Advanced search options

Advanced Search Options 🞨

Browse by author name (“Author name starts with…”).

Find ETDs with:

in
/  
in
/  
in
/  
in

Written in Published in Earliest date Latest date

Sorted by

Results per page:

Sorted by: relevance · author · university · dateNew search

You searched for +publisher:"University of Hong Kong" +contributor:("Vanhoutte, PMGR"). Showing records 1 – 20 of 20 total matches.

Search Limiters

Last 2 Years | English Only

No search limiters apply to these results.

▼ Search Limiters


University of Hong Kong

1. Mao, Xiaowen. Peroxynitrite/Ho-1 interaction in propofol post-conditioning protection against myocardial ischemia reperfusion injury.

Degree: PhD, 2013, University of Hong Kong

 Coronary artery disease limits myocardial blood flow and results in myocardial infarction. Reperfusion therapies restore coronary flow, but may also cause myocardial ischemia reperfusion injury… (more)

Subjects/Keywords: Myocardial reperfusion; Reperfusion injury

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Mao, X. (2013). Peroxynitrite/Ho-1 interaction in propofol post-conditioning protection against myocardial ischemia reperfusion injury. (Doctoral Dissertation). University of Hong Kong. Retrieved from Mao, X. [毛晓雯]. (2013). Peroxynitrite/Ho-1 interaction in propofol post-conditioning protection against myocardial ischemia reperfusion injury. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5108641 ; http://dx.doi.org/10.5353/th_b5108641 ; http://hdl.handle.net/10722/193463

Chicago Manual of Style (16th Edition):

Mao, Xiaowen. “Peroxynitrite/Ho-1 interaction in propofol post-conditioning protection against myocardial ischemia reperfusion injury.” 2013. Doctoral Dissertation, University of Hong Kong. Accessed December 15, 2019. Mao, X. [毛晓雯]. (2013). Peroxynitrite/Ho-1 interaction in propofol post-conditioning protection against myocardial ischemia reperfusion injury. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5108641 ; http://dx.doi.org/10.5353/th_b5108641 ; http://hdl.handle.net/10722/193463.

MLA Handbook (7th Edition):

Mao, Xiaowen. “Peroxynitrite/Ho-1 interaction in propofol post-conditioning protection against myocardial ischemia reperfusion injury.” 2013. Web. 15 Dec 2019.

Vancouver:

Mao X. Peroxynitrite/Ho-1 interaction in propofol post-conditioning protection against myocardial ischemia reperfusion injury. [Internet] [Doctoral dissertation]. University of Hong Kong; 2013. [cited 2019 Dec 15]. Available from: Mao, X. [毛晓雯]. (2013). Peroxynitrite/Ho-1 interaction in propofol post-conditioning protection against myocardial ischemia reperfusion injury. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5108641 ; http://dx.doi.org/10.5353/th_b5108641 ; http://hdl.handle.net/10722/193463.

Council of Science Editors:

Mao X. Peroxynitrite/Ho-1 interaction in propofol post-conditioning protection against myocardial ischemia reperfusion injury. [Doctoral Dissertation]. University of Hong Kong; 2013. Available from: Mao, X. [毛晓雯]. (2013). Peroxynitrite/Ho-1 interaction in propofol post-conditioning protection against myocardial ischemia reperfusion injury. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5108641 ; http://dx.doi.org/10.5353/th_b5108641 ; http://hdl.handle.net/10722/193463


University of Hong Kong

2. Bai, Bo. Post-translational modification of SIRT1 during endothelial senescence and vascular aging : molecular mechanisms and pathophysiological implications.

Degree: PhD, 2013, University of Hong Kong

 Endothelial senescence represents one of the major characteristics of vascular aging contributing to the development of cardiovascular diseases. SIRT1 is a NAD+-dependent enzyme catalyzing the… (more)

Subjects/Keywords: Vascular endothelium - Aging; Sirtuins

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Bai, B. (2013). Post-translational modification of SIRT1 during endothelial senescence and vascular aging : molecular mechanisms and pathophysiological implications. (Doctoral Dissertation). University of Hong Kong. Retrieved from Bai, B. [白波]. (2013). Post-translational modification of SIRT1 during endothelial senescence and vascular aging : molecular mechanisms and pathophysiological implications. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5177330 ; http://dx.doi.org/10.5353/th_b5177330 ; http://hdl.handle.net/10722/196472

Chicago Manual of Style (16th Edition):

Bai, Bo. “Post-translational modification of SIRT1 during endothelial senescence and vascular aging : molecular mechanisms and pathophysiological implications.” 2013. Doctoral Dissertation, University of Hong Kong. Accessed December 15, 2019. Bai, B. [白波]. (2013). Post-translational modification of SIRT1 during endothelial senescence and vascular aging : molecular mechanisms and pathophysiological implications. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5177330 ; http://dx.doi.org/10.5353/th_b5177330 ; http://hdl.handle.net/10722/196472.

MLA Handbook (7th Edition):

Bai, Bo. “Post-translational modification of SIRT1 during endothelial senescence and vascular aging : molecular mechanisms and pathophysiological implications.” 2013. Web. 15 Dec 2019.

Vancouver:

Bai B. Post-translational modification of SIRT1 during endothelial senescence and vascular aging : molecular mechanisms and pathophysiological implications. [Internet] [Doctoral dissertation]. University of Hong Kong; 2013. [cited 2019 Dec 15]. Available from: Bai, B. [白波]. (2013). Post-translational modification of SIRT1 during endothelial senescence and vascular aging : molecular mechanisms and pathophysiological implications. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5177330 ; http://dx.doi.org/10.5353/th_b5177330 ; http://hdl.handle.net/10722/196472.

