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You searched for +publisher:"University of Arizona" +contributor:("Futscher, Bernard W."). Showing records 1 – 22 of 22 total matches.

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University of Arizona

1. Muñoz-Rodríguez, José Luis. Postpartum Breast Cancer in Hispanic Women: Epigenetics and microRNAs .

Degree: 2015, University of Arizona

 The risk of breast cancer transiently increases immediately following pregnancy. Hispanic women have one of the highest rates of postpartum breast cancers of all racial/ethnic… (more)

Subjects/Keywords: microRNAs; postpartum breast cancer; Pharmacology & Toxicology; epigenetics

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APA (6th Edition):

Muñoz-Rodríguez, J. L. (2015). Postpartum Breast Cancer in Hispanic Women: Epigenetics and microRNAs . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/603490

Chicago Manual of Style (16th Edition):

Muñoz-Rodríguez, José Luis. “Postpartum Breast Cancer in Hispanic Women: Epigenetics and microRNAs .” 2015. Doctoral Dissertation, University of Arizona. Accessed March 07, 2021. http://hdl.handle.net/10150/603490.

MLA Handbook (7th Edition):

Muñoz-Rodríguez, José Luis. “Postpartum Breast Cancer in Hispanic Women: Epigenetics and microRNAs .” 2015. Web. 07 Mar 2021.

Vancouver:

Muñoz-Rodríguez JL. Postpartum Breast Cancer in Hispanic Women: Epigenetics and microRNAs . [Internet] [Doctoral dissertation]. University of Arizona; 2015. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/10150/603490.

Council of Science Editors:

Muñoz-Rodríguez JL. Postpartum Breast Cancer in Hispanic Women: Epigenetics and microRNAs . [Doctoral Dissertation]. University of Arizona; 2015. Available from: http://hdl.handle.net/10150/603490


University of Arizona

2. Griggs, Chanel Antoinette. Lysine Deacetylase Inhibitors Disrupt Glucocorticoid Receptor-Mediated Transcription and Circadian Rhythm .

Degree: 2018, University of Arizona

 Glucocorticoids mediate their anti-inflammatory and immunosuppressive effects through activation of the glucocorticoid receptor (GR), a nuclear receptor, and subsequent effects on gene transcription. GR use… (more)

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APA (6th Edition):

Griggs, C. A. (2018). Lysine Deacetylase Inhibitors Disrupt Glucocorticoid Receptor-Mediated Transcription and Circadian Rhythm . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/627675

Chicago Manual of Style (16th Edition):

Griggs, Chanel Antoinette. “Lysine Deacetylase Inhibitors Disrupt Glucocorticoid Receptor-Mediated Transcription and Circadian Rhythm .” 2018. Doctoral Dissertation, University of Arizona. Accessed March 07, 2021. http://hdl.handle.net/10150/627675.

MLA Handbook (7th Edition):

Griggs, Chanel Antoinette. “Lysine Deacetylase Inhibitors Disrupt Glucocorticoid Receptor-Mediated Transcription and Circadian Rhythm .” 2018. Web. 07 Mar 2021.

Vancouver:

Griggs CA. Lysine Deacetylase Inhibitors Disrupt Glucocorticoid Receptor-Mediated Transcription and Circadian Rhythm . [Internet] [Doctoral dissertation]. University of Arizona; 2018. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/10150/627675.

Council of Science Editors:

Griggs CA. Lysine Deacetylase Inhibitors Disrupt Glucocorticoid Receptor-Mediated Transcription and Circadian Rhythm . [Doctoral Dissertation]. University of Arizona; 2018. Available from: http://hdl.handle.net/10150/627675


University of Arizona

3. Villeneuve, Nicole Frances. Mechanistic Study of USP15-Dependent Deubiquitination and Characterization of Natural Compounds that Modulate the Nrf2-Keap1 Antioxidant Response .

Degree: 2011, University of Arizona

 Nrf2 (NF-E2-related factor 2) is a transcription factor that regulates a battery of downstream genes that contain an antioxidant response element (ARE) in their promoters,… (more)

Subjects/Keywords: cinnamic aldehyde; Keap1; Nrf2; oridonin; ubiquitination; USP15

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APA (6th Edition):

Villeneuve, N. F. (2011). Mechanistic Study of USP15-Dependent Deubiquitination and Characterization of Natural Compounds that Modulate the Nrf2-Keap1 Antioxidant Response . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/145720

Chicago Manual of Style (16th Edition):

Villeneuve, Nicole Frances. “Mechanistic Study of USP15-Dependent Deubiquitination and Characterization of Natural Compounds that Modulate the Nrf2-Keap1 Antioxidant Response .” 2011. Doctoral Dissertation, University of Arizona. Accessed March 07, 2021. http://hdl.handle.net/10150/145720.

