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You searched for +publisher:"University of Kansas" +contributor:("Apte, Udayan"). Showing records 1 – 20 of 20 total matches.

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University of Kansas

1. Poudel, Samikshya. Role of Yes-Associated Protein (YAP) in Liver Injury and Regeneration following Acetaminophen Overdose.

Degree: MS, Pharmacology, Toxicology & Therapeutics, 2016, University of Kansas

 Acetaminophen (APAP) overdose is the major cause of Acute Liver Failure (ALF) in the Western world. Treatment options for APAP-induced ALF are limited. Studies have… (more)

Subjects/Keywords: Toxicology

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APA (6th Edition):

Poudel, S. (2016). Role of Yes-Associated Protein (YAP) in Liver Injury and Regeneration following Acetaminophen Overdose. (Masters Thesis). University of Kansas. Retrieved from http://hdl.handle.net/1808/24805

Chicago Manual of Style (16th Edition):

Poudel, Samikshya. “Role of Yes-Associated Protein (YAP) in Liver Injury and Regeneration following Acetaminophen Overdose.” 2016. Masters Thesis, University of Kansas. Accessed June 24, 2019. http://hdl.handle.net/1808/24805.

MLA Handbook (7th Edition):

Poudel, Samikshya. “Role of Yes-Associated Protein (YAP) in Liver Injury and Regeneration following Acetaminophen Overdose.” 2016. Web. 24 Jun 2019.

Vancouver:

Poudel S. Role of Yes-Associated Protein (YAP) in Liver Injury and Regeneration following Acetaminophen Overdose. [Internet] [Masters thesis]. University of Kansas; 2016. [cited 2019 Jun 24]. Available from: http://hdl.handle.net/1808/24805.

Council of Science Editors:

Poudel S. Role of Yes-Associated Protein (YAP) in Liver Injury and Regeneration following Acetaminophen Overdose. [Masters Thesis]. University of Kansas; 2016. Available from: http://hdl.handle.net/1808/24805


University of Kansas

2. McGreal, Steven. The Role of O-GlcNAc in Liver Injury and Regeneration.

Degree: PhD, Pharmacology, Toxicology & Therapeutics, 2017, University of Kansas

 O-GlcNAcylation is a covalent attachment of a single N-acetyl glucosamine to a serine or threonine residue of a protein. Unlike other forms of protein glycosylation,… (more)

Subjects/Keywords: Toxicology; Acetaminophen; Liver; O-GlcNAc; regeneration

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APA (6th Edition):

McGreal, S. (2017). The Role of O-GlcNAc in Liver Injury and Regeneration. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/27029

Chicago Manual of Style (16th Edition):

McGreal, Steven. “The Role of O-GlcNAc in Liver Injury and Regeneration.” 2017. Doctoral Dissertation, University of Kansas. Accessed June 24, 2019. http://hdl.handle.net/1808/27029.

MLA Handbook (7th Edition):

McGreal, Steven. “The Role of O-GlcNAc in Liver Injury and Regeneration.” 2017. Web. 24 Jun 2019.

Vancouver:

McGreal S. The Role of O-GlcNAc in Liver Injury and Regeneration. [Internet] [Doctoral dissertation]. University of Kansas; 2017. [cited 2019 Jun 24]. Available from: http://hdl.handle.net/1808/27029.

Council of Science Editors:

McGreal S. The Role of O-GlcNAc in Liver Injury and Regeneration. [Doctoral Dissertation]. University of Kansas; 2017. Available from: http://hdl.handle.net/1808/27029


University of Kansas

3. Woolbright, Ben. The role of bile acids during cholestasis in mice and man.

Degree: PhD, Pharmacology, Toxicology & Therapeutics, 2015, University of Kansas

 Cholestasis is a reduction in bile flow that occurs during numerous pathologies. Cholestasis leads to significant liver toxicity, biliary hyperplasia, and liver cirrhosis. The molecular… (more)

Subjects/Keywords: Toxicology; Pathology; Pharmacology; apoptosis; bile acid; cholestasis; hepatocytes; inflammation; neutrophil

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APA (6th Edition):

Woolbright, B. (2015). The role of bile acids during cholestasis in mice and man. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/19455

Chicago Manual of Style (16th Edition):

Woolbright, Ben. “The role of bile acids during cholestasis in mice and man.” 2015. Doctoral Dissertation, University of Kansas. Accessed June 24, 2019. http://hdl.handle.net/1808/19455.

MLA Handbook (7th Edition):

Woolbright, Ben. “The role of bile acids during cholestasis in mice and man.” 2015. Web. 24 Jun 2019.

Vancouver:

Woolbright B. The role of bile acids during cholestasis in mice and man. [Internet] [Doctoral dissertation]. University of Kansas; 2015. [cited 2019 Jun 24]. Available from: http://hdl.handle.net/1808/19455.