Council of Science Editors:

Bai B. Post-translational modification of SIRT1 during endothelial senescence and vascular aging : molecular mechanisms and pathophysiological implications. [Doctoral Dissertation]. University of Hong Kong; 2013. Available from: Bai, B. [白波]. (2013). Post-translational modification of SIRT1 during endothelial senescence and vascular aging : molecular mechanisms and pathophysiological implications. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5177330 ; http://dx.doi.org/10.5353/th_b5177330 ; http://hdl.handle.net/10722/196472


University of Hong Kong

3. Zou, Qian. Hypertension favors the endothelial non-neuronal cholinergic system.

Degree: PhD, 2013, University of Hong Kong

 This thesis investigates the involvement of the non-neuronal cholinergic system in endothelium-dependent relaxations and the impact of hypertension on the function of this system. In… (more)

Subjects/Keywords: Hypertension; Endothelium; Cholinergic mechanisms

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Zou, Q. (2013). Hypertension favors the endothelial non-neuronal cholinergic system. (Doctoral Dissertation). University of Hong Kong. Retrieved from Zou, Q. [鄒倩]. (2013). Hypertension favors the endothelial non-neuronal cholinergic system. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5177329 ; http://dx.doi.org/10.5353/th_b5177329 ; http://hdl.handle.net/10722/196476

Chicago Manual of Style (16th Edition):

Zou, Qian. “Hypertension favors the endothelial non-neuronal cholinergic system.” 2013. Doctoral Dissertation, University of Hong Kong. Accessed December 15, 2019. Zou, Q. [鄒倩]. (2013). Hypertension favors the endothelial non-neuronal cholinergic system. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5177329 ; http://dx.doi.org/10.5353/th_b5177329 ; http://hdl.handle.net/10722/196476.

MLA Handbook (7th Edition):

Zou, Qian. “Hypertension favors the endothelial non-neuronal cholinergic system.” 2013. Web. 15 Dec 2019.

Vancouver:

Zou Q. Hypertension favors the endothelial non-neuronal cholinergic system. [Internet] [Doctoral dissertation]. University of Hong Kong; 2013. [cited 2019 Dec 15]. Available from: Zou, Q. [鄒倩]. (2013). Hypertension favors the endothelial non-neuronal cholinergic system. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5177329 ; http://dx.doi.org/10.5353/th_b5177329 ; http://hdl.handle.net/10722/196476.

Council of Science Editors:

Zou Q. Hypertension favors the endothelial non-neuronal cholinergic system. [Doctoral Dissertation]. University of Hong Kong; 2013. Available from: Zou, Q. [鄒倩]. (2013). Hypertension favors the endothelial non-neuronal cholinergic system. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5177329 ; http://dx.doi.org/10.5353/th_b5177329 ; http://hdl.handle.net/10722/196476


University of Hong Kong

4. Li, Jie. Over-expression of human SIRT1 prevents ageing-induced endothelial dysfunction : eNOS dependent and independent mechanisms.

Degree: M. Phil., 2013, University of Hong Kong

In blood vessel of mature animal, endothelial cells remain quiescent for years, before apoptosis and being replaced by newly generated endothelial cells. During aging, this… (more)

Subjects/Keywords: Vascular endothelium - Aging; Sirtuins

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Li, J. (2013). Over-expression of human SIRT1 prevents ageing-induced endothelial dysfunction : eNOS dependent and independent mechanisms. (Masters Thesis). University of Hong Kong. Retrieved from Li, J. [李杰]. (2013). Over-expression of human SIRT1 prevents ageing-induced endothelial dysfunction : eNOS dependent and independent mechanisms. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5194769 ; http://dx.doi.org/10.5353/th_b5194769 ; http://hdl.handle.net/10722/197537

Chicago Manual of Style (16th Edition):

Li, Jie. “Over-expression of human SIRT1 prevents ageing-induced endothelial dysfunction : eNOS dependent and independent mechanisms.” 2013. Masters Thesis, University of Hong Kong. Accessed December 15, 2019. Li, J. [李杰]. (2013). Over-expression of human SIRT1 prevents ageing-induced endothelial dysfunction : eNOS dependent and independent mechanisms. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5194769 ; http://dx.doi.org/10.5353/th_b5194769 ; http://hdl.handle.net/10722/197537.

MLA Handbook (7th Edition):

Li, Jie. “Over-expression of human SIRT1 prevents ageing-induced endothelial dysfunction : eNOS dependent and independent mechanisms.” 2013. Web. 15 Dec 2019.

Vancouver:

Li J. Over-expression of human SIRT1 prevents ageing-induced endothelial dysfunction : eNOS dependent and independent mechanisms. [Internet] [Masters thesis]. University of Hong Kong; 2013. [cited 2019 Dec 15]. Available from: Li, J. [李杰]. (2013). Over-expression of human SIRT1 prevents ageing-induced endothelial dysfunction : eNOS dependent and independent mechanisms. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5194769 ; http://dx.doi.org/10.5353/th_b5194769 ; http://hdl.handle.net/10722/197537.

Council of Science Editors:

Li J. Over-expression of human SIRT1 prevents ageing-induced endothelial dysfunction : eNOS dependent and independent mechanisms. [Masters Thesis]. University of Hong Kong; 2013. Available from: Li, J. [李杰]. (2013). Over-expression of human SIRT1 prevents ageing-induced endothelial dysfunction : eNOS dependent and independent mechanisms. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5194769 ; http://dx.doi.org/10.5353/th_b5194769 ; http://hdl.handle.net/10722/197537


University of Hong Kong

5. 徐承; Xu, Cheng. Role of adipose SIRT1 in regulating systemic energy metabolism.

Degree: PhD, 2013, University of Hong Kong

SIRT1 (sirtuin 1), a mammalian ortholog of the longevity regulator yeast Sir2p, elicits diversified functions by mediating NAD+ -dependent deacetylation of protein targets. SIRT1 contributes… (more)

Subjects/Keywords: Sirtuins.; Energy metabolism.

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

徐承; Xu, C. (2013). Role of adipose SIRT1 in regulating systemic energy metabolism. (Doctoral Dissertation). University of Hong Kong. Retrieved from Xu, C. [徐承]. (2013). Role of adipose SIRT1 in regulating systemic energy metabolism. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5016268 ; http://dx.doi.org/10.5353/th_b5016268 ; http://hdl.handle.net/10722/183054

Chicago Manual of Style (16th Edition):

徐承; Xu, Cheng. “Role of adipose SIRT1 in regulating systemic energy metabolism.” 2013. Doctoral Dissertation, University of Hong Kong. Accessed December 15, 2019. Xu, C. [徐承]. (2013). Role of adipose SIRT1 in regulating systemic energy metabolism. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5016268 ; http://dx.doi.org/10.5353/th_b5016268 ; http://hdl.handle.net/10722/183054.

MLA Handbook (7th Edition):

徐承; Xu, Cheng. “Role of adipose SIRT1 in regulating systemic energy metabolism.” 2013. Web. 15 Dec 2019.

Vancouver:

徐承; Xu C. Role of adipose SIRT1 in regulating systemic energy metabolism. [Internet] [Doctoral dissertation]. University of Hong Kong; 2013. [cited 2019 Dec 15]. Available from: Xu, C. [徐承]. (2013). Role of adipose SIRT1 in regulating systemic energy metabolism. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5016268 ; http://dx.doi.org/10.5353/th_b5016268 ; http://hdl.handle.net/10722/183054.

