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You searched for +publisher:"University of Houston" +contributor:("Webb, Paul"). Showing records 1 – 10 of 10 total matches.

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University of Houston

1. Butler, Ryan 1986-. Physiological and pathological aspects of the ligand-activated transcription factors: AhR and ERβ.

Degree: PhD, Biology, 2013, University of Houston

 Ligand-activated transcription factors are a diverse group of proteins that are involved a variety of physiological processes. The purpose of these studies was to investigate… (more)

Subjects/Keywords: Aryl hydrocarbon receptor; Estrogen receptors; Transcription factors

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APA (6th Edition):

Butler, R. 1. (2013). Physiological and pathological aspects of the ligand-activated transcription factors: AhR and ERβ. (Doctoral Dissertation). University of Houston. Retrieved from http://hdl.handle.net/10657/956

Chicago Manual of Style (16th Edition):

Butler, Ryan 1986-. “Physiological and pathological aspects of the ligand-activated transcription factors: AhR and ERβ.” 2013. Doctoral Dissertation, University of Houston. Accessed January 25, 2021. http://hdl.handle.net/10657/956.

MLA Handbook (7th Edition):

Butler, Ryan 1986-. “Physiological and pathological aspects of the ligand-activated transcription factors: AhR and ERβ.” 2013. Web. 25 Jan 2021.

Vancouver:

Butler R1. Physiological and pathological aspects of the ligand-activated transcription factors: AhR and ERβ. [Internet] [Doctoral dissertation]. University of Houston; 2013. [cited 2021 Jan 25]. Available from: http://hdl.handle.net/10657/956.

Council of Science Editors:

Butler R1. Physiological and pathological aspects of the ligand-activated transcription factors: AhR and ERβ. [Doctoral Dissertation]. University of Houston; 2013. Available from: http://hdl.handle.net/10657/956


University of Houston

2. Jonsson, Philip 1985-. Transcriptional Control of Oncogenic Processes in Breast Cancer Cells by the Estrogen Receptors.

Degree: PhD, Biology, 2014, University of Houston

 The estrogen receptors are fundamental factors in human biology. As transcriptional factors regulating gene programs controlling many processes in the body, they are key in… (more)

Subjects/Keywords: Estrogen receptors; Breast cancer

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APA (6th Edition):

Jonsson, P. 1. (2014). Transcriptional Control of Oncogenic Processes in Breast Cancer Cells by the Estrogen Receptors. (Doctoral Dissertation). University of Houston. Retrieved from http://hdl.handle.net/10657/1949

Chicago Manual of Style (16th Edition):

Jonsson, Philip 1985-. “Transcriptional Control of Oncogenic Processes in Breast Cancer Cells by the Estrogen Receptors.” 2014. Doctoral Dissertation, University of Houston. Accessed January 25, 2021. http://hdl.handle.net/10657/1949.

MLA Handbook (7th Edition):

Jonsson, Philip 1985-. “Transcriptional Control of Oncogenic Processes in Breast Cancer Cells by the Estrogen Receptors.” 2014. Web. 25 Jan 2021.

Vancouver:

Jonsson P1. Transcriptional Control of Oncogenic Processes in Breast Cancer Cells by the Estrogen Receptors. [Internet] [Doctoral dissertation]. University of Houston; 2014. [cited 2021 Jan 25]. Available from: http://hdl.handle.net/10657/1949.

Council of Science Editors:

Jonsson P1. Transcriptional Control of Oncogenic Processes in Breast Cancer Cells by the Estrogen Receptors. [Doctoral Dissertation]. University of Houston; 2014. Available from: http://hdl.handle.net/10657/1949


University of Houston

3. Rajapaksa, R. P. Gayani Kumudu 1981-. Understanding the Tumor Repressive Function of Estrogen Receptor Beta in Breast Cancer.

