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You searched for +publisher:"University of Kansas" +contributor:("Ding, Wen-Xing"). Showing records 1 – 18 of 18 total matches.

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University of Kansas

1. Woolbright, Ben. The role of bile acids during cholestasis in mice and man.

Degree: PhD, Pharmacology, Toxicology & Therapeutics, 2015, University of Kansas

 Cholestasis is a reduction in bile flow that occurs during numerous pathologies. Cholestasis leads to significant liver toxicity, biliary hyperplasia, and liver cirrhosis. The molecular… (more)

Subjects/Keywords: Toxicology; Pathology; Pharmacology; apoptosis; bile acid; cholestasis; hepatocytes; inflammation; neutrophil

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APA (6th Edition):

Woolbright, B. (2015). The role of bile acids during cholestasis in mice and man. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/19455

Chicago Manual of Style (16th Edition):

Woolbright, Ben. “The role of bile acids during cholestasis in mice and man.” 2015. Doctoral Dissertation, University of Kansas. Accessed July 23, 2019. http://hdl.handle.net/1808/19455.

MLA Handbook (7th Edition):

Woolbright, Ben. “The role of bile acids during cholestasis in mice and man.” 2015. Web. 23 Jul 2019.

Vancouver:

Woolbright B. The role of bile acids during cholestasis in mice and man. [Internet] [Doctoral dissertation]. University of Kansas; 2015. [cited 2019 Jul 23]. Available from: http://hdl.handle.net/1808/19455.

Council of Science Editors:

Woolbright B. The role of bile acids during cholestasis in mice and man. [Doctoral Dissertation]. University of Kansas; 2015. Available from: http://hdl.handle.net/1808/19455


University of Kansas

2. Polireddy, Kishore. Identifying Novel Inhibitors of Epithelial to Mesenchymal Transition (EMT) for Targeting Pancreatic Cancer Metastasis and Cancer Stem Cells.

Degree: PhD, Pharmacology, Toxicology & Therapeutics, 2015, University of Kansas

 Pancreatic cancer is the fourth leading cause of cancer-related death in the United States, and it is expected to become the second-leading cause of cancer-related… (more)

Subjects/Keywords: Oncology; Biology; Cancer stem cells; EMT; Pancreatic cancer

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APA (6th Edition):

Polireddy, K. (2015). Identifying Novel Inhibitors of Epithelial to Mesenchymal Transition (EMT) for Targeting Pancreatic Cancer Metastasis and Cancer Stem Cells. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/21926

Chicago Manual of Style (16th Edition):

Polireddy, Kishore. “Identifying Novel Inhibitors of Epithelial to Mesenchymal Transition (EMT) for Targeting Pancreatic Cancer Metastasis and Cancer Stem Cells.” 2015. Doctoral Dissertation, University of Kansas. Accessed July 23, 2019. http://hdl.handle.net/1808/21926.

MLA Handbook (7th Edition):

Polireddy, Kishore. “Identifying Novel Inhibitors of Epithelial to Mesenchymal Transition (EMT) for Targeting Pancreatic Cancer Metastasis and Cancer Stem Cells.” 2015. Web. 23 Jul 2019.

Vancouver:

Polireddy K. Identifying Novel Inhibitors of Epithelial to Mesenchymal Transition (EMT) for Targeting Pancreatic Cancer Metastasis and Cancer Stem Cells. [Internet] [Doctoral dissertation]. University of Kansas; 2015. [cited 2019 Jul 23]. Available from: http://hdl.handle.net/1808/21926.

Council of Science Editors:

Polireddy K. Identifying Novel Inhibitors of Epithelial to Mesenchymal Transition (EMT) for Targeting Pancreatic Cancer Metastasis and Cancer Stem Cells. [Doctoral Dissertation]. University of Kansas; 2015. Available from: http://hdl.handle.net/1808/21926


University of Kansas

3. Xie, Yuchao. ACETAMINOPHEN HEPATOTOXICITY IN PRIMARY HUMAN HEPATOCYTES AND MICE.

Degree: PhD, Pharmacology, Toxicology & Therapeutics, 2016, University of Kansas

 Acetaminophen is the most prevalent cause of acute liver failure (ALF) and drug-induced liver injury (DILI) in western countries. Extensive studies have revealed important intracellular… (more)

Subjects/Keywords: Toxicology

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APA (6th Edition):

Xie, Y. (2016). ACETAMINOPHEN HEPATOTOXICITY IN PRIMARY HUMAN HEPATOCYTES AND MICE. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/22502

Chicago Manual of Style (16th Edition):

Xie, Yuchao. “ACETAMINOPHEN HEPATOTOXICITY IN PRIMARY HUMAN HEPATOCYTES AND MICE.” 2016. Doctoral Dissertation, University of Kansas. Accessed July 23, 2019. http://hdl.handle.net/1808/22502.

