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You searched for +publisher:"University of Arizona" +contributor:("Futscher, Bernard"). Showing records 1 – 30 of 35 total matches.

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University of Arizona

1. Woods, Dana. Localization and Mechanism of MORT in Human Mammary Epithelial Cell Mortal to Immortal Transition .

Degree: 2019, University of Arizona

 Cancer is quickly becoming the leading cause of death in the United States. Despite research making headway with early detection techniques and effective treatments, the… (more)

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APA (6th Edition):

Woods, D. (2019). Localization and Mechanism of MORT in Human Mammary Epithelial Cell Mortal to Immortal Transition . (Masters Thesis). University of Arizona. Retrieved from http://hdl.handle.net/10150/632540

Chicago Manual of Style (16th Edition):

Woods, Dana. “Localization and Mechanism of MORT in Human Mammary Epithelial Cell Mortal to Immortal Transition .” 2019. Masters Thesis, University of Arizona. Accessed November 17, 2019. http://hdl.handle.net/10150/632540.

MLA Handbook (7th Edition):

Woods, Dana. “Localization and Mechanism of MORT in Human Mammary Epithelial Cell Mortal to Immortal Transition .” 2019. Web. 17 Nov 2019.

Vancouver:

Woods D. Localization and Mechanism of MORT in Human Mammary Epithelial Cell Mortal to Immortal Transition . [Internet] [Masters thesis]. University of Arizona; 2019. [cited 2019 Nov 17]. Available from: http://hdl.handle.net/10150/632540.

Council of Science Editors:

Woods D. Localization and Mechanism of MORT in Human Mammary Epithelial Cell Mortal to Immortal Transition . [Masters Thesis]. University of Arizona; 2019. Available from: http://hdl.handle.net/10150/632540


University of Arizona

2. Muñoz-Rodríguez, José Luis. Postpartum Breast Cancer in Hispanic Women: Epigenetics and microRNAs .

Degree: 2015, University of Arizona

 The risk of breast cancer transiently increases immediately following pregnancy. Hispanic women have one of the highest rates of postpartum breast cancers of all racial/ethnic… (more)

Subjects/Keywords: microRNAs; postpartum breast cancer; Pharmacology & Toxicology; epigenetics

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APA (6th Edition):

Muñoz-Rodríguez, J. L. (2015). Postpartum Breast Cancer in Hispanic Women: Epigenetics and microRNAs . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/603490

Chicago Manual of Style (16th Edition):

Muñoz-Rodríguez, José Luis. “Postpartum Breast Cancer in Hispanic Women: Epigenetics and microRNAs .” 2015. Doctoral Dissertation, University of Arizona. Accessed November 17, 2019. http://hdl.handle.net/10150/603490.

MLA Handbook (7th Edition):

Muñoz-Rodríguez, José Luis. “Postpartum Breast Cancer in Hispanic Women: Epigenetics and microRNAs .” 2015. Web. 17 Nov 2019.

Vancouver:

Muñoz-Rodríguez JL. Postpartum Breast Cancer in Hispanic Women: Epigenetics and microRNAs . [Internet] [Doctoral dissertation]. University of Arizona; 2015. [cited 2019 Nov 17]. Available from: http://hdl.handle.net/10150/603490.

Council of Science Editors:

Muñoz-Rodríguez JL. Postpartum Breast Cancer in Hispanic Women: Epigenetics and microRNAs . [Doctoral Dissertation]. University of Arizona; 2015. Available from: http://hdl.handle.net/10150/603490


University of Arizona

3. Griggs, Chanel Antoinette. Lysine Deacetylase Inhibitors Disrupt Glucocorticoid Receptor-Mediated Transcription and Circadian Rhythm .

Degree: 2018, University of Arizona

 Glucocorticoids mediate their anti-inflammatory and immunosuppressive effects through activation of the glucocorticoid receptor (GR), a nuclear receptor, and subsequent effects on gene transcription. GR use… (more)

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APA (6th Edition):

Griggs, C. A. (2018). Lysine Deacetylase Inhibitors Disrupt Glucocorticoid Receptor-Mediated Transcription and Circadian Rhythm . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/627675

Chicago Manual of Style (16th Edition):

Griggs, Chanel Antoinette. “Lysine Deacetylase Inhibitors Disrupt Glucocorticoid Receptor-Mediated Transcription and Circadian Rhythm .” 2018. Doctoral Dissertation, University of Arizona. Accessed November 17, 2019. http://hdl.handle.net/10150/627675.

MLA Handbook (7th Edition):

Griggs, Chanel Antoinette. “Lysine Deacetylase Inhibitors Disrupt Glucocorticoid Receptor-Mediated Transcription and Circadian Rhythm .” 2018. Web. 17 Nov 2019.

Vancouver:

Griggs CA. Lysine Deacetylase Inhibitors Disrupt Glucocorticoid Receptor-Mediated Transcription and Circadian Rhythm . [Internet] [Doctoral dissertation]. University of Arizona; 2018. [cited 2019 Nov 17]. Available from: http://hdl.handle.net/10150/627675.

Council of Science Editors:

Griggs CA. Lysine Deacetylase Inhibitors Disrupt Glucocorticoid Receptor-Mediated Transcription and Circadian Rhythm . [Doctoral Dissertation]. University of Arizona; 2018. Available from: http://hdl.handle.net/10150/627675


University of Arizona

4. Goldenberg, Joshua. Assessments of Tumor Metabolism with CEST and DCE MRI .

Degree: 2018, University of Arizona

 This dissertation begins with a comprehensive review of the assessment of tumor metabolism highlighting the various applications of chemical exchange saturation transfer (CEST) MRI. The… (more)

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APA (6th Edition):

Goldenberg, J. (2018). Assessments of Tumor Metabolism with CEST and DCE MRI . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/627748

Chicago Manual of Style (16th Edition):

Goldenberg, Joshua. “Assessments of Tumor Metabolism with CEST and DCE MRI .” 2018. Doctoral Dissertation, University of Arizona. Accessed November 17, 2019. http://hdl.handle.net/10150/627748.

MLA Handbook (7th Edition):

Goldenberg, Joshua. “Assessments of Tumor Metabolism with CEST and DCE MRI .” 2018. Web. 17 Nov 2019.

Vancouver:

Goldenberg J. Assessments of Tumor Metabolism with CEST and DCE MRI . [Internet] [Doctoral dissertation]. University of Arizona; 2018. [cited 2019 Nov 17]. Available from: http://hdl.handle.net/10150/627748.

