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You searched for +publisher:"University of Arizona" +contributor:("Chen, Qin"). Showing records 1 – 27 of 27 total matches.

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University of Arizona

1. Maltagliati, Anthony. Nrf2: A Candidate Therapeutic Target to Dampen Oxidative Stress in Acute Myocardial Infarction .

Degree: 2016, University of Arizona

 This literature review posits that the transcription factor Nuclear factor (erythroid-derived 2)-like 2 (Nrf2) is an attractive candidate therapeutic target in the setting of acute… (more)

Subjects/Keywords: MI; myocardial infarction; Nrf2; oxidative stress; reactive oxygen species; heart attack

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APA (6th Edition):

Maltagliati, A. (2016). Nrf2: A Candidate Therapeutic Target to Dampen Oxidative Stress in Acute Myocardial Infarction . (Masters Thesis). University of Arizona. Retrieved from http://hdl.handle.net/10150/623086

Chicago Manual of Style (16th Edition):

Maltagliati, Anthony. “Nrf2: A Candidate Therapeutic Target to Dampen Oxidative Stress in Acute Myocardial Infarction .” 2016. Masters Thesis, University of Arizona. Accessed February 21, 2020. http://hdl.handle.net/10150/623086.

MLA Handbook (7th Edition):

Maltagliati, Anthony. “Nrf2: A Candidate Therapeutic Target to Dampen Oxidative Stress in Acute Myocardial Infarction .” 2016. Web. 21 Feb 2020.

Vancouver:

Maltagliati A. Nrf2: A Candidate Therapeutic Target to Dampen Oxidative Stress in Acute Myocardial Infarction . [Internet] [Masters thesis]. University of Arizona; 2016. [cited 2020 Feb 21]. Available from: http://hdl.handle.net/10150/623086.

Council of Science Editors:

Maltagliati A. Nrf2: A Candidate Therapeutic Target to Dampen Oxidative Stress in Acute Myocardial Infarction . [Masters Thesis]. University of Arizona; 2016. Available from: http://hdl.handle.net/10150/623086


University of Arizona

2. Braileanu, Anthony Leo. Teaching an Old Dog New Tricks: Introducing a Hypothetical Mechanism of Synergy Between Metformin and New Generation mTOR Inhibitors in the Fight Against Cancer .

Degree: 2019, University of Arizona

 mTOR is a serine-threonine kinase and the central component of the complexes mTORC1 and mTORC2. The protein mTORC1 is most associated with global regulation of… (more)

Subjects/Keywords: breast cancer; cancer; metformin; mTOR; rapamycin; synergy

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APA (6th Edition):

Braileanu, A. L. (2019). Teaching an Old Dog New Tricks: Introducing a Hypothetical Mechanism of Synergy Between Metformin and New Generation mTOR Inhibitors in the Fight Against Cancer . (Masters Thesis). University of Arizona. Retrieved from http://hdl.handle.net/10150/636653

Chicago Manual of Style (16th Edition):

Braileanu, Anthony Leo. “Teaching an Old Dog New Tricks: Introducing a Hypothetical Mechanism of Synergy Between Metformin and New Generation mTOR Inhibitors in the Fight Against Cancer .” 2019. Masters Thesis, University of Arizona. Accessed February 21, 2020. http://hdl.handle.net/10150/636653.

MLA Handbook (7th Edition):

Braileanu, Anthony Leo. “Teaching an Old Dog New Tricks: Introducing a Hypothetical Mechanism of Synergy Between Metformin and New Generation mTOR Inhibitors in the Fight Against Cancer .” 2019. Web. 21 Feb 2020.

Vancouver:

Braileanu AL. Teaching an Old Dog New Tricks: Introducing a Hypothetical Mechanism of Synergy Between Metformin and New Generation mTOR Inhibitors in the Fight Against Cancer . [Internet] [Masters thesis]. University of Arizona; 2019. [cited 2020 Feb 21]. Available from: http://hdl.handle.net/10150/636653.

Council of Science Editors:

Braileanu AL. Teaching an Old Dog New Tricks: Introducing a Hypothetical Mechanism of Synergy Between Metformin and New Generation mTOR Inhibitors in the Fight Against Cancer . [Masters Thesis]. University of Arizona; 2019. Available from: http://hdl.handle.net/10150/636653


University of Arizona

3. Kerins, Michael John. Ironing Out Roles and Regulation of NRF2 in a Hereditary Cancer Syndrome .

Degree: 2019, University of Arizona

 The deadly kidney cancers associated with hereditary leiomyomatosis and renal cell cancer (HLRCC) show activation of the nuclear factor (erythroid 2)-like 2 (NFE2L2, NRF2) transcription… (more)

Subjects/Keywords: Cancer; ferroptosis; HLRCC; iron; NRF2; signaling

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APA (6th Edition):

Kerins, M. J. (2019). Ironing Out Roles and Regulation of NRF2 in a Hereditary Cancer Syndrome . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/633229

Chicago Manual of Style (16th Edition):

Kerins, Michael John. “Ironing Out Roles and Regulation of NRF2 in a Hereditary Cancer Syndrome .” 2019. Doctoral Dissertation, University of Arizona. Accessed February 21, 2020. http://hdl.handle.net/10150/633229.

MLA Handbook (7th Edition):

Kerins, Michael John. “Ironing Out Roles and Regulation of NRF2 in a Hereditary Cancer Syndrome .” 2019. Web. 21 Feb 2020.

Vancouver:

Kerins MJ. Ironing Out Roles and Regulation of NRF2 in a Hereditary Cancer Syndrome . [Internet] [Doctoral dissertation]. University of Arizona; 2019. [cited 2020 Feb 21]. Available from: http://hdl.handle.net/10150/633229.