Degree: PhD, Biology, 2014, University of Houston

 Estrogen receptors play a key role in breast cancer, and understanding the mechanisms of action is important in clinical management of the disease. Here we… (more)

Subjects/Keywords: Breast cancer; ERβ; Unfolded protein response; Epithelial-to-mesenchymal transition; Tumor repressor

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APA (6th Edition):

Rajapaksa, R. P. G. K. 1. (2014). Understanding the Tumor Repressive Function of Estrogen Receptor Beta in Breast Cancer. (Doctoral Dissertation). University of Houston. Retrieved from http://hdl.handle.net/10657/1919

Chicago Manual of Style (16th Edition):

Rajapaksa, R P Gayani Kumudu 1981-. “Understanding the Tumor Repressive Function of Estrogen Receptor Beta in Breast Cancer.” 2014. Doctoral Dissertation, University of Houston. Accessed January 25, 2021. http://hdl.handle.net/10657/1919.

MLA Handbook (7th Edition):

Rajapaksa, R P Gayani Kumudu 1981-. “Understanding the Tumor Repressive Function of Estrogen Receptor Beta in Breast Cancer.” 2014. Web. 25 Jan 2021.

Vancouver:

Rajapaksa RPGK1. Understanding the Tumor Repressive Function of Estrogen Receptor Beta in Breast Cancer. [Internet] [Doctoral dissertation]. University of Houston; 2014. [cited 2021 Jan 25]. Available from: http://hdl.handle.net/10657/1919.

Council of Science Editors:

Rajapaksa RPGK1. Understanding the Tumor Repressive Function of Estrogen Receptor Beta in Breast Cancer. [Doctoral Dissertation]. University of Houston; 2014. Available from: http://hdl.handle.net/10657/1919


University of Houston

4. Lin, Jean Z. 1984-. Thyroid Hormone Receptor Regulation of Cholesterol and Lipid Metabolism.

Degree: PhD, Biology, 2014, University of Houston

 Thyroid hormone receptor (TR) activation affects numerous metabolic processes involved in lipid and glucose metabolism, with implications for diseases such as obesity, diabetes, and hyperlipidemia.… (more)

Subjects/Keywords: Thyroid hormone; Thyroid hormone receptor; Cholesterol; Thermogenesis

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APA (6th Edition):

Lin, J. Z. 1. (2014). Thyroid Hormone Receptor Regulation of Cholesterol and Lipid Metabolism. (Doctoral Dissertation). University of Houston. Retrieved from http://hdl.handle.net/10657/5359

Chicago Manual of Style (16th Edition):

Lin, Jean Z 1984-. “Thyroid Hormone Receptor Regulation of Cholesterol and Lipid Metabolism.” 2014. Doctoral Dissertation, University of Houston. Accessed January 25, 2021. http://hdl.handle.net/10657/5359.

MLA Handbook (7th Edition):

Lin, Jean Z 1984-. “Thyroid Hormone Receptor Regulation of Cholesterol and Lipid Metabolism.” 2014. Web. 25 Jan 2021.

Vancouver:

Lin JZ1. Thyroid Hormone Receptor Regulation of Cholesterol and Lipid Metabolism. [Internet] [Doctoral dissertation]. University of Houston; 2014. [cited 2021 Jan 25]. Available from: http://hdl.handle.net/10657/5359.

Council of Science Editors:

Lin JZ1. Thyroid Hormone Receptor Regulation of Cholesterol and Lipid Metabolism. [Doctoral Dissertation]. University of Houston; 2014. Available from: http://hdl.handle.net/10657/5359


University of Houston

5. Dey, Prasenjit 1978-. Antiproliferative and pro-­apoptotic actions of Estrogen receptor β in prostate cancer.

Degree: PhD, Biochemistry, 2013, University of Houston

 High Gleason grade prostate cancers are aggressive. Currently, the major target for treatment is the androgen receptor. Recent literature points towards a tumor suppressive role… (more)

Subjects/Keywords: Prostate cancer; Estrogen receptor beta; Apoptosis; Proliferation; FOXO3a; PUMA; Bone metastasis; Biochemistry

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APA (6th Edition):

Dey, P. 1. (2013). Antiproliferative and pro-­apoptotic actions of Estrogen receptor β in prostate cancer. (Doctoral Dissertation). University of Houston. Retrieved from http://hdl.handle.net/10657/955

Chicago Manual of Style (16th Edition):

Dey, Prasenjit 1978-. “Antiproliferative and pro-­apoptotic actions of Estrogen receptor β in prostate cancer.” 2013. Doctoral Dissertation, University of Houston. Accessed January 25, 2021. http://hdl.handle.net/10657/955.

