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You searched for +publisher:"University of Kansas" +contributor:("Copple, Bryan"). Showing records 1 – 6 of 6 total matches.

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University of Kansas

1. Allen, Katryn Miller. Mechanisms of Proinflammatory Signaling during Cholestasis.

Degree: PhD, Pharmacology, Toxicology & Therapeutics, 2011, University of Kansas

 Cholestasis is a condition in which bile flow from the liver to the intestines is inhibited. Loss of bile flow leads to increased concentrations of… (more)

Subjects/Keywords: Toxicology; Bile acids; Cholestasis; Early growth response factor-1; Inflammation

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Allen, K. M. (2011). Mechanisms of Proinflammatory Signaling during Cholestasis. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/7918

Chicago Manual of Style (16th Edition):

Allen, Katryn Miller. “Mechanisms of Proinflammatory Signaling during Cholestasis.” 2011. Doctoral Dissertation, University of Kansas. Accessed April 25, 2019. http://hdl.handle.net/1808/7918.

MLA Handbook (7th Edition):

Allen, Katryn Miller. “Mechanisms of Proinflammatory Signaling during Cholestasis.” 2011. Web. 25 Apr 2019.

Vancouver:

Allen KM. Mechanisms of Proinflammatory Signaling during Cholestasis. [Internet] [Doctoral dissertation]. University of Kansas; 2011. [cited 2019 Apr 25]. Available from: http://hdl.handle.net/1808/7918.

Council of Science Editors:

Allen KM. Mechanisms of Proinflammatory Signaling during Cholestasis. [Doctoral Dissertation]. University of Kansas; 2011. Available from: http://hdl.handle.net/1808/7918


University of Kansas

2. Saito, Chieko. PROTECTIVE STRATEGIES AGAINST ACETAMINOPHEN INDUCED HEPATOTOXICITY.

Degree: PhD, Pharmacology, Toxicology & Therapeutics, 2010, University of Kansas

 Acetaminophen (APAP) is a widely used analgesic, which is safe at therapeutic levels. APAP is mainly conjugated with glucuronic acid and sulfate to form water-soluble,… (more)

Subjects/Keywords: Health sciences; Toxicology; Acetaminophen; C-jun n-terminal kinase; Glutathione; Metallothionein; N-acetylcysteine

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Saito, C. (2010). PROTECTIVE STRATEGIES AGAINST ACETAMINOPHEN INDUCED HEPATOTOXICITY. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/6369

Chicago Manual of Style (16th Edition):

Saito, Chieko. “PROTECTIVE STRATEGIES AGAINST ACETAMINOPHEN INDUCED HEPATOTOXICITY.” 2010. Doctoral Dissertation, University of Kansas. Accessed April 25, 2019. http://hdl.handle.net/1808/6369.

MLA Handbook (7th Edition):

Saito, Chieko. “PROTECTIVE STRATEGIES AGAINST ACETAMINOPHEN INDUCED HEPATOTOXICITY.” 2010. Web. 25 Apr 2019.

Vancouver:

Saito C. PROTECTIVE STRATEGIES AGAINST ACETAMINOPHEN INDUCED HEPATOTOXICITY. [Internet] [Doctoral dissertation]. University of Kansas; 2010. [cited 2019 Apr 25]. Available from: http://hdl.handle.net/1808/6369.

Council of Science Editors:

Saito C. PROTECTIVE STRATEGIES AGAINST ACETAMINOPHEN INDUCED HEPATOTOXICITY. [Doctoral Dissertation]. University of Kansas; 2010. Available from: http://hdl.handle.net/1808/6369


University of Kansas

3. Song, Peizhen. EFFECT OF BILE ACID FEEDING AND SEQUESTRATION ON LIVER BILE ACID COMPOSITION AND GENE REGULATION IN MICE.

