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You searched for +publisher:"University of Alabama – Birmingham" +contributor:("Yoder, Bradley K."). Showing records 1 – 19 of 19 total matches.

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1. Nie, Shuyi. Regulation of vertebrate gastrulation by ErbB signaling.

Degree: PhD, 2007, University of Alabama – Birmingham

ErbB receptor tyrosine kinases have long been implicated in cancer formation and progression by regulating cell division, migration, and survival. ErbBs are also essential in… (more)

Subjects/Keywords: Cell Movement  – physiology <; br>; Gastrula  – physiology <; br>; Morphogenesis  – physiology <; br>; Receptor, Epidermal Growth Factor  – metabolism <; br>; Signal Transduction  – physiology <; br>; Xenopus  – embryology

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APA (6th Edition):

Nie, S. (2007). Regulation of vertebrate gastrulation by ErbB signaling. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,93

Chicago Manual of Style (16th Edition):

Nie, Shuyi. “Regulation of vertebrate gastrulation by ErbB signaling.” 2007. Doctoral Dissertation, University of Alabama – Birmingham. Accessed April 01, 2020. http://contentdm.mhsl.uab.edu/u?/etd,93.

MLA Handbook (7th Edition):

Nie, Shuyi. “Regulation of vertebrate gastrulation by ErbB signaling.” 2007. Web. 01 Apr 2020.

Vancouver:

Nie S. Regulation of vertebrate gastrulation by ErbB signaling. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2007. [cited 2020 Apr 01]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,93.

Council of Science Editors:

Nie S. Regulation of vertebrate gastrulation by ErbB signaling. [Doctoral Dissertation]. University of Alabama – Birmingham; 2007. Available from: http://contentdm.mhsl.uab.edu/u?/etd,93

2. Hock, Thomas D. Regulation of the human heme oxygenase-1 gene.

Degree: PhD, 2007, University of Alabama – Birmingham

The heme oxygenase-1 (HO-1) gene encodes a microsomal enzyme that catalyzes the conversion of heme to carbon monoxide, Iron, and biliverdin. HO-1 transcription is induced… (more)

Subjects/Keywords: Heme Oxygenase-1  – genetics <; br>; Proto-Oncogene Proteins c-jun  – metabolism <; br>; Transcription Factors  – metabolism

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APA (6th Edition):

Hock, T. D. (2007). Regulation of the human heme oxygenase-1 gene. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,126

Chicago Manual of Style (16th Edition):

Hock, Thomas D. “Regulation of the human heme oxygenase-1 gene.” 2007. Doctoral Dissertation, University of Alabama – Birmingham. Accessed April 01, 2020. http://contentdm.mhsl.uab.edu/u?/etd,126.

MLA Handbook (7th Edition):

Hock, Thomas D. “Regulation of the human heme oxygenase-1 gene.” 2007. Web. 01 Apr 2020.

Vancouver:

Hock TD. Regulation of the human heme oxygenase-1 gene. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2007. [cited 2020 Apr 01]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,126.

Council of Science Editors:

Hock TD. Regulation of the human heme oxygenase-1 gene. [Doctoral Dissertation]. University of Alabama – Birmingham; 2007. Available from: http://contentdm.mhsl.uab.edu/u?/etd,126

3. Liu, Jia. Increased hexosamine biosynthesis and protein O-GLCNAC protect isolated rat heart from ischemia/reperfusion injury.

Degree: PhD, 2006, University of Alabama – Birmingham

Increased levels of protein-associated O-linked N-acetylglucosamine (O-GlcNAc) have been correlated with increased tolerance to stress. Therefore the goal of this study was to determine whether… (more)

Subjects/Keywords: Acetylglucosamine  – metabolism <; br>; Hexosamines  – biosynthesis <; br>; Reperfusion Injury  – prevention & control

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APA (6th Edition):

Liu, J. (2006). Increased hexosamine biosynthesis and protein O-GLCNAC protect isolated rat heart from ischemia/reperfusion injury. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,307

Chicago Manual of Style (16th Edition):

Liu, Jia. “Increased hexosamine biosynthesis and protein O-GLCNAC protect isolated rat heart from ischemia/reperfusion injury.” 2006. Doctoral Dissertation, University of Alabama – Birmingham. Accessed April 01, 2020. http://contentdm.mhsl.uab.edu/u?/etd,307.

