.handle.net/1808/7717


University of Kansas

26. Wang, Pan. Computational Molecular Modeling Studies of the Interactions of Estrogens with Their Receptors and Intracellular Estrogen Binding Protein PDIp.

Degree: PhD, Pharmacology, Toxicology & Therapeutics, 2010, University of Kansas

 The endogenous estrogens are vitally-important female sex hormones with diverse biological functions. Disruption of their actions contributes to the pathogenesis of a number of disease… (more)

Subjects/Keywords: Pharmacology; Antiestrogen; Estrogen; Estrogen receptor; Molecular docking; Molecular modeling; Pdip

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APA (6th Edition):

Wang, P. (2010). Computational Molecular Modeling Studies of the Interactions of Estrogens with Their Receptors and Intracellular Estrogen Binding Protein PDIp. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/7432

Chicago Manual of Style (16th Edition):

Wang, Pan. “Computational Molecular Modeling Studies of the Interactions of Estrogens with Their Receptors and Intracellular Estrogen Binding Protein PDIp.” 2010. Doctoral Dissertation, University of Kansas. Accessed January 19, 2021. http://hdl.handle.net/1808/7432.

MLA Handbook (7th Edition):

Wang, Pan. “Computational Molecular Modeling Studies of the Interactions of Estrogens with Their Receptors and Intracellular Estrogen Binding Protein PDIp.” 2010. Web. 19 Jan 2021.

Vancouver:

Wang P. Computational Molecular Modeling Studies of the Interactions of Estrogens with Their Receptors and Intracellular Estrogen Binding Protein PDIp. [Internet] [Doctoral dissertation]. University of Kansas; 2010. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/1808/7432.

Council of Science Editors:

Wang P. Computational Molecular Modeling Studies of the Interactions of Estrogens with Their Receptors and Intracellular Estrogen Binding Protein PDIp. [Doctoral Dissertation]. University of Kansas; 2010. Available from: http://hdl.handle.net/1808/7432


University of Kansas

27. Song, Peizhen. EFFECT OF BILE ACID FEEDING AND SEQUESTRATION ON LIVER BILE ACID COMPOSITION AND GENE REGULATION IN MICE.

Degree: PhD, Pharmacology, Toxicology & Therapeutics, 2010, University of Kansas

 The dissertation investigates the non-hepatotoxic doses of five bile acids (BAs) in the feed of mice, as well as adaptations in the expression of genes… (more)

Subjects/Keywords: Toxicology; Pathology; Bile acids; Cholestyramine resin; Cyp7a1; Cyp8b1; Fgf15; Fxr

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APA (6th Edition):

Song, P. (2010). EFFECT OF BILE ACID FEEDING AND SEQUESTRATION ON LIVER BILE ACID COMPOSITION AND GENE REGULATION IN MICE. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/7756

Chicago Manual of Style (16th Edition):

Song, Peizhen. “EFFECT OF BILE ACID FEEDING AND SEQUESTRATION ON LIVER BILE ACID COMPOSITION AND GENE REGULATION IN MICE.” 2010. Doctoral Dissertation, University of Kansas. Accessed January 19, 2021. http://hdl.handle.net/1808/7756.

MLA Handbook (7th Edition):

Song, Peizhen. “EFFECT OF BILE ACID FEEDING AND SEQUESTRATION ON LIVER BILE ACID COMPOSITION AND GENE REGULATION IN MICE.” 2010. Web. 19 Jan 2021.

Vancouver:

Song P. EFFECT OF BILE ACID FEEDING AND SEQUESTRATION ON LIVER BILE ACID COMPOSITION AND GENE REGULATION IN MICE. [Internet] [Doctoral dissertation]. University of Kansas; 2010. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/1808/7756.