MLA Handbook (7th Edition):

Villeneuve, Nicole Frances. “Mechanistic Study of USP15-Dependent Deubiquitination and Characterization of Natural Compounds that Modulate the Nrf2-Keap1 Antioxidant Response .” 2011. Web. 07 Mar 2021.

Vancouver:

Villeneuve NF. Mechanistic Study of USP15-Dependent Deubiquitination and Characterization of Natural Compounds that Modulate the Nrf2-Keap1 Antioxidant Response . [Internet] [Doctoral dissertation]. University of Arizona; 2011. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/10150/145720.

Council of Science Editors:

Villeneuve NF. Mechanistic Study of USP15-Dependent Deubiquitination and Characterization of Natural Compounds that Modulate the Nrf2-Keap1 Antioxidant Response . [Doctoral Dissertation]. University of Arizona; 2011. Available from: http://hdl.handle.net/10150/145720


University of Arizona

4. Wnek, Shawn Michael. Mechanisms of malignant transformation of human urothelial cells by monomethylarsonous acid .

Degree: 2011, University of Arizona

 Sources of arsenic exposure include air, water, and food from both natural and anthropogenic sources. Arsenic is categorized as a human carcinogen, and is associated… (more)

Subjects/Keywords: DNA damage; DNA repair; monomethylarsonous acid; Pharmacology & Toxicology; arsenic; bladder cancer

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APA (6th Edition):

Wnek, S. M. (2011). Mechanisms of malignant transformation of human urothelial cells by monomethylarsonous acid . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/201495

Chicago Manual of Style (16th Edition):

Wnek, Shawn Michael. “Mechanisms of malignant transformation of human urothelial cells by monomethylarsonous acid .” 2011. Doctoral Dissertation, University of Arizona. Accessed March 07, 2021. http://hdl.handle.net/10150/201495.

MLA Handbook (7th Edition):

Wnek, Shawn Michael. “Mechanisms of malignant transformation of human urothelial cells by monomethylarsonous acid .” 2011. Web. 07 Mar 2021.

Vancouver:

Wnek SM. Mechanisms of malignant transformation of human urothelial cells by monomethylarsonous acid . [Internet] [Doctoral dissertation]. University of Arizona; 2011. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/10150/201495.

Council of Science Editors:

Wnek SM. Mechanisms of malignant transformation of human urothelial cells by monomethylarsonous acid . [Doctoral Dissertation]. University of Arizona; 2011. Available from: http://hdl.handle.net/10150/201495


University of Arizona

5. Gomez Rubio, Paulina. DETERMINANTS OF INTERINDIVIDUAL VARIABILITY IN ARSENIC SECONDARY METHYLATION EFFICIENCY IN A POPULATION FROM NORTHWEST MEXICO .

Degree: 2011, University of Arizona

 Chronic environmental exposure to inorganic arsenic is widely associated with human disease. Low human arsenic secondary methylation efficiency (SME), represented by high urinary monomethylarsonic acid… (more)

Subjects/Keywords: AS3MT; BMI; methylation; MMA; Pharmacology & Toxicology; Ancestry; Arsenic

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APA (6th Edition):

Gomez Rubio, P. (2011). DETERMINANTS OF INTERINDIVIDUAL VARIABILITY IN ARSENIC SECONDARY METHYLATION EFFICIENCY IN A POPULATION FROM NORTHWEST MEXICO . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/203433

Chicago Manual of Style (16th Edition):

Gomez Rubio, Paulina. “DETERMINANTS OF INTERINDIVIDUAL VARIABILITY IN ARSENIC SECONDARY METHYLATION EFFICIENCY IN A POPULATION FROM NORTHWEST MEXICO .” 2011. Doctoral Dissertation, University of Arizona. Accessed March 07, 2021. http://hdl.handle.net/10150/203433.

MLA Handbook (7th Edition):

Gomez Rubio, Paulina. “DETERMINANTS OF INTERINDIVIDUAL VARIABILITY IN ARSENIC SECONDARY METHYLATION EFFICIENCY IN A POPULATION FROM NORTHWEST MEXICO .” 2011. Web. 07 Mar 2021.

Vancouver:

Gomez Rubio P. DETERMINANTS OF INTERINDIVIDUAL VARIABILITY IN ARSENIC SECONDARY METHYLATION EFFICIENCY IN A POPULATION FROM NORTHWEST MEXICO . [Internet] [Doctoral dissertation]. University of Arizona; 2011. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/10150/203433.

Council of Science Editors:

Gomez Rubio P. DETERMINANTS OF INTERINDIVIDUAL VARIABILITY IN ARSENIC SECONDARY METHYLATION EFFICIENCY IN A POPULATION FROM NORTHWEST MEXICO . [Doctoral Dissertation]. University of Arizona; 2011. Available from: http://hdl.handle.net/10150/203433


University of Arizona

6. Tula Sanchez, Ana A. Elucidation of the Mechanisms of Resistance and Sensitivity to Histone Deacetylase Inhibitor, PXD101, in Diffuse Large B-Cell Lymphoma (DLBCL) .