MLA Handbook (7th Edition):

Liu, Jia. “Increased hexosamine biosynthesis and protein O-GLCNAC protect isolated rat heart from ischemia/reperfusion injury.” 2006. Web. 01 Apr 2020.

Vancouver:

Liu J. Increased hexosamine biosynthesis and protein O-GLCNAC protect isolated rat heart from ischemia/reperfusion injury. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2006. [cited 2020 Apr 01]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,307.

Council of Science Editors:

Liu J. Increased hexosamine biosynthesis and protein O-GLCNAC protect isolated rat heart from ischemia/reperfusion injury. [Doctoral Dissertation]. University of Alabama – Birmingham; 2006. Available from: http://contentdm.mhsl.uab.edu/u?/etd,307

4. Tower-Gilchrist, Cristy (Cristy Davette). The role of protein folding and protein trafficking in human disease.

Degree: PhD, 2011, University of Alabama – Birmingham

Project 1: Expansion of CAG repeats encoding glutamine in huntingtin and ataxin 3 causes the neurodegenerative diseases Huntington's disease (HD) and spinocerebellar ataxia 3 (SCA3),… (more)

Subjects/Keywords: Cilia<; br>; Cytomegalovirus  – enzymology<; br>; Huntington Disease  – virology<; br>; Neurons  – metabolism<; br>; Peptides  – antagonists & inhibitors<; br>; Phosphotransferases (Alcohol Group Acceptor)  – physiology<; br>; Spinocerebellar Ataxias  – virology

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APA (6th Edition):

Tower-Gilchrist, C. (. D. (2011). The role of protein folding and protein trafficking in human disease. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,964

Chicago Manual of Style (16th Edition):

Tower-Gilchrist, Cristy (Cristy Davette). “The role of protein folding and protein trafficking in human disease.” 2011. Doctoral Dissertation, University of Alabama – Birmingham. Accessed April 01, 2020. http://contentdm.mhsl.uab.edu/u?/etd,964.

MLA Handbook (7th Edition):

Tower-Gilchrist, Cristy (Cristy Davette). “The role of protein folding and protein trafficking in human disease.” 2011. Web. 01 Apr 2020.

Vancouver:

Tower-Gilchrist C(D. The role of protein folding and protein trafficking in human disease. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2011. [cited 2020 Apr 01]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,964.

Council of Science Editors:

Tower-Gilchrist C(D. The role of protein folding and protein trafficking in human disease. [Doctoral Dissertation]. University of Alabama – Birmingham; 2011. Available from: http://contentdm.mhsl.uab.edu/u?/etd,964

5. Bolisetty, Subhashini. Role Of Heme Oxygenase-1 In Acute Kidney Injury.

Degree: PhD, 2010, University of Alabama – Birmingham

Acute kidney injury (AKI), defined as the rapid loss of kidney function, is often seen in the setting of multiple organ failure in critically ill… (more)

Subjects/Keywords: Acute Kidney Injury – enzymology.<; br>; Autophagy.<; br>; Cisplatin – adverse effects.<; br>; Heme Oxygenase-1 – metabolism.<; br>; Kidney Tubules, Proximal – enzymology.<; br>; Mitochondria – physiology<; br>; Reactive Oxygen Species – metabolism

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APA (6th Edition):

Bolisetty, S. (2010). Role Of Heme Oxygenase-1 In Acute Kidney Injury. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1402

Chicago Manual of Style (16th Edition):

Bolisetty, Subhashini. “Role Of Heme Oxygenase-1 In Acute Kidney Injury.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed April 01, 2020. http://contentdm.mhsl.uab.edu/u?/etd,1402.

MLA Handbook (7th Edition):

Bolisetty, Subhashini. “Role Of Heme Oxygenase-1 In Acute Kidney Injury.” 2010. Web. 01 Apr 2020.

