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You searched for +publisher:"University of Arizona" +contributor:("Gandolfi, A. Jay"). Showing records 1 – 30 of 43 total matches.

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University of Arizona

1. Wnek, Shawn Michael. Mechanisms of malignant transformation of human urothelial cells by monomethylarsonous acid .

Degree: 2011, University of Arizona

 Sources of arsenic exposure include air, water, and food from both natural and anthropogenic sources. Arsenic is categorized as a human carcinogen, and is associated… (more)

Subjects/Keywords: DNA damage; DNA repair; monomethylarsonous acid; Pharmacology & Toxicology; arsenic; bladder cancer

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APA (6th Edition):

Wnek, S. M. (2011). Mechanisms of malignant transformation of human urothelial cells by monomethylarsonous acid . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/201495

Chicago Manual of Style (16th Edition):

Wnek, Shawn Michael. “Mechanisms of malignant transformation of human urothelial cells by monomethylarsonous acid .” 2011. Doctoral Dissertation, University of Arizona. Accessed March 08, 2021. http://hdl.handle.net/10150/201495.

MLA Handbook (7th Edition):

Wnek, Shawn Michael. “Mechanisms of malignant transformation of human urothelial cells by monomethylarsonous acid .” 2011. Web. 08 Mar 2021.

Vancouver:

Wnek SM. Mechanisms of malignant transformation of human urothelial cells by monomethylarsonous acid . [Internet] [Doctoral dissertation]. University of Arizona; 2011. [cited 2021 Mar 08]. Available from: http://hdl.handle.net/10150/201495.

Council of Science Editors:

Wnek SM. Mechanisms of malignant transformation of human urothelial cells by monomethylarsonous acid . [Doctoral Dissertation]. University of Arizona; 2011. Available from: http://hdl.handle.net/10150/201495


University of Arizona

2. Yellowhair, Monica. The Chemical-Induced Genotoxicity of Depleted Uranium .

Degree: 2011, University of Arizona

 Uranium has been mined for many years and used for fuel for nuclear reactors and materials for atomic weapons, ammunition, and armor. While the radioactivity… (more)

Subjects/Keywords: genotoxicity; metals; Pharmacology & Toxicology; depleted uranium; DNA damage

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APA (6th Edition):

Yellowhair, M. (2011). The Chemical-Induced Genotoxicity of Depleted Uranium . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/202509

Chicago Manual of Style (16th Edition):

Yellowhair, Monica. “The Chemical-Induced Genotoxicity of Depleted Uranium .” 2011. Doctoral Dissertation, University of Arizona. Accessed March 08, 2021. http://hdl.handle.net/10150/202509.

MLA Handbook (7th Edition):

Yellowhair, Monica. “The Chemical-Induced Genotoxicity of Depleted Uranium .” 2011. Web. 08 Mar 2021.

Vancouver:

Yellowhair M. The Chemical-Induced Genotoxicity of Depleted Uranium . [Internet] [Doctoral dissertation]. University of Arizona; 2011. [cited 2021 Mar 08]. Available from: http://hdl.handle.net/10150/202509.

Council of Science Editors:

Yellowhair M. The Chemical-Induced Genotoxicity of Depleted Uranium . [Doctoral Dissertation]. University of Arizona; 2011. Available from: http://hdl.handle.net/10150/202509


University of Arizona

3. Gomez Rubio, Paulina. DETERMINANTS OF INTERINDIVIDUAL VARIABILITY IN ARSENIC SECONDARY METHYLATION EFFICIENCY IN A POPULATION FROM NORTHWEST MEXICO .

Degree: 2011, University of Arizona

 Chronic environmental exposure to inorganic arsenic is widely associated with human disease. Low human arsenic secondary methylation efficiency (SME), represented by high urinary monomethylarsonic acid… (more)

Subjects/Keywords: AS3MT; BMI; methylation; MMA; Pharmacology & Toxicology; Ancestry; Arsenic

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APA (6th Edition):

Gomez Rubio, P. (2011). DETERMINANTS OF INTERINDIVIDUAL VARIABILITY IN ARSENIC SECONDARY METHYLATION EFFICIENCY IN A POPULATION FROM NORTHWEST MEXICO . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/203433

Chicago Manual of Style (16th Edition):

Gomez Rubio, Paulina. “DETERMINANTS OF INTERINDIVIDUAL VARIABILITY IN ARSENIC SECONDARY METHYLATION EFFICIENCY IN A POPULATION FROM NORTHWEST MEXICO .” 2011. Doctoral Dissertation, University of Arizona. Accessed March 08, 2021. http://hdl.handle.net/10150/203433.

MLA Handbook (7th Edition):

Gomez Rubio, Paulina. “DETERMINANTS OF INTERINDIVIDUAL VARIABILITY IN ARSENIC SECONDARY METHYLATION EFFICIENCY IN A POPULATION FROM NORTHWEST MEXICO .” 2011. Web. 08 Mar 2021.

Vancouver:

Gomez Rubio P. DETERMINANTS OF INTERINDIVIDUAL VARIABILITY IN ARSENIC SECONDARY METHYLATION EFFICIENCY IN A POPULATION FROM NORTHWEST MEXICO . [Internet] [Doctoral dissertation]. University of Arizona; 2011. [cited 2021 Mar 08]. Available from: http://hdl.handle.net/10150/203433.

Council of Science Editors:

Gomez Rubio P. DETERMINANTS OF INTERINDIVIDUAL VARIABILITY IN ARSENIC SECONDARY METHYLATION EFFICIENCY IN A POPULATION FROM NORTHWEST MEXICO . [Doctoral Dissertation]. University of Arizona; 2011. Available from: http://hdl.handle.net/10150/203433


University of Arizona

4. Merrell, Matthew David. Xenosensor Regulation of Enzymes and Transporters in Drug Exposure and Disease .

Degree: 2011, University of Arizona

 A large and varied array of xenobiotics (foreign chemicals) enters into our bodies every day. In order to prevent toxicity resulting from xenobiotic accumulation, the… (more)

Subjects/Keywords: Disease; Liver; Metabolism; Xenobiotic; Xenosensor

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APA (6th Edition):

Merrell, M. D. (2011). Xenosensor Regulation of Enzymes and Transporters in Drug Exposure and Disease . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/194051

Chicago Manual of Style (16th Edition):

Merrell, Matthew David. “Xenosensor Regulation of Enzymes and Transporters in Drug Exposure and Disease .” 2011. Doctoral Dissertation, University of Arizona. Accessed March 08, 2021. http://hdl.handle.net/10150/194051.

MLA Handbook (7th Edition):

Merrell, Matthew David. “Xenosensor Regulation of Enzymes and Transporters in Drug Exposure and Disease .” 2011. Web. 08 Mar 2021.

