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You searched for +publisher:"University of Houston" +contributor:("Gunaratne, Preethi H."). Showing records 1 – 16 of 16 total matches.

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University of Houston

1. Jonsson, Philip 1985-. Transcriptional Control of Oncogenic Processes in Breast Cancer Cells by the Estrogen Receptors.

Degree: PhD, Biology, 2014, University of Houston

 The estrogen receptors are fundamental factors in human biology. As transcriptional factors regulating gene programs controlling many processes in the body, they are key in… (more)

Subjects/Keywords: Estrogen receptors; Breast cancer

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APA (6th Edition):

Jonsson, P. 1. (2014). Transcriptional Control of Oncogenic Processes in Breast Cancer Cells by the Estrogen Receptors. (Doctoral Dissertation). University of Houston. Retrieved from http://hdl.handle.net/10657/1949

Chicago Manual of Style (16th Edition):

Jonsson, Philip 1985-. “Transcriptional Control of Oncogenic Processes in Breast Cancer Cells by the Estrogen Receptors.” 2014. Doctoral Dissertation, University of Houston. Accessed January 19, 2021. http://hdl.handle.net/10657/1949.

MLA Handbook (7th Edition):

Jonsson, Philip 1985-. “Transcriptional Control of Oncogenic Processes in Breast Cancer Cells by the Estrogen Receptors.” 2014. Web. 19 Jan 2021.

Vancouver:

Jonsson P1. Transcriptional Control of Oncogenic Processes in Breast Cancer Cells by the Estrogen Receptors. [Internet] [Doctoral dissertation]. University of Houston; 2014. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/10657/1949.

Council of Science Editors:

Jonsson P1. Transcriptional Control of Oncogenic Processes in Breast Cancer Cells by the Estrogen Receptors. [Doctoral Dissertation]. University of Houston; 2014. Available from: http://hdl.handle.net/10657/1949


University of Houston

2. Rajapaksa, R. P. Gayani Kumudu 1981-. Understanding the Tumor Repressive Function of Estrogen Receptor Beta in Breast Cancer.

Degree: PhD, Biology, 2014, University of Houston

 Estrogen receptors play a key role in breast cancer, and understanding the mechanisms of action is important in clinical management of the disease. Here we… (more)

Subjects/Keywords: Breast cancer; ERβ; Unfolded protein response; Epithelial-to-mesenchymal transition; Tumor repressor

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APA (6th Edition):

Rajapaksa, R. P. G. K. 1. (2014). Understanding the Tumor Repressive Function of Estrogen Receptor Beta in Breast Cancer. (Doctoral Dissertation). University of Houston. Retrieved from http://hdl.handle.net/10657/1919

Chicago Manual of Style (16th Edition):

Rajapaksa, R P Gayani Kumudu 1981-. “Understanding the Tumor Repressive Function of Estrogen Receptor Beta in Breast Cancer.” 2014. Doctoral Dissertation, University of Houston. Accessed January 19, 2021. http://hdl.handle.net/10657/1919.

MLA Handbook (7th Edition):

Rajapaksa, R P Gayani Kumudu 1981-. “Understanding the Tumor Repressive Function of Estrogen Receptor Beta in Breast Cancer.” 2014. Web. 19 Jan 2021.

Vancouver:

Rajapaksa RPGK1. Understanding the Tumor Repressive Function of Estrogen Receptor Beta in Breast Cancer. [Internet] [Doctoral dissertation]. University of Houston; 2014. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/10657/1919.

Council of Science Editors:

Rajapaksa RPGK1. Understanding the Tumor Repressive Function of Estrogen Receptor Beta in Breast Cancer. [Doctoral Dissertation]. University of Houston; 2014. Available from: http://hdl.handle.net/10657/1919


University of Houston

3. Wang, Jun 1988-. The Mechanisms of Tumor-suppressive miRNAs in Mammary Stem Cell Development and Triple-negative Breast Cancer.