Council of Science Editors:

徐承; Xu C. Role of adipose SIRT1 in regulating systemic energy metabolism. [Doctoral Dissertation]. University of Hong Kong; 2013. Available from: Xu, C. [徐承]. (2013). Role of adipose SIRT1 in regulating systemic energy metabolism. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5016268 ; http://dx.doi.org/10.5353/th_b5016268 ; http://hdl.handle.net/10722/183054


University of Hong Kong

6. Liang, Yan. Endothelial LKB1/AMPK signaling pathway in regulating energy and vascular homeostasis.

Degree: PhD, 2013, University of Hong Kong

Liver kinase B1 (LKB1), a serine/threonine kinase, is responsible for the activation of AMP-activated protein kinase (AMPK), the master regulator of energy metabolism. LKB1/AMPK signaling… (more)

Subjects/Keywords: Homeostasis; Protein kinases; Blood-vessels - Physiology; Cellular signal transduction

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Liang, Y. (2013). Endothelial LKB1/AMPK signaling pathway in regulating energy and vascular homeostasis. (Doctoral Dissertation). University of Hong Kong. Retrieved from Liang, Y. [梁艳]. (2013). Endothelial LKB1/AMPK signaling pathway in regulating energy and vascular homeostasis. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5108678 ; http://dx.doi.org/10.5353/th_b5108678 ; http://hdl.handle.net/10722/193460

Chicago Manual of Style (16th Edition):

Liang, Yan. “Endothelial LKB1/AMPK signaling pathway in regulating energy and vascular homeostasis.” 2013. Doctoral Dissertation, University of Hong Kong. Accessed December 15, 2019. Liang, Y. [梁艳]. (2013). Endothelial LKB1/AMPK signaling pathway in regulating energy and vascular homeostasis. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5108678 ; http://dx.doi.org/10.5353/th_b5108678 ; http://hdl.handle.net/10722/193460.

MLA Handbook (7th Edition):

Liang, Yan. “Endothelial LKB1/AMPK signaling pathway in regulating energy and vascular homeostasis.” 2013. Web. 15 Dec 2019.

Vancouver:

Liang Y. Endothelial LKB1/AMPK signaling pathway in regulating energy and vascular homeostasis. [Internet] [Doctoral dissertation]. University of Hong Kong; 2013. [cited 2019 Dec 15]. Available from: Liang, Y. [梁艳]. (2013). Endothelial LKB1/AMPK signaling pathway in regulating energy and vascular homeostasis. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5108678 ; http://dx.doi.org/10.5353/th_b5108678 ; http://hdl.handle.net/10722/193460.

Council of Science Editors:

Liang Y. Endothelial LKB1/AMPK signaling pathway in regulating energy and vascular homeostasis. [Doctoral Dissertation]. University of Hong Kong; 2013. Available from: Liang, Y. [梁艳]. (2013). Endothelial LKB1/AMPK signaling pathway in regulating energy and vascular homeostasis. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5108678 ; http://dx.doi.org/10.5353/th_b5108678 ; http://hdl.handle.net/10722/193460


University of Hong Kong

7. Zhao, Yingzi. Acute and chronic impact of pressure on vascular responsiveness.

Degree: PhD, 2014, University of Hong Kong

 Hypertension leads to vascular complications including endothelial dysfunction, heart failure and stroke. The purpose of the present studies was to investigate the chronic and acute… (more)

Subjects/Keywords: Blood-vessels; Hypertension

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Zhao, Y. (2014). Acute and chronic impact of pressure on vascular responsiveness. (Doctoral Dissertation). University of Hong Kong. Retrieved from Zhao, Y. [趙瑩子]. (2014). Acute and chronic impact of pressure on vascular responsiveness. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5334859 ; http://dx.doi.org/10.5353/th_b5334859 ; http://hdl.handle.net/10722/207185

Chicago Manual of Style (16th Edition):

Zhao, Yingzi. “Acute and chronic impact of pressure on vascular responsiveness.” 2014. Doctoral Dissertation, University of Hong Kong. Accessed December 15, 2019. Zhao, Y. [趙瑩子]. (2014). Acute and chronic impact of pressure on vascular responsiveness. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5334859 ; http://dx.doi.org/10.5353/th_b5334859 ; http://hdl.handle.net/10722/207185.

MLA Handbook (7th Edition):

Zhao, Yingzi. “Acute and chronic impact of pressure on vascular responsiveness.” 2014. Web. 15 Dec 2019.

Vancouver:

Zhao Y. Acute and chronic impact of pressure on vascular responsiveness. [Internet] [Doctoral dissertation]. University of Hong Kong; 2014. [cited 2019 Dec 15]. Available from: Zhao, Y. [趙瑩子]. (2014). Acute and chronic impact of pressure on vascular responsiveness. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5334859 ; http://dx.doi.org/10.5353/th_b5334859 ; http://hdl.handle.net/10722/207185.

Council of Science Editors:

Zhao Y. Acute and chronic impact of pressure on vascular responsiveness. [Doctoral Dissertation]. University of Hong Kong; 2014. Available from: Zhao, Y. [趙瑩子]. (2014). Acute and chronic impact of pressure on vascular responsiveness. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5334859 ; http://dx.doi.org/10.5353/th_b5334859 ; http://hdl.handle.net/10722/207185


University of Hong Kong

8. 李卓明; Li, Zhuoming. Heme oxygenase-1 and endothelial dysfunction in the spontaneously hypertensive rat.

Degree: PhD, 2012, University of Hong Kong

 The endothelium is important for the regulation of vascular tone. In diseases like hypertension, the endothelial cells become dysfunctional. This dysfunction is characterized by nitric… (more)

Subjects/Keywords: Heme oxygenase.; Rats as laboratory animals.; Hypertension - Animal models.; Endothelium.

