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You searched for +publisher:"University of Alabama – Birmingham" +contributor:("Welch, Danny R.<br>"). Showing records 1 – 6 of 6 total matches.

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1. Khotskaya, Yekaterina B. Role of syndecan-1 as key regulator of multiple myeloma pathogenesis.

Degree: PhD, 2009, University of Alabama – Birmingham

Syndecan-1 (CD138), a transmembrane heparan sulfate-bearing proteoglycan, is expressed at high levels on most myeloma cells and is shed into the microenvironment. In patients, high… (more)

Subjects/Keywords: Gene Expression Regulation, Neoplastic<; br>; Melanoma  – metabolism<; br>; Neoplasm Proteins  – biosynthesis<; br>; Neovascularization, Pathologic  – metabolism<; br>; Syndecan-1  – biosynthesis

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Khotskaya, Y. B. (2009). Role of syndecan-1 as key regulator of multiple myeloma pathogenesis. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,766

Chicago Manual of Style (16th Edition):

Khotskaya, Yekaterina B. “Role of syndecan-1 as key regulator of multiple myeloma pathogenesis.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed February 17, 2020. http://contentdm.mhsl.uab.edu/u?/etd,766.

MLA Handbook (7th Edition):

Khotskaya, Yekaterina B. “Role of syndecan-1 as key regulator of multiple myeloma pathogenesis.” 2009. Web. 17 Feb 2020.

Vancouver:

Khotskaya YB. Role of syndecan-1 as key regulator of multiple myeloma pathogenesis. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2020 Feb 17]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,766.

Council of Science Editors:

Khotskaya YB. Role of syndecan-1 as key regulator of multiple myeloma pathogenesis. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,766

2. Geng, Ying. p53-dependent neural stem cell death regulation.

Degree: PhD, 2008, University of Alabama – Birmingham

Regulation of neural precursor cell (NPC) death is important for both normal brain development and prevention of brain tumor formation. The tumor suppressor p53 is… (more)

Subjects/Keywords: Antineoplastic Agents  – pharmacology<; br>; Apoptosis  – physiology bcl-2-Associated X Protein  – physiology<; br>; Brain Neoplasms  – genetics Cerebellum  – cytology<; br>; Chloroquine  – pharmacology<; br>; Cytoplasm  – metabolism<; br>; Glioma  – genetics Neurons  – cytology<; br>; Stem Cells  – cytology Transcriptional Activation  – physiology<; br>; Tumor Suppressor Protein p53  – genetics

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APA (6th Edition):

Geng, Y. (2008). p53-dependent neural stem cell death regulation. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,767

Chicago Manual of Style (16th Edition):

Geng, Ying. “p53-dependent neural stem cell death regulation.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed February 17, 2020. http://contentdm.mhsl.uab.edu/u?/etd,767.

MLA Handbook (7th Edition):

Geng, Ying. “p53-dependent neural stem cell death regulation.” 2008. Web. 17 Feb 2020.

Vancouver:

Geng Y. p53-dependent neural stem cell death regulation. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2020 Feb 17]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,767.

Council of Science Editors:

Geng Y. p53-dependent neural stem cell death regulation. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,767

3. Ritchie, Joseph P. Heparanase drives the aggressive myeloma phenotype: preclinical development of a heparanase inhibitor for the treatment of multiple myeloma.

Degree: PhD, 2010, University of Alabama – Birmingham

Heparanase, an endoglycosidase which cleaves heparan sulfate chains at specific sites, is rarely expressed in normal tissues but becomes evident in many human cancers. We… (more)

Subjects/Keywords: Heparin  – metabolism<; br>; Heparitin Sulfate  – metabolism<; br>; Multiple Myeloma  – pathology<; br>; Neoplasm Invasiveness<; br>; Syndecan-1  – metabolism

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APA (6th Edition):

Ritchie, J. P. (2010). Heparanase drives the aggressive myeloma phenotype: preclinical development of a heparanase inhibitor for the treatment of multiple myeloma. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,852

Chicago Manual of Style (16th Edition):

Ritchie, Joseph P. “Heparanase drives the aggressive myeloma phenotype: preclinical development of a heparanase inhibitor for the treatment of multiple myeloma.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed February 17, 2020. http://contentdm.mhsl.uab.edu/u?/etd,852.

MLA Handbook (7th Edition):

Ritchie, Joseph P. “Heparanase drives the aggressive myeloma phenotype: preclinical development of a heparanase inhibitor for the treatment of multiple myeloma.” 2010. Web. 17 Feb 2020.

Vancouver:

Ritchie JP. Heparanase drives the aggressive myeloma phenotype: preclinical development of a heparanase inhibitor for the treatment of multiple myeloma. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2020 Feb 17]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,852.