Council of Science Editors:

Song P. EFFECT OF BILE ACID FEEDING AND SEQUESTRATION ON LIVER BILE ACID COMPOSITION AND GENE REGULATION IN MICE. [Doctoral Dissertation]. University of Kansas; 2010. Available from: http://hdl.handle.net/1808/7756


University of Kansas

28. Bu, Pengli. RETINOID-INDUCED APOPTOSIS AND PROLIFERATION OF HEPATOCYTES ARE MEDIATED BY DISTINCT NUCLEAR RECEPTORS.

Degree: PhD, Pharmacology, Toxicology & Therapeutics, 2009, University of Kansas

 Retinoids, derivatives of vitamin A, are important signaling molecules regulating cellular homeostasis including differentiation, apoptosis, and proliferation. In this dissertation, we examined the versatile effects… (more)

Subjects/Keywords: Health sciences; Toxicology; Pharmacology; Apoptosis; Hepatocyte; Nuclear receptors; Proliferation; Retinoid

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APA (6th Edition):

Bu, P. (2009). RETINOID-INDUCED APOPTOSIS AND PROLIFERATION OF HEPATOCYTES ARE MEDIATED BY DISTINCT NUCLEAR RECEPTORS. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/5385

Chicago Manual of Style (16th Edition):

Bu, Pengli. “RETINOID-INDUCED APOPTOSIS AND PROLIFERATION OF HEPATOCYTES ARE MEDIATED BY DISTINCT NUCLEAR RECEPTORS.” 2009. Doctoral Dissertation, University of Kansas. Accessed January 19, 2021. http://hdl.handle.net/1808/5385.

MLA Handbook (7th Edition):

Bu, Pengli. “RETINOID-INDUCED APOPTOSIS AND PROLIFERATION OF HEPATOCYTES ARE MEDIATED BY DISTINCT NUCLEAR RECEPTORS.” 2009. Web. 19 Jan 2021.

Vancouver:

Bu P. RETINOID-INDUCED APOPTOSIS AND PROLIFERATION OF HEPATOCYTES ARE MEDIATED BY DISTINCT NUCLEAR RECEPTORS. [Internet] [Doctoral dissertation]. University of Kansas; 2009. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/1808/5385.

Council of Science Editors:

Bu P. RETINOID-INDUCED APOPTOSIS AND PROLIFERATION OF HEPATOCYTES ARE MEDIATED BY DISTINCT NUCLEAR RECEPTORS. [Doctoral Dissertation]. University of Kansas; 2009. Available from: http://hdl.handle.net/1808/5385


University of Kansas

29. Weaver, Yi Miao. STRUCTURAL REQUIREMENTS OF ORGANIC ANION TRANSPORTING POLYPEPTIDE MEDIATED TRANSPORT.

Degree: PhD, Pharmacology, Toxicology & Therapeutics, 2010, University of Kansas

 The organic anion transporting polypeptides (human: OATP; other: Oatp) form a mammalian transporter superfamily that mediates the transport of structurally unrelated compounds across the cell… (more)

Subjects/Keywords: Health sciences; Pharmacology; Biology; Physiology; Oatp; Organic anion; Perfluoronated carboxylates; Pfca; Structure; Transporters

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APA (6th Edition):

Weaver, Y. M. (2010). STRUCTURAL REQUIREMENTS OF ORGANIC ANION TRANSPORTING POLYPEPTIDE MEDIATED TRANSPORT. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/7394

Chicago Manual of Style (16th Edition):

Weaver, Yi Miao. “STRUCTURAL REQUIREMENTS OF ORGANIC ANION TRANSPORTING POLYPEPTIDE MEDIATED TRANSPORT.” 2010. Doctoral Dissertation, University of Kansas. Accessed January 19, 2021. http://hdl.handle.net/1808/7394.

MLA Handbook (7th Edition):

Weaver, Yi Miao. “STRUCTURAL REQUIREMENTS OF ORGANIC ANION TRANSPORTING POLYPEPTIDE MEDIATED TRANSPORT.” 2010. Web. 19 Jan 2021.

Vancouver:

Weaver YM. STRUCTURAL REQUIREMENTS OF ORGANIC ANION TRANSPORTING POLYPEPTIDE MEDIATED TRANSPORT. [Internet] [Doctoral dissertation]. University of Kansas; 2010. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/1808/7394.

Council of Science Editors:

Weaver YM. STRUCTURAL REQUIREMENTS OF ORGANIC ANION TRANSPORTING POLYPEPTIDE MEDIATED TRANSPORT. [Doctoral Dissertation]. University of Kansas; 2010. Available from: http://hdl.handle.net/1808/7394

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