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You searched for +publisher:"University of Houston" +contributor:("Frigo, Daniel E."). Showing records 1 – 15 of 15 total matches.

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University of Houston

1. -6543-5086. Regulation of Nutrient Uptake in Prostate Cancer.

Degree: PhD, Cell and Molecular Biology, 2017, University of Houston

 Prostate cancer is a hormone-driven malignancy that relies on the function of the androgen receptor (AR). AR is a transcription factor that regulates the expression… (more)

Subjects/Keywords: Glutamine; Glucose metabolism; Prostate cancer; MYC; MTOR; SLC1A4; SLC1A5; AMPK; CaMKK2; SLC2A12; GLUT12; TBC1D4

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APA (6th Edition):

-6543-5086. (2017). Regulation of Nutrient Uptake in Prostate Cancer. (Doctoral Dissertation). University of Houston. Retrieved from http://hdl.handle.net/10657/4533

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Chicago Manual of Style (16th Edition):

-6543-5086. “Regulation of Nutrient Uptake in Prostate Cancer.” 2017. Doctoral Dissertation, University of Houston. Accessed January 16, 2021. http://hdl.handle.net/10657/4533.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

MLA Handbook (7th Edition):

-6543-5086. “Regulation of Nutrient Uptake in Prostate Cancer.” 2017. Web. 16 Jan 2021.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Vancouver:

-6543-5086. Regulation of Nutrient Uptake in Prostate Cancer. [Internet] [Doctoral dissertation]. University of Houston; 2017. [cited 2021 Jan 16]. Available from: http://hdl.handle.net/10657/4533.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Council of Science Editors:

-6543-5086. Regulation of Nutrient Uptake in Prostate Cancer. [Doctoral Dissertation]. University of Houston; 2017. Available from: http://hdl.handle.net/10657/4533

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete


University of Houston

2. Wang, Jun 1988-. The Mechanisms of Tumor-suppressive miRNAs in Mammary Stem Cell Development and Triple-negative Breast Cancer.

Degree: PhD, Biology, 2015, University of Houston

 In this thesis, we investigated the mechanisms of two tumor-suppressive miRNAs in triple-negative breast cancer (TNBC). This undifferentiated subtype of breast cancer shows similarity of… (more)

Subjects/Keywords: MicroRNAs (miRNA); Breast cancer

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APA (6th Edition):

Wang, J. 1. (2015). The Mechanisms of Tumor-suppressive miRNAs in Mammary Stem Cell Development and Triple-negative Breast Cancer. (Doctoral Dissertation). University of Houston. Retrieved from http://hdl.handle.net/10657/4876

Chicago Manual of Style (16th Edition):

Wang, Jun 1988-. “The Mechanisms of Tumor-suppressive miRNAs in Mammary Stem Cell Development and Triple-negative Breast Cancer.” 2015. Doctoral Dissertation, University of Houston. Accessed January 16, 2021. http://hdl.handle.net/10657/4876.

MLA Handbook (7th Edition):

Wang, Jun 1988-. “The Mechanisms of Tumor-suppressive miRNAs in Mammary Stem Cell Development and Triple-negative Breast Cancer.” 2015. Web. 16 Jan 2021.

Vancouver:

Wang J1. The Mechanisms of Tumor-suppressive miRNAs in Mammary Stem Cell Development and Triple-negative Breast Cancer. [Internet] [Doctoral dissertation]. University of Houston; 2015. [cited 2021 Jan 16]. Available from: http://hdl.handle.net/10657/4876.