Council of Science Editors:

Ritchie JP. Heparanase drives the aggressive myeloma phenotype: preclinical development of a heparanase inhibitor for the treatment of multiple myeloma. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,852

4. Evans, Lynda Michele. The effect of stearic acid on breast cancer development and progression.

Degree: PhD, 2009, University of Alabama – Birmingham

Stearate is an 18-carbon saturated fatty acid that is found in many foods in the western diet including beef and chocolate. Cell culture studies indicate… (more)

Subjects/Keywords: Antineoplastic Agents  – administration & dosage<; br>; Apoptosis<; br>; Breast Neoplasms  – metabolism<; br>; Breast Neoplasms  – pathology<; br>; Dietary Fats<; br>; Neoplasm Metastasis<; br>; Stearates  – therapeutic use<; br>; Stearic Acids  – administration & dosage

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APA (6th Edition):

Evans, L. M. (2009). The effect of stearic acid on breast cancer development and progression. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,906

Chicago Manual of Style (16th Edition):

Evans, Lynda Michele. “The effect of stearic acid on breast cancer development and progression.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed February 17, 2020. http://contentdm.mhsl.uab.edu/u?/etd,906.

MLA Handbook (7th Edition):

Evans, Lynda Michele. “The effect of stearic acid on breast cancer development and progression.” 2009. Web. 17 Feb 2020.

Vancouver:

Evans LM. The effect of stearic acid on breast cancer development and progression. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2020 Feb 17]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,906.

Council of Science Editors:

Evans LM. The effect of stearic acid on breast cancer development and progression. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,906

5. Ashley, Jason Waid. Significance and regulation of CD68 expression in the osteoclast.

Degree: PhD, 2011, University of Alabama – Birmingham

The mucin-like Lysosome Associated Membrane Protein (LAMP) family member CD68 is a primarily myeloid lineage restricted transmembrane protein that is expressed in macrophages and osteoclasts.… (more)

Subjects/Keywords: Antigens, CD  – genetics<; br>; Antigens, Differentiation, Myelomonocytic  – genetics<; br>; Biological Assay  – methods<; br>; Bone and Bones<; br>; Drug Evaluation, Preclinical  – methods<; br>; Gene Deletion<; br>; High-Throughput Screening Assays  – methods<; br>; Macrophages  – drug effects<; br>; Osteoclasts<; br>; Receptor Activator of Nuclear Factor-kappa B  – metabolism<; br>; Signal Transduction  – physiology

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APA (6th Edition):

Ashley, J. W. (2011). Significance and regulation of CD68 expression in the osteoclast. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1033

Chicago Manual of Style (16th Edition):

Ashley, Jason Waid. “Significance and regulation of CD68 expression in the osteoclast.” 2011. Doctoral Dissertation, University of Alabama – Birmingham. Accessed February 17, 2020. http://contentdm.mhsl.uab.edu/u?/etd,1033.

MLA Handbook (7th Edition):

Ashley, Jason Waid. “Significance and regulation of CD68 expression in the osteoclast.” 2011. Web. 17 Feb 2020.

Vancouver:

Ashley JW. Significance and regulation of CD68 expression in the osteoclast. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2011. [cited 2020 Feb 17]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1033.

Council of Science Editors:

Ashley JW. Significance and regulation of CD68 expression in the osteoclast. [Doctoral Dissertation]. University of Alabama – Birmingham; 2011. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1033

6. Fetterman, Jessica L. Mitochondrial Genetics And Function In Cardiovascular Disease Susceptibility.

Degree: PhD, 2011, University of Alabama – Birmingham

While progress has been made in understanding the development and progression of cardiovascular disease (CVD), the mechanisms of CVD risk and initiation are not completely… (more)

Subjects/Keywords: Antigens – metabolism.<; br>; Hematopoietic Stem Cell Transplantation – methods.<; br>; Hematopoietic Stem Cells – metabolism.<; br>; HIV-1<; br>; Immunologic Memory – genetics.<; br>; Lentivirus.<; br>; Neoplasms – immunology<; br>; T-Lymphocytes, Cytotoxic – immunology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Fetterman, J. L. (2011). Mitochondrial Genetics And Function In Cardiovascular Disease Susceptibility. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1395

Chicago Manual of Style (16th Edition):

Fetterman, Jessica L. “Mitochondrial Genetics And Function In Cardiovascular Disease Susceptibility.” 2011. Doctoral Dissertation, University of Alabama – Birmingham. Accessed February 17, 2020. http://contentdm.mhsl.uab.edu/u?/etd,1395.

MLA Handbook (7th Edition):

Fetterman, Jessica L. “Mitochondrial Genetics And Function In Cardiovascular Disease Susceptibility.” 2011. Web. 17 Feb 2020.

Vancouver:

Fetterman JL. Mitochondrial Genetics And Function In Cardiovascular Disease Susceptibility. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2011. [cited 2020 Feb 17]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1395.

Council of Science Editors:

Fetterman JL. Mitochondrial Genetics And Function In Cardiovascular Disease Susceptibility. [Doctoral Dissertation]. University of Alabama – Birmingham; 2011. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1395

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