MLA Handbook (7th Edition):

Xie, Yuchao. “ACETAMINOPHEN HEPATOTOXICITY IN PRIMARY HUMAN HEPATOCYTES AND MICE.” 2016. Web. 23 Jul 2019.

Vancouver:

Xie Y. ACETAMINOPHEN HEPATOTOXICITY IN PRIMARY HUMAN HEPATOCYTES AND MICE. [Internet] [Doctoral dissertation]. University of Kansas; 2016. [cited 2019 Jul 23]. Available from: http://hdl.handle.net/1808/22502.

Council of Science Editors:

Xie Y. ACETAMINOPHEN HEPATOTOXICITY IN PRIMARY HUMAN HEPATOCYTES AND MICE. [Doctoral Dissertation]. University of Kansas; 2016. Available from: http://hdl.handle.net/1808/22502


University of Kansas

4. Du, Kuo. MITOCHONDRIAL OXIDATIVE STRESS AND BIOGENESIS DURING ACETAMINOPHEN HEPATOTOXICITY.

Degree: PhD, Pharmacology, Toxicology & Therapeutics, 2017, University of Kansas

 Acetaminophen (APAP) overdose causes severe hepatotoxicity in animals and humans. Although numerous studies have established the existence of an extensive oxidative stress during APAP hepatotoxicity,… (more)

Subjects/Keywords: Toxicology; Pharmacology; Molecular biology; Acetaminophen; liver toxicity; metformin; mitochondria; mito-tempo; oxidative stress

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APA (6th Edition):

Du, K. (2017). MITOCHONDRIAL OXIDATIVE STRESS AND BIOGENESIS DURING ACETAMINOPHEN HEPATOTOXICITY. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/26928

Chicago Manual of Style (16th Edition):

Du, Kuo. “MITOCHONDRIAL OXIDATIVE STRESS AND BIOGENESIS DURING ACETAMINOPHEN HEPATOTOXICITY.” 2017. Doctoral Dissertation, University of Kansas. Accessed July 23, 2019. http://hdl.handle.net/1808/26928.

MLA Handbook (7th Edition):

Du, Kuo. “MITOCHONDRIAL OXIDATIVE STRESS AND BIOGENESIS DURING ACETAMINOPHEN HEPATOTOXICITY.” 2017. Web. 23 Jul 2019.

Vancouver:

Du K. MITOCHONDRIAL OXIDATIVE STRESS AND BIOGENESIS DURING ACETAMINOPHEN HEPATOTOXICITY. [Internet] [Doctoral dissertation]. University of Kansas; 2017. [cited 2019 Jul 23]. Available from: http://hdl.handle.net/1808/26928.

Council of Science Editors:

Du K. MITOCHONDRIAL OXIDATIVE STRESS AND BIOGENESIS DURING ACETAMINOPHEN HEPATOTOXICITY. [Doctoral Dissertation]. University of Kansas; 2017. Available from: http://hdl.handle.net/1808/26928


University of Kansas

5. Weemhoff, James Lawrence. Mechanistic Biomarkers in Acute Liver Injury.

Degree: PhD, Pharmacology, Toxicology & Therapeutics, 2017, University of Kansas

 Acute liver failure continues to be a major medical problem. There are many underlying causes of acute liver failure, but drug induced liver injury is… (more)

Subjects/Keywords: Toxicology; Acetaminophen; Biomarkers; HepaRG; Hypoxic Hepatitis; Liver Injury; Liver Transplantation

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APA (6th Edition):

Weemhoff, J. L. (2017). Mechanistic Biomarkers in Acute Liver Injury. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/27044

Chicago Manual of Style (16th Edition):

Weemhoff, James Lawrence. “Mechanistic Biomarkers in Acute Liver Injury.” 2017. Doctoral Dissertation, University of Kansas. Accessed July 23, 2019. http://hdl.handle.net/1808/27044.

MLA Handbook (7th Edition):

Weemhoff, James Lawrence. “Mechanistic Biomarkers in Acute Liver Injury.” 2017. Web. 23 Jul 2019.

