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You searched for +publisher:"University of Arizona" +contributor:("Lybarger, Lonnie"). Showing records 1 – 30 of 48 total matches.

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University of Arizona

1. Bhagwandin, Candida B. The Regulatory Mechanisms Governing The E3 Ubiquitin Ligase, Membrane-Associated RING-CH1 (MARCH1), And The Consequences Of Dysregulation On Metabolism .

Degree: 2014, University of Arizona

 Membrane-Associated RING-CH1 (MARCH1) is a highly-conserved E3 ubiquitin ligase identified as a potent regulator of the immune modulatory molecules, Major Histocompatibility Complex II (MHC-II), and… (more)

Subjects/Keywords: Cell Biology & Anatomy

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APA (6th Edition):

Bhagwandin, C. B. (2014). The Regulatory Mechanisms Governing The E3 Ubiquitin Ligase, Membrane-Associated RING-CH1 (MARCH1), And The Consequences Of Dysregulation On Metabolism . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/333037

Chicago Manual of Style (16th Edition):

Bhagwandin, Candida B. “The Regulatory Mechanisms Governing The E3 Ubiquitin Ligase, Membrane-Associated RING-CH1 (MARCH1), And The Consequences Of Dysregulation On Metabolism .” 2014. Doctoral Dissertation, University of Arizona. Accessed November 22, 2019. http://hdl.handle.net/10150/333037.

MLA Handbook (7th Edition):

Bhagwandin, Candida B. “The Regulatory Mechanisms Governing The E3 Ubiquitin Ligase, Membrane-Associated RING-CH1 (MARCH1), And The Consequences Of Dysregulation On Metabolism .” 2014. Web. 22 Nov 2019.

Vancouver:

Bhagwandin CB. The Regulatory Mechanisms Governing The E3 Ubiquitin Ligase, Membrane-Associated RING-CH1 (MARCH1), And The Consequences Of Dysregulation On Metabolism . [Internet] [Doctoral dissertation]. University of Arizona; 2014. [cited 2019 Nov 22]. Available from: http://hdl.handle.net/10150/333037.

Council of Science Editors:

Bhagwandin CB. The Regulatory Mechanisms Governing The E3 Ubiquitin Ligase, Membrane-Associated RING-CH1 (MARCH1), And The Consequences Of Dysregulation On Metabolism . [Doctoral Dissertation]. University of Arizona; 2014. Available from: http://hdl.handle.net/10150/333037


University of Arizona

2. Millner, Rodrick Antonio. Investigating How High Ferritin Levels are Associated with Gestational Diabetes .

Degree: 2019, University of Arizona

 Gestational diabetes mellites (GDM) is now a widely recognized condition that affects over 10% of mothers within the United States. The CDC estimates between 2-10%… (more)

Subjects/Keywords: Ferritin; Gestational Diabetes; Hepcidin; Interleukin-6

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APA (6th Edition):

Millner, R. A. (2019). Investigating How High Ferritin Levels are Associated with Gestational Diabetes . (Masters Thesis). University of Arizona. Retrieved from http://hdl.handle.net/10150/632548

Chicago Manual of Style (16th Edition):

Millner, Rodrick Antonio. “Investigating How High Ferritin Levels are Associated with Gestational Diabetes .” 2019. Masters Thesis, University of Arizona. Accessed November 22, 2019. http://hdl.handle.net/10150/632548.

MLA Handbook (7th Edition):

Millner, Rodrick Antonio. “Investigating How High Ferritin Levels are Associated with Gestational Diabetes .” 2019. Web. 22 Nov 2019.

Vancouver:

Millner RA. Investigating How High Ferritin Levels are Associated with Gestational Diabetes . [Internet] [Masters thesis]. University of Arizona; 2019. [cited 2019 Nov 22]. Available from: http://hdl.handle.net/10150/632548.

Council of Science Editors:

Millner RA. Investigating How High Ferritin Levels are Associated with Gestational Diabetes . [Masters Thesis]. University of Arizona; 2019. Available from: http://hdl.handle.net/10150/632548


University of Arizona

3. Essif, Jacob Nathaniel. Targeting Polyploidy in Diffuse Large B-Cell Lymphoma .

Degree: 2019, University of Arizona

 Diffuse large B-cell lymphoma (DLBCL) accounts for thirty-one percent of B-cell Non-Hodgkin lymphomas (NHL) diagnosed in the United States (16). The current treatment is a… (more)

Subjects/Keywords: Alisertib; AurkinA; Aurora A Kinase; Diffuse Large B-Cell Lymphoma; Polyploidy; TPX2

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APA (6th Edition):

Essif, J. N. (2019). Targeting Polyploidy in Diffuse Large B-Cell Lymphoma . (Masters Thesis). University of Arizona. Retrieved from http://hdl.handle.net/10150/633227

Chicago Manual of Style (16th Edition):

Essif, Jacob Nathaniel. “Targeting Polyploidy in Diffuse Large B-Cell Lymphoma .” 2019. Masters Thesis, University of Arizona. Accessed November 22, 2019. http://hdl.handle.net/10150/633227.

MLA Handbook (7th Edition):

Essif, Jacob Nathaniel. “Targeting Polyploidy in Diffuse Large B-Cell Lymphoma .” 2019. Web. 22 Nov 2019.

Vancouver:

Essif JN. Targeting Polyploidy in Diffuse Large B-Cell Lymphoma . [Internet] [Masters thesis]. University of Arizona; 2019. [cited 2019 Nov 22]. Available from: http://hdl.handle.net/10150/633227.

Council of Science Editors:

Essif JN. Targeting Polyploidy in Diffuse Large B-Cell Lymphoma . [Masters Thesis]. University of Arizona; 2019. Available from: http://hdl.handle.net/10150/633227


University of Arizona

4. Likens, Jacob Andrew. The Pathophysiology of Chronic Stroke Infarcts: What Happens After Brain Tissue Dies? .

Degree: 2018, University of Arizona

 A stroke can occur when blood flow to a specific area of the brain is interrupted. There has been extensive research in both animal models… (more)

Subjects/Keywords: Alzheimer's disease; chronic; IDE; insulin degrading enzyme; liquefactive necrosis; stroke

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APA (6th Edition):

Likens, J. A. (2018). The Pathophysiology of Chronic Stroke Infarcts: What Happens After Brain Tissue Dies? . (Masters Thesis). University of Arizona. Retrieved from http://hdl.handle.net/10150/628067

Chicago Manual of Style (16th Edition):

Likens, Jacob Andrew. “The Pathophysiology of Chronic Stroke Infarcts: What Happens After Brain Tissue Dies? .” 2018. Masters Thesis, University of Arizona. Accessed November 22, 2019. http://hdl.handle.net/10150/628067.