MLA Handbook (7th Edition):

Dey, Prasenjit 1978-. “Antiproliferative and pro-­apoptotic actions of Estrogen receptor β in prostate cancer.” 2013. Web. 25 Jan 2021.

Vancouver:

Dey P1. Antiproliferative and pro-­apoptotic actions of Estrogen receptor β in prostate cancer. [Internet] [Doctoral dissertation]. University of Houston; 2013. [cited 2021 Jan 25]. Available from: http://hdl.handle.net/10657/955.

Council of Science Editors:

Dey P1. Antiproliferative and pro-­apoptotic actions of Estrogen receptor β in prostate cancer. [Doctoral Dissertation]. University of Houston; 2013. Available from: http://hdl.handle.net/10657/955


University of Houston

6. Katchy, Anne Chinenye 1984-. Exploring estrogen receptor gene regulatory mechanism in breast cancer.

Degree: PhD, Biochemistry, 2013, University of Houston

 Estrogen has vital roles in development and maintenance of mammary gland and supports the growth of the majority of primary breast cancer. It carries out… (more)

Subjects/Keywords: Estrogen receptors; MicroRNAs (miRNA); Breast cancer; Bisphenol A; Phytoestrogen; Biochemistry

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APA (6th Edition):

Katchy, A. C. 1. (2013). Exploring estrogen receptor gene regulatory mechanism in breast cancer. (Doctoral Dissertation). University of Houston. Retrieved from http://hdl.handle.net/10657/1061

Chicago Manual of Style (16th Edition):

Katchy, Anne Chinenye 1984-. “Exploring estrogen receptor gene regulatory mechanism in breast cancer.” 2013. Doctoral Dissertation, University of Houston. Accessed January 25, 2021. http://hdl.handle.net/10657/1061.

MLA Handbook (7th Edition):

Katchy, Anne Chinenye 1984-. “Exploring estrogen receptor gene regulatory mechanism in breast cancer.” 2013. Web. 25 Jan 2021.

Vancouver:

Katchy AC1. Exploring estrogen receptor gene regulatory mechanism in breast cancer. [Internet] [Doctoral dissertation]. University of Houston; 2013. [cited 2021 Jan 25]. Available from: http://hdl.handle.net/10657/1061.

Council of Science Editors:

Katchy AC1. Exploring estrogen receptor gene regulatory mechanism in breast cancer. [Doctoral Dissertation]. University of Houston; 2013. Available from: http://hdl.handle.net/10657/1061


University of Houston

7. Carruthers, Carl W., Jr. 1971-. Phosphorylation of Glucocorticoid Receptor tau1c Transactivation Domain Enhances Binding to CREB Binding Protein (CBP) TAZ2 and a Microfluidic, High Throughput Protein Crystal Growth Method for Microgravity.

Degree: PhD, Biochemistry, 2014, University of Houston

 The glucocorticoid receptor (GR) N-terminal domain (NTD) contains a transactivation domain (activation function 1; AF-1). Although GR AF-1 is phosphorylated, effects of GR phosphorylation upon… (more)

Subjects/Keywords: Phosphorylation; Glucocorticoids; Glucocorticoid receptors; Tau1c; Transactivation; CBP; Transcriptional adaptor zinc binding (TAZ) 2

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APA (6th Edition):

Carruthers, Carl W., J. 1. (2014). Phosphorylation of Glucocorticoid Receptor tau1c Transactivation Domain Enhances Binding to CREB Binding Protein (CBP) TAZ2 and a Microfluidic, High Throughput Protein Crystal Growth Method for Microgravity. (Doctoral Dissertation). University of Houston. Retrieved from http://hdl.handle.net/10657/1918

Chicago Manual of Style (16th Edition):

Carruthers, Carl W., Jr 1971-. “Phosphorylation of Glucocorticoid Receptor tau1c Transactivation Domain Enhances Binding to CREB Binding Protein (CBP) TAZ2 and a Microfluidic, High Throughput Protein Crystal Growth Method for Microgravity.” 2014. Doctoral Dissertation, University of Houston. Accessed January 25, 2021. http://hdl.handle.net/10657/1918.