Vancouver:

Merrell MD. Xenosensor Regulation of Enzymes and Transporters in Drug Exposure and Disease . [Internet] [Doctoral dissertation]. University of Arizona; 2011. [cited 2021 Mar 08]. Available from: http://hdl.handle.net/10150/194051.

Council of Science Editors:

Merrell MD. Xenosensor Regulation of Enzymes and Transporters in Drug Exposure and Disease . [Doctoral Dissertation]. University of Arizona; 2011. Available from: http://hdl.handle.net/10150/194051


University of Arizona

5. Hardwick, Rhiannon Nicole. Nash Alters Drug Metabolizing Enzyme and Transporter Expression Resulting in Significant Consequences for Pharmaceutical Disposition and Toxicity .

Degree: 2012, University of Arizona

 The body encounters an innumerable amount of foreign substances, termed xenobiotics, which it must remove in order to prevent damage to cells and organs. This… (more)

Subjects/Keywords: drug transporters; nonalcoholic fatty liver disease; nonalcoholic steatohepatitis; Pharmacology & Toxicology; drug disposition; drug metabolizing enzymes

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APA (6th Edition):

Hardwick, R. N. (2012). Nash Alters Drug Metabolizing Enzyme and Transporter Expression Resulting in Significant Consequences for Pharmaceutical Disposition and Toxicity . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/247281

Chicago Manual of Style (16th Edition):

Hardwick, Rhiannon Nicole. “Nash Alters Drug Metabolizing Enzyme and Transporter Expression Resulting in Significant Consequences for Pharmaceutical Disposition and Toxicity .” 2012. Doctoral Dissertation, University of Arizona. Accessed March 08, 2021. http://hdl.handle.net/10150/247281.

MLA Handbook (7th Edition):

Hardwick, Rhiannon Nicole. “Nash Alters Drug Metabolizing Enzyme and Transporter Expression Resulting in Significant Consequences for Pharmaceutical Disposition and Toxicity .” 2012. Web. 08 Mar 2021.

Vancouver:

Hardwick RN. Nash Alters Drug Metabolizing Enzyme and Transporter Expression Resulting in Significant Consequences for Pharmaceutical Disposition and Toxicity . [Internet] [Doctoral dissertation]. University of Arizona; 2012. [cited 2021 Mar 08]. Available from: http://hdl.handle.net/10150/247281.

Council of Science Editors:

Hardwick RN. Nash Alters Drug Metabolizing Enzyme and Transporter Expression Resulting in Significant Consequences for Pharmaceutical Disposition and Toxicity . [Doctoral Dissertation]. University of Arizona; 2012. Available from: http://hdl.handle.net/10150/247281


University of Arizona

6. Jimenez-Canet, Mark. Coordinated Regulation Of Hepatic And Renal Membrane Transporters In Experimental Nonalcoholic Steatohepatitis .

Degree: 2014, University of Arizona

 Inter-individual variability in drug response is a significant clinical concern and may lead to the development of adverse drug reactions, which are currently a top-ten… (more)

Subjects/Keywords: liver disease; membrane transporters; pharmacokinetics; Pharmacology & Toxicology; adverse drug reactions

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APA (6th Edition):

Jimenez-Canet, M. (2014). Coordinated Regulation Of Hepatic And Renal Membrane Transporters In Experimental Nonalcoholic Steatohepatitis . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/333487

Chicago Manual of Style (16th Edition):

Jimenez-Canet, Mark. “Coordinated Regulation Of Hepatic And Renal Membrane Transporters In Experimental Nonalcoholic Steatohepatitis .” 2014. Doctoral Dissertation, University of Arizona. Accessed March 08, 2021. http://hdl.handle.net/10150/333487.

MLA Handbook (7th Edition):

Jimenez-Canet, Mark. “Coordinated Regulation Of Hepatic And Renal Membrane Transporters In Experimental Nonalcoholic Steatohepatitis .” 2014. Web. 08 Mar 2021.

Vancouver:

Jimenez-Canet M. Coordinated Regulation Of Hepatic And Renal Membrane Transporters In Experimental Nonalcoholic Steatohepatitis . [Internet] [Doctoral dissertation]. University of Arizona; 2014. [cited 2021 Mar 08]. Available from: http://hdl.handle.net/10150/333487.

Council of Science Editors:

Jimenez-Canet M. Coordinated Regulation Of Hepatic And Renal Membrane Transporters In Experimental Nonalcoholic Steatohepatitis . [Doctoral Dissertation]. University of Arizona; 2014. Available from: http://hdl.handle.net/10150/333487

7. Medeiros, Matthew Keane. Genomic Response in Human Urothelial Cells Exposed Chronically to Monomethylarsonous Acid .

Degree: 2013, University of Arizona

 Bladder cancer has been associated with chronic arsenic exposure. Monomethylarsonous acid [MMA(III)] is a metabolite of inorganic arsenic biotransformation and has been shown to transform… (more)

Subjects/Keywords: bladder; cancer biology; gene expression; gene microarray; urothelial cells; Pharmacology & Toxicology; arsenic

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APA (6th Edition):

Medeiros, M. K. (2013). Genomic Response in Human Urothelial Cells Exposed Chronically to Monomethylarsonous Acid . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/299107

Chicago Manual of Style (16th Edition):

Medeiros, Matthew Keane. “Genomic Response in Human Urothelial Cells Exposed Chronically to Monomethylarsonous Acid .” 2013. Doctoral Dissertation, University of Arizona. Accessed March 08, 2021. http://hdl.handle.net/10150/299107.

MLA Handbook (7th Edition):

Medeiros, Matthew Keane. “Genomic Response in Human Urothelial Cells Exposed Chronically to Monomethylarsonous Acid .” 2013. Web. 08 Mar 2021.

Vancouver:

Medeiros MK. Genomic Response in Human Urothelial Cells Exposed Chronically to Monomethylarsonous Acid . [Internet] [Doctoral dissertation]. University of Arizona; 2013. [cited 2021 Mar 08]. Available from: http://hdl.handle.net/10150/299107.

Council of Science Editors:

Medeiros MK. Genomic Response in Human Urothelial Cells Exposed Chronically to Monomethylarsonous Acid . [Doctoral Dissertation]. University of Arizona; 2013. Available from: http://hdl.handle.net/10150/299107


University of Arizona

8. RUEGG, CHARLES EDWARD. MECHANISMS UNDERLYING REGIOSELECTIVE ACUTE TUBULAR NECROSIS OF RENAL PROXIMAL TUBULAR SEGMENTS .

Degree: 1987, University of Arizona

 The convoluted (CPT) and straight (SPT) portions of the renal proximal tubule are susceptible to injury by a wide variety of chemical agents. These agents… (more)

Subjects/Keywords: Kidney tubules  – Wounds and injuries.; Kidneys  – Necrosis.