Council of Science Editors:

Woolbright B. The role of bile acids during cholestasis in mice and man. [Doctoral Dissertation]. University of Kansas; 2015. Available from: http://hdl.handle.net/1808/19455


University of Kansas

4. Zhao, Wen. Identification and characterization of the transporters involved in the disposition of perfluoroalkyl substances.

Degree: PhD, Pharmacology, Toxicology & Therapeutics, 2016, University of Kansas

 Perfluoroalkyl substances (PFASs), including perfluoroalkyl carboxylates (PFCAs) and perfluoroalkyl sulfonates (PFSAs), are persistent amphiphilic chemicals many of which are distributed ubiquitously in the environment and… (more)

Subjects/Keywords: Pharmacology; Toxicology; Drug transporters; Enterohepatic circulation; Perfluoroalkyl substances; PFBS; PFHxS; PFOS

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APA (6th Edition):

Zhao, W. (2016). Identification and characterization of the transporters involved in the disposition of perfluoroalkyl substances. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/24802

Chicago Manual of Style (16th Edition):

Zhao, Wen. “Identification and characterization of the transporters involved in the disposition of perfluoroalkyl substances.” 2016. Doctoral Dissertation, University of Kansas. Accessed June 24, 2019. http://hdl.handle.net/1808/24802.

MLA Handbook (7th Edition):

Zhao, Wen. “Identification and characterization of the transporters involved in the disposition of perfluoroalkyl substances.” 2016. Web. 24 Jun 2019.

Vancouver:

Zhao W. Identification and characterization of the transporters involved in the disposition of perfluoroalkyl substances. [Internet] [Doctoral dissertation]. University of Kansas; 2016. [cited 2019 Jun 24]. Available from: http://hdl.handle.net/1808/24802.

Council of Science Editors:

Zhao W. Identification and characterization of the transporters involved in the disposition of perfluoroalkyl substances. [Doctoral Dissertation]. University of Kansas; 2016. Available from: http://hdl.handle.net/1808/24802


University of Kansas

5. Du, Kuo. MITOCHONDRIAL OXIDATIVE STRESS AND BIOGENESIS DURING ACETAMINOPHEN HEPATOTOXICITY.

Degree: PhD, Pharmacology, Toxicology & Therapeutics, 2017, University of Kansas

 Acetaminophen (APAP) overdose causes severe hepatotoxicity in animals and humans. Although numerous studies have established the existence of an extensive oxidative stress during APAP hepatotoxicity,… (more)

Subjects/Keywords: Toxicology; Pharmacology; Molecular biology; Acetaminophen; liver toxicity; metformin; mitochondria; mito-tempo; oxidative stress

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APA (6th Edition):

Du, K. (2017). MITOCHONDRIAL OXIDATIVE STRESS AND BIOGENESIS DURING ACETAMINOPHEN HEPATOTOXICITY. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/26928

Chicago Manual of Style (16th Edition):

Du, Kuo. “MITOCHONDRIAL OXIDATIVE STRESS AND BIOGENESIS DURING ACETAMINOPHEN HEPATOTOXICITY.” 2017. Doctoral Dissertation, University of Kansas. Accessed June 24, 2019. http://hdl.handle.net/1808/26928.

MLA Handbook (7th Edition):

Du, Kuo. “MITOCHONDRIAL OXIDATIVE STRESS AND BIOGENESIS DURING ACETAMINOPHEN HEPATOTOXICITY.” 2017. Web. 24 Jun 2019.

Vancouver:

Du K. MITOCHONDRIAL OXIDATIVE STRESS AND BIOGENESIS DURING ACETAMINOPHEN HEPATOTOXICITY. [Internet] [Doctoral dissertation]. University of Kansas; 2017. [cited 2019 Jun 24]. Available from: http://hdl.handle.net/1808/26928.

Council of Science Editors:

Du K. MITOCHONDRIAL OXIDATIVE STRESS AND BIOGENESIS DURING ACETAMINOPHEN HEPATOTOXICITY. [Doctoral Dissertation]. University of Kansas; 2017. Available from: http://hdl.handle.net/1808/26928


University of Kansas

6. Weemhoff, James Lawrence. Mechanistic Biomarkers in Acute Liver Injury.

Degree: PhD, Pharmacology, Toxicology & Therapeutics, 2017, University of Kansas

 Acute liver failure continues to be a major medical problem. There are many underlying causes of acute liver failure, but drug induced liver injury is… (more)

Subjects/Keywords: Toxicology; Acetaminophen; Biomarkers; HepaRG; Hypoxic Hepatitis; Liver Injury; Liver Transplantation

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APA (6th Edition):

Weemhoff, J. L. (2017). Mechanistic Biomarkers in Acute Liver Injury. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/27044

Chicago Manual of Style (16th Edition):

Weemhoff, James Lawrence. “Mechanistic Biomarkers in Acute Liver Injury.” 2017. Doctoral Dissertation, University of Kansas. Accessed June 24, 2019. http://hdl.handle.net/1808/27044.