Council of Science Editors:

Wang J1. The Mechanisms of Tumor-suppressive miRNAs in Mammary Stem Cell Development and Triple-negative Breast Cancer. [Doctoral Dissertation]. University of Houston; 2015. Available from: http://hdl.handle.net/10657/4876


University of Houston

3. Reins, Rose 1976-. Function of Vitamin D at the Ocular Surface and Its Role during Corneal Inflammation.

Degree: PhD, Biology and Biochemistry, 2015, University of Houston

 Although first recognized for its role in calcium regulation and bone health, there has been an explosion of research demonstrating that vitamin D is an… (more)

Subjects/Keywords: Vitamin D; Cornea; Ocular surface; Inflammation; Antimicrobial peptide; Wounding; Dry eye diseases; Immunomodulation

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APA (6th Edition):

Reins, R. 1. (2015). Function of Vitamin D at the Ocular Surface and Its Role during Corneal Inflammation. (Doctoral Dissertation). University of Houston. Retrieved from http://hdl.handle.net/10657/1981

Chicago Manual of Style (16th Edition):

Reins, Rose 1976-. “Function of Vitamin D at the Ocular Surface and Its Role during Corneal Inflammation.” 2015. Doctoral Dissertation, University of Houston. Accessed January 16, 2021. http://hdl.handle.net/10657/1981.

MLA Handbook (7th Edition):

Reins, Rose 1976-. “Function of Vitamin D at the Ocular Surface and Its Role during Corneal Inflammation.” 2015. Web. 16 Jan 2021.

Vancouver:

Reins R1. Function of Vitamin D at the Ocular Surface and Its Role during Corneal Inflammation. [Internet] [Doctoral dissertation]. University of Houston; 2015. [cited 2021 Jan 16]. Available from: http://hdl.handle.net/10657/1981.

Council of Science Editors:

Reins R1. Function of Vitamin D at the Ocular Surface and Its Role during Corneal Inflammation. [Doctoral Dissertation]. University of Houston; 2015. Available from: http://hdl.handle.net/10657/1981


University of Houston

4. -8379-9505. Modulation of Nuclear Receptor Functions in Breast and Pancreatic Cancers.

Degree: PhD, Cell and Molecular Biology, 2015, University of Houston

 Alcohol Regulates Genes that are Associated with Response to Endocrine Therapy and Attenuates the Actions of Tamoxifen in Breast Cancer Cells. Pancreatic ductal adenocarcinoma (PDAC)… (more)

Subjects/Keywords: LXR; Pancreatic cancer; ER; Breast cancer; Alcohol

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APA (6th Edition):

-8379-9505. (2015). Modulation of Nuclear Receptor Functions in Breast and Pancreatic Cancers. (Doctoral Dissertation). University of Houston. Retrieved from http://hdl.handle.net/10657/5367

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Chicago Manual of Style (16th Edition):

-8379-9505. “Modulation of Nuclear Receptor Functions in Breast and Pancreatic Cancers.” 2015. Doctoral Dissertation, University of Houston. Accessed January 16, 2021. http://hdl.handle.net/10657/5367.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

MLA Handbook (7th Edition):

-8379-9505. “Modulation of Nuclear Receptor Functions in Breast and Pancreatic Cancers.” 2015. Web. 16 Jan 2021.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Vancouver:

-8379-9505. Modulation of Nuclear Receptor Functions in Breast and Pancreatic Cancers. [Internet] [Doctoral dissertation]. University of Houston; 2015. [cited 2021 Jan 16]. Available from: http://hdl.handle.net/10657/5367.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Council of Science Editors:

-8379-9505. Modulation of Nuclear Receptor Functions in Breast and Pancreatic Cancers. [Doctoral Dissertation]. University of Houston; 2015. Available from: http://hdl.handle.net/10657/5367

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete


University of Houston

5. Mehta, Fabiola Melissa 1985-. Suppression of Cervical Cancer by the Progesterone Receptor Signaling.

Degree: PhD, Biology, 2016, University of Houston

 Cervical cancer is the fourth-most common cancer in woman and fourth leading cause of cancer death worldwide. The majority of cervical cancer is associated with… (more)

Subjects/Keywords: Progesterone receptor; Cervical cancer; Female reproductive tract

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APA (6th Edition):

Mehta, F. M. 1. (2016). Suppression of Cervical Cancer by the Progesterone Receptor Signaling. (Doctoral Dissertation). University of Houston. Retrieved from http://hdl.handle.net/10657/3271

Chicago Manual of Style (16th Edition):

Mehta, Fabiola Melissa 1985-. “Suppression of Cervical Cancer by the Progesterone Receptor Signaling.” 2016. Doctoral Dissertation, University of Houston. Accessed January 16, 2021. http://hdl.handle.net/10657/3271.