Council of Science Editors:

Goldenberg J. Assessments of Tumor Metabolism with CEST and DCE MRI . [Doctoral Dissertation]. University of Arizona; 2018. Available from: http://hdl.handle.net/10150/627748


University of Arizona

5. Kacsinta, Apollo Daniel. Determining Biological Effectors of alpha6 Integrin Cleavage .

Degree: 2010, University of Arizona

 Cancer metastasis is a multi–step process that initiates with a tumor cell obtaining the ability to migrate. A multitude of changes occur in such a… (more)

Subjects/Keywords: Actin; Integrin; Prostate Cancer; uPAR

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APA (6th Edition):

Kacsinta, A. D. (2010). Determining Biological Effectors of alpha6 Integrin Cleavage . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/193604

Chicago Manual of Style (16th Edition):

Kacsinta, Apollo Daniel. “Determining Biological Effectors of alpha6 Integrin Cleavage .” 2010. Doctoral Dissertation, University of Arizona. Accessed November 17, 2019. http://hdl.handle.net/10150/193604.

MLA Handbook (7th Edition):

Kacsinta, Apollo Daniel. “Determining Biological Effectors of alpha6 Integrin Cleavage .” 2010. Web. 17 Nov 2019.

Vancouver:

Kacsinta AD. Determining Biological Effectors of alpha6 Integrin Cleavage . [Internet] [Doctoral dissertation]. University of Arizona; 2010. [cited 2019 Nov 17]. Available from: http://hdl.handle.net/10150/193604.

Council of Science Editors:

Kacsinta AD. Determining Biological Effectors of alpha6 Integrin Cleavage . [Doctoral Dissertation]. University of Arizona; 2010. Available from: http://hdl.handle.net/10150/193604


University of Arizona

6. Villeneuve, Nicole Frances. Mechanistic Study of USP15-Dependent Deubiquitination and Characterization of Natural Compounds that Modulate the Nrf2-Keap1 Antioxidant Response .

Degree: 2011, University of Arizona

 Nrf2 (NF-E2-related factor 2) is a transcription factor that regulates a battery of downstream genes that contain an antioxidant response element (ARE) in their promoters,… (more)

Subjects/Keywords: cinnamic aldehyde; Keap1; Nrf2; oridonin; ubiquitination; USP15

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APA (6th Edition):

Villeneuve, N. F. (2011). Mechanistic Study of USP15-Dependent Deubiquitination and Characterization of Natural Compounds that Modulate the Nrf2-Keap1 Antioxidant Response . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/145720

Chicago Manual of Style (16th Edition):

Villeneuve, Nicole Frances. “Mechanistic Study of USP15-Dependent Deubiquitination and Characterization of Natural Compounds that Modulate the Nrf2-Keap1 Antioxidant Response .” 2011. Doctoral Dissertation, University of Arizona. Accessed November 17, 2019. http://hdl.handle.net/10150/145720.

MLA Handbook (7th Edition):

Villeneuve, Nicole Frances. “Mechanistic Study of USP15-Dependent Deubiquitination and Characterization of Natural Compounds that Modulate the Nrf2-Keap1 Antioxidant Response .” 2011. Web. 17 Nov 2019.

Vancouver:

Villeneuve NF. Mechanistic Study of USP15-Dependent Deubiquitination and Characterization of Natural Compounds that Modulate the Nrf2-Keap1 Antioxidant Response . [Internet] [Doctoral dissertation]. University of Arizona; 2011. [cited 2019 Nov 17]. Available from: http://hdl.handle.net/10150/145720.

Council of Science Editors:

Villeneuve NF. Mechanistic Study of USP15-Dependent Deubiquitination and Characterization of Natural Compounds that Modulate the Nrf2-Keap1 Antioxidant Response . [Doctoral Dissertation]. University of Arizona; 2011. Available from: http://hdl.handle.net/10150/145720


University of Arizona

7. High, Rachel. Evaluations of pancreatic cancer with acidoCEST MRI .

Degree: 2019, University of Arizona

 Pancreatic ductal adenocarcinoma (PDAC) is a virulent disease which readily develops resistances to prevailing chemotherapies. PDAC is often not diagnosed until the disease presents in… (more)

Subjects/Keywords: acidosis; cancer; MRI; pancreatic; PDAC; pH

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APA (6th Edition):

High, R. (2019). Evaluations of pancreatic cancer with acidoCEST MRI . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/631951

Chicago Manual of Style (16th Edition):

High, Rachel. “Evaluations of pancreatic cancer with acidoCEST MRI .” 2019. Doctoral Dissertation, University of Arizona. Accessed November 17, 2019. http://hdl.handle.net/10150/631951.

MLA Handbook (7th Edition):

High, Rachel. “Evaluations of pancreatic cancer with acidoCEST MRI .” 2019. Web. 17 Nov 2019.

Vancouver:

High R. Evaluations of pancreatic cancer with acidoCEST MRI . [Internet] [Doctoral dissertation]. University of Arizona; 2019. [cited 2019 Nov 17]. Available from: http://hdl.handle.net/10150/631951.

Council of Science Editors:

High R. Evaluations of pancreatic cancer with acidoCEST MRI . [Doctoral Dissertation]. University of Arizona; 2019. Available from: http://hdl.handle.net/10150/631951


University of Arizona

8. Kadiyala, Vineela. Class I Lysine Deacetylases Facilitate Glucocorticoid Receptor-Mediated Transcriptional Activation .

Degree: 2013, University of Arizona

 Glucocorticoid receptor (GR) is known to associate with KATs and KDACs to regulate transcription. The current model of GR-mediated transcription focuses on agonist-dependent recruitment of… (more)

Subjects/Keywords: Lysine deaectylases; Transcription; Chemistry; Glucocorticoid receptor

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APA (6th Edition):

Kadiyala, V. (2013). Class I Lysine Deacetylases Facilitate Glucocorticoid Receptor-Mediated Transcriptional Activation . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/311669

Chicago Manual of Style (16th Edition):

Kadiyala, Vineela. “Class I Lysine Deacetylases Facilitate Glucocorticoid Receptor-Mediated Transcriptional Activation .” 2013. Doctoral Dissertation, University of Arizona. Accessed November 17, 2019. http://hdl.handle.net/10150/311669.

MLA Handbook (7th Edition):

Kadiyala, Vineela. “Class I Lysine Deacetylases Facilitate Glucocorticoid Receptor-Mediated Transcriptional Activation .” 2013. Web. 17 Nov 2019.

Vancouver:

Kadiyala V. Class I Lysine Deacetylases Facilitate Glucocorticoid Receptor-Mediated Transcriptional Activation . [Internet] [Doctoral dissertation]. University of Arizona; 2013. [cited 2019 Nov 17]. Available from: http://hdl.handle.net/10150/311669.