Degree: 2013, University of Arizona

 Although curable in the majority of cases, Diffuse Large B-cell Lymphoma (DLBCL), the most prevalent Non-Hodgkin Lymphoma (NHL) throughout the world, is still fatal for… (more)

Subjects/Keywords: HDACs; PXD101; Pharmacology & Toxicology; DLBCL

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APA (6th Edition):

Tula Sanchez, A. A. (2013). Elucidation of the Mechanisms of Resistance and Sensitivity to Histone Deacetylase Inhibitor, PXD101, in Diffuse Large B-Cell Lymphoma (DLBCL) . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/301692

Chicago Manual of Style (16th Edition):

Tula Sanchez, Ana A. “Elucidation of the Mechanisms of Resistance and Sensitivity to Histone Deacetylase Inhibitor, PXD101, in Diffuse Large B-Cell Lymphoma (DLBCL) .” 2013. Doctoral Dissertation, University of Arizona. Accessed March 07, 2021. http://hdl.handle.net/10150/301692.

MLA Handbook (7th Edition):

Tula Sanchez, Ana A. “Elucidation of the Mechanisms of Resistance and Sensitivity to Histone Deacetylase Inhibitor, PXD101, in Diffuse Large B-Cell Lymphoma (DLBCL) .” 2013. Web. 07 Mar 2021.

Vancouver:

Tula Sanchez AA. Elucidation of the Mechanisms of Resistance and Sensitivity to Histone Deacetylase Inhibitor, PXD101, in Diffuse Large B-Cell Lymphoma (DLBCL) . [Internet] [Doctoral dissertation]. University of Arizona; 2013. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/10150/301692.

Council of Science Editors:

Tula Sanchez AA. Elucidation of the Mechanisms of Resistance and Sensitivity to Histone Deacetylase Inhibitor, PXD101, in Diffuse Large B-Cell Lymphoma (DLBCL) . [Doctoral Dissertation]. University of Arizona; 2013. Available from: http://hdl.handle.net/10150/301692

7. Severson, Paul Leamon. Epigenomic Actions of Environmental Arsenicals .

Degree: 2013, University of Arizona

 Epigenetic dysfunction is a known contributor in carcinogenesis, and is emerging as a mechanism involved in toxicant-induced malignant transformation for environmental carcinogens such as arsenicals.… (more)

Subjects/Keywords: DNA methylation; Epigenetics; Histone methylation; Malignant Transformation; Zinc Finger Genes; Pharmacology & Toxicology; Arsenic

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APA (6th Edition):

Severson, P. L. (2013). Epigenomic Actions of Environmental Arsenicals . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/299122

Chicago Manual of Style (16th Edition):

Severson, Paul Leamon. “Epigenomic Actions of Environmental Arsenicals .” 2013. Doctoral Dissertation, University of Arizona. Accessed March 07, 2021. http://hdl.handle.net/10150/299122.

MLA Handbook (7th Edition):

Severson, Paul Leamon. “Epigenomic Actions of Environmental Arsenicals .” 2013. Web. 07 Mar 2021.

Vancouver:

Severson PL. Epigenomic Actions of Environmental Arsenicals . [Internet] [Doctoral dissertation]. University of Arizona; 2013. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/10150/299122.

Council of Science Editors:

Severson PL. Epigenomic Actions of Environmental Arsenicals . [Doctoral Dissertation]. University of Arizona; 2013. Available from: http://hdl.handle.net/10150/299122


University of Arizona

8. Dodge, Jonathan Eldon. Selective variegated methylation of the p15/INK4B CpG island is a high frequency event in acute myeloid leukemia (AML) .

Degree: 2000, University of Arizona

 We attempted to define target genes that were inactivated in acute myeloid leukemia (AML) by DNA methylation. We hypothesized that hypermethylation of 51 CpG islands… (more)

Subjects/Keywords: Biology, Molecular.; Health Sciences, Pharmacology.; Health Sciences, Oncology.

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APA (6th Edition):

Dodge, J. E. (2000). Selective variegated methylation of the p15/INK4B CpG island is a high frequency event in acute myeloid leukemia (AML) . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/284143

Chicago Manual of Style (16th Edition):

Dodge, Jonathan Eldon. “Selective variegated methylation of the p15/INK4B CpG island is a high frequency event in acute myeloid leukemia (AML) .” 2000. Doctoral Dissertation, University of Arizona. Accessed March 07, 2021. http://hdl.handle.net/10150/284143.