MLA Handbook (7th Edition):

Weemhoff, James Lawrence. “Mechanistic Biomarkers in Acute Liver Injury.” 2017. Web. 24 Jun 2019.

Vancouver:

Weemhoff JL. Mechanistic Biomarkers in Acute Liver Injury. [Internet] [Doctoral dissertation]. University of Kansas; 2017. [cited 2019 Jun 24]. Available from: http://hdl.handle.net/1808/27044.

Council of Science Editors:

Weemhoff JL. Mechanistic Biomarkers in Acute Liver Injury. [Doctoral Dissertation]. University of Kansas; 2017. Available from: http://hdl.handle.net/1808/27044


University of Kansas

7. Chavan, Hemantkumar Dilip. XENOBIOTIC REGULATION OF THE ATP BINDING CASSETTE TRANSPORTER ABCB6 AND ITS SIGNIFICANCE TO HEPATIC HEME HOMEOSTASIS.

Degree: PhD, Pharmacology, Toxicology & Therapeutics, 2013, University of Kansas

 Heme is indispensable for mammalian life. It is an essential component of numerous heme proteins, with functions including oxygen transport and storage, energy metabolism, drug… (more)

Subjects/Keywords: Toxicology; Pharmaceutical sciences; Pharmacology; Abc transporter; Cytochrome p450; Heme; Liver; Mitochondria; Nuclear receptors

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APA (6th Edition):

Chavan, H. D. (2013). XENOBIOTIC REGULATION OF THE ATP BINDING CASSETTE TRANSPORTER ABCB6 AND ITS SIGNIFICANCE TO HEPATIC HEME HOMEOSTASIS. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/14190

Chicago Manual of Style (16th Edition):

Chavan, Hemantkumar Dilip. “XENOBIOTIC REGULATION OF THE ATP BINDING CASSETTE TRANSPORTER ABCB6 AND ITS SIGNIFICANCE TO HEPATIC HEME HOMEOSTASIS.” 2013. Doctoral Dissertation, University of Kansas. Accessed June 24, 2019. http://hdl.handle.net/1808/14190.

MLA Handbook (7th Edition):

Chavan, Hemantkumar Dilip. “XENOBIOTIC REGULATION OF THE ATP BINDING CASSETTE TRANSPORTER ABCB6 AND ITS SIGNIFICANCE TO HEPATIC HEME HOMEOSTASIS.” 2013. Web. 24 Jun 2019.

Vancouver:

Chavan HD. XENOBIOTIC REGULATION OF THE ATP BINDING CASSETTE TRANSPORTER ABCB6 AND ITS SIGNIFICANCE TO HEPATIC HEME HOMEOSTASIS. [Internet] [Doctoral dissertation]. University of Kansas; 2013. [cited 2019 Jun 24]. Available from: http://hdl.handle.net/1808/14190.

Council of Science Editors:

Chavan HD. XENOBIOTIC REGULATION OF THE ATP BINDING CASSETTE TRANSPORTER ABCB6 AND ITS SIGNIFICANCE TO HEPATIC HEME HOMEOSTASIS. [Doctoral Dissertation]. University of Kansas; 2013. Available from: http://hdl.handle.net/1808/14190

8. Williams, Jessica A. R. The Role of Parkin and Mitophagy in Acetaminophen and Alcohol-induced Liver Injuries.

Degree: PhD, Pharmacology, Toxicology & Therapeutics, 2015, University of Kansas

 Acetaminophen (APAP) is the leading cause of acute liver failure in the United States, and alcoholic liver disease (ALD) is a worldwide health problem that… (more)

Subjects/Keywords: Toxicology; Acetaminophen; Alcohol; Autophagy; Liver Injury; Mitophagy; Parkin

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APA (6th Edition):

Williams, J. A. R. (2015). The Role of Parkin and Mitophagy in Acetaminophen and Alcohol-induced Liver Injuries. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/19470

Chicago Manual of Style (16th Edition):

Williams, Jessica A R. “The Role of Parkin and Mitophagy in Acetaminophen and Alcohol-induced Liver Injuries.” 2015. Doctoral Dissertation, University of Kansas. Accessed June 24, 2019. http://hdl.handle.net/1808/19470.

MLA Handbook (7th Edition):

Williams, Jessica A R. “The Role of Parkin and Mitophagy in Acetaminophen and Alcohol-induced Liver Injuries.” 2015. Web. 24 Jun 2019.

Vancouver:

Williams JAR. The Role of Parkin and Mitophagy in Acetaminophen and Alcohol-induced Liver Injuries. [Internet] [Doctoral dissertation]. University of Kansas; 2015. [cited 2019 Jun 24]. Available from: http://hdl.handle.net/1808/19470.