Council of Science Editors:

Kadiyala V. Class I Lysine Deacetylases Facilitate Glucocorticoid Receptor-Mediated Transcriptional Activation . [Doctoral Dissertation]. University of Arizona; 2013. Available from: http://hdl.handle.net/10150/311669


University of Arizona

9. Patrick, Nina M. Class I Lysine Deacetylases Facilitate Glucocorticoid-Mediated Gene Activation and Repression .

Degree: 2015, University of Arizona

 Lysine acetyltransferases (KATs) and lysine deacetylases (KDACs) are known to cooperate with the glucocorticoid receptor (GR) to regulate transcription. The current model of GR-mediated transcription… (more)

Subjects/Keywords: Gene Activation; Gene Repression; Glucocorticoid Receptor; KDAC; LSD1; Pharmaceutical Sciences; Deacetylase

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APA (6th Edition):

Patrick, N. M. (2015). Class I Lysine Deacetylases Facilitate Glucocorticoid-Mediated Gene Activation and Repression . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/594555

Chicago Manual of Style (16th Edition):

Patrick, Nina M. “Class I Lysine Deacetylases Facilitate Glucocorticoid-Mediated Gene Activation and Repression .” 2015. Doctoral Dissertation, University of Arizona. Accessed November 17, 2019. http://hdl.handle.net/10150/594555.

MLA Handbook (7th Edition):

Patrick, Nina M. “Class I Lysine Deacetylases Facilitate Glucocorticoid-Mediated Gene Activation and Repression .” 2015. Web. 17 Nov 2019.

Vancouver:

Patrick NM. Class I Lysine Deacetylases Facilitate Glucocorticoid-Mediated Gene Activation and Repression . [Internet] [Doctoral dissertation]. University of Arizona; 2015. [cited 2019 Nov 17]. Available from: http://hdl.handle.net/10150/594555.

Council of Science Editors:

Patrick NM. Class I Lysine Deacetylases Facilitate Glucocorticoid-Mediated Gene Activation and Repression . [Doctoral Dissertation]. University of Arizona; 2015. Available from: http://hdl.handle.net/10150/594555


University of Arizona

10. Rheinheimer, Brenna Ann. Alternative Transcription Of The SLIT2/Mir-218-1 Transcriptional Axis Mediates Pancreatic Cancer Invasion .

Degree: 2016, University of Arizona

 The development of several organ systems through modeling and shaping of the tissue structure occurs from signaling through axon guidance molecules. The Slit family of… (more)

Subjects/Keywords: Epigenetics; Invasion; miRNA; Pancreatic cancer; Transcriptional regulation; Cancer Biology; Axon guidance molecules

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APA (6th Edition):

Rheinheimer, B. A. (2016). Alternative Transcription Of The SLIT2/Mir-218-1 Transcriptional Axis Mediates Pancreatic Cancer Invasion . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/605118

Chicago Manual of Style (16th Edition):

Rheinheimer, Brenna Ann. “Alternative Transcription Of The SLIT2/Mir-218-1 Transcriptional Axis Mediates Pancreatic Cancer Invasion .” 2016. Doctoral Dissertation, University of Arizona. Accessed November 17, 2019. http://hdl.handle.net/10150/605118.

MLA Handbook (7th Edition):

Rheinheimer, Brenna Ann. “Alternative Transcription Of The SLIT2/Mir-218-1 Transcriptional Axis Mediates Pancreatic Cancer Invasion .” 2016. Web. 17 Nov 2019.

Vancouver:

Rheinheimer BA. Alternative Transcription Of The SLIT2/Mir-218-1 Transcriptional Axis Mediates Pancreatic Cancer Invasion . [Internet] [Doctoral dissertation]. University of Arizona; 2016. [cited 2019 Nov 17]. Available from: http://hdl.handle.net/10150/605118.

Council of Science Editors:

Rheinheimer BA. Alternative Transcription Of The SLIT2/Mir-218-1 Transcriptional Axis Mediates Pancreatic Cancer Invasion . [Doctoral Dissertation]. University of Arizona; 2016. Available from: http://hdl.handle.net/10150/605118


University of Arizona

11. Wnek, Shawn Michael. Mechanisms of malignant transformation of human urothelial cells by monomethylarsonous acid .

Degree: 2011, University of Arizona

 Sources of arsenic exposure include air, water, and food from both natural and anthropogenic sources. Arsenic is categorized as a human carcinogen, and is associated… (more)

Subjects/Keywords: DNA damage; DNA repair; monomethylarsonous acid; Pharmacology & Toxicology; arsenic; bladder cancer

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APA (6th Edition):

Wnek, S. M. (2011). Mechanisms of malignant transformation of human urothelial cells by monomethylarsonous acid . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/201495

Chicago Manual of Style (16th Edition):

Wnek, Shawn Michael. “Mechanisms of malignant transformation of human urothelial cells by monomethylarsonous acid .” 2011. Doctoral Dissertation, University of Arizona. Accessed November 17, 2019. http://hdl.handle.net/10150/201495.

MLA Handbook (7th Edition):

Wnek, Shawn Michael. “Mechanisms of malignant transformation of human urothelial cells by monomethylarsonous acid .” 2011. Web. 17 Nov 2019.

Vancouver:

Wnek SM. Mechanisms of malignant transformation of human urothelial cells by monomethylarsonous acid . [Internet] [Doctoral dissertation]. University of Arizona; 2011. [cited 2019 Nov 17]. Available from: http://hdl.handle.net/10150/201495.

Council of Science Editors:

Wnek SM. Mechanisms of malignant transformation of human urothelial cells by monomethylarsonous acid . [Doctoral Dissertation]. University of Arizona; 2011. Available from: http://hdl.handle.net/10150/201495


University of Arizona

12. Gomez Rubio, Paulina. DETERMINANTS OF INTERINDIVIDUAL VARIABILITY IN ARSENIC SECONDARY METHYLATION EFFICIENCY IN A POPULATION FROM NORTHWEST MEXICO .

Degree: 2011, University of Arizona

 Chronic environmental exposure to inorganic arsenic is widely associated with human disease. Low human arsenic secondary methylation efficiency (SME), represented by high urinary monomethylarsonic acid… (more)

Subjects/Keywords: AS3MT; BMI; methylation; MMA; Pharmacology & Toxicology; Ancestry; Arsenic

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APA (6th Edition):

Gomez Rubio, P. (2011). DETERMINANTS OF INTERINDIVIDUAL VARIABILITY IN ARSENIC SECONDARY METHYLATION EFFICIENCY IN A POPULATION FROM NORTHWEST MEXICO . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/203433

Chicago Manual of Style (16th Edition):

Gomez Rubio, Paulina. “DETERMINANTS OF INTERINDIVIDUAL VARIABILITY IN ARSENIC SECONDARY METHYLATION EFFICIENCY IN A POPULATION FROM NORTHWEST MEXICO .” 2011. Doctoral Dissertation, University of Arizona. Accessed November 17, 2019. http://hdl.handle.net/10150/203433.