Degree: PhD, Pharmacology, Toxicology & Therapeutics, 2010, University of Kansas

 The dissertation investigates the non-hepatotoxic doses of five bile acids (BAs) in the feed of mice, as well as adaptations in the expression of genes… (more)

Subjects/Keywords: Toxicology; Pathology; Bile acids; Cholestyramine resin; Cyp7a1; Cyp8b1; Fgf15; Fxr

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Song, P. (2010). EFFECT OF BILE ACID FEEDING AND SEQUESTRATION ON LIVER BILE ACID COMPOSITION AND GENE REGULATION IN MICE. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/7756

Chicago Manual of Style (16th Edition):

Song, Peizhen. “EFFECT OF BILE ACID FEEDING AND SEQUESTRATION ON LIVER BILE ACID COMPOSITION AND GENE REGULATION IN MICE.” 2010. Doctoral Dissertation, University of Kansas. Accessed April 25, 2019. http://hdl.handle.net/1808/7756.

MLA Handbook (7th Edition):

Song, Peizhen. “EFFECT OF BILE ACID FEEDING AND SEQUESTRATION ON LIVER BILE ACID COMPOSITION AND GENE REGULATION IN MICE.” 2010. Web. 25 Apr 2019.

Vancouver:

Song P. EFFECT OF BILE ACID FEEDING AND SEQUESTRATION ON LIVER BILE ACID COMPOSITION AND GENE REGULATION IN MICE. [Internet] [Doctoral dissertation]. University of Kansas; 2010. [cited 2019 Apr 25]. Available from: http://hdl.handle.net/1808/7756.

Council of Science Editors:

Song P. EFFECT OF BILE ACID FEEDING AND SEQUESTRATION ON LIVER BILE ACID COMPOSITION AND GENE REGULATION IN MICE. [Doctoral Dissertation]. University of Kansas; 2010. Available from: http://hdl.handle.net/1808/7756


University of Kansas

4. Chao, Jie. MECHANISMS OF MICROVASCULAR INFLAMMATION INDUCED BY ALVEOLAR HYPOXIA.

Degree: PhD, Molecular & Integrative Physiology, 2010, University of Kansas

 Alveolar hypoxia is observed in a number of clinical settings, and is frequently associated with systemic effects, many of which present an inflammatory component. Reduction… (more)

Subjects/Keywords: Biology; Physiology; Alveolar macrophage; Hypoxia; Inflammation; Mast cell; Mcp-1; Microcirculation

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Chao, J. (2010). MECHANISMS OF MICROVASCULAR INFLAMMATION INDUCED BY ALVEOLAR HYPOXIA. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/7425

Chicago Manual of Style (16th Edition):

Chao, Jie. “MECHANISMS OF MICROVASCULAR INFLAMMATION INDUCED BY ALVEOLAR HYPOXIA.” 2010. Doctoral Dissertation, University of Kansas. Accessed April 25, 2019. http://hdl.handle.net/1808/7425.

MLA Handbook (7th Edition):

Chao, Jie. “MECHANISMS OF MICROVASCULAR INFLAMMATION INDUCED BY ALVEOLAR HYPOXIA.” 2010. Web. 25 Apr 2019.

Vancouver:

Chao J. MECHANISMS OF MICROVASCULAR INFLAMMATION INDUCED BY ALVEOLAR HYPOXIA. [Internet] [Doctoral dissertation]. University of Kansas; 2010. [cited 2019 Apr 25]. Available from: http://hdl.handle.net/1808/7425.

Council of Science Editors:

Chao J. MECHANISMS OF MICROVASCULAR INFLAMMATION INDUCED BY ALVEOLAR HYPOXIA. [Doctoral Dissertation]. University of Kansas; 2010. Available from: http://hdl.handle.net/1808/7425


University of Kansas

5. Rook, Jerri Michelle. Mechanisms of Cutaneous Wound Healing are Mediated via Peripheral Neuropeptide Activity.

Degree: PH.D., Pharmacology, Toxicology & Therapeutics, 2008, University of Kansas

 Topically applied morphine is routinely used to alleviate pain in cutaneous wounds such as burns and pressure sores, yet evidence suggests the topical administration of… (more)

Subjects/Keywords: Health sciences; Pharmacology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Rook, J. M. (2008). Mechanisms of Cutaneous Wound Healing are Mediated via Peripheral Neuropeptide Activity. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/4232

Chicago Manual of Style (16th Edition):

Rook, Jerri Michelle. “Mechanisms of Cutaneous Wound Healing are Mediated via Peripheral Neuropeptide Activity.” 2008. Doctoral Dissertation, University of Kansas. Accessed April 25, 2019. http://hdl.handle.net/1808/4232.