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APA (6th Edition):

RUEGG, C. E. (1987). MECHANISMS UNDERLYING REGIOSELECTIVE ACUTE TUBULAR NECROSIS OF RENAL PROXIMAL TUBULAR SEGMENTS . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/184162

Chicago Manual of Style (16th Edition):

RUEGG, CHARLES EDWARD. “MECHANISMS UNDERLYING REGIOSELECTIVE ACUTE TUBULAR NECROSIS OF RENAL PROXIMAL TUBULAR SEGMENTS .” 1987. Doctoral Dissertation, University of Arizona. Accessed March 08, 2021. http://hdl.handle.net/10150/184162.

MLA Handbook (7th Edition):

RUEGG, CHARLES EDWARD. “MECHANISMS UNDERLYING REGIOSELECTIVE ACUTE TUBULAR NECROSIS OF RENAL PROXIMAL TUBULAR SEGMENTS .” 1987. Web. 08 Mar 2021.

Vancouver:

RUEGG CE. MECHANISMS UNDERLYING REGIOSELECTIVE ACUTE TUBULAR NECROSIS OF RENAL PROXIMAL TUBULAR SEGMENTS . [Internet] [Doctoral dissertation]. University of Arizona; 1987. [cited 2021 Mar 08]. Available from: http://hdl.handle.net/10150/184162.

Council of Science Editors:

RUEGG CE. MECHANISMS UNDERLYING REGIOSELECTIVE ACUTE TUBULAR NECROSIS OF RENAL PROXIMAL TUBULAR SEGMENTS . [Doctoral Dissertation]. University of Arizona; 1987. Available from: http://hdl.handle.net/10150/184162


University of Arizona

9. Peraza, Marjorie Aida. Subcytotoxic inorganic arsenic effects on mitochondria in a human proximal tubule cell line: Implications on mechanism of cell death .

Degree: 2003, University of Arizona

 Arsenic is an environmental toxicant and a human carcinogen. The kidney is a known target organ for arsenic (As) and is critical for both arsenic… (more)

Subjects/Keywords: Biology, Cell.; Health Sciences, Toxicology.

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APA (6th Edition):

Peraza, M. A. (2003). Subcytotoxic inorganic arsenic effects on mitochondria in a human proximal tubule cell line: Implications on mechanism of cell death . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/280493

Chicago Manual of Style (16th Edition):

Peraza, Marjorie Aida. “Subcytotoxic inorganic arsenic effects on mitochondria in a human proximal tubule cell line: Implications on mechanism of cell death .” 2003. Doctoral Dissertation, University of Arizona. Accessed March 08, 2021. http://hdl.handle.net/10150/280493.

MLA Handbook (7th Edition):

Peraza, Marjorie Aida. “Subcytotoxic inorganic arsenic effects on mitochondria in a human proximal tubule cell line: Implications on mechanism of cell death .” 2003. Web. 08 Mar 2021.

Vancouver:

Peraza MA. Subcytotoxic inorganic arsenic effects on mitochondria in a human proximal tubule cell line: Implications on mechanism of cell death . [Internet] [Doctoral dissertation]. University of Arizona; 2003. [cited 2021 Mar 08]. Available from: http://hdl.handle.net/10150/280493.

Council of Science Editors:

Peraza MA. Subcytotoxic inorganic arsenic effects on mitochondria in a human proximal tubule cell line: Implications on mechanism of cell death . [Doctoral Dissertation]. University of Arizona; 2003. Available from: http://hdl.handle.net/10150/280493


University of Arizona

10. Waters, Stephen Joseph. In vivo and in vitro toxicity of M-741 (3,15 di-(5,5-dimethyl-3-N(-(cyclopropylmethylinium)-(N-propylinium));-1-cyclohex-1-enyl);-7,11,18,21-tetraoxa-3,15-diazatrispiro (5.2.2.5.2.2); heneicosane) .

Degree: 1996, University of Arizona

 M-741 (3,15 di-[5,5-dimethyl-3-N[-(cyclopropylmethylinium)-(N-propylinium]-1-cyclohex-1-enyl]-7,11,18,21-tetraoxa-3,15-diazatrispiro [5.2.2.5.2.2] heneicosane produces hepatotoxicity in rats following intravenous administration. Hepatocellular pathology is characterized by parenchymal cell necrosis and inflammatory cell infiltration. Electron… (more)

Subjects/Keywords: Health Sciences, Pharmacology.; Health Sciences, Pharmacology.

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APA (6th Edition):

Waters, S. J. (1996). In vivo and in vitro toxicity of M-741 (3,15 di-(5,5-dimethyl-3-N(-(cyclopropylmethylinium)-(N-propylinium));-1-cyclohex-1-enyl);-7,11,18,21-tetraoxa-3,15-diazatrispiro (5.2.2.5.2.2); heneicosane) . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/282154

Chicago Manual of Style (16th Edition):

Waters, Stephen Joseph. “In vivo and in vitro toxicity of M-741 (3,15 di-(5,5-dimethyl-3-N(-(cyclopropylmethylinium)-(N-propylinium));-1-cyclohex-1-enyl);-7,11,18,21-tetraoxa-3,15-diazatrispiro (5.2.2.5.2.2); heneicosane) .” 1996. Doctoral Dissertation, University of Arizona. Accessed March 08, 2021. http://hdl.handle.net/10150/282154.

MLA Handbook (7th Edition):

Waters, Stephen Joseph. “In vivo and in vitro toxicity of M-741 (3,15 di-(5,5-dimethyl-3-N(-(cyclopropylmethylinium)-(N-propylinium));-1-cyclohex-1-enyl);-7,11,18,21-tetraoxa-3,15-diazatrispiro (5.2.2.5.2.2); heneicosane) .” 1996. Web. 08 Mar 2021.

Vancouver:

Waters SJ. In vivo and in vitro toxicity of M-741 (3,15 di-(5,5-dimethyl-3-N(-(cyclopropylmethylinium)-(N-propylinium));-1-cyclohex-1-enyl);-7,11,18,21-tetraoxa-3,15-diazatrispiro (5.2.2.5.2.2); heneicosane) . [Internet] [Doctoral dissertation]. University of Arizona; 1996. [cited 2021 Mar 08]. Available from: http://hdl.handle.net/10150/282154.