Vancouver:

Bolisetty S. Role Of Heme Oxygenase-1 In Acute Kidney Injury. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2020 Apr 01]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1402.

Council of Science Editors:

Bolisetty S. Role Of Heme Oxygenase-1 In Acute Kidney Injury. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1402

6. Chang, Ching-Fang. The Role Of Primary Cilia In Articular Cartilage And Growth Plate.

Degree: 2012, University of Alabama – Birmingham

Primary cilia are now considered an important organelle required for many key steps during developmental processes including skeletal development. Articular cartilage and growth plate are… (more)

Subjects/Keywords: Arthritis, Experimental – pathology.<; br>; Cartilage, Articular – ultrastructure.<; br>; Chondrocytes – ultrastructure.<; br>; Cilia – pathology<; br>; Osteoarthritis – pathology

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APA (6th Edition):

Chang, C. (2012). The Role Of Primary Cilia In Articular Cartilage And Growth Plate. (Thesis). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1486

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Chang, Ching-Fang. “The Role Of Primary Cilia In Articular Cartilage And Growth Plate.” 2012. Thesis, University of Alabama – Birmingham. Accessed April 01, 2020. http://contentdm.mhsl.uab.edu/u?/etd,1486.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Chang, Ching-Fang. “The Role Of Primary Cilia In Articular Cartilage And Growth Plate.” 2012. Web. 01 Apr 2020.

Vancouver:

Chang C. The Role Of Primary Cilia In Articular Cartilage And Growth Plate. [Internet] [Thesis]. University of Alabama – Birmingham; 2012. [cited 2020 Apr 01]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1486.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Chang C. The Role Of Primary Cilia In Articular Cartilage And Growth Plate. [Thesis]. University of Alabama – Birmingham; 2012. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1486

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

7. Boddu, Ravindra. Characterizing The Transcriptional Complexity Of Pkhd1/pkhd1.

Degree: 2012, University of Alabama – Birmingham

Mutations in <italic>PKHD1</italic> gene cause autosomal recessive polycystic kidney disease. Our previous studies have shown that human <italic>PKHD1</italic> and its mouse orthologue, <italic>Pkhd1</italic>, undergo an… (more)

Subjects/Keywords: Alternative Splicing<; br>; Disease Models, Animal<; br>; Kidney Tubules, Collecting – metabolism.<; br>; Polycystic Kidney, Autosomal Recessive – genetics.<; br>; Receptors, Cell Surface – genetics.<; br>; Translocation, Genetic

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APA (6th Edition):

Boddu, R. (2012). Characterizing The Transcriptional Complexity Of Pkhd1/pkhd1. (Thesis). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1504

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Boddu, Ravindra. “Characterizing The Transcriptional Complexity Of Pkhd1/pkhd1.” 2012. Thesis, University of Alabama – Birmingham. Accessed April 01, 2020. http://contentdm.mhsl.uab.edu/u?/etd,1504.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Boddu, Ravindra. “Characterizing The Transcriptional Complexity Of Pkhd1/pkhd1.” 2012. Web. 01 Apr 2020.

Vancouver:

Boddu R. Characterizing The Transcriptional Complexity Of Pkhd1/pkhd1. [Internet] [Thesis]. University of Alabama – Birmingham; 2012. [cited 2020 Apr 01]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1504.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Boddu R. Characterizing The Transcriptional Complexity Of Pkhd1/pkhd1. [Thesis]. University of Alabama – Birmingham; 2012. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1504

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

8. Champattanachai, Voraratt. Effects of hexosamine biosynthesis on an in vitro model of cardiac ischemia.

Degree: PhD, 2008, University of Alabama – Birmingham

Increased levels of protein O-linked-N-acetylglucosamine (O-GlcNAc) have been correlated with increased cell survival following stress. Therefore the goal of this study was to determine whether… (more)

Subjects/Keywords: Acetylglucosamine  – metabolism <; br>; Glucosamine  – pharmacology <; br>; Glycoproteins  – metabolism <; br>; Mitochondria  – metabolism <; br>; Myocardial Reperfusion Injury  – metabolism <; br>; Myocardial Reperfusion Injury  – pathology <; br>; Myocytes, Cardiac  – metabolism <; br>; Myocytes, Cardiac  – pathology

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APA (6th Edition):

Champattanachai, V. (2008). Effects of hexosamine biosynthesis on an in vitro model of cardiac ischemia. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,194

Chicago Manual of Style (16th Edition):

Champattanachai, Voraratt. “Effects of hexosamine biosynthesis on an in vitro model of cardiac ischemia.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed April 01, 2020. http://contentdm.mhsl.uab.edu/u?/etd,194.