MLA Handbook (7th Edition):

Rook, Jerri Michelle. “Mechanisms of Cutaneous Wound Healing are Mediated via Peripheral Neuropeptide Activity.” 2008. Web. 25 Apr 2019.

Vancouver:

Rook JM. Mechanisms of Cutaneous Wound Healing are Mediated via Peripheral Neuropeptide Activity. [Internet] [Doctoral dissertation]. University of Kansas; 2008. [cited 2019 Apr 25]. Available from: http://hdl.handle.net/1808/4232.

Council of Science Editors:

Rook JM. Mechanisms of Cutaneous Wound Healing are Mediated via Peripheral Neuropeptide Activity. [Doctoral Dissertation]. University of Kansas; 2008. Available from: http://hdl.handle.net/1808/4232


University of Kansas

6. Reisman, Scott Aaron. PHARMACOLOGIC AND TRANSGENIC ACTIVATION OF NUCLEAR FACTOR-ERYTHROID 2-RELATED FACTOR 2 (NRF2) ALTERS KINETICS AND TOXICODYNAMICS OF XENOBIOTICS.

Degree: PhD, Pharmacology, Toxicology & Therapeutics, 2008, University of Kansas

 Nuclear factor erythroid 2-related factor 2 (Nrf2) is a transcription factor, which, upon translocation into the nucleus, is capable of inducing a variety of cytoprotective… (more)

Subjects/Keywords: Health sciences; Toxicology; Pharmacology; Acetaminophen; Electrophilic stress; Glutathione-s-transferases; Nad(p)h:quinone oxidoreductase; Nrf2; Oxidative stress

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Reisman, S. A. (2008). PHARMACOLOGIC AND TRANSGENIC ACTIVATION OF NUCLEAR FACTOR-ERYTHROID 2-RELATED FACTOR 2 (NRF2) ALTERS KINETICS AND TOXICODYNAMICS OF XENOBIOTICS. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/4325

Chicago Manual of Style (16th Edition):

Reisman, Scott Aaron. “PHARMACOLOGIC AND TRANSGENIC ACTIVATION OF NUCLEAR FACTOR-ERYTHROID 2-RELATED FACTOR 2 (NRF2) ALTERS KINETICS AND TOXICODYNAMICS OF XENOBIOTICS.” 2008. Doctoral Dissertation, University of Kansas. Accessed April 25, 2019. http://hdl.handle.net/1808/4325.

MLA Handbook (7th Edition):

Reisman, Scott Aaron. “PHARMACOLOGIC AND TRANSGENIC ACTIVATION OF NUCLEAR FACTOR-ERYTHROID 2-RELATED FACTOR 2 (NRF2) ALTERS KINETICS AND TOXICODYNAMICS OF XENOBIOTICS.” 2008. Web. 25 Apr 2019.

Vancouver:

Reisman SA. PHARMACOLOGIC AND TRANSGENIC ACTIVATION OF NUCLEAR FACTOR-ERYTHROID 2-RELATED FACTOR 2 (NRF2) ALTERS KINETICS AND TOXICODYNAMICS OF XENOBIOTICS. [Internet] [Doctoral dissertation]. University of Kansas; 2008. [cited 2019 Apr 25]. Available from: http://hdl.handle.net/1808/4325.

Council of Science Editors:

Reisman SA. PHARMACOLOGIC AND TRANSGENIC ACTIVATION OF NUCLEAR FACTOR-ERYTHROID 2-RELATED FACTOR 2 (NRF2) ALTERS KINETICS AND TOXICODYNAMICS OF XENOBIOTICS. [Doctoral Dissertation]. University of Kansas; 2008. Available from: http://hdl.handle.net/1808/4325

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