Vancouver:

Weemhoff JL. Mechanistic Biomarkers in Acute Liver Injury. [Internet] [Doctoral dissertation]. University of Kansas; 2017. [cited 2019 Jul 23]. Available from: http://hdl.handle.net/1808/27044.

Council of Science Editors:

Weemhoff JL. Mechanistic Biomarkers in Acute Liver Injury. [Doctoral Dissertation]. University of Kansas; 2017. Available from: http://hdl.handle.net/1808/27044

6. Williams, Jessica A. R. The Role of Parkin and Mitophagy in Acetaminophen and Alcohol-induced Liver Injuries.

Degree: PhD, Pharmacology, Toxicology & Therapeutics, 2015, University of Kansas

 Acetaminophen (APAP) is the leading cause of acute liver failure in the United States, and alcoholic liver disease (ALD) is a worldwide health problem that… (more)

Subjects/Keywords: Toxicology; Acetaminophen; Alcohol; Autophagy; Liver Injury; Mitophagy; Parkin

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APA (6th Edition):

Williams, J. A. R. (2015). The Role of Parkin and Mitophagy in Acetaminophen and Alcohol-induced Liver Injuries. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/19470

Chicago Manual of Style (16th Edition):

Williams, Jessica A R. “The Role of Parkin and Mitophagy in Acetaminophen and Alcohol-induced Liver Injuries.” 2015. Doctoral Dissertation, University of Kansas. Accessed July 23, 2019. http://hdl.handle.net/1808/19470.

MLA Handbook (7th Edition):

Williams, Jessica A R. “The Role of Parkin and Mitophagy in Acetaminophen and Alcohol-induced Liver Injuries.” 2015. Web. 23 Jul 2019.

Vancouver:

Williams JAR. The Role of Parkin and Mitophagy in Acetaminophen and Alcohol-induced Liver Injuries. [Internet] [Doctoral dissertation]. University of Kansas; 2015. [cited 2019 Jul 23]. Available from: http://hdl.handle.net/1808/19470.

Council of Science Editors:

Williams JAR. The Role of Parkin and Mitophagy in Acetaminophen and Alcohol-induced Liver Injuries. [Doctoral Dissertation]. University of Kansas; 2015. Available from: http://hdl.handle.net/1808/19470


University of Kansas

7. McCracken, Jennifer M. HEPATIC WOUND HEALING FOLLOWING ACUTE AND CHRONIC LIVER INJURY: A POTENTIAL ROLE FOR THE HYALURONAN NETWORK.

Degree: PhD, Pharmacology, Toxicology & Therapeutics, 2017, University of Kansas

 Chronic liver disease is the 12th leading cause of death in the United States and consists of a continuum of pathologies. Following initial injury, a… (more)

Subjects/Keywords: Toxicology; hyaluronan; hyaluronan mediated motility receptor; liver injury; liver wound healing

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APA (6th Edition):

McCracken, J. M. (2017). HEPATIC WOUND HEALING FOLLOWING ACUTE AND CHRONIC LIVER INJURY: A POTENTIAL ROLE FOR THE HYALURONAN NETWORK. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/26930

Chicago Manual of Style (16th Edition):

McCracken, Jennifer M. “HEPATIC WOUND HEALING FOLLOWING ACUTE AND CHRONIC LIVER INJURY: A POTENTIAL ROLE FOR THE HYALURONAN NETWORK.” 2017. Doctoral Dissertation, University of Kansas. Accessed July 23, 2019. http://hdl.handle.net/1808/26930.

MLA Handbook (7th Edition):

McCracken, Jennifer M. “HEPATIC WOUND HEALING FOLLOWING ACUTE AND CHRONIC LIVER INJURY: A POTENTIAL ROLE FOR THE HYALURONAN NETWORK.” 2017. Web. 23 Jul 2019.

Vancouver:

McCracken JM. HEPATIC WOUND HEALING FOLLOWING ACUTE AND CHRONIC LIVER INJURY: A POTENTIAL ROLE FOR THE HYALURONAN NETWORK. [Internet] [Doctoral dissertation]. University of Kansas; 2017. [cited 2019 Jul 23]. Available from: http://hdl.handle.net/1808/26930.