Council of Science Editors:

Kerins MJ. Ironing Out Roles and Regulation of NRF2 in a Hereditary Cancer Syndrome . [Doctoral Dissertation]. University of Arizona; 2019. Available from: http://hdl.handle.net/10150/633229


University of Arizona

4. Davis, Melissa Ann. Fetal Programming of Metabolic Disease: The Role of Catecholamines .

Degree: 2019, University of Arizona

 The objective of this dissertation research was to determine the chronic actions of catecholamines on insulin-stimulated glucose metabolism in the following models: 1) norepinephrine-infused ovine… (more)

Subjects/Keywords: adrenal demedullation; catecholamines; fetal development; growth restriction; hyperinsulinemic-euglycemic clamp; insulin-stimulated glucose metabolism

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APA (6th Edition):

Davis, M. A. (2019). Fetal Programming of Metabolic Disease: The Role of Catecholamines . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/634248

Chicago Manual of Style (16th Edition):

Davis, Melissa Ann. “Fetal Programming of Metabolic Disease: The Role of Catecholamines .” 2019. Doctoral Dissertation, University of Arizona. Accessed February 21, 2020. http://hdl.handle.net/10150/634248.

MLA Handbook (7th Edition):

Davis, Melissa Ann. “Fetal Programming of Metabolic Disease: The Role of Catecholamines .” 2019. Web. 21 Feb 2020.

Vancouver:

Davis MA. Fetal Programming of Metabolic Disease: The Role of Catecholamines . [Internet] [Doctoral dissertation]. University of Arizona; 2019. [cited 2020 Feb 21]. Available from: http://hdl.handle.net/10150/634248.

Council of Science Editors:

Davis MA. Fetal Programming of Metabolic Disease: The Role of Catecholamines . [Doctoral Dissertation]. University of Arizona; 2019. Available from: http://hdl.handle.net/10150/634248


University of Arizona

5. Montoya, Jesse Alan. Blood Product Administration and Kidney Function as a Mortality Indicator for VA-ECMO: A Retrospective Review of a Single Institution .

Degree: 2019, University of Arizona

 Background Veno-arterial extracorporeal membrane oxygenation (VA ECMO) is a rapidly growing treatment for critically ill patients. The management of this life-saving therapy is extremely complicated;… (more)

Subjects/Keywords: AKI; ECMO; Kidney; Mortality; Product; VA ECMO

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APA (6th Edition):

Montoya, J. A. (2019). Blood Product Administration and Kidney Function as a Mortality Indicator for VA-ECMO: A Retrospective Review of a Single Institution . (Masters Thesis). University of Arizona. Retrieved from http://hdl.handle.net/10150/633245

Chicago Manual of Style (16th Edition):

Montoya, Jesse Alan. “Blood Product Administration and Kidney Function as a Mortality Indicator for VA-ECMO: A Retrospective Review of a Single Institution .” 2019. Masters Thesis, University of Arizona. Accessed February 21, 2020. http://hdl.handle.net/10150/633245.

MLA Handbook (7th Edition):

Montoya, Jesse Alan. “Blood Product Administration and Kidney Function as a Mortality Indicator for VA-ECMO: A Retrospective Review of a Single Institution .” 2019. Web. 21 Feb 2020.

Vancouver:

Montoya JA. Blood Product Administration and Kidney Function as a Mortality Indicator for VA-ECMO: A Retrospective Review of a Single Institution . [Internet] [Masters thesis]. University of Arizona; 2019. [cited 2020 Feb 21]. Available from: http://hdl.handle.net/10150/633245.

Council of Science Editors:

Montoya JA. Blood Product Administration and Kidney Function as a Mortality Indicator for VA-ECMO: A Retrospective Review of a Single Institution . [Masters Thesis]. University of Arizona; 2019. Available from: http://hdl.handle.net/10150/633245


University of Arizona

6. Villeneuve, Nicole Frances. Mechanistic Study of USP15-Dependent Deubiquitination and Characterization of Natural Compounds that Modulate the Nrf2-Keap1 Antioxidant Response .

Degree: 2011, University of Arizona

 Nrf2 (NF-E2-related factor 2) is a transcription factor that regulates a battery of downstream genes that contain an antioxidant response element (ARE) in their promoters,… (more)

Subjects/Keywords: cinnamic aldehyde; Keap1; Nrf2; oridonin; ubiquitination; USP15

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APA (6th Edition):

Villeneuve, N. F. (2011). Mechanistic Study of USP15-Dependent Deubiquitination and Characterization of Natural Compounds that Modulate the Nrf2-Keap1 Antioxidant Response . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/145720

Chicago Manual of Style (16th Edition):

Villeneuve, Nicole Frances. “Mechanistic Study of USP15-Dependent Deubiquitination and Characterization of Natural Compounds that Modulate the Nrf2-Keap1 Antioxidant Response .” 2011. Doctoral Dissertation, University of Arizona. Accessed February 21, 2020. http://hdl.handle.net/10150/145720.

MLA Handbook (7th Edition):

Villeneuve, Nicole Frances. “Mechanistic Study of USP15-Dependent Deubiquitination and Characterization of Natural Compounds that Modulate the Nrf2-Keap1 Antioxidant Response .” 2011. Web. 21 Feb 2020.

Vancouver:

Villeneuve NF. Mechanistic Study of USP15-Dependent Deubiquitination and Characterization of Natural Compounds that Modulate the Nrf2-Keap1 Antioxidant Response . [Internet] [Doctoral dissertation]. University of Arizona; 2011. [cited 2020 Feb 21]. Available from: http://hdl.handle.net/10150/145720.

Council of Science Editors:

Villeneuve NF. Mechanistic Study of USP15-Dependent Deubiquitination and Characterization of Natural Compounds that Modulate the Nrf2-Keap1 Antioxidant Response . [Doctoral Dissertation]. University of Arizona; 2011. Available from: http://hdl.handle.net/10150/145720


University of Arizona

7. Merrell, Matthew David. Xenosensor Regulation of Enzymes and Transporters in Drug Exposure and Disease .

Degree: 2011, University of Arizona

 A large and varied array of xenobiotics (foreign chemicals) enters into our bodies every day. In order to prevent toxicity resulting from xenobiotic accumulation, the… (more)

Subjects/Keywords: Disease; Liver; Metabolism; Xenobiotic; Xenosensor

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APA (6th Edition):

Merrell, M. D. (2011). Xenosensor Regulation of Enzymes and Transporters in Drug Exposure and Disease . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/194051

Chicago Manual of Style (16th Edition):

Merrell, Matthew David. “Xenosensor Regulation of Enzymes and Transporters in Drug Exposure and Disease .” 2011. Doctoral Dissertation, University of Arizona. Accessed February 21, 2020. http://hdl.handle.net/10150/194051.