Council of Science Editors:

Williams JAR. The Role of Parkin and Mitophagy in Acetaminophen and Alcohol-induced Liver Injuries. [Doctoral Dissertation]. University of Kansas; 2015. Available from: http://hdl.handle.net/1808/19470


University of Kansas

9. Fu, Zidong. Regulation of Xenobiotic and Bile Acid Metabolism by the Anti-aging Intervention Calorie Restriction in Mice.

Degree: PhD, Pharmacology, Toxicology & Therapeutics, 2013, University of Kansas

 Calorie restriction (CR), defined as reduced calorie intake without causing malnutrition, is the best-known intervention to increase life span and slow aging-related diseases in various… (more)

Subjects/Keywords: Toxicology; Pharmacology; Animal sciences; aging; bile acids; calorie restriction; xenobiotic metabolism and detoxification

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APA (6th Edition):

Fu, Z. (2013). Regulation of Xenobiotic and Bile Acid Metabolism by the Anti-aging Intervention Calorie Restriction in Mice. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/19597

Chicago Manual of Style (16th Edition):

Fu, Zidong. “Regulation of Xenobiotic and Bile Acid Metabolism by the Anti-aging Intervention Calorie Restriction in Mice.” 2013. Doctoral Dissertation, University of Kansas. Accessed June 24, 2019. http://hdl.handle.net/1808/19597.

MLA Handbook (7th Edition):

Fu, Zidong. “Regulation of Xenobiotic and Bile Acid Metabolism by the Anti-aging Intervention Calorie Restriction in Mice.” 2013. Web. 24 Jun 2019.

Vancouver:

Fu Z. Regulation of Xenobiotic and Bile Acid Metabolism by the Anti-aging Intervention Calorie Restriction in Mice. [Internet] [Doctoral dissertation]. University of Kansas; 2013. [cited 2019 Jun 24]. Available from: http://hdl.handle.net/1808/19597.

Council of Science Editors:

Fu Z. Regulation of Xenobiotic and Bile Acid Metabolism by the Anti-aging Intervention Calorie Restriction in Mice. [Doctoral Dissertation]. University of Kansas; 2013. Available from: http://hdl.handle.net/1808/19597


University of Kansas

10. Bimali, Milan. A Likelihood Based Approach to the Assessment of Large Sample Convergence and Model Based Clustering.

Degree: PhD, Biostatistics, 2015, University of Kansas

 The likelihood is a function of model parameter(s) and data using a pre-defined probability density function (pdf). Thus, the likelihood can be viewed as model-data… (more)

Subjects/Keywords: Biostatistics; Asymptotic Convergence; Clustering; Fisher Information; Maximum Likelihood Estimator; Relative Likelihood Function; Simulation

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APA (6th Edition):

Bimali, M. (2015). A Likelihood Based Approach to the Assessment of Large Sample Convergence and Model Based Clustering. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/21918

Chicago Manual of Style (16th Edition):

Bimali, Milan. “A Likelihood Based Approach to the Assessment of Large Sample Convergence and Model Based Clustering.” 2015. Doctoral Dissertation, University of Kansas. Accessed June 24, 2019. http://hdl.handle.net/1808/21918.

MLA Handbook (7th Edition):

Bimali, Milan. “A Likelihood Based Approach to the Assessment of Large Sample Convergence and Model Based Clustering.” 2015. Web. 24 Jun 2019.

Vancouver:

Bimali M. A Likelihood Based Approach to the Assessment of Large Sample Convergence and Model Based Clustering. [Internet] [Doctoral dissertation]. University of Kansas; 2015. [cited 2019 Jun 24]. Available from: http://hdl.handle.net/1808/21918.

Council of Science Editors:

Bimali M. A Likelihood Based Approach to the Assessment of Large Sample Convergence and Model Based Clustering. [Doctoral Dissertation]. University of Kansas; 2015. Available from: http://hdl.handle.net/1808/21918


University of Kansas

11. Cox, Josiah. The Role of FOXO3 in Mechanisms of Doxorubicin Resistance in Hepatocellular Carcinoma.

Degree: PhD, Microbiology, Molecular Genetics & Immunology, 2016, University of Kansas

 Trans-arterial chemoembolization (TACE) with doxorubicin is commonly used to treat hepatocellular carcinoma (HCC), but has limited efficacy due to a high level of resistance. The… (more)

Subjects/Keywords: Cellular biology; Medicine

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APA (6th Edition):

Cox, J. (2016). The Role of FOXO3 in Mechanisms of Doxorubicin Resistance in Hepatocellular Carcinoma. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/25878

Chicago Manual of Style (16th Edition):

Cox, Josiah. “The Role of FOXO3 in Mechanisms of Doxorubicin Resistance in Hepatocellular Carcinoma.” 2016. Doctoral Dissertation, University of Kansas. Accessed June 24, 2019. http://hdl.handle.net/1808/25878.