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

李卓明; Li, Z. (2012). Heme oxygenase-1 and endothelial dysfunction in the spontaneously hypertensive rat. (Doctoral Dissertation). University of Hong Kong. Retrieved from Li, Z. [李卓明]. (2012). Heme oxygenase-1 and endothelial dysfunction in the spontaneously hypertensive rat. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4852173 ; http://dx.doi.org/10.5353/th_b4852173 ; http://hdl.handle.net/10722/179982

Chicago Manual of Style (16th Edition):

李卓明; Li, Zhuoming. “Heme oxygenase-1 and endothelial dysfunction in the spontaneously hypertensive rat.” 2012. Doctoral Dissertation, University of Hong Kong. Accessed December 15, 2019. Li, Z. [李卓明]. (2012). Heme oxygenase-1 and endothelial dysfunction in the spontaneously hypertensive rat. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4852173 ; http://dx.doi.org/10.5353/th_b4852173 ; http://hdl.handle.net/10722/179982.

MLA Handbook (7th Edition):

李卓明; Li, Zhuoming. “Heme oxygenase-1 and endothelial dysfunction in the spontaneously hypertensive rat.” 2012. Web. 15 Dec 2019.

Vancouver:

李卓明; Li Z. Heme oxygenase-1 and endothelial dysfunction in the spontaneously hypertensive rat. [Internet] [Doctoral dissertation]. University of Hong Kong; 2012. [cited 2019 Dec 15]. Available from: Li, Z. [李卓明]. (2012). Heme oxygenase-1 and endothelial dysfunction in the spontaneously hypertensive rat. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4852173 ; http://dx.doi.org/10.5353/th_b4852173 ; http://hdl.handle.net/10722/179982.

Council of Science Editors:

李卓明; Li Z. Heme oxygenase-1 and endothelial dysfunction in the spontaneously hypertensive rat. [Doctoral Dissertation]. University of Hong Kong; 2012. Available from: Li, Z. [李卓明]. (2012). Heme oxygenase-1 and endothelial dysfunction in the spontaneously hypertensive rat. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4852173 ; http://dx.doi.org/10.5353/th_b4852173 ; http://hdl.handle.net/10722/179982


University of Hong Kong

9. Yang, Bo. Role of lipocalin-2 in cardiac dysfunction associated with aging and dietary obesity.

Degree: PhD, 2012, University of Hong Kong

 Obesity is closely related to many medical complications such as type 2 diabetes, hypertension and heart failure. Obesity and other factors, including elevated blood glucose… (more)

Subjects/Keywords: Obesity - Complications.; Cardiovascular system - Diseases.; Aging.; Glycoproteins.

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Yang, B. (2012). Role of lipocalin-2 in cardiac dysfunction associated with aging and dietary obesity. (Doctoral Dissertation). University of Hong Kong. Retrieved from Yang, B. [杨波]. (2012). Role of lipocalin-2 in cardiac dysfunction associated with aging and dietary obesity. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4786964 ; http://dx.doi.org/10.5353/th_b4786964 ; http://hdl.handle.net/10722/161524

Chicago Manual of Style (16th Edition):

Yang, Bo. “Role of lipocalin-2 in cardiac dysfunction associated with aging and dietary obesity.” 2012. Doctoral Dissertation, University of Hong Kong. Accessed December 15, 2019. Yang, B. [杨波]. (2012). Role of lipocalin-2 in cardiac dysfunction associated with aging and dietary obesity. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4786964 ; http://dx.doi.org/10.5353/th_b4786964 ; http://hdl.handle.net/10722/161524.

MLA Handbook (7th Edition):

Yang, Bo. “Role of lipocalin-2 in cardiac dysfunction associated with aging and dietary obesity.” 2012. Web. 15 Dec 2019.

Vancouver:

Yang B. Role of lipocalin-2 in cardiac dysfunction associated with aging and dietary obesity. [Internet] [Doctoral dissertation]. University of Hong Kong; 2012. [cited 2019 Dec 15]. Available from: Yang, B. [杨波]. (2012). Role of lipocalin-2 in cardiac dysfunction associated with aging and dietary obesity. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4786964 ; http://dx.doi.org/10.5353/th_b4786964 ; http://hdl.handle.net/10722/161524.

Council of Science Editors:

Yang B. Role of lipocalin-2 in cardiac dysfunction associated with aging and dietary obesity. [Doctoral Dissertation]. University of Hong Kong; 2012. Available from: Yang, B. [杨波]. (2012). Role of lipocalin-2 in cardiac dysfunction associated with aging and dietary obesity. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4786964 ; http://dx.doi.org/10.5353/th_b4786964 ; http://hdl.handle.net/10722/161524


University of Hong Kong

10. Wong, Yue-ling. The role of adipocyte fatty acid binding protein in the pathogenesis of non-alcoholic fatty liver disease.

Degree: M. Phil., 2010, University of Hong Kong

published_or_final_version

Medicine

Master

Master of Philosophy

Advisors/Committee Members: Vanhoutte, PMGR, Xu, A.

Subjects/Keywords: Fatty liver - Pathogenesis.; Fatty acid-binding proteins.

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Wong, Y. (2010). The role of adipocyte fatty acid binding protein in the pathogenesis of non-alcoholic fatty liver disease. (Masters Thesis). University of Hong Kong. Retrieved from Wong, Y. [黃愉鈴]. (2010). The role of adipocyte fatty acid binding protein in the pathogenesis of non-alcoholic fatty liver disease. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4516487 ; http://dx.doi.org/10.5353/th_b4516487 ; http://hdl.handle.net/10722/131774

Chicago Manual of Style (16th Edition):

Wong, Yue-ling. “The role of adipocyte fatty acid binding protein in the pathogenesis of non-alcoholic fatty liver disease.” 2010. Masters Thesis, University of Hong Kong. Accessed December 15, 2019. Wong, Y. [黃愉鈴]. (2010). The role of adipocyte fatty acid binding protein in the pathogenesis of non-alcoholic fatty liver disease. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4516487 ; http://dx.doi.org/10.5353/th_b4516487 ; http://hdl.handle.net/10722/131774.

MLA Handbook (7th Edition):

Wong, Yue-ling. “The role of adipocyte fatty acid binding protein in the pathogenesis of non-alcoholic fatty liver disease.” 2010. Web. 15 Dec 2019.

Vancouver:

Wong Y. The role of adipocyte fatty acid binding protein in the pathogenesis of non-alcoholic fatty liver disease. [Internet] [Masters thesis]. University of Hong Kong; 2010. [cited 2019 Dec 15]. Available from: Wong, Y. [黃愉鈴]. (2010). The role of adipocyte fatty acid binding protein in the pathogenesis of non-alcoholic fatty liver disease. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4516487 ; http://dx.doi.org/10.5353/th_b4516487 ; http://hdl.handle.net/10722/131774.

