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You searched for +publisher:"University of Alabama – Birmingham" +contributor:("Welch, Danny R."). Showing records 1 – 16 of 16 total matches.

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1. Anderson, Joshua C. Thrombospondin type-1 repeats and their potential role in inhibiting glioblastoma angiogenesis.

Degree: PhD, 2008, University of Alabama – Birmingham

Thrombospondin-1 (TSP-1) is a 420 kilodalton homotrimeric protein; it is one of the first identified anti-angiogenic proteins found to be expressed in some normal tissues.… (more)

Subjects/Keywords: Angiogenesis Inhibitors  – therapeutic use <; br>; Brain Neoplasms <; br>; Extracellular Matrix  – physiology <; br>; Glioblastoma  – blood supply <; br>; Glioma <; br>; Neovascularization, Pathologic <; br>; Oligopeptides  – therapeutic use

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APA (6th Edition):

Anderson, J. C. (2008). Thrombospondin type-1 repeats and their potential role in inhibiting glioblastoma angiogenesis. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,318

Chicago Manual of Style (16th Edition):

Anderson, Joshua C. “Thrombospondin type-1 repeats and their potential role in inhibiting glioblastoma angiogenesis.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed December 14, 2019. http://contentdm.mhsl.uab.edu/u?/etd,318.

MLA Handbook (7th Edition):

Anderson, Joshua C. “Thrombospondin type-1 repeats and their potential role in inhibiting glioblastoma angiogenesis.” 2008. Web. 14 Dec 2019.

Vancouver:

Anderson JC. Thrombospondin type-1 repeats and their potential role in inhibiting glioblastoma angiogenesis. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2019 Dec 14]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,318.

Council of Science Editors:

Anderson JC. Thrombospondin type-1 repeats and their potential role in inhibiting glioblastoma angiogenesis. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,318

2. Cody, James Joseph. A dual-action, armed replicating adenovirus for the treatment of bone metastases of breast cancer.

Degree: PhD, 2008, University of Alabama – Birmingham

Most patients with advanced breast cancer develop osteolytic bone metastases, which have numerous complications. Because current therapies are not curative, new treatments are needed. Conditionally… (more)

Subjects/Keywords: Adenoviridae  – genetics <; br>; Bone Neoplasms  – secondary <; br>; Breast Neoplasms <; br>; Gene Expression Regulation, Neoplastic <; br>; Neoplasm Metastasis  – genetics <; br>; Neoplasm Metastasis  – therapy <; br>; Virus Replication  – genetics

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APA (6th Edition):

Cody, J. J. (2008). A dual-action, armed replicating adenovirus for the treatment of bone metastases of breast cancer. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,320

Chicago Manual of Style (16th Edition):

Cody, James Joseph. “A dual-action, armed replicating adenovirus for the treatment of bone metastases of breast cancer.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed December 14, 2019. http://contentdm.mhsl.uab.edu/u?/etd,320.

MLA Handbook (7th Edition):

Cody, James Joseph. “A dual-action, armed replicating adenovirus for the treatment of bone metastases of breast cancer.” 2008. Web. 14 Dec 2019.

Vancouver:

Cody JJ. A dual-action, armed replicating adenovirus for the treatment of bone metastases of breast cancer. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2019 Dec 14]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,320.

Council of Science Editors:

Cody JJ. A dual-action, armed replicating adenovirus for the treatment of bone metastases of breast cancer. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,320

3. Roarty, Kevin Patrick. The role of TGF-[beta] and Wnt5a in mammary gland development and tumorigenesis.

Degree: PhD, 2008, University of Alabama – Birmingham

Breast cancer is the second most common cancer worldwide behind lung cancer, affecting women of all ages, races, ethnicities, and socioeconomic strata. Concerted efforts have… (more)

Subjects/Keywords: Gene Expression Regulation, Developmental <; br>; Mammary Glands, Animal  – growth & development <; br>; Mammary Glands, Animal  – metabolism <; br>; Transforming Growth Factor beta1  – pharmacology <; br>; Wnt Proteins  – metabolism

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APA (6th Edition):

Roarty, K. P. (2008). The role of TGF-[beta] and Wnt5a in mammary gland development and tumorigenesis. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,337

Chicago Manual of Style (16th Edition):

Roarty, Kevin Patrick. “The role of TGF-[beta] and Wnt5a in mammary gland development and tumorigenesis.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed December 14, 2019. http://contentdm.mhsl.uab.edu/u?/etd,337.

MLA Handbook (7th Edition):

Roarty, Kevin Patrick. “The role of TGF-[beta] and Wnt5a in mammary gland development and tumorigenesis.” 2008. Web. 14 Dec 2019.

Vancouver:

Roarty KP. The role of TGF-[beta] and Wnt5a in mammary gland development and tumorigenesis. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2019 Dec 14]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,337.