MLA Handbook (7th Edition):

Cox, Josiah. “The Role of FOXO3 in Mechanisms of Doxorubicin Resistance in Hepatocellular Carcinoma.” 2016. Web. 24 Jun 2019.

Vancouver:

Cox J. The Role of FOXO3 in Mechanisms of Doxorubicin Resistance in Hepatocellular Carcinoma. [Internet] [Doctoral dissertation]. University of Kansas; 2016. [cited 2019 Jun 24]. Available from: http://hdl.handle.net/1808/25878.

Council of Science Editors:

Cox J. The Role of FOXO3 in Mechanisms of Doxorubicin Resistance in Hepatocellular Carcinoma. [Doctoral Dissertation]. University of Kansas; 2016. Available from: http://hdl.handle.net/1808/25878


University of Kansas

12. Borude, Prachi C. ROLE OF DNA DAMAGE RESPONSE IN LIVER REGENERATION AFTER APAP OVERDOSE INDUCED ALF.

Degree: PhD, Pharmacology, Toxicology & Therapeutics, 2017, University of Kansas

 Acetaminophen (APAP) overdose is a leading cause of acute liver failure (ALF) with limited treatment options. The mechanisms of APAP-induced liver injury include formation of… (more)

Subjects/Keywords: Toxicology

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APA (6th Edition):

Borude, P. C. (2017). ROLE OF DNA DAMAGE RESPONSE IN LIVER REGENERATION AFTER APAP OVERDOSE INDUCED ALF. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/27046

Chicago Manual of Style (16th Edition):

Borude, Prachi C. “ROLE OF DNA DAMAGE RESPONSE IN LIVER REGENERATION AFTER APAP OVERDOSE INDUCED ALF.” 2017. Doctoral Dissertation, University of Kansas. Accessed June 24, 2019. http://hdl.handle.net/1808/27046.

MLA Handbook (7th Edition):

Borude, Prachi C. “ROLE OF DNA DAMAGE RESPONSE IN LIVER REGENERATION AFTER APAP OVERDOSE INDUCED ALF.” 2017. Web. 24 Jun 2019.

Vancouver:

Borude PC. ROLE OF DNA DAMAGE RESPONSE IN LIVER REGENERATION AFTER APAP OVERDOSE INDUCED ALF. [Internet] [Doctoral dissertation]. University of Kansas; 2017. [cited 2019 Jun 24]. Available from: http://hdl.handle.net/1808/27046.

Council of Science Editors:

Borude PC. ROLE OF DNA DAMAGE RESPONSE IN LIVER REGENERATION AFTER APAP OVERDOSE INDUCED ALF. [Doctoral Dissertation]. University of Kansas; 2017. Available from: http://hdl.handle.net/1808/27046


University of Kansas

13. Bhushan, Bharat. MECHANISMS OF LIVER REGENERATION AFTER ACETAMINOPHEN-INDUCED HEPATOTOXICITY IN MICE.

Degree: PhD, Pharmacology, Toxicology & Therapeutics, 2016, University of Kansas

 Overdose of acetaminophen (APAP) is the major cause of acute liver failure (ALF) in the western world with very limited treatment options. Recent studies suggest… (more)

Subjects/Keywords: Toxicology; Pharmacology; Acetaminophen; EGF receptor; Liver; mice model; Regeneration; Wnt-Beta Catenin-GSK3 Beta pathway

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APA (6th Edition):

Bhushan, B. (2016). MECHANISMS OF LIVER REGENERATION AFTER ACETAMINOPHEN-INDUCED HEPATOTOXICITY IN MICE. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/25366

Chicago Manual of Style (16th Edition):

Bhushan, Bharat. “MECHANISMS OF LIVER REGENERATION AFTER ACETAMINOPHEN-INDUCED HEPATOTOXICITY IN MICE.” 2016. Doctoral Dissertation, University of Kansas. Accessed June 24, 2019. http://hdl.handle.net/1808/25366.

MLA Handbook (7th Edition):

Bhushan, Bharat. “MECHANISMS OF LIVER REGENERATION AFTER ACETAMINOPHEN-INDUCED HEPATOTOXICITY IN MICE.” 2016. Web. 24 Jun 2019.

Vancouver:

Bhushan B. MECHANISMS OF LIVER REGENERATION AFTER ACETAMINOPHEN-INDUCED HEPATOTOXICITY IN MICE. [Internet] [Doctoral dissertation]. University of Kansas; 2016. [cited 2019 Jun 24]. Available from: http://hdl.handle.net/1808/25366.