Degree: PhD, Biology, 2015, University of Houston

 In this thesis, we investigated the mechanisms of two tumor-suppressive miRNAs in triple-negative breast cancer (TNBC). This undifferentiated subtype of breast cancer shows similarity of… (more)

Subjects/Keywords: MicroRNAs (miRNA); Breast cancer

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APA (6th Edition):

Wang, J. 1. (2015). The Mechanisms of Tumor-suppressive miRNAs in Mammary Stem Cell Development and Triple-negative Breast Cancer. (Doctoral Dissertation). University of Houston. Retrieved from http://hdl.handle.net/10657/4876

Chicago Manual of Style (16th Edition):

Wang, Jun 1988-. “The Mechanisms of Tumor-suppressive miRNAs in Mammary Stem Cell Development and Triple-negative Breast Cancer.” 2015. Doctoral Dissertation, University of Houston. Accessed January 19, 2021. http://hdl.handle.net/10657/4876.

MLA Handbook (7th Edition):

Wang, Jun 1988-. “The Mechanisms of Tumor-suppressive miRNAs in Mammary Stem Cell Development and Triple-negative Breast Cancer.” 2015. Web. 19 Jan 2021.

Vancouver:

Wang J1. The Mechanisms of Tumor-suppressive miRNAs in Mammary Stem Cell Development and Triple-negative Breast Cancer. [Internet] [Doctoral dissertation]. University of Houston; 2015. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/10657/4876.

Council of Science Editors:

Wang J1. The Mechanisms of Tumor-suppressive miRNAs in Mammary Stem Cell Development and Triple-negative Breast Cancer. [Doctoral Dissertation]. University of Houston; 2015. Available from: http://hdl.handle.net/10657/4876


University of Houston

4. Anderson, Marc 1981-. An Account of Chemical and Mechanical Regulation of TRPC6 Channels in Podocytes.

Degree: PhD, Biology, 2013, University of Houston

 Podocytes play a dynamic role in regulating glomerular filtration. The focus here is on the regulatory mechanisms of podocyte expressed transient receptor potential 6 (TRPC6)… (more)

Subjects/Keywords: Podocytes; TRPC6 channels; N-methyl-D-aspartate (NMDA); Receptors; Biology

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APA (6th Edition):

Anderson, M. 1. (2013). An Account of Chemical and Mechanical Regulation of TRPC6 Channels in Podocytes. (Doctoral Dissertation). University of Houston. Retrieved from http://hdl.handle.net/10657/921

Chicago Manual of Style (16th Edition):

Anderson, Marc 1981-. “An Account of Chemical and Mechanical Regulation of TRPC6 Channels in Podocytes.” 2013. Doctoral Dissertation, University of Houston. Accessed January 19, 2021. http://hdl.handle.net/10657/921.

MLA Handbook (7th Edition):

Anderson, Marc 1981-. “An Account of Chemical and Mechanical Regulation of TRPC6 Channels in Podocytes.” 2013. Web. 19 Jan 2021.

Vancouver:

Anderson M1. An Account of Chemical and Mechanical Regulation of TRPC6 Channels in Podocytes. [Internet] [Doctoral dissertation]. University of Houston; 2013. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/10657/921.

Council of Science Editors:

Anderson M1. An Account of Chemical and Mechanical Regulation of TRPC6 Channels in Podocytes. [Doctoral Dissertation]. University of Houston; 2013. Available from: http://hdl.handle.net/10657/921


University of Houston

5. Folly-Kossi, Helena 1984-. Nuclear Receptors in Breast Cancer.

Degree: PhD, Cell and Molecular Biology, 2017, University of Houston

 Breast cancer (BC) is classified into four major molecular subtypes. The most predominant subtypes, luminal A and B are hormone receptor-positive BC (ERα +/PR+) which… (more)

Subjects/Keywords: Nuclear receptors; Breast cancer; Survival analysis; Endocrine therapy; Tamoxifen resistance; Glucocorticoid receptors; Estrogen receptors; Clinical data

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APA (6th Edition):

Folly-Kossi, H. 1. (2017). Nuclear Receptors in Breast Cancer. (Doctoral Dissertation). University of Houston. Retrieved from http://hdl.handle.net/10657/4586

Chicago Manual of Style (16th Edition):

Folly-Kossi, Helena 1984-. “Nuclear Receptors in Breast Cancer.” 2017. Doctoral Dissertation, University of Houston. Accessed January 19, 2021. http://hdl.handle.net/10657/4586.

MLA Handbook (7th Edition):

Folly-Kossi, Helena 1984-. “Nuclear Receptors in Breast Cancer.” 2017. Web. 19 Jan 2021.