MLA Handbook (7th Edition):

Mehta, Fabiola Melissa 1985-. “Suppression of Cervical Cancer by the Progesterone Receptor Signaling.” 2016. Web. 16 Jan 2021.

Vancouver:

Mehta FM1. Suppression of Cervical Cancer by the Progesterone Receptor Signaling. [Internet] [Doctoral dissertation]. University of Houston; 2016. [cited 2021 Jan 16]. Available from: http://hdl.handle.net/10657/3271.

Council of Science Editors:

Mehta FM1. Suppression of Cervical Cancer by the Progesterone Receptor Signaling. [Doctoral Dissertation]. University of Houston; 2016. Available from: http://hdl.handle.net/10657/3271


University of Houston

6. Tennakoon, Jayantha 1969-. Impact of Dicer on the Embryonic Stem Cell Epigenome and Androgen Mediated AMPK-PGC-1α Signaling in Prostate Cancer.

Degree: PhD, Biology, 2013, University of Houston

 What mechanisms govern a cellular phenotype is a fascinating question for which answers are yet being sought. The work presented in this dissertation is an… (more)

Subjects/Keywords: Dicer; Mouse embryonic stem cells; Epigenome; Androgen signaling; AMPK; PGC1a; Prostate cancer; Biology

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APA (6th Edition):

Tennakoon, J. 1. (2013). Impact of Dicer on the Embryonic Stem Cell Epigenome and Androgen Mediated AMPK-PGC-1α Signaling in Prostate Cancer. (Doctoral Dissertation). University of Houston. Retrieved from http://hdl.handle.net/10657/1027

Chicago Manual of Style (16th Edition):

Tennakoon, Jayantha 1969-. “Impact of Dicer on the Embryonic Stem Cell Epigenome and Androgen Mediated AMPK-PGC-1α Signaling in Prostate Cancer.” 2013. Doctoral Dissertation, University of Houston. Accessed January 16, 2021. http://hdl.handle.net/10657/1027.

MLA Handbook (7th Edition):

Tennakoon, Jayantha 1969-. “Impact of Dicer on the Embryonic Stem Cell Epigenome and Androgen Mediated AMPK-PGC-1α Signaling in Prostate Cancer.” 2013. Web. 16 Jan 2021.

Vancouver:

Tennakoon J1. Impact of Dicer on the Embryonic Stem Cell Epigenome and Androgen Mediated AMPK-PGC-1α Signaling in Prostate Cancer. [Internet] [Doctoral dissertation]. University of Houston; 2013. [cited 2021 Jan 16]. Available from: http://hdl.handle.net/10657/1027.

Council of Science Editors:

Tennakoon J1. Impact of Dicer on the Embryonic Stem Cell Epigenome and Androgen Mediated AMPK-PGC-1α Signaling in Prostate Cancer. [Doctoral Dissertation]. University of Houston; 2013. Available from: http://hdl.handle.net/10657/1027


University of Houston

7. Cuko, Efrosini 1987-. Elucidating the Role of Glucose Metabolism in Prostate Cancer.

Degree: PhD, Cell and Molecular Biology, 2015, University of Houston

 Prostate cancer is a complex disease. Despite the progress in early detection and treatment, the progression to castration-resistant prostate cancer (CRPC) is inevitable for some… (more)

Subjects/Keywords: Prostate cancer; Pentose phosphate pathway; Glycolysis; Glucose transporters; AMPK; CaMKK2; PPP; G6PD; PFKFB2; PFKFB3; MTOR; GLUT12; TBC1D4

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APA (6th Edition):

Cuko, E. 1. (2015). Elucidating the Role of Glucose Metabolism in Prostate Cancer. (Doctoral Dissertation). University of Houston. Retrieved from http://hdl.handle.net/10657/5371

Chicago Manual of Style (16th Edition):

Cuko, Efrosini 1987-. “Elucidating the Role of Glucose Metabolism in Prostate Cancer.” 2015. Doctoral Dissertation, University of Houston. Accessed January 16, 2021. http://hdl.handle.net/10657/5371.