Council of Science Editors:

Bhushan B. MECHANISMS OF LIVER REGENERATION AFTER ACETAMINOPHEN-INDUCED HEPATOTOXICITY IN MICE. [Doctoral Dissertation]. University of Kansas; 2016. Available from: http://hdl.handle.net/1808/25366


University of Kansas

14. Raman, Archana. THE ROLE OF MATRICELLULAR SIGNALING IN POLYCYSTIC KIDNEY DISEASE.

Degree: PhD, Molecular & Integrative Physiology, 2017, University of Kansas

 Polycystic kidney disease (PKD) is characterized by excessive enlargement of the kidney, due to the hyperplastic growth of renal epithelial cells, giving rise to fluid-filled… (more)

Subjects/Keywords: Physiology; Autosomal dominant Polycystic kidney disease; ECM; Integrin-linked kinase; Integrins; Kidney; Periostin

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APA (6th Edition):

Raman, A. (2017). THE ROLE OF MATRICELLULAR SIGNALING IN POLYCYSTIC KIDNEY DISEASE. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/26892

Chicago Manual of Style (16th Edition):

Raman, Archana. “THE ROLE OF MATRICELLULAR SIGNALING IN POLYCYSTIC KIDNEY DISEASE.” 2017. Doctoral Dissertation, University of Kansas. Accessed June 24, 2019. http://hdl.handle.net/1808/26892.

MLA Handbook (7th Edition):

Raman, Archana. “THE ROLE OF MATRICELLULAR SIGNALING IN POLYCYSTIC KIDNEY DISEASE.” 2017. Web. 24 Jun 2019.

Vancouver:

Raman A. THE ROLE OF MATRICELLULAR SIGNALING IN POLYCYSTIC KIDNEY DISEASE. [Internet] [Doctoral dissertation]. University of Kansas; 2017. [cited 2019 Jun 24]. Available from: http://hdl.handle.net/1808/26892.

Council of Science Editors:

Raman A. THE ROLE OF MATRICELLULAR SIGNALING IN POLYCYSTIC KIDNEY DISEASE. [Doctoral Dissertation]. University of Kansas; 2017. Available from: http://hdl.handle.net/1808/26892

15. Walesky, Chad Michael. Role of Hepatocyte Nuclear Factor 4 alpha in Hepatocyte Proliferation.

Degree: PhD, Pharmacology, Toxicology & Therapeutics, 2014, University of Kansas

 Hepatocyte Nuclear Factor 4 alpha (HNF4α) is the master regulator of hepatocyte differentiation. It is involved in the up-regulation of genes involved in many classic… (more)

Subjects/Keywords: Toxicology; Cancer; Gene expression; Hepatocyte; Hepatocyte nuclear factor 4; Liver; Proliferation

University of Kansas Medical Center under a standard 12-h light/dark cycle with access to chow and… 

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APA (6th Edition):

Walesky, C. M. (2014). Role of Hepatocyte Nuclear Factor 4 alpha in Hepatocyte Proliferation. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/14505

Chicago Manual of Style (16th Edition):

Walesky, Chad Michael. “Role of Hepatocyte Nuclear Factor 4 alpha in Hepatocyte Proliferation.” 2014. Doctoral Dissertation, University of Kansas. Accessed June 24, 2019. http://hdl.handle.net/1808/14505.

MLA Handbook (7th Edition):

Walesky, Chad Michael. “Role of Hepatocyte Nuclear Factor 4 alpha in Hepatocyte Proliferation.” 2014. Web. 24 Jun 2019.

Vancouver:

Walesky CM. Role of Hepatocyte Nuclear Factor 4 alpha in Hepatocyte Proliferation. [Internet] [Doctoral dissertation]. University of Kansas; 2014. [cited 2019 Jun 24]. Available from: http://hdl.handle.net/1808/14505.

Council of Science Editors:

Walesky CM. Role of Hepatocyte Nuclear Factor 4 alpha in Hepatocyte Proliferation. [Doctoral Dissertation]. University of Kansas; 2014. Available from: http://hdl.handle.net/1808/14505

16. Manley, Sharon. ROLE OF BILE ACIDS AND FARNESOID X RECEPTOR IN HEPATIC AUTOPHAGY AND ITS IMPLICATIONS IN ETHANOL-INDUCED HEPATOTOXICITY.

Degree: PhD, Pharmacology, Toxicology & Therapeutics, 2014, University of Kansas

 Retention of bile acids (BAs) in the liver during cholestasis plays an important role in the development of cholestatic liver injury. Several studies have reported… (more)

Subjects/Keywords: Toxicology; Alcoholic Liver Disease; Autophagy; Bile Acids; Farnesoid X Receptor; Forkhead Box O3a; Mitochondrial Spheroid

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APA (6th Edition):

Manley, S. (2014). ROLE OF BILE ACIDS AND FARNESOID X RECEPTOR IN HEPATIC AUTOPHAGY AND ITS IMPLICATIONS IN ETHANOL-INDUCED HEPATOTOXICITY. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/18425

Chicago Manual of Style (16th Edition):

Manley, Sharon. “ROLE OF BILE ACIDS AND FARNESOID X RECEPTOR IN HEPATIC AUTOPHAGY AND ITS IMPLICATIONS IN ETHANOL-INDUCED HEPATOTOXICITY.” 2014. Doctoral Dissertation, University of Kansas. Accessed June 24, 2019. http://hdl.handle.net/1808/18425.