MLA Handbook (7th Edition):

Dodge, Jonathan Eldon. “Selective variegated methylation of the p15/INK4B CpG island is a high frequency event in acute myeloid leukemia (AML) .” 2000. Web. 07 Mar 2021.

Vancouver:

Dodge JE. Selective variegated methylation of the p15/INK4B CpG island is a high frequency event in acute myeloid leukemia (AML) . [Internet] [Doctoral dissertation]. University of Arizona; 2000. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/10150/284143.

Council of Science Editors:

Dodge JE. Selective variegated methylation of the p15/INK4B CpG island is a high frequency event in acute myeloid leukemia (AML) . [Doctoral Dissertation]. University of Arizona; 2000. Available from: http://hdl.handle.net/10150/284143


University of Arizona

9. Rice, Judd Christopher. Epigenetic silencing of BRCA1 and maspin in sporadic breast cancer .

Degree: 2000, University of Arizona

 The RNA expression of the tumor suppressor genes BRCA1 and maspin are frequently decreased or lost in sporadic breast cancer. We hypothesized that inactivation of… (more)

Subjects/Keywords: Biology, Molecular.; Biology, Genetics.; Health Sciences, Oncology.

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APA (6th Edition):

Rice, J. C. (2000). Epigenetic silencing of BRCA1 and maspin in sporadic breast cancer . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/289142

Chicago Manual of Style (16th Edition):

Rice, Judd Christopher. “Epigenetic silencing of BRCA1 and maspin in sporadic breast cancer .” 2000. Doctoral Dissertation, University of Arizona. Accessed March 07, 2021. http://hdl.handle.net/10150/289142.

MLA Handbook (7th Edition):

Rice, Judd Christopher. “Epigenetic silencing of BRCA1 and maspin in sporadic breast cancer .” 2000. Web. 07 Mar 2021.

Vancouver:

Rice JC. Epigenetic silencing of BRCA1 and maspin in sporadic breast cancer . [Internet] [Doctoral dissertation]. University of Arizona; 2000. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/10150/289142.

Council of Science Editors:

Rice JC. Epigenetic silencing of BRCA1 and maspin in sporadic breast cancer . [Doctoral Dissertation]. University of Arizona; 2000. Available from: http://hdl.handle.net/10150/289142

10. High, Rachel. Evaluations of pancreatic cancer with acidoCEST MRI .

Degree: 2019, University of Arizona

 Pancreatic ductal adenocarcinoma (PDAC) is a virulent disease which readily develops resistances to prevailing chemotherapies. PDAC is often not diagnosed until the disease presents in… (more)

Subjects/Keywords: acidosis; cancer; MRI; pancreatic; PDAC; pH

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APA (6th Edition):

High, R. (2019). Evaluations of pancreatic cancer with acidoCEST MRI . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/631951

Chicago Manual of Style (16th Edition):

High, Rachel. “Evaluations of pancreatic cancer with acidoCEST MRI .” 2019. Doctoral Dissertation, University of Arizona. Accessed March 07, 2021. http://hdl.handle.net/10150/631951.

MLA Handbook (7th Edition):

High, Rachel. “Evaluations of pancreatic cancer with acidoCEST MRI .” 2019. Web. 07 Mar 2021.

Vancouver:

High R. Evaluations of pancreatic cancer with acidoCEST MRI . [Internet] [Doctoral dissertation]. University of Arizona; 2019. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/10150/631951.

Council of Science Editors:

High R. Evaluations of pancreatic cancer with acidoCEST MRI . [Doctoral Dissertation]. University of Arizona; 2019. Available from: http://hdl.handle.net/10150/631951

11. Sollome, James Jerome. Heregulin Activates a Novel HER2/HER3-MTK1-GIT1/ERK1/2 MAPK Signaling Pathway .

Degree: 2014, University of Arizona

 Human MAP3K4 (MTK1) functions upstream of mitogen activated protein kinases (MAPKs). In the studies presented herein, MTK1 is shown to be required for human epidermal… (more)

Subjects/Keywords: Pharmacology & Toxicology

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APA (6th Edition):

Sollome, J. J. (2014). Heregulin Activates a Novel HER2/HER3-MTK1-GIT1/ERK1/2 MAPK Signaling Pathway . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/315554

Chicago Manual of Style (16th Edition):

Sollome, James Jerome. “Heregulin Activates a Novel HER2/HER3-MTK1-GIT1/ERK1/2 MAPK Signaling Pathway .” 2014. Doctoral Dissertation, University of Arizona. Accessed March 07, 2021. http://hdl.handle.net/10150/315554.

MLA Handbook (7th Edition):

Sollome, James Jerome. “Heregulin Activates a Novel HER2/HER3-MTK1-GIT1/ERK1/2 MAPK Signaling Pathway .” 2014. Web. 07 Mar 2021.

