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You searched for +publisher:"University of Florida" +contributor:("Dunn, William A."). Showing records 1 – 16 of 16 total matches.

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University of Florida

1. Nicks, Jessica Renee. Therapeutic Strategies for Treating Neuromuscular Deficits in Murine Models of Peripheral Neuropathy.

Degree: PhD, Medical Sciences - Neuroscience (IDP), 2013, University of Florida

 Hereditary demyelinating neuropathies affect 1:2500 people, and currently there is no effective treatment.  One of the most common neuropathies, Charcot-Marie-Tooth diseases (CMT) is modeled by… (more)

Subjects/Keywords: Antibodies; Diseases; Dosage; Innervation; Muscles; Myelin; Myelin proteins; Nerves; Peripheral nervous system diseases; Schwann cells; myelin  – neuromuscular  – peripheral

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APA (6th Edition):

Nicks, J. R. (2013). Therapeutic Strategies for Treating Neuromuscular Deficits in Murine Models of Peripheral Neuropathy. (Doctoral Dissertation). University of Florida. Retrieved from http://ufdc.ufl.edu/UFE0046177

Chicago Manual of Style (16th Edition):

Nicks, Jessica Renee. “Therapeutic Strategies for Treating Neuromuscular Deficits in Murine Models of Peripheral Neuropathy.” 2013. Doctoral Dissertation, University of Florida. Accessed September 20, 2019. http://ufdc.ufl.edu/UFE0046177.

MLA Handbook (7th Edition):

Nicks, Jessica Renee. “Therapeutic Strategies for Treating Neuromuscular Deficits in Murine Models of Peripheral Neuropathy.” 2013. Web. 20 Sep 2019.

Vancouver:

Nicks JR. Therapeutic Strategies for Treating Neuromuscular Deficits in Murine Models of Peripheral Neuropathy. [Internet] [Doctoral dissertation]. University of Florida; 2013. [cited 2019 Sep 20]. Available from: http://ufdc.ufl.edu/UFE0046177.

Council of Science Editors:

Nicks JR. Therapeutic Strategies for Treating Neuromuscular Deficits in Murine Models of Peripheral Neuropathy. [Doctoral Dissertation]. University of Florida; 2013. Available from: http://ufdc.ufl.edu/UFE0046177


University of Florida

2. Biel, Thomas G. Mitofusin 2 Mediates Sirtuin 1 Induced Autophagy to Suppress Liver Ischemia/Reperfusion Injury.

Degree: PhD, Medical Sciences - Physiology and Pharmacology (IDP), 2014, University of Florida

 Hepatic ischemia/reperfusion (I/R) injury causes organ damage that can lead to liver failure and mortality after resection and transplantation surgeries. Currently, there are no therapeutic… (more)

Subjects/Keywords: Acetylation; Cell death; Cytoprotection; Death; Hepatocytes; Ischemia; Liver; Mitochondria; Physical trauma; Reperfusion; autophagy  – hepatocytes  – ischemia  – liver  – mitofusin2  – reperfusion  – resection  – sirtuin1

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APA (6th Edition):

Biel, T. G. (2014). Mitofusin 2 Mediates Sirtuin 1 Induced Autophagy to Suppress Liver Ischemia/Reperfusion Injury. (Doctoral Dissertation). University of Florida. Retrieved from http://ufdc.ufl.edu/UFE0047316

Chicago Manual of Style (16th Edition):

Biel, Thomas G. “Mitofusin 2 Mediates Sirtuin 1 Induced Autophagy to Suppress Liver Ischemia/Reperfusion Injury.” 2014. Doctoral Dissertation, University of Florida. Accessed September 20, 2019. http://ufdc.ufl.edu/UFE0047316.

MLA Handbook (7th Edition):

Biel, Thomas G. “Mitofusin 2 Mediates Sirtuin 1 Induced Autophagy to Suppress Liver Ischemia/Reperfusion Injury.” 2014. Web. 20 Sep 2019.

Vancouver:

Biel TG. Mitofusin 2 Mediates Sirtuin 1 Induced Autophagy to Suppress Liver Ischemia/Reperfusion Injury. [Internet] [Doctoral dissertation]. University of Florida; 2014. [cited 2019 Sep 20]. Available from: http://ufdc.ufl.edu/UFE0047316.

Council of Science Editors:

Biel TG. Mitofusin 2 Mediates Sirtuin 1 Induced Autophagy to Suppress Liver Ischemia/Reperfusion Injury. [Doctoral Dissertation]. University of Florida; 2014. Available from: http://ufdc.ufl.edu/UFE0047316


University of Florida

3. Karch, Celeste. Mechanisms of Superoxide Dismutase 1 Aggregate Formation in Familial Amyotrophic Lateral Sclerosis.

Degree: PhD, Medical Sciences - Neuroscience (IDP), 2009, University of Florida

 Familial amyotrophic lateral sclerosis (FALS) is a late onset neurodegenerative disease that is characterized by selective death of neurons in the upper and lower motor… (more)

Subjects/Keywords: Aggregation; Amino acids; Amyotrophic lateral sclerosis; Cell aggregates; Crystallins; Diseases; Disulfides; Genetic mutation; Spinal cord; Superoxides; amyotrophic, disease, dismustase, lateral, misfolding, models, mouse, neurodegenerative, protein, sclerosis, superoxide

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APA (6th Edition):

Karch, C. (2009). Mechanisms of Superoxide Dismutase 1 Aggregate Formation in Familial Amyotrophic Lateral Sclerosis. (Doctoral Dissertation). University of Florida. Retrieved from http://ufdc.ufl.edu/UFE0024366

Chicago Manual of Style (16th Edition):

Karch, Celeste. “Mechanisms of Superoxide Dismutase 1 Aggregate Formation in Familial Amyotrophic Lateral Sclerosis.” 2009. Doctoral Dissertation, University of Florida. Accessed September 20, 2019. http://ufdc.ufl.edu/UFE0024366.

MLA Handbook (7th Edition):

Karch, Celeste. “Mechanisms of Superoxide Dismutase 1 Aggregate Formation in Familial Amyotrophic Lateral Sclerosis.” 2009. Web. 20 Sep 2019.

Vancouver:

Karch C. Mechanisms of Superoxide Dismutase 1 Aggregate Formation in Familial Amyotrophic Lateral Sclerosis. [Internet] [Doctoral dissertation]. University of Florida; 2009. [cited 2019 Sep 20]. Available from: http://ufdc.ufl.edu/UFE0024366.

Council of Science Editors:

Karch C. Mechanisms of Superoxide Dismutase 1 Aggregate Formation in Familial Amyotrophic Lateral Sclerosis. [Doctoral Dissertation]. University of Florida; 2009. Available from: http://ufdc.ufl.edu/UFE0024366


University of Florida

4. Salganik, Maxim. Phenotype of Mutants in pH-Sensitive Regions of the AAV Capsid and Evidence for a pH-Induced Capsid Protease.

Degree: PhD, Medical Sciences - Immunology and Microbiology (IDP), 2012, University of Florida

 Adeno-Associated Virus (AAV) is a small, non-pathogenic parvovirus that hasgained popularity as a gene therapy vector in clinical trials and a gene transfer tool inresearch.… (more)

Subjects/Keywords: Amino acids; Capsid; DNA; Genomes; Infections; pH; Proteins; Receptors; String quartets; Virology; aav  – protease  – virus

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APA (6th Edition):

Salganik, M. (2012). Phenotype of Mutants in pH-Sensitive Regions of the AAV Capsid and Evidence for a pH-Induced Capsid Protease. (Doctoral Dissertation). University of Florida. Retrieved from http://ufdc.ufl.edu/UFE0044946

Chicago Manual of Style (16th Edition):

Salganik, Maxim. “Phenotype of Mutants in pH-Sensitive Regions of the AAV Capsid and Evidence for a pH-Induced Capsid Protease.” 2012. Doctoral Dissertation, University of Florida. Accessed September 20, 2019. http://ufdc.ufl.edu/UFE0044946.

MLA Handbook (7th Edition):

Salganik, Maxim. “Phenotype of Mutants in pH-Sensitive Regions of the AAV Capsid and Evidence for a pH-Induced Capsid Protease.” 2012. Web. 20 Sep 2019.

Vancouver:

Salganik M. Phenotype of Mutants in pH-Sensitive Regions of the AAV Capsid and Evidence for a pH-Induced Capsid Protease. [Internet] [Doctoral dissertation]. University of Florida; 2012. [cited 2019 Sep 20]. Available from: http://ufdc.ufl.edu/UFE0044946.

Council of Science Editors:

Salganik M. Phenotype of Mutants in pH-Sensitive Regions of the AAV Capsid and Evidence for a pH-Induced Capsid Protease. [Doctoral Dissertation]. University of Florida; 2012. Available from: http://ufdc.ufl.edu/UFE0044946


University of Florida

5. Chastain-Gross, Ryan Paul. The Role of the Bacterial Gene Pg0717 in Periodontitis.

Degree: PhD, Medical Sciences - Immunology and Microbiology (IDP), 2015, University of Florida

 Periodontitis is a class of diseases that cause oral destruction in both adults and children. Porphyromonas gingivalis is an oral bacterium that is strongly associated… (more)

Subjects/Keywords: Bacteria; Diseases; Endosomes; Infections; Periodontal diseases; Periodontitis; Porphyromonas gingivalis; Proteins; Receptors; Species; association  – clinical  – gingivalis  – homologues  – invasion  – periodontitis  – porphyromonas  – protein  – trafficking  – virulence

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APA (6th Edition):

Chastain-Gross, R. P. (2015). The Role of the Bacterial Gene Pg0717 in Periodontitis. (Doctoral Dissertation). University of Florida. Retrieved from http://ufdc.ufl.edu/UFE0047502

Chicago Manual of Style (16th Edition):

Chastain-Gross, Ryan Paul. “The Role of the Bacterial Gene Pg0717 in Periodontitis.” 2015. Doctoral Dissertation, University of Florida. Accessed September 20, 2019. http://ufdc.ufl.edu/UFE0047502.

MLA Handbook (7th Edition):

Chastain-Gross, Ryan Paul. “The Role of the Bacterial Gene Pg0717 in Periodontitis.” 2015. Web. 20 Sep 2019.

Vancouver:

Chastain-Gross RP. The Role of the Bacterial Gene Pg0717 in Periodontitis. [Internet] [Doctoral dissertation]. University of Florida; 2015. [cited 2019 Sep 20]. Available from: http://ufdc.ufl.edu/UFE0047502.

Council of Science Editors:

Chastain-Gross RP. The Role of the Bacterial Gene Pg0717 in Periodontitis. [Doctoral Dissertation]. University of Florida; 2015. Available from: http://ufdc.ufl.edu/UFE0047502


University of Florida

6. Rangaraju, Sunitha. Quality Control Pathways in the Peripheral Nerve with Disease and Aging.

Degree: PhD, Medical Sciences - Neuroscience (IDP), 2010, University of Florida

 QUALITY CONTROL PATHWAYS IN THE PERIPHERAL NERVE WITH DISEASE AND AGING By Sunitha Rangaraju May 2010 Chair: Lucia Notterpek Major: Medical Sciences?Neuroscience Peripheral myelin protein… (more)

Subjects/Keywords: Antibodies; Cell aggregates; Diseases; Myelin; Myelin proteins; Myelination; Nerves; Peripheral nerves; Rats; T tests; aging, autophagy, chaperones, myelin, oxidative, peripheral, quality, schwann

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APA (6th Edition):

Rangaraju, S. (2010). Quality Control Pathways in the Peripheral Nerve with Disease and Aging. (Doctoral Dissertation). University of Florida. Retrieved from http://ufdc.ufl.edu/UFE0041506

Chicago Manual of Style (16th Edition):

Rangaraju, Sunitha. “Quality Control Pathways in the Peripheral Nerve with Disease and Aging.” 2010. Doctoral Dissertation, University of Florida. Accessed September 20, 2019. http://ufdc.ufl.edu/UFE0041506.

MLA Handbook (7th Edition):

Rangaraju, Sunitha. “Quality Control Pathways in the Peripheral Nerve with Disease and Aging.” 2010. Web. 20 Sep 2019.

Vancouver:

Rangaraju S. Quality Control Pathways in the Peripheral Nerve with Disease and Aging. [Internet] [Doctoral dissertation]. University of Florida; 2010. [cited 2019 Sep 20]. Available from: http://ufdc.ufl.edu/UFE0041506.

Council of Science Editors:

Rangaraju S. Quality Control Pathways in the Peripheral Nerve with Disease and Aging. [Doctoral Dissertation]. University of Florida; 2010. Available from: http://ufdc.ufl.edu/UFE0041506


University of Florida

7. Prudencio-Alvarez, Mercedes. Implication of Detergent-Insoluble Aggregates of Superoxide Dismutase 1 in Familial Amyotrophic Lateral Sclerosis.

Degree: PhD, Medical Sciences - Neuroscience (IDP), 2009, University of Florida

 IMPLICATION OF DETERGENT-INSOLUBLE AGGREGATES OF SUPEROXIDE DISMUTASE 1 IN FAMILIAL AMYOTROPHIC LATERAL SCLEROSIS Mutations in superoxide dismutase 1 (SOD1) are responsible for 10-20% of the… (more)

Subjects/Keywords: Aggregation; Amyotrophic lateral sclerosis; Cell aggregates; Detergents; Diseases; Genetic mutation; Inclusion bodies; Mice; Mutant proteins; Superoxides; aggregation, amyotrophic, misfolding, motorneuron, neurodegeneration, sclerosis, transgenics

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APA (6th Edition):

Prudencio-Alvarez, M. (2009). Implication of Detergent-Insoluble Aggregates of Superoxide Dismutase 1 in Familial Amyotrophic Lateral Sclerosis. (Doctoral Dissertation). University of Florida. Retrieved from http://ufdc.ufl.edu/UFE0025156

Chicago Manual of Style (16th Edition):

Prudencio-Alvarez, Mercedes. “Implication of Detergent-Insoluble Aggregates of Superoxide Dismutase 1 in Familial Amyotrophic Lateral Sclerosis.” 2009. Doctoral Dissertation, University of Florida. Accessed September 20, 2019. http://ufdc.ufl.edu/UFE0025156.

MLA Handbook (7th Edition):

Prudencio-Alvarez, Mercedes. “Implication of Detergent-Insoluble Aggregates of Superoxide Dismutase 1 in Familial Amyotrophic Lateral Sclerosis.” 2009. Web. 20 Sep 2019.

Vancouver:

Prudencio-Alvarez M. Implication of Detergent-Insoluble Aggregates of Superoxide Dismutase 1 in Familial Amyotrophic Lateral Sclerosis. [Internet] [Doctoral dissertation]. University of Florida; 2009. [cited 2019 Sep 20]. Available from: http://ufdc.ufl.edu/UFE0025156.

Council of Science Editors:

Prudencio-Alvarez M. Implication of Detergent-Insoluble Aggregates of Superoxide Dismutase 1 in Familial Amyotrophic Lateral Sclerosis. [Doctoral Dissertation]. University of Florida; 2009. Available from: http://ufdc.ufl.edu/UFE0025156

8. Barrett, Amanda Gene. Pg0553 Plays a Significant Role in Por Secretion and Gingipains Processing in Porphyromonas Gingivalis Strain W83.

Degree: PhD, Medical Sciences - Immunology and Microbiology (IDP), 2013, University of Florida

 Porphyromonas gingivalis (Pg) is a periodontal pathogen that has been detected in atherosclerotic plaque and has been shown to invade and persist within human coronary… (more)

Subjects/Keywords: Atherosclerosis; Diseases; Endothelial cells; Infections; Pathogens; Periodontal diseases; Porphyromonas gingivalis; Proteins; Secretion; Virulence; gingipains  – pg0553  – porphyromonas  – porss

…Dissertation Presented to the Graduate School of the University of Florida in Partial Fulfillment of… 

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APA (6th Edition):

Barrett, A. G. (2013). Pg0553 Plays a Significant Role in Por Secretion and Gingipains Processing in Porphyromonas Gingivalis Strain W83. (Doctoral Dissertation). University of Florida. Retrieved from http://ufdc.ufl.edu/UFE0045196

Chicago Manual of Style (16th Edition):

Barrett, Amanda Gene. “Pg0553 Plays a Significant Role in Por Secretion and Gingipains Processing in Porphyromonas Gingivalis Strain W83.” 2013. Doctoral Dissertation, University of Florida. Accessed September 20, 2019. http://ufdc.ufl.edu/UFE0045196.

MLA Handbook (7th Edition):

Barrett, Amanda Gene. “Pg0553 Plays a Significant Role in Por Secretion and Gingipains Processing in Porphyromonas Gingivalis Strain W83.” 2013. Web. 20 Sep 2019.

Vancouver:

Barrett AG. Pg0553 Plays a Significant Role in Por Secretion and Gingipains Processing in Porphyromonas Gingivalis Strain W83. [Internet] [Doctoral dissertation]. University of Florida; 2013. [cited 2019 Sep 20]. Available from: http://ufdc.ufl.edu/UFE0045196.

Council of Science Editors:

Barrett AG. Pg0553 Plays a Significant Role in Por Secretion and Gingipains Processing in Porphyromonas Gingivalis Strain W83. [Doctoral Dissertation]. University of Florida; 2013. Available from: http://ufdc.ufl.edu/UFE0045196


University of Florida

9. Schroder, Laura Aaron. Characterizing the Assembly of the Membranes that Sequester Peroxisomes during Autophagy.

Degree: PhD, Medical Sciences - Molecular Cell Biology (IDP), 2007, University of Florida

Subjects/Keywords: Amino acids; Cells; Genomics; Jury sequestration; Nitrogen; Peroxisomes; Pichia; Proteins; Vacuoles; Yeasts; atg, autophagosome, autophagy, degradation, lysosome, macropexophagy, micropexophagy, mipa, peroxisome, pexophagosome, pexophagy, vacuole, yeast

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APA (6th Edition):

Schroder, L. A. (2007). Characterizing the Assembly of the Membranes that Sequester Peroxisomes during Autophagy. (Doctoral Dissertation). University of Florida. Retrieved from http://ufdc.ufl.edu/UFE0021725

Chicago Manual of Style (16th Edition):

Schroder, Laura Aaron. “Characterizing the Assembly of the Membranes that Sequester Peroxisomes during Autophagy.” 2007. Doctoral Dissertation, University of Florida. Accessed September 20, 2019. http://ufdc.ufl.edu/UFE0021725.

MLA Handbook (7th Edition):

Schroder, Laura Aaron. “Characterizing the Assembly of the Membranes that Sequester Peroxisomes during Autophagy.” 2007. Web. 20 Sep 2019.

Vancouver:

Schroder LA. Characterizing the Assembly of the Membranes that Sequester Peroxisomes during Autophagy. [Internet] [Doctoral dissertation]. University of Florida; 2007. [cited 2019 Sep 20]. Available from: http://ufdc.ufl.edu/UFE0021725.

Council of Science Editors:

Schroder LA. Characterizing the Assembly of the Membranes that Sequester Peroxisomes during Autophagy. [Doctoral Dissertation]. University of Florida; 2007. Available from: http://ufdc.ufl.edu/UFE0021725


University of Florida

10. Xiao, Carl. Development of Novel Therapies for Liver Disease Associated with Misfolded Z Alpha-1-Antitrypsin Accumulation and Disposal.

Degree: PhD, Genetics and Genomics, 2012, University of Florida

Subjects/Keywords: Antibodies; Cell lines; Gene therapy; Hepatocytes; Liver; Plasmids; Polymerization; Polymers; Secretion; Transfection; alpha1antitrypsin; erad; gene-therapy; liver-disease; proteasome; scfv; upr

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APA (6th Edition):

Xiao, C. (2012). Development of Novel Therapies for Liver Disease Associated with Misfolded Z Alpha-1-Antitrypsin Accumulation and Disposal. (Doctoral Dissertation). University of Florida. Retrieved from http://ufdc.ufl.edu/UFE0044879

Chicago Manual of Style (16th Edition):

Xiao, Carl. “Development of Novel Therapies for Liver Disease Associated with Misfolded Z Alpha-1-Antitrypsin Accumulation and Disposal.” 2012. Doctoral Dissertation, University of Florida. Accessed September 20, 2019. http://ufdc.ufl.edu/UFE0044879.

MLA Handbook (7th Edition):

Xiao, Carl. “Development of Novel Therapies for Liver Disease Associated with Misfolded Z Alpha-1-Antitrypsin Accumulation and Disposal.” 2012. Web. 20 Sep 2019.

Vancouver:

Xiao C. Development of Novel Therapies for Liver Disease Associated with Misfolded Z Alpha-1-Antitrypsin Accumulation and Disposal. [Internet] [Doctoral dissertation]. University of Florida; 2012. [cited 2019 Sep 20]. Available from: http://ufdc.ufl.edu/UFE0044879.

Council of Science Editors:

Xiao C. Development of Novel Therapies for Liver Disease Associated with Misfolded Z Alpha-1-Antitrypsin Accumulation and Disposal. [Doctoral Dissertation]. University of Florida; 2012. Available from: http://ufdc.ufl.edu/UFE0044879


University of Florida

11. Sadasivan, Shankar. Role of Autophagy in Neuronal Injury Models of the Central Nervous System.

Degree: PhD, Medical Sciences - Neuroscience (IDP), 2008, University of Florida

 Autophagy is an intracellular phenonmenon which is induced under conditions of stress. It is characterized by the presence of double membrane vesicles in the cytoplasm,… (more)

Subjects/Keywords: Amino acids; Antibodies; Apoptosis; Brain; Cell death; Death; Neurons; Physical trauma; Starvation; Traumatic brain injury; apoptosis, autophagic, autophagy, brain, excitotoxicity

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APA (6th Edition):

Sadasivan, S. (2008). Role of Autophagy in Neuronal Injury Models of the Central Nervous System. (Doctoral Dissertation). University of Florida. Retrieved from http://ufdc.ufl.edu/UFE0022094

Chicago Manual of Style (16th Edition):

Sadasivan, Shankar. “Role of Autophagy in Neuronal Injury Models of the Central Nervous System.” 2008. Doctoral Dissertation, University of Florida. Accessed September 20, 2019. http://ufdc.ufl.edu/UFE0022094.

MLA Handbook (7th Edition):

Sadasivan, Shankar. “Role of Autophagy in Neuronal Injury Models of the Central Nervous System.” 2008. Web. 20 Sep 2019.

Vancouver:

Sadasivan S. Role of Autophagy in Neuronal Injury Models of the Central Nervous System. [Internet] [Doctoral dissertation]. University of Florida; 2008. [cited 2019 Sep 20]. Available from: http://ufdc.ufl.edu/UFE0022094.

Council of Science Editors:

Sadasivan S. Role of Autophagy in Neuronal Injury Models of the Central Nervous System. [Doctoral Dissertation]. University of Florida; 2008. Available from: http://ufdc.ufl.edu/UFE0022094


University of Florida

12. Sampson, Edith M. Streptococcus Mutans OMZ175 Invasion of Human Tissues in Culture.

Degree: PhD, Medical Sciences - Immunology and Microbiology (IDP), 2014, University of Florida

Subjects/Keywords: Antibiotics; Apoptosis; Atherosclerosis; Bacteria; Cell growth; Cells; Diseases; Endothelial cells; Infections; Streptococcus mutans; atherosclerosis; cardiovascular; cnm; invasion; streptococcus

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APA (6th Edition):

Sampson, E. M. (2014). Streptococcus Mutans OMZ175 Invasion of Human Tissues in Culture. (Doctoral Dissertation). University of Florida. Retrieved from http://ufdc.ufl.edu/UFE0046195

Chicago Manual of Style (16th Edition):

Sampson, Edith M. “Streptococcus Mutans OMZ175 Invasion of Human Tissues in Culture.” 2014. Doctoral Dissertation, University of Florida. Accessed September 20, 2019. http://ufdc.ufl.edu/UFE0046195.

MLA Handbook (7th Edition):

Sampson, Edith M. “Streptococcus Mutans OMZ175 Invasion of Human Tissues in Culture.” 2014. Web. 20 Sep 2019.

Vancouver:

Sampson EM. Streptococcus Mutans OMZ175 Invasion of Human Tissues in Culture. [Internet] [Doctoral dissertation]. University of Florida; 2014. [cited 2019 Sep 20]. Available from: http://ufdc.ufl.edu/UFE0046195.

Council of Science Editors:

Sampson EM. Streptococcus Mutans OMZ175 Invasion of Human Tissues in Culture. [Doctoral Dissertation]. University of Florida; 2014. Available from: http://ufdc.ufl.edu/UFE0046195


University of Florida

13. Vale Cruz, Dustin Shawn, 1977-. Role of Activating Transcription Factor-2 in Control of Pocket Protein Expression and Regulation of Chondrocyte Growth.

Degree: PhD, Medical Sciences - Molecular Cell Biology (IDP), 2008, University of Florida

 Endochondral ossification is the process of skeletal bone growth through the formation of a cartilage template that subsequently undergoes mineralization to form trabecular bone. Genetic… (more)

Subjects/Keywords: Activating transcription factors; Bones; Cartilage; Cell cycle; Chondrocytes; Collagens; Cyclins; Epiphyses; Neonates; Skeleton; chondrocytes, endorchondral, retinoblastoma, skeleton

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APA (6th Edition):

Vale Cruz, Dustin Shawn, 1. (2008). Role of Activating Transcription Factor-2 in Control of Pocket Protein Expression and Regulation of Chondrocyte Growth. (Doctoral Dissertation). University of Florida. Retrieved from http://ufdc.ufl.edu/UFE0021887

Chicago Manual of Style (16th Edition):

Vale Cruz, Dustin Shawn, 1977-. “Role of Activating Transcription Factor-2 in Control of Pocket Protein Expression and Regulation of Chondrocyte Growth.” 2008. Doctoral Dissertation, University of Florida. Accessed September 20, 2019. http://ufdc.ufl.edu/UFE0021887.

MLA Handbook (7th Edition):

Vale Cruz, Dustin Shawn, 1977-. “Role of Activating Transcription Factor-2 in Control of Pocket Protein Expression and Regulation of Chondrocyte Growth.” 2008. Web. 20 Sep 2019.

Vancouver:

Vale Cruz, Dustin Shawn 1. Role of Activating Transcription Factor-2 in Control of Pocket Protein Expression and Regulation of Chondrocyte Growth. [Internet] [Doctoral dissertation]. University of Florida; 2008. [cited 2019 Sep 20]. Available from: http://ufdc.ufl.edu/UFE0021887.

Council of Science Editors:

Vale Cruz, Dustin Shawn 1. Role of Activating Transcription Factor-2 in Control of Pocket Protein Expression and Regulation of Chondrocyte Growth. [Doctoral Dissertation]. University of Florida; 2008. Available from: http://ufdc.ufl.edu/UFE0021887


University of Florida

14. Thiaville, Michelle. Transcriptional Regulation of the Sodium-Coupled Neutral Amino Acid Transporter (SNAT2) Gene by Amino Acid Availability.

Degree: PhD, Medical Sciences - Biochemistry and Molecular Biology (IDP), 2008, University of Florida

 Limited intake of nutrients, especially amino acids, can alter general growth and development as well as disease states such as diabetes and solid tumor growth.… (more)

Subjects/Keywords: Amino acids; Gene expression; Genes; Hep G2 cells; Messenger RNA; Microelectromechanical systems; Phosphorylation; RNA; Small interfering RNA; Starvation; aare, asns, atf4, cebpbeta, erk, mek, snat2

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APA (6th Edition):

Thiaville, M. (2008). Transcriptional Regulation of the Sodium-Coupled Neutral Amino Acid Transporter (SNAT2) Gene by Amino Acid Availability. (Doctoral Dissertation). University of Florida. Retrieved from http://ufdc.ufl.edu/UFE0022014

Chicago Manual of Style (16th Edition):

Thiaville, Michelle. “Transcriptional Regulation of the Sodium-Coupled Neutral Amino Acid Transporter (SNAT2) Gene by Amino Acid Availability.” 2008. Doctoral Dissertation, University of Florida. Accessed September 20, 2019. http://ufdc.ufl.edu/UFE0022014.

MLA Handbook (7th Edition):

Thiaville, Michelle. “Transcriptional Regulation of the Sodium-Coupled Neutral Amino Acid Transporter (SNAT2) Gene by Amino Acid Availability.” 2008. Web. 20 Sep 2019.

Vancouver:

Thiaville M. Transcriptional Regulation of the Sodium-Coupled Neutral Amino Acid Transporter (SNAT2) Gene by Amino Acid Availability. [Internet] [Doctoral dissertation]. University of Florida; 2008. [cited 2019 Sep 20]. Available from: http://ufdc.ufl.edu/UFE0022014.

Council of Science Editors:

Thiaville M. Transcriptional Regulation of the Sodium-Coupled Neutral Amino Acid Transporter (SNAT2) Gene by Amino Acid Availability. [Doctoral Dissertation]. University of Florida; 2008. Available from: http://ufdc.ufl.edu/UFE0022014


University of Florida

15. Hunt, Darrell. Regulation of Breast Cancer Cell Growth and Survival by the Interaction of Focal Adhesion Kinase and Vascular Endothelial Growth Factor Receptor-3.

Degree: PhD, Medical Sciences - Molecular Cell Biology (IDP), 2008, University of Florida

 Vascular endothelial growth factor receptor-3 (VEGFR-3, flt-4) is a receptor tyrosine that is overexpressed in a variety of human carcinomas, but its role in carcinogenesis… (more)

Subjects/Keywords: Apoptosis; Breast cancer; Cell growth; Cell lines; Cells; Focal adhesions; Ligands; Molecules; Receptors; Tumors; breast, cancer, kinases, vegfr

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Hunt, D. (2008). Regulation of Breast Cancer Cell Growth and Survival by the Interaction of Focal Adhesion Kinase and Vascular Endothelial Growth Factor Receptor-3. (Doctoral Dissertation). University of Florida. Retrieved from http://ufdc.ufl.edu/UFE0022461

Chicago Manual of Style (16th Edition):

Hunt, Darrell. “Regulation of Breast Cancer Cell Growth and Survival by the Interaction of Focal Adhesion Kinase and Vascular Endothelial Growth Factor Receptor-3.” 2008. Doctoral Dissertation, University of Florida. Accessed September 20, 2019. http://ufdc.ufl.edu/UFE0022461.

MLA Handbook (7th Edition):

Hunt, Darrell. “Regulation of Breast Cancer Cell Growth and Survival by the Interaction of Focal Adhesion Kinase and Vascular Endothelial Growth Factor Receptor-3.” 2008. Web. 20 Sep 2019.

Vancouver:

Hunt D. Regulation of Breast Cancer Cell Growth and Survival by the Interaction of Focal Adhesion Kinase and Vascular Endothelial Growth Factor Receptor-3. [Internet] [Doctoral dissertation]. University of Florida; 2008. [cited 2019 Sep 20]. Available from: http://ufdc.ufl.edu/UFE0022461.

Council of Science Editors:

Hunt D. Regulation of Breast Cancer Cell Growth and Survival by the Interaction of Focal Adhesion Kinase and Vascular Endothelial Growth Factor Receptor-3. [Doctoral Dissertation]. University of Florida; 2008. Available from: http://ufdc.ufl.edu/UFE0022461


University of Florida

16. Mccall, Angela Layne. Correction of Autophagic Accumulation in Skeletal Muscle of a Pompe Disease Mouse Model following Gene Therapy Administration.

Degree: PhD, Medical Sciences - Genetics (IDP), 2017, University of Florida

Subjects/Keywords: aav; autophagy; muscle; pompe

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Mccall, A. L. (2017). Correction of Autophagic Accumulation in Skeletal Muscle of a Pompe Disease Mouse Model following Gene Therapy Administration. (Doctoral Dissertation). University of Florida. Retrieved from http://ufdc.ufl.edu/UFE0051039

Chicago Manual of Style (16th Edition):

Mccall, Angela Layne. “Correction of Autophagic Accumulation in Skeletal Muscle of a Pompe Disease Mouse Model following Gene Therapy Administration.” 2017. Doctoral Dissertation, University of Florida. Accessed September 20, 2019. http://ufdc.ufl.edu/UFE0051039.

MLA Handbook (7th Edition):

Mccall, Angela Layne. “Correction of Autophagic Accumulation in Skeletal Muscle of a Pompe Disease Mouse Model following Gene Therapy Administration.” 2017. Web. 20 Sep 2019.

Vancouver:

Mccall AL. Correction of Autophagic Accumulation in Skeletal Muscle of a Pompe Disease Mouse Model following Gene Therapy Administration. [Internet] [Doctoral dissertation]. University of Florida; 2017. [cited 2019 Sep 20]. Available from: http://ufdc.ufl.edu/UFE0051039.

Council of Science Editors:

Mccall AL. Correction of Autophagic Accumulation in Skeletal Muscle of a Pompe Disease Mouse Model following Gene Therapy Administration. [Doctoral Dissertation]. University of Florida; 2017. Available from: http://ufdc.ufl.edu/UFE0051039

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