MLA Handbook (7th Edition):

Gomez Rubio, Paulina. “DETERMINANTS OF INTERINDIVIDUAL VARIABILITY IN ARSENIC SECONDARY METHYLATION EFFICIENCY IN A POPULATION FROM NORTHWEST MEXICO .” 2011. Web. 17 Nov 2019.

Vancouver:

Gomez Rubio P. DETERMINANTS OF INTERINDIVIDUAL VARIABILITY IN ARSENIC SECONDARY METHYLATION EFFICIENCY IN A POPULATION FROM NORTHWEST MEXICO . [Internet] [Doctoral dissertation]. University of Arizona; 2011. [cited 2019 Nov 17]. Available from: http://hdl.handle.net/10150/203433.

Council of Science Editors:

Gomez Rubio P. DETERMINANTS OF INTERINDIVIDUAL VARIABILITY IN ARSENIC SECONDARY METHYLATION EFFICIENCY IN A POPULATION FROM NORTHWEST MEXICO . [Doctoral Dissertation]. University of Arizona; 2011. Available from: http://hdl.handle.net/10150/203433


University of Arizona

13. Gustafson, Heather Lynn. Role of Manganese Superoxide Dismutase in Chemotherapy-induced Oxidative Stress .

Degree: 2011, University of Arizona

 Existing treatments for mantle cell lymphoma (MCL) are non-curative, demonstrating a need for a refined treatment approach. Recent clinical trials have shown promising results with… (more)

Subjects/Keywords: Oxidative Stress; Cancer Biology; Mantle Cell Lymphoma; mTOR inhibitors

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APA (6th Edition):

Gustafson, H. L. (2011). Role of Manganese Superoxide Dismutase in Chemotherapy-induced Oxidative Stress . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/203510

Chicago Manual of Style (16th Edition):

Gustafson, Heather Lynn. “Role of Manganese Superoxide Dismutase in Chemotherapy-induced Oxidative Stress .” 2011. Doctoral Dissertation, University of Arizona. Accessed November 17, 2019. http://hdl.handle.net/10150/203510.

MLA Handbook (7th Edition):

Gustafson, Heather Lynn. “Role of Manganese Superoxide Dismutase in Chemotherapy-induced Oxidative Stress .” 2011. Web. 17 Nov 2019.

Vancouver:

Gustafson HL. Role of Manganese Superoxide Dismutase in Chemotherapy-induced Oxidative Stress . [Internet] [Doctoral dissertation]. University of Arizona; 2011. [cited 2019 Nov 17]. Available from: http://hdl.handle.net/10150/203510.

Council of Science Editors:

Gustafson HL. Role of Manganese Superoxide Dismutase in Chemotherapy-induced Oxidative Stress . [Doctoral Dissertation]. University of Arizona; 2011. Available from: http://hdl.handle.net/10150/203510


University of Arizona

14. Tula Sanchez, Ana A. Elucidation of the Mechanisms of Resistance and Sensitivity to Histone Deacetylase Inhibitor, PXD101, in Diffuse Large B-Cell Lymphoma (DLBCL) .

Degree: 2013, University of Arizona

 Although curable in the majority of cases, Diffuse Large B-cell Lymphoma (DLBCL), the most prevalent Non-Hodgkin Lymphoma (NHL) throughout the world, is still fatal for… (more)

Subjects/Keywords: HDACs; PXD101; Pharmacology & Toxicology; DLBCL

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APA (6th Edition):

Tula Sanchez, A. A. (2013). Elucidation of the Mechanisms of Resistance and Sensitivity to Histone Deacetylase Inhibitor, PXD101, in Diffuse Large B-Cell Lymphoma (DLBCL) . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/301692

Chicago Manual of Style (16th Edition):

Tula Sanchez, Ana A. “Elucidation of the Mechanisms of Resistance and Sensitivity to Histone Deacetylase Inhibitor, PXD101, in Diffuse Large B-Cell Lymphoma (DLBCL) .” 2013. Doctoral Dissertation, University of Arizona. Accessed November 17, 2019. http://hdl.handle.net/10150/301692.

MLA Handbook (7th Edition):

Tula Sanchez, Ana A. “Elucidation of the Mechanisms of Resistance and Sensitivity to Histone Deacetylase Inhibitor, PXD101, in Diffuse Large B-Cell Lymphoma (DLBCL) .” 2013. Web. 17 Nov 2019.

Vancouver:

Tula Sanchez AA. Elucidation of the Mechanisms of Resistance and Sensitivity to Histone Deacetylase Inhibitor, PXD101, in Diffuse Large B-Cell Lymphoma (DLBCL) . [Internet] [Doctoral dissertation]. University of Arizona; 2013. [cited 2019 Nov 17]. Available from: http://hdl.handle.net/10150/301692.

Council of Science Editors:

Tula Sanchez AA. Elucidation of the Mechanisms of Resistance and Sensitivity to Histone Deacetylase Inhibitor, PXD101, in Diffuse Large B-Cell Lymphoma (DLBCL) . [Doctoral Dissertation]. University of Arizona; 2013. Available from: http://hdl.handle.net/10150/301692

15. Severson, Paul Leamon. Epigenomic Actions of Environmental Arsenicals .

Degree: 2013, University of Arizona

 Epigenetic dysfunction is a known contributor in carcinogenesis, and is emerging as a mechanism involved in toxicant-induced malignant transformation for environmental carcinogens such as arsenicals.… (more)

Subjects/Keywords: DNA methylation; Epigenetics; Histone methylation; Malignant Transformation; Zinc Finger Genes; Pharmacology & Toxicology; Arsenic

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APA (6th Edition):

Severson, P. L. (2013). Epigenomic Actions of Environmental Arsenicals . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/299122

Chicago Manual of Style (16th Edition):

Severson, Paul Leamon. “Epigenomic Actions of Environmental Arsenicals .” 2013. Doctoral Dissertation, University of Arizona. Accessed November 17, 2019. http://hdl.handle.net/10150/299122.

MLA Handbook (7th Edition):

Severson, Paul Leamon. “Epigenomic Actions of Environmental Arsenicals .” 2013. Web. 17 Nov 2019.

Vancouver:

Severson PL. Epigenomic Actions of Environmental Arsenicals . [Internet] [Doctoral dissertation]. University of Arizona; 2013. [cited 2019 Nov 17]. Available from: http://hdl.handle.net/10150/299122.

Council of Science Editors:

Severson PL. Epigenomic Actions of Environmental Arsenicals . [Doctoral Dissertation]. University of Arizona; 2013. Available from: http://hdl.handle.net/10150/299122


University of Arizona

16. Dodge, Jonathan Eldon. Selective variegated methylation of the p15/INK4B CpG island is a high frequency event in acute myeloid leukemia (AML) .