MLA Handbook (7th Edition):

Merrell, Matthew David. “Xenosensor Regulation of Enzymes and Transporters in Drug Exposure and Disease .” 2011. Web. 21 Feb 2020.

Vancouver:

Merrell MD. Xenosensor Regulation of Enzymes and Transporters in Drug Exposure and Disease . [Internet] [Doctoral dissertation]. University of Arizona; 2011. [cited 2020 Feb 21]. Available from: http://hdl.handle.net/10150/194051.

Council of Science Editors:

Merrell MD. Xenosensor Regulation of Enzymes and Transporters in Drug Exposure and Disease . [Doctoral Dissertation]. University of Arizona; 2011. Available from: http://hdl.handle.net/10150/194051


University of Arizona

8. Cheng, Yaofeng. Roles of organic cation transporters on the disposition of N-butylpyridinium chloride and structurally related ionic liquids .

Degree: 2010, University of Arizona

 Studies in this dissertation were conducted to explore the roles of organic cation transporters (OCTs) in the disposition of N-butylpyridinium Chloride (NBuPy-Cl) and structurally related… (more)

Subjects/Keywords: Ionic liquids; N-butylpyridinium chloride; organic cation transporters; pharmacokinetics; SAR; toxicity

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APA (6th Edition):

Cheng, Y. (2010). Roles of organic cation transporters on the disposition of N-butylpyridinium chloride and structurally related ionic liquids . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/195472

Chicago Manual of Style (16th Edition):

Cheng, Yaofeng. “Roles of organic cation transporters on the disposition of N-butylpyridinium chloride and structurally related ionic liquids .” 2010. Doctoral Dissertation, University of Arizona. Accessed February 21, 2020. http://hdl.handle.net/10150/195472.

MLA Handbook (7th Edition):

Cheng, Yaofeng. “Roles of organic cation transporters on the disposition of N-butylpyridinium chloride and structurally related ionic liquids .” 2010. Web. 21 Feb 2020.

Vancouver:

Cheng Y. Roles of organic cation transporters on the disposition of N-butylpyridinium chloride and structurally related ionic liquids . [Internet] [Doctoral dissertation]. University of Arizona; 2010. [cited 2020 Feb 21]. Available from: http://hdl.handle.net/10150/195472.

Council of Science Editors:

Cheng Y. Roles of organic cation transporters on the disposition of N-butylpyridinium chloride and structurally related ionic liquids . [Doctoral Dissertation]. University of Arizona; 2010. Available from: http://hdl.handle.net/10150/195472


University of Arizona

9. Torres Urquidy, Oscar Hernando. Copper Resistant Bacteria Better Tolerate Commercially Available Antimicrobial Treatments Based in Silver and Silver-Copper Ions .

Degree: 2011, University of Arizona

 In the current study, the antibacterial efficacy of zeolites containing silver or copper ions or a combination of these metals was assessed against several diverse… (more)

Subjects/Keywords: copper; copper resistant; silver; zeolite; Soil, Water & Environmental Science; antibacterial; bacteria

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APA (6th Edition):

Torres Urquidy, O. H. (2011). Copper Resistant Bacteria Better Tolerate Commercially Available Antimicrobial Treatments Based in Silver and Silver-Copper Ions . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/202734

Chicago Manual of Style (16th Edition):

Torres Urquidy, Oscar Hernando. “Copper Resistant Bacteria Better Tolerate Commercially Available Antimicrobial Treatments Based in Silver and Silver-Copper Ions .” 2011. Doctoral Dissertation, University of Arizona. Accessed February 21, 2020. http://hdl.handle.net/10150/202734.

MLA Handbook (7th Edition):

Torres Urquidy, Oscar Hernando. “Copper Resistant Bacteria Better Tolerate Commercially Available Antimicrobial Treatments Based in Silver and Silver-Copper Ions .” 2011. Web. 21 Feb 2020.

Vancouver:

Torres Urquidy OH. Copper Resistant Bacteria Better Tolerate Commercially Available Antimicrobial Treatments Based in Silver and Silver-Copper Ions . [Internet] [Doctoral dissertation]. University of Arizona; 2011. [cited 2020 Feb 21]. Available from: http://hdl.handle.net/10150/202734.

Council of Science Editors:

Torres Urquidy OH. Copper Resistant Bacteria Better Tolerate Commercially Available Antimicrobial Treatments Based in Silver and Silver-Copper Ions . [Doctoral Dissertation]. University of Arizona; 2011. Available from: http://hdl.handle.net/10150/202734


University of Arizona

10. Qiao, Shuxi. Pharmacological Modulation of Oxidative and Proteotoxic Stress for Antimelanoma Intervention .

Degree: 2013, University of Arizona

 Cumulative evidence suggests that constitutively elevated levels of proteotoxic stress represent a specific vulnerability of malignant cells that can be targeted by pharmacological modulation of… (more)

Subjects/Keywords: Pharmacology & Toxicology

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APA (6th Edition):

Qiao, S. (2013). Pharmacological Modulation of Oxidative and Proteotoxic Stress for Antimelanoma Intervention . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/311348

Chicago Manual of Style (16th Edition):

Qiao, Shuxi. “Pharmacological Modulation of Oxidative and Proteotoxic Stress for Antimelanoma Intervention .” 2013. Doctoral Dissertation, University of Arizona. Accessed February 21, 2020. http://hdl.handle.net/10150/311348.

MLA Handbook (7th Edition):

Qiao, Shuxi. “Pharmacological Modulation of Oxidative and Proteotoxic Stress for Antimelanoma Intervention .” 2013. Web. 21 Feb 2020.