Vancouver:

Folly-Kossi H1. Nuclear Receptors in Breast Cancer. [Internet] [Doctoral dissertation]. University of Houston; 2017. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/10657/4586.

Council of Science Editors:

Folly-Kossi H1. Nuclear Receptors in Breast Cancer. [Doctoral Dissertation]. University of Houston; 2017. Available from: http://hdl.handle.net/10657/4586


University of Houston

6. Lama, Chamala 1979-. Identification and Characterization of Sex-Specific Transcripts in the Blood-Brain Barrier of Drosophila melanogaster.

Degree: PhD, Biology, 2013, University of Houston

 In Drosophila, a male displays a series of complex stereotypic acts in courting a female. This behavior is mainly controlled by male specific transcription factors… (more)

Subjects/Keywords: Blood-Brain Barrier; Drosophila; Courtship; Sex-specific transcripts; MiRNAs; Sinu; Hr46; Qvr; MiR-184

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APA (6th Edition):

Lama, C. 1. (2013). Identification and Characterization of Sex-Specific Transcripts in the Blood-Brain Barrier of Drosophila melanogaster. (Doctoral Dissertation). University of Houston. Retrieved from http://hdl.handle.net/10657/1236

Chicago Manual of Style (16th Edition):

Lama, Chamala 1979-. “Identification and Characterization of Sex-Specific Transcripts in the Blood-Brain Barrier of Drosophila melanogaster.” 2013. Doctoral Dissertation, University of Houston. Accessed January 19, 2021. http://hdl.handle.net/10657/1236.

MLA Handbook (7th Edition):

Lama, Chamala 1979-. “Identification and Characterization of Sex-Specific Transcripts in the Blood-Brain Barrier of Drosophila melanogaster.” 2013. Web. 19 Jan 2021.

Vancouver:

Lama C1. Identification and Characterization of Sex-Specific Transcripts in the Blood-Brain Barrier of Drosophila melanogaster. [Internet] [Doctoral dissertation]. University of Houston; 2013. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/10657/1236.

Council of Science Editors:

Lama C1. Identification and Characterization of Sex-Specific Transcripts in the Blood-Brain Barrier of Drosophila melanogaster. [Doctoral Dissertation]. University of Houston; 2013. Available from: http://hdl.handle.net/10657/1236


University of Houston

7. Petkova, Tihomira D. 1974-. TOWARDS RETINA REGENERATION: THE EPIGENETIC BASIS OF NEURONAL AND AXON GUIDANCE GENE REGULATION.

Degree: PhD, Physiological Optics and Vision Science, 2012, University of Houston

 Degenerative disorders, such as glaucoma are among the leading causes of irreversible blindness worldwide. With these disorders affecting a larger fraction of the population yearly,… (more)

Subjects/Keywords: Retinal gene regulation; DNA methylation; Math5/Atoh7; EphA5; Wnt signaling; Müller glia; Stem cells; Retinal regeneration

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APA (6th Edition):

Petkova, T. D. 1. (2012). TOWARDS RETINA REGENERATION: THE EPIGENETIC BASIS OF NEURONAL AND AXON GUIDANCE GENE REGULATION. (Doctoral Dissertation). University of Houston. Retrieved from http://hdl.handle.net/10657/1560

Chicago Manual of Style (16th Edition):

Petkova, Tihomira D 1974-. “TOWARDS RETINA REGENERATION: THE EPIGENETIC BASIS OF NEURONAL AND AXON GUIDANCE GENE REGULATION.” 2012. Doctoral Dissertation, University of Houston. Accessed January 19, 2021. http://hdl.handle.net/10657/1560.

MLA Handbook (7th Edition):

Petkova, Tihomira D 1974-. “TOWARDS RETINA REGENERATION: THE EPIGENETIC BASIS OF NEURONAL AND AXON GUIDANCE GENE REGULATION.” 2012. Web. 19 Jan 2021.

Vancouver:

Petkova TD1. TOWARDS RETINA REGENERATION: THE EPIGENETIC BASIS OF NEURONAL AND AXON GUIDANCE GENE REGULATION. [Internet] [Doctoral dissertation]. University of Houston; 2012. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/10657/1560.