Council of Science Editors:

Waters SJ. In vivo and in vitro toxicity of M-741 (3,15 di-(5,5-dimethyl-3-N(-(cyclopropylmethylinium)-(N-propylinium));-1-cyclohex-1-enyl);-7,11,18,21-tetraoxa-3,15-diazatrispiro (5.2.2.5.2.2); heneicosane) . [Doctoral Dissertation]. University of Arizona; 1996. Available from: http://hdl.handle.net/10150/282154


University of Arizona

11. Kirkpatrick, Donald Scott. The effects of arsenic on the ubiquitin dependent proteolytic pathway .

Degree: 2003, University of Arizona

 Arsenic is a potent toxicant and known human carcinogen. Arsenite [As (III)], a major inorganic form of arsenic, can stimulate oxidative stress, decrease DNA repair,… (more)

Subjects/Keywords: Health Sciences; Toxicology.

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APA (6th Edition):

Kirkpatrick, D. S. (2003). The effects of arsenic on the ubiquitin dependent proteolytic pathway . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/289893

Chicago Manual of Style (16th Edition):

Kirkpatrick, Donald Scott. “The effects of arsenic on the ubiquitin dependent proteolytic pathway .” 2003. Doctoral Dissertation, University of Arizona. Accessed March 08, 2021. http://hdl.handle.net/10150/289893.

MLA Handbook (7th Edition):

Kirkpatrick, Donald Scott. “The effects of arsenic on the ubiquitin dependent proteolytic pathway .” 2003. Web. 08 Mar 2021.

Vancouver:

Kirkpatrick DS. The effects of arsenic on the ubiquitin dependent proteolytic pathway . [Internet] [Doctoral dissertation]. University of Arizona; 2003. [cited 2021 Mar 08]. Available from: http://hdl.handle.net/10150/289893.

Council of Science Editors:

Kirkpatrick DS. The effects of arsenic on the ubiquitin dependent proteolytic pathway . [Doctoral Dissertation]. University of Arizona; 2003. Available from: http://hdl.handle.net/10150/289893


University of Arizona

12. Reid, Lynnda Louise, 1949-. Use of staphylococcal enterotoxin A-induced interleukin 2 production as an indicator of immunotoxicity .

Degree: 1990, University of Arizona

 An in vitro assay measuring IL-2 production as an index of compromised immune function was developed using primary splenocytes from female C57BL/6 mice pretreated with… (more)

Subjects/Keywords: Health Sciences, Pharmacology.; Health Sciences, Immunology.

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APA (6th Edition):

Reid, Lynnda Louise, 1. (1990). Use of staphylococcal enterotoxin A-induced interleukin 2 production as an indicator of immunotoxicity . (Masters Thesis). University of Arizona. Retrieved from http://hdl.handle.net/10150/278371

Chicago Manual of Style (16th Edition):

Reid, Lynnda Louise, 1949-. “Use of staphylococcal enterotoxin A-induced interleukin 2 production as an indicator of immunotoxicity .” 1990. Masters Thesis, University of Arizona. Accessed March 08, 2021. http://hdl.handle.net/10150/278371.

MLA Handbook (7th Edition):

Reid, Lynnda Louise, 1949-. “Use of staphylococcal enterotoxin A-induced interleukin 2 production as an indicator of immunotoxicity .” 1990. Web. 08 Mar 2021.

Vancouver:

Reid, Lynnda Louise 1. Use of staphylococcal enterotoxin A-induced interleukin 2 production as an indicator of immunotoxicity . [Internet] [Masters thesis]. University of Arizona; 1990. [cited 2021 Mar 08]. Available from: http://hdl.handle.net/10150/278371.

Council of Science Editors:

Reid, Lynnda Louise 1. Use of staphylococcal enterotoxin A-induced interleukin 2 production as an indicator of immunotoxicity . [Masters Thesis]. University of Arizona; 1990. Available from: http://hdl.handle.net/10150/278371

13. Bolt, Alicia Marie. Arsenite Alters Lysosome-Mediated Degradation and the Autophagy Process Leading to Immunosuppression in Human B-Lymphoblastoid Cell Lines .

Degree: 2012, University of Arizona

 The immune system is a target of arsenic toxicity. Epidemiological data have shown that arsenic exposure is associated with characteristics of immunosuppression. Human B-lymphoblastoid cell… (more)

Subjects/Keywords: Immunotoxicity; Lymphoblastoid; Proteotoxicity; Pharmacology & Toxicology; Arsenic; Autophagy

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APA (6th Edition):

Bolt, A. M. (2012). Arsenite Alters Lysosome-Mediated Degradation and the Autophagy Process Leading to Immunosuppression in Human B-Lymphoblastoid Cell Lines . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/222895

Chicago Manual of Style (16th Edition):

Bolt, Alicia Marie. “Arsenite Alters Lysosome-Mediated Degradation and the Autophagy Process Leading to Immunosuppression in Human B-Lymphoblastoid Cell Lines .” 2012. Doctoral Dissertation, University of Arizona. Accessed March 08, 2021. http://hdl.handle.net/10150/222895.

MLA Handbook (7th Edition):

Bolt, Alicia Marie. “Arsenite Alters Lysosome-Mediated Degradation and the Autophagy Process Leading to Immunosuppression in Human B-Lymphoblastoid Cell Lines .” 2012. Web. 08 Mar 2021.

Vancouver:

Bolt AM. Arsenite Alters Lysosome-Mediated Degradation and the Autophagy Process Leading to Immunosuppression in Human B-Lymphoblastoid Cell Lines . [Internet] [Doctoral dissertation]. University of Arizona; 2012. [cited 2021 Mar 08]. Available from: http://hdl.handle.net/10150/222895.

Council of Science Editors:

Bolt AM. Arsenite Alters Lysosome-Mediated Degradation and the Autophagy Process Leading to Immunosuppression in Human B-Lymphoblastoid Cell Lines . [Doctoral Dissertation]. University of Arizona; 2012. Available from: http://hdl.handle.net/10150/222895


University of Arizona

14. Wolfgang, Grushenka Hope Isabella. S-(1,2-dichlorovinyl)-L-cysteine induced cellular injury in rabbit renal cortical slices .

Degree: 1988, University of Arizona

 The proximal tubule of the kidney is a target for a wide variety of chemical agents, both inorganic and organic. An in vitro model to… (more)

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APA (6th Edition):

Wolfgang, G. H. I. (1988). S-(1,2-dichlorovinyl)-L-cysteine induced cellular injury in rabbit renal cortical slices . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/184504

Chicago Manual of Style (16th Edition):

Wolfgang, Grushenka Hope Isabella. “S-(1,2-dichlorovinyl)-L-cysteine induced cellular injury in rabbit renal cortical slices .” 1988. Doctoral Dissertation, University of Arizona. Accessed March 08, 2021. http://hdl.handle.net/10150/184504.

MLA Handbook (7th Edition):

Wolfgang, Grushenka Hope Isabella. “S-(1,2-dichlorovinyl)-L-cysteine induced cellular injury in rabbit renal cortical slices .” 1988. Web. 08 Mar 2021.