MLA Handbook (7th Edition):

Champattanachai, Voraratt. “Effects of hexosamine biosynthesis on an in vitro model of cardiac ischemia.” 2008. Web. 01 Apr 2020.

Vancouver:

Champattanachai V. Effects of hexosamine biosynthesis on an in vitro model of cardiac ischemia. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2020 Apr 01]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,194.

Council of Science Editors:

Champattanachai V. Effects of hexosamine biosynthesis on an in vitro model of cardiac ischemia. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,194

9. Gaines, Jasmine P. The role of sperm protein 17 (Sp17) in somatic cells and cancer.

Degree: PhD, 2006, University of Alabama – Birmingham

Sperm protein 17 (Sp17) was initially described as a member of the rabbit sperm autoantigen family and was proposed to participate in the acrosome reaction… (more)

Subjects/Keywords: Antigens <; br>; Spermatozoa <; br>; Somatic cells <; br>; Centrosomes<; br>; Aneuploidy <; br>; Protein-protein interactions <; br>; Chromosome abnormalities <; br>; Carcinogenesis

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APA (6th Edition):

Gaines, J. P. (2006). The role of sperm protein 17 (Sp17) in somatic cells and cancer. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,366

Chicago Manual of Style (16th Edition):

Gaines, Jasmine P. “The role of sperm protein 17 (Sp17) in somatic cells and cancer.” 2006. Doctoral Dissertation, University of Alabama – Birmingham. Accessed April 01, 2020. http://contentdm.mhsl.uab.edu/u?/etd,366.

MLA Handbook (7th Edition):

Gaines, Jasmine P. “The role of sperm protein 17 (Sp17) in somatic cells and cancer.” 2006. Web. 01 Apr 2020.

Vancouver:

Gaines JP. The role of sperm protein 17 (Sp17) in somatic cells and cancer. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2006. [cited 2020 Apr 01]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,366.

Council of Science Editors:

Gaines JP. The role of sperm protein 17 (Sp17) in somatic cells and cancer. [Doctoral Dissertation]. University of Alabama – Birmingham; 2006. Available from: http://contentdm.mhsl.uab.edu/u?/etd,366

10. Williams, Corey L. Analysis of cystic kidney disease-related genes in Caenorhabditis elegans.

Degree: PhD, 2009, University of Alabama – Birmingham

Cilia are evolutionarily conserved, membrane-bound, microtubule-based organelles found on a diverse array of cell types in eukaryotic organisms. Inherited diseases of cilia protein dysfunction include… (more)

Subjects/Keywords: Caenorhabditis elegans  – cytology<; br>; Caenorhabditis elegans  – metabolism<; br>; Caenorhabditis elegans Proteins  – metabolism<; br>; Cilia  – metabolism<; br>; Protein Transport

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APA (6th Edition):

Williams, C. L. (2009). Analysis of cystic kidney disease-related genes in Caenorhabditis elegans. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,407

Chicago Manual of Style (16th Edition):

Williams, Corey L. “Analysis of cystic kidney disease-related genes in Caenorhabditis elegans.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed April 01, 2020. http://contentdm.mhsl.uab.edu/u?/etd,407.

MLA Handbook (7th Edition):

Williams, Corey L. “Analysis of cystic kidney disease-related genes in Caenorhabditis elegans.” 2009. Web. 01 Apr 2020.

Vancouver:

Williams CL. Analysis of cystic kidney disease-related genes in Caenorhabditis elegans. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2020 Apr 01]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,407.