Degree: 2000, University of Arizona

 We attempted to define target genes that were inactivated in acute myeloid leukemia (AML) by DNA methylation. We hypothesized that hypermethylation of 51 CpG islands… (more)

Subjects/Keywords: Biology, Molecular.; Health Sciences, Pharmacology.; Health Sciences, Oncology.

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APA (6th Edition):

Dodge, J. E. (2000). Selective variegated methylation of the p15/INK4B CpG island is a high frequency event in acute myeloid leukemia (AML) . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/284143

Chicago Manual of Style (16th Edition):

Dodge, Jonathan Eldon. “Selective variegated methylation of the p15/INK4B CpG island is a high frequency event in acute myeloid leukemia (AML) .” 2000. Doctoral Dissertation, University of Arizona. Accessed November 17, 2019. http://hdl.handle.net/10150/284143.

MLA Handbook (7th Edition):

Dodge, Jonathan Eldon. “Selective variegated methylation of the p15/INK4B CpG island is a high frequency event in acute myeloid leukemia (AML) .” 2000. Web. 17 Nov 2019.

Vancouver:

Dodge JE. Selective variegated methylation of the p15/INK4B CpG island is a high frequency event in acute myeloid leukemia (AML) . [Internet] [Doctoral dissertation]. University of Arizona; 2000. [cited 2019 Nov 17]. Available from: http://hdl.handle.net/10150/284143.

Council of Science Editors:

Dodge JE. Selective variegated methylation of the p15/INK4B CpG island is a high frequency event in acute myeloid leukemia (AML) . [Doctoral Dissertation]. University of Arizona; 2000. Available from: http://hdl.handle.net/10150/284143


University of Arizona

17. Rice, Judd Christopher. Epigenetic silencing of BRCA1 and maspin in sporadic breast cancer .

Degree: 2000, University of Arizona

 The RNA expression of the tumor suppressor genes BRCA1 and maspin are frequently decreased or lost in sporadic breast cancer. We hypothesized that inactivation of… (more)

Subjects/Keywords: Biology, Molecular.; Biology, Genetics.; Health Sciences, Oncology.

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APA (6th Edition):

Rice, J. C. (2000). Epigenetic silencing of BRCA1 and maspin in sporadic breast cancer . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/289142

Chicago Manual of Style (16th Edition):

Rice, Judd Christopher. “Epigenetic silencing of BRCA1 and maspin in sporadic breast cancer .” 2000. Doctoral Dissertation, University of Arizona. Accessed November 17, 2019. http://hdl.handle.net/10150/289142.

MLA Handbook (7th Edition):

Rice, Judd Christopher. “Epigenetic silencing of BRCA1 and maspin in sporadic breast cancer .” 2000. Web. 17 Nov 2019.

Vancouver:

Rice JC. Epigenetic silencing of BRCA1 and maspin in sporadic breast cancer . [Internet] [Doctoral dissertation]. University of Arizona; 2000. [cited 2019 Nov 17]. Available from: http://hdl.handle.net/10150/289142.

Council of Science Editors:

Rice JC. Epigenetic silencing of BRCA1 and maspin in sporadic breast cancer . [Doctoral Dissertation]. University of Arizona; 2000. Available from: http://hdl.handle.net/10150/289142


University of Arizona

18. Watts, George Somerset, 1971-. The role of 5-methylcytosine in the expression of O(6)-methylguanine DNA-methyltransferase .

Degree: 1998, University of Arizona

 Regulation of the gene which encodes the human DNA repair enzyme O6-methylguanine DNA methyltransferase (MGMT) is of clinical interest and provides a gene model for… (more)

Subjects/Keywords: Biology, Molecular.; Health Sciences, Pharmacology.

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APA (6th Edition):

Watts, George Somerset, 1. (1998). The role of 5-methylcytosine in the expression of O(6)-methylguanine DNA-methyltransferase . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/282599

Chicago Manual of Style (16th Edition):

Watts, George Somerset, 1971-. “The role of 5-methylcytosine in the expression of O(6)-methylguanine DNA-methyltransferase .” 1998. Doctoral Dissertation, University of Arizona. Accessed November 17, 2019. http://hdl.handle.net/10150/282599.

MLA Handbook (7th Edition):

Watts, George Somerset, 1971-. “The role of 5-methylcytosine in the expression of O(6)-methylguanine DNA-methyltransferase .” 1998. Web. 17 Nov 2019.

Vancouver:

Watts, George Somerset 1. The role of 5-methylcytosine in the expression of O(6)-methylguanine DNA-methyltransferase . [Internet] [Doctoral dissertation]. University of Arizona; 1998. [cited 2019 Nov 17]. Available from: http://hdl.handle.net/10150/282599.

Council of Science Editors:

Watts, George Somerset 1. The role of 5-methylcytosine in the expression of O(6)-methylguanine DNA-methyltransferase . [Doctoral Dissertation]. University of Arizona; 1998. Available from: http://hdl.handle.net/10150/282599

19. Medeiros, Matthew Keane. Genomic Response in Human Urothelial Cells Exposed Chronically to Monomethylarsonous Acid .

Degree: 2013, University of Arizona

 Bladder cancer has been associated with chronic arsenic exposure. Monomethylarsonous acid [MMA(III)] is a metabolite of inorganic arsenic biotransformation and has been shown to transform… (more)

Subjects/Keywords: bladder; cancer biology; gene expression; gene microarray; urothelial cells; Pharmacology & Toxicology; arsenic

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APA (6th Edition):

Medeiros, M. K. (2013). Genomic Response in Human Urothelial Cells Exposed Chronically to Monomethylarsonous Acid . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/299107

Chicago Manual of Style (16th Edition):

Medeiros, Matthew Keane. “Genomic Response in Human Urothelial Cells Exposed Chronically to Monomethylarsonous Acid .” 2013. Doctoral Dissertation, University of Arizona. Accessed November 17, 2019. http://hdl.handle.net/10150/299107.

MLA Handbook (7th Edition):

Medeiros, Matthew Keane. “Genomic Response in Human Urothelial Cells Exposed Chronically to Monomethylarsonous Acid .” 2013. Web. 17 Nov 2019.

Vancouver:

Medeiros MK. Genomic Response in Human Urothelial Cells Exposed Chronically to Monomethylarsonous Acid . [Internet] [Doctoral dissertation]. University of Arizona; 2013. [cited 2019 Nov 17]. Available from: http://hdl.handle.net/10150/299107.