Vancouver:

Qiao S. Pharmacological Modulation of Oxidative and Proteotoxic Stress for Antimelanoma Intervention . [Internet] [Doctoral dissertation]. University of Arizona; 2013. [cited 2020 Feb 21]. Available from: http://hdl.handle.net/10150/311348.

Council of Science Editors:

Qiao S. Pharmacological Modulation of Oxidative and Proteotoxic Stress for Antimelanoma Intervention . [Doctoral Dissertation]. University of Arizona; 2013. Available from: http://hdl.handle.net/10150/311348


University of Arizona

11. Lau, Alexandria G. The Role of the Nrf2-Keap1 Pathway in Autophagy and How it Contributes to Arsenic Carcinogenicity .

Degree: 2012, University of Arizona

 NF-E2-related factor 2 (Nrf2) is a transcription factor that is responsible for maintaining cellular homeostasis by controlling the fate of cells through transcriptional upregulation of… (more)

Subjects/Keywords: autophagy; Keap1; Nrf2; p62; Pharmacology & Toxicology; Antioxidant response; arsenic

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APA (6th Edition):

Lau, A. G. (2012). The Role of the Nrf2-Keap1 Pathway in Autophagy and How it Contributes to Arsenic Carcinogenicity . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/228515

Chicago Manual of Style (16th Edition):

Lau, Alexandria G. “The Role of the Nrf2-Keap1 Pathway in Autophagy and How it Contributes to Arsenic Carcinogenicity .” 2012. Doctoral Dissertation, University of Arizona. Accessed February 21, 2020. http://hdl.handle.net/10150/228515.

MLA Handbook (7th Edition):

Lau, Alexandria G. “The Role of the Nrf2-Keap1 Pathway in Autophagy and How it Contributes to Arsenic Carcinogenicity .” 2012. Web. 21 Feb 2020.

Vancouver:

Lau AG. The Role of the Nrf2-Keap1 Pathway in Autophagy and How it Contributes to Arsenic Carcinogenicity . [Internet] [Doctoral dissertation]. University of Arizona; 2012. [cited 2020 Feb 21]. Available from: http://hdl.handle.net/10150/228515.

Council of Science Editors:

Lau AG. The Role of the Nrf2-Keap1 Pathway in Autophagy and How it Contributes to Arsenic Carcinogenicity . [Doctoral Dissertation]. University of Arizona; 2012. Available from: http://hdl.handle.net/10150/228515

12. Aguilar, David Christopher. GILZ: A Novel Glucocorticoid Induced Cytoprotective Protein in Cardiomyocytes .

Degree: 2012, University of Arizona

 Glucocorticoids (GCs) are frequently prescribed pharmacological agents most notably for their immunosuppressant effects. Endogenous GCs mediate biological processes such as energy metabolism and tissue development.… (more)

Subjects/Keywords: doxorubicin; glucocorticoid induced leucine zipper; H9C2 rat cardiomyocytes; primary neonatal cardiomyocytes; Medical Pharmacology; corticosterone; dexamethasone

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APA (6th Edition):

Aguilar, D. C. (2012). GILZ: A Novel Glucocorticoid Induced Cytoprotective Protein in Cardiomyocytes . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/228177

Chicago Manual of Style (16th Edition):

Aguilar, David Christopher. “GILZ: A Novel Glucocorticoid Induced Cytoprotective Protein in Cardiomyocytes .” 2012. Doctoral Dissertation, University of Arizona. Accessed February 21, 2020. http://hdl.handle.net/10150/228177.

MLA Handbook (7th Edition):

Aguilar, David Christopher. “GILZ: A Novel Glucocorticoid Induced Cytoprotective Protein in Cardiomyocytes .” 2012. Web. 21 Feb 2020.

Vancouver:

Aguilar DC. GILZ: A Novel Glucocorticoid Induced Cytoprotective Protein in Cardiomyocytes . [Internet] [Doctoral dissertation]. University of Arizona; 2012. [cited 2020 Feb 21]. Available from: http://hdl.handle.net/10150/228177.

Council of Science Editors:

Aguilar DC. GILZ: A Novel Glucocorticoid Induced Cytoprotective Protein in Cardiomyocytes . [Doctoral Dissertation]. University of Arizona; 2012. Available from: http://hdl.handle.net/10150/228177


University of Arizona

13. Tu, Chunyi. The cellular and molecular mechanisms of oxidant-induced cardiomyocyte hypertrophy .

Degree: 2002, University of Arizona

 Cardiac hypertrophy is a common consequence of many cardiovascular diseases. It is often a transition to heart failure. Although oxidants have been indicated to mediate… (more)

Subjects/Keywords: Biology, Cell.; Health Sciences, Toxicology.; Health Sciences, Pharmacology.

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APA (6th Edition):

Tu, C. (2002). The cellular and molecular mechanisms of oxidant-induced cardiomyocyte hypertrophy . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/280216

Chicago Manual of Style (16th Edition):

Tu, Chunyi. “The cellular and molecular mechanisms of oxidant-induced cardiomyocyte hypertrophy .” 2002. Doctoral Dissertation, University of Arizona. Accessed February 21, 2020. http://hdl.handle.net/10150/280216.

MLA Handbook (7th Edition):

Tu, Chunyi. “The cellular and molecular mechanisms of oxidant-induced cardiomyocyte hypertrophy .” 2002. Web. 21 Feb 2020.

Vancouver:

Tu C. The cellular and molecular mechanisms of oxidant-induced cardiomyocyte hypertrophy . [Internet] [Doctoral dissertation]. University of Arizona; 2002. [cited 2020 Feb 21]. Available from: http://hdl.handle.net/10150/280216.