Council of Science Editors:

Williams CL. Analysis of cystic kidney disease-related genes in Caenorhabditis elegans. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,407

11. Davenport, James Robert. The role of the primary cilium in energy and glucose metabolism.

Degree: PhD, 2007, University of Alabama – Birmingham

Virtually ignored for years as a useless organelle, the primary cilium has emerged as an essential signaling center in both development and maintenance of tissues… (more)

Subjects/Keywords: Cilia  – metabolism<; br>; Flagella  – metabolism<; br>; Kidney  – metabolism<; br>; Kidney Diseases, Cystic  – metabolism<; br>; Obesity  – metabolism<; br>; Pancreas  – abnormalities<; br>; Pancreas  – pathology<; br>; Tumor Suppressor Proteins

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APA (6th Edition):

Davenport, J. R. (2007). The role of the primary cilium in energy and glucose metabolism. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,516

Chicago Manual of Style (16th Edition):

Davenport, James Robert. “The role of the primary cilium in energy and glucose metabolism.” 2007. Doctoral Dissertation, University of Alabama – Birmingham. Accessed April 01, 2020. http://contentdm.mhsl.uab.edu/u?/etd,516.

MLA Handbook (7th Edition):

Davenport, James Robert. “The role of the primary cilium in energy and glucose metabolism.” 2007. Web. 01 Apr 2020.

Vancouver:

Davenport JR. The role of the primary cilium in energy and glucose metabolism. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2007. [cited 2020 Apr 01]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,516.

Council of Science Editors:

Davenport JR. The role of the primary cilium in energy and glucose metabolism. [Doctoral Dissertation]. University of Alabama – Birmingham; 2007. Available from: http://contentdm.mhsl.uab.edu/u?/etd,516

12. Fan-Minogue, Hua. Understanding the molecular mechanism of eukaryotic translation termination: functional analysis of ribosomal RNA and eukaryotic release factor one.

Degree: PhD, 2007, University of Alabama – Birmingham

Translation termination, the last step of protein synthesis, is mediated by release factors, which recognize the stop signal in messenger RNA (mRNA) and relay the… (more)

Subjects/Keywords: Aminoglycosides  – pharmacology<; br>; Codon, Terminator<; br>; Euplotes  – genetics<; br>; Protein Biosynthesis<; br>; RNA, Fungal  – drug effects<; br>; Saccharomyces cerevisiae  – drug effects Saccharomyces cerevisiae  – genetics

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APA (6th Edition):

Fan-Minogue, H. (2007). Understanding the molecular mechanism of eukaryotic translation termination: functional analysis of ribosomal RNA and eukaryotic release factor one. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,519

Chicago Manual of Style (16th Edition):

Fan-Minogue, Hua. “Understanding the molecular mechanism of eukaryotic translation termination: functional analysis of ribosomal RNA and eukaryotic release factor one.” 2007. Doctoral Dissertation, University of Alabama – Birmingham. Accessed April 01, 2020. http://contentdm.mhsl.uab.edu/u?/etd,519.

MLA Handbook (7th Edition):

Fan-Minogue, Hua. “Understanding the molecular mechanism of eukaryotic translation termination: functional analysis of ribosomal RNA and eukaryotic release factor one.” 2007. Web. 01 Apr 2020.

Vancouver:

Fan-Minogue H. Understanding the molecular mechanism of eukaryotic translation termination: functional analysis of ribosomal RNA and eukaryotic release factor one. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2007. [cited 2020 Apr 01]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,519.

Council of Science Editors:

Fan-Minogue H. Understanding the molecular mechanism of eukaryotic translation termination: functional analysis of ribosomal RNA and eukaryotic release factor one. [Doctoral Dissertation]. University of Alabama – Birmingham; 2007. Available from: http://contentdm.mhsl.uab.edu/u?/etd,519

13. Matthews, Tori A. Pathological modifications of tau induce toxicity and facilitate cell death.

Degree: PhD, 2009, University of Alabama – Birmingham

lzheimer’s disease (AD) is a progressive neurodegenerative disorder characterized by two major pathophysiological hallmarks, beta-amyloid (A[Beta]) plaques and tau tangles. In AD and other tau… (more)

Subjects/Keywords: Caspases  – metabolism<; br>; Cell Death  – physiology<; br>; Microtubules  – metabolism<; br>; tau Proteins  – physiology

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APA (6th Edition):

Matthews, T. A. (2009). Pathological modifications of tau induce toxicity and facilitate cell death. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,607

Chicago Manual of Style (16th Edition):

Matthews, Tori A. “Pathological modifications of tau induce toxicity and facilitate cell death.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed April 01, 2020. http://contentdm.mhsl.uab.edu/u?/etd,607.