Council of Science Editors:

Wong Y. The role of adipocyte fatty acid binding protein in the pathogenesis of non-alcoholic fatty liver disease. [Masters Thesis]. University of Hong Kong; 2010. Available from: Wong, Y. [黃愉鈴]. (2010). The role of adipocyte fatty acid binding protein in the pathogenesis of non-alcoholic fatty liver disease. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4516487 ; http://dx.doi.org/10.5353/th_b4516487 ; http://hdl.handle.net/10722/131774


University of Hong Kong

11. Lee, Yuk-kwan, Mary. Molecular changes in regenerated and senescent cultured endothelial cells.

Degree: PhD, 2006, University of Hong Kong

abstract

published_or_final_version

Pharmacology

Doctoral

Doctor of Philosophy

Advisors/Committee Members: Man, RYK, Vanhoutte, PMGR.

Subjects/Keywords: Endothelium.; Cytogenetics.; Atherosclerosis - Animal models.; Atherosclerosis - Molecular aspects.

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Lee, Yuk-kwan, M. (2006). Molecular changes in regenerated and senescent cultured endothelial cells. (Doctoral Dissertation). University of Hong Kong. Retrieved from Lee, Y. M. [李玉筠]. (2006). Molecular changes in regenerated and senescent cultured endothelial cells. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b3876551 ; http://dx.doi.org/10.5353/th_b3876551 ; http://hdl.handle.net/10722/52313

Chicago Manual of Style (16th Edition):

Lee, Yuk-kwan, Mary. “Molecular changes in regenerated and senescent cultured endothelial cells.” 2006. Doctoral Dissertation, University of Hong Kong. Accessed December 15, 2019. Lee, Y. M. [李玉筠]. (2006). Molecular changes in regenerated and senescent cultured endothelial cells. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b3876551 ; http://dx.doi.org/10.5353/th_b3876551 ; http://hdl.handle.net/10722/52313.

MLA Handbook (7th Edition):

Lee, Yuk-kwan, Mary. “Molecular changes in regenerated and senescent cultured endothelial cells.” 2006. Web. 15 Dec 2019.

Vancouver:

Lee, Yuk-kwan M. Molecular changes in regenerated and senescent cultured endothelial cells. [Internet] [Doctoral dissertation]. University of Hong Kong; 2006. [cited 2019 Dec 15]. Available from: Lee, Y. M. [李玉筠]. (2006). Molecular changes in regenerated and senescent cultured endothelial cells. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b3876551 ; http://dx.doi.org/10.5353/th_b3876551 ; http://hdl.handle.net/10722/52313.

Council of Science Editors:

Lee, Yuk-kwan M. Molecular changes in regenerated and senescent cultured endothelial cells. [Doctoral Dissertation]. University of Hong Kong; 2006. Available from: Lee, Y. M. [李玉筠]. (2006). Molecular changes in regenerated and senescent cultured endothelial cells. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b3876551 ; http://dx.doi.org/10.5353/th_b3876551 ; http://hdl.handle.net/10722/52313


University of Hong Kong

12. Shi, Yi. Endothelial dysfunction and changes in vascular smooth muscle responsiveness in femoral arteries of rats with type I diabetes.

Degree: PhD, 2006, University of Hong Kong

published_or_final_version

abstract

Pharmacology

Doctoral

Doctor of Philosophy

Advisors/Committee Members: Vanhoutte, PMGR, Man, RYK.

Subjects/Keywords: Smooth muscle - Physiology.; Endothelium.; Smooth muscle - Effect of drugs on.; Diabetes - Complications - Animal models.

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Shi, Y. (2006). Endothelial dysfunction and changes in vascular smooth muscle responsiveness in femoral arteries of rats with type I diabetes. (Doctoral Dissertation). University of Hong Kong. Retrieved from Shi, Y. [史懿]. (2006). Endothelial dysfunction and changes in vascular smooth muscle responsiveness in femoral arteries of rats with type I diabetes. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b3828607 ; http://dx.doi.org/10.5353/th_b3828607 ; http://hdl.handle.net/10722/52322

Chicago Manual of Style (16th Edition):

Shi, Yi. “Endothelial dysfunction and changes in vascular smooth muscle responsiveness in femoral arteries of rats with type I diabetes.” 2006. Doctoral Dissertation, University of Hong Kong. Accessed December 15, 2019. Shi, Y. [史懿]. (2006). Endothelial dysfunction and changes in vascular smooth muscle responsiveness in femoral arteries of rats with type I diabetes. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b3828607 ; http://dx.doi.org/10.5353/th_b3828607 ; http://hdl.handle.net/10722/52322.

MLA Handbook (7th Edition):

Shi, Yi. “Endothelial dysfunction and changes in vascular smooth muscle responsiveness in femoral arteries of rats with type I diabetes.” 2006. Web. 15 Dec 2019.

Vancouver:

Shi Y. Endothelial dysfunction and changes in vascular smooth muscle responsiveness in femoral arteries of rats with type I diabetes. [Internet] [Doctoral dissertation]. University of Hong Kong; 2006. [cited 2019 Dec 15]. Available from: Shi, Y. [史懿]. (2006). Endothelial dysfunction and changes in vascular smooth muscle responsiveness in femoral arteries of rats with type I diabetes. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b3828607 ; http://dx.doi.org/10.5353/th_b3828607 ; http://hdl.handle.net/10722/52322.

Council of Science Editors:

Shi Y. Endothelial dysfunction and changes in vascular smooth muscle responsiveness in femoral arteries of rats with type I diabetes. [Doctoral Dissertation]. University of Hong Kong; 2006. Available from: Shi, Y. [史懿]. (2006). Endothelial dysfunction and changes in vascular smooth muscle responsiveness in femoral arteries of rats with type I diabetes. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b3828607 ; http://dx.doi.org/10.5353/th_b3828607 ; http://hdl.handle.net/10722/52322


University of Hong Kong

13. Li, Huiying. Adipocyte fatty acid-binding protein: a link between inflammation and vascular dysfunction.

Degree: M. Phil., 2010, University of Hong Kong

published_or_final_version

Pharmacology and Pharmacy

Master

Master of Philosophy

Advisors/Committee Members: Xu, A, Vanhoutte, PMGR.

Subjects/Keywords: Vascular endothelium.; Fatty acid-binding proteins.