Council of Science Editors:

Medeiros MK. Genomic Response in Human Urothelial Cells Exposed Chronically to Monomethylarsonous Acid . [Doctoral Dissertation]. University of Arizona; 2013. Available from: http://hdl.handle.net/10150/299107

20. Bolt, Alicia Marie. Arsenite Alters Lysosome-Mediated Degradation and the Autophagy Process Leading to Immunosuppression in Human B-Lymphoblastoid Cell Lines .

Degree: 2012, University of Arizona

 The immune system is a target of arsenic toxicity. Epidemiological data have shown that arsenic exposure is associated with characteristics of immunosuppression. Human B-lymphoblastoid cell… (more)

Subjects/Keywords: Immunotoxicity; Lymphoblastoid; Proteotoxicity; Pharmacology & Toxicology; Arsenic; Autophagy

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APA (6th Edition):

Bolt, A. M. (2012). Arsenite Alters Lysosome-Mediated Degradation and the Autophagy Process Leading to Immunosuppression in Human B-Lymphoblastoid Cell Lines . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/222895

Chicago Manual of Style (16th Edition):

Bolt, Alicia Marie. “Arsenite Alters Lysosome-Mediated Degradation and the Autophagy Process Leading to Immunosuppression in Human B-Lymphoblastoid Cell Lines .” 2012. Doctoral Dissertation, University of Arizona. Accessed November 17, 2019. http://hdl.handle.net/10150/222895.

MLA Handbook (7th Edition):

Bolt, Alicia Marie. “Arsenite Alters Lysosome-Mediated Degradation and the Autophagy Process Leading to Immunosuppression in Human B-Lymphoblastoid Cell Lines .” 2012. Web. 17 Nov 2019.

Vancouver:

Bolt AM. Arsenite Alters Lysosome-Mediated Degradation and the Autophagy Process Leading to Immunosuppression in Human B-Lymphoblastoid Cell Lines . [Internet] [Doctoral dissertation]. University of Arizona; 2012. [cited 2019 Nov 17]. Available from: http://hdl.handle.net/10150/222895.

Council of Science Editors:

Bolt AM. Arsenite Alters Lysosome-Mediated Degradation and the Autophagy Process Leading to Immunosuppression in Human B-Lymphoblastoid Cell Lines . [Doctoral Dissertation]. University of Arizona; 2012. Available from: http://hdl.handle.net/10150/222895

21. Sollome, James Jerome. Heregulin Activates a Novel HER2/HER3-MTK1-GIT1/ERK1/2 MAPK Signaling Pathway .

Degree: 2014, University of Arizona

 Human MAP3K4 (MTK1) functions upstream of mitogen activated protein kinases (MAPKs). In the studies presented herein, MTK1 is shown to be required for human epidermal… (more)

Subjects/Keywords: Pharmacology & Toxicology

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APA (6th Edition):

Sollome, J. J. (2014). Heregulin Activates a Novel HER2/HER3-MTK1-GIT1/ERK1/2 MAPK Signaling Pathway . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/315554

Chicago Manual of Style (16th Edition):

Sollome, James Jerome. “Heregulin Activates a Novel HER2/HER3-MTK1-GIT1/ERK1/2 MAPK Signaling Pathway .” 2014. Doctoral Dissertation, University of Arizona. Accessed November 17, 2019. http://hdl.handle.net/10150/315554.

MLA Handbook (7th Edition):

Sollome, James Jerome. “Heregulin Activates a Novel HER2/HER3-MTK1-GIT1/ERK1/2 MAPK Signaling Pathway .” 2014. Web. 17 Nov 2019.

Vancouver:

Sollome JJ. Heregulin Activates a Novel HER2/HER3-MTK1-GIT1/ERK1/2 MAPK Signaling Pathway . [Internet] [Doctoral dissertation]. University of Arizona; 2014. [cited 2019 Nov 17]. Available from: http://hdl.handle.net/10150/315554.

Council of Science Editors:

Sollome JJ. Heregulin Activates a Novel HER2/HER3-MTK1-GIT1/ERK1/2 MAPK Signaling Pathway . [Doctoral Dissertation]. University of Arizona; 2014. Available from: http://hdl.handle.net/10150/315554


University of Arizona

22. Oshiro, Marc Makoto. Role of interleukin-6 signaling via the Jak/Stat pathway in human myeloma and effects on mediators of cell survival and proliferation .

Degree: 2000, University of Arizona

 Multiple myeloma is a B-cell malignancy characterized by the latent accumulation in bone marrow of monoclonal plasma cells with a low proliferative index and an… (more)

Subjects/Keywords: Biology, Cell.; Health Sciences, Toxicology.; Health Sciences, Pharmacology.

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APA (6th Edition):

Oshiro, M. M. (2000). Role of interleukin-6 signaling via the Jak/Stat pathway in human myeloma and effects on mediators of cell survival and proliferation . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/284289

Chicago Manual of Style (16th Edition):

Oshiro, Marc Makoto. “Role of interleukin-6 signaling via the Jak/Stat pathway in human myeloma and effects on mediators of cell survival and proliferation .” 2000. Doctoral Dissertation, University of Arizona. Accessed November 17, 2019. http://hdl.handle.net/10150/284289.

MLA Handbook (7th Edition):

Oshiro, Marc Makoto. “Role of interleukin-6 signaling via the Jak/Stat pathway in human myeloma and effects on mediators of cell survival and proliferation .” 2000. Web. 17 Nov 2019.

Vancouver:

Oshiro MM. Role of interleukin-6 signaling via the Jak/Stat pathway in human myeloma and effects on mediators of cell survival and proliferation . [Internet] [Doctoral dissertation]. University of Arizona; 2000. [cited 2019 Nov 17]. Available from: http://hdl.handle.net/10150/284289.

Council of Science Editors:

Oshiro MM. Role of interleukin-6 signaling via the Jak/Stat pathway in human myeloma and effects on mediators of cell survival and proliferation . [Doctoral Dissertation]. University of Arizona; 2000. Available from: http://hdl.handle.net/10150/284289


University of Arizona

23. Wozniak, Ryan Joseph. Mechanisms Underlying the Pharmacologic Reversal of Genetic and Epigenetic Components of Tumor Suppressor Gene Silencing in Human Breast Cancer .

Degree: 2006, University of Arizona

 In women, tumors of the breast remain the most frequently diagnosed malignancy and the second leading cause of cancer-related deaths. One of the hallmarks of… (more)

Subjects/Keywords: Pharmacology; Cancer; Genetics; Epigenetics

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APA (6th Edition):

Wozniak, R. J. (2006). Mechanisms Underlying the Pharmacologic Reversal of Genetic and Epigenetic Components of Tumor Suppressor Gene Silencing in Human Breast Cancer . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/195193

Chicago Manual of Style (16th Edition):

Wozniak, Ryan Joseph. “Mechanisms Underlying the Pharmacologic Reversal of Genetic and Epigenetic Components of Tumor Suppressor Gene Silencing in Human Breast Cancer .” 2006. Doctoral Dissertation, University of Arizona. Accessed November 17, 2019. http://hdl.handle.net/10150/195193.