MLA Handbook (7th Edition):

Matthews, Tori A. “Pathological modifications of tau induce toxicity and facilitate cell death.” 2009. Web. 01 Apr 2020.

Vancouver:

Matthews TA. Pathological modifications of tau induce toxicity and facilitate cell death. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2020 Apr 01]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,607.

Council of Science Editors:

Matthews TA. Pathological modifications of tau induce toxicity and facilitate cell death. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,607

14. Olteanu, Dragos S. Dysregulated ENAC and NHE function in cilium-deficient renal collecting duct cell monolayers : a model of polycystic kidney disease.

Degree: PhD, 2007, University of Alabama – Birmingham

Polycystic kidney disease in both its recessive and dominant forms involves the remodeling of the kidney and extra-renal tissues where parts of the tissue break… (more)

Subjects/Keywords: Cilia  – metabolism<; br>; Epithelial Cells<; br>; Kidney<; br>; Polycystic Kidney, Autosomal Recessive  – metabolism<; br>; Sodium  – metabolism<; br>; Sodium Channels  – metabolism

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APA (6th Edition):

Olteanu, D. S. (2007). Dysregulated ENAC and NHE function in cilium-deficient renal collecting duct cell monolayers : a model of polycystic kidney disease. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,610

Chicago Manual of Style (16th Edition):

Olteanu, Dragos S. “Dysregulated ENAC and NHE function in cilium-deficient renal collecting duct cell monolayers : a model of polycystic kidney disease.” 2007. Doctoral Dissertation, University of Alabama – Birmingham. Accessed April 01, 2020. http://contentdm.mhsl.uab.edu/u?/etd,610.

MLA Handbook (7th Edition):

Olteanu, Dragos S. “Dysregulated ENAC and NHE function in cilium-deficient renal collecting duct cell monolayers : a model of polycystic kidney disease.” 2007. Web. 01 Apr 2020.

Vancouver:

Olteanu DS. Dysregulated ENAC and NHE function in cilium-deficient renal collecting duct cell monolayers : a model of polycystic kidney disease. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2007. [cited 2020 Apr 01]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,610.

Council of Science Editors:

Olteanu DS. Dysregulated ENAC and NHE function in cilium-deficient renal collecting duct cell monolayers : a model of polycystic kidney disease. [Doctoral Dissertation]. University of Alabama – Birmingham; 2007. Available from: http://contentdm.mhsl.uab.edu/u?/etd,610

15. Winkelbauer, Marlene Elizabeth. Elucidating the role of nephronophthisis proteins utilizing Caenorhabditis elegans as a model.

Degree: PhD, 2007, University of Alabama – Birmingham

Numerous disorders are characterized by the presence of cystic lesions within the kidney. The proteins associated with these disorders often localize to cilia, and improper… (more)

Subjects/Keywords: Caenorhabditis elegans  – genetics<; br>; Caenorhabditis elegans Proteins  – genetics<; br>; Cilia  – metabolism<; br>; Neurons, Afferent  – physiology<; br>; Protein Transport<; br>; Transcription Factors  – metabolism

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Winkelbauer, M. E. (2007). Elucidating the role of nephronophthisis proteins utilizing Caenorhabditis elegans as a model. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,619

Chicago Manual of Style (16th Edition):

Winkelbauer, Marlene Elizabeth. “Elucidating the role of nephronophthisis proteins utilizing Caenorhabditis elegans as a model.” 2007. Doctoral Dissertation, University of Alabama – Birmingham. Accessed April 01, 2020. http://contentdm.mhsl.uab.edu/u?/etd,619.