Vancouver:

Wolfgang GHI. S-(1,2-dichlorovinyl)-L-cysteine induced cellular injury in rabbit renal cortical slices . [Internet] [Doctoral dissertation]. University of Arizona; 1988. [cited 2021 Mar 08]. Available from: http://hdl.handle.net/10150/184504.

Council of Science Editors:

Wolfgang GHI. S-(1,2-dichlorovinyl)-L-cysteine induced cellular injury in rabbit renal cortical slices . [Doctoral Dissertation]. University of Arizona; 1988. Available from: http://hdl.handle.net/10150/184504

15. Wu, Tongde. Post-Transcriptional Regulation of Nrf2: Novel Mechanisms beyond Keap1 .

Degree: 2013, University of Arizona

 Nrf2 (NF-E2-related factor 2) is a transcription factor that regulates a battery of downstream genes that contain the antioxidant response element (ARE) in their promoter… (more)

Subjects/Keywords: Pharmacology & Toxicology

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APA (6th Edition):

Wu, T. (2013). Post-Transcriptional Regulation of Nrf2: Novel Mechanisms beyond Keap1 . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/301750

Chicago Manual of Style (16th Edition):

Wu, Tongde. “Post-Transcriptional Regulation of Nrf2: Novel Mechanisms beyond Keap1 .” 2013. Doctoral Dissertation, University of Arizona. Accessed March 08, 2021. http://hdl.handle.net/10150/301750.

MLA Handbook (7th Edition):

Wu, Tongde. “Post-Transcriptional Regulation of Nrf2: Novel Mechanisms beyond Keap1 .” 2013. Web. 08 Mar 2021.

Vancouver:

Wu T. Post-Transcriptional Regulation of Nrf2: Novel Mechanisms beyond Keap1 . [Internet] [Doctoral dissertation]. University of Arizona; 2013. [cited 2021 Mar 08]. Available from: http://hdl.handle.net/10150/301750.

Council of Science Editors:

Wu T. Post-Transcriptional Regulation of Nrf2: Novel Mechanisms beyond Keap1 . [Doctoral Dissertation]. University of Arizona; 2013. Available from: http://hdl.handle.net/10150/301750


University of Arizona

16. Casarez, Elizabeth Anne Joseph. Microbial regulation of arsenic bioavailability and toxicity in soils .

Degree: 2004, University of Arizona

 The biogeochemical cycle of arsenic (As) is very complex. The current practice of measuring total arsenic concentrations, especially in soil environments, ignores any differences in… (more)

Subjects/Keywords: Health Sciences, Toxicology.; Biology, Microbiology.; Environmental Sciences.

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APA (6th Edition):

Casarez, E. A. J. (2004). Microbial regulation of arsenic bioavailability and toxicity in soils . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/280678

Chicago Manual of Style (16th Edition):

Casarez, Elizabeth Anne Joseph. “Microbial regulation of arsenic bioavailability and toxicity in soils .” 2004. Doctoral Dissertation, University of Arizona. Accessed March 08, 2021. http://hdl.handle.net/10150/280678.

MLA Handbook (7th Edition):

Casarez, Elizabeth Anne Joseph. “Microbial regulation of arsenic bioavailability and toxicity in soils .” 2004. Web. 08 Mar 2021.

Vancouver:

Casarez EAJ. Microbial regulation of arsenic bioavailability and toxicity in soils . [Internet] [Doctoral dissertation]. University of Arizona; 2004. [cited 2021 Mar 08]. Available from: http://hdl.handle.net/10150/280678.

Council of Science Editors:

Casarez EAJ. Microbial regulation of arsenic bioavailability and toxicity in soils . [Doctoral Dissertation]. University of Arizona; 2004. Available from: http://hdl.handle.net/10150/280678


University of Arizona

17. El. Sisi, Alaa El. Din El. Sayed. Elucidation of the mechanism of vitamin A potentiation of carbon tetrachloride-induced liver injury .

Degree: 1987, University of Arizona

 The liver is a target tissue for Vitamin A toxicity. High doses of Vitamin A have been shown to produce hepatomegaly, portal hypertension, fatty liver,… (more)

Subjects/Keywords: Vitamin A  – Toxicology.; Liver  – Wounds and injuries.

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APA (6th Edition):

El. Sisi, A. E. D. E. S. (1987). Elucidation of the mechanism of vitamin A potentiation of carbon tetrachloride-induced liver injury . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/184291

Chicago Manual of Style (16th Edition):

El. Sisi, Alaa El Din El Sayed. “Elucidation of the mechanism of vitamin A potentiation of carbon tetrachloride-induced liver injury .” 1987. Doctoral Dissertation, University of Arizona. Accessed March 08, 2021. http://hdl.handle.net/10150/184291.

MLA Handbook (7th Edition):

El. Sisi, Alaa El Din El Sayed. “Elucidation of the mechanism of vitamin A potentiation of carbon tetrachloride-induced liver injury .” 1987. Web. 08 Mar 2021.

Vancouver:

El. Sisi AEDES. Elucidation of the mechanism of vitamin A potentiation of carbon tetrachloride-induced liver injury . [Internet] [Doctoral dissertation]. University of Arizona; 1987. [cited 2021 Mar 08]. Available from: http://hdl.handle.net/10150/184291.

Council of Science Editors:

El. Sisi AEDES. Elucidation of the mechanism of vitamin A potentiation of carbon tetrachloride-induced liver injury . [Doctoral Dissertation]. University of Arizona; 1987. Available from: http://hdl.handle.net/10150/184291


University of Arizona

18. Shubat, Pamela Jane. Monocrotaline toxicity and pulmonary arteries .

Degree: 1988, University of Arizona

 Monocrotaline is a pyrrolizidine alkaloid found in plants implicated in livestock and human poisoning. Laboratory rats given monocrotaline develop pulmonary hypertension and right heart hypertrophy… (more)

Subjects/Keywords: Pyrrolizidines.; Pulmonary artery  – Effect of drugs on.; Pulmonary pharmacology.; Heart  – Effect of chemicals on.

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APA (6th Edition):

Shubat, P. J. (1988). Monocrotaline toxicity and pulmonary arteries . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/184533

Chicago Manual of Style (16th Edition):

Shubat, Pamela Jane. “Monocrotaline toxicity and pulmonary arteries .” 1988. Doctoral Dissertation, University of Arizona. Accessed March 08, 2021. http://hdl.handle.net/10150/184533.

MLA Handbook (7th Edition):

Shubat, Pamela Jane. “Monocrotaline toxicity and pulmonary arteries .” 1988. Web. 08 Mar 2021.