MLA Handbook (7th Edition):

Carruthers, Carl W., Jr 1971-. “Phosphorylation of Glucocorticoid Receptor tau1c Transactivation Domain Enhances Binding to CREB Binding Protein (CBP) TAZ2 and a Microfluidic, High Throughput Protein Crystal Growth Method for Microgravity.” 2014. Web. 25 Jan 2021.

Vancouver:

Carruthers, Carl W. J1. Phosphorylation of Glucocorticoid Receptor tau1c Transactivation Domain Enhances Binding to CREB Binding Protein (CBP) TAZ2 and a Microfluidic, High Throughput Protein Crystal Growth Method for Microgravity. [Internet] [Doctoral dissertation]. University of Houston; 2014. [cited 2021 Jan 25]. Available from: http://hdl.handle.net/10657/1918.

Council of Science Editors:

Carruthers, Carl W. J1. Phosphorylation of Glucocorticoid Receptor tau1c Transactivation Domain Enhances Binding to CREB Binding Protein (CBP) TAZ2 and a Microfluidic, High Throughput Protein Crystal Growth Method for Microgravity. [Doctoral Dissertation]. University of Houston; 2014. Available from: http://hdl.handle.net/10657/1918


University of Houston

8. Pinto, Caroline Lucia 1981-. In Vitro Assays and Zebrafish as an in Vivo Model to Study Nuclear Receptor Signaling.

Degree: PhD, Biochemistry, 2014, University of Houston

 Humans are exposed daily to cocktails of chemicals from different sources. Some of these chemicals can interfere with hormonal signaling, and are referred to as… (more)

Subjects/Keywords: Zebrafish; Nuclear receptors; Estrogen receptors; Liver X receptor; Peroxisome proliferator-activated receptor gamma; Endocrine disruptors; Lipid metabolism; Lipids; Yolk; Retina; Lenses; Eye

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APA (6th Edition):

Pinto, C. L. 1. (2014). In Vitro Assays and Zebrafish as an in Vivo Model to Study Nuclear Receptor Signaling. (Doctoral Dissertation). University of Houston. Retrieved from http://hdl.handle.net/10657/4737

Chicago Manual of Style (16th Edition):

Pinto, Caroline Lucia 1981-. “In Vitro Assays and Zebrafish as an in Vivo Model to Study Nuclear Receptor Signaling.” 2014. Doctoral Dissertation, University of Houston. Accessed January 25, 2021. http://hdl.handle.net/10657/4737.

MLA Handbook (7th Edition):

Pinto, Caroline Lucia 1981-. “In Vitro Assays and Zebrafish as an in Vivo Model to Study Nuclear Receptor Signaling.” 2014. Web. 25 Jan 2021.

Vancouver:

Pinto CL1. In Vitro Assays and Zebrafish as an in Vivo Model to Study Nuclear Receptor Signaling. [Internet] [Doctoral dissertation]. University of Houston; 2014. [cited 2021 Jan 25]. Available from: http://hdl.handle.net/10657/4737.

Council of Science Editors:

Pinto CL1. In Vitro Assays and Zebrafish as an in Vivo Model to Study Nuclear Receptor Signaling. [Doctoral Dissertation]. University of Houston; 2014. Available from: http://hdl.handle.net/10657/4737


University of Houston

9. Nikollo, Foti 1986-. Exploring the Tumor Suppressive Roles of Estrogen Receptor β in Lung and Breast Cancer.

Degree: PhD, Biochemistry, University of Houston

 Estrogens represent a subclass of steroid hormones that by regulating cell growth and differentiation, influence normal physiology as well as pathology. The effects of estrogen… (more)

Subjects/Keywords: Estrogen receptor beta; EGFR; NSCLC; RAS; DNA damage response; P53; EMT; Basal-like; E-cadherin

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APA (6th Edition):

Nikollo, F. 1. (n.d.). Exploring the Tumor Suppressive Roles of Estrogen Receptor β in Lung and Breast Cancer. (Doctoral Dissertation). University of Houston. Retrieved from http://hdl.handle.net/10657/2043

Note: this citation may be lacking information needed for this citation format:
No year of publication.