Council of Science Editors:

Petkova TD1. TOWARDS RETINA REGENERATION: THE EPIGENETIC BASIS OF NEURONAL AND AXON GUIDANCE GENE REGULATION. [Doctoral Dissertation]. University of Houston; 2012. Available from: http://hdl.handle.net/10657/1560


University of Houston

8. Shen, Xiaopeng 1990-. miR-322/503 Cluster Drives Cardiac Differentiation by Inhibiting CUG-binding Protein 1.

Degree: PhD, Biochemistry, 2014, University of Houston

 Understanding the mechanisms of early cardiac fate determination may lead to better approaches in promoting heart regeneration after injury. MicroRNAs (miRNAs) involved in the process… (more)

Subjects/Keywords: MicroRNAs (miRNA); Celf1; Cardiac differentiation; Neural differentiation

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APA (6th Edition):

Shen, X. 1. (2014). miR-322/503 Cluster Drives Cardiac Differentiation by Inhibiting CUG-binding Protein 1. (Doctoral Dissertation). University of Houston. Retrieved from http://hdl.handle.net/10657/5669

Chicago Manual of Style (16th Edition):

Shen, Xiaopeng 1990-. “miR-322/503 Cluster Drives Cardiac Differentiation by Inhibiting CUG-binding Protein 1.” 2014. Doctoral Dissertation, University of Houston. Accessed January 19, 2021. http://hdl.handle.net/10657/5669.

MLA Handbook (7th Edition):

Shen, Xiaopeng 1990-. “miR-322/503 Cluster Drives Cardiac Differentiation by Inhibiting CUG-binding Protein 1.” 2014. Web. 19 Jan 2021.

Vancouver:

Shen X1. miR-322/503 Cluster Drives Cardiac Differentiation by Inhibiting CUG-binding Protein 1. [Internet] [Doctoral dissertation]. University of Houston; 2014. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/10657/5669.

Council of Science Editors:

Shen X1. miR-322/503 Cluster Drives Cardiac Differentiation by Inhibiting CUG-binding Protein 1. [Doctoral Dissertation]. University of Houston; 2014. Available from: http://hdl.handle.net/10657/5669


University of Houston

9. Kim, Jong Hwan 1983-. MESP1's Epigenetic and Transcriptional Role during Mesendoderm Formation.

Degree: PhD, Biochemistry, 2016, University of Houston

 MESP1 is a basic helix loop helix transcription factor that is essential for the survival and development of mouse embryogenesis. It is the earliest marker… (more)

Subjects/Keywords: Helix loop; Mesendoderm

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APA (6th Edition):

Kim, J. H. 1. (2016). MESP1's Epigenetic and Transcriptional Role during Mesendoderm Formation. (Doctoral Dissertation). University of Houston. Retrieved from http://hdl.handle.net/10657/5393

Chicago Manual of Style (16th Edition):

Kim, Jong Hwan 1983-. “MESP1's Epigenetic and Transcriptional Role during Mesendoderm Formation.” 2016. Doctoral Dissertation, University of Houston. Accessed January 19, 2021. http://hdl.handle.net/10657/5393.

MLA Handbook (7th Edition):

Kim, Jong Hwan 1983-. “MESP1's Epigenetic and Transcriptional Role during Mesendoderm Formation.” 2016. Web. 19 Jan 2021.

Vancouver:

Kim JH1. MESP1's Epigenetic and Transcriptional Role during Mesendoderm Formation. [Internet] [Doctoral dissertation]. University of Houston; 2016. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/10657/5393.

Council of Science Editors:

Kim JH1. MESP1's Epigenetic and Transcriptional Role during Mesendoderm Formation. [Doctoral Dissertation]. University of Houston; 2016. Available from: http://hdl.handle.net/10657/5393


University of Houston

10. Tennakoon, Jayantha 1969-. Impact of Dicer on the Embryonic Stem Cell Epigenome and Androgen Mediated AMPK-PGC-1α Signaling in Prostate Cancer.