MLA Handbook (7th Edition):

Wozniak, Ryan Joseph. “Mechanisms Underlying the Pharmacologic Reversal of Genetic and Epigenetic Components of Tumor Suppressor Gene Silencing in Human Breast Cancer .” 2006. Web. 17 Nov 2019.

Vancouver:

Wozniak RJ. Mechanisms Underlying the Pharmacologic Reversal of Genetic and Epigenetic Components of Tumor Suppressor Gene Silencing in Human Breast Cancer . [Internet] [Doctoral dissertation]. University of Arizona; 2006. [cited 2019 Nov 17]. Available from: http://hdl.handle.net/10150/195193.

Council of Science Editors:

Wozniak RJ. Mechanisms Underlying the Pharmacologic Reversal of Genetic and Epigenetic Components of Tumor Suppressor Gene Silencing in Human Breast Cancer . [Doctoral Dissertation]. University of Arizona; 2006. Available from: http://hdl.handle.net/10150/195193


University of Arizona

24. Jensen, Taylor Jacob. EPIGENETIC REMODELING DURING ARSENICAL-INDUCED MALIGNANT TRANSFORMATION .

Degree: 2008, University of Arizona

 Humans are exposed to arsenicals through many routes with the most common being drinking water. Exposure to arsenic has been associated with an increased incidence… (more)

Subjects/Keywords: Arsenic; Carcinogenesis; DNA Methylation; Epigenetic; Histone Acetylation; MMA(III)

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APA (6th Edition):

Jensen, T. J. (2008). EPIGENETIC REMODELING DURING ARSENICAL-INDUCED MALIGNANT TRANSFORMATION . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/193558

Chicago Manual of Style (16th Edition):

Jensen, Taylor Jacob. “EPIGENETIC REMODELING DURING ARSENICAL-INDUCED MALIGNANT TRANSFORMATION .” 2008. Doctoral Dissertation, University of Arizona. Accessed November 17, 2019. http://hdl.handle.net/10150/193558.

MLA Handbook (7th Edition):

Jensen, Taylor Jacob. “EPIGENETIC REMODELING DURING ARSENICAL-INDUCED MALIGNANT TRANSFORMATION .” 2008. Web. 17 Nov 2019.

Vancouver:

Jensen TJ. EPIGENETIC REMODELING DURING ARSENICAL-INDUCED MALIGNANT TRANSFORMATION . [Internet] [Doctoral dissertation]. University of Arizona; 2008. [cited 2019 Nov 17]. Available from: http://hdl.handle.net/10150/193558.

Council of Science Editors:

Jensen TJ. EPIGENETIC REMODELING DURING ARSENICAL-INDUCED MALIGNANT TRANSFORMATION . [Doctoral Dissertation]. University of Arizona; 2008. Available from: http://hdl.handle.net/10150/193558


University of Arizona

25. Manza, Linda Lee. Characterization of Protein Sumoylation in Response to Alkylation Stress in HEK 293 Cells .

Degree: 2007, University of Arizona

 Stress conditions such as heat shock, UV, alkylating agents, and H2O2 have been shown to result in the modification of a variety of protein targets… (more)

Subjects/Keywords: Sumoylation; alkylation; hydroxynonenal; LC-MS-MS analysis

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APA (6th Edition):

Manza, L. L. (2007). Characterization of Protein Sumoylation in Response to Alkylation Stress in HEK 293 Cells . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/193944

Chicago Manual of Style (16th Edition):

Manza, Linda Lee. “Characterization of Protein Sumoylation in Response to Alkylation Stress in HEK 293 Cells .” 2007. Doctoral Dissertation, University of Arizona. Accessed November 17, 2019. http://hdl.handle.net/10150/193944.

MLA Handbook (7th Edition):

Manza, Linda Lee. “Characterization of Protein Sumoylation in Response to Alkylation Stress in HEK 293 Cells .” 2007. Web. 17 Nov 2019.

Vancouver:

Manza LL. Characterization of Protein Sumoylation in Response to Alkylation Stress in HEK 293 Cells . [Internet] [Doctoral dissertation]. University of Arizona; 2007. [cited 2019 Nov 17]. Available from: http://hdl.handle.net/10150/193944.

Council of Science Editors:

Manza LL. Characterization of Protein Sumoylation in Response to Alkylation Stress in HEK 293 Cells . [Doctoral Dissertation]. University of Arizona; 2007. Available from: http://hdl.handle.net/10150/193944


University of Arizona

26. Beilke, Lisa D. Mechanisms of Hepatoprotection in a Murine Model of Bile Acid-Induced Intrahepatic Cholestasis .

Degree: 2008, University of Arizona

 There are many causes of cholestasis, which results when the flow of bile acids is slowed or stopped. Bile acids are hydrophobic molecules synthesized from… (more)

Subjects/Keywords: Cholestasis; bile acid; biosynthesis; apoptosis; constitutive androstane receptor

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APA (6th Edition):

Beilke, L. D. (2008). Mechanisms of Hepatoprotection in a Murine Model of Bile Acid-Induced Intrahepatic Cholestasis . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/194264

Chicago Manual of Style (16th Edition):

Beilke, Lisa D. “Mechanisms of Hepatoprotection in a Murine Model of Bile Acid-Induced Intrahepatic Cholestasis .” 2008. Doctoral Dissertation, University of Arizona. Accessed November 17, 2019. http://hdl.handle.net/10150/194264.

MLA Handbook (7th Edition):

Beilke, Lisa D. “Mechanisms of Hepatoprotection in a Murine Model of Bile Acid-Induced Intrahepatic Cholestasis .” 2008. Web. 17 Nov 2019.

Vancouver:

Beilke LD. Mechanisms of Hepatoprotection in a Murine Model of Bile Acid-Induced Intrahepatic Cholestasis . [Internet] [Doctoral dissertation]. University of Arizona; 2008. [cited 2019 Nov 17]. Available from: http://hdl.handle.net/10150/194264.

Council of Science Editors:

Beilke LD. Mechanisms of Hepatoprotection in a Murine Model of Bile Acid-Induced Intrahepatic Cholestasis . [Doctoral Dissertation]. University of Arizona; 2008. Available from: http://hdl.handle.net/10150/194264


University of Arizona

27. Palumbo, SunMi Lee. Characterization of Secondary DNA Structures Formed in the c-myb and hTERT Promoters and Their Potential Role in the Regulation of Transcription .