Council of Science Editors:

McCracken JM. HEPATIC WOUND HEALING FOLLOWING ACUTE AND CHRONIC LIVER INJURY: A POTENTIAL ROLE FOR THE HYALURONAN NETWORK. [Doctoral Dissertation]. University of Kansas; 2017. Available from: http://hdl.handle.net/1808/26930


University of Kansas

8. Fu, Zidong. Regulation of Xenobiotic and Bile Acid Metabolism by the Anti-aging Intervention Calorie Restriction in Mice.

Degree: PhD, Pharmacology, Toxicology & Therapeutics, 2013, University of Kansas

 Calorie restriction (CR), defined as reduced calorie intake without causing malnutrition, is the best-known intervention to increase life span and slow aging-related diseases in various… (more)

Subjects/Keywords: Toxicology; Pharmacology; Animal sciences; aging; bile acids; calorie restriction; xenobiotic metabolism and detoxification

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APA (6th Edition):

Fu, Z. (2013). Regulation of Xenobiotic and Bile Acid Metabolism by the Anti-aging Intervention Calorie Restriction in Mice. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/19597

Chicago Manual of Style (16th Edition):

Fu, Zidong. “Regulation of Xenobiotic and Bile Acid Metabolism by the Anti-aging Intervention Calorie Restriction in Mice.” 2013. Doctoral Dissertation, University of Kansas. Accessed July 23, 2019. http://hdl.handle.net/1808/19597.

MLA Handbook (7th Edition):

Fu, Zidong. “Regulation of Xenobiotic and Bile Acid Metabolism by the Anti-aging Intervention Calorie Restriction in Mice.” 2013. Web. 23 Jul 2019.

Vancouver:

Fu Z. Regulation of Xenobiotic and Bile Acid Metabolism by the Anti-aging Intervention Calorie Restriction in Mice. [Internet] [Doctoral dissertation]. University of Kansas; 2013. [cited 2019 Jul 23]. Available from: http://hdl.handle.net/1808/19597.

Council of Science Editors:

Fu Z. Regulation of Xenobiotic and Bile Acid Metabolism by the Anti-aging Intervention Calorie Restriction in Mice. [Doctoral Dissertation]. University of Kansas; 2013. Available from: http://hdl.handle.net/1808/19597


University of Kansas

9. Jones-Jamtgaard, Kellyann Nadira. Hepatitis C Virus Suppresses Autophagosome Degradation by Arl8b-mediated Lysosomal Positioning.

Degree: PhD, Microbiology, Molecular Genetics & Immunology, 2016, University of Kansas

 HCV infection increases steady-state autophagosome numbers but the mechanism of this effect is poorly understood. Flux assays suggest that an increase in autophagosomes results from… (more)

Subjects/Keywords: Virology; Microbiology; Arl8b; autophagy; GTPase; Hepatitis C Virus; lysosome

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APA (6th Edition):

Jones-Jamtgaard, K. N. (2016). Hepatitis C Virus Suppresses Autophagosome Degradation by Arl8b-mediated Lysosomal Positioning. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/25879

Chicago Manual of Style (16th Edition):

Jones-Jamtgaard, Kellyann Nadira. “Hepatitis C Virus Suppresses Autophagosome Degradation by Arl8b-mediated Lysosomal Positioning.” 2016. Doctoral Dissertation, University of Kansas. Accessed July 23, 2019. http://hdl.handle.net/1808/25879.

MLA Handbook (7th Edition):

Jones-Jamtgaard, Kellyann Nadira. “Hepatitis C Virus Suppresses Autophagosome Degradation by Arl8b-mediated Lysosomal Positioning.” 2016. Web. 23 Jul 2019.

Vancouver:

Jones-Jamtgaard KN. Hepatitis C Virus Suppresses Autophagosome Degradation by Arl8b-mediated Lysosomal Positioning. [Internet] [Doctoral dissertation]. University of Kansas; 2016. [cited 2019 Jul 23]. Available from: http://hdl.handle.net/1808/25879.