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Li, H. (2010). Adipocyte fatty acid-binding protein: a link between inflammation and vascular dysfunction. (Masters Thesis). University of Hong Kong. Retrieved from Li, H. [李慧颖]. (2010). Adipocyte fatty acid-binding protein : a link between inflammation and vascular dysfunction. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4424871 ; http://dx.doi.org/10.5353/th_b4424871 ; http://hdl.handle.net/10722/133169

Chicago Manual of Style (16th Edition):

Li, Huiying. “Adipocyte fatty acid-binding protein: a link between inflammation and vascular dysfunction.” 2010. Masters Thesis, University of Hong Kong. Accessed December 15, 2019. Li, H. [李慧颖]. (2010). Adipocyte fatty acid-binding protein : a link between inflammation and vascular dysfunction. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4424871 ; http://dx.doi.org/10.5353/th_b4424871 ; http://hdl.handle.net/10722/133169.

MLA Handbook (7th Edition):

Li, Huiying. “Adipocyte fatty acid-binding protein: a link between inflammation and vascular dysfunction.” 2010. Web. 15 Dec 2019.

Vancouver:

Li H. Adipocyte fatty acid-binding protein: a link between inflammation and vascular dysfunction. [Internet] [Masters thesis]. University of Hong Kong; 2010. [cited 2019 Dec 15]. Available from: Li, H. [李慧颖]. (2010). Adipocyte fatty acid-binding protein : a link between inflammation and vascular dysfunction. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4424871 ; http://dx.doi.org/10.5353/th_b4424871 ; http://hdl.handle.net/10722/133169.

Council of Science Editors:

Li H. Adipocyte fatty acid-binding protein: a link between inflammation and vascular dysfunction. [Masters Thesis]. University of Hong Kong; 2010. Available from: Li, H. [李慧颖]. (2010). Adipocyte fatty acid-binding protein : a link between inflammation and vascular dysfunction. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4424871 ; http://dx.doi.org/10.5353/th_b4424871 ; http://hdl.handle.net/10722/133169


University of Hong Kong

14. Wong, Sze-ka. Vascular effects of vitamin D3 on endothelium-dependent contractions in SHR aorta.

Degree: PhD, 2010, University of Hong Kong

published_or_final_version

Pharmacology and Pharmacy

Doctoral

Doctor of Philosophy

Advisors/Committee Members: Man, RYK, Vanhoutte, PMGR.

Subjects/Keywords: Blood-vessels - Contraction.; Vascular endothelium.; Vitamin D.; Aorta.; Hypertension - Animal models.; Rats - Physiology.

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Wong, S. (2010). Vascular effects of vitamin D3 on endothelium-dependent contractions in SHR aorta. (Doctoral Dissertation). University of Hong Kong. Retrieved from Wong, S. [黃思伽]. (2010). Vascular effects of vitamin D3 on endothelium-dependent contractions in SHR aorta. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4587171 ; http://dx.doi.org/10.5353/th_b4587171 ; http://hdl.handle.net/10722/134016

Chicago Manual of Style (16th Edition):

Wong, Sze-ka. “Vascular effects of vitamin D3 on endothelium-dependent contractions in SHR aorta.” 2010. Doctoral Dissertation, University of Hong Kong. Accessed December 15, 2019. Wong, S. [黃思伽]. (2010). Vascular effects of vitamin D3 on endothelium-dependent contractions in SHR aorta. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4587171 ; http://dx.doi.org/10.5353/th_b4587171 ; http://hdl.handle.net/10722/134016.

MLA Handbook (7th Edition):

Wong, Sze-ka. “Vascular effects of vitamin D3 on endothelium-dependent contractions in SHR aorta.” 2010. Web. 15 Dec 2019.

Vancouver:

Wong S. Vascular effects of vitamin D3 on endothelium-dependent contractions in SHR aorta. [Internet] [Doctoral dissertation]. University of Hong Kong; 2010. [cited 2019 Dec 15]. Available from: Wong, S. [黃思伽]. (2010). Vascular effects of vitamin D3 on endothelium-dependent contractions in SHR aorta. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4587171 ; http://dx.doi.org/10.5353/th_b4587171 ; http://hdl.handle.net/10722/134016.

Council of Science Editors:

Wong S. Vascular effects of vitamin D3 on endothelium-dependent contractions in SHR aorta. [Doctoral Dissertation]. University of Hong Kong; 2010. Available from: Wong, S. [黃思伽]. (2010). Vascular effects of vitamin D3 on endothelium-dependent contractions in SHR aorta. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4587171 ; http://dx.doi.org/10.5353/th_b4587171 ; http://hdl.handle.net/10722/134016


University of Hong Kong

15. Liang, Chaofan. Toll-like receptor 4 plays a key role in insulin resistance and endothelial dysfunction.

Degree: PhD, 2011, University of Hong Kong

published_or_final_version

Pharmacology and Pharmacy

Doctoral

Doctor of Philosophy

Advisors/Committee Members: Vanhoutte, PMGR, Xu, A.

Subjects/Keywords: Cell receptors.; Insulin resistance.; Endothelium.

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Liang, C. (2011). Toll-like receptor 4 plays a key role in insulin resistance and endothelial dysfunction. (Doctoral Dissertation). University of Hong Kong. Retrieved from Liang, C. [梁超凡]. (2011). Toll-like receptor 4 plays a key role in insulin resistance and endothelial dysfunction. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4702892 ; http://dx.doi.org/10.5353/th_b4702892 ; http://hdl.handle.net/10722/144792

Chicago Manual of Style (16th Edition):

Liang, Chaofan. “Toll-like receptor 4 plays a key role in insulin resistance and endothelial dysfunction.” 2011. Doctoral Dissertation, University of Hong Kong. Accessed December 15, 2019. Liang, C. [梁超凡]. (2011). Toll-like receptor 4 plays a key role in insulin resistance and endothelial dysfunction. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4702892 ; http://dx.doi.org/10.5353/th_b4702892 ; http://hdl.handle.net/10722/144792.

MLA Handbook (7th Edition):

Liang, Chaofan. “Toll-like receptor 4 plays a key role in insulin resistance and endothelial dysfunction.” 2011. Web. 15 Dec 2019.

Vancouver:

Liang C. Toll-like receptor 4 plays a key role in insulin resistance and endothelial dysfunction. [Internet] [Doctoral dissertation]. University of Hong Kong; 2011. [cited 2019 Dec 15]. Available from: Liang, C. [梁超凡]. (2011). Toll-like receptor 4 plays a key role in insulin resistance and endothelial dysfunction. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4702892 ; http://dx.doi.org/10.5353/th_b4702892 ; http://hdl.handle.net/10722/144792.