Vancouver:

Shubat PJ. Monocrotaline toxicity and pulmonary arteries . [Internet] [Doctoral dissertation]. University of Arizona; 1988. [cited 2021 Mar 08]. Available from: http://hdl.handle.net/10150/184533.

Council of Science Editors:

Shubat PJ. Monocrotaline toxicity and pulmonary arteries . [Doctoral Dissertation]. University of Arizona; 1988. Available from: http://hdl.handle.net/10150/184533


University of Arizona

19. Wild, Stacie Lynn. Pyrrolizidine alkaloids: Hepatic metabolism and extrahepatic toxicity .

Degree: 1994, University of Arizona

 Pyrrolizidine alkaloids are proposed to be metabolized in the liver to reactive pyrrole species, or dehydroalkaloids. These reactive pyrroles are hypothesized to be responsible for… (more)

Subjects/Keywords: Dissertations, Academic.; Toxicology.; Pharmacology.

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APA (6th Edition):

Wild, S. L. (1994). Pyrrolizidine alkaloids: Hepatic metabolism and extrahepatic toxicity . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/186599

Chicago Manual of Style (16th Edition):

Wild, Stacie Lynn. “Pyrrolizidine alkaloids: Hepatic metabolism and extrahepatic toxicity .” 1994. Doctoral Dissertation, University of Arizona. Accessed March 08, 2021. http://hdl.handle.net/10150/186599.

MLA Handbook (7th Edition):

Wild, Stacie Lynn. “Pyrrolizidine alkaloids: Hepatic metabolism and extrahepatic toxicity .” 1994. Web. 08 Mar 2021.

Vancouver:

Wild SL. Pyrrolizidine alkaloids: Hepatic metabolism and extrahepatic toxicity . [Internet] [Doctoral dissertation]. University of Arizona; 1994. [cited 2021 Mar 08]. Available from: http://hdl.handle.net/10150/186599.

Council of Science Editors:

Wild SL. Pyrrolizidine alkaloids: Hepatic metabolism and extrahepatic toxicity . [Doctoral Dissertation]. University of Arizona; 1994. Available from: http://hdl.handle.net/10150/186599


University of Arizona

20. Klimecki, Walter Thomas. P-glycoprotein: Expression and function in normal circulating leukocytes .

Degree: 1994, University of Arizona

 P-glycoprotein (P-gly) is a well characterized membrane protein, expressed in cancer cells, functioning as an efflux pump. This function confers drug-resistance. P-gly is also expressed… (more)

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APA (6th Edition):

Klimecki, W. T. (1994). P-glycoprotein: Expression and function in normal circulating leukocytes . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/186715

Chicago Manual of Style (16th Edition):

Klimecki, Walter Thomas. “P-glycoprotein: Expression and function in normal circulating leukocytes .” 1994. Doctoral Dissertation, University of Arizona. Accessed March 08, 2021. http://hdl.handle.net/10150/186715.

MLA Handbook (7th Edition):

Klimecki, Walter Thomas. “P-glycoprotein: Expression and function in normal circulating leukocytes .” 1994. Web. 08 Mar 2021.

Vancouver:

Klimecki WT. P-glycoprotein: Expression and function in normal circulating leukocytes . [Internet] [Doctoral dissertation]. University of Arizona; 1994. [cited 2021 Mar 08]. Available from: http://hdl.handle.net/10150/186715.

Council of Science Editors:

Klimecki WT. P-glycoprotein: Expression and function in normal circulating leukocytes . [Doctoral Dissertation]. University of Arizona; 1994. Available from: http://hdl.handle.net/10150/186715


University of Arizona

21. Bogdan, Gregory Michael. A 450K arsenite-binding protein of the rabbit liver cytosol .

Degree: 1994, University of Arizona

 Inorganic arsenic administered to animals is primarily associated with liver cytosolic macromolecules. The specifics of this binding and its role in arsenic toxicity have never… (more)

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APA (6th Edition):

Bogdan, G. M. (1994). A 450K arsenite-binding protein of the rabbit liver cytosol . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/186945

Chicago Manual of Style (16th Edition):

Bogdan, Gregory Michael. “A 450K arsenite-binding protein of the rabbit liver cytosol .” 1994. Doctoral Dissertation, University of Arizona. Accessed March 08, 2021. http://hdl.handle.net/10150/186945.

MLA Handbook (7th Edition):

Bogdan, Gregory Michael. “A 450K arsenite-binding protein of the rabbit liver cytosol .” 1994. Web. 08 Mar 2021.

Vancouver:

Bogdan GM. A 450K arsenite-binding protein of the rabbit liver cytosol . [Internet] [Doctoral dissertation]. University of Arizona; 1994. [cited 2021 Mar 08]. Available from: http://hdl.handle.net/10150/186945.

Council of Science Editors:

Bogdan GM. A 450K arsenite-binding protein of the rabbit liver cytosol . [Doctoral Dissertation]. University of Arizona; 1994. Available from: http://hdl.handle.net/10150/186945


University of Arizona

22. Poet, Torka Sue. Cytochromes P450 involved in the bioactivation of cocaine in the rat .

Degree: 1995, University of Arizona

 Cytochrome P450 enzyme inducters and inhibitors were used to elucidate the isoforms involved in the bioactivation of cocaine in the rat. Phenobarbital and dexamethasone induction… (more)

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APA (6th Edition):

Poet, T. S. (1995). Cytochromes P450 involved in the bioactivation of cocaine in the rat . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/187215

Chicago Manual of Style (16th Edition):

Poet, Torka Sue. “Cytochromes P450 involved in the bioactivation of cocaine in the rat .” 1995. Doctoral Dissertation, University of Arizona. Accessed March 08, 2021. http://hdl.handle.net/10150/187215.

MLA Handbook (7th Edition):

Poet, Torka Sue. “Cytochromes P450 involved in the bioactivation of cocaine in the rat .” 1995. Web. 08 Mar 2021.

Vancouver:

Poet TS. Cytochromes P450 involved in the bioactivation of cocaine in the rat . [Internet] [Doctoral dissertation]. University of Arizona; 1995. [cited 2021 Mar 08]. Available from: http://hdl.handle.net/10150/187215.

Council of Science Editors:

Poet TS. Cytochromes P450 involved in the bioactivation of cocaine in the rat . [Doctoral Dissertation]. University of Arizona; 1995. Available from: http://hdl.handle.net/10150/187215


University of Arizona

23. Sauer, John-Michael. All- trans-retinol modulation of chemically-induced pulmonary and hepatic toxicity .

Degree: 1996, University of Arizona

 It has been previously reported that acute hypervitaminosis A in rats dramatically increases the hepatotoxicity of a number of chemicals. This potentiation appears to be… (more)

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APA (6th Edition):

Sauer, J. (1996). All- trans-retinol modulation of chemically-induced pulmonary and hepatic toxicity . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/187501

Chicago Manual of Style (16th Edition):

Sauer, John-Michael. “All- trans-retinol modulation of chemically-induced pulmonary and hepatic toxicity .” 1996. Doctoral Dissertation, University of Arizona. Accessed March 08, 2021. http://hdl.handle.net/10150/187501.