Council of Science Editors:

Roarty KP. The role of TGF-[beta] and Wnt5a in mammary gland development and tumorigenesis. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,337

4. Whitsett, Timothy Glynn. Breast cancer chemoprevention with the natural polyphenols resveratrol and genistein, alone and in combination.

Degree: PhD, 2007, University of Alabama – Birmingham

It has been established that the environment, including the diet, plays a critical role in a woman’s risk of breast cancer. Two dietary polyphenols that… (more)

Subjects/Keywords: Chemoprevention  – methods<; br>; Gene Expression Regulation  – drug effects<; br>; Genistein  – therapeutic use<; br>; Mammary Neoplasms, Experimental  – prevention & control<; br>; Stilbenes  – therapeutic use

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APA (6th Edition):

Whitsett, T. G. (2007). Breast cancer chemoprevention with the natural polyphenols resveratrol and genistein, alone and in combination. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,539

Chicago Manual of Style (16th Edition):

Whitsett, Timothy Glynn. “Breast cancer chemoprevention with the natural polyphenols resveratrol and genistein, alone and in combination.” 2007. Doctoral Dissertation, University of Alabama – Birmingham. Accessed December 14, 2019. http://contentdm.mhsl.uab.edu/u?/etd,539.

MLA Handbook (7th Edition):

Whitsett, Timothy Glynn. “Breast cancer chemoprevention with the natural polyphenols resveratrol and genistein, alone and in combination.” 2007. Web. 14 Dec 2019.

Vancouver:

Whitsett TG. Breast cancer chemoprevention with the natural polyphenols resveratrol and genistein, alone and in combination. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2007. [cited 2019 Dec 14]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,539.

Council of Science Editors:

Whitsett TG. Breast cancer chemoprevention with the natural polyphenols resveratrol and genistein, alone and in combination. [Doctoral Dissertation]. University of Alabama – Birmingham; 2007. Available from: http://contentdm.mhsl.uab.edu/u?/etd,539

5. Lin, Victor T. G. Regulation Of P27kip1 By Trip6 And Its Implications In Cancer Progression.

Degree: 2012, University of Alabama – Birmingham

TRIP6 is a focal adhesion molecule that functions as an adaptor protein to mediate diverse cellular functions, including motility and antiapoptotic signaling, through a wide… (more)

Subjects/Keywords: Actins – metabolism<; /br>; Adaptor Proteins, Signal Transducing.<; /br>; Cell Transformation, Neoplastic – genetics<; /br>; Cyclin-Dependent Kinase Inhibitor p27.<; /br>; Glioma<; /br>; LIM Domain Proteins.<; /br>; Signal Transduction<; /br>; Transcription Factors<; /br>; Transcriptional Activation

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APA (6th Edition):

Lin, V. T. G. (2012). Regulation Of P27kip1 By Trip6 And Its Implications In Cancer Progression. (Thesis). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1649

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lin, Victor T G. “Regulation Of P27kip1 By Trip6 And Its Implications In Cancer Progression.” 2012. Thesis, University of Alabama – Birmingham. Accessed December 14, 2019. http://contentdm.mhsl.uab.edu/u?/etd,1649.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lin, Victor T G. “Regulation Of P27kip1 By Trip6 And Its Implications In Cancer Progression.” 2012. Web. 14 Dec 2019.

Vancouver:

Lin VTG. Regulation Of P27kip1 By Trip6 And Its Implications In Cancer Progression. [Internet] [Thesis]. University of Alabama – Birmingham; 2012. [cited 2019 Dec 14]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1649.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lin VTG. Regulation Of P27kip1 By Trip6 And Its Implications In Cancer Progression. [Thesis]. University of Alabama – Birmingham; 2012. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1649

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

6. Nash, Kevin T. (Kevin Tyler). KISS1 metastasis suppressor secretion is required for metastasis suppression.

Degree: PhD, 2006, University of Alabama – Birmingham

Failure to reduce the number of cancer deaths over the last 50 years is due to the inability to selectively target metastatic disease. Recently, the… (more)

Subjects/Keywords: Gene Expression Regulation, Neoplastic <; br>; Melanoma  – metabolism <; br>; Melanoma  – secretion <; br>; Neoplasm Metastasis  – prevention & control <; br>; Proteins  – physiology <; br>; Receptors, G-Protein-Coupled  – metabolism <; br>; Tumor Suppressor Proteins  – secretion

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APA (6th Edition):

Nash, K. T. (. T. (2006). KISS1 metastasis suppressor secretion is required for metastasis suppression. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,380

Chicago Manual of Style (16th Edition):

Nash, Kevin T (Kevin Tyler). “KISS1 metastasis suppressor secretion is required for metastasis suppression.” 2006. Doctoral Dissertation, University of Alabama – Birmingham. Accessed December 14, 2019. http://contentdm.mhsl.uab.edu/u?/etd,380.