Vancouver:

Sollome JJ. Heregulin Activates a Novel HER2/HER3-MTK1-GIT1/ERK1/2 MAPK Signaling Pathway . [Internet] [Doctoral dissertation]. University of Arizona; 2014. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/10150/315554.

Council of Science Editors:

Sollome JJ. Heregulin Activates a Novel HER2/HER3-MTK1-GIT1/ERK1/2 MAPK Signaling Pathway . [Doctoral Dissertation]. University of Arizona; 2014. Available from: http://hdl.handle.net/10150/315554


University of Arizona

12. Watts, George Somerset, 1971-. The role of 5-methylcytosine in the expression of O(6)-methylguanine DNA-methyltransferase .

Degree: 1998, University of Arizona

 Regulation of the gene which encodes the human DNA repair enzyme O6-methylguanine DNA methyltransferase (MGMT) is of clinical interest and provides a gene model for… (more)

Subjects/Keywords: Biology, Molecular.; Health Sciences, Pharmacology.

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APA (6th Edition):

Watts, George Somerset, 1. (1998). The role of 5-methylcytosine in the expression of O(6)-methylguanine DNA-methyltransferase . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/282599

Chicago Manual of Style (16th Edition):

Watts, George Somerset, 1971-. “The role of 5-methylcytosine in the expression of O(6)-methylguanine DNA-methyltransferase .” 1998. Doctoral Dissertation, University of Arizona. Accessed March 07, 2021. http://hdl.handle.net/10150/282599.

MLA Handbook (7th Edition):

Watts, George Somerset, 1971-. “The role of 5-methylcytosine in the expression of O(6)-methylguanine DNA-methyltransferase .” 1998. Web. 07 Mar 2021.

Vancouver:

Watts, George Somerset 1. The role of 5-methylcytosine in the expression of O(6)-methylguanine DNA-methyltransferase . [Internet] [Doctoral dissertation]. University of Arizona; 1998. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/10150/282599.

Council of Science Editors:

Watts, George Somerset 1. The role of 5-methylcytosine in the expression of O(6)-methylguanine DNA-methyltransferase . [Doctoral Dissertation]. University of Arizona; 1998. Available from: http://hdl.handle.net/10150/282599

13. Medeiros, Matthew Keane. Genomic Response in Human Urothelial Cells Exposed Chronically to Monomethylarsonous Acid .

Degree: 2013, University of Arizona

 Bladder cancer has been associated with chronic arsenic exposure. Monomethylarsonous acid [MMA(III)] is a metabolite of inorganic arsenic biotransformation and has been shown to transform… (more)

Subjects/Keywords: bladder; cancer biology; gene expression; gene microarray; urothelial cells; Pharmacology & Toxicology; arsenic

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APA (6th Edition):

Medeiros, M. K. (2013). Genomic Response in Human Urothelial Cells Exposed Chronically to Monomethylarsonous Acid . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/299107

Chicago Manual of Style (16th Edition):

Medeiros, Matthew Keane. “Genomic Response in Human Urothelial Cells Exposed Chronically to Monomethylarsonous Acid .” 2013. Doctoral Dissertation, University of Arizona. Accessed March 07, 2021. http://hdl.handle.net/10150/299107.

MLA Handbook (7th Edition):

Medeiros, Matthew Keane. “Genomic Response in Human Urothelial Cells Exposed Chronically to Monomethylarsonous Acid .” 2013. Web. 07 Mar 2021.

Vancouver:

Medeiros MK. Genomic Response in Human Urothelial Cells Exposed Chronically to Monomethylarsonous Acid . [Internet] [Doctoral dissertation]. University of Arizona; 2013. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/10150/299107.

Council of Science Editors:

Medeiros MK. Genomic Response in Human Urothelial Cells Exposed Chronically to Monomethylarsonous Acid . [Doctoral Dissertation]. University of Arizona; 2013. Available from: http://hdl.handle.net/10150/299107


University of Arizona

14. Oshiro, Marc Makoto. Role of interleukin-6 signaling via the Jak/Stat pathway in human myeloma and effects on mediators of cell survival and proliferation .

Degree: 2000, University of Arizona

 Multiple myeloma is a B-cell malignancy characterized by the latent accumulation in bone marrow of monoclonal plasma cells with a low proliferative index and an… (more)

Subjects/Keywords: Biology, Cell.; Health Sciences, Toxicology.; Health Sciences, Pharmacology.

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APA (6th Edition):

Oshiro, M. M. (2000). Role of interleukin-6 signaling via the Jak/Stat pathway in human myeloma and effects on mediators of cell survival and proliferation . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/284289

Chicago Manual of Style (16th Edition):

Oshiro, Marc Makoto. “Role of interleukin-6 signaling via the Jak/Stat pathway in human myeloma and effects on mediators of cell survival and proliferation .” 2000. Doctoral Dissertation, University of Arizona. Accessed March 07, 2021. http://hdl.handle.net/10150/284289.