Council of Science Editors:

Tu C. The cellular and molecular mechanisms of oxidant-induced cardiomyocyte hypertrophy . [Doctoral Dissertation]. University of Arizona; 2002. Available from: http://hdl.handle.net/10150/280216

14. Wu, Tongde. Post-Transcriptional Regulation of Nrf2: Novel Mechanisms beyond Keap1 .

Degree: 2013, University of Arizona

 Nrf2 (NF-E2-related factor 2) is a transcription factor that regulates a battery of downstream genes that contain the antioxidant response element (ARE) in their promoter… (more)

Subjects/Keywords: Pharmacology & Toxicology

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APA (6th Edition):

Wu, T. (2013). Post-Transcriptional Regulation of Nrf2: Novel Mechanisms beyond Keap1 . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/301750

Chicago Manual of Style (16th Edition):

Wu, Tongde. “Post-Transcriptional Regulation of Nrf2: Novel Mechanisms beyond Keap1 .” 2013. Doctoral Dissertation, University of Arizona. Accessed February 21, 2020. http://hdl.handle.net/10150/301750.

MLA Handbook (7th Edition):

Wu, Tongde. “Post-Transcriptional Regulation of Nrf2: Novel Mechanisms beyond Keap1 .” 2013. Web. 21 Feb 2020.

Vancouver:

Wu T. Post-Transcriptional Regulation of Nrf2: Novel Mechanisms beyond Keap1 . [Internet] [Doctoral dissertation]. University of Arizona; 2013. [cited 2020 Feb 21]. Available from: http://hdl.handle.net/10150/301750.

Council of Science Editors:

Wu T. Post-Transcriptional Regulation of Nrf2: Novel Mechanisms beyond Keap1 . [Doctoral Dissertation]. University of Arizona; 2013. Available from: http://hdl.handle.net/10150/301750


University of Arizona

15. Dedek, Matthew Milan. The Melanocortin System: Structure Activity Relationships of Alpha-N-Methylated MT-II Analogues and Mutation Studies of Human Melanocortin Receptor Subtypes 1 and 4 .

Degree: 2007, University of Arizona

 The melanocortin system regulates various physiological processes including feeding behavior, sexual function, skin pigmentation and photoprotection via five G-protein coupled receptors and several endogenous ligands.… (more)

Subjects/Keywords: structure activity relationship; melanocortin 1 receptor; melanocortin; MT-II; mutagenesis; melanocortin 4 receptor

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APA (6th Edition):

Dedek, M. M. (2007). The Melanocortin System: Structure Activity Relationships of Alpha-N-Methylated MT-II Analogues and Mutation Studies of Human Melanocortin Receptor Subtypes 1 and 4 . (Masters Thesis). University of Arizona. Retrieved from http://hdl.handle.net/10150/193306

Chicago Manual of Style (16th Edition):

Dedek, Matthew Milan. “The Melanocortin System: Structure Activity Relationships of Alpha-N-Methylated MT-II Analogues and Mutation Studies of Human Melanocortin Receptor Subtypes 1 and 4 .” 2007. Masters Thesis, University of Arizona. Accessed February 21, 2020. http://hdl.handle.net/10150/193306.

MLA Handbook (7th Edition):

Dedek, Matthew Milan. “The Melanocortin System: Structure Activity Relationships of Alpha-N-Methylated MT-II Analogues and Mutation Studies of Human Melanocortin Receptor Subtypes 1 and 4 .” 2007. Web. 21 Feb 2020.

Vancouver:

Dedek MM. The Melanocortin System: Structure Activity Relationships of Alpha-N-Methylated MT-II Analogues and Mutation Studies of Human Melanocortin Receptor Subtypes 1 and 4 . [Internet] [Masters thesis]. University of Arizona; 2007. [cited 2020 Feb 21]. Available from: http://hdl.handle.net/10150/193306.

Council of Science Editors:

Dedek MM. The Melanocortin System: Structure Activity Relationships of Alpha-N-Methylated MT-II Analogues and Mutation Studies of Human Melanocortin Receptor Subtypes 1 and 4 . [Masters Thesis]. University of Arizona; 2007. Available from: http://hdl.handle.net/10150/193306


University of Arizona

16. Manza, Linda Lee. Characterization of Protein Sumoylation in Response to Alkylation Stress in HEK 293 Cells .

Degree: 2007, University of Arizona

 Stress conditions such as heat shock, UV, alkylating agents, and H2O2 have been shown to result in the modification of a variety of protein targets… (more)

Subjects/Keywords: Sumoylation; alkylation; hydroxynonenal; LC-MS-MS analysis

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APA (6th Edition):

Manza, L. L. (2007). Characterization of Protein Sumoylation in Response to Alkylation Stress in HEK 293 Cells . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/193944

Chicago Manual of Style (16th Edition):

Manza, Linda Lee. “Characterization of Protein Sumoylation in Response to Alkylation Stress in HEK 293 Cells .” 2007. Doctoral Dissertation, University of Arizona. Accessed February 21, 2020. http://hdl.handle.net/10150/193944.

MLA Handbook (7th Edition):

Manza, Linda Lee. “Characterization of Protein Sumoylation in Response to Alkylation Stress in HEK 293 Cells .” 2007. Web. 21 Feb 2020.

Vancouver:

Manza LL. Characterization of Protein Sumoylation in Response to Alkylation Stress in HEK 293 Cells . [Internet] [Doctoral dissertation]. University of Arizona; 2007. [cited 2020 Feb 21]. Available from: http://hdl.handle.net/10150/193944.

Council of Science Editors:

Manza LL. Characterization of Protein Sumoylation in Response to Alkylation Stress in HEK 293 Cells . [Doctoral Dissertation]. University of Arizona; 2007. Available from: http://hdl.handle.net/10150/193944


University of Arizona

17. Purdom-Dickinson, Sally Elizabeth. Early Responses to Oxidative Stress In Heart Cells: Signals From The Cell Membrane To The Nucleus and Beyond .

Degree: 2005, University of Arizona

 Oxidative stress is known to contribute to many forms of heart disease. Oxidants such as H₂O₂ can cause hypertrophy of cardiomyocytes (CMCs). Heart fibroblasts (HFs)… (more)

Subjects/Keywords: oxidant; Nrf2; cardiomyocyte; fibroblast; cytoprotection; PI3Kinase

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APA (6th Edition):

Purdom-Dickinson, S. E. (2005). Early Responses to Oxidative Stress In Heart Cells: Signals From The Cell Membrane To The Nucleus and Beyond . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/194381

Chicago Manual of Style (16th Edition):

Purdom-Dickinson, Sally Elizabeth. “Early Responses to Oxidative Stress In Heart Cells: Signals From The Cell Membrane To The Nucleus and Beyond .” 2005. Doctoral Dissertation, University of Arizona. Accessed February 21, 2020. http://hdl.handle.net/10150/194381.