MLA Handbook (7th Edition):

Cuko, Efrosini 1987-. “Elucidating the Role of Glucose Metabolism in Prostate Cancer.” 2015. Web. 16 Jan 2021.

Vancouver:

Cuko E1. Elucidating the Role of Glucose Metabolism in Prostate Cancer. [Internet] [Doctoral dissertation]. University of Houston; 2015. [cited 2021 Jan 16]. Available from: http://hdl.handle.net/10657/5371.

Council of Science Editors:

Cuko E1. Elucidating the Role of Glucose Metabolism in Prostate Cancer. [Doctoral Dissertation]. University of Houston; 2015. Available from: http://hdl.handle.net/10657/5371


University of Houston

8. -9636-8146. Long Noncoding RNAs in Cardiac and Skeletal Muscle Differentiation during Mouse Embryogenesis.

Degree: PhD, Biology, 2017, University of Houston

 Development is a multistep process that involves a close co-ordination of gene regulatory networks. Lineage specification is a crucial step involved in embryogenesis and understanding… (more)

Subjects/Keywords: Long non coding RNA; RNA; Platr14; Cardiac; Skeletal myogenesis; Differentiation

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APA (6th Edition):

-9636-8146. (2017). Long Noncoding RNAs in Cardiac and Skeletal Muscle Differentiation during Mouse Embryogenesis. (Doctoral Dissertation). University of Houston. Retrieved from http://hdl.handle.net/10657/4789

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Chicago Manual of Style (16th Edition):

-9636-8146. “Long Noncoding RNAs in Cardiac and Skeletal Muscle Differentiation during Mouse Embryogenesis.” 2017. Doctoral Dissertation, University of Houston. Accessed January 16, 2021. http://hdl.handle.net/10657/4789.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

MLA Handbook (7th Edition):

-9636-8146. “Long Noncoding RNAs in Cardiac and Skeletal Muscle Differentiation during Mouse Embryogenesis.” 2017. Web. 16 Jan 2021.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Vancouver:

-9636-8146. Long Noncoding RNAs in Cardiac and Skeletal Muscle Differentiation during Mouse Embryogenesis. [Internet] [Doctoral dissertation]. University of Houston; 2017. [cited 2021 Jan 16]. Available from: http://hdl.handle.net/10657/4789.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Council of Science Editors:

-9636-8146. Long Noncoding RNAs in Cardiac and Skeletal Muscle Differentiation during Mouse Embryogenesis. [Doctoral Dissertation]. University of Houston; 2017. Available from: http://hdl.handle.net/10657/4789

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete


University of Houston

9. -3163-8114. Novel Mechanisms of Androgen Receptor Signaling in Prostate Cancer.

Degree: PhD, Biology, 2015, University of Houston

 Androgens regulate the physiological development of the prostate and the pathology of prostate cancer. Androgen-receptor (AR)-mediated transcriptional activity is a driver of prostate cancer (PCa)… (more)

Subjects/Keywords: Androgen receptor; Prostate cancer; SH3YL1; Ubinuclein1 (UBN1); Autophagy; Polyproline

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APA (6th Edition):

-3163-8114. (2015). Novel Mechanisms of Androgen Receptor Signaling in Prostate Cancer. (Doctoral Dissertation). University of Houston. Retrieved from http://hdl.handle.net/10657/4885

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Chicago Manual of Style (16th Edition):

-3163-8114. “Novel Mechanisms of Androgen Receptor Signaling in Prostate Cancer.” 2015. Doctoral Dissertation, University of Houston. Accessed January 16, 2021. http://hdl.handle.net/10657/4885.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

MLA Handbook (7th Edition):

-3163-8114. “Novel Mechanisms of Androgen Receptor Signaling in Prostate Cancer.” 2015. Web. 16 Jan 2021.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Vancouver:

-3163-8114. Novel Mechanisms of Androgen Receptor Signaling in Prostate Cancer. [Internet] [Doctoral dissertation]. University of Houston; 2015. [cited 2021 Jan 16]. Available from: http://hdl.handle.net/10657/4885.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Council of Science Editors:

-3163-8114. Novel Mechanisms of Androgen Receptor Signaling in Prostate Cancer. [Doctoral Dissertation]. University of Houston; 2015. Available from: http://hdl.handle.net/10657/4885

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete


University of Houston

10. Son, Jieun 1985-. Mechanism of Estrogen Receptor Alpha in Cervical Carcinogenesis.

Degree: PhD, Cell and Molecular Biology, 2017, University of Houston

 Most cervical cancers are associated with high-risk human papillomaviruses (HPVs). HPVs display tumorigenic functions in host cells mainly through viral oncogenes E6 and E7 that… (more)

Subjects/Keywords: Estrogen receptor alpha; Cervical cancer

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APA (6th Edition):

Son, J. 1. (2017). Mechanism of Estrogen Receptor Alpha in Cervical Carcinogenesis. (Doctoral Dissertation). University of Houston. Retrieved from http://hdl.handle.net/10657/4602

Chicago Manual of Style (16th Edition):

Son, Jieun 1985-. “Mechanism of Estrogen Receptor Alpha in Cervical Carcinogenesis.” 2017. Doctoral Dissertation, University of Houston. Accessed January 16, 2021. http://hdl.handle.net/10657/4602.

MLA Handbook (7th Edition):

Son, Jieun 1985-. “Mechanism of Estrogen Receptor Alpha in Cervical Carcinogenesis.” 2017. Web. 16 Jan 2021.

Vancouver:

Son J1. Mechanism of Estrogen Receptor Alpha in Cervical Carcinogenesis. [Internet] [Doctoral dissertation]. University of Houston; 2017. [cited 2021 Jan 16]. Available from: http://hdl.handle.net/10657/4602.

Council of Science Editors:

Son J1. Mechanism of Estrogen Receptor Alpha in Cervical Carcinogenesis. [Doctoral Dissertation]. University of Houston; 2017. Available from: http://hdl.handle.net/10657/4602

11. Swaby, Tyrek Nesta 1982-. Design and Synthesis of 4-Hydroxy Proline Based Library for Pharmaceutical Uses.

Degree: MS, Chemistry, 2013, University of Houston

 Trans-4-hydroxy proline is readily accessible, stereochemically rich molecule that offers multiple sites for functionality. This functionality can be used to attach different pharmacophores in an… (more)

Subjects/Keywords: Proline; Small Molecule Library; Drug design; Scaffold Modification; Chemistry

Page 1 Page 2 Page 3 Page 4 Page 5 Page 6 Page 7

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APA (6th Edition):

Swaby, T. N. 1. (2013). Design and Synthesis of 4-Hydroxy Proline Based Library for Pharmaceutical Uses. (Masters Thesis). University of Houston. Retrieved from http://hdl.handle.net/10657/838

Chicago Manual of Style (16th Edition):

Swaby, Tyrek Nesta 1982-. “Design and Synthesis of 4-Hydroxy Proline Based Library for Pharmaceutical Uses.” 2013. Masters Thesis, University of Houston. Accessed January 16, 2021. http://hdl.handle.net/10657/838.

MLA Handbook (7th Edition):

Swaby, Tyrek Nesta 1982-. “Design and Synthesis of 4-Hydroxy Proline Based Library for Pharmaceutical Uses.” 2013. Web. 16 Jan 2021.

Vancouver:

Swaby TN1. Design and Synthesis of 4-Hydroxy Proline Based Library for Pharmaceutical Uses. [Internet] [Masters thesis]. University of Houston; 2013. [cited 2021 Jan 16]. Available from: http://hdl.handle.net/10657/838.

Council of Science Editors:

Swaby TN1. Design and Synthesis of 4-Hydroxy Proline Based Library for Pharmaceutical Uses. [Masters Thesis]. University of Houston; 2013. Available from: http://hdl.handle.net/10657/838


University of Houston

12. -5121-5234. Inhibition of Fatty Acid Amide Hydrolase Increases Rates of Apoptosis in Cell Line Models of DLBCL and Breast Cancer.

