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You searched for +publisher:"University of Arizona" +contributor:("Schroeder, Joyce A."). Showing records 1 – 22 of 22 total matches.

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University of Arizona

1. Makhani, Kiran. Mechanism of Action of ERBB Decoy Cancer Therapeutic Peptide SAH5 .

Degree: 2017, University of Arizona

 Breast cancer is the most prevalent type of cancer and second leading cause of death in women. Among others, the triple negative breast cancer (TNBC)… (more)

Subjects/Keywords: Breast cancer; Calcium; ERBB decoy peptide; IP3R; Reactive oxygen species

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APA (6th Edition):

Makhani, K. (2017). Mechanism of Action of ERBB Decoy Cancer Therapeutic Peptide SAH5 . (Masters Thesis). University of Arizona. Retrieved from http://hdl.handle.net/10150/626139

Chicago Manual of Style (16th Edition):

Makhani, Kiran. “Mechanism of Action of ERBB Decoy Cancer Therapeutic Peptide SAH5 .” 2017. Masters Thesis, University of Arizona. Accessed March 04, 2021. http://hdl.handle.net/10150/626139.

MLA Handbook (7th Edition):

Makhani, Kiran. “Mechanism of Action of ERBB Decoy Cancer Therapeutic Peptide SAH5 .” 2017. Web. 04 Mar 2021.

Vancouver:

Makhani K. Mechanism of Action of ERBB Decoy Cancer Therapeutic Peptide SAH5 . [Internet] [Masters thesis]. University of Arizona; 2017. [cited 2021 Mar 04]. Available from: http://hdl.handle.net/10150/626139.

Council of Science Editors:

Makhani K. Mechanism of Action of ERBB Decoy Cancer Therapeutic Peptide SAH5 . [Masters Thesis]. University of Arizona; 2017. Available from: http://hdl.handle.net/10150/626139


University of Arizona

2. Su, Hsin-Yuan. Therapeutic Potential of EGFR Derived Peptides in Breast Cancer .

Degree: 2013, University of Arizona

 The epidermal growth factor receptor (EGFR) belongs to the erbB family of receptor tyrosine kinases which consists of four members (EGFR, ErbB2, ErbB3 and ErbB4).… (more)

Subjects/Keywords: EGFR; juxtamembrane domain; MUC1; nuclear EGFR; peptide; Cancer Biology; breast cancer

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APA (6th Edition):

Su, H. (2013). Therapeutic Potential of EGFR Derived Peptides in Breast Cancer . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/293486

Chicago Manual of Style (16th Edition):

Su, Hsin-Yuan. “Therapeutic Potential of EGFR Derived Peptides in Breast Cancer .” 2013. Doctoral Dissertation, University of Arizona. Accessed March 04, 2021. http://hdl.handle.net/10150/293486.

MLA Handbook (7th Edition):

Su, Hsin-Yuan. “Therapeutic Potential of EGFR Derived Peptides in Breast Cancer .” 2013. Web. 04 Mar 2021.

Vancouver:

Su H. Therapeutic Potential of EGFR Derived Peptides in Breast Cancer . [Internet] [Doctoral dissertation]. University of Arizona; 2013. [cited 2021 Mar 04]. Available from: http://hdl.handle.net/10150/293486.

Council of Science Editors:

Su H. Therapeutic Potential of EGFR Derived Peptides in Breast Cancer . [Doctoral Dissertation]. University of Arizona; 2013. Available from: http://hdl.handle.net/10150/293486


University of Arizona

3. Hart, Matthew Robert. Understanding EGFR Modification, Trafficking, and the Importance of its Juxtamembrane Domain in Cancer .

Degree: 2013, University of Arizona

 Much of what is known about the role of the ERBB family in cellular biology and in cancer has to do with canonical downstream signaling… (more)

Subjects/Keywords: EGFR; Peptide; Ubiquitin; Genetics; Cancer

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APA (6th Edition):

Hart, M. R. (2013). Understanding EGFR Modification, Trafficking, and the Importance of its Juxtamembrane Domain in Cancer . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/293476

Chicago Manual of Style (16th Edition):

Hart, Matthew Robert. “Understanding EGFR Modification, Trafficking, and the Importance of its Juxtamembrane Domain in Cancer .” 2013. Doctoral Dissertation, University of Arizona. Accessed March 04, 2021. http://hdl.handle.net/10150/293476.

MLA Handbook (7th Edition):

Hart, Matthew Robert. “Understanding EGFR Modification, Trafficking, and the Importance of its Juxtamembrane Domain in Cancer .” 2013. Web. 04 Mar 2021.

Vancouver:

Hart MR. Understanding EGFR Modification, Trafficking, and the Importance of its Juxtamembrane Domain in Cancer . [Internet] [Doctoral dissertation]. University of Arizona; 2013. [cited 2021 Mar 04]. Available from: http://hdl.handle.net/10150/293476.