MLA Handbook (7th Edition):

Winkelbauer, Marlene Elizabeth. “Elucidating the role of nephronophthisis proteins utilizing Caenorhabditis elegans as a model.” 2007. Web. 01 Apr 2020.

Vancouver:

Winkelbauer ME. Elucidating the role of nephronophthisis proteins utilizing Caenorhabditis elegans as a model. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2007. [cited 2020 Apr 01]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,619.

Council of Science Editors:

Winkelbauer ME. Elucidating the role of nephronophthisis proteins utilizing Caenorhabditis elegans as a model. [Doctoral Dissertation]. University of Alabama – Birmingham; 2007. Available from: http://contentdm.mhsl.uab.edu/u?/etd,619

16. Lehman, Jonathan Merle. Characterizing the role of primary cilia in the hair follicle and skin.

Degree: PhD, 2009, University of Alabama – Birmingham

Primary cilia have been established as a nearly ubiquitous microtubule based signaling organelle, yet their function in many organs, including the hair and skin, is… (more)

Subjects/Keywords: Cilia  – physiology<; br>; Hair Follicle  – cytology<; br>; Hair Follicle  – growth & development<; br>; Hedgehog Proteins  – metabolism<; br>; Hypotrichosis  – physiopathology<; br>; Morphogenesis  – physiology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Lehman, J. M. (2009). Characterizing the role of primary cilia in the hair follicle and skin. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,681

Chicago Manual of Style (16th Edition):

Lehman, Jonathan Merle. “Characterizing the role of primary cilia in the hair follicle and skin.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed April 01, 2020. http://contentdm.mhsl.uab.edu/u?/etd,681.

MLA Handbook (7th Edition):

Lehman, Jonathan Merle. “Characterizing the role of primary cilia in the hair follicle and skin.” 2009. Web. 01 Apr 2020.

Vancouver:

Lehman JM. Characterizing the role of primary cilia in the hair follicle and skin. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2020 Apr 01]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,681.

Council of Science Editors:

Lehman JM. Characterizing the role of primary cilia in the hair follicle and skin. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,681

17. Kwon, Yeon-Jin. The Role of Gli1 in ERα-negative breast cancer : promoting survival, migration, invasion, and metastasis.

Degree: PhD, 2010, University of Alabama – Birmingham

Glioma-associated oncogene homolog 1 (Gli1) is a well-known oncogene and a transcription factor that mediates several signaling pathways important for tumor progression, such as hedgehog,… (more)

Subjects/Keywords: Breast Neoplasms  – metabolism<; br>; Breast Neoplasms  – pathology<; br>; Cell Movement<; br>; Estrogen Receptor alpha  – deficiency<; br>; Matrix Metalloproteinase 11  – metabolism<; br>; Transcription Factors  – metabolism<; br>; Up-Regulation

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APA (6th Edition):

Kwon, Y. (2010). The Role of Gli1 in ERα-negative breast cancer : promoting survival, migration, invasion, and metastasis. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,913

Chicago Manual of Style (16th Edition):

Kwon, Yeon-Jin. “The Role of Gli1 in ERα-negative breast cancer : promoting survival, migration, invasion, and metastasis.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed April 01, 2020. http://contentdm.mhsl.uab.edu/u?/etd,913.

MLA Handbook (7th Edition):

Kwon, Yeon-Jin. “The Role of Gli1 in ERα-negative breast cancer : promoting survival, migration, invasion, and metastasis.” 2010. Web. 01 Apr 2020.

Vancouver:

Kwon Y. The Role of Gli1 in ERα-negative breast cancer : promoting survival, migration, invasion, and metastasis. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2020 Apr 01]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,913.

Council of Science Editors:

Kwon Y. The Role of Gli1 in ERα-negative breast cancer : promoting survival, migration, invasion, and metastasis. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,913

18. Hollingsworth, T.j. The Role Of Autosomal Dominant Retinitis Pigmentosa Rhodopsin Mutant Ter349glu In Rod Cell Biogenesis And Retinal Inflammation.