Chicago Manual of Style (16th Edition):

Nikollo, Foti 1986-. “Exploring the Tumor Suppressive Roles of Estrogen Receptor β in Lung and Breast Cancer.” Doctoral Dissertation, University of Houston. Accessed January 25, 2021. http://hdl.handle.net/10657/2043.

Note: this citation may be lacking information needed for this citation format:
No year of publication.

MLA Handbook (7th Edition):

Nikollo, Foti 1986-. “Exploring the Tumor Suppressive Roles of Estrogen Receptor β in Lung and Breast Cancer.” Web. 25 Jan 2021.

Note: this citation may be lacking information needed for this citation format:
No year of publication.

Vancouver:

Nikollo F1. Exploring the Tumor Suppressive Roles of Estrogen Receptor β in Lung and Breast Cancer. [Internet] [Doctoral dissertation]. University of Houston; [cited 2021 Jan 25]. Available from: http://hdl.handle.net/10657/2043.

Note: this citation may be lacking information needed for this citation format:
No year of publication.

Council of Science Editors:

Nikollo F1. Exploring the Tumor Suppressive Roles of Estrogen Receptor β in Lung and Breast Cancer. [Doctoral Dissertation]. University of Houston; Available from: http://hdl.handle.net/10657/2043

Note: this citation may be lacking information needed for this citation format:
No year of publication.


University of Houston

10. Bado, Igor Landry 1986-. Understanding the Tumor Suppressive Functions of Estrogen Receptor β in Breast Cancer.

Degree: PhD, Biology, University of Houston

 Breast cancer is a heterogeneous disease with regard to clinical outcome and molecular characteristics. Some breast cancers are more aggressive because they express mutant proteins… (more)

Subjects/Keywords: Breast cancer; Cancer; Triple negative breast cancer cells (TNBC); ERβ1; P53; P63; EMT; Invasion; Metastasis; Luminal breast cancer; ERα

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APA (6th Edition):

Bado, I. L. 1. (n.d.). Understanding the Tumor Suppressive Functions of Estrogen Receptor β in Breast Cancer. (Doctoral Dissertation). University of Houston. Retrieved from http://hdl.handle.net/10657/3620

Note: this citation may be lacking information needed for this citation format:
No year of publication.

Chicago Manual of Style (16th Edition):

Bado, Igor Landry 1986-. “Understanding the Tumor Suppressive Functions of Estrogen Receptor β in Breast Cancer.” Doctoral Dissertation, University of Houston. Accessed January 25, 2021. http://hdl.handle.net/10657/3620.

Note: this citation may be lacking information needed for this citation format:
No year of publication.

MLA Handbook (7th Edition):

Bado, Igor Landry 1986-. “Understanding the Tumor Suppressive Functions of Estrogen Receptor β in Breast Cancer.” Web. 25 Jan 2021.

Note: this citation may be lacking information needed for this citation format:
No year of publication.

Vancouver:

Bado IL1. Understanding the Tumor Suppressive Functions of Estrogen Receptor β in Breast Cancer. [Internet] [Doctoral dissertation]. University of Houston; [cited 2021 Jan 25]. Available from: http://hdl.handle.net/10657/3620.

Note: this citation may be lacking information needed for this citation format:
No year of publication.

Council of Science Editors:

Bado IL1. Understanding the Tumor Suppressive Functions of Estrogen Receptor β in Breast Cancer. [Doctoral Dissertation]. University of Houston; Available from: http://hdl.handle.net/10657/3620

Note: this citation may be lacking information needed for this citation format:
No year of publication.

.