Council of Science Editors:

Liang C. Toll-like receptor 4 plays a key role in insulin resistance and endothelial dysfunction. [Doctoral Dissertation]. University of Hong Kong; 2011. Available from: Liang, C. [梁超凡]. (2011). Toll-like receptor 4 plays a key role in insulin resistance and endothelial dysfunction. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4702892 ; http://dx.doi.org/10.5353/th_b4702892 ; http://hdl.handle.net/10722/144792


University of Hong Kong

16. 陳翹昕.; Chan, Kiu-yan, Calvin. Challenges to arterial endothelial function.

Degree: PhD, 2011, University of Hong Kong

published_or_final_version

Pharmacology and Pharmacy

Doctoral

Doctor of Philosophy

Advisors/Committee Members: Vanhoutte, PMGR, Mak, JCW.

Subjects/Keywords: Vascular endothelium.; Arteries.; Blood-vessels - Contraction.

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

陳翹昕.; Chan, Kiu-yan, C. (2011). Challenges to arterial endothelial function. (Doctoral Dissertation). University of Hong Kong. Retrieved from Chan, K. C. [陳翹昕]. (2011). Challenges to arterial endothelial function. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4691658 ; http://dx.doi.org/10.5353/th_b4691658 ; http://hdl.handle.net/10722/144151

Chicago Manual of Style (16th Edition):

陳翹昕.; Chan, Kiu-yan, Calvin. “Challenges to arterial endothelial function.” 2011. Doctoral Dissertation, University of Hong Kong. Accessed December 15, 2019. Chan, K. C. [陳翹昕]. (2011). Challenges to arterial endothelial function. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4691658 ; http://dx.doi.org/10.5353/th_b4691658 ; http://hdl.handle.net/10722/144151.

MLA Handbook (7th Edition):

陳翹昕.; Chan, Kiu-yan, Calvin. “Challenges to arterial endothelial function.” 2011. Web. 15 Dec 2019.

Vancouver:

陳翹昕.; Chan, Kiu-yan C. Challenges to arterial endothelial function. [Internet] [Doctoral dissertation]. University of Hong Kong; 2011. [cited 2019 Dec 15]. Available from: Chan, K. C. [陳翹昕]. (2011). Challenges to arterial endothelial function. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4691658 ; http://dx.doi.org/10.5353/th_b4691658 ; http://hdl.handle.net/10722/144151.

Council of Science Editors:

陳翹昕.; Chan, Kiu-yan C. Challenges to arterial endothelial function. [Doctoral Dissertation]. University of Hong Kong; 2011. Available from: Chan, K. C. [陳翹昕]. (2011). Challenges to arterial endothelial function. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4691658 ; http://dx.doi.org/10.5353/th_b4691658 ; http://hdl.handle.net/10722/144151


University of Hong Kong

17. Qu, Chen. Modulation of endothelium-dependent contractions by chronic inhibitionof nitric oxide synthase in the rat aorta.

Degree: M. Phil., 2008, University of Hong Kong

published_or_final_version

Pharmacology

Master

Master of Philosophy

Advisors/Committee Members: Vanhoutte, PMGR, Leung, SWS, Man, RYK.

Subjects/Keywords: Cyclooxygenases.; Nitric-oxide synthase.; Vascular endothelium.; Blood-vessels - Contraction.; Arteries.

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Qu, C. (2008). Modulation of endothelium-dependent contractions by chronic inhibitionof nitric oxide synthase in the rat aorta. (Masters Thesis). University of Hong Kong. Retrieved from Qu, C. [屈晨]. (2008). Modulation of endothelium-dependent contractions by chronic inhibition of nitric oxide synthase in the rat aorta. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4129083 ; http://dx.doi.org/10.5353/th_b4129083 ; http://hdl.handle.net/10722/52314

Chicago Manual of Style (16th Edition):

Qu, Chen. “Modulation of endothelium-dependent contractions by chronic inhibitionof nitric oxide synthase in the rat aorta.” 2008. Masters Thesis, University of Hong Kong. Accessed December 15, 2019. Qu, C. [屈晨]. (2008). Modulation of endothelium-dependent contractions by chronic inhibition of nitric oxide synthase in the rat aorta. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4129083 ; http://dx.doi.org/10.5353/th_b4129083 ; http://hdl.handle.net/10722/52314.

MLA Handbook (7th Edition):

Qu, Chen. “Modulation of endothelium-dependent contractions by chronic inhibitionof nitric oxide synthase in the rat aorta.” 2008. Web. 15 Dec 2019.

Vancouver:

Qu C. Modulation of endothelium-dependent contractions by chronic inhibitionof nitric oxide synthase in the rat aorta. [Internet] [Masters thesis]. University of Hong Kong; 2008. [cited 2019 Dec 15]. Available from: Qu, C. [屈晨]. (2008). Modulation of endothelium-dependent contractions by chronic inhibition of nitric oxide synthase in the rat aorta. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4129083 ; http://dx.doi.org/10.5353/th_b4129083 ; http://hdl.handle.net/10722/52314.

Council of Science Editors:

Qu C. Modulation of endothelium-dependent contractions by chronic inhibitionof nitric oxide synthase in the rat aorta. [Masters Thesis]. University of Hong Kong; 2008. Available from: Qu, C. [屈晨]. (2008). Modulation of endothelium-dependent contractions by chronic inhibition of nitric oxide synthase in the rat aorta. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4129083 ; http://dx.doi.org/10.5353/th_b4129083 ; http://hdl.handle.net/10722/52314


University of Hong Kong

18. Ng, Wai-hung, William. Modulation of vascular function by genistein through cAMP-PKA signaling cascade in porcine coronary artery.

Degree: M. Phil., 2006, University of Hong Kong

published_or_final_version

abstract

Pharmacology

Master

Master of Philosophy

Advisors/Committee Members: Vanhoutte, PMGR, Leung, GPH, Man, RYK.

Subjects/Keywords: Nitric oxide.; Isoflavones.; Coronary arteries

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Ng, Wai-hung, W. (2006). Modulation of vascular function by genistein through cAMP-PKA signaling cascade in porcine coronary artery. (Masters Thesis). University of Hong Kong. Retrieved from Ng, W. W. [伍偉鴻]. (2006). Modulation of vascular function by genistein through cAMP-PKA signaling cascade in porcine coronary artery. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b3719125 ; http://dx.doi.org/10.5353/th_b3719125 ; http://hdl.handle.net/10722/52318

Chicago Manual of Style (16th Edition):

Ng, Wai-hung, William. “Modulation of vascular function by genistein through cAMP-PKA signaling cascade in porcine coronary artery.” 2006. Masters Thesis, University of Hong Kong. Accessed December 15, 2019. Ng, W. W. [伍偉鴻]. (2006). Modulation of vascular function by genistein through cAMP-PKA signaling cascade in porcine coronary artery. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b3719125 ; http://dx.doi.org/10.5353/th_b3719125 ; http://hdl.handle.net/10722/52318.

MLA Handbook (7th Edition):

Ng, Wai-hung, William. “Modulation of vascular function by genistein through cAMP-PKA signaling cascade in porcine coronary artery.” 2006. Web. 15 Dec 2019.

Vancouver:

Ng, Wai-hung W. Modulation of vascular function by genistein through cAMP-PKA signaling cascade in porcine coronary artery. [Internet] [Masters thesis]. University of Hong Kong; 2006. [cited 2019 Dec 15]. Available from: Ng, W. W. [伍偉鴻]. (2006). Modulation of vascular function by genistein through cAMP-PKA signaling cascade in porcine coronary artery. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b3719125 ; http://dx.doi.org/10.5353/th_b3719125 ; http://hdl.handle.net/10722/52318.

Council of Science Editors:

Ng, Wai-hung W. Modulation of vascular function by genistein through cAMP-PKA signaling cascade in porcine coronary artery. [Masters Thesis]. University of Hong Kong; 2006. Available from: Ng, W. W. [伍偉鴻]. (2006). Modulation of vascular function by genistein through cAMP-PKA signaling cascade in porcine coronary artery. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b3719125 ; http://dx.doi.org/10.5353/th_b3719125 ; http://hdl.handle.net/10722/52318


University of Hong Kong

19. Wong, Sze-wan, Emily. Vascular effects of the intravenous anaesthetic dexmedetomidine.

Degree: PhD, 2011, University of Hong Kong

published_or_final_version

Pharmacology and Pharmacy

Doctoral

Doctor of Philosophy

Advisors/Committee Members: Ng, JKF, Vanhoutte, PMGR, Man, RYK.

Subjects/Keywords: Intravenous anesthesia.; Adrenergic alpha blockers.; Sedatives - Therapeutic use.; Analgesics - Therapeutic use.

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Wong, Sze-wan, E. (2011). Vascular effects of the intravenous anaesthetic dexmedetomidine. (Doctoral Dissertation). University of Hong Kong. Retrieved from Wong, S. E. [黃詩韻]. (2011). Vascular effects of the intravenous anaesthetic dexmedetomidine. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4692561 ; http://dx.doi.org/10.5353/th_b4692561 ; http://hdl.handle.net/10722/144159

Chicago Manual of Style (16th Edition):

Wong, Sze-wan, Emily. “Vascular effects of the intravenous anaesthetic dexmedetomidine.” 2011. Doctoral Dissertation, University of Hong Kong. Accessed December 15, 2019. Wong, S. E. [黃詩韻]. (2011). Vascular effects of the intravenous anaesthetic dexmedetomidine. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4692561 ; http://dx.doi.org/10.5353/th_b4692561 ; http://hdl.handle.net/10722/144159.

MLA Handbook (7th Edition):

Wong, Sze-wan, Emily. “Vascular effects of the intravenous anaesthetic dexmedetomidine.” 2011. Web. 15 Dec 2019.

Vancouver:

Wong, Sze-wan E. Vascular effects of the intravenous anaesthetic dexmedetomidine. [Internet] [Doctoral dissertation]. University of Hong Kong; 2011. [cited 2019 Dec 15]. Available from: Wong, S. E. [黃詩韻]. (2011). Vascular effects of the intravenous anaesthetic dexmedetomidine. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4692561 ; http://dx.doi.org/10.5353/th_b4692561 ; http://hdl.handle.net/10722/144159.

Council of Science Editors:

Wong, Sze-wan E. Vascular effects of the intravenous anaesthetic dexmedetomidine. [Doctoral Dissertation]. University of Hong Kong; 2011. Available from: Wong, S. E. [黃詩韻]. (2011). Vascular effects of the intravenous anaesthetic dexmedetomidine. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4692561 ; http://dx.doi.org/10.5353/th_b4692561 ; http://hdl.handle.net/10722/144159


University of Hong Kong

20. Sit, Sai-man. Potential protective effect of ergothioneine on endothelial function.

Degree: M. Phil., 2011, University of Hong Kong

published_or_final_version

Pharmacology and Pharmacy

Master

Master of Philosophy

Advisors/Committee Members: Leung, GPH, Vanhoutte, PMGR, Man, RYK.

Subjects/Keywords: Antioxidants.; Sulphur amino acids.; Vascular endothelium.

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Sit, S. (2011). Potential protective effect of ergothioneine on endothelial function. (Masters Thesis). University of Hong Kong. Retrieved from Sit, S. [薛世文]. (2011). Potential protective effect of ergothioneine on endothelial function. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4694178 ; http://dx.doi.org/10.5353/th_b4694178 ; http://hdl.handle.net/10722/144173

Chicago Manual of Style (16th Edition):

Sit, Sai-man. “Potential protective effect of ergothioneine on endothelial function.” 2011. Masters Thesis, University of Hong Kong. Accessed December 15, 2019. Sit, S. [薛世文]. (2011). Potential protective effect of ergothioneine on endothelial function. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4694178 ; http://dx.doi.org/10.5353/th_b4694178 ; http://hdl.handle.net/10722/144173.

MLA Handbook (7th Edition):

Sit, Sai-man. “Potential protective effect of ergothioneine on endothelial function.” 2011. Web. 15 Dec 2019.

Vancouver:

Sit S. Potential protective effect of ergothioneine on endothelial function. [Internet] [Masters thesis]. University of Hong Kong; 2011. [cited 2019 Dec 15]. Available from: Sit, S. [薛世文]. (2011). Potential protective effect of ergothioneine on endothelial function. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4694178 ; http://dx.doi.org/10.5353/th_b4694178 ; http://hdl.handle.net/10722/144173.

Council of Science Editors:

Sit S. Potential protective effect of ergothioneine on endothelial function. [Masters Thesis]. University of Hong Kong; 2011. Available from: Sit, S. [薛世文]. (2011). Potential protective effect of ergothioneine on endothelial function. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4694178 ; http://dx.doi.org/10.5353/th_b4694178 ; http://hdl.handle.net/10722/144173

.