MLA Handbook (7th Edition):

Purdom-Dickinson, Sally Elizabeth. “Early Responses to Oxidative Stress In Heart Cells: Signals From The Cell Membrane To The Nucleus and Beyond .” 2005. Web. 21 Feb 2020.

Vancouver:

Purdom-Dickinson SE. Early Responses to Oxidative Stress In Heart Cells: Signals From The Cell Membrane To The Nucleus and Beyond . [Internet] [Doctoral dissertation]. University of Arizona; 2005. [cited 2020 Feb 21]. Available from: http://hdl.handle.net/10150/194381.

Council of Science Editors:

Purdom-Dickinson SE. Early Responses to Oxidative Stress In Heart Cells: Signals From The Cell Membrane To The Nucleus and Beyond . [Doctoral Dissertation]. University of Arizona; 2005. Available from: http://hdl.handle.net/10150/194381


University of Arizona

18. Rajapaksa, Kathila Seuwandhi. The Role of Ovarian Metabolism in 4-Vinylcyclohexene Metabolites and 7,12-Dimethylbenz[a]anthracene Induced Ovotoxicity in Mice .

Degree: 2007, University of Arizona

 Ovarian toxicants 4-vinylcychlohexene (VCH) and 7,12-dimethylbenz[a]anthracene (DMBA) requires bioactivation to induce follicle loss. VCH is bioactivated to monoepoxides (1,2-VCM and 7,8-VCM), and subsequently to an… (more)

Subjects/Keywords: VCM; VCD; DMBA; CYP 2E1; GSH; mEH

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APA (6th Edition):

Rajapaksa, K. S. (2007). The Role of Ovarian Metabolism in 4-Vinylcyclohexene Metabolites and 7,12-Dimethylbenz[a]anthracene Induced Ovotoxicity in Mice . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/194403

Chicago Manual of Style (16th Edition):

Rajapaksa, Kathila Seuwandhi. “The Role of Ovarian Metabolism in 4-Vinylcyclohexene Metabolites and 7,12-Dimethylbenz[a]anthracene Induced Ovotoxicity in Mice .” 2007. Doctoral Dissertation, University of Arizona. Accessed February 21, 2020. http://hdl.handle.net/10150/194403.

MLA Handbook (7th Edition):

Rajapaksa, Kathila Seuwandhi. “The Role of Ovarian Metabolism in 4-Vinylcyclohexene Metabolites and 7,12-Dimethylbenz[a]anthracene Induced Ovotoxicity in Mice .” 2007. Web. 21 Feb 2020.

Vancouver:

Rajapaksa KS. The Role of Ovarian Metabolism in 4-Vinylcyclohexene Metabolites and 7,12-Dimethylbenz[a]anthracene Induced Ovotoxicity in Mice . [Internet] [Doctoral dissertation]. University of Arizona; 2007. [cited 2020 Feb 21]. Available from: http://hdl.handle.net/10150/194403.

Council of Science Editors:

Rajapaksa KS. The Role of Ovarian Metabolism in 4-Vinylcyclohexene Metabolites and 7,12-Dimethylbenz[a]anthracene Induced Ovotoxicity in Mice . [Doctoral Dissertation]. University of Arizona; 2007. Available from: http://hdl.handle.net/10150/194403


University of Arizona

19. Sun, Haipeng. Regulation of Cyclooxygenase Gene Expression by Glucocorticoids in Cardiomyocytes .

Degree: 2007, University of Arizona

 Glucocorticoids (GCs) are endogenous steroid hormones that regulate a number of critical physiological processes. Psychological stress increases the level of GCs in the circulating system.… (more)

Subjects/Keywords: glucocorticoid; COX-1; COX-2; cardiomyocytes; gene; regulation

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APA (6th Edition):

Sun, H. (2007). Regulation of Cyclooxygenase Gene Expression by Glucocorticoids in Cardiomyocytes . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/194896

Chicago Manual of Style (16th Edition):

Sun, Haipeng. “Regulation of Cyclooxygenase Gene Expression by Glucocorticoids in Cardiomyocytes .” 2007. Doctoral Dissertation, University of Arizona. Accessed February 21, 2020. http://hdl.handle.net/10150/194896.

MLA Handbook (7th Edition):

Sun, Haipeng. “Regulation of Cyclooxygenase Gene Expression by Glucocorticoids in Cardiomyocytes .” 2007. Web. 21 Feb 2020.

Vancouver:

Sun H. Regulation of Cyclooxygenase Gene Expression by Glucocorticoids in Cardiomyocytes . [Internet] [Doctoral dissertation]. University of Arizona; 2007. [cited 2020 Feb 21]. Available from: http://hdl.handle.net/10150/194896.

Council of Science Editors:

Sun H. Regulation of Cyclooxygenase Gene Expression by Glucocorticoids in Cardiomyocytes . [Doctoral Dissertation]. University of Arizona; 2007. Available from: http://hdl.handle.net/10150/194896


University of Arizona

20. Bredfeldt, Tiffany Gail. Low-Level Arsenic Toxicity in Human Bladder Cells .

Degree: 2006, University of Arizona

 Arsenic is a human bladder carcinogen. Inorganic arsenic and methylated metabolites are excreted from the human body in urine. This study investigates the effects of… (more)

Subjects/Keywords: arsenic; arsenic methylation; carcinogenesis; bladder cancer

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APA (6th Edition):

Bredfeldt, T. G. (2006). Low-Level Arsenic Toxicity in Human Bladder Cells . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/195157

Chicago Manual of Style (16th Edition):

Bredfeldt, Tiffany Gail. “Low-Level Arsenic Toxicity in Human Bladder Cells .” 2006. Doctoral Dissertation, University of Arizona. Accessed February 21, 2020. http://hdl.handle.net/10150/195157.