MLA Handbook (7th Edition):

Nash, Kevin T (Kevin Tyler). “KISS1 metastasis suppressor secretion is required for metastasis suppression.” 2006. Web. 14 Dec 2019.

Vancouver:

Nash KT(T. KISS1 metastasis suppressor secretion is required for metastasis suppression. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2006. [cited 2019 Dec 14]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,380.

Council of Science Editors:

Nash KT(T. KISS1 metastasis suppressor secretion is required for metastasis suppression. [Doctoral Dissertation]. University of Alabama – Birmingham; 2006. Available from: http://contentdm.mhsl.uab.edu/u?/etd,380

7. Khotskaya, Yekaterina B. Role of syndecan-1 as key regulator of multiple myeloma pathogenesis.

Degree: PhD, 2009, University of Alabama – Birmingham

Syndecan-1 (CD138), a transmembrane heparan sulfate-bearing proteoglycan, is expressed at high levels on most myeloma cells and is shed into the microenvironment. In patients, high… (more)

Subjects/Keywords: Gene Expression Regulation, Neoplastic<; br>; Melanoma  – metabolism<; br>; Neoplasm Proteins  – biosynthesis<; br>; Neovascularization, Pathologic  – metabolism<; br>; Syndecan-1  – biosynthesis

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APA (6th Edition):

Khotskaya, Y. B. (2009). Role of syndecan-1 as key regulator of multiple myeloma pathogenesis. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,766

Chicago Manual of Style (16th Edition):

Khotskaya, Yekaterina B. “Role of syndecan-1 as key regulator of multiple myeloma pathogenesis.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed December 14, 2019. http://contentdm.mhsl.uab.edu/u?/etd,766.

MLA Handbook (7th Edition):

Khotskaya, Yekaterina B. “Role of syndecan-1 as key regulator of multiple myeloma pathogenesis.” 2009. Web. 14 Dec 2019.

Vancouver:

Khotskaya YB. Role of syndecan-1 as key regulator of multiple myeloma pathogenesis. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2019 Dec 14]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,766.

Council of Science Editors:

Khotskaya YB. Role of syndecan-1 as key regulator of multiple myeloma pathogenesis. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,766

8. Geng, Ying. p53-dependent neural stem cell death regulation.

Degree: PhD, 2008, University of Alabama – Birmingham

Regulation of neural precursor cell (NPC) death is important for both normal brain development and prevention of brain tumor formation. The tumor suppressor p53 is… (more)

Subjects/Keywords: Antineoplastic Agents  – pharmacology<; br>; Apoptosis  – physiology bcl-2-Associated X Protein  – physiology<; br>; Brain Neoplasms  – genetics Cerebellum  – cytology<; br>; Chloroquine  – pharmacology<; br>; Cytoplasm  – metabolism<; br>; Glioma  – genetics Neurons  – cytology<; br>; Stem Cells  – cytology Transcriptional Activation  – physiology<; br>; Tumor Suppressor Protein p53  – genetics

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APA (6th Edition):

Geng, Y. (2008). p53-dependent neural stem cell death regulation. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,767

Chicago Manual of Style (16th Edition):

Geng, Ying. “p53-dependent neural stem cell death regulation.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed December 14, 2019. http://contentdm.mhsl.uab.edu/u?/etd,767.

MLA Handbook (7th Edition):

Geng, Ying. “p53-dependent neural stem cell death regulation.” 2008. Web. 14 Dec 2019.

Vancouver:

Geng Y. p53-dependent neural stem cell death regulation. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2019 Dec 14]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,767.

Council of Science Editors:

Geng Y. p53-dependent neural stem cell death regulation. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,767

9. Ritchie, Joseph P. Heparanase drives the aggressive myeloma phenotype: preclinical development of a heparanase inhibitor for the treatment of multiple myeloma.

Degree: PhD, 2010, University of Alabama – Birmingham

Heparanase, an endoglycosidase which cleaves heparan sulfate chains at specific sites, is rarely expressed in normal tissues but becomes evident in many human cancers. We… (more)

Subjects/Keywords: Heparin  – metabolism<; br>; Heparitin Sulfate  – metabolism<; br>; Multiple Myeloma  – pathology<; br>; Neoplasm Invasiveness<; br>; Syndecan-1  – metabolism

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APA (6th Edition):

Ritchie, J. P. (2010). Heparanase drives the aggressive myeloma phenotype: preclinical development of a heparanase inhibitor for the treatment of multiple myeloma. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,852

Chicago Manual of Style (16th Edition):

Ritchie, Joseph P. “Heparanase drives the aggressive myeloma phenotype: preclinical development of a heparanase inhibitor for the treatment of multiple myeloma.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed December 14, 2019. http://contentdm.mhsl.uab.edu/u?/etd,852.