Degree: MS, Biomedical Engineering, University of Houston

 Despite recent advances in cancer treatment, cancers with high heterogeneity such as DLBCL and breast cancer remain difficult to treat, with many patients having few… (more)

Subjects/Keywords: FAAH; Fatty Acid Amide Hydrolase; DLBCL; Breast cancer; CB1; CB2; Endocannabinoids; Apoptosis; URB 597

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APA (6th Edition):

-5121-5234. (n.d.). Inhibition of Fatty Acid Amide Hydrolase Increases Rates of Apoptosis in Cell Line Models of DLBCL and Breast Cancer. (Masters Thesis). University of Houston. Retrieved from http://hdl.handle.net/10657/3576

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
No year of publication.

Chicago Manual of Style (16th Edition):

-5121-5234. “Inhibition of Fatty Acid Amide Hydrolase Increases Rates of Apoptosis in Cell Line Models of DLBCL and Breast Cancer.” Masters Thesis, University of Houston. Accessed January 16, 2021. http://hdl.handle.net/10657/3576.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
No year of publication.

MLA Handbook (7th Edition):

-5121-5234. “Inhibition of Fatty Acid Amide Hydrolase Increases Rates of Apoptosis in Cell Line Models of DLBCL and Breast Cancer.” Web. 16 Jan 2021.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
No year of publication.

Vancouver:

-5121-5234. Inhibition of Fatty Acid Amide Hydrolase Increases Rates of Apoptosis in Cell Line Models of DLBCL and Breast Cancer. [Internet] [Masters thesis]. University of Houston; [cited 2021 Jan 16]. Available from: http://hdl.handle.net/10657/3576.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
No year of publication.

Council of Science Editors:

-5121-5234. Inhibition of Fatty Acid Amide Hydrolase Increases Rates of Apoptosis in Cell Line Models of DLBCL and Breast Cancer. [Masters Thesis]. University of Houston; Available from: http://hdl.handle.net/10657/3576

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
No year of publication.


University of Houston

13. Khan, Ayesha 1985-. mTOR-Mediated Regulation of Metabolic Pathways in Prostate Cancer.

Degree: MS, Biology, University of Houston

 Metabolic reprogramming is one of the key features of many cancers including prostate cancer. Interestingly, most prostate cancers demonstrate a marked shift towards increased mitochondrial… (more)

Subjects/Keywords: Prostate; Pathways

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APA (6th Edition):

Khan, A. 1. (n.d.). mTOR-Mediated Regulation of Metabolic Pathways in Prostate Cancer. (Masters Thesis). University of Houston. Retrieved from http://hdl.handle.net/10657/3567

Note: this citation may be lacking information needed for this citation format:
No year of publication.

Chicago Manual of Style (16th Edition):

Khan, Ayesha 1985-. “mTOR-Mediated Regulation of Metabolic Pathways in Prostate Cancer.” Masters Thesis, University of Houston. Accessed January 16, 2021. http://hdl.handle.net/10657/3567.

Note: this citation may be lacking information needed for this citation format:
No year of publication.

MLA Handbook (7th Edition):

Khan, Ayesha 1985-. “mTOR-Mediated Regulation of Metabolic Pathways in Prostate Cancer.” Web. 16 Jan 2021.

Note: this citation may be lacking information needed for this citation format:
No year of publication.

Vancouver:

Khan A1. mTOR-Mediated Regulation of Metabolic Pathways in Prostate Cancer. [Internet] [Masters thesis]. University of Houston; [cited 2021 Jan 16]. Available from: http://hdl.handle.net/10657/3567.

Note: this citation may be lacking information needed for this citation format:
No year of publication.

Council of Science Editors:

Khan A1. mTOR-Mediated Regulation of Metabolic Pathways in Prostate Cancer. [Masters Thesis]. University of Houston; Available from: http://hdl.handle.net/10657/3567

Note: this citation may be lacking information needed for this citation format:
No year of publication.