MLA Handbook (7th Edition):

Likens, Jacob Andrew. “The Pathophysiology of Chronic Stroke Infarcts: What Happens After Brain Tissue Dies? .” 2018. Web. 22 Nov 2019.

Vancouver:

Likens JA. The Pathophysiology of Chronic Stroke Infarcts: What Happens After Brain Tissue Dies? . [Internet] [Masters thesis]. University of Arizona; 2018. [cited 2019 Nov 22]. Available from: http://hdl.handle.net/10150/628067.

Council of Science Editors:

Likens JA. The Pathophysiology of Chronic Stroke Infarcts: What Happens After Brain Tissue Dies? . [Masters Thesis]. University of Arizona; 2018. Available from: http://hdl.handle.net/10150/628067


University of Arizona

5. Diaz, David Andrew. Glyphosate Holds Potential to Induce Dysbiosis Associated Pathologies in Humans .

Degree: 2018, University of Arizona

 There is increasing evidence of a deep integration between microbes and human physiological function. With such extensive communication in both directions, it is arguable that… (more)

Subjects/Keywords: Dysbiosis; Glyphosate; Inflammation; Metabolism; Microbiome

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APA (6th Edition):

Diaz, D. A. (2018). Glyphosate Holds Potential to Induce Dysbiosis Associated Pathologies in Humans . (Masters Thesis). University of Arizona. Retrieved from http://hdl.handle.net/10150/628080

Chicago Manual of Style (16th Edition):

Diaz, David Andrew. “Glyphosate Holds Potential to Induce Dysbiosis Associated Pathologies in Humans .” 2018. Masters Thesis, University of Arizona. Accessed November 22, 2019. http://hdl.handle.net/10150/628080.

MLA Handbook (7th Edition):

Diaz, David Andrew. “Glyphosate Holds Potential to Induce Dysbiosis Associated Pathologies in Humans .” 2018. Web. 22 Nov 2019.

Vancouver:

Diaz DA. Glyphosate Holds Potential to Induce Dysbiosis Associated Pathologies in Humans . [Internet] [Masters thesis]. University of Arizona; 2018. [cited 2019 Nov 22]. Available from: http://hdl.handle.net/10150/628080.

Council of Science Editors:

Diaz DA. Glyphosate Holds Potential to Induce Dysbiosis Associated Pathologies in Humans . [Masters Thesis]. University of Arizona; 2018. Available from: http://hdl.handle.net/10150/628080


University of Arizona

6. Jabbour, Maurice E. REGULATION AND FUNCTION OF MARCH1: MODULATION OF IMMUNITY THROUGH UBIQUITINATION .

Degree: 2010, University of Arizona

 The activation of the immune system, particularly adaptive immunity, in response to a pathogen ( e.g. viruses) relies on complex network of cell interactions including… (more)

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APA (6th Edition):

Jabbour, M. E. (2010). REGULATION AND FUNCTION OF MARCH1: MODULATION OF IMMUNITY THROUGH UBIQUITINATION . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/196151

Chicago Manual of Style (16th Edition):

Jabbour, Maurice E. “REGULATION AND FUNCTION OF MARCH1: MODULATION OF IMMUNITY THROUGH UBIQUITINATION .” 2010. Doctoral Dissertation, University of Arizona. Accessed November 22, 2019. http://hdl.handle.net/10150/196151.

MLA Handbook (7th Edition):

Jabbour, Maurice E. “REGULATION AND FUNCTION OF MARCH1: MODULATION OF IMMUNITY THROUGH UBIQUITINATION .” 2010. Web. 22 Nov 2019.

Vancouver:

Jabbour ME. REGULATION AND FUNCTION OF MARCH1: MODULATION OF IMMUNITY THROUGH UBIQUITINATION . [Internet] [Doctoral dissertation]. University of Arizona; 2010. [cited 2019 Nov 22]. Available from: http://hdl.handle.net/10150/196151.

Council of Science Editors:

Jabbour ME. REGULATION AND FUNCTION OF MARCH1: MODULATION OF IMMUNITY THROUGH UBIQUITINATION . [Doctoral Dissertation]. University of Arizona; 2010. Available from: http://hdl.handle.net/10150/196151


University of Arizona

7. Babaria, Arati. Molecular Mechanisms that Underlie Duchenne Muscular Dystrophy .

Degree: 2016, University of Arizona

 Duchenne muscular dystrophy is an inherited, X-linked recessive skeletal muscle disorder that is characterized by mutations in the dystrophin gene [1]. Therefore, the disease affects… (more)

Subjects/Keywords: Muscular Dystrophy; Cellular and Molecular Medicine; Duchenne

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APA (6th Edition):

Babaria, A. (2016). Molecular Mechanisms that Underlie Duchenne Muscular Dystrophy . (Masters Thesis). University of Arizona. Retrieved from http://hdl.handle.net/10150/612573

Chicago Manual of Style (16th Edition):

Babaria, Arati. “Molecular Mechanisms that Underlie Duchenne Muscular Dystrophy .” 2016. Masters Thesis, University of Arizona. Accessed November 22, 2019. http://hdl.handle.net/10150/612573.

MLA Handbook (7th Edition):

Babaria, Arati. “Molecular Mechanisms that Underlie Duchenne Muscular Dystrophy .” 2016. Web. 22 Nov 2019.

Vancouver:

Babaria A. Molecular Mechanisms that Underlie Duchenne Muscular Dystrophy . [Internet] [Masters thesis]. University of Arizona; 2016. [cited 2019 Nov 22]. Available from: http://hdl.handle.net/10150/612573.

Council of Science Editors:

Babaria A. Molecular Mechanisms that Underlie Duchenne Muscular Dystrophy . [Masters Thesis]. University of Arizona; 2016. Available from: http://hdl.handle.net/10150/612573


University of Arizona

8. Mogor, Odinaka P. Sickle Cell Anemia: The Effects of Hydroxyurea on Red Blood Cell Polymerization Reversal .

Degree: 2017, University of Arizona

 Sickle cell anemia – the most common form of sickle cell disease (SCD) – is a debilitating condition that presents with health complications ranging from… (more)

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APA (6th Edition):

Mogor, O. P. (2017). Sickle Cell Anemia: The Effects of Hydroxyurea on Red Blood Cell Polymerization Reversal . (Masters Thesis). University of Arizona. Retrieved from http://hdl.handle.net/10150/626380

Chicago Manual of Style (16th Edition):

Mogor, Odinaka P. “Sickle Cell Anemia: The Effects of Hydroxyurea on Red Blood Cell Polymerization Reversal .” 2017. Masters Thesis, University of Arizona. Accessed November 22, 2019. http://hdl.handle.net/10150/626380.

MLA Handbook (7th Edition):

Mogor, Odinaka P. “Sickle Cell Anemia: The Effects of Hydroxyurea on Red Blood Cell Polymerization Reversal .” 2017. Web. 22 Nov 2019.

Vancouver:

Mogor OP. Sickle Cell Anemia: The Effects of Hydroxyurea on Red Blood Cell Polymerization Reversal . [Internet] [Masters thesis]. University of Arizona; 2017. [cited 2019 Nov 22]. Available from: http://hdl.handle.net/10150/626380.

Council of Science Editors:

Mogor OP. Sickle Cell Anemia: The Effects of Hydroxyurea on Red Blood Cell Polymerization Reversal . [Masters Thesis]. University of Arizona; 2017. Available from: http://hdl.handle.net/10150/626380


University of Arizona

9. Calderon, Stephanie Marie. CC16 Depletion in the Lung Due to Early Life Biomass Exposures .

Degree: 2019, University of Arizona

 Millions of people across the globe are affected by respiratory diseases that include chronic obstructive pulmonary disease (COPD), asthma, emphysema, as well as cancer. COPD… (more)

Subjects/Keywords: Lung; CC16; Club cells; Biomass exposure

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APA (6th Edition):

Calderon, S. M. (2019). CC16 Depletion in the Lung Due to Early Life Biomass Exposures . (Masters Thesis). University of Arizona. Retrieved from http://hdl.handle.net/10150/631918

Chicago Manual of Style (16th Edition):

Calderon, Stephanie Marie. “CC16 Depletion in the Lung Due to Early Life Biomass Exposures .” 2019. Masters Thesis, University of Arizona. Accessed November 22, 2019. http://hdl.handle.net/10150/631918.

MLA Handbook (7th Edition):

Calderon, Stephanie Marie. “CC16 Depletion in the Lung Due to Early Life Biomass Exposures .” 2019. Web. 22 Nov 2019.

Vancouver:

Calderon SM. CC16 Depletion in the Lung Due to Early Life Biomass Exposures . [Internet] [Masters thesis]. University of Arizona; 2019. [cited 2019 Nov 22]. Available from: http://hdl.handle.net/10150/631918.

Council of Science Editors:

Calderon SM. CC16 Depletion in the Lung Due to Early Life Biomass Exposures . [Masters Thesis]. University of Arizona; 2019. Available from: http://hdl.handle.net/10150/631918


University of Arizona

10. Padilla-Rodriguez, Marco. Regulation of Cancer Cell Invasion by the Estrogen Receptor in ER+ Breast Cancer .

Degree: 2018, University of Arizona

 Estrogen receptor-positive (ER+) breast tumors account for 75% of diagnosed breast cancer in the United States. These breast tumors are defined by their dependency on… (more)

Subjects/Keywords: Actin Remodeling; Breast Cancer; Estrogen Receptor; EVL; Invasion

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APA (6th Edition):

Padilla-Rodriguez, M. (2018). Regulation of Cancer Cell Invasion by the Estrogen Receptor in ER+ Breast Cancer . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/628456

Chicago Manual of Style (16th Edition):

Padilla-Rodriguez, Marco. “Regulation of Cancer Cell Invasion by the Estrogen Receptor in ER+ Breast Cancer .” 2018. Doctoral Dissertation, University of Arizona. Accessed November 22, 2019. http://hdl.handle.net/10150/628456.

MLA Handbook (7th Edition):

Padilla-Rodriguez, Marco. “Regulation of Cancer Cell Invasion by the Estrogen Receptor in ER+ Breast Cancer .” 2018. Web. 22 Nov 2019.

Vancouver:

Padilla-Rodriguez M. Regulation of Cancer Cell Invasion by the Estrogen Receptor in ER+ Breast Cancer . [Internet] [Doctoral dissertation]. University of Arizona; 2018. [cited 2019 Nov 22]. Available from: http://hdl.handle.net/10150/628456.

Council of Science Editors:

Padilla-Rodriguez M. Regulation of Cancer Cell Invasion by the Estrogen Receptor in ER+ Breast Cancer . [Doctoral Dissertation]. University of Arizona; 2018. Available from: http://hdl.handle.net/10150/628456


University of Arizona

11. Puleo, Julieann Isabel. Mechanosensing during Directed Cell Migration Requires Dynamic Actin Polymerization at Focal Adhesions .

Degree: 2019, University of Arizona

 The tumor microenvironment consists of a complex arrangement of both biochemical and mechanical components, which collectively regulate fundamental cellular processes including cell migration. The directional… (more)

Subjects/Keywords: chemotaxis; durotaxis; invasion; mechanosensing; microenvironment; migration

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APA (6th Edition):

Puleo, J. I. (2019). Mechanosensing during Directed Cell Migration Requires Dynamic Actin Polymerization at Focal Adhesions . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/631909

Chicago Manual of Style (16th Edition):

Puleo, Julieann Isabel. “Mechanosensing during Directed Cell Migration Requires Dynamic Actin Polymerization at Focal Adhesions .” 2019. Doctoral Dissertation, University of Arizona. Accessed November 22, 2019. http://hdl.handle.net/10150/631909.

MLA Handbook (7th Edition):

Puleo, Julieann Isabel. “Mechanosensing during Directed Cell Migration Requires Dynamic Actin Polymerization at Focal Adhesions .” 2019. Web. 22 Nov 2019.

Vancouver:

Puleo JI. Mechanosensing during Directed Cell Migration Requires Dynamic Actin Polymerization at Focal Adhesions . [Internet] [Doctoral dissertation]. University of Arizona; 2019. [cited 2019 Nov 22]. Available from: http://hdl.handle.net/10150/631909.

Council of Science Editors:

Puleo JI. Mechanosensing during Directed Cell Migration Requires Dynamic Actin Polymerization at Focal Adhesions . [Doctoral Dissertation]. University of Arizona; 2019. Available from: http://hdl.handle.net/10150/631909


University of Arizona

12. Caviness, Katie Elizabeth. Complex Gene Expression And Interplay Of The UL136 Protein Isoforms Influence Human Cytomegalovirus Persistence .

Degree: 2015, University of Arizona

 Human cytomegalovirus (HCMV), a beta herpesvirus, persists indefinitely in the human host through a life-long, latent infection. HCMV is associated with life threatening pathologies in… (more)

Subjects/Keywords: Gene Expression; Herpesvirus; Isoforms; Latency; Persistence; Genetics; Cytomegalovirus

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APA (6th Edition):

Caviness, K. E. (2015). Complex Gene Expression And Interplay Of The UL136 Protein Isoforms Influence Human Cytomegalovirus Persistence . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/556472

Chicago Manual of Style (16th Edition):

Caviness, Katie Elizabeth. “Complex Gene Expression And Interplay Of The UL136 Protein Isoforms Influence Human Cytomegalovirus Persistence .” 2015. Doctoral Dissertation, University of Arizona. Accessed November 22, 2019. http://hdl.handle.net/10150/556472.

MLA Handbook (7th Edition):

Caviness, Katie Elizabeth. “Complex Gene Expression And Interplay Of The UL136 Protein Isoforms Influence Human Cytomegalovirus Persistence .” 2015. Web. 22 Nov 2019.

Vancouver:

Caviness KE. Complex Gene Expression And Interplay Of The UL136 Protein Isoforms Influence Human Cytomegalovirus Persistence . [Internet] [Doctoral dissertation]. University of Arizona; 2015. [cited 2019 Nov 22]. Available from: http://hdl.handle.net/10150/556472.

Council of Science Editors:

Caviness KE. Complex Gene Expression And Interplay Of The UL136 Protein Isoforms Influence Human Cytomegalovirus Persistence . [Doctoral Dissertation]. University of Arizona; 2015. Available from: http://hdl.handle.net/10150/556472


University of Arizona

13. Grainger, Lora Ann. Characterization of the Transcripts that Encode pUL138, a Latency Determinant, During Human Cytomegalovirus Infection .

Degree: 2010, University of Arizona

 Mechanisms involved in the establishment of HCMV latency are poorly understood, however, work in our laboratory has demonstrated the ULb' encoded protein, pUL138, as the… (more)

Subjects/Keywords: Human Cytomegalovirus; IRES; Latency; pUL138; Translation Initiation; Type I IFN

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APA (6th Edition):

Grainger, L. A. (2010). Characterization of the Transcripts that Encode pUL138, a Latency Determinant, During Human Cytomegalovirus Infection . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/195915

Chicago Manual of Style (16th Edition):

Grainger, Lora Ann. “Characterization of the Transcripts that Encode pUL138, a Latency Determinant, During Human Cytomegalovirus Infection .” 2010. Doctoral Dissertation, University of Arizona. Accessed November 22, 2019. http://hdl.handle.net/10150/195915.

MLA Handbook (7th Edition):

Grainger, Lora Ann. “Characterization of the Transcripts that Encode pUL138, a Latency Determinant, During Human Cytomegalovirus Infection .” 2010. Web. 22 Nov 2019.

Vancouver:

Grainger LA. Characterization of the Transcripts that Encode pUL138, a Latency Determinant, During Human Cytomegalovirus Infection . [Internet] [Doctoral dissertation]. University of Arizona; 2010. [cited 2019 Nov 22]. Available from: http://hdl.handle.net/10150/195915.

Council of Science Editors:

Grainger LA. Characterization of the Transcripts that Encode pUL138, a Latency Determinant, During Human Cytomegalovirus Infection . [Doctoral Dissertation]. University of Arizona; 2010. Available from: http://hdl.handle.net/10150/195915


University of Arizona

14. Banuelos, Alma. CDX2 Alone Plays an Important Role in Epithelial Phenotypic Changes in Barrett's Metaplasia in the Esophagus, and Esophageal Submucosal Glands Seem to Be the Predominant Cell of Origin for Barrett’s Esophagus .

Degree: 2019, University of Arizona

 Gastroesophageal Reflux Disease (GERD) is a long-known disorder caused by back flow of acid (also known as acid reflux) from the stomach into the esophagus.… (more)

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APA (6th Edition):

Banuelos, A. (2019). CDX2 Alone Plays an Important Role in Epithelial Phenotypic Changes in Barrett's Metaplasia in the Esophagus, and Esophageal Submucosal Glands Seem to Be the Predominant Cell of Origin for Barrett’s Esophagus . (Masters Thesis). University of Arizona. Retrieved from http://hdl.handle.net/10150/634289

Chicago Manual of Style (16th Edition):

Banuelos, Alma. “CDX2 Alone Plays an Important Role in Epithelial Phenotypic Changes in Barrett's Metaplasia in the Esophagus, and Esophageal Submucosal Glands Seem to Be the Predominant Cell of Origin for Barrett’s Esophagus .” 2019. Masters Thesis, University of Arizona. Accessed November 22, 2019. http://hdl.handle.net/10150/634289.

MLA Handbook (7th Edition):

Banuelos, Alma. “CDX2 Alone Plays an Important Role in Epithelial Phenotypic Changes in Barrett's Metaplasia in the Esophagus, and Esophageal Submucosal Glands Seem to Be the Predominant Cell of Origin for Barrett’s Esophagus .” 2019. Web. 22 Nov 2019.

Vancouver:

Banuelos A. CDX2 Alone Plays an Important Role in Epithelial Phenotypic Changes in Barrett's Metaplasia in the Esophagus, and Esophageal Submucosal Glands Seem to Be the Predominant Cell of Origin for Barrett’s Esophagus . [Internet] [Masters thesis]. University of Arizona; 2019. [cited 2019 Nov 22]. Available from: http://hdl.handle.net/10150/634289.

Council of Science Editors:

Banuelos A. CDX2 Alone Plays an Important Role in Epithelial Phenotypic Changes in Barrett's Metaplasia in the Esophagus, and Esophageal Submucosal Glands Seem to Be the Predominant Cell of Origin for Barrett’s Esophagus . [Masters Thesis]. University of Arizona; 2019. Available from: http://hdl.handle.net/10150/634289


University of Arizona

15. Ayala, Dilia. The Metabolic Relationship between Type 2 Diabetes and Alzheimer's Disease .

Degree: 2019, University of Arizona

 Regulation of blood glucose is responsible for maintaining a continuous supply of energy moving throughout the human body; excess glucose is toxic, while insufficient glucose… (more)

Subjects/Keywords: AD; Alzheimer's Disease; Amyloid plaques; insulin resistance; T2D; Tau

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APA (6th Edition):

Ayala, D. (2019). The Metabolic Relationship between Type 2 Diabetes and Alzheimer's Disease . (Masters Thesis). University of Arizona. Retrieved from http://hdl.handle.net/10150/634290

Chicago Manual of Style (16th Edition):

Ayala, Dilia. “The Metabolic Relationship between Type 2 Diabetes and Alzheimer's Disease .” 2019. Masters Thesis, University of Arizona. Accessed November 22, 2019. http://hdl.handle.net/10150/634290.

MLA Handbook (7th Edition):

Ayala, Dilia. “The Metabolic Relationship between Type 2 Diabetes and Alzheimer's Disease .” 2019. Web. 22 Nov 2019.

Vancouver:

Ayala D. The Metabolic Relationship between Type 2 Diabetes and Alzheimer's Disease . [Internet] [Masters thesis]. University of Arizona; 2019. [cited 2019 Nov 22]. Available from: http://hdl.handle.net/10150/634290.

Council of Science Editors:

Ayala D. The Metabolic Relationship between Type 2 Diabetes and Alzheimer's Disease . [Masters Thesis]. University of Arizona; 2019. Available from: http://hdl.handle.net/10150/634290


University of Arizona

16. Tseng, Roger Sean. High-risk HPV: From Infection to Cervical Cancer Progression .

Degree: 2015, University of Arizona

 Human papillomaviruses (HPV) are small non-enveloped viruses that infect basal cells. Most HPV infections are mild and develop warts at the site of infection. However,… (more)

Subjects/Keywords: Cancer Biology

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APA (6th Edition):

Tseng, R. S. (2015). High-risk HPV: From Infection to Cervical Cancer Progression . (Masters Thesis). University of Arizona. Retrieved from http://hdl.handle.net/10150/604865

Chicago Manual of Style (16th Edition):

Tseng, Roger Sean. “High-risk HPV: From Infection to Cervical Cancer Progression .” 2015. Masters Thesis, University of Arizona. Accessed November 22, 2019. http://hdl.handle.net/10150/604865.

MLA Handbook (7th Edition):

Tseng, Roger Sean. “High-risk HPV: From Infection to Cervical Cancer Progression .” 2015. Web. 22 Nov 2019.

Vancouver:

Tseng RS. High-risk HPV: From Infection to Cervical Cancer Progression . [Internet] [Masters thesis]. University of Arizona; 2015. [cited 2019 Nov 22]. Available from: http://hdl.handle.net/10150/604865.

Council of Science Editors:

Tseng RS. High-risk HPV: From Infection to Cervical Cancer Progression . [Masters Thesis]. University of Arizona; 2015. Available from: http://hdl.handle.net/10150/604865


University of Arizona

17. Li, Shuaizhi. Cytosolic Glutathione Reducing Potential is Important for Membrane Penetration of HPV16 at the Trans-Golgi Network .

Degree: 2016, University of Arizona

 High-risk human papillomaviruses (HPVs) cause 5% of all human cancers worldwide. The HPV capsid consists of 72 disulfide-linked pentamers of major capsid protein L1 and… (more)

Subjects/Keywords: HPV16; Minor capsid protein L2; Cellular and Molecular Medicine; Cytosolic glutathione

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APA (6th Edition):

Li, S. (2016). Cytosolic Glutathione Reducing Potential is Important for Membrane Penetration of HPV16 at the Trans-Golgi Network . (Masters Thesis). University of Arizona. Retrieved from http://hdl.handle.net/10150/612410

Chicago Manual of Style (16th Edition):

Li, Shuaizhi. “Cytosolic Glutathione Reducing Potential is Important for Membrane Penetration of HPV16 at the Trans-Golgi Network .” 2016. Masters Thesis, University of Arizona. Accessed November 22, 2019. http://hdl.handle.net/10150/612410.

MLA Handbook (7th Edition):

Li, Shuaizhi. “Cytosolic Glutathione Reducing Potential is Important for Membrane Penetration of HPV16 at the Trans-Golgi Network .” 2016. Web. 22 Nov 2019.

Vancouver:

Li S. Cytosolic Glutathione Reducing Potential is Important for Membrane Penetration of HPV16 at the Trans-Golgi Network . [Internet] [Masters thesis]. University of Arizona; 2016. [cited 2019 Nov 22]. Available from: http://hdl.handle.net/10150/612410.

Council of Science Editors:

Li S. Cytosolic Glutathione Reducing Potential is Important for Membrane Penetration of HPV16 at the Trans-Golgi Network . [Masters Thesis]. University of Arizona; 2016. Available from: http://hdl.handle.net/10150/612410


University of Arizona

18. Paluszcyk, Chana Renee. The Future of Myasthenia Gravis: Exploring the Onset, Progression and Implications of Disease .

Degree: 2016, University of Arizona

 Myasthenia gravis (MG) is an autoimmune disease whose name means "grave muscular weakness". MG is a rare disease affecting only 200-400 persons per million and… (more)

Subjects/Keywords: Complement system; Myasthenia gravis; nicotinic acetylcholine receptor; Cellular and Molecular Medicine; Autoimmune

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APA (6th Edition):

Paluszcyk, C. R. (2016). The Future of Myasthenia Gravis: Exploring the Onset, Progression and Implications of Disease . (Masters Thesis). University of Arizona. Retrieved from http://hdl.handle.net/10150/613371

Chicago Manual of Style (16th Edition):

Paluszcyk, Chana Renee. “The Future of Myasthenia Gravis: Exploring the Onset, Progression and Implications of Disease .” 2016. Masters Thesis, University of Arizona. Accessed November 22, 2019. http://hdl.handle.net/10150/613371.

MLA Handbook (7th Edition):

Paluszcyk, Chana Renee. “The Future of Myasthenia Gravis: Exploring the Onset, Progression and Implications of Disease .” 2016. Web. 22 Nov 2019.

Vancouver:

Paluszcyk CR. The Future of Myasthenia Gravis: Exploring the Onset, Progression and Implications of Disease . [Internet] [Masters thesis]. University of Arizona; 2016. [cited 2019 Nov 22]. Available from: http://hdl.handle.net/10150/613371.

Council of Science Editors:

Paluszcyk CR. The Future of Myasthenia Gravis: Exploring the Onset, Progression and Implications of Disease . [Masters Thesis]. University of Arizona; 2016. Available from: http://hdl.handle.net/10150/613371


University of Arizona

19. Abdelhabib, Mohamed. Causes of Organ Rejection in Kidney Transplantation and a New Proposed Strategy to Improve Survival of the Graft .

Degree: 2018, University of Arizona

 The kidney is an essential organ that serves a crucial role in preserving homeostasis by filtering blood, regulating fluid levels and maintaining acid/base balance. Any… (more)

Subjects/Keywords: Immune evasion; Kidney failure; Kidney transplant; Mechanism of transplant rejection; Transplant rejection

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APA (6th Edition):

Abdelhabib, M. (2018). Causes of Organ Rejection in Kidney Transplantation and a New Proposed Strategy to Improve Survival of the Graft . (Masters Thesis). University of Arizona. Retrieved from http://hdl.handle.net/10150/628057

Chicago Manual of Style (16th Edition):

Abdelhabib, Mohamed. “Causes of Organ Rejection in Kidney Transplantation and a New Proposed Strategy to Improve Survival of the Graft .” 2018. Masters Thesis, University of Arizona. Accessed November 22, 2019. http://hdl.handle.net/10150/628057.

MLA Handbook (7th Edition):

Abdelhabib, Mohamed. “Causes of Organ Rejection in Kidney Transplantation and a New Proposed Strategy to Improve Survival of the Graft .” 2018. Web. 22 Nov 2019.

Vancouver:

Abdelhabib M. Causes of Organ Rejection in Kidney Transplantation and a New Proposed Strategy to Improve Survival of the Graft . [Internet] [Masters thesis]. University of Arizona; 2018. [cited 2019 Nov 22]. Available from: http://hdl.handle.net/10150/628057.

Council of Science Editors:

Abdelhabib M. Causes of Organ Rejection in Kidney Transplantation and a New Proposed Strategy to Improve Survival of the Graft . [Masters Thesis]. University of Arizona; 2018. Available from: http://hdl.handle.net/10150/628057


University of Arizona

20. Chung, Amanda. Liquefaction of the Brain Following Stroke Shares Multiple Characteristics with Atherosclerosis and Mediates Secondary Neurodegeneration in an Osteopontin-Dependent Mechanism .

Degree: 2018, University of Arizona

 The response to ischemic injury in the brain is different to the response to ischemic injury in other organs and tissues. Almost exclusive to the… (more)

Subjects/Keywords: Atherosclerosis; Liquefaction; Osteopontin; Stroke

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APA (6th Edition):

Chung, A. (2018). Liquefaction of the Brain Following Stroke Shares Multiple Characteristics with Atherosclerosis and Mediates Secondary Neurodegeneration in an Osteopontin-Dependent Mechanism . (Masters Thesis). University of Arizona. Retrieved from http://hdl.handle.net/10150/628068

Chicago Manual of Style (16th Edition):

Chung, Amanda. “Liquefaction of the Brain Following Stroke Shares Multiple Characteristics with Atherosclerosis and Mediates Secondary Neurodegeneration in an Osteopontin-Dependent Mechanism .” 2018. Masters Thesis, University of Arizona. Accessed November 22, 2019. http://hdl.handle.net/10150/628068.

MLA Handbook (7th Edition):

Chung, Amanda. “Liquefaction of the Brain Following Stroke Shares Multiple Characteristics with Atherosclerosis and Mediates Secondary Neurodegeneration in an Osteopontin-Dependent Mechanism .” 2018. Web. 22 Nov 2019.

Vancouver:

Chung A. Liquefaction of the Brain Following Stroke Shares Multiple Characteristics with Atherosclerosis and Mediates Secondary Neurodegeneration in an Osteopontin-Dependent Mechanism . [Internet] [Masters thesis]. University of Arizona; 2018. [cited 2019 Nov 22]. Available from: http://hdl.handle.net/10150/628068.

Council of Science Editors:

Chung A. Liquefaction of the Brain Following Stroke Shares Multiple Characteristics with Atherosclerosis and Mediates Secondary Neurodegeneration in an Osteopontin-Dependent Mechanism . [Masters Thesis]. University of Arizona; 2018. Available from: http://hdl.handle.net/10150/628068


University of Arizona

21. Centuori, Sara Mozelle. NEGATIVE REGULATION OF REGULATORY T CELLS BY MYELOID-DERIVED SUPPRESSOR CELLS IN CANCER .

Degree: 2011, University of Arizona

 Myeloid-derived suppressor cells (MDSC) and regulatory T cells (Treg) play an essential role in the immunosuppressive networks that contribute to tumor immune evasion. The mechanisms… (more)

Subjects/Keywords: Cancer; immunosuppressive cells; MDSC; Myeloid-derived suppressor cells; Regulatory T cells; Treg

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APA (6th Edition):

Centuori, S. M. (2011). NEGATIVE REGULATION OF REGULATORY T CELLS BY MYELOID-DERIVED SUPPRESSOR CELLS IN CANCER . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/145099

Chicago Manual of Style (16th Edition):

Centuori, Sara Mozelle. “NEGATIVE REGULATION OF REGULATORY T CELLS BY MYELOID-DERIVED SUPPRESSOR CELLS IN CANCER .” 2011. Doctoral Dissertation, University of Arizona. Accessed November 22, 2019. http://hdl.handle.net/10150/145099.

MLA Handbook (7th Edition):

Centuori, Sara Mozelle. “NEGATIVE REGULATION OF REGULATORY T CELLS BY MYELOID-DERIVED SUPPRESSOR CELLS IN CANCER .” 2011. Web. 22 Nov 2019.

Vancouver:

Centuori SM. NEGATIVE REGULATION OF REGULATORY T CELLS BY MYELOID-DERIVED SUPPRESSOR CELLS IN CANCER . [Internet] [Doctoral dissertation]. University of Arizona; 2011. [cited 2019 Nov 22]. Available from: http://hdl.handle.net/10150/145099.

Council of Science Editors:

Centuori SM. NEGATIVE REGULATION OF REGULATORY T CELLS BY MYELOID-DERIVED SUPPRESSOR CELLS IN CANCER . [Doctoral Dissertation]. University of Arizona; 2011. Available from: http://hdl.handle.net/10150/145099


University of Arizona

22. Felix, Krysta M. The Gut Microbiota Crosstalk with the Host Immune System in Health and Disease .

Degree: 2018, University of Arizona

 The gut microbiota both regulates and stimulates the immune system to impact many aspects of immunity, including the response to infection and the development of… (more)

Subjects/Keywords: arthritis; microbiota; neutrophil; P2X7; pneumonia; SFB

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APA (6th Edition):

Felix, K. M. (2018). The Gut Microbiota Crosstalk with the Host Immune System in Health and Disease . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/628475

Chicago Manual of Style (16th Edition):

Felix, Krysta M. “The Gut Microbiota Crosstalk with the Host Immune System in Health and Disease .” 2018. Doctoral Dissertation, University of Arizona. Accessed November 22, 2019. http://hdl.handle.net/10150/628475.

MLA Handbook (7th Edition):

Felix, Krysta M. “The Gut Microbiota Crosstalk with the Host Immune System in Health and Disease .” 2018. Web. 22 Nov 2019.

Vancouver:

Felix KM. The Gut Microbiota Crosstalk with the Host Immune System in Health and Disease . [Internet] [Doctoral dissertation]. University of Arizona; 2018. [cited 2019 Nov 22]. Available from: http://hdl.handle.net/10150/628475.

Council of Science Editors:

Felix KM. The Gut Microbiota Crosstalk with the Host Immune System in Health and Disease . [Doctoral Dissertation]. University of Arizona; 2018. Available from: http://hdl.handle.net/10150/628475


University of Arizona

23. Tuladhar, Shraddha. Toxoplasma Strain-specific Early Immune Responses Determines Sub-acute CNS Immune Responses .

Degree: 2018, University of Arizona

 Toxoplasma gondii is an obligate intracellular parasite that infects up to 1/3 of the world’s population. Genetically distinct Toxoplasma strains show differences in acute and… (more)

Subjects/Keywords: CNS; Host-pathogen; Immune Response; parasites; ROP16; Toxoplasma gondii

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APA (6th Edition):

Tuladhar, S. (2018). Toxoplasma Strain-specific Early Immune Responses Determines Sub-acute CNS Immune Responses . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/630215

Chicago Manual of Style (16th Edition):

Tuladhar, Shraddha. “Toxoplasma Strain-specific Early Immune Responses Determines Sub-acute CNS Immune Responses .” 2018. Doctoral Dissertation, University of Arizona. Accessed November 22, 2019. http://hdl.handle.net/10150/630215.

MLA Handbook (7th Edition):

Tuladhar, Shraddha. “Toxoplasma Strain-specific Early Immune Responses Determines Sub-acute CNS Immune Responses .” 2018. Web. 22 Nov 2019.

Vancouver:

Tuladhar S. Toxoplasma Strain-specific Early Immune Responses Determines Sub-acute CNS Immune Responses . [Internet] [Doctoral dissertation]. University of Arizona; 2018. [cited 2019 Nov 22]. Available from: http://hdl.handle.net/10150/630215.

Council of Science Editors:

Tuladhar S. Toxoplasma Strain-specific Early Immune Responses Determines Sub-acute CNS Immune Responses . [Doctoral Dissertation]. University of Arizona; 2018. Available from: http://hdl.handle.net/10150/630215


University of Arizona

24. Kim, Won J. Neisseria gonorrhoeae Survival in the Host .

Degree: 2018, University of Arizona

 A Gram-negative human pathogen Neisseria gonorrhoeae (Ngo), which causes sexually transmitted disease gonorrhea, infects hundreds of millions of people each year. Gonorrhea patients experience symptoms… (more)

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APA (6th Edition):

Kim, W. J. (2018). Neisseria gonorrhoeae Survival in the Host . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/631384

Chicago Manual of Style (16th Edition):

Kim, Won J. “Neisseria gonorrhoeae Survival in the Host .” 2018. Doctoral Dissertation, University of Arizona. Accessed November 22, 2019. http://hdl.handle.net/10150/631384.

MLA Handbook (7th Edition):

Kim, Won J. “Neisseria gonorrhoeae Survival in the Host .” 2018. Web. 22 Nov 2019.

Vancouver:

Kim WJ. Neisseria gonorrhoeae Survival in the Host . [Internet] [Doctoral dissertation]. University of Arizona; 2018. [cited 2019 Nov 22]. Available from: http://hdl.handle.net/10150/631384.

Council of Science Editors:

Kim WJ. Neisseria gonorrhoeae Survival in the Host . [Doctoral Dissertation]. University of Arizona; 2018. Available from: http://hdl.handle.net/10150/631384


University of Arizona

25. Renkema, Kristin. Differential Maintenance, Function, and Transcriptional Profile of CD8⁺ T cells with Age .

Degree: 2013, University of Arizona

 Infectious diseases remain amongst leading causes of death in people aged 65 years and older; therefore, much research is focused on determining the immune components… (more)

Subjects/Keywords: CD8⁺ T cell; Homeostasis; Immunobiology; Aging

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APA (6th Edition):

Renkema, K. (2013). Differential Maintenance, Function, and Transcriptional Profile of CD8⁺ T cells with Age . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/293484

Chicago Manual of Style (16th Edition):

Renkema, Kristin. “Differential Maintenance, Function, and Transcriptional Profile of CD8⁺ T cells with Age .” 2013. Doctoral Dissertation, University of Arizona. Accessed November 22, 2019. http://hdl.handle.net/10150/293484.

MLA Handbook (7th Edition):

Renkema, Kristin. “Differential Maintenance, Function, and Transcriptional Profile of CD8⁺ T cells with Age .” 2013. Web. 22 Nov 2019.

Vancouver:

Renkema K. Differential Maintenance, Function, and Transcriptional Profile of CD8⁺ T cells with Age . [Internet] [Doctoral dissertation]. University of Arizona; 2013. [cited 2019 Nov 22]. Available from: http://hdl.handle.net/10150/293484.

Council of Science Editors:

Renkema K. Differential Maintenance, Function, and Transcriptional Profile of CD8⁺ T cells with Age . [Doctoral Dissertation]. University of Arizona; 2013. Available from: http://hdl.handle.net/10150/293484


University of Arizona

26. Brownlee, Christopher William. Cell Cycle-Dependent Regulation of Centriole Duplication .

Degree: 2013, University of Arizona

 Centrosomes are organelles that promote microtubule growth. Normally, a single centrosome duplicates once each cell cycle to guide assembly of a bipolar mitotic spindle, ensuring… (more)

Subjects/Keywords: Centriole Duplication; Centrioles; Plk4; PP2A; STIL; Cell Biology & Anatomy; Ana2

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APA (6th Edition):

Brownlee, C. W. (2013). Cell Cycle-Dependent Regulation of Centriole Duplication . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/311316

Chicago Manual of Style (16th Edition):

Brownlee, Christopher William. “Cell Cycle-Dependent Regulation of Centriole Duplication .” 2013. Doctoral Dissertation, University of Arizona. Accessed November 22, 2019. http://hdl.handle.net/10150/311316.

MLA Handbook (7th Edition):

Brownlee, Christopher William. “Cell Cycle-Dependent Regulation of Centriole Duplication .” 2013. Web. 22 Nov 2019.

Vancouver:

Brownlee CW. Cell Cycle-Dependent Regulation of Centriole Duplication . [Internet] [Doctoral dissertation]. University of Arizona; 2013. [cited 2019 Nov 22]. Available from: http://hdl.handle.net/10150/311316.

Council of Science Editors:

Brownlee CW. Cell Cycle-Dependent Regulation of Centriole Duplication . [Doctoral Dissertation]. University of Arizona; 2013. Available from: http://hdl.handle.net/10150/311316


University of Arizona

27. Nworu, Chinedu Uzoma. Using the Xenopus Model to Elucidate the Functional Roles of Leiomodin3 and Tropomodulin4 (Tmod4) During Skeletal Muscle Development .

Degree: 2013, University of Arizona

 Having an in vivo model of development that develops quickly and efficiently is important for investigators to elucidate the critical steps, components and signaling pathways… (more)

Subjects/Keywords: sarcomere; skeletal muscle development; thin filament regulation; Tmod; Xenopus; Cell Biology & Anatomy; Lmod

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APA (6th Edition):

Nworu, C. U. (2013). Using the Xenopus Model to Elucidate the Functional Roles of Leiomodin3 and Tropomodulin4 (Tmod4) During Skeletal Muscle Development . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/311689

Chicago Manual of Style (16th Edition):

Nworu, Chinedu Uzoma. “Using the Xenopus Model to Elucidate the Functional Roles of Leiomodin3 and Tropomodulin4 (Tmod4) During Skeletal Muscle Development .” 2013. Doctoral Dissertation, University of Arizona. Accessed November 22, 2019. http://hdl.handle.net/10150/311689.

MLA Handbook (7th Edition):

Nworu, Chinedu Uzoma. “Using the Xenopus Model to Elucidate the Functional Roles of Leiomodin3 and Tropomodulin4 (Tmod4) During Skeletal Muscle Development .” 2013. Web. 22 Nov 2019.

Vancouver:

Nworu CU. Using the Xenopus Model to Elucidate the Functional Roles of Leiomodin3 and Tropomodulin4 (Tmod4) During Skeletal Muscle Development . [Internet] [Doctoral dissertation]. University of Arizona; 2013. [cited 2019 Nov 22]. Available from: http://hdl.handle.net/10150/311689.

Council of Science Editors:

Nworu CU. Using the Xenopus Model to Elucidate the Functional Roles of Leiomodin3 and Tropomodulin4 (Tmod4) During Skeletal Muscle Development . [Doctoral Dissertation]. University of Arizona; 2013. Available from: http://hdl.handle.net/10150/311689


University of Arizona

28. Nye, Jonathan. Utilizing S2 Cells to Study the Molecular Mechanisms Regulating Centriole Duplication .

Degree: 2014, University of Arizona

 Centrosomes are complex organelles consisting of a pair of small microtubule based structures called centrioles embedded in an amorphous cloud of pericentriolar material (PCM). These… (more)

Subjects/Keywords: Cell Biology & Anatomy

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APA (6th Edition):

Nye, J. (2014). Utilizing S2 Cells to Study the Molecular Mechanisms Regulating Centriole Duplication . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/332835

Chicago Manual of Style (16th Edition):

Nye, Jonathan. “Utilizing S2 Cells to Study the Molecular Mechanisms Regulating Centriole Duplication .” 2014. Doctoral Dissertation, University of Arizona. Accessed November 22, 2019. http://hdl.handle.net/10150/332835.

MLA Handbook (7th Edition):

Nye, Jonathan. “Utilizing S2 Cells to Study the Molecular Mechanisms Regulating Centriole Duplication .” 2014. Web. 22 Nov 2019.

Vancouver:

Nye J. Utilizing S2 Cells to Study the Molecular Mechanisms Regulating Centriole Duplication . [Internet] [Doctoral dissertation]. University of Arizona; 2014. [cited 2019 Nov 22]. Available from: http://hdl.handle.net/10150/332835.

Council of Science Editors:

Nye J. Utilizing S2 Cells to Study the Molecular Mechanisms Regulating Centriole Duplication . [Doctoral Dissertation]. University of Arizona; 2014. Available from: http://hdl.handle.net/10150/332835


University of Arizona

29. Deshpande, Neha Rajendra. Impact of Time on the Size, Shape and Effector Responses of Cd4⁺ T Cells .

Degree: 2015, University of Arizona

 T cells discriminate self from foreign peptides presented in the context of self-major histocompatibility complex (pMHC) molecules via clonotypic T cell receptors (TCRs). CD8⁺ T… (more)

Subjects/Keywords: Immunobiology

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APA (6th Edition):

Deshpande, N. R. (2015). Impact of Time on the Size, Shape and Effector Responses of Cd4⁺ T Cells . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/556850

Chicago Manual of Style (16th Edition):

Deshpande, Neha Rajendra. “Impact of Time on the Size, Shape and Effector Responses of Cd4⁺ T Cells .” 2015. Doctoral Dissertation, University of Arizona. Accessed November 22, 2019. http://hdl.handle.net/10150/556850.

MLA Handbook (7th Edition):

Deshpande, Neha Rajendra. “Impact of Time on the Size, Shape and Effector Responses of Cd4⁺ T Cells .” 2015. Web. 22 Nov 2019.

Vancouver:

Deshpande NR. Impact of Time on the Size, Shape and Effector Responses of Cd4⁺ T Cells . [Internet] [Doctoral dissertation]. University of Arizona; 2015. [cited 2019 Nov 22]. Available from: http://hdl.handle.net/10150/556850.

Council of Science Editors:

Deshpande NR. Impact of Time on the Size, Shape and Effector Responses of Cd4⁺ T Cells . [Doctoral Dissertation]. University of Arizona; 2015. Available from: http://hdl.handle.net/10150/556850


University of Arizona

30. Johnson, Debra L. Engagement of T cells with Antigen Presenting Cells is Dependent on Clathrin-Independent Endocytic Trafficking: The Role of Arf6 and Rab22 .

Degree: 2016, University of Arizona

 The clathrin-independent endosomal system is required for cellular homeostasis and specialized modifications of the plasma membrane such as cell spreading and polarization. Clathrin-independent endocytosis (CIE)… (more)

Subjects/Keywords: Clathrin-independent; endocytosis; membrane trafficking; Rab22; T cells; Cellular and Molecular Medicine; Arf6

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APA (6th Edition):

Johnson, D. L. (2016). Engagement of T cells with Antigen Presenting Cells is Dependent on Clathrin-Independent Endocytic Trafficking: The Role of Arf6 and Rab22 . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/612373

Chicago Manual of Style (16th Edition):

Johnson, Debra L. “Engagement of T cells with Antigen Presenting Cells is Dependent on Clathrin-Independent Endocytic Trafficking: The Role of Arf6 and Rab22 .” 2016. Doctoral Dissertation, University of Arizona. Accessed November 22, 2019. http://hdl.handle.net/10150/612373.

MLA Handbook (7th Edition):

Johnson, Debra L. “Engagement of T cells with Antigen Presenting Cells is Dependent on Clathrin-Independent Endocytic Trafficking: The Role of Arf6 and Rab22 .” 2016. Web. 22 Nov 2019.

Vancouver:

Johnson DL. Engagement of T cells with Antigen Presenting Cells is Dependent on Clathrin-Independent Endocytic Trafficking: The Role of Arf6 and Rab22 . [Internet] [Doctoral dissertation]. University of Arizona; 2016. [cited 2019 Nov 22]. Available from: http://hdl.handle.net/10150/612373.

Council of Science Editors:

Johnson DL. Engagement of T cells with Antigen Presenting Cells is Dependent on Clathrin-Independent Endocytic Trafficking: The Role of Arf6 and Rab22 . [Doctoral Dissertation]. University of Arizona; 2016. Available from: http://hdl.handle.net/10150/612373

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