Degree: PhD, Biology, 2013, University of Houston

 What mechanisms govern a cellular phenotype is a fascinating question for which answers are yet being sought. The work presented in this dissertation is an… (more)

Subjects/Keywords: Dicer; Mouse embryonic stem cells; Epigenome; Androgen signaling; AMPK; PGC1a; Prostate cancer; Biology

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APA (6th Edition):

Tennakoon, J. 1. (2013). Impact of Dicer on the Embryonic Stem Cell Epigenome and Androgen Mediated AMPK-PGC-1α Signaling in Prostate Cancer. (Doctoral Dissertation). University of Houston. Retrieved from http://hdl.handle.net/10657/1027

Chicago Manual of Style (16th Edition):

Tennakoon, Jayantha 1969-. “Impact of Dicer on the Embryonic Stem Cell Epigenome and Androgen Mediated AMPK-PGC-1α Signaling in Prostate Cancer.” 2013. Doctoral Dissertation, University of Houston. Accessed January 19, 2021. http://hdl.handle.net/10657/1027.

MLA Handbook (7th Edition):

Tennakoon, Jayantha 1969-. “Impact of Dicer on the Embryonic Stem Cell Epigenome and Androgen Mediated AMPK-PGC-1α Signaling in Prostate Cancer.” 2013. Web. 19 Jan 2021.

Vancouver:

Tennakoon J1. Impact of Dicer on the Embryonic Stem Cell Epigenome and Androgen Mediated AMPK-PGC-1α Signaling in Prostate Cancer. [Internet] [Doctoral dissertation]. University of Houston; 2013. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/10657/1027.

Council of Science Editors:

Tennakoon J1. Impact of Dicer on the Embryonic Stem Cell Epigenome and Androgen Mediated AMPK-PGC-1α Signaling in Prostate Cancer. [Doctoral Dissertation]. University of Houston; 2013. Available from: http://hdl.handle.net/10657/1027


University of Houston

11. Valenzuela, Nicolas 1988-. A Role for the Histone Chaperone Hira in Muscle Hypertrophy, Cellular Stress Responses, and Developmental Gene Expression.

Degree: PhD, Biochemistry, 2016, University of Houston

 Chromatin modifications play a pivotal role in regulating gene expression. The deposition of histone variants by histone chaperones into the nucleosome plays a large role… (more)

Subjects/Keywords: Cardiomyocyte; Myocyte; H3.3; HIRA; Heart health; Skeletal muscle; Histone; Histone Chaperone

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APA (6th Edition):

Valenzuela, N. 1. (2016). A Role for the Histone Chaperone Hira in Muscle Hypertrophy, Cellular Stress Responses, and Developmental Gene Expression. (Doctoral Dissertation). University of Houston. Retrieved from http://hdl.handle.net/10657/1938

Chicago Manual of Style (16th Edition):

Valenzuela, Nicolas 1988-. “A Role for the Histone Chaperone Hira in Muscle Hypertrophy, Cellular Stress Responses, and Developmental Gene Expression.” 2016. Doctoral Dissertation, University of Houston. Accessed January 19, 2021. http://hdl.handle.net/10657/1938.

MLA Handbook (7th Edition):

Valenzuela, Nicolas 1988-. “A Role for the Histone Chaperone Hira in Muscle Hypertrophy, Cellular Stress Responses, and Developmental Gene Expression.” 2016. Web. 19 Jan 2021.

Vancouver:

Valenzuela N1. A Role for the Histone Chaperone Hira in Muscle Hypertrophy, Cellular Stress Responses, and Developmental Gene Expression. [Internet] [Doctoral dissertation]. University of Houston; 2016. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/10657/1938.

Council of Science Editors:

Valenzuela N1. A Role for the Histone Chaperone Hira in Muscle Hypertrophy, Cellular Stress Responses, and Developmental Gene Expression. [Doctoral Dissertation]. University of Houston; 2016. Available from: http://hdl.handle.net/10657/1938


University of Houston

12. -8465-3791. Defining Anti-Tumorigenic and Anti-Inflammatory Effects Mediated By Estrogen Receptor Beta In Colon Epithelial Cells.

Degree: PhD, Biology and Biochemistry, 2015, University of Houston

 Despite its slow development and our capacity for early detection using endoscopy, colorectal cancer remains the second leading cause of cancer death in the United… (more)

Subjects/Keywords: Estrogen receptor beta; Colon cancer epithelial cells

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APA (6th Edition):

-8465-3791. (2015). Defining Anti-Tumorigenic and Anti-Inflammatory Effects Mediated By Estrogen Receptor Beta In Colon Epithelial Cells. (Doctoral Dissertation). University of Houston. Retrieved from http://hdl.handle.net/10657/2018

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Author name may be incomplete

Chicago Manual of Style (16th Edition):

-8465-3791. “Defining Anti-Tumorigenic and Anti-Inflammatory Effects Mediated By Estrogen Receptor Beta In Colon Epithelial Cells.” 2015. Doctoral Dissertation, University of Houston. Accessed January 19, 2021. http://hdl.handle.net/10657/2018.

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Author name may be incomplete

MLA Handbook (7th Edition):

-8465-3791. “Defining Anti-Tumorigenic and Anti-Inflammatory Effects Mediated By Estrogen Receptor Beta In Colon Epithelial Cells.” 2015. Web. 19 Jan 2021.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Vancouver:

-8465-3791. Defining Anti-Tumorigenic and Anti-Inflammatory Effects Mediated By Estrogen Receptor Beta In Colon Epithelial Cells. [Internet] [Doctoral dissertation]. University of Houston; 2015. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/10657/2018.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Council of Science Editors:

-8465-3791. Defining Anti-Tumorigenic and Anti-Inflammatory Effects Mediated By Estrogen Receptor Beta In Colon Epithelial Cells. [Doctoral Dissertation]. University of Houston; 2015. Available from: http://hdl.handle.net/10657/2018

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Author name may be incomplete


University of Houston

13. Hernandez Herrera, Anadulce 1982-. A Genome-Wide Search for Tumor Suppressor MicroRNAs in Ovarian Cancer.

Degree: PhD, Biochemistry, 2014, University of Houston

 Ovarian cancer is one of the most lethal cancers among women. The Cancer Genome Atlas (TCGA) is a collaborative effort, which seeks to characterize the… (more)

Subjects/Keywords: MicroRNAs (miRNA); Ovarian cancer; The cancer genome atlas (TCGA)

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APA (6th Edition):

Hernandez Herrera, A. 1. (2014). A Genome-Wide Search for Tumor Suppressor MicroRNAs in Ovarian Cancer. (Doctoral Dissertation). University of Houston. Retrieved from http://hdl.handle.net/10657/4756

Chicago Manual of Style (16th Edition):

Hernandez Herrera, Anadulce 1982-. “A Genome-Wide Search for Tumor Suppressor MicroRNAs in Ovarian Cancer.” 2014. Doctoral Dissertation, University of Houston. Accessed January 19, 2021. http://hdl.handle.net/10657/4756.

MLA Handbook (7th Edition):

Hernandez Herrera, Anadulce 1982-. “A Genome-Wide Search for Tumor Suppressor MicroRNAs in Ovarian Cancer.” 2014. Web. 19 Jan 2021.

Vancouver:

Hernandez Herrera A1. A Genome-Wide Search for Tumor Suppressor MicroRNAs in Ovarian Cancer. [Internet] [Doctoral dissertation]. University of Houston; 2014. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/10657/4756.

Council of Science Editors:

Hernandez Herrera A1. A Genome-Wide Search for Tumor Suppressor MicroRNAs in Ovarian Cancer. [Doctoral Dissertation]. University of Houston; 2014. Available from: http://hdl.handle.net/10657/4756


University of Houston

14. Ghosh, Rajib 1979-. Redefining tumor suppressor microRNAs: functional complexities & nanoparticle mediated delivery.

Degree: PhD, Biology, 2012, University of Houston

 MicroRNAs (miRNAs) are 22-24 nucleotides non-coding RNAs that can simultaneously influence the levels of multiple target genes and have the potential to strongly silence multiple… (more)

Subjects/Keywords: MicroRNAs (miRNA); Cancer; Neuroblastoma; Neurosciences; Ovarian cancer; Gold nanoparticles; In vitro; Biology

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APA (6th Edition):

Ghosh, R. 1. (2012). Redefining tumor suppressor microRNAs: functional complexities & nanoparticle mediated delivery. (Doctoral Dissertation). University of Houston. Retrieved from http://hdl.handle.net/10657/836

Chicago Manual of Style (16th Edition):

Ghosh, Rajib 1979-. “Redefining tumor suppressor microRNAs: functional complexities & nanoparticle mediated delivery.” 2012. Doctoral Dissertation, University of Houston. Accessed January 19, 2021. http://hdl.handle.net/10657/836.