MLA Handbook (7th Edition):

Sauer, John-Michael. “All- trans-retinol modulation of chemically-induced pulmonary and hepatic toxicity .” 1996. Web. 08 Mar 2021.

Vancouver:

Sauer J. All- trans-retinol modulation of chemically-induced pulmonary and hepatic toxicity . [Internet] [Doctoral dissertation]. University of Arizona; 1996. [cited 2021 Mar 08]. Available from: http://hdl.handle.net/10150/187501.

Council of Science Editors:

Sauer J. All- trans-retinol modulation of chemically-induced pulmonary and hepatic toxicity . [Doctoral Dissertation]. University of Arizona; 1996. Available from: http://hdl.handle.net/10150/187501


University of Arizona

24. Jensen, Taylor Jacob. EPIGENETIC REMODELING DURING ARSENICAL-INDUCED MALIGNANT TRANSFORMATION .

Degree: 2008, University of Arizona

 Humans are exposed to arsenicals through many routes with the most common being drinking water. Exposure to arsenic has been associated with an increased incidence… (more)

Subjects/Keywords: Arsenic; Carcinogenesis; DNA Methylation; Epigenetic; Histone Acetylation; MMA(III)

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APA (6th Edition):

Jensen, T. J. (2008). EPIGENETIC REMODELING DURING ARSENICAL-INDUCED MALIGNANT TRANSFORMATION . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/193558

Chicago Manual of Style (16th Edition):

Jensen, Taylor Jacob. “EPIGENETIC REMODELING DURING ARSENICAL-INDUCED MALIGNANT TRANSFORMATION .” 2008. Doctoral Dissertation, University of Arizona. Accessed March 08, 2021. http://hdl.handle.net/10150/193558.

MLA Handbook (7th Edition):

Jensen, Taylor Jacob. “EPIGENETIC REMODELING DURING ARSENICAL-INDUCED MALIGNANT TRANSFORMATION .” 2008. Web. 08 Mar 2021.

Vancouver:

Jensen TJ. EPIGENETIC REMODELING DURING ARSENICAL-INDUCED MALIGNANT TRANSFORMATION . [Internet] [Doctoral dissertation]. University of Arizona; 2008. [cited 2021 Mar 08]. Available from: http://hdl.handle.net/10150/193558.

Council of Science Editors:

Jensen TJ. EPIGENETIC REMODELING DURING ARSENICAL-INDUCED MALIGNANT TRANSFORMATION . [Doctoral Dissertation]. University of Arizona; 2008. Available from: http://hdl.handle.net/10150/193558


University of Arizona

25. Lickteig, Andrew Joseph. Drug Metabolizing Enzyme, Drug Transporter Expression And Drug Disposition Are Altered In Models Of Inflammatory Liver Disease .

Degree: 2007, University of Arizona

 Correct dosing in pharmacotherapeutics is based on the idea that too much of a drug will cause toxicity, while too little will result in failure… (more)

Subjects/Keywords: drug transporters; Mrp; acetaminophen; inflammation; nonalcoholic steatohepatitis; NASH

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APA (6th Edition):

Lickteig, A. J. (2007). Drug Metabolizing Enzyme, Drug Transporter Expression And Drug Disposition Are Altered In Models Of Inflammatory Liver Disease . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/193836

Chicago Manual of Style (16th Edition):

Lickteig, Andrew Joseph. “Drug Metabolizing Enzyme, Drug Transporter Expression And Drug Disposition Are Altered In Models Of Inflammatory Liver Disease .” 2007. Doctoral Dissertation, University of Arizona. Accessed March 08, 2021. http://hdl.handle.net/10150/193836.

MLA Handbook (7th Edition):

Lickteig, Andrew Joseph. “Drug Metabolizing Enzyme, Drug Transporter Expression And Drug Disposition Are Altered In Models Of Inflammatory Liver Disease .” 2007. Web. 08 Mar 2021.

Vancouver:

Lickteig AJ. Drug Metabolizing Enzyme, Drug Transporter Expression And Drug Disposition Are Altered In Models Of Inflammatory Liver Disease . [Internet] [Doctoral dissertation]. University of Arizona; 2007. [cited 2021 Mar 08]. Available from: http://hdl.handle.net/10150/193836.

Council of Science Editors:

Lickteig AJ. Drug Metabolizing Enzyme, Drug Transporter Expression And Drug Disposition Are Altered In Models Of Inflammatory Liver Disease . [Doctoral Dissertation]. University of Arizona; 2007. Available from: http://hdl.handle.net/10150/193836


University of Arizona

26. Manza, Linda Lee. Characterization of Protein Sumoylation in Response to Alkylation Stress in HEK 293 Cells .

Degree: 2007, University of Arizona

 Stress conditions such as heat shock, UV, alkylating agents, and H2O2 have been shown to result in the modification of a variety of protein targets… (more)

Subjects/Keywords: Sumoylation; alkylation; hydroxynonenal; LC-MS-MS analysis

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APA (6th Edition):

Manza, L. L. (2007). Characterization of Protein Sumoylation in Response to Alkylation Stress in HEK 293 Cells . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/193944

Chicago Manual of Style (16th Edition):

Manza, Linda Lee. “Characterization of Protein Sumoylation in Response to Alkylation Stress in HEK 293 Cells .” 2007. Doctoral Dissertation, University of Arizona. Accessed March 08, 2021. http://hdl.handle.net/10150/193944.

MLA Handbook (7th Edition):

Manza, Linda Lee. “Characterization of Protein Sumoylation in Response to Alkylation Stress in HEK 293 Cells .” 2007. Web. 08 Mar 2021.

Vancouver:

Manza LL. Characterization of Protein Sumoylation in Response to Alkylation Stress in HEK 293 Cells . [Internet] [Doctoral dissertation]. University of Arizona; 2007. [cited 2021 Mar 08]. Available from: http://hdl.handle.net/10150/193944.