MLA Handbook (7th Edition):

Ritchie, Joseph P. “Heparanase drives the aggressive myeloma phenotype: preclinical development of a heparanase inhibitor for the treatment of multiple myeloma.” 2010. Web. 14 Dec 2019.

Vancouver:

Ritchie JP. Heparanase drives the aggressive myeloma phenotype: preclinical development of a heparanase inhibitor for the treatment of multiple myeloma. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2019 Dec 14]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,852.

Council of Science Editors:

Ritchie JP. Heparanase drives the aggressive myeloma phenotype: preclinical development of a heparanase inhibitor for the treatment of multiple myeloma. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,852

10. Evans, Lynda Michele. The effect of stearic acid on breast cancer development and progression.

Degree: PhD, 2009, University of Alabama – Birmingham

Stearate is an 18-carbon saturated fatty acid that is found in many foods in the western diet including beef and chocolate. Cell culture studies indicate… (more)

Subjects/Keywords: Antineoplastic Agents  – administration & dosage<; br>; Apoptosis<; br>; Breast Neoplasms  – metabolism<; br>; Breast Neoplasms  – pathology<; br>; Dietary Fats<; br>; Neoplasm Metastasis<; br>; Stearates  – therapeutic use<; br>; Stearic Acids  – administration & dosage

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APA (6th Edition):

Evans, L. M. (2009). The effect of stearic acid on breast cancer development and progression. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,906

Chicago Manual of Style (16th Edition):

Evans, Lynda Michele. “The effect of stearic acid on breast cancer development and progression.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed December 14, 2019. http://contentdm.mhsl.uab.edu/u?/etd,906.

MLA Handbook (7th Edition):

Evans, Lynda Michele. “The effect of stearic acid on breast cancer development and progression.” 2009. Web. 14 Dec 2019.

Vancouver:

Evans LM. The effect of stearic acid on breast cancer development and progression. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2019 Dec 14]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,906.

Council of Science Editors:

Evans LM. The effect of stearic acid on breast cancer development and progression. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,906

11. Kwon, Yeon-Jin. The Role of Gli1 in ERα-negative breast cancer : promoting survival, migration, invasion, and metastasis.

Degree: PhD, 2010, University of Alabama – Birmingham

Glioma-associated oncogene homolog 1 (Gli1) is a well-known oncogene and a transcription factor that mediates several signaling pathways important for tumor progression, such as hedgehog,… (more)

Subjects/Keywords: Breast Neoplasms  – metabolism<; br>; Breast Neoplasms  – pathology<; br>; Cell Movement<; br>; Estrogen Receptor alpha  – deficiency<; br>; Matrix Metalloproteinase 11  – metabolism<; br>; Transcription Factors  – metabolism<; br>; Up-Regulation

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APA (6th Edition):

Kwon, Y. (2010). The Role of Gli1 in ERα-negative breast cancer : promoting survival, migration, invasion, and metastasis. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,913

Chicago Manual of Style (16th Edition):

Kwon, Yeon-Jin. “The Role of Gli1 in ERα-negative breast cancer : promoting survival, migration, invasion, and metastasis.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed December 14, 2019. http://contentdm.mhsl.uab.edu/u?/etd,913.

MLA Handbook (7th Edition):

Kwon, Yeon-Jin. “The Role of Gli1 in ERα-negative breast cancer : promoting survival, migration, invasion, and metastasis.” 2010. Web. 14 Dec 2019.

Vancouver:

Kwon Y. The Role of Gli1 in ERα-negative breast cancer : promoting survival, migration, invasion, and metastasis. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2019 Dec 14]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,913.

Council of Science Editors:

Kwon Y. The Role of Gli1 in ERα-negative breast cancer : promoting survival, migration, invasion, and metastasis. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,913

12. Ashley, Jason Waid. Significance and regulation of CD68 expression in the osteoclast.

Degree: PhD, 2011, University of Alabama – Birmingham

The mucin-like Lysosome Associated Membrane Protein (LAMP) family member CD68 is a primarily myeloid lineage restricted transmembrane protein that is expressed in macrophages and osteoclasts.… (more)

Subjects/Keywords: Antigens, CD  – genetics<; br>; Antigens, Differentiation, Myelomonocytic  – genetics<; br>; Biological Assay  – methods<; br>; Bone and Bones<; br>; Drug Evaluation, Preclinical  – methods<; br>; Gene Deletion<; br>; High-Throughput Screening Assays  – methods<; br>; Macrophages  – drug effects<; br>; Osteoclasts<; br>; Receptor Activator of Nuclear Factor-kappa B  – metabolism<; br>; Signal Transduction  – physiology

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APA (6th Edition):

Ashley, J. W. (2011). Significