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You searched for +publisher:"University of Arizona" +contributor:("Zhang, Donna"). Showing records 1 – 27 of 27 total matches.

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University of Arizona

1. Harder, Bryan. Molecular and Chemical Modulation of NRF2 for Disease Intervention .

Degree: 2017, University of Arizona

 Cells are frequently exposed to endogenous and environmental stressors that pose a threat to homeostatic cellular conditions. In response to such stress, the cell activates… (more)

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APA (6th Edition):

Harder, B. (2017). Molecular and Chemical Modulation of NRF2 for Disease Intervention . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/624590

Chicago Manual of Style (16th Edition):

Harder, Bryan. “Molecular and Chemical Modulation of NRF2 for Disease Intervention .” 2017. Doctoral Dissertation, University of Arizona. Accessed January 19, 2020. http://hdl.handle.net/10150/624590.

MLA Handbook (7th Edition):

Harder, Bryan. “Molecular and Chemical Modulation of NRF2 for Disease Intervention .” 2017. Web. 19 Jan 2020.

Vancouver:

Harder B. Molecular and Chemical Modulation of NRF2 for Disease Intervention . [Internet] [Doctoral dissertation]. University of Arizona; 2017. [cited 2020 Jan 19]. Available from: http://hdl.handle.net/10150/624590.

Council of Science Editors:

Harder B. Molecular and Chemical Modulation of NRF2 for Disease Intervention . [Doctoral Dissertation]. University of Arizona; 2017. Available from: http://hdl.handle.net/10150/624590


University of Arizona

2. Rojo de la Vega Guinea, Elisa Montserrat. Characterization of the Molecular Mechanisms of Xenobiotic-Induced NRF2 Activation for Disease Prevention and Intervention .

Degree: 2018, University of Arizona

 The transcription factor nuclear factor erythroid 2-related factor 2 (NRF2) is the master regulator of the cellular antioxidant response upon exposure to xenobiotics. In addition,… (more)

Subjects/Keywords: arsenic; bixin; cancer; NRF2; oxidative stress; ultraviolet radiation

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APA (6th Edition):

Rojo de la Vega Guinea, E. M. (2018). Characterization of the Molecular Mechanisms of Xenobiotic-Induced NRF2 Activation for Disease Prevention and Intervention . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/627744

Chicago Manual of Style (16th Edition):

Rojo de la Vega Guinea, Elisa Montserrat. “Characterization of the Molecular Mechanisms of Xenobiotic-Induced NRF2 Activation for Disease Prevention and Intervention .” 2018. Doctoral Dissertation, University of Arizona. Accessed January 19, 2020. http://hdl.handle.net/10150/627744.

MLA Handbook (7th Edition):

Rojo de la Vega Guinea, Elisa Montserrat. “Characterization of the Molecular Mechanisms of Xenobiotic-Induced NRF2 Activation for Disease Prevention and Intervention .” 2018. Web. 19 Jan 2020.

Vancouver:

Rojo de la Vega Guinea EM. Characterization of the Molecular Mechanisms of Xenobiotic-Induced NRF2 Activation for Disease Prevention and Intervention . [Internet] [Doctoral dissertation]. University of Arizona; 2018. [cited 2020 Jan 19]. Available from: http://hdl.handle.net/10150/627744.

Council of Science Editors:

Rojo de la Vega Guinea EM. Characterization of the Molecular Mechanisms of Xenobiotic-Induced NRF2 Activation for Disease Prevention and Intervention . [Doctoral Dissertation]. University of Arizona; 2018. Available from: http://hdl.handle.net/10150/627744


University of Arizona

3. Shi, Taoda. Synthesis and Biological Evaluation of Protein Tyrosine Phosphatase Inhibitors .

Degree: 2017, University of Arizona

 Protein tyrosine phosphatases (PTP) are a group of enzymes that regulate signal transduction by removing a phosphate group from tyrosine residues of their substrates. Despite… (more)

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APA (6th Edition):

Shi, T. (2017). Synthesis and Biological Evaluation of Protein Tyrosine Phosphatase Inhibitors . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/625892

Chicago Manual of Style (16th Edition):

Shi, Taoda. “Synthesis and Biological Evaluation of Protein Tyrosine Phosphatase Inhibitors .” 2017. Doctoral Dissertation, University of Arizona. Accessed January 19, 2020. http://hdl.handle.net/10150/625892.

MLA Handbook (7th Edition):

Shi, Taoda. “Synthesis and Biological Evaluation of Protein Tyrosine Phosphatase Inhibitors .” 2017. Web. 19 Jan 2020.

Vancouver:

Shi T. Synthesis and Biological Evaluation of Protein Tyrosine Phosphatase Inhibitors . [Internet] [Doctoral dissertation]. University of Arizona; 2017. [cited 2020 Jan 19]. Available from: http://hdl.handle.net/10150/625892.

Council of Science Editors:

Shi T. Synthesis and Biological Evaluation of Protein Tyrosine Phosphatase Inhibitors . [Doctoral Dissertation]. University of Arizona; 2017. Available from: http://hdl.handle.net/10150/625892


University of Arizona

4. Kerins, Michael John. Ironing Out Roles and Regulation of NRF2 in a Hereditary Cancer Syndrome .

Degree: 2019, University of Arizona

 The deadly kidney cancers associated with hereditary leiomyomatosis and renal cell cancer (HLRCC) show activation of the nuclear factor (erythroid 2)-like 2 (NFE2L2, NRF2) transcription… (more)

Subjects/Keywords: Cancer; ferroptosis; HLRCC; iron; NRF2; signaling

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APA (6th Edition):

Kerins, M. J. (2019). Ironing Out Roles and Regulation of NRF2 in a Hereditary Cancer Syndrome . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/633229

Chicago Manual of Style (16th Edition):

Kerins, Michael John. “Ironing Out Roles and Regulation of NRF2 in a Hereditary Cancer Syndrome .” 2019. Doctoral Dissertation, University of Arizona. Accessed January 19, 2020. http://hdl.handle.net/10150/633229.

MLA Handbook (7th Edition):

Kerins, Michael John. “Ironing Out Roles and Regulation of NRF2 in a Hereditary Cancer Syndrome .” 2019. Web. 19 Jan 2020.

Vancouver:

Kerins MJ. Ironing Out Roles and Regulation of NRF2 in a Hereditary Cancer Syndrome . [Internet] [Doctoral dissertation]. University of Arizona; 2019. [cited 2020 Jan 19]. Available from: http://hdl.handle.net/10150/633229.

Council of Science Editors:

Kerins MJ. Ironing Out Roles and Regulation of NRF2 in a Hereditary Cancer Syndrome . [Doctoral Dissertation]. University of Arizona; 2019. Available from: http://hdl.handle.net/10150/633229


University of Arizona

5. Lau, Alexandria G. The Role of the Nrf2-Keap1 Pathway in Autophagy and How it Contributes to Arsenic Carcinogenicity .

Degree: 2012, University of Arizona

 NF-E2-related factor 2 (Nrf2) is a transcription factor that is responsible for maintaining cellular homeostasis by controlling the fate of cells through transcriptional upregulation of… (more)

Subjects/Keywords: autophagy; Keap1; Nrf2; p62; Pharmacology & Toxicology; Antioxidant response; arsenic

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APA (6th Edition):

Lau, A. G. (2012). The Role of the Nrf2-Keap1 Pathway in Autophagy and How it Contributes to Arsenic Carcinogenicity . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/228515

Chicago Manual of Style (16th Edition):

Lau, Alexandria G. “The Role of the Nrf2-Keap1 Pathway in Autophagy and How it Contributes to Arsenic Carcinogenicity .” 2012. Doctoral Dissertation, University of Arizona. Accessed January 19, 2020. http://hdl.handle.net/10150/228515.

MLA Handbook (7th Edition):

Lau, Alexandria G. “The Role of the Nrf2-Keap1 Pathway in Autophagy and How it Contributes to Arsenic Carcinogenicity .” 2012. Web. 19 Jan 2020.

Vancouver:

Lau AG. The Role of the Nrf2-Keap1 Pathway in Autophagy and How it Contributes to Arsenic Carcinogenicity . [Internet] [Doctoral dissertation]. University of Arizona; 2012. [cited 2020 Jan 19]. Available from: http://hdl.handle.net/10150/228515.

Council of Science Editors:

Lau AG. The Role of the Nrf2-Keap1 Pathway in Autophagy and How it Contributes to Arsenic Carcinogenicity . [Doctoral Dissertation]. University of Arizona; 2012. Available from: http://hdl.handle.net/10150/228515


University of Arizona

6. Villeneuve, Nicole Frances. Mechanistic Study of USP15-Dependent Deubiquitination and Characterization of Natural Compounds that Modulate the Nrf2-Keap1 Antioxidant Response .

Degree: 2011, University of Arizona

 Nrf2 (NF-E2-related factor 2) is a transcription factor that regulates a battery of downstream genes that contain an antioxidant response element (ARE) in their promoters,… (more)

Subjects/Keywords: cinnamic aldehyde; Keap1; Nrf2; oridonin; ubiquitination; USP15

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APA (6th Edition):

Villeneuve, N. F. (2011). Mechanistic Study of USP15-Dependent Deubiquitination and Characterization of Natural Compounds that Modulate the Nrf2-Keap1 Antioxidant Response . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/145720

Chicago Manual of Style (16th Edition):

Villeneuve, Nicole Frances. “Mechanistic Study of USP15-Dependent Deubiquitination and Characterization of Natural Compounds that Modulate the Nrf2-Keap1 Antioxidant Response .” 2011. Doctoral Dissertation, University of Arizona. Accessed January 19, 2020. http://hdl.handle.net/10150/145720.

MLA Handbook (7th Edition):

Villeneuve, Nicole Frances. “Mechanistic Study of USP15-Dependent Deubiquitination and Characterization of Natural Compounds that Modulate the Nrf2-Keap1 Antioxidant Response .” 2011. Web. 19 Jan 2020.

Vancouver:

Villeneuve NF. Mechanistic Study of USP15-Dependent Deubiquitination and Characterization of Natural Compounds that Modulate the Nrf2-Keap1 Antioxidant Response . [Internet] [Doctoral dissertation]. University of Arizona; 2011. [cited 2020 Jan 19]. Available from: http://hdl.handle.net/10150/145720.

Council of Science Editors:

Villeneuve NF. Mechanistic Study of USP15-Dependent Deubiquitination and Characterization of Natural Compounds that Modulate the Nrf2-Keap1 Antioxidant Response . [Doctoral Dissertation]. University of Arizona; 2011. Available from: http://hdl.handle.net/10150/145720


University of Arizona

7. Wang, Shue. Probing Single Cell Gene Expression in Tissue Morphogenesis and Angiogenesis .

Degree: 2015, University of Arizona

 The fascinating capability of cellular self-organization during tissue development and repair is a central question in developmental biology and regenerative medicine. Understanding the dynamic morphogenic… (more)

Subjects/Keywords: gene expression; gold nanoparticles; molecular probe; Single cell analysis; Tissue morphogenesis; Mechanical Engineering; Angiogenesis

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APA (6th Edition):

Wang, S. (2015). Probing Single Cell Gene Expression in Tissue Morphogenesis and Angiogenesis . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/560919

Chicago Manual of Style (16th Edition):

Wang, Shue. “Probing Single Cell Gene Expression in Tissue Morphogenesis and Angiogenesis .” 2015. Doctoral Dissertation, University of Arizona. Accessed January 19, 2020. http://hdl.handle.net/10150/560919.

MLA Handbook (7th Edition):

Wang, Shue. “Probing Single Cell Gene Expression in Tissue Morphogenesis and Angiogenesis .” 2015. Web. 19 Jan 2020.

Vancouver:

Wang S. Probing Single Cell Gene Expression in Tissue Morphogenesis and Angiogenesis . [Internet] [Doctoral dissertation]. University of Arizona; 2015. [cited 2020 Jan 19]. Available from: http://hdl.handle.net/10150/560919.

Council of Science Editors:

Wang S. Probing Single Cell Gene Expression in Tissue Morphogenesis and Angiogenesis . [Doctoral Dissertation]. University of Arizona; 2015. Available from: http://hdl.handle.net/10150/560919


University of Arizona

8. Cholanians, Aram B. Dopamine Induced Post-Translational Modifications of α-Synuclein and the Role of Arsenic in the Development of Parkinson's Disease and Other Synucleinopathies .

Degree: 2016, University of Arizona

 Synucleinopathies are a family of neurodegenerative diseases, with the distinctive pathological feature of Lewy bodies, which include Parkinson’s disease. Lewy bodies are intracellular inclusions filled… (more)

Subjects/Keywords: alpha-synuclein; arsenic; dopamine; oligomers; Parkinson's Disease; Pharmacology & Toxicology; aggregates

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APA (6th Edition):

Cholanians, A. B. (2016). Dopamine Induced Post-Translational Modifications of α-Synuclein and the Role of Arsenic in the Development of Parkinson's Disease and Other Synucleinopathies . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/612093

Chicago Manual of Style (16th Edition):

Cholanians, Aram B. “Dopamine Induced Post-Translational Modifications of α-Synuclein and the Role of Arsenic in the Development of Parkinson's Disease and Other Synucleinopathies .” 2016. Doctoral Dissertation, University of Arizona. Accessed January 19, 2020. http://hdl.handle.net/10150/612093.

MLA Handbook (7th Edition):

Cholanians, Aram B. “Dopamine Induced Post-Translational Modifications of α-Synuclein and the Role of Arsenic in the Development of Parkinson's Disease and Other Synucleinopathies .” 2016. Web. 19 Jan 2020.

Vancouver:

Cholanians AB. Dopamine Induced Post-Translational Modifications of α-Synuclein and the Role of Arsenic in the Development of Parkinson's Disease and Other Synucleinopathies . [Internet] [Doctoral dissertation]. University of Arizona; 2016. [cited 2020 Jan 19]. Available from: http://hdl.handle.net/10150/612093.

Council of Science Editors:

Cholanians AB. Dopamine Induced Post-Translational Modifications of α-Synuclein and the Role of Arsenic in the Development of Parkinson's Disease and Other Synucleinopathies . [Doctoral Dissertation]. University of Arizona; 2016. Available from: http://hdl.handle.net/10150/612093


University of Arizona

9. Sapiro, Jessica M. Molecular Mechanisms Associated with All-Trans-Retinoic Acid-Mediated Cytoprotection against Renal Cell Injury .

Degree: 2017, University of Arizona

 Chemical-induced nephrotoxicity is a major cause of acute kidney injury. My dissertation reveals that all-trans-retinoic acid (ATRA) affords cytoprotection against renal cell injury. Pretreatment with… (more)

Subjects/Keywords: Kidney; Toxicology; Vitamin A

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APA (6th Edition):

Sapiro, J. M. (2017). Molecular Mechanisms Associated with All-Trans-Retinoic Acid-Mediated Cytoprotection against Renal Cell Injury . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/625452

Chicago Manual of Style (16th Edition):

Sapiro, Jessica M. “Molecular Mechanisms Associated with All-Trans-Retinoic Acid-Mediated Cytoprotection against Renal Cell Injury .” 2017. Doctoral Dissertation, University of Arizona. Accessed January 19, 2020. http://hdl.handle.net/10150/625452.

MLA Handbook (7th Edition):

Sapiro, Jessica M. “Molecular Mechanisms Associated with All-Trans-Retinoic Acid-Mediated Cytoprotection against Renal Cell Injury .” 2017. Web. 19 Jan 2020.

Vancouver:

Sapiro JM. Molecular Mechanisms Associated with All-Trans-Retinoic Acid-Mediated Cytoprotection against Renal Cell Injury . [Internet] [Doctoral dissertation]. University of Arizona; 2017. [cited 2020 Jan 19]. Available from: http://hdl.handle.net/10150/625452.

Council of Science Editors:

Sapiro JM. Molecular Mechanisms Associated with All-Trans-Retinoic Acid-Mediated Cytoprotection against Renal Cell Injury . [Doctoral Dissertation]. University of Arizona; 2017. Available from: http://hdl.handle.net/10150/625452


University of Arizona

10. Fan, Xiaoyu. Function and Regulation of Intestinal Cytochrome P450 .

Degree: 2019, University of Arizona

 The overall goal of this thesis was to study the function and regulation of intestinal P450s. The central hypothesis is that disease state and environmental… (more)

Subjects/Keywords: Drug metabolism; Intestinal P450s

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APA (6th Edition):

Fan, X. (2019). Function and Regulation of Intestinal Cytochrome P450 . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/634254

Chicago Manual of Style (16th Edition):

Fan, Xiaoyu. “Function and Regulation of Intestinal Cytochrome P450 .” 2019. Doctoral Dissertation, University of Arizona. Accessed January 19, 2020. http://hdl.handle.net/10150/634254.

MLA Handbook (7th Edition):

Fan, Xiaoyu. “Function and Regulation of Intestinal Cytochrome P450 .” 2019. Web. 19 Jan 2020.

Vancouver:

Fan X. Function and Regulation of Intestinal Cytochrome P450 . [Internet] [Doctoral dissertation]. University of Arizona; 2019. [cited 2020 Jan 19]. Available from: http://hdl.handle.net/10150/634254.

Council of Science Editors:

Fan X. Function and Regulation of Intestinal Cytochrome P450 . [Doctoral Dissertation]. University of Arizona; 2019. Available from: http://hdl.handle.net/10150/634254


University of Arizona

11. Merrell, Matthew David. Xenosensor Regulation of Enzymes and Transporters in Drug Exposure and Disease .

Degree: 2011, University of Arizona

 A large and varied array of xenobiotics (foreign chemicals) enters into our bodies every day. In order to prevent toxicity resulting from xenobiotic accumulation, the… (more)

Subjects/Keywords: Disease; Liver; Metabolism; Xenobiotic; Xenosensor

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APA (6th Edition):

Merrell, M. D. (2011). Xenosensor Regulation of Enzymes and Transporters in Drug Exposure and Disease . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/194051

Chicago Manual of Style (16th Edition):

Merrell, Matthew David. “Xenosensor Regulation of Enzymes and Transporters in Drug Exposure and Disease .” 2011. Doctoral Dissertation, University of Arizona. Accessed January 19, 2020. http://hdl.handle.net/10150/194051.

MLA Handbook (7th Edition):

Merrell, Matthew David. “Xenosensor Regulation of Enzymes and Transporters in Drug Exposure and Disease .” 2011. Web. 19 Jan 2020.

Vancouver:

Merrell MD. Xenosensor Regulation of Enzymes and Transporters in Drug Exposure and Disease . [Internet] [Doctoral dissertation]. University of Arizona; 2011. [cited 2020 Jan 19]. Available from: http://hdl.handle.net/10150/194051.

Council of Science Editors:

Merrell MD. Xenosensor Regulation of Enzymes and Transporters in Drug Exposure and Disease . [Doctoral Dissertation]. University of Arizona; 2011. Available from: http://hdl.handle.net/10150/194051


University of Arizona

12. Goldman, Aaron. The Role of Bile Acids in the Progression of Squamous Epithelium to Barrett's Esophagus and Esophageal Adenocarcinoma .

Degree: 2010, University of Arizona

 Barrett's esophagus (BE) is a premalignant disease associated with esophageal adenocarcinoma (EAC). This condition is highly associated with gastroesophageal reflux disease (GERD) which is characterized… (more)

Subjects/Keywords: biochemistry; Carcinogenesis; DNA damage; Ion dynamics; Nitric oxide; physiology

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APA (6th Edition):

Goldman, A. (2010). The Role of Bile Acids in the Progression of Squamous Epithelium to Barrett's Esophagus and Esophageal Adenocarcinoma . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/195890

Chicago Manual of Style (16th Edition):

Goldman, Aaron. “The Role of Bile Acids in the Progression of Squamous Epithelium to Barrett's Esophagus and Esophageal Adenocarcinoma .” 2010. Doctoral Dissertation, University of Arizona. Accessed January 19, 2020. http://hdl.handle.net/10150/195890.

MLA Handbook (7th Edition):

Goldman, Aaron. “The Role of Bile Acids in the Progression of Squamous Epithelium to Barrett's Esophagus and Esophageal Adenocarcinoma .” 2010. Web. 19 Jan 2020.

Vancouver:

Goldman A. The Role of Bile Acids in the Progression of Squamous Epithelium to Barrett's Esophagus and Esophageal Adenocarcinoma . [Internet] [Doctoral dissertation]. University of Arizona; 2010. [cited 2020 Jan 19]. Available from: http://hdl.handle.net/10150/195890.

Council of Science Editors:

Goldman A. The Role of Bile Acids in the Progression of Squamous Epithelium to Barrett's Esophagus and Esophageal Adenocarcinoma . [Doctoral Dissertation]. University of Arizona; 2010. Available from: http://hdl.handle.net/10150/195890


University of Arizona

13. Wnek, Shawn Michael. Mechanisms of malignant transformation of human urothelial cells by monomethylarsonous acid .

Degree: 2011, University of Arizona

 Sources of arsenic exposure include air, water, and food from both natural and anthropogenic sources. Arsenic is categorized as a human carcinogen, and is associated… (more)

Subjects/Keywords: DNA damage; DNA repair; monomethylarsonous acid; Pharmacology & Toxicology; arsenic; bladder cancer

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APA (6th Edition):

Wnek, S. M. (2011). Mechanisms of malignant transformation of human urothelial cells by monomethylarsonous acid . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/201495

Chicago Manual of Style (16th Edition):

Wnek, Shawn Michael. “Mechanisms of malignant transformation of human urothelial cells by monomethylarsonous acid .” 2011. Doctoral Dissertation, University of Arizona. Accessed January 19, 2020. http://hdl.handle.net/10150/201495.

MLA Handbook (7th Edition):

Wnek, Shawn Michael. “Mechanisms of malignant transformation of human urothelial cells by monomethylarsonous acid .” 2011. Web. 19 Jan 2020.

Vancouver:

Wnek SM. Mechanisms of malignant transformation of human urothelial cells by monomethylarsonous acid . [Internet] [Doctoral dissertation]. University of Arizona; 2011. [cited 2020 Jan 19]. Available from: http://hdl.handle.net/10150/201495.

Council of Science Editors:

Wnek SM. Mechanisms of malignant transformation of human urothelial cells by monomethylarsonous acid . [Doctoral Dissertation]. University of Arizona; 2011. Available from: http://hdl.handle.net/10150/201495


University of Arizona

14. Mastrandrea, Nicholas Joseph. Pentoxifylline As An Adjuvant Treatment In Renal Cell Carcinoma .

Degree: 2014, University of Arizona

 Cyclin D1, a proto-oncogene, is required for progression from the G1 phase into the S phase of the cell cycle. Over-expression of cyclin D1 causes… (more)

Subjects/Keywords: ACHN; Cyclin D1; Pentoxifylline; Renal Cell Carcinoma; Pharmacology & Toxicology; 4EBP1

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APA (6th Edition):

Mastrandrea, N. J. (2014). Pentoxifylline As An Adjuvant Treatment In Renal Cell Carcinoma . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/337293

Chicago Manual of Style (16th Edition):

Mastrandrea, Nicholas Joseph. “Pentoxifylline As An Adjuvant Treatment In Renal Cell Carcinoma .” 2014. Doctoral Dissertation, University of Arizona. Accessed January 19, 2020. http://hdl.handle.net/10150/337293.

MLA Handbook (7th Edition):

Mastrandrea, Nicholas Joseph. “Pentoxifylline As An Adjuvant Treatment In Renal Cell Carcinoma .” 2014. Web. 19 Jan 2020.

Vancouver:

Mastrandrea NJ. Pentoxifylline As An Adjuvant Treatment In Renal Cell Carcinoma . [Internet] [Doctoral dissertation]. University of Arizona; 2014. [cited 2020 Jan 19]. Available from: http://hdl.handle.net/10150/337293.

Council of Science Editors:

Mastrandrea NJ. Pentoxifylline As An Adjuvant Treatment In Renal Cell Carcinoma . [Doctoral Dissertation]. University of Arizona; 2014. Available from: http://hdl.handle.net/10150/337293


University of Arizona

15. Tula Sanchez, Ana A. Elucidation of the Mechanisms of Resistance and Sensitivity to Histone Deacetylase Inhibitor, PXD101, in Diffuse Large B-Cell Lymphoma (DLBCL) .

Degree: 2013, University of Arizona

 Although curable in the majority of cases, Diffuse Large B-cell Lymphoma (DLBCL), the most prevalent Non-Hodgkin Lymphoma (NHL) throughout the world, is still fatal for… (more)

Subjects/Keywords: HDACs; PXD101; Pharmacology & Toxicology; DLBCL

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APA (6th Edition):

Tula Sanchez, A. A. (2013). Elucidation of the Mechanisms of Resistance and Sensitivity to Histone Deacetylase Inhibitor, PXD101, in Diffuse Large B-Cell Lymphoma (DLBCL) . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/301692

Chicago Manual of Style (16th Edition):

Tula Sanchez, Ana A. “Elucidation of the Mechanisms of Resistance and Sensitivity to Histone Deacetylase Inhibitor, PXD101, in Diffuse Large B-Cell Lymphoma (DLBCL) .” 2013. Doctoral Dissertation, University of Arizona. Accessed January 19, 2020. http://hdl.handle.net/10150/301692.

MLA Handbook (7th Edition):

Tula Sanchez, Ana A. “Elucidation of the Mechanisms of Resistance and Sensitivity to Histone Deacetylase Inhibitor, PXD101, in Diffuse Large B-Cell Lymphoma (DLBCL) .” 2013. Web. 19 Jan 2020.

Vancouver:

Tula Sanchez AA. Elucidation of the Mechanisms of Resistance and Sensitivity to Histone Deacetylase Inhibitor, PXD101, in Diffuse Large B-Cell Lymphoma (DLBCL) . [Internet] [Doctoral dissertation]. University of Arizona; 2013. [cited 2020 Jan 19]. Available from: http://hdl.handle.net/10150/301692.

Council of Science Editors:

Tula Sanchez AA. Elucidation of the Mechanisms of Resistance and Sensitivity to Histone Deacetylase Inhibitor, PXD101, in Diffuse Large B-Cell Lymphoma (DLBCL) . [Doctoral Dissertation]. University of Arizona; 2013. Available from: http://hdl.handle.net/10150/301692


University of Arizona

16. Hardwick, Rhiannon Nicole. Nash Alters Drug Metabolizing Enzyme and Transporter Expression Resulting in Significant Consequences for Pharmaceutical Disposition and Toxicity .

Degree: 2012, University of Arizona

 The body encounters an innumerable amount of foreign substances, termed xenobiotics, which it must remove in order to prevent damage to cells and organs. This… (more)

Subjects/Keywords: drug transporters; nonalcoholic fatty liver disease; nonalcoholic steatohepatitis; Pharmacology & Toxicology; drug disposition; drug metabolizing enzymes

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APA (6th Edition):

Hardwick, R. N. (2012). Nash Alters Drug Metabolizing Enzyme and Transporter Expression Resulting in Significant Consequences for Pharmaceutical Disposition and Toxicity . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/247281

Chicago Manual of Style (16th Edition):

Hardwick, Rhiannon Nicole. “Nash Alters Drug Metabolizing Enzyme and Transporter Expression Resulting in Significant Consequences for Pharmaceutical Disposition and Toxicity .” 2012. Doctoral Dissertation, University of Arizona. Accessed January 19, 2020. http://hdl.handle.net/10150/247281.

MLA Handbook (7th Edition):

Hardwick, Rhiannon Nicole. “Nash Alters Drug Metabolizing Enzyme and Transporter Expression Resulting in Significant Consequences for Pharmaceutical Disposition and Toxicity .” 2012. Web. 19 Jan 2020.

Vancouver:

Hardwick RN. Nash Alters Drug Metabolizing Enzyme and Transporter Expression Resulting in Significant Consequences for Pharmaceutical Disposition and Toxicity . [Internet] [Doctoral dissertation]. University of Arizona; 2012. [cited 2020 Jan 19]. Available from: http://hdl.handle.net/10150/247281.

Council of Science Editors:

Hardwick RN. Nash Alters Drug Metabolizing Enzyme and Transporter Expression Resulting in Significant Consequences for Pharmaceutical Disposition and Toxicity . [Doctoral Dissertation]. University of Arizona; 2012. Available from: http://hdl.handle.net/10150/247281

17. Wu, Tongde. Post-Transcriptional Regulation of Nrf2: Novel Mechanisms beyond Keap1 .

Degree: 2013, University of Arizona

 Nrf2 (NF-E2-related factor 2) is a transcription factor that regulates a battery of downstream genes that contain the antioxidant response element (ARE) in their promoter… (more)

Subjects/Keywords: Pharmacology & Toxicology

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APA (6th Edition):

Wu, T. (2013). Post-Transcriptional Regulation of Nrf2: Novel Mechanisms beyond Keap1 . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/301750

Chicago Manual of Style (16th Edition):

Wu, Tongde. “Post-Transcriptional Regulation of Nrf2: Novel Mechanisms beyond Keap1 .” 2013. Doctoral Dissertation, University of Arizona. Accessed January 19, 2020. http://hdl.handle.net/10150/301750.

MLA Handbook (7th Edition):

Wu, Tongde. “Post-Transcriptional Regulation of Nrf2: Novel Mechanisms beyond Keap1 .” 2013. Web. 19 Jan 2020.

Vancouver:

Wu T. Post-Transcriptional Regulation of Nrf2: Novel Mechanisms beyond Keap1 . [Internet] [Doctoral dissertation]. University of Arizona; 2013. [cited 2020 Jan 19]. Available from: http://hdl.handle.net/10150/301750.

Council of Science Editors:

Wu T. Post-Transcriptional Regulation of Nrf2: Novel Mechanisms beyond Keap1 . [Doctoral Dissertation]. University of Arizona; 2013. Available from: http://hdl.handle.net/10150/301750

18. Islas Robles, Argel. Identification of Poly(ADP-Ribose)-Associated Proteins and their Potential Role in ROS-Mediated Cell Death .

Degree: 2019, University of Arizona

 2,3,5-Tris-(glutathion-S-yl)hydroquinone (TGHQ) is a nephrotoxic and nephrocarcinogenic metabolite of hydroquinone. TGHQ can redox cycle, generating reactive oxygen species (ROS) which cause DNA strand breaks, hyperactivation… (more)

Subjects/Keywords: Cell death; PARP-1; Poly(ADP-ribosylation); ROS; TFII-I; TRPC3

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APA (6th Edition):

Islas Robles, A. (2019). Identification of Poly(ADP-Ribose)-Associated Proteins and their Potential Role in ROS-Mediated Cell Death . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/634402

Chicago Manual of Style (16th Edition):

Islas Robles, Argel. “Identification of Poly(ADP-Ribose)-Associated Proteins and their Potential Role in ROS-Mediated Cell Death .” 2019. Doctoral Dissertation, University of Arizona. Accessed January 19, 2020. http://hdl.handle.net/10150/634402.

MLA Handbook (7th Edition):

Islas Robles, Argel. “Identification of Poly(ADP-Ribose)-Associated Proteins and their Potential Role in ROS-Mediated Cell Death .” 2019. Web. 19 Jan 2020.

Vancouver:

Islas Robles A. Identification of Poly(ADP-Ribose)-Associated Proteins and their Potential Role in ROS-Mediated Cell Death . [Internet] [Doctoral dissertation]. University of Arizona; 2019. [cited 2020 Jan 19]. Available from: http://hdl.handle.net/10150/634402.

Council of Science Editors:

Islas Robles A. Identification of Poly(ADP-Ribose)-Associated Proteins and their Potential Role in ROS-Mediated Cell Death . [Doctoral Dissertation]. University of Arizona; 2019. Available from: http://hdl.handle.net/10150/634402

19. Cabello, Christopher Michael. Identification of Experimental Prooxidants Targeting the Redox Vulnerability of Malignant Melanoma .

Degree: 2013, University of Arizona

 Cumulative evidence suggests that redox dysregulation in cancer cells represents a chemical vulnerability that can be targeted by pharmacological modulation of cellular oxidative stress. According… (more)

Subjects/Keywords: Cinnamaldehyde; Dichlorophenolindophenol; Dihydroartemesinin; Melanoma; Redox; Pharmaceutical Sciences; Cancer

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APA (6th Edition):

Cabello, C. M. (2013). Identification of Experimental Prooxidants Targeting the Redox Vulnerability of Malignant Melanoma . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/293427

Chicago Manual of Style (16th Edition):

Cabello, Christopher Michael. “Identification of Experimental Prooxidants Targeting the Redox Vulnerability of Malignant Melanoma .” 2013. Doctoral Dissertation, University of Arizona. Accessed January 19, 2020. http://hdl.handle.net/10150/293427.

MLA Handbook (7th Edition):

Cabello, Christopher Michael. “Identification of Experimental Prooxidants Targeting the Redox Vulnerability of Malignant Melanoma .” 2013. Web. 19 Jan 2020.

Vancouver:

Cabello CM. Identification of Experimental Prooxidants Targeting the Redox Vulnerability of Malignant Melanoma . [Internet] [Doctoral dissertation]. University of Arizona; 2013. [cited 2020 Jan 19]. Available from: http://hdl.handle.net/10150/293427.

Council of Science Editors:

Cabello CM. Identification of Experimental Prooxidants Targeting the Redox Vulnerability of Malignant Melanoma . [Doctoral Dissertation]. University of Arizona; 2013. Available from: http://hdl.handle.net/10150/293427

20. Dzierlenga, Anika L. Mechanism and Functional Consequence of MRP2 Mislocalization in Nonalcoholic Steatohepatitis .

Degree: 2016, University of Arizona

 Adverse drug reactions (ADRs) are a pervasive complication in the realm of pharmacotherapy. At the root of ADRs lies interindividual variability in drug response, which… (more)

Subjects/Keywords: Liver; Pharmacokinetics; Transporter; Pharmacology & Toxicology; Adverse drug reactions

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APA (6th Edition):

Dzierlenga, A. L. (2016). Mechanism and Functional Consequence of MRP2 Mislocalization in Nonalcoholic Steatohepatitis . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/613592

Chicago Manual of Style (16th Edition):

Dzierlenga, Anika L. “Mechanism and Functional Consequence of MRP2 Mislocalization in Nonalcoholic Steatohepatitis .” 2016. Doctoral Dissertation, University of Arizona. Accessed January 19, 2020. http://hdl.handle.net/10150/613592.

MLA Handbook (7th Edition):

Dzierlenga, Anika L. “Mechanism and Functional Consequence of MRP2 Mislocalization in Nonalcoholic Steatohepatitis .” 2016. Web. 19 Jan 2020.

Vancouver:

Dzierlenga AL. Mechanism and Functional Consequence of MRP2 Mislocalization in Nonalcoholic Steatohepatitis . [Internet] [Doctoral dissertation]. University of Arizona; 2016. [cited 2020 Jan 19]. Available from: http://hdl.handle.net/10150/613592.

Council of Science Editors:

Dzierlenga AL. Mechanism and Functional Consequence of MRP2 Mislocalization in Nonalcoholic Steatohepatitis . [Doctoral Dissertation]. University of Arizona; 2016. Available from: http://hdl.handle.net/10150/613592

21. Tillotson, Joseph. Targeting Enzymes Involved in Protein Translation and Quality Control as Potential Cancer Therapeutics .

Degree: 2016, University of Arizona

 Activation of pathways resulting in an overexpression of oncoproteins, reliant on cap-dependent translation, or mutations of key proteins in a pathway can be advantageous to… (more)

Subjects/Keywords: Pharmacology & Toxicology

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APA (6th Edition):

Tillotson, J. (2016). Targeting Enzymes Involved in Protein Translation and Quality Control as Potential Cancer Therapeutics . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/621777

Chicago Manual of Style (16th Edition):

Tillotson, Joseph. “Targeting Enzymes Involved in Protein Translation and Quality Control as Potential Cancer Therapeutics .” 2016. Doctoral Dissertation, University of Arizona. Accessed January 19, 2020. http://hdl.handle.net/10150/621777.

MLA Handbook (7th Edition):

Tillotson, Joseph. “Targeting Enzymes Involved in Protein Translation and Quality Control as Potential Cancer Therapeutics .” 2016. Web. 19 Jan 2020.

Vancouver:

Tillotson J. Targeting Enzymes Involved in Protein Translation and Quality Control as Potential Cancer Therapeutics . [Internet] [Doctoral dissertation]. University of Arizona; 2016. [cited 2020 Jan 19]. Available from: http://hdl.handle.net/10150/621777.

Council of Science Editors:

Tillotson J. Targeting Enzymes Involved in Protein Translation and Quality Control as Potential Cancer Therapeutics . [Doctoral Dissertation]. University of Arizona; 2016. Available from: http://hdl.handle.net/10150/621777

22. Li, Hui. Nonalcoholic Steatohepatitis Alters Phase I Drug Metabolism .

Degree: 2018, University of Arizona

 Variable drug responses (VDRs) are dependent on inter-individual variability in the activity of drug-metabolizing enzymes. As the most common chronic liver disease in children and… (more)

Subjects/Keywords: Adverse Drug Reactions; Alcohol Metabolism; Cytochrome p450; Nonalcoholic Steatohepatitis; Phase I Metabolism

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APA (6th Edition):

Li, H. (2018). Nonalcoholic Steatohepatitis Alters Phase I Drug Metabolism . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/627774

Chicago Manual of Style (16th Edition):

Li, Hui. “Nonalcoholic Steatohepatitis Alters Phase I Drug Metabolism .” 2018. Doctoral Dissertation, University of Arizona. Accessed January 19, 2020. http://hdl.handle.net/10150/627774.

MLA Handbook (7th Edition):

Li, Hui. “Nonalcoholic Steatohepatitis Alters Phase I Drug Metabolism .” 2018. Web. 19 Jan 2020.

Vancouver:

Li H. Nonalcoholic Steatohepatitis Alters Phase I Drug Metabolism . [Internet] [Doctoral dissertation]. University of Arizona; 2018. [cited 2020 Jan 19]. Available from: http://hdl.handle.net/10150/627774.

Council of Science Editors:

Li H. Nonalcoholic Steatohepatitis Alters Phase I Drug Metabolism . [Doctoral Dissertation]. University of Arizona; 2018. Available from: http://hdl.handle.net/10150/627774

23. Bolt, Alicia Marie. Arsenite Alters Lysosome-Mediated Degradation and the Autophagy Process Leading to Immunosuppression in Human B-Lymphoblastoid Cell Lines .

Degree: 2012, University of Arizona

 The immune system is a target of arsenic toxicity. Epidemiological data have shown that arsenic exposure is associated with characteristics of immunosuppression. Human B-lymphoblastoid cell… (more)

Subjects/Keywords: Immunotoxicity; Lymphoblastoid; Proteotoxicity; Pharmacology & Toxicology; Arsenic; Autophagy

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APA (6th Edition):

Bolt, A. M. (2012). Arsenite Alters Lysosome-Mediated Degradation and the Autophagy Process Leading to Immunosuppression in Human B-Lymphoblastoid Cell Lines . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/222895

Chicago Manual of Style (16th Edition):

Bolt, Alicia Marie. “Arsenite Alters Lysosome-Mediated Degradation and the Autophagy Process Leading to Immunosuppression in Human B-Lymphoblastoid Cell Lines .” 2012. Doctoral Dissertation, University of Arizona. Accessed January 19, 2020. http://hdl.handle.net/10150/222895.

MLA Handbook (7th Edition):

Bolt, Alicia Marie. “Arsenite Alters Lysosome-Mediated Degradation and the Autophagy Process Leading to Immunosuppression in Human B-Lymphoblastoid Cell Lines .” 2012. Web. 19 Jan 2020.

Vancouver:

Bolt AM. Arsenite Alters Lysosome-Mediated Degradation and the Autophagy Process Leading to Immunosuppression in Human B-Lymphoblastoid Cell Lines . [Internet] [Doctoral dissertation]. University of Arizona; 2012. [cited 2020 Jan 19]. Available from: http://hdl.handle.net/10150/222895.

Council of Science Editors:

Bolt AM. Arsenite Alters Lysosome-Mediated Degradation and the Autophagy Process Leading to Immunosuppression in Human B-Lymphoblastoid Cell Lines . [Doctoral Dissertation]. University of Arizona; 2012. Available from: http://hdl.handle.net/10150/222895

24. Medeiros, Matthew Keane. Genomic Response in Human Urothelial Cells Exposed Chronically to Monomethylarsonous Acid .

Degree: 2013, University of Arizona

 Bladder cancer has been associated with chronic arsenic exposure. Monomethylarsonous acid [MMA(III)] is a metabolite of inorganic arsenic biotransformation and has been shown to transform… (more)

Subjects/Keywords: bladder; cancer biology; gene expression; gene microarray; urothelial cells; Pharmacology & Toxicology; arsenic

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APA (6th Edition):

Medeiros, M. K. (2013). Genomic Response in Human Urothelial Cells Exposed Chronically to Monomethylarsonous Acid . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/299107

Chicago Manual of Style (16th Edition):

Medeiros, Matthew Keane. “Genomic Response in Human Urothelial Cells Exposed Chronically to Monomethylarsonous Acid .” 2013. Doctoral Dissertation, University of Arizona. Accessed January 19, 2020. http://hdl.handle.net/10150/299107.

MLA Handbook (7th Edition):

Medeiros, Matthew Keane. “Genomic Response in Human Urothelial Cells Exposed Chronically to Monomethylarsonous Acid .” 2013. Web. 19 Jan 2020.

Vancouver:

Medeiros MK. Genomic Response in Human Urothelial Cells Exposed Chronically to Monomethylarsonous Acid . [Internet] [Doctoral dissertation]. University of Arizona; 2013. [cited 2020 Jan 19]. Available from: http://hdl.handle.net/10150/299107.

Council of Science Editors:

Medeiros MK. Genomic Response in Human Urothelial Cells Exposed Chronically to Monomethylarsonous Acid . [Doctoral Dissertation]. University of Arizona; 2013. Available from: http://hdl.handle.net/10150/299107

25. Lake, April D. Hepatic Stress Response Mechanisms in Progressive Human Nonalcoholic Fatty Liver Disease .

Degree: 2013, University of Arizona

 Nonalcoholic fatty liver disease (NAFLD) has become a worldwide, chronic liver disease of increasing clinical significance. It is closely associated with the rising epidemics of… (more)

Subjects/Keywords: Metabolism and Transport; Metabolomics; Nonalcoholic Fatty Liver Disease; Stress; Transcriptomics; Pharmacology & Toxicology; Liver

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APA (6th Edition):

Lake, A. D. (2013). Hepatic Stress Response Mechanisms in Progressive Human Nonalcoholic Fatty Liver Disease . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/299078

Chicago Manual of Style (16th Edition):

Lake, April D. “Hepatic Stress Response Mechanisms in Progressive Human Nonalcoholic Fatty Liver Disease .” 2013. Doctoral Dissertation, University of Arizona. Accessed January 19, 2020. http://hdl.handle.net/10150/299078.

MLA Handbook (7th Edition):

Lake, April D. “Hepatic Stress Response Mechanisms in Progressive Human Nonalcoholic Fatty Liver Disease .” 2013. Web. 19 Jan 2020.

Vancouver:

Lake AD. Hepatic Stress Response Mechanisms in Progressive Human Nonalcoholic Fatty Liver Disease . [Internet] [Doctoral dissertation]. University of Arizona; 2013. [cited 2020 Jan 19]. Available from: http://hdl.handle.net/10150/299078.

Council of Science Editors:

Lake AD. Hepatic Stress Response Mechanisms in Progressive Human Nonalcoholic Fatty Liver Disease . [Doctoral Dissertation]. University of Arizona; 2013. Available from: http://hdl.handle.net/10150/299078

26. Canatsey, Ryan Douglas. Combination Therapeutic Strategies Targeting Growth and Metabolic Pathways in Prostate Cancer .

Degree: 2016, University of Arizona

 Despite recent advances, prognosis in metastatic prostate cancer remains poor. As with other cancers, tumor heterogeneity is an increasingly evident contributor in prostate tumorigenesis and… (more)

Subjects/Keywords: diagnostics; immunohistochemistry; pentoxifylline; prostate cancer; signaling; Pharmacology & Toxicology; combination therapies

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APA (6th Edition):

Canatsey, R. D. (2016). Combination Therapeutic Strategies Targeting Growth and Metabolic Pathways in Prostate Cancer . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/613236

Chicago Manual of Style (16th Edition):

Canatsey, Ryan Douglas. “Combination Therapeutic Strategies Targeting Growth and Metabolic Pathways in Prostate Cancer .” 2016. Doctoral Dissertation, University of Arizona. Accessed January 19, 2020. http://hdl.handle.net/10150/613236.

MLA Handbook (7th Edition):

Canatsey, Ryan Douglas. “Combination Therapeutic Strategies Targeting Growth and Metabolic Pathways in Prostate Cancer .” 2016. Web. 19 Jan 2020.

Vancouver:

Canatsey RD. Combination Therapeutic Strategies Targeting Growth and Metabolic Pathways in Prostate Cancer . [Internet] [Doctoral dissertation]. University of Arizona; 2016. [cited 2020 Jan 19]. Available from: http://hdl.handle.net/10150/613236.

Council of Science Editors:

Canatsey RD. Combination Therapeutic Strategies Targeting Growth and Metabolic Pathways in Prostate Cancer . [Doctoral Dissertation]. University of Arizona; 2016. Available from: http://hdl.handle.net/10150/613236


University of Arizona

27. Sun, Zheng. Mechanistic Study of Nucleocytoplasmic Trafficking and Reversible Acetylation in Modulating the NRF2-Dependent Antioxidant Response .

Degree: 2008, University of Arizona

 To maintain intracellular redox homeostasis, genes encoding many endogenous antioxidants and phase II detoxification enzymes are transcriptionally upregulated upon deleterious oxidative stress through the cis-… (more)

Subjects/Keywords: acetylation; antioxidant response element; Keap1; Nrf2; oxidative stress; p300/CBP

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APA (6th Edition):

Sun, Z. (2008). Mechanistic Study of Nucleocytoplasmic Trafficking and Reversible Acetylation in Modulating the NRF2-Dependent Antioxidant Response . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/194904

Chicago Manual of Style (16th Edition):

Sun, Zheng. “Mechanistic Study of Nucleocytoplasmic Trafficking and Reversible Acetylation in Modulating the NRF2-Dependent Antioxidant Response .” 2008. Doctoral Dissertation, University of Arizona. Accessed January 19, 2020. http://hdl.handle.net/10150/194904.

MLA Handbook (7th Edition):

Sun, Zheng. “Mechanistic Study of Nucleocytoplasmic Trafficking and Reversible Acetylation in Modulating the NRF2-Dependent Antioxidant Response .” 2008. Web. 19 Jan 2020.

Vancouver:

Sun Z. Mechanistic Study of Nucleocytoplasmic Trafficking and Reversible Acetylation in Modulating the NRF2-Dependent Antioxidant Response . [Internet] [Doctoral dissertation]. University of Arizona; 2008. [cited 2020 Jan 19]. Available from: http://hdl.handle.net/10150/194904.

Council of Science Editors:

Sun Z. Mechanistic Study of Nucleocytoplasmic Trafficking and Reversible Acetylation in Modulating the NRF2-Dependent Antioxidant Response . [Doctoral Dissertation]. University of Arizona; 2008. Available from: http://hdl.handle.net/10150/194904

.