Council of Science Editors:

Manza LL. Characterization of Protein Sumoylation in Response to Alkylation Stress in HEK 293 Cells . [Doctoral Dissertation]. University of Arizona; 2007. Available from: http://hdl.handle.net/10150/193944


University of Arizona

27. Beilke, Lisa D. Mechanisms of Hepatoprotection in a Murine Model of Bile Acid-Induced Intrahepatic Cholestasis .

Degree: 2008, University of Arizona

 There are many causes of cholestasis, which results when the flow of bile acids is slowed or stopped. Bile acids are hydrophobic molecules synthesized from… (more)

Subjects/Keywords: Cholestasis; bile acid; biosynthesis; apoptosis; constitutive androstane receptor

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APA (6th Edition):

Beilke, L. D. (2008). Mechanisms of Hepatoprotection in a Murine Model of Bile Acid-Induced Intrahepatic Cholestasis . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/194264

Chicago Manual of Style (16th Edition):

Beilke, Lisa D. “Mechanisms of Hepatoprotection in a Murine Model of Bile Acid-Induced Intrahepatic Cholestasis .” 2008. Doctoral Dissertation, University of Arizona. Accessed March 08, 2021. http://hdl.handle.net/10150/194264.

MLA Handbook (7th Edition):

Beilke, Lisa D. “Mechanisms of Hepatoprotection in a Murine Model of Bile Acid-Induced Intrahepatic Cholestasis .” 2008. Web. 08 Mar 2021.

Vancouver:

Beilke LD. Mechanisms of Hepatoprotection in a Murine Model of Bile Acid-Induced Intrahepatic Cholestasis . [Internet] [Doctoral dissertation]. University of Arizona; 2008. [cited 2021 Mar 08]. Available from: http://hdl.handle.net/10150/194264.

Council of Science Editors:

Beilke LD. Mechanisms of Hepatoprotection in a Murine Model of Bile Acid-Induced Intrahepatic Cholestasis . [Doctoral Dissertation]. University of Arizona; 2008. Available from: http://hdl.handle.net/10150/194264


University of Arizona

28. Bredfeldt, Tiffany Gail. Low-Level Arsenic Toxicity in Human Bladder Cells .

Degree: 2006, University of Arizona

 Arsenic is a human bladder carcinogen. Inorganic arsenic and methylated metabolites are excreted from the human body in urine. This study investigates the effects of… (more)

Subjects/Keywords: arsenic; arsenic methylation; carcinogenesis; bladder cancer

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APA (6th Edition):

Bredfeldt, T. G. (2006). Low-Level Arsenic Toxicity in Human Bladder Cells . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/195157

Chicago Manual of Style (16th Edition):

Bredfeldt, Tiffany Gail. “Low-Level Arsenic Toxicity in Human Bladder Cells .” 2006. Doctoral Dissertation, University of Arizona. Accessed March 08, 2021. http://hdl.handle.net/10150/195157.

MLA Handbook (7th Edition):

Bredfeldt, Tiffany Gail. “Low-Level Arsenic Toxicity in Human Bladder Cells .” 2006. Web. 08 Mar 2021.

Vancouver:

Bredfeldt TG. Low-Level Arsenic Toxicity in Human Bladder Cells . [Internet] [Doctoral dissertation]. University of Arizona; 2006. [cited 2021 Mar 08]. Available from: http://hdl.handle.net/10150/195157.

Council of Science Editors:

Bredfeldt TG. Low-Level Arsenic Toxicity in Human Bladder Cells . [Doctoral Dissertation]. University of Arizona; 2006. Available from: http://hdl.handle.net/10150/195157


University of Arizona

29. Bruhn, Roberta L. Relationship Between Glaucoma and Selenium Levels in Plasma and Aqueous Humor .

Degree: 2008, University of Arizona

 Purpose: To determine the association of plasma and aqueous humor selenium with glaucoma; and to determine those factors influencing biological levels of selenium in patients… (more)

Subjects/Keywords: epidemiology; glaucoma; selenium; aqueous humor

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APA (6th Edition):

Bruhn, R. L. (2008). Relationship Between Glaucoma and Selenium Levels in Plasma and Aqueous Humor . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/195335

Chicago Manual of Style (16th Edition):

Bruhn, Roberta L. “Relationship Between Glaucoma and Selenium Levels in Plasma and Aqueous Humor .” 2008. Doctoral Dissertation, University of Arizona. Accessed March 08, 2021. http://hdl.handle.net/10150/195335.

MLA Handbook (7th Edition):

Bruhn, Roberta L. “Relationship Between Glaucoma and Selenium Levels in Plasma and Aqueous Humor .” 2008. Web. 08 Mar 2021.

Vancouver:

Bruhn RL. Relationship Between Glaucoma and Selenium Levels in Plasma and Aqueous Humor . [Internet] [Doctoral dissertation]. University of Arizona; 2008. [cited 2021 Mar 08]. Available from: http://hdl.handle.net/10150/195335.

Council of Science Editors:

Bruhn RL. Relationship Between Glaucoma and Selenium Levels in Plasma and Aqueous Humor . [Doctoral Dissertation]. University of Arizona; 2008. Available from: http://hdl.handle.net/10150/195335


University of Arizona

30. Eblin, Kylee Elaine. Arsenical-induced Reactive Oxygen Species Lead to Altered Cellular Signaling and Phenotypic Alterations in Human Bladder Cells .

Degree: 2008, University of Arizona

 Arsenical-induced carcinogenesis in human bladder has been established through epidemiological evidence, but unfortunately, no mode of action had been determined for this phenomenon. UROtsa cells,… (more)

Subjects/Keywords: Arsenite; Monomethylarsonous acid; ROS; MAPK signaling

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APA (6th Edition):

Eblin, K. E. (2008). Arsenical-induced Reactive Oxygen Species Lead to Altered Cellular Signaling and Phenotypic Alterations in Human Bladder Cells . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/195706

Chicago Manual of Style (16th Edition):

Eblin, Kylee Elaine. “Arsenical-induced Reactive Oxygen Species Lead to Altered Cellular Signaling and Phenotypic Alterations in Human Bladder Cells .” 2008. Doctoral Dissertation, University of Arizona. Accessed March 08, 2021. http://hdl.handle.net/10150/195706.

MLA Handbook (7th Edition):

Eblin, Kylee Elaine. “Arsenical-induced Reactive Oxygen Species Lead to Altered Cellular Signaling and Phenotypic Alterations in Human Bladder Cells .” 2008. Web. 08 Mar 2021.

Vancouver:

Eblin KE. Arsenical-induced Reactive Oxygen Species Lead to Altered Cellular Signaling and Phenotypic Alterations in Human Bladder Cells . [Internet] [Doctoral dissertation]. University of Arizona; 2008. [cited 2021 Mar 08]. Available from: http://hdl.handle.net/10150/195706.

Council of Science Editors:

Eblin KE. Arsenical-induced Reactive Oxygen Species Lead to Altered Cellular Signaling and Phenotypic Alterations in Human Bladder Cells . [Doctoral Dissertation]. University of Arizona; 2008. Available from: http://hdl.handle.net/10150/195706

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