Council of Science Editors:

Jones-Jamtgaard KN. Hepatitis C Virus Suppresses Autophagosome Degradation by Arl8b-mediated Lysosomal Positioning. [Doctoral Dissertation]. University of Kansas; 2016. Available from: http://hdl.handle.net/1808/25879


University of Kansas

10. Borude, Prachi C. ROLE OF DNA DAMAGE RESPONSE IN LIVER REGENERATION AFTER APAP OVERDOSE INDUCED ALF.

Degree: PhD, Pharmacology, Toxicology & Therapeutics, 2017, University of Kansas

 Acetaminophen (APAP) overdose is a leading cause of acute liver failure (ALF) with limited treatment options. The mechanisms of APAP-induced liver injury include formation of… (more)

Subjects/Keywords: Toxicology

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APA (6th Edition):

Borude, P. C. (2017). ROLE OF DNA DAMAGE RESPONSE IN LIVER REGENERATION AFTER APAP OVERDOSE INDUCED ALF. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/27046

Chicago Manual of Style (16th Edition):

Borude, Prachi C. “ROLE OF DNA DAMAGE RESPONSE IN LIVER REGENERATION AFTER APAP OVERDOSE INDUCED ALF.” 2017. Doctoral Dissertation, University of Kansas. Accessed July 23, 2019. http://hdl.handle.net/1808/27046.

MLA Handbook (7th Edition):

Borude, Prachi C. “ROLE OF DNA DAMAGE RESPONSE IN LIVER REGENERATION AFTER APAP OVERDOSE INDUCED ALF.” 2017. Web. 23 Jul 2019.

Vancouver:

Borude PC. ROLE OF DNA DAMAGE RESPONSE IN LIVER REGENERATION AFTER APAP OVERDOSE INDUCED ALF. [Internet] [Doctoral dissertation]. University of Kansas; 2017. [cited 2019 Jul 23]. Available from: http://hdl.handle.net/1808/27046.

Council of Science Editors:

Borude PC. ROLE OF DNA DAMAGE RESPONSE IN LIVER REGENERATION AFTER APAP OVERDOSE INDUCED ALF. [Doctoral Dissertation]. University of Kansas; 2017. Available from: http://hdl.handle.net/1808/27046


University of Kansas

11. New, Jacob. Autophagy in Head and Neck Cancer Associated Fibroblasts.

Degree: PhD, Anatomy & Cell Biology, 2018, University of Kansas

 Head and neck squamous cell carcinoma (HNSCC) is a devastating disease. Despite therapeutic advancements, little change in 5-year survival has been made for patients with… (more)

Subjects/Keywords: Cellular biology; Autophagy; Head and Neck Cancer; Tumor Microenvironment

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APA (6th Edition):

New, J. (2018). Autophagy in Head and Neck Cancer Associated Fibroblasts. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/27070

Chicago Manual of Style (16th Edition):

New, Jacob. “Autophagy in Head and Neck Cancer Associated Fibroblasts.” 2018. Doctoral Dissertation, University of Kansas. Accessed July 23, 2019. http://hdl.handle.net/1808/27070.

MLA Handbook (7th Edition):

New, Jacob. “Autophagy in Head and Neck Cancer Associated Fibroblasts.” 2018. Web. 23 Jul 2019.

Vancouver:

New J. Autophagy in Head and Neck Cancer Associated Fibroblasts. [Internet] [Doctoral dissertation]. University of Kansas; 2018. [cited 2019 Jul 23]. Available from: http://hdl.handle.net/1808/27070.

Council of Science Editors:

New J. Autophagy in Head and Neck Cancer Associated Fibroblasts. [Doctoral Dissertation]. University of Kansas; 2018. Available from: http://hdl.handle.net/1808/27070


University of Kansas

12. Bastola, Prabhakar. Targeting vulnerabilities through inhibiting the valosin-containing protein (VCP/p97) in ovarian cancer.

Degree: PhD, Pharmacology, Toxicology & Therapeutics, 2018, University of Kansas

 Ovarian cancer is the fifth leading cause of cancer-related death among women and the deadliest of all gynecological cancers. Treatment failure is a major contributing… (more)

Subjects/Keywords: Pharmacology; endoplasmic reticulum stress; ovarian cancer; protein quality control; unfolded protein response; Valosin-containing protein; VCP/p97 inhibitors

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APA (6th Edition):

Bastola, P. (2018). Targeting vulnerabilities through inhibiting the valosin-containing protein (VCP/p97) in ovarian cancer. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/27334

Chicago Manual of Style (16th Edition):

Bastola, Prabhakar. “Targeting vulnerabilities through inhibiting the valosin-containing protein (VCP/p97) in ovarian cancer.” 2018. Doctoral Dissertation, University of Kansas. Accessed July 23, 2019. http://hdl.handle.net/1808/27334.

MLA Handbook (7th Edition):

Bastola, Prabhakar. “Targeting vulnerabilities through inhibiting the valosin-containing protein (VCP/p97) in ovarian cancer.” 2018. Web. 23 Jul 2019.