MLA Handbook (7th Edition):

Bredfeldt, Tiffany Gail. “Low-Level Arsenic Toxicity in Human Bladder Cells .” 2006. Web. 21 Feb 2020.

Vancouver:

Bredfeldt TG. Low-Level Arsenic Toxicity in Human Bladder Cells . [Internet] [Doctoral dissertation]. University of Arizona; 2006. [cited 2020 Feb 21]. Available from: http://hdl.handle.net/10150/195157.

Council of Science Editors:

Bredfeldt TG. Low-Level Arsenic Toxicity in Human Bladder Cells . [Doctoral Dissertation]. University of Arizona; 2006. Available from: http://hdl.handle.net/10150/195157


University of Arizona

21. Xie, Lifang. Proteomic and Microarray Identification of Novel Cardiac Specific Indicators of Oxidative Injury and Mechanism of Action .

Degree: 2007, University of Arizona

 Cardiovascular disease (CVD) is the leading cause of death in the United States. Oxidative stress plays an important role in the pathogenesis of CVD. Heart… (more)

Subjects/Keywords: Heart failure; Biomarker; Proteomics; Microarray

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APA (6th Edition):

Xie, L. (2007). Proteomic and Microarray Identification of Novel Cardiac Specific Indicators of Oxidative Injury and Mechanism of Action . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/195214

Chicago Manual of Style (16th Edition):

Xie, Lifang. “Proteomic and Microarray Identification of Novel Cardiac Specific Indicators of Oxidative Injury and Mechanism of Action .” 2007. Doctoral Dissertation, University of Arizona. Accessed February 21, 2020. http://hdl.handle.net/10150/195214.

MLA Handbook (7th Edition):

Xie, Lifang. “Proteomic and Microarray Identification of Novel Cardiac Specific Indicators of Oxidative Injury and Mechanism of Action .” 2007. Web. 21 Feb 2020.

Vancouver:

Xie L. Proteomic and Microarray Identification of Novel Cardiac Specific Indicators of Oxidative Injury and Mechanism of Action . [Internet] [Doctoral dissertation]. University of Arizona; 2007. [cited 2020 Feb 21]. Available from: http://hdl.handle.net/10150/195214.

Council of Science Editors:

Xie L. Proteomic and Microarray Identification of Novel Cardiac Specific Indicators of Oxidative Injury and Mechanism of Action . [Doctoral Dissertation]. University of Arizona; 2007. Available from: http://hdl.handle.net/10150/195214


University of Arizona

22. Choi, Ji-Eun. The Role of Adiponectin in Ischemia-Reperfusion Injury in the Type 2 Diabetic Heart .

Degree: 2008, University of Arizona

 Cardiovascular disease (CVD) is the leading cause of death in the United States, and the risk and severity of CVD are increased with type 2… (more)

Subjects/Keywords: Physiological Sciences

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APA (6th Edition):

Choi, J. (2008). The Role of Adiponectin in Ischemia-Reperfusion Injury in the Type 2 Diabetic Heart . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/195495

Chicago Manual of Style (16th Edition):

Choi, Ji-Eun. “The Role of Adiponectin in Ischemia-Reperfusion Injury in the Type 2 Diabetic Heart .” 2008. Doctoral Dissertation, University of Arizona. Accessed February 21, 2020. http://hdl.handle.net/10150/195495.

MLA Handbook (7th Edition):

Choi, Ji-Eun. “The Role of Adiponectin in Ischemia-Reperfusion Injury in the Type 2 Diabetic Heart .” 2008. Web. 21 Feb 2020.

Vancouver:

Choi J. The Role of Adiponectin in Ischemia-Reperfusion Injury in the Type 2 Diabetic Heart . [Internet] [Doctoral dissertation]. University of Arizona; 2008. [cited 2020 Feb 21]. Available from: http://hdl.handle.net/10150/195495.

Council of Science Editors:

Choi J. The Role of Adiponectin in Ischemia-Reperfusion Injury in the Type 2 Diabetic Heart . [Doctoral Dissertation]. University of Arizona; 2008. Available from: http://hdl.handle.net/10150/195495


University of Arizona

23. Mayelzadeh, Foroozan. DNA Binding and Selective Gene Induction by Different Forms of the P53 Protein .

Degree: 2006, University of Arizona

 When cells are challenged by genotoxic stress, the tumor suppressor protein p53 promotes adaptive responses, including cell cycle arrest, DNA repair, or apoptosis. How these… (more)

Subjects/Keywords: Genetics

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APA (6th Edition):

Mayelzadeh, F. (2006). DNA Binding and Selective Gene Induction by Different Forms of the P53 Protein . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/193992

Chicago Manual of Style (16th Edition):

Mayelzadeh, Foroozan. “DNA Binding and Selective Gene Induction by Different Forms of the P53 Protein .” 2006. Doctoral Dissertation, University of Arizona. Accessed February 21, 2020. http://hdl.handle.net/10150/193992.

MLA Handbook (7th Edition):

Mayelzadeh, Foroozan. “DNA Binding and Selective Gene Induction by Different Forms of the P53 Protein .” 2006. Web. 21 Feb 2020.

Vancouver:

Mayelzadeh F. DNA Binding and Selective Gene Induction by Different Forms of the P53 Protein . [Internet] [Doctoral dissertation]. University of Arizona; 2006. [cited 2020 Feb 21]. Available from: http://hdl.handle.net/10150/193992.