Degree: 2013, University of Alabama – Birmingham

Retinitis pigmentosa is one of the most common inherited blinding disorders, affecting 1 in 4000 individuals world-wide. Approximately 30% of retinitis pigmentosa cases are dominantly… (more)

Subjects/Keywords: Genes, Dominant – genetics.<; br>; Mutant Proteins – metabolism.<; br>; Mutation – genetics<; br>; Retinal Degeneration – metabolism.<; br>; Retinal Rod Photoreceptor Cells<; br>; Retinitis Pigmentosa<; br>; Rhodopsin – metabolism.

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APA (6th Edition):

Hollingsworth, T. j. (2013). The Role Of Autosomal Dominant Retinitis Pigmentosa Rhodopsin Mutant Ter349glu In Rod Cell Biogenesis And Retinal Inflammation. (Thesis). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1785

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hollingsworth, T j. “The Role Of Autosomal Dominant Retinitis Pigmentosa Rhodopsin Mutant Ter349glu In Rod Cell Biogenesis And Retinal Inflammation.” 2013. Thesis, University of Alabama – Birmingham. Accessed April 01, 2020. http://contentdm.mhsl.uab.edu/u?/etd,1785.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hollingsworth, T j. “The Role Of Autosomal Dominant Retinitis Pigmentosa Rhodopsin Mutant Ter349glu In Rod Cell Biogenesis And Retinal Inflammation.” 2013. Web. 01 Apr 2020.

Vancouver:

Hollingsworth Tj. The Role Of Autosomal Dominant Retinitis Pigmentosa Rhodopsin Mutant Ter349glu In Rod Cell Biogenesis And Retinal Inflammation. [Internet] [Thesis]. University of Alabama – Birmingham; 2013. [cited 2020 Apr 01]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1785.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hollingsworth Tj. The Role Of Autosomal Dominant Retinitis Pigmentosa Rhodopsin Mutant Ter349glu In Rod Cell Biogenesis And Retinal Inflammation. [Thesis]. University of Alabama – Birmingham; 2013. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1785

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

19. Pyle, Louise Clare. Cystic fibrosis transmembrane conductance regulator (CFTR) modulators and their mechanistic basis: relevance to emerging therapies for cystic fibrosis.

Degree: PhD, 2009, University of Alabama – Birmingham

Cystic fibrosis (CF) is a lethal genetic disorder leading to pulmonary decline and premature death. The gene responsible for CF, the cystic fibrosis transmembrane conductance… (more)

Subjects/Keywords: Biological Markers  – metabolism<; br>; Cystic Fibrosis  – metabolism<; br>; Cystic Fibrosis Transmembrane Conductance Regulator<; br>; High-Throughput Screening Assays<; br>; Ion Channel Gating  – drug effects<; br>; Membrane Transport Modulators  – pharmacology<; br>; Mutant Proteins  – metabolism<; br>; Protein Structure, Tertiary  – genetics<; br>; Quercetin  – pharmacology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Pyle, L. C. (2009). Cystic fibrosis transmembrane conductance regulator (CFTR) modulators and their mechanistic basis: relevance to emerging therapies for cystic fibrosis. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1106

Chicago Manual of Style (16th Edition):

Pyle, Louise Clare. “Cystic fibrosis transmembrane conductance regulator (CFTR) modulators and their mechanistic basis: relevance to emerging therapies for cystic fibrosis.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed April 01, 2020. http://contentdm.mhsl.uab.edu/u?/etd,1106.

MLA Handbook (7th Edition):

Pyle, Louise Clare. “Cystic fibrosis transmembrane conductance regulator (CFTR) modulators and their mechanistic basis: relevance to emerging therapies for cystic fibrosis.” 2009. Web. 01 Apr 2020.

Vancouver:

Pyle LC. Cystic fibrosis transmembrane conductance regulator (CFTR) modulators and their mechanistic basis: relevance to emerging therapies for cystic fibrosis. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2020 Apr 01]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1106.

Council of Science Editors:

Pyle LC. Cystic fibrosis transmembrane conductance regulator (CFTR) modulators and their mechanistic basis: relevance to emerging therapies for cystic fibrosis. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1106

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