Degree: 2009, University of Arizona

 In this dissertation, the formation of unusual G-quadruplexes in the critical regions of the c-myb and hTERT promoters for control of promoter activity was investigated.The… (more)

Subjects/Keywords: c-myb; G-quadruplex; hTERT; secondary DNA structure; transcription

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APA (6th Edition):

Palumbo, S. L. (2009). Characterization of Secondary DNA Structures Formed in the c-myb and hTERT Promoters and Their Potential Role in the Regulation of Transcription . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/194266

Chicago Manual of Style (16th Edition):

Palumbo, SunMi Lee. “Characterization of Secondary DNA Structures Formed in the c-myb and hTERT Promoters and Their Potential Role in the Regulation of Transcription .” 2009. Doctoral Dissertation, University of Arizona. Accessed November 17, 2019. http://hdl.handle.net/10150/194266.

MLA Handbook (7th Edition):

Palumbo, SunMi Lee. “Characterization of Secondary DNA Structures Formed in the c-myb and hTERT Promoters and Their Potential Role in the Regulation of Transcription .” 2009. Web. 17 Nov 2019.

Vancouver:

Palumbo SL. Characterization of Secondary DNA Structures Formed in the c-myb and hTERT Promoters and Their Potential Role in the Regulation of Transcription . [Internet] [Doctoral dissertation]. University of Arizona; 2009. [cited 2019 Nov 17]. Available from: http://hdl.handle.net/10150/194266.

Council of Science Editors:

Palumbo SL. Characterization of Secondary DNA Structures Formed in the c-myb and hTERT Promoters and Their Potential Role in the Regulation of Transcription . [Doctoral Dissertation]. University of Arizona; 2009. Available from: http://hdl.handle.net/10150/194266


University of Arizona

28. Sun, Haipeng. Regulation of Cyclooxygenase Gene Expression by Glucocorticoids in Cardiomyocytes .

Degree: 2007, University of Arizona

 Glucocorticoids (GCs) are endogenous steroid hormones that regulate a number of critical physiological processes. Psychological stress increases the level of GCs in the circulating system.… (more)

Subjects/Keywords: glucocorticoid; COX-1; COX-2; cardiomyocytes; gene; regulation

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APA (6th Edition):

Sun, H. (2007). Regulation of Cyclooxygenase Gene Expression by Glucocorticoids in Cardiomyocytes . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/194896

Chicago Manual of Style (16th Edition):

Sun, Haipeng. “Regulation of Cyclooxygenase Gene Expression by Glucocorticoids in Cardiomyocytes .” 2007. Doctoral Dissertation, University of Arizona. Accessed November 17, 2019. http://hdl.handle.net/10150/194896.

MLA Handbook (7th Edition):

Sun, Haipeng. “Regulation of Cyclooxygenase Gene Expression by Glucocorticoids in Cardiomyocytes .” 2007. Web. 17 Nov 2019.

Vancouver:

Sun H. Regulation of Cyclooxygenase Gene Expression by Glucocorticoids in Cardiomyocytes . [Internet] [Doctoral dissertation]. University of Arizona; 2007. [cited 2019 Nov 17]. Available from: http://hdl.handle.net/10150/194896.

Council of Science Editors:

Sun H. Regulation of Cyclooxygenase Gene Expression by Glucocorticoids in Cardiomyocytes . [Doctoral Dissertation]. University of Arizona; 2007. Available from: http://hdl.handle.net/10150/194896


University of Arizona

29. Bredfeldt, Tiffany Gail. Low-Level Arsenic Toxicity in Human Bladder Cells .

Degree: 2006, University of Arizona

 Arsenic is a human bladder carcinogen. Inorganic arsenic and methylated metabolites are excreted from the human body in urine. This study investigates the effects of… (more)

Subjects/Keywords: arsenic; arsenic methylation; carcinogenesis; bladder cancer

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APA (6th Edition):

Bredfeldt, T. G. (2006). Low-Level Arsenic Toxicity in Human Bladder Cells . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/195157

Chicago Manual of Style (16th Edition):

Bredfeldt, Tiffany Gail. “Low-Level Arsenic Toxicity in Human Bladder Cells .” 2006. Doctoral Dissertation, University of Arizona. Accessed November 17, 2019. http://hdl.handle.net/10150/195157.

MLA Handbook (7th Edition):

Bredfeldt, Tiffany Gail. “Low-Level Arsenic Toxicity in Human Bladder Cells .” 2006. Web. 17 Nov 2019.

Vancouver:

Bredfeldt TG. Low-Level Arsenic Toxicity in Human Bladder Cells . [Internet] [Doctoral dissertation]. University of Arizona; 2006. [cited 2019 Nov 17]. Available from: http://hdl.handle.net/10150/195157.

Council of Science Editors:

Bredfeldt TG. Low-Level Arsenic Toxicity in Human Bladder Cells . [Doctoral Dissertation]. University of Arizona; 2006. Available from: http://hdl.handle.net/10150/195157


University of Arizona

30. Eblin, Kylee Elaine. Arsenical-induced Reactive Oxygen Species Lead to Altered Cellular Signaling and Phenotypic Alterations in Human Bladder Cells .

Degree: 2008, University of Arizona

 Arsenical-induced carcinogenesis in human bladder has been established through epidemiological evidence, but unfortunately, no mode of action had been determined for this phenomenon. UROtsa cells,… (more)

Subjects/Keywords: Arsenite; Monomethylarsonous acid; ROS; MAPK signaling

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APA (6th Edition):

Eblin, K. E. (2008). Arsenical-induced Reactive Oxygen Species Lead to Altered Cellular Signaling and Phenotypic Alterations in Human Bladder Cells . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/195706

Chicago Manual of Style (16th Edition):

Eblin, Kylee Elaine. “Arsenical-induced Reactive Oxygen Species Lead to Altered Cellular Signaling and Phenotypic Alterations in Human Bladder Cells .” 2008. Doctoral Dissertation, University of Arizona. Accessed November 17, 2019. http://hdl.handle.net/10150/195706.

MLA Handbook (7th Edition):

Eblin, Kylee Elaine. “Arsenical-induced Reactive Oxygen Species Lead to Altered Cellular Signaling and Phenotypic Alterations in Human Bladder Cells .” 2008. Web. 17 Nov 2019.

Vancouver:

Eblin KE. Arsenical-induced Reactive Oxygen Species Lead to Altered Cellular Signaling and Phenotypic Alterations in Human Bladder Cells . [Internet] [Doctoral dissertation]. University of Arizona; 2008. [cited 2019 Nov 17]. Available from: http://hdl.handle.net/10150/195706.

Council of Science Editors:

Eblin KE. Arsenical-induced Reactive Oxygen Species Lead to Altered Cellular Signaling and Phenotypic Alterations in Human Bladder Cells . [Doctoral Dissertation]. University of Arizona; 2008. Available from: http://hdl.handle.net/10150/195706

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