MLA Handbook (7th Edition):

Ghosh, Rajib 1979-. “Redefining tumor suppressor microRNAs: functional complexities & nanoparticle mediated delivery.” 2012. Web. 19 Jan 2021.

Vancouver:

Ghosh R1. Redefining tumor suppressor microRNAs: functional complexities & nanoparticle mediated delivery. [Internet] [Doctoral dissertation]. University of Houston; 2012. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/10657/836.

Council of Science Editors:

Ghosh R1. Redefining tumor suppressor microRNAs: functional complexities & nanoparticle mediated delivery. [Doctoral Dissertation]. University of Houston; 2012. Available from: http://hdl.handle.net/10657/836

15. Chaney, Shawnta Y. Retinal development and age related degeneration following gestational lead exposure.

Degree: PhD, Biochemistry, 2013, University of Houston

 PURPOSE: Gestational lead exposure (GLE) increased and prolonged retinal progenitor cell proliferation in mice, resulting in a dose-dependent increase in two late-born retinal neurons: rod… (more)

Subjects/Keywords: Retinal development; Toxicology; Lead; Biochemistry

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APA (6th Edition):

Chaney, S. Y. (2013). Retinal development and age related degeneration following gestational lead exposure. (Doctoral Dissertation). University of Houston. Retrieved from http://hdl.handle.net/10657/1041

Chicago Manual of Style (16th Edition):

Chaney, Shawnta Y. “Retinal development and age related degeneration following gestational lead exposure.” 2013. Doctoral Dissertation, University of Houston. Accessed January 19, 2021. http://hdl.handle.net/10657/1041.

MLA Handbook (7th Edition):

Chaney, Shawnta Y. “Retinal development and age related degeneration following gestational lead exposure.” 2013. Web. 19 Jan 2021.

Vancouver:

Chaney SY. Retinal development and age related degeneration following gestational lead exposure. [Internet] [Doctoral dissertation]. University of Houston; 2013. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/10657/1041.

Council of Science Editors:

Chaney SY. Retinal development and age related degeneration following gestational lead exposure. [Doctoral Dissertation]. University of Houston; 2013. Available from: http://hdl.handle.net/10657/1041


University of Houston

16. Rababa'H, Abeer 1981-. Human Signaling Scaffold Protein (mAKAP) Polymorphisms: Role in Heart Failure.

Degree: Pharmacology, Pharmacology PhD, University of Houston

 Protein kinase-A (PKA) substrate phosphorylation is facilitated through its co-localization with its signaling partner by A-kinase anchoring proteins (AKAPs). mAKAP (muscle-selective AKAP) localizes PKA and… (more)

Subjects/Keywords: Heart failure; Protein kinase A; MAKAP; Hypertrophy

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APA (6th Edition):

Rababa'H, A. 1. (n.d.). Human Signaling Scaffold Protein (mAKAP) Polymorphisms: Role in Heart Failure. (Doctoral Dissertation). University of Houston. Retrieved from http://hdl.handle.net/10657/2616

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Chicago Manual of Style (16th Edition):

Rababa'H, Abeer 1981-. “Human Signaling Scaffold Protein (mAKAP) Polymorphisms: Role in Heart Failure.” Doctoral Dissertation, University of Houston. Accessed January 19, 2021. http://hdl.handle.net/10657/2616.

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MLA Handbook (7th Edition):

Rababa'H, Abeer 1981-. “Human Signaling Scaffold Protein (mAKAP) Polymorphisms: Role in Heart Failure.” Web. 19 Jan 2021.

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Vancouver:

Rababa'H A1. Human Signaling Scaffold Protein (mAKAP) Polymorphisms: Role in Heart Failure. [Internet] [Doctoral dissertation]. University of Houston; [cited 2021 Jan 19]. Available from: http://hdl.handle.net/10657/2616.

Note: this citation may be lacking information needed for this citation format:
No year of publication.

Council of Science Editors:

Rababa'H A1. Human Signaling Scaffold Protein (mAKAP) Polymorphisms: Role in Heart Failure. [Doctoral Dissertation]. University of Houston; Available from: http://hdl.handle.net/10657/2616

Note: this citation may be lacking information needed for this citation format:
No year of publication.

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