Vancouver:

Bastola P. Targeting vulnerabilities through inhibiting the valosin-containing protein (VCP/p97) in ovarian cancer. [Internet] [Doctoral dissertation]. University of Kansas; 2018. [cited 2019 Jul 23]. Available from: http://hdl.handle.net/1808/27334.

Council of Science Editors:

Bastola P. Targeting vulnerabilities through inhibiting the valosin-containing protein (VCP/p97) in ovarian cancer. [Doctoral Dissertation]. University of Kansas; 2018. Available from: http://hdl.handle.net/1808/27334


University of Kansas

13. Fang, Pingping. CHARACTERIZATION OF MOLECULAR AND GENETIC DETERMINANTS OF POLY (ADP-RIBOSE) POLYMERASE INHIBITOR SENSITIVITY IN OVARIAN CANCER.

Degree: PhD, Pharmacology, Toxicology & Therapeutics, 2018, University of Kansas

 Ovarian cancer is the most leathal cancer among gynecologic cancers in the United States and is the fifth leading cause of cancer death among American… (more)

Subjects/Keywords: Biology; Cellular biology; Pharmacology; adaptive response; DNA repair; homologous recombination; ovarian cancer; PARP inhibitor; resistance

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APA (6th Edition):

Fang, P. (2018). CHARACTERIZATION OF MOLECULAR AND GENETIC DETERMINANTS OF POLY (ADP-RIBOSE) POLYMERASE INHIBITOR SENSITIVITY IN OVARIAN CANCER. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/27335

Chicago Manual of Style (16th Edition):

Fang, Pingping. “CHARACTERIZATION OF MOLECULAR AND GENETIC DETERMINANTS OF POLY (ADP-RIBOSE) POLYMERASE INHIBITOR SENSITIVITY IN OVARIAN CANCER.” 2018. Doctoral Dissertation, University of Kansas. Accessed July 23, 2019. http://hdl.handle.net/1808/27335.

MLA Handbook (7th Edition):

Fang, Pingping. “CHARACTERIZATION OF MOLECULAR AND GENETIC DETERMINANTS OF POLY (ADP-RIBOSE) POLYMERASE INHIBITOR SENSITIVITY IN OVARIAN CANCER.” 2018. Web. 23 Jul 2019.

Vancouver:

Fang P. CHARACTERIZATION OF MOLECULAR AND GENETIC DETERMINANTS OF POLY (ADP-RIBOSE) POLYMERASE INHIBITOR SENSITIVITY IN OVARIAN CANCER. [Internet] [Doctoral dissertation]. University of Kansas; 2018. [cited 2019 Jul 23]. Available from: http://hdl.handle.net/1808/27335.

Council of Science Editors:

Fang P. CHARACTERIZATION OF MOLECULAR AND GENETIC DETERMINANTS OF POLY (ADP-RIBOSE) POLYMERASE INHIBITOR SENSITIVITY IN OVARIAN CANCER. [Doctoral Dissertation]. University of Kansas; 2018. Available from: http://hdl.handle.net/1808/27335


University of Kansas

14. Bhushan, Bharat. MECHANISMS OF LIVER REGENERATION AFTER ACETAMINOPHEN-INDUCED HEPATOTOXICITY IN MICE.

Degree: PhD, Pharmacology, Toxicology & Therapeutics, 2016, University of Kansas

 Overdose of acetaminophen (APAP) is the major cause of acute liver failure (ALF) in the western world with very limited treatment options. Recent studies suggest… (more)

Subjects/Keywords: Toxicology; Pharmacology; Acetaminophen; EGF receptor; Liver; mice model; Regeneration; Wnt-Beta Catenin-GSK3 Beta pathway

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APA (6th Edition):

Bhushan, B. (2016). MECHANISMS OF LIVER REGENERATION AFTER ACETAMINOPHEN-INDUCED HEPATOTOXICITY IN MICE. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/25366

Chicago Manual of Style (16th Edition):

Bhushan, Bharat. “MECHANISMS OF LIVER REGENERATION AFTER ACETAMINOPHEN-INDUCED HEPATOTOXICITY IN MICE.” 2016. Doctoral Dissertation, University of Kansas. Accessed July 23, 2019. http://hdl.handle.net/1808/25366.