Council of Science Editors:

Hart MR. Understanding EGFR Modification, Trafficking, and the Importance of its Juxtamembrane Domain in Cancer . [Doctoral Dissertation]. University of Arizona; 2013. Available from: http://hdl.handle.net/10150/293476


University of Arizona

4. Russ, Atlantis Dawn. HUGL and the Role of Polarity in Breast Cancer .

Degree: 2013, University of Arizona

 Loss of polarity is a defining characteristic of epithelial cancers. The cytoskeletal proteins, HUGL1 and HUGL2, mediate polarity in epithelial cells through diversified roles in… (more)

Subjects/Keywords: HUGL; Lgl; polarity; Genetics; cancer stem cell

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APA (6th Edition):

Russ, A. D. (2013). HUGL and the Role of Polarity in Breast Cancer . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/293491

Chicago Manual of Style (16th Edition):

Russ, Atlantis Dawn. “HUGL and the Role of Polarity in Breast Cancer .” 2013. Doctoral Dissertation, University of Arizona. Accessed March 04, 2021. http://hdl.handle.net/10150/293491.

MLA Handbook (7th Edition):

Russ, Atlantis Dawn. “HUGL and the Role of Polarity in Breast Cancer .” 2013. Web. 04 Mar 2021.

Vancouver:

Russ AD. HUGL and the Role of Polarity in Breast Cancer . [Internet] [Doctoral dissertation]. University of Arizona; 2013. [cited 2021 Mar 04]. Available from: http://hdl.handle.net/10150/293491.

Council of Science Editors:

Russ AD. HUGL and the Role of Polarity in Breast Cancer . [Doctoral Dissertation]. University of Arizona; 2013. Available from: http://hdl.handle.net/10150/293491


University of Arizona

5. Horm, Teresa Marie. The Roles of MUC1 and EGFR in Breast Cancer Progression and Mammary Lactation .

Degree: 2013, University of Arizona

 The relationship between MUC1 and EGFR has been characterized by our lab to be highly tumorigenic. A peptide therapeutic was developed in our lab to… (more)

Subjects/Keywords: EGFR; epithelial polarity; metastasis; MUC1; Molecular & Cellular Biology; breast cancer

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APA (6th Edition):

Horm, T. M. (2013). The Roles of MUC1 and EGFR in Breast Cancer Progression and Mammary Lactation . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/293562

Chicago Manual of Style (16th Edition):

Horm, Teresa Marie. “The Roles of MUC1 and EGFR in Breast Cancer Progression and Mammary Lactation .” 2013. Doctoral Dissertation, University of Arizona. Accessed March 04, 2021. http://hdl.handle.net/10150/293562.

MLA Handbook (7th Edition):

Horm, Teresa Marie. “The Roles of MUC1 and EGFR in Breast Cancer Progression and Mammary Lactation .” 2013. Web. 04 Mar 2021.

Vancouver:

Horm TM. The Roles of MUC1 and EGFR in Breast Cancer Progression and Mammary Lactation . [Internet] [Doctoral dissertation]. University of Arizona; 2013. [cited 2021 Mar 04]. Available from: http://hdl.handle.net/10150/293562.

Council of Science Editors:

Horm TM. The Roles of MUC1 and EGFR in Breast Cancer Progression and Mammary Lactation . [Doctoral Dissertation]. University of Arizona; 2013. Available from: http://hdl.handle.net/10150/293562


University of Arizona

6. Soyfer, Eli Michael. Understanding the Mechanism of Cancer Therapeutic SAH5-EJ1 in Targeting Breast Cancer Metastasis .

Degree: 2020, University of Arizona

 While many breast cancer subtypes overexpress members of the ERBB family of receptor tyrosine kinases (including the Epidermal Growth Factor Receptor/EGFR, HER2, ERBB3 and ERBB4),… (more)

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APA (6th Edition):

Soyfer, E. M. (2020). Understanding the Mechanism of Cancer Therapeutic SAH5-EJ1 in Targeting Breast Cancer Metastasis . (Masters Thesis). University of Arizona. Retrieved from http://hdl.handle.net/10150/641712

Chicago Manual of Style (16th Edition):

Soyfer, Eli Michael. “Understanding the Mechanism of Cancer Therapeutic SAH5-EJ1 in Targeting Breast Cancer Metastasis .” 2020. Masters Thesis, University of Arizona. Accessed March 04, 2021. http://hdl.handle.net/10150/641712.

MLA Handbook (7th Edition):

Soyfer, Eli Michael. “Understanding the Mechanism of Cancer Therapeutic SAH5-EJ1 in Targeting Breast Cancer Metastasis .” 2020. Web. 04 Mar 2021.

Vancouver:

Soyfer EM. Understanding the Mechanism of Cancer Therapeutic SAH5-EJ1 in Targeting Breast Cancer Metastasis . [Internet] [Masters thesis]. University of Arizona; 2020. [cited 2021 Mar 04]. Available from: http://hdl.handle.net/10150/641712.

Council of Science Editors:

Soyfer EM. Understanding the Mechanism of Cancer Therapeutic SAH5-EJ1 in Targeting Breast Cancer Metastasis . [Masters Thesis]. University of Arizona; 2020. Available from: http://hdl.handle.net/10150/641712


University of Arizona

7. Hunjan, Anoop Singh. Chronic Arsenite Exposure in Lung Epithelium Modulates Endocytosis .

Degree: 2017, University of Arizona

 Arsenic exposure in humans has been implicated in the development of a myriad of non-cancerous and cancerous diseases. A reductionist approach to understanding this unusual… (more)

Subjects/Keywords: Albumin; Arsenic; BEAS-2B; Endocytosis; LDL; Transferrin

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APA (6th Edition):

Hunjan, A. S. (2017). Chronic Arsenite Exposure in Lung Epithelium Modulates Endocytosis . (Masters Thesis). University of Arizona. Retrieved from http://hdl.handle.net/10150/626765

Chicago Manual of Style (16th Edition):

Hunjan, Anoop Singh. “Chronic Arsenite Exposure in Lung Epithelium Modulates Endocytosis .” 2017. Masters Thesis, University of Arizona. Accessed March 04, 2021. http://hdl.handle.net/10150/626765.

MLA Handbook (7th Edition):

Hunjan, Anoop Singh. “Chronic Arsenite Exposure in Lung Epithelium Modulates Endocytosis .” 2017. Web. 04 Mar 2021.

Vancouver:

Hunjan AS. Chronic Arsenite Exposure in Lung Epithelium Modulates Endocytosis . [Internet] [Masters thesis]. University of Arizona; 2017. [cited 2021 Mar 04]. Available from: http://hdl.handle.net/10150/626765.

Council of Science Editors:

Hunjan AS. Chronic Arsenite Exposure in Lung Epithelium Modulates Endocytosis . [Masters Thesis]. University of Arizona; 2017. Available from: http://hdl.handle.net/10150/626765


University of Arizona

8. Harwood, Samuel John. Human Cytomegalovirus Use and Manipulation of Host Phospholipids .

Degree: 2019, University of Arizona

 Human cytomegalovirus (HCMV) is a widely-spread β-herpesvirus that causes a congenital infection that results in devastating disabilities in newborns. Infection also causes life-threatening disease in… (more)

Subjects/Keywords: HCMV; Lipids; Mass Spectrometry; Metabolism

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APA (6th Edition):

Harwood, S. J. (2019). Human Cytomegalovirus Use and Manipulation of Host Phospholipids . (Masters Thesis). University of Arizona. Retrieved from http://hdl.handle.net/10150/632563

Chicago Manual of Style (16th Edition):

Harwood, Samuel John. “Human Cytomegalovirus Use and Manipulation of Host Phospholipids .” 2019. Masters Thesis, University of Arizona. Accessed March 04, 2021. http://hdl.handle.net/10150/632563.

MLA Handbook (7th Edition):

Harwood, Samuel John. “Human Cytomegalovirus Use and Manipulation of Host Phospholipids .” 2019. Web. 04 Mar 2021.

Vancouver:

Harwood SJ. Human Cytomegalovirus Use and Manipulation of Host Phospholipids . [Internet] [Masters thesis]. University of Arizona; 2019. [cited 2021 Mar 04]. Available from: http://hdl.handle.net/10150/632563.

Council of Science Editors:

Harwood SJ. Human Cytomegalovirus Use and Manipulation of Host Phospholipids . [Masters Thesis]. University of Arizona; 2019. Available from: http://hdl.handle.net/10150/632563


University of Arizona

9. Cheng, Julia N. The Roles of Estrogen and Transforming Growth Factor-β Signaling Pathways in Estrogen Receptor-Positive Breast Cancer Osteolytic Bone Metastasis Progression .

Degree: 2020, University of Arizona

 Tumors that express the estrogen receptor alpha (ERa) are the most commonly diagnosed breast cancer subtype (>70%), and also have the highest predilection to form… (more)

Subjects/Keywords: breast cancer; estrogen; metastasis

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APA (6th Edition):

Cheng, J. N. (2020). The Roles of Estrogen and Transforming Growth Factor-β Signaling Pathways in Estrogen Receptor-Positive Breast Cancer Osteolytic Bone Metastasis Progression . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/648593

Chicago Manual of Style (16th Edition):

Cheng, Julia N. “The Roles of Estrogen and Transforming Growth Factor-β Signaling Pathways in Estrogen Receptor-Positive Breast Cancer Osteolytic Bone Metastasis Progression .” 2020. Doctoral Dissertation, University of Arizona. Accessed March 04, 2021. http://hdl.handle.net/10150/648593.

MLA Handbook (7th Edition):

Cheng, Julia N. “The Roles of Estrogen and Transforming Growth Factor-β Signaling Pathways in Estrogen Receptor-Positive Breast Cancer Osteolytic Bone Metastasis Progression .” 2020. Web. 04 Mar 2021.

Vancouver:

Cheng JN. The Roles of Estrogen and Transforming Growth Factor-β Signaling Pathways in Estrogen Receptor-Positive Breast Cancer Osteolytic Bone Metastasis Progression . [Internet] [Doctoral dissertation]. University of Arizona; 2020. [cited 2021 Mar 04]. Available from: http://hdl.handle.net/10150/648593.

Council of Science Editors:

Cheng JN. The Roles of Estrogen and Transforming Growth Factor-β Signaling Pathways in Estrogen Receptor-Positive Breast Cancer Osteolytic Bone Metastasis Progression . [Doctoral Dissertation]. University of Arizona; 2020. Available from: http://hdl.handle.net/10150/648593

10. Pei, Fen. Cell-Based Mechanism Mediating Prion Loss and Stability .

Degree: 2017, University of Arizona

 The prion protein underlies several previously inexplicable phenomena, including transmissible neurodegenerative disease in mammals and the non-Mendelian inheritance of unique traits in fungi. These proteins… (more)

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APA (6th Edition):

Pei, F. (2017). Cell-Based Mechanism Mediating Prion Loss and Stability . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/626311

Chicago Manual of Style (16th Edition):

Pei, Fen. “Cell-Based Mechanism Mediating Prion Loss and Stability .” 2017. Doctoral Dissertation, University of Arizona. Accessed March 04, 2021. http://hdl.handle.net/10150/626311.

MLA Handbook (7th Edition):

Pei, Fen. “Cell-Based Mechanism Mediating Prion Loss and Stability .” 2017. Web. 04 Mar 2021.

Vancouver:

Pei F. Cell-Based Mechanism Mediating Prion Loss and Stability . [Internet] [Doctoral dissertation]. University of Arizona; 2017. [cited 2021 Mar 04]. Available from: http://hdl.handle.net/10150/626311.

Council of Science Editors:

Pei F. Cell-Based Mechanism Mediating Prion Loss and Stability . [Doctoral Dissertation]. University of Arizona; 2017. Available from: http://hdl.handle.net/10150/626311


University of Arizona

11. Moses, Sylvestor Andrea. Targeting Pleckstrin Homology Domains for the Inhibition of Cancer Growth and Metastasis .

Degree: 2013, University of Arizona

 Pleckstrin homology (PH) domains are structurally conserved domains, which generally bind to phosphatidylinositol phosphate (PtdInsP) lipids. They are present in a variety of proteins, including… (more)

Subjects/Keywords: Drug Discovery; Metastasis; Pleckstrin Homology Domains; Small Molecule Inhibitors; Tiam1; Molecular & Cellular Biology; Akt

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APA (6th Edition):

Moses, S. A. (2013). Targeting Pleckstrin Homology Domains for the Inhibition of Cancer Growth and Metastasis . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/293751

Chicago Manual of Style (16th Edition):

Moses, Sylvestor Andrea. “Targeting Pleckstrin Homology Domains for the Inhibition of Cancer Growth and Metastasis .” 2013. Doctoral Dissertation, University of Arizona. Accessed March 04, 2021. http://hdl.handle.net/10150/293751.

MLA Handbook (7th Edition):

Moses, Sylvestor Andrea. “Targeting Pleckstrin Homology Domains for the Inhibition of Cancer Growth and Metastasis .” 2013. Web. 04 Mar 2021.

Vancouver:

Moses SA. Targeting Pleckstrin Homology Domains for the Inhibition of Cancer Growth and Metastasis . [Internet] [Doctoral dissertation]. University of Arizona; 2013. [cited 2021 Mar 04]. Available from: http://hdl.handle.net/10150/293751.

Council of Science Editors:

Moses SA. Targeting Pleckstrin Homology Domains for the Inhibition of Cancer Growth and Metastasis . [Doctoral Dissertation]. University of Arizona; 2013. Available from: http://hdl.handle.net/10150/293751

12. Greenwood, Erin Barbara. Polarity as a Regulator of Metaplasia .

Degree: 2016, University of Arizona

 Cell polarity is an important regulator of cellular processes and is vital in helping to prevent metaplasia and tumorigenesis. There are three many polarity complexes… (more)

Subjects/Keywords: Hugl1; Llgl1; Mgl1; Migration; Taz; Molecular & Cellular Biology; Epidermal Growth Factor Receptor

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APA (6th Edition):

Greenwood, E. B. (2016). Polarity as a Regulator of Metaplasia . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/621753

Chicago Manual of Style (16th Edition):

Greenwood, Erin Barbara. “Polarity as a Regulator of Metaplasia .” 2016. Doctoral Dissertation, University of Arizona. Accessed March 04, 2021. http://hdl.handle.net/10150/621753.

MLA Handbook (7th Edition):

Greenwood, Erin Barbara. “Polarity as a Regulator of Metaplasia .” 2016. Web. 04 Mar 2021.

Vancouver:

Greenwood EB. Polarity as a Regulator of Metaplasia . [Internet] [Doctoral dissertation]. University of Arizona; 2016. [cited 2021 Mar 04]. Available from: http://hdl.handle.net/10150/621753.

Council of Science Editors:

Greenwood EB. Polarity as a Regulator of Metaplasia . [Doctoral Dissertation]. University of Arizona; 2016. Available from: http://hdl.handle.net/10150/621753


University of Arizona

13. Vargas, Ashley Joy. Assessing the Role of Dietary Polyamines on the Continuum of Colorectal Carcinoma .

Degree: 2013, University of Arizona

 Putrescine, spermidine and spermine are the polyamines biosynthesized by human cells via ornithine decarboxylase (ODC) and are also sourced from the diet. Polyamines are required… (more)

Subjects/Keywords: Colorectal cancer; Dietary polyamines; Polyamines; Putrescine; Spermidine; Nutritional Sciences; Colorectal adenoma

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APA (6th Edition):

Vargas, A. J. (2013). Assessing the Role of Dietary Polyamines on the Continuum of Colorectal Carcinoma . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/293416

Chicago Manual of Style (16th Edition):

Vargas, Ashley Joy. “Assessing the Role of Dietary Polyamines on the Continuum of Colorectal Carcinoma .” 2013. Doctoral Dissertation, University of Arizona. Accessed March 04, 2021. http://hdl.handle.net/10150/293416.

MLA Handbook (7th Edition):

Vargas, Ashley Joy. “Assessing the Role of Dietary Polyamines on the Continuum of Colorectal Carcinoma .” 2013. Web. 04 Mar 2021.

Vancouver:

Vargas AJ. Assessing the Role of Dietary Polyamines on the Continuum of Colorectal Carcinoma . [Internet] [Doctoral dissertation]. University of Arizona; 2013. [cited 2021 Mar 04]. Available from: http://hdl.handle.net/10150/293416.

Council of Science Editors:

Vargas AJ. Assessing the Role of Dietary Polyamines on the Continuum of Colorectal Carcinoma . [Doctoral Dissertation]. University of Arizona; 2013. Available from: http://hdl.handle.net/10150/293416

14. Maisel, Sabrina. The Role of Alternative Epidermal Growth Factor Receptor Trafficking in Driving Cancer Progression .

Degree: 2017, University of Arizona

 The Epidermal Growth Factor Receptor (EGFR) is associated with a variety of cancers, including brain, lung, cervix, renal and breast. It is part of a… (more)

Subjects/Keywords: epidermal growth factor receptor; llgl1; migration; MUC1; Retrograde Trafficking

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APA (6th Edition):

Maisel, S. (2017). The Role of Alternative Epidermal Growth Factor Receptor Trafficking in Driving Cancer Progression . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/624472

Chicago Manual of Style (16th Edition):

Maisel, Sabrina. “The Role of Alternative Epidermal Growth Factor Receptor Trafficking in Driving Cancer Progression .” 2017. Doctoral Dissertation, University of Arizona. Accessed March 04, 2021. http://hdl.handle.net/10150/624472.

MLA Handbook (7th Edition):

Maisel, Sabrina. “The Role of Alternative Epidermal Growth Factor Receptor Trafficking in Driving Cancer Progression .” 2017. Web. 04 Mar 2021.

Vancouver:

Maisel S. The Role of Alternative Epidermal Growth Factor Receptor Trafficking in Driving Cancer Progression . [Internet] [Doctoral dissertation]. University of Arizona; 2017. [cited 2021 Mar 04]. Available from: http://hdl.handle.net/10150/624472.

Council of Science Editors:

Maisel S. The Role of Alternative Epidermal Growth Factor Receptor Trafficking in Driving Cancer Progression . [Doctoral Dissertation]. University of Arizona; 2017. Available from: http://hdl.handle.net/10150/624472

15. vanGils Louderbough, Jeanne Marguerite. CD44 and Hyaluronan in the Regulation of Mammary Gland Development and Breast Cancer Progression .

Degree: 2011, University of Arizona

 Metastasis is the leading cause of death in patients with cancer, and the extracellular matrix is critical to cancer dissemination. The adhesion receptor, CD44, mediates… (more)

Subjects/Keywords: Breast Cancer; CD44; Hyaluronan; Mammary Gland Development

…Requirements For the Degree of DOCTOR OF PHILOSOPHY In the Graduate College THE UNIVERSITY OF ARIZONA… …2011 THE UNIVERSITY OF ARIZONA GRADUATE COLLEGE As members of the Dissertation Committee… …University of Arizona and is deposited in the University Library to be made available to borrowers… 

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APA (6th Edition):

vanGils Louderbough, J. M. (2011). CD44 and Hyaluronan in the Regulation of Mammary Gland Development and Breast Cancer Progression . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/146075

Chicago Manual of Style (16th Edition):

vanGils Louderbough, Jeanne Marguerite. “CD44 and Hyaluronan in the Regulation of Mammary Gland Development and Breast Cancer Progression .” 2011. Doctoral Dissertation, University of Arizona. Accessed March 04, 2021. http://hdl.handle.net/10150/146075.

MLA Handbook (7th Edition):

vanGils Louderbough, Jeanne Marguerite. “CD44 and Hyaluronan in the Regulation of Mammary Gland Development and Breast Cancer Progression .” 2011. Web. 04 Mar 2021.

Vancouver:

vanGils Louderbough JM. CD44 and Hyaluronan in the Regulation of Mammary Gland Development and Breast Cancer Progression . [Internet] [Doctoral dissertation]. University of Arizona; 2011. [cited 2021 Mar 04]. Available from: http://hdl.handle.net/10150/146075.

Council of Science Editors:

vanGils Louderbough JM. CD44 and Hyaluronan in the Regulation of Mammary Gland Development and Breast Cancer Progression . [Doctoral Dissertation]. University of Arizona; 2011. Available from: http://hdl.handle.net/10150/146075

16. Wright, Laura E. Curcuminoids in the Prevention of Osteoclast-Mediated Bone Resorption in Translational Models of Postmenopausal Osteoporosis and Lytic Breast Cancer Bone Metastasis .

Degree: 2012, University of Arizona

 The studies presented in this dissertation offer evidence to support the hypothesis that polyphenolic curcuminoids isolated from the plant turmeric (Curcuma longa L.) are bone-protective… (more)

Subjects/Keywords: Curcuminoids; Metastasis; Osteoclast; Osteoporosis; Physiological Sciences; Bone; Breast Cancer

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APA (6th Edition):

Wright, L. E. (2012). Curcuminoids in the Prevention of Osteoclast-Mediated Bone Resorption in Translational Models of Postmenopausal Osteoporosis and Lytic Breast Cancer Bone Metastasis . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/222897

Chicago Manual of Style (16th Edition):

Wright, Laura E. “Curcuminoids in the Prevention of Osteoclast-Mediated Bone Resorption in Translational Models of Postmenopausal Osteoporosis and Lytic Breast Cancer Bone Metastasis .” 2012. Doctoral Dissertation, University of Arizona. Accessed March 04, 2021. http://hdl.handle.net/10150/222897.

MLA Handbook (7th Edition):

Wright, Laura E. “Curcuminoids in the Prevention of Osteoclast-Mediated Bone Resorption in Translational Models of Postmenopausal Osteoporosis and Lytic Breast Cancer Bone Metastasis .” 2012. Web. 04 Mar 2021.

Vancouver:

Wright LE. Curcuminoids in the Prevention of Osteoclast-Mediated Bone Resorption in Translational Models of Postmenopausal Osteoporosis and Lytic Breast Cancer Bone Metastasis . [Internet] [Doctoral dissertation]. University of Arizona; 2012. [cited 2021 Mar 04]. Available from: http://hdl.handle.net/10150/222897.

Council of Science Editors:

Wright LE. Curcuminoids in the Prevention of Osteoclast-Mediated Bone Resorption in Translational Models of Postmenopausal Osteoporosis and Lytic Breast Cancer Bone Metastasis . [Doctoral Dissertation]. University of Arizona; 2012. Available from: http://hdl.handle.net/10150/222897


University of Arizona

17. Lopez, Jose Ignacio. CD44 Attenuates Metastasis During Breast Cancer Progression .

Degree: 2008, University of Arizona

 Progression to metastatic disease is the leading cause of deaths resulting from breast cancer. Understanding the mechanisms underlying a cell's ability to move away from… (more)

Subjects/Keywords: CD44; Hyaluronan; Breast Cancer

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APA (6th Edition):

Lopez, J. I. (2008). CD44 Attenuates Metastasis During Breast Cancer Progression . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/193882

Chicago Manual of Style (16th Edition):

Lopez, Jose Ignacio. “CD44 Attenuates Metastasis During Breast Cancer Progression .” 2008. Doctoral Dissertation, University of Arizona. Accessed March 04, 2021. http://hdl.handle.net/10150/193882.

MLA Handbook (7th Edition):

Lopez, Jose Ignacio. “CD44 Attenuates Metastasis During Breast Cancer Progression .” 2008. Web. 04 Mar 2021.

Vancouver:

Lopez JI. CD44 Attenuates Metastasis During Breast Cancer Progression . [Internet] [Doctoral dissertation]. University of Arizona; 2008. [cited 2021 Mar 04]. Available from: http://hdl.handle.net/10150/193882.

Council of Science Editors:

Lopez JI. CD44 Attenuates Metastasis During Breast Cancer Progression . [Doctoral Dissertation]. University of Arizona; 2008. Available from: http://hdl.handle.net/10150/193882


University of Arizona

18. Basu Roy, Upal Kunal. Regulation and Function of Caveolin-1 in Colorectal Carcinogenesis .

Degree: 2007, University of Arizona

 Colon cancer is the second leading cause of cancer deaths in the United States of America. It is caused by the accumulation of mutations in… (more)

Subjects/Keywords: Molecular & Cellular Biology

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APA (6th Edition):

Basu Roy, U. K. (2007). Regulation and Function of Caveolin-1 in Colorectal Carcinogenesis . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/194031

Chicago Manual of Style (16th Edition):

Basu Roy, Upal Kunal. “Regulation and Function of Caveolin-1 in Colorectal Carcinogenesis .” 2007. Doctoral Dissertation, University of Arizona. Accessed March 04, 2021. http://hdl.handle.net/10150/194031.

MLA Handbook (7th Edition):

Basu Roy, Upal Kunal. “Regulation and Function of Caveolin-1 in Colorectal Carcinogenesis .” 2007. Web. 04 Mar 2021.

Vancouver:

Basu Roy UK. Regulation and Function of Caveolin-1 in Colorectal Carcinogenesis . [Internet] [Doctoral dissertation]. University of Arizona; 2007. [cited 2021 Mar 04]. Available from: http://hdl.handle.net/10150/194031.

Council of Science Editors:

Basu Roy UK. Regulation and Function of Caveolin-1 in Colorectal Carcinogenesis . [Doctoral Dissertation]. University of Arizona; 2007. Available from: http://hdl.handle.net/10150/194031


University of Arizona

19. Bitler, Benjamin Guy. DETERMINING THE ROLE OF MUC1 AND BETA-CATENIN ON THE EPIDERMAL GROWTH FACTOR RECEPTOR SIGNALING AND LOCALIZATION IN BREAST CANCER .

Degree: 2010, University of Arizona

 The epidermal growth factor family of receptors is important in the development and progression of many types of cancers including, breast, lung, and glioblastoma. The… (more)

Subjects/Keywords: beta-catenin; breast cancer; EGFR; MUC1; Nuclear Localization; Trafficking

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APA (6th Edition):

Bitler, B. G. (2010). DETERMINING THE ROLE OF MUC1 AND BETA-CATENIN ON THE EPIDERMAL GROWTH FACTOR RECEPTOR SIGNALING AND LOCALIZATION IN BREAST CANCER . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/194738

Chicago Manual of Style (16th Edition):

Bitler, Benjamin Guy. “DETERMINING THE ROLE OF MUC1 AND BETA-CATENIN ON THE EPIDERMAL GROWTH FACTOR RECEPTOR SIGNALING AND LOCALIZATION IN BREAST CANCER .” 2010. Doctoral Dissertation, University of Arizona. Accessed March 04, 2021. http://hdl.handle.net/10150/194738.

MLA Handbook (7th Edition):

Bitler, Benjamin Guy. “DETERMINING THE ROLE OF MUC1 AND BETA-CATENIN ON THE EPIDERMAL GROWTH FACTOR RECEPTOR SIGNALING AND LOCALIZATION IN BREAST CANCER .” 2010. Web. 04 Mar 2021.

Vancouver:

Bitler BG. DETERMINING THE ROLE OF MUC1 AND BETA-CATENIN ON THE EPIDERMAL GROWTH FACTOR RECEPTOR SIGNALING AND LOCALIZATION IN BREAST CANCER . [Internet] [Doctoral dissertation]. University of Arizona; 2010. [cited 2021 Mar 04]. Available from: http://hdl.handle.net/10150/194738.

Council of Science Editors:

Bitler BG. DETERMINING THE ROLE OF MUC1 AND BETA-CATENIN ON THE EPIDERMAL GROWTH FACTOR RECEPTOR SIGNALING AND LOCALIZATION IN BREAST CANCER . [Doctoral Dissertation]. University of Arizona; 2010. Available from: http://hdl.handle.net/10150/194738


University of Arizona

20. Thompson, Airlia Camille Simone. THE ROLE OF DIFFERENT ADIPOCYTE SIZE POPULATIONS IN THE MEDIATION OF OBESITY-RELATED INSULIN RESISTANCE AND INFLAMMATION .

Degree: 2008, University of Arizona

 Insulin resistance, the cause of type 2 diabetes mellitus, is intimately linked to the dysregulation of adipose tissue. Recent decades have witnessed the discovery and… (more)

Subjects/Keywords: Adipocyte; Adiponectin; Diabetes; Inflammation; Obesity

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APA (6th Edition):

Thompson, A. C. S. (2008). THE ROLE OF DIFFERENT ADIPOCYTE SIZE POPULATIONS IN THE MEDIATION OF OBESITY-RELATED INSULIN RESISTANCE AND INFLAMMATION . (Masters Thesis). University of Arizona. Retrieved from http://hdl.handle.net/10150/193412

Chicago Manual of Style (16th Edition):

Thompson, Airlia Camille Simone. “THE ROLE OF DIFFERENT ADIPOCYTE SIZE POPULATIONS IN THE MEDIATION OF OBESITY-RELATED INSULIN RESISTANCE AND INFLAMMATION .” 2008. Masters Thesis, University of Arizona. Accessed March 04, 2021. http://hdl.handle.net/10150/193412.

MLA Handbook (7th Edition):

Thompson, Airlia Camille Simone. “THE ROLE OF DIFFERENT ADIPOCYTE SIZE POPULATIONS IN THE MEDIATION OF OBESITY-RELATED INSULIN RESISTANCE AND INFLAMMATION .” 2008. Web. 04 Mar 2021.

Vancouver:

Thompson ACS. THE ROLE OF DIFFERENT ADIPOCYTE SIZE POPULATIONS IN THE MEDIATION OF OBESITY-RELATED INSULIN RESISTANCE AND INFLAMMATION . [Internet] [Masters thesis]. University of Arizona; 2008. [cited 2021 Mar 04]. Available from: http://hdl.handle.net/10150/193412.

Council of Science Editors:

Thompson ACS. THE ROLE OF DIFFERENT ADIPOCYTE SIZE POPULATIONS IN THE MEDIATION OF OBESITY-RELATED INSULIN RESISTANCE AND INFLAMMATION . [Masters Thesis]. University of Arizona; 2008. Available from: http://hdl.handle.net/10150/193412


University of Arizona

21. Stevens, Mark V. Distinct Functions of MEKK3 and MEKK4 in Heart Valve Morphogenesis .

Degree: 2008, University of Arizona

 Congenital heart defects (CHDs) occur in 5% of births. While gene mutations have been identified in CHD patients, not much is known about coordinated signaling… (more)

Subjects/Keywords: Endocardial cushion; Epithelial to Mesenchymal Transition; Heart; MEKK3; MEKK4; Valve

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APA (6th Edition):

Stevens, M. V. (2008). Distinct Functions of MEKK3 and MEKK4 in Heart Valve Morphogenesis . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/194848

Chicago Manual of Style (16th Edition):

Stevens, Mark V. “Distinct Functions of MEKK3 and MEKK4 in Heart Valve Morphogenesis .” 2008. Doctoral Dissertation, University of Arizona. Accessed March 04, 2021. http://hdl.handle.net/10150/194848.

MLA Handbook (7th Edition):

Stevens, Mark V. “Distinct Functions of MEKK3 and MEKK4 in Heart Valve Morphogenesis .” 2008. Web. 04 Mar 2021.

Vancouver:

Stevens MV. Distinct Functions of MEKK3 and MEKK4 in Heart Valve Morphogenesis . [Internet] [Doctoral dissertation]. University of Arizona; 2008. [cited 2021 Mar 04]. Available from: http://hdl.handle.net/10150/194848.

Council of Science Editors:

Stevens MV. Distinct Functions of MEKK3 and MEKK4 in Heart Valve Morphogenesis . [Doctoral Dissertation]. University of Arizona; 2008. Available from: http://hdl.handle.net/10150/194848


University of Arizona

22. Flowers, Margaret. In Vitro and In Vivo Effects of Conjugated Linoleic Acid on Mammary Tumorigenesis .

Degree: 2008, University of Arizona

 Conjugated linoleic acid (CLA) exhibits multiple biological and molecular activities that have made it the subject of considerable nutrition-related research. Numerous studies support broad acting… (more)

Subjects/Keywords: breast cancer; conjugate linoleic acid; ERBB2; FASN; HER2/neu; PyV-mT

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APA (6th Edition):

Flowers, M. (2008). In Vitro and In Vivo Effects of Conjugated Linoleic Acid on Mammary Tumorigenesis . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/195806

Chicago Manual of Style (16th Edition):

Flowers, Margaret. “In Vitro and In Vivo Effects of Conjugated Linoleic Acid on Mammary Tumorigenesis .” 2008. Doctoral Dissertation, University of Arizona. Accessed March 04, 2021. http://hdl.handle.net/10150/195806.

MLA Handbook (7th Edition):

Flowers, Margaret. “In Vitro and In Vivo Effects of Conjugated Linoleic Acid on Mammary Tumorigenesis .” 2008. Web. 04 Mar 2021.

Vancouver:

Flowers M. In Vitro and In Vivo Effects of Conjugated Linoleic Acid on Mammary Tumorigenesis . [Internet] [Doctoral dissertation]. University of Arizona; 2008. [cited 2021 Mar 04]. Available from: http://hdl.handle.net/10150/195806.

Council of Science Editors:

Flowers M. In Vitro and In Vivo Effects of Conjugated Linoleic Acid on Mammary Tumorigenesis . [Doctoral Dissertation]. University of Arizona; 2008. Available from: http://hdl.handle.net/10150/195806

.