MLA Handbook (7th Edition):

Oshiro, Marc Makoto. “Role of interleukin-6 signaling via the Jak/Stat pathway in human myeloma and effects on mediators of cell survival and proliferation .” 2000. Web. 07 Mar 2021.

Vancouver:

Oshiro MM. Role of interleukin-6 signaling via the Jak/Stat pathway in human myeloma and effects on mediators of cell survival and proliferation . [Internet] [Doctoral dissertation]. University of Arizona; 2000. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/10150/284289.

Council of Science Editors:

Oshiro MM. Role of interleukin-6 signaling via the Jak/Stat pathway in human myeloma and effects on mediators of cell survival and proliferation . [Doctoral Dissertation]. University of Arizona; 2000. Available from: http://hdl.handle.net/10150/284289


University of Arizona

15. Wozniak, Ryan Joseph. Mechanisms Underlying the Pharmacologic Reversal of Genetic and Epigenetic Components of Tumor Suppressor Gene Silencing in Human Breast Cancer .

Degree: 2006, University of Arizona

 In women, tumors of the breast remain the most frequently diagnosed malignancy and the second leading cause of cancer-related deaths. One of the hallmarks of… (more)

Subjects/Keywords: Pharmacology; Cancer; Genetics; Epigenetics

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APA (6th Edition):

Wozniak, R. J. (2006). Mechanisms Underlying the Pharmacologic Reversal of Genetic and Epigenetic Components of Tumor Suppressor Gene Silencing in Human Breast Cancer . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/195193

Chicago Manual of Style (16th Edition):

Wozniak, Ryan Joseph. “Mechanisms Underlying the Pharmacologic Reversal of Genetic and Epigenetic Components of Tumor Suppressor Gene Silencing in Human Breast Cancer .” 2006. Doctoral Dissertation, University of Arizona. Accessed March 07, 2021. http://hdl.handle.net/10150/195193.

MLA Handbook (7th Edition):

Wozniak, Ryan Joseph. “Mechanisms Underlying the Pharmacologic Reversal of Genetic and Epigenetic Components of Tumor Suppressor Gene Silencing in Human Breast Cancer .” 2006. Web. 07 Mar 2021.

Vancouver:

Wozniak RJ. Mechanisms Underlying the Pharmacologic Reversal of Genetic and Epigenetic Components of Tumor Suppressor Gene Silencing in Human Breast Cancer . [Internet] [Doctoral dissertation]. University of Arizona; 2006. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/10150/195193.

Council of Science Editors:

Wozniak RJ. Mechanisms Underlying the Pharmacologic Reversal of Genetic and Epigenetic Components of Tumor Suppressor Gene Silencing in Human Breast Cancer . [Doctoral Dissertation]. University of Arizona; 2006. Available from: http://hdl.handle.net/10150/195193


University of Arizona

16. Jensen, Taylor Jacob. EPIGENETIC REMODELING DURING ARSENICAL-INDUCED MALIGNANT TRANSFORMATION .

Degree: 2008, University of Arizona

 Humans are exposed to arsenicals through many routes with the most common being drinking water. Exposure to arsenic has been associated with an increased incidence… (more)

Subjects/Keywords: Arsenic; Carcinogenesis; DNA Methylation; Epigenetic; Histone Acetylation; MMA(III)

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Jensen, T. J. (2008). EPIGENETIC REMODELING DURING ARSENICAL-INDUCED MALIGNANT TRANSFORMATION . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/193558

Chicago Manual of Style (16th Edition):

Jensen, Taylor Jacob. “EPIGENETIC REMODELING DURING ARSENICAL-INDUCED MALIGNANT TRANSFORMATION .” 2008. Doctoral Dissertation, University of Arizona. Accessed March 07, 2021. http://hdl.handle.net/10150/193558.

MLA Handbook (7th Edition):

Jensen, Taylor Jacob. “EPIGENETIC REMODELING DURING ARSENICAL-INDUCED MALIGNANT TRANSFORMATION .” 2008. Web. 07 Mar 2021.

Vancouver:

Jensen TJ. EPIGENETIC REMODELING DURING ARSENICAL-INDUCED MALIGNANT TRANSFORMATION . [Internet] [Doctoral dissertation]. University of Arizona; 2008. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/10150/193558.