MLA Handbook (7th Edition):

Bhushan, Bharat. “MECHANISMS OF LIVER REGENERATION AFTER ACETAMINOPHEN-INDUCED HEPATOTOXICITY IN MICE.” 2016. Web. 23 Jul 2019.

Vancouver:

Bhushan B. MECHANISMS OF LIVER REGENERATION AFTER ACETAMINOPHEN-INDUCED HEPATOTOXICITY IN MICE. [Internet] [Doctoral dissertation]. University of Kansas; 2016. [cited 2019 Jul 23]. Available from: http://hdl.handle.net/1808/25366.

Council of Science Editors:

Bhushan B. MECHANISMS OF LIVER REGENERATION AFTER ACETAMINOPHEN-INDUCED HEPATOTOXICITY IN MICE. [Doctoral Dissertation]. University of Kansas; 2016. Available from: http://hdl.handle.net/1808/25366

15. Manley, Sharon. ROLE OF BILE ACIDS AND FARNESOID X RECEPTOR IN HEPATIC AUTOPHAGY AND ITS IMPLICATIONS IN ETHANOL-INDUCED HEPATOTOXICITY.

Degree: PhD, Pharmacology, Toxicology & Therapeutics, 2014, University of Kansas

 Retention of bile acids (BAs) in the liver during cholestasis plays an important role in the development of cholestatic liver injury. Several studies have reported… (more)

Subjects/Keywords: Toxicology; Alcoholic Liver Disease; Autophagy; Bile Acids; Farnesoid X Receptor; Forkhead Box O3a; Mitochondrial Spheroid

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APA (6th Edition):

Manley, S. (2014). ROLE OF BILE ACIDS AND FARNESOID X RECEPTOR IN HEPATIC AUTOPHAGY AND ITS IMPLICATIONS IN ETHANOL-INDUCED HEPATOTOXICITY. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/18425

Chicago Manual of Style (16th Edition):

Manley, Sharon. “ROLE OF BILE ACIDS AND FARNESOID X RECEPTOR IN HEPATIC AUTOPHAGY AND ITS IMPLICATIONS IN ETHANOL-INDUCED HEPATOTOXICITY.” 2014. Doctoral Dissertation, University of Kansas. Accessed July 23, 2019. http://hdl.handle.net/1808/18425.

MLA Handbook (7th Edition):

Manley, Sharon. “ROLE OF BILE ACIDS AND FARNESOID X RECEPTOR IN HEPATIC AUTOPHAGY AND ITS IMPLICATIONS IN ETHANOL-INDUCED HEPATOTOXICITY.” 2014. Web. 23 Jul 2019.

Vancouver:

Manley S. ROLE OF BILE ACIDS AND FARNESOID X RECEPTOR IN HEPATIC AUTOPHAGY AND ITS IMPLICATIONS IN ETHANOL-INDUCED HEPATOTOXICITY. [Internet] [Doctoral dissertation]. University of Kansas; 2014. [cited 2019 Jul 23]. Available from: http://hdl.handle.net/1808/18425.

Council of Science Editors:

Manley S. ROLE OF BILE ACIDS AND FARNESOID X RECEPTOR IN HEPATIC AUTOPHAGY AND ITS IMPLICATIONS IN ETHANOL-INDUCED HEPATOTOXICITY. [Doctoral Dissertation]. University of Kansas; 2014. Available from: http://hdl.handle.net/1808/18425

16. McGill, Mitchell Ryan. Acetaminophen Hepatotoxicity in Humans and Mice.

Degree: PhD, Pharmacology, Toxicology & Therapeutics, 2013, University of Kansas

 Acetaminophen (APAP) is a popular analgesic and antipyretic. Most of a therapeutic dose is glucuronidated or sulfated and excreted. A small amount is converted by… (more)

Subjects/Keywords: Toxicology; Physiology; Pathology; Acetaminophen; Human; Liver; Mitochondria; Translational research

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APA (6th Edition):

McGill, M. R. (2013). Acetaminophen Hepatotoxicity in Humans and Mice. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/12178

Chicago Manual of Style (16th Edition):

McGill, Mitchell Ryan. “Acetaminophen Hepatotoxicity in Humans and Mice.” 2013. Doctoral Dissertation, University of Kansas. Accessed July 23, 2019. http://hdl.handle.net/1808/12178.

MLA Handbook (7th Edition):

McGill, Mitchell Ryan. “Acetaminophen Hepatotoxicity in Humans and Mice.” 2013. Web. 23 Jul 2019.