MLA Handbook (7th Edition):

Manley, Sharon. “ROLE OF BILE ACIDS AND FARNESOID X RECEPTOR IN HEPATIC AUTOPHAGY AND ITS IMPLICATIONS IN ETHANOL-INDUCED HEPATOTOXICITY.” 2014. Web. 24 Jun 2019.

Vancouver:

Manley S. ROLE OF BILE ACIDS AND FARNESOID X RECEPTOR IN HEPATIC AUTOPHAGY AND ITS IMPLICATIONS IN ETHANOL-INDUCED HEPATOTOXICITY. [Internet] [Doctoral dissertation]. University of Kansas; 2014. [cited 2019 Jun 24]. Available from: http://hdl.handle.net/1808/18425.

Council of Science Editors:

Manley S. ROLE OF BILE ACIDS AND FARNESOID X RECEPTOR IN HEPATIC AUTOPHAGY AND ITS IMPLICATIONS IN ETHANOL-INDUCED HEPATOTOXICITY. [Doctoral Dissertation]. University of Kansas; 2014. Available from: http://hdl.handle.net/1808/18425

17. Williams, Clarence David. Role of Interleukin-1beta and neutrophil activation during acetaminophen overdose.

Degree: PhD, Pharmacology, Toxicology & Therapeutics, 2012, University of Kansas

 Acetaminophen (APAP) overdose is the leading cause of acute liver failure in the US. APAP is metabolized to a reactive metabolite that causes hepatotoxicity in… (more)

Subjects/Keywords: Toxicology; Acetaminophen; Il-1beta; Neutrophil

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APA (6th Edition):

Williams, C. D. (2012). Role of Interleukin-1beta and neutrophil activation during acetaminophen overdose. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/10290

Chicago Manual of Style (16th Edition):

Williams, Clarence David. “Role of Interleukin-1beta and neutrophil activation during acetaminophen overdose.” 2012. Doctoral Dissertation, University of Kansas. Accessed June 24, 2019. http://hdl.handle.net/1808/10290.

MLA Handbook (7th Edition):

Williams, Clarence David. “Role of Interleukin-1beta and neutrophil activation during acetaminophen overdose.” 2012. Web. 24 Jun 2019.

Vancouver:

Williams CD. Role of Interleukin-1beta and neutrophil activation during acetaminophen overdose. [Internet] [Doctoral dissertation]. University of Kansas; 2012. [cited 2019 Jun 24]. Available from: http://hdl.handle.net/1808/10290.

Council of Science Editors:

Williams CD. Role of Interleukin-1beta and neutrophil activation during acetaminophen overdose. [Doctoral Dissertation]. University of Kansas; 2012. Available from: http://hdl.handle.net/1808/10290

18. Hays, Amanda Lynne. Targeting Organic Anion Transporting Polypeptides in Cancer to Improve Diagnostics and Therapy.

Degree: PhD, Pharmacology, Toxicology & Therapeutics, 2012, University of Kansas

 Organic Anion Transporting Polypeptides (OATPs) are multispecific transport proteins that mediate the uptake of numerous endogenous and exogenous compounds into cells. Recently, OATPs have been… (more)

Subjects/Keywords: Pharmacology; Cancer; Oatp; Transporters

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APA (6th Edition):

Hays, A. L. (2012). Targeting Organic Anion Transporting Polypeptides in Cancer to Improve Diagnostics and Therapy. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/11446

Chicago Manual of Style (16th Edition):

Hays, Amanda Lynne. “Targeting Organic Anion Transporting Polypeptides in Cancer to Improve Diagnostics and Therapy.” 2012. Doctoral Dissertation, University of Kansas. Accessed June 24, 2019. http://hdl.handle.net/1808/11446.

MLA Handbook (7th Edition):

Hays, Amanda Lynne. “Targeting Organic Anion Transporting Polypeptides in Cancer to Improve Diagnostics and Therapy.” 2012. Web. 24 Jun 2019.

Vancouver:

Hays AL. Targeting Organic Anion Transporting Polypeptides in Cancer to Improve Diagnostics and Therapy. [Internet] [Doctoral dissertation]. University of Kansas; 2012. [cited 2019 Jun 24]. Available from: http://hdl.handle.net/1808/11446.