University of Houston

14. Nikollo, Foti 1986-. Exploring the Tumor Suppressive Roles of Estrogen Receptor β in Lung and Breast Cancer.

Degree: PhD, Biochemistry, University of Houston

 Estrogens represent a subclass of steroid hormones that by regulating cell growth and differentiation, influence normal physiology as well as pathology. The effects of estrogen… (more)

Subjects/Keywords: Estrogen receptor beta; EGFR; NSCLC; RAS; DNA damage response; P53; EMT; Basal-like; E-cadherin

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APA (6th Edition):

Nikollo, F. 1. (n.d.). Exploring the Tumor Suppressive Roles of Estrogen Receptor β in Lung and Breast Cancer. (Doctoral Dissertation). University of Houston. Retrieved from http://hdl.handle.net/10657/2043

Note: this citation may be lacking information needed for this citation format:
No year of publication.

Chicago Manual of Style (16th Edition):

Nikollo, Foti 1986-. “Exploring the Tumor Suppressive Roles of Estrogen Receptor β in Lung and Breast Cancer.” Doctoral Dissertation, University of Houston. Accessed January 16, 2021. http://hdl.handle.net/10657/2043.

Note: this citation may be lacking information needed for this citation format:
No year of publication.

MLA Handbook (7th Edition):

Nikollo, Foti 1986-. “Exploring the Tumor Suppressive Roles of Estrogen Receptor β in Lung and Breast Cancer.” Web. 16 Jan 2021.

Note: this citation may be lacking information needed for this citation format:
No year of publication.

Vancouver:

Nikollo F1. Exploring the Tumor Suppressive Roles of Estrogen Receptor β in Lung and Breast Cancer. [Internet] [Doctoral dissertation]. University of Houston; [cited 2021 Jan 16]. Available from: http://hdl.handle.net/10657/2043.

Note: this citation may be lacking information needed for this citation format:
No year of publication.

Council of Science Editors:

Nikollo F1. Exploring the Tumor Suppressive Roles of Estrogen Receptor β in Lung and Breast Cancer. [Doctoral Dissertation]. University of Houston; Available from: http://hdl.handle.net/10657/2043

Note: this citation may be lacking information needed for this citation format:
No year of publication.


University of Houston

15. Bado, Igor Landry 1986-. Understanding the Tumor Suppressive Functions of Estrogen Receptor β in Breast Cancer.

Degree: PhD, Biology, University of Houston

 Breast cancer is a heterogeneous disease with regard to clinical outcome and molecular characteristics. Some breast cancers are more aggressive because they express mutant proteins… (more)

Subjects/Keywords: Breast cancer; Cancer; Triple negative breast cancer cells (TNBC); ERβ1; P53; P63; EMT; Invasion; Metastasis; Luminal breast cancer; ERα

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APA (6th Edition):

Bado, I. L. 1. (n.d.). Understanding the Tumor Suppressive Functions of Estrogen Receptor β in Breast Cancer. (Doctoral Dissertation). University of Houston. Retrieved from http://hdl.handle.net/10657/3620

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Chicago Manual of Style (16th Edition):

Bado, Igor Landry 1986-. “Understanding the Tumor Suppressive Functions of Estrogen Receptor β in Breast Cancer.” Doctoral Dissertation, University of Houston. Accessed January 16, 2021. http://hdl.handle.net/10657/3620.

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No year of publication.

MLA Handbook (7th Edition):

Bado, Igor Landry 1986-. “Understanding the Tumor Suppressive Functions of Estrogen Receptor β in Breast Cancer.” Web. 16 Jan 2021.

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Vancouver:

Bado IL1. Understanding the Tumor Suppressive Functions of Estrogen Receptor β in Breast Cancer. [Internet] [Doctoral dissertation]. University of Houston; [cited 2021 Jan 16]. Available from: http://hdl.handle.net/10657/3620.

Note: this citation may be lacking information needed for this citation format:
No year of publication.

Council of Science Editors:

Bado IL1. Understanding the Tumor Suppressive Functions of Estrogen Receptor β in Breast Cancer. [Doctoral Dissertation]. University of Houston; Available from: http://hdl.handle.net/10657/3620

Note: this citation may be lacking information needed for this citation format:
No year of publication.

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