Council of Science Editors:

Jensen TJ. EPIGENETIC REMODELING DURING ARSENICAL-INDUCED MALIGNANT TRANSFORMATION . [Doctoral Dissertation]. University of Arizona; 2008. Available from: http://hdl.handle.net/10150/193558


University of Arizona

17. Manza, Linda Lee. Characterization of Protein Sumoylation in Response to Alkylation Stress in HEK 293 Cells .

Degree: 2007, University of Arizona

 Stress conditions such as heat shock, UV, alkylating agents, and H2O2 have been shown to result in the modification of a variety of protein targets… (more)

Subjects/Keywords: Sumoylation; alkylation; hydroxynonenal; LC-MS-MS analysis

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APA (6th Edition):

Manza, L. L. (2007). Characterization of Protein Sumoylation in Response to Alkylation Stress in HEK 293 Cells . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/193944

Chicago Manual of Style (16th Edition):

Manza, Linda Lee. “Characterization of Protein Sumoylation in Response to Alkylation Stress in HEK 293 Cells .” 2007. Doctoral Dissertation, University of Arizona. Accessed March 07, 2021. http://hdl.handle.net/10150/193944.

MLA Handbook (7th Edition):

Manza, Linda Lee. “Characterization of Protein Sumoylation in Response to Alkylation Stress in HEK 293 Cells .” 2007. Web. 07 Mar 2021.

Vancouver:

Manza LL. Characterization of Protein Sumoylation in Response to Alkylation Stress in HEK 293 Cells . [Internet] [Doctoral dissertation]. University of Arizona; 2007. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/10150/193944.

Council of Science Editors:

Manza LL. Characterization of Protein Sumoylation in Response to Alkylation Stress in HEK 293 Cells . [Doctoral Dissertation]. University of Arizona; 2007. Available from: http://hdl.handle.net/10150/193944


University of Arizona

18. Beilke, Lisa D. Mechanisms of Hepatoprotection in a Murine Model of Bile Acid-Induced Intrahepatic Cholestasis .

Degree: 2008, University of Arizona

 There are many causes of cholestasis, which results when the flow of bile acids is slowed or stopped. Bile acids are hydrophobic molecules synthesized from… (more)

Subjects/Keywords: Cholestasis; bile acid; biosynthesis; apoptosis; constitutive androstane receptor

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APA (6th Edition):

Beilke, L. D. (2008). Mechanisms of Hepatoprotection in a Murine Model of Bile Acid-Induced Intrahepatic Cholestasis . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/194264

Chicago Manual of Style (16th Edition):

Beilke, Lisa D. “Mechanisms of Hepatoprotection in a Murine Model of Bile Acid-Induced Intrahepatic Cholestasis .” 2008. Doctoral Dissertation, University of Arizona. Accessed March 07, 2021. http://hdl.handle.net/10150/194264.

MLA Handbook (7th Edition):

Beilke, Lisa D. “Mechanisms of Hepatoprotection in a Murine Model of Bile Acid-Induced Intrahepatic Cholestasis .” 2008. Web. 07 Mar 2021.

Vancouver:

Beilke LD. Mechanisms of Hepatoprotection in a Murine Model of Bile Acid-Induced Intrahepatic Cholestasis . [Internet] [Doctoral dissertation]. University of Arizona; 2008. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/10150/194264.

Council of Science Editors:

Beilke LD. Mechanisms of Hepatoprotection in a Murine Model of Bile Acid-Induced Intrahepatic Cholestasis . [Doctoral Dissertation]. University of Arizona; 2008. Available from: http://hdl.handle.net/10150/194264


University of Arizona

19. Palumbo, SunMi Lee. Characterization of Secondary DNA Structures Formed in the c-myb and hTERT Promoters and Their Potential Role in the Regulation of Transcription .

Degree: 2009, University of Arizona

 In this dissertation, the formation of unusual G-quadruplexes in the critical regions of the c-myb and hTERT promoters for control of promoter activity was investigated.The… (more)

Subjects/Keywords: c-myb; G-quadruplex; hTERT; secondary DNA structure; transcription

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Palumbo, S. L. (2009). Characterization of Secondary DNA Structures Formed in the c-myb and hTERT Promoters and Their Potential Role in the Regulation of Transcription . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/194266

Chicago Manual of Style (16th Edition):

Palumbo, SunMi Lee. “Characterization of Secondary DNA Structures Formed in the c-myb and hTERT Promoters and Their Potential Role in the Regulation of Transcription .” 2009. Doctoral Dissertation, University of Arizona. Accessed March 07, 2021. http://hdl.handle.net/10150/194266.

MLA Handbook (7th Edition):

Palumbo, SunMi Lee. “Characterization of Secondary DNA Structures Formed in the c-myb and hTERT Promoters and Their Potential Role in the Regulation of Transcription .” 2009. Web. 07 Mar 2021.

Vancouver:

Palumbo SL. Characterization of Secondary DNA Structures Formed in the c-myb and hTERT Promoters and Their Potential Role in the Regulation of Transcription . [Internet] [Doctoral dissertation]. University of Arizona; 2009. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/10150/194266.