Council of Science Editors:

Hays AL. Targeting Organic Anion Transporting Polypeptides in Cancer to Improve Diagnostics and Therapy. [Doctoral Dissertation]. University of Kansas; 2012. Available from: http://hdl.handle.net/1808/11446

19. Thomas, Ann M. THE ROLE OF EPIGENETICS IN TRANSCRIPTIONAL REGULATION OF FXR AND SILENCING FXR EXPRESSION IN HUMAN COLON CANCER.

Degree: PhD, Pharmacology, Toxicology & Therapeutics, 2011, University of Kansas

 Farnesoid X receptor (FXR) is a ligand activated transcription factor belonging to the nuclear receptor superfamily and bile acids are its endogenous ligands. FXR is… (more)

Subjects/Keywords: Cellular biology; Colon cancer; DNA methylation; Epigenetics; Farnesoid x receptor; Nuclear receptors

…Toxicology & Therapeutics at the University of Kansas Medical Center, particularly members of the… 

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APA (6th Edition):

Thomas, A. M. (2011). THE ROLE OF EPIGENETICS IN TRANSCRIPTIONAL REGULATION OF FXR AND SILENCING FXR EXPRESSION IN HUMAN COLON CANCER. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/8386

Chicago Manual of Style (16th Edition):

Thomas, Ann M. “THE ROLE OF EPIGENETICS IN TRANSCRIPTIONAL REGULATION OF FXR AND SILENCING FXR EXPRESSION IN HUMAN COLON CANCER.” 2011. Doctoral Dissertation, University of Kansas. Accessed June 24, 2019. http://hdl.handle.net/1808/8386.

MLA Handbook (7th Edition):

Thomas, Ann M. “THE ROLE OF EPIGENETICS IN TRANSCRIPTIONAL REGULATION OF FXR AND SILENCING FXR EXPRESSION IN HUMAN COLON CANCER.” 2011. Web. 24 Jun 2019.

Vancouver:

Thomas AM. THE ROLE OF EPIGENETICS IN TRANSCRIPTIONAL REGULATION OF FXR AND SILENCING FXR EXPRESSION IN HUMAN COLON CANCER. [Internet] [Doctoral dissertation]. University of Kansas; 2011. [cited 2019 Jun 24]. Available from: http://hdl.handle.net/1808/8386.

Council of Science Editors:

Thomas AM. THE ROLE OF EPIGENETICS IN TRANSCRIPTIONAL REGULATION OF FXR AND SILENCING FXR EXPRESSION IN HUMAN COLON CANCER. [Doctoral Dissertation]. University of Kansas; 2011. Available from: http://hdl.handle.net/1808/8386

20. Zhan, Le. STUDY OF FXR IN PRIMARY HUMAN HEPATOCYTES AND FXR REGULATED BA HOMEOSTASIS IN PARENTERAL NUTRITION ASSOCIATED LIVER DISEASES.

Degree: PhD, Pharmacology, Toxicology & Therapeutics, 2014, University of Kansas

 Farnesoid X receptor (FXR, NR1H4) is a ligand activated transcription factor belonging to the nuclear receptor (NR) superfamily, and is highly expressed in the liver,… (more)

Subjects/Keywords: Pharmacology; Toxicology; BA; ChIP-seq; FXR; humans versus mice; liver; PNALD

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Zhan, L. (2014). STUDY OF FXR IN PRIMARY HUMAN HEPATOCYTES AND FXR REGULATED BA HOMEOSTASIS IN PARENTERAL NUTRITION ASSOCIATED LIVER DISEASES. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/19570

Chicago Manual of Style (16th Edition):

Zhan, Le. “STUDY OF FXR IN PRIMARY HUMAN HEPATOCYTES AND FXR REGULATED BA HOMEOSTASIS IN PARENTERAL NUTRITION ASSOCIATED LIVER DISEASES.” 2014. Doctoral Dissertation, University of Kansas. Accessed June 24, 2019. http://hdl.handle.net/1808/19570.

MLA Handbook (7th Edition):

Zhan, Le. “STUDY OF FXR IN PRIMARY HUMAN HEPATOCYTES AND FXR REGULATED BA HOMEOSTASIS IN PARENTERAL NUTRITION ASSOCIATED LIVER DISEASES.” 2014. Web. 24 Jun 2019.

Vancouver:

Zhan L. STUDY OF FXR IN PRIMARY HUMAN HEPATOCYTES AND FXR REGULATED BA HOMEOSTASIS IN PARENTERAL NUTRITION ASSOCIATED LIVER DISEASES. [Internet] [Doctoral dissertation]. University of Kansas; 2014. [cited 2019 Jun 24]. Available from: http://hdl.handle.net/1808/19570.

Council of Science Editors:

Zhan L. STUDY OF FXR IN PRIMARY HUMAN HEPATOCYTES AND FXR REGULATED BA HOMEOSTASIS IN PARENTERAL NUTRITION ASSOCIATED LIVER DISEASES. [Doctoral Dissertation]. University of Kansas; 2014. Available from: http://hdl.handle.net/1808/19570

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