Vancouver:

McGill MR. Acetaminophen Hepatotoxicity in Humans and Mice. [Internet] [Doctoral dissertation]. University of Kansas; 2013. [cited 2019 Jul 23]. Available from: http://hdl.handle.net/1808/12178.

Council of Science Editors:

McGill MR. Acetaminophen Hepatotoxicity in Humans and Mice. [Doctoral Dissertation]. University of Kansas; 2013. Available from: http://hdl.handle.net/1808/12178

17. Zhan, Le. STUDY OF FXR IN PRIMARY HUMAN HEPATOCYTES AND FXR REGULATED BA HOMEOSTASIS IN PARENTERAL NUTRITION ASSOCIATED LIVER DISEASES.

Degree: PhD, Pharmacology, Toxicology & Therapeutics, 2014, University of Kansas

 Farnesoid X receptor (FXR, NR1H4) is a ligand activated transcription factor belonging to the nuclear receptor (NR) superfamily, and is highly expressed in the liver,… (more)

Subjects/Keywords: Pharmacology; Toxicology; BA; ChIP-seq; FXR; humans versus mice; liver; PNALD

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APA (6th Edition):

Zhan, L. (2014). STUDY OF FXR IN PRIMARY HUMAN HEPATOCYTES AND FXR REGULATED BA HOMEOSTASIS IN PARENTERAL NUTRITION ASSOCIATED LIVER DISEASES. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/19570

Chicago Manual of Style (16th Edition):

Zhan, Le. “STUDY OF FXR IN PRIMARY HUMAN HEPATOCYTES AND FXR REGULATED BA HOMEOSTASIS IN PARENTERAL NUTRITION ASSOCIATED LIVER DISEASES.” 2014. Doctoral Dissertation, University of Kansas. Accessed July 23, 2019. http://hdl.handle.net/1808/19570.

MLA Handbook (7th Edition):

Zhan, Le. “STUDY OF FXR IN PRIMARY HUMAN HEPATOCYTES AND FXR REGULATED BA HOMEOSTASIS IN PARENTERAL NUTRITION ASSOCIATED LIVER DISEASES.” 2014. Web. 23 Jul 2019.

Vancouver:

Zhan L. STUDY OF FXR IN PRIMARY HUMAN HEPATOCYTES AND FXR REGULATED BA HOMEOSTASIS IN PARENTERAL NUTRITION ASSOCIATED LIVER DISEASES. [Internet] [Doctoral dissertation]. University of Kansas; 2014. [cited 2019 Jul 23]. Available from: http://hdl.handle.net/1808/19570.

Council of Science Editors:

Zhan L. STUDY OF FXR IN PRIMARY HUMAN HEPATOCYTES AND FXR REGULATED BA HOMEOSTASIS IN PARENTERAL NUTRITION ASSOCIATED LIVER DISEASES. [Doctoral Dissertation]. University of Kansas; 2014. Available from: http://hdl.handle.net/1808/19570

18. Cheng, Erdong. Mechanism of cap-snatching in hantaviruses.

Degree: PhD, Microbiology, Molecular Genetics & Immunology, 2014, University of Kansas

 Hantaviruses are enveloped RNA viruses belong to the Family Bunyaviridae, which cause hemorrhagic fever with renal syndrome (HFRS) and hantavirus cardiopulmonary syndrome (HCPS) in humans.… (more)

Subjects/Keywords: Microbiology; Virology; Cap-snatching; Hantavirus; P-body; Transcription

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Cheng, E. (2014). Mechanism of cap-snatching in hantaviruses. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/18420

Chicago Manual of Style (16th Edition):

Cheng, Erdong. “Mechanism of cap-snatching in hantaviruses.” 2014. Doctoral Dissertation, University of Kansas. Accessed July 23, 2019. http://hdl.handle.net/1808/18420.

MLA Handbook (7th Edition):

Cheng, Erdong. “Mechanism of cap-snatching in hantaviruses.” 2014. Web. 23 Jul 2019.

Vancouver:

Cheng E. Mechanism of cap-snatching in hantaviruses. [Internet] [Doctoral dissertation]. University of Kansas; 2014. [cited 2019 Jul 23]. Available from: http://hdl.handle.net/1808/18420.

Council of Science Editors:

Cheng E. Mechanism of cap-snatching in hantaviruses. [Doctoral Dissertation]. University of Kansas; 2014. Available from: http://hdl.handle.net/1808/18420

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