Council of Science Editors:

Mayelzadeh F. DNA Binding and Selective Gene Induction by Different Forms of the P53 Protein . [Doctoral Dissertation]. University of Arizona; 2006. Available from: http://hdl.handle.net/10150/193992


University of Arizona

24. Morrissy, Stephen J. Regulation Of Anti-Oxidant and Anti-Apoptotic Genes By Progesterone in Cardiomyocytes .

Degree: 2007, University of Arizona

 The anthracycline quinone, doxorubicin (Adriamycin) is an antineoplastic agent that has substantial therapeutic activity against a broad variety of human cancers. Unfortunately, the use of… (more)

Subjects/Keywords: Progesterone; Bcl-XL; NQO1; Apoptosis; Anti-Oxidant

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APA (6th Edition):

Morrissy, S. J. (2007). Regulation Of Anti-Oxidant and Anti-Apoptotic Genes By Progesterone in Cardiomyocytes . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/194129

Chicago Manual of Style (16th Edition):

Morrissy, Stephen J. “Regulation Of Anti-Oxidant and Anti-Apoptotic Genes By Progesterone in Cardiomyocytes .” 2007. Doctoral Dissertation, University of Arizona. Accessed February 21, 2020. http://hdl.handle.net/10150/194129.

MLA Handbook (7th Edition):

Morrissy, Stephen J. “Regulation Of Anti-Oxidant and Anti-Apoptotic Genes By Progesterone in Cardiomyocytes .” 2007. Web. 21 Feb 2020.

Vancouver:

Morrissy SJ. Regulation Of Anti-Oxidant and Anti-Apoptotic Genes By Progesterone in Cardiomyocytes . [Internet] [Doctoral dissertation]. University of Arizona; 2007. [cited 2020 Feb 21]. Available from: http://hdl.handle.net/10150/194129.

Council of Science Editors:

Morrissy SJ. Regulation Of Anti-Oxidant and Anti-Apoptotic Genes By Progesterone in Cardiomyocytes . [Doctoral Dissertation]. University of Arizona; 2007. Available from: http://hdl.handle.net/10150/194129


University of Arizona

25. Xie, Ruiyu. Reactive Oxygen Species-Induced Necrotic Cell Death .

Degree: 2009, University of Arizona

 Mechanisms of cell death extend beyond the simple apoptosis/necrosis relationship to include regulated modes of cell death that do not readily fit either of the… (more)

Subjects/Keywords: calcium; cell death; mechanism; necrosis; PARP; reactive oxygen species

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APA (6th Edition):

Xie, R. (2009). Reactive Oxygen Species-Induced Necrotic Cell Death . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/195215

Chicago Manual of Style (16th Edition):

Xie, Ruiyu. “Reactive Oxygen Species-Induced Necrotic Cell Death .” 2009. Doctoral Dissertation, University of Arizona. Accessed February 21, 2020. http://hdl.handle.net/10150/195215.

MLA Handbook (7th Edition):

Xie, Ruiyu. “Reactive Oxygen Species-Induced Necrotic Cell Death .” 2009. Web. 21 Feb 2020.

Vancouver:

Xie R. Reactive Oxygen Species-Induced Necrotic Cell Death . [Internet] [Doctoral dissertation]. University of Arizona; 2009. [cited 2020 Feb 21]. Available from: http://hdl.handle.net/10150/195215.

Council of Science Editors:

Xie R. Reactive Oxygen Species-Induced Necrotic Cell Death . [Doctoral Dissertation]. University of Arizona; 2009. Available from: http://hdl.handle.net/10150/195215


University of Arizona

26. Catania, Jeffrey Mark. Derivations of Tissue Slice Technology as Toxicological Screening Systems .

Degree: 2006, University of Arizona

 In vitro toxicology studies are hindered by the use of specific cellular systems which solely examine one cell type. Precision-cut tissue slices mimic specific organ… (more)

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APA (6th Edition):

Catania, J. M. (2006). Derivations of Tissue Slice Technology as Toxicological Screening Systems . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/195419

Chicago Manual of Style (16th Edition):

Catania, Jeffrey Mark. “Derivations of Tissue Slice Technology as Toxicological Screening Systems .” 2006. Doctoral Dissertation, University of Arizona. Accessed February 21, 2020. http://hdl.handle.net/10150/195419.

MLA Handbook (7th Edition):

Catania, Jeffrey Mark. “Derivations of Tissue Slice Technology as Toxicological Screening Systems .” 2006. Web. 21 Feb 2020.

Vancouver:

Catania JM. Derivations of Tissue Slice Technology as Toxicological Screening Systems . [Internet] [Doctoral dissertation]. University of Arizona; 2006. [cited 2020 Feb 21]. Available from: http://hdl.handle.net/10150/195419.

Council of Science Editors:

Catania JM. Derivations of Tissue Slice Technology as Toxicological Screening Systems . [Doctoral Dissertation]. University of Arizona; 2006. Available from: http://hdl.handle.net/10150/195419


University of Arizona

27. Labenski, Matthew Thomas. Identification and Characterization of Quinone-Thioether Protein Adducts In Vivo .

Degree: 2008, University of Arizona

 Quinones represent an important class of endogenous compounds such as neurotransmitters and coenzyme Q10, electrophilic xenobiotics and environmental toxicants that have known reactivity based on… (more)

Subjects/Keywords: nephrotoxicity; protein adduction; Quinone-thioether; reactive oxygen species

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APA (6th Edition):

Labenski, M. T. (2008). Identification and Characterization of Quinone-Thioether Protein Adducts In Vivo . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/193748

Chicago Manual of Style (16th Edition):

Labenski, Matthew Thomas. “Identification and Characterization of Quinone-Thioether Protein Adducts In Vivo .” 2008. Doctoral Dissertation, University of Arizona. Accessed February 21, 2020. http://hdl.handle.net/10150/193748.

MLA Handbook (7th Edition):

Labenski, Matthew Thomas. “Identification and Characterization of Quinone-Thioether Protein Adducts In Vivo .” 2008. Web. 21 Feb 2020.

Vancouver:

Labenski MT. Identification and Characterization of Quinone-Thioether Protein Adducts In Vivo . [Internet] [Doctoral dissertation]. University of Arizona; 2008. [cited 2020 Feb 21]. Available from: http://hdl.handle.net/10150/193748.

Council of Science Editors:

Labenski MT. Identification and Characterization of Quinone-Thioether Protein Adducts In Vivo . [Doctoral Dissertation]. University of Arizona; 2008. Available from: http://hdl.handle.net/10150/193748

.