Council of Science Editors:

Palumbo SL. Characterization of Secondary DNA Structures Formed in the c-myb and hTERT Promoters and Their Potential Role in the Regulation of Transcription . [Doctoral Dissertation]. University of Arizona; 2009. Available from: http://hdl.handle.net/10150/194266


University of Arizona

20. Hong, Fei. ANALYSIS OF ELECTROPHILE-INDUCED NRF2 GENE ACTIVATION .

Degree: 2005, University of Arizona

 Activation of the transcription factor Nrf2 regulates expression of phase II enzymes and other adaptive responses to electrophile and oxidant stress. Nrf2 concentrations are regulated… (more)

Subjects/Keywords: Pharmaceutical Sciences

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APA (6th Edition):

Hong, F. (2005). ANALYSIS OF ELECTROPHILE-INDUCED NRF2 GENE ACTIVATION . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/196091

Chicago Manual of Style (16th Edition):

Hong, Fei. “ANALYSIS OF ELECTROPHILE-INDUCED NRF2 GENE ACTIVATION .” 2005. Doctoral Dissertation, University of Arizona. Accessed March 07, 2021. http://hdl.handle.net/10150/196091.

MLA Handbook (7th Edition):

Hong, Fei. “ANALYSIS OF ELECTROPHILE-INDUCED NRF2 GENE ACTIVATION .” 2005. Web. 07 Mar 2021.

Vancouver:

Hong F. ANALYSIS OF ELECTROPHILE-INDUCED NRF2 GENE ACTIVATION . [Internet] [Doctoral dissertation]. University of Arizona; 2005. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/10150/196091.

Council of Science Editors:

Hong F. ANALYSIS OF ELECTROPHILE-INDUCED NRF2 GENE ACTIVATION . [Doctoral Dissertation]. University of Arizona; 2005. Available from: http://hdl.handle.net/10150/196091


University of Arizona

21. Stevens, Mark V. Distinct Functions of MEKK3 and MEKK4 in Heart Valve Morphogenesis .

Degree: 2008, University of Arizona

 Congenital heart defects (CHDs) occur in 5% of births. While gene mutations have been identified in CHD patients, not much is known about coordinated signaling… (more)

Subjects/Keywords: Endocardial cushion; Epithelial to Mesenchymal Transition; Heart; MEKK3; MEKK4; Valve

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Stevens, M. V. (2008). Distinct Functions of MEKK3 and MEKK4 in Heart Valve Morphogenesis . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/194848

Chicago Manual of Style (16th Edition):

Stevens, Mark V. “Distinct Functions of MEKK3 and MEKK4 in Heart Valve Morphogenesis .” 2008. Doctoral Dissertation, University of Arizona. Accessed March 07, 2021. http://hdl.handle.net/10150/194848.

MLA Handbook (7th Edition):

Stevens, Mark V. “Distinct Functions of MEKK3 and MEKK4 in Heart Valve Morphogenesis .” 2008. Web. 07 Mar 2021.

Vancouver:

Stevens MV. Distinct Functions of MEKK3 and MEKK4 in Heart Valve Morphogenesis . [Internet] [Doctoral dissertation]. University of Arizona; 2008. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/10150/194848.

Council of Science Editors:

Stevens MV. Distinct Functions of MEKK3 and MEKK4 in Heart Valve Morphogenesis . [Doctoral Dissertation]. University of Arizona; 2008. Available from: http://hdl.handle.net/10150/194848


University of Arizona

22. Catania, Jeffrey Mark. Derivations of Tissue Slice Technology as Toxicological Screening Systems .

Degree: 2006, University of Arizona

 In vitro toxicology studies are hindered by the use of specific cellular systems which solely examine one cell type. Precision-cut tissue slices mimic specific organ… (more)

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APA (6th Edition):

Catania, J. M. (2006). Derivations of Tissue Slice Technology as Toxicological Screening Systems . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/195419

Chicago Manual of Style (16th Edition):

Catania, Jeffrey Mark. “Derivations of Tissue Slice Technology as Toxicological Screening Systems .” 2006. Doctoral Dissertation, University of Arizona. Accessed March 07, 2021. http://hdl.handle.net/10150/195419.

MLA Handbook (7th Edition):

Catania, Jeffrey Mark. “Derivations of Tissue Slice Technology as Toxicological Screening Systems .” 2006. Web. 07 Mar 2021.

Vancouver:

Catania JM. Derivations of Tissue Slice Technology as Toxicological Screening Systems . [Internet] [Doctoral dissertation]. University of Arizona; 2006. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/10150/195419.

Council of Science Editors:

Catania JM. Derivations of Tissue Slice Technology as Toxicological Screening Systems . [Doctoral Dissertation]. University of Arizona; 2006. Available from: http://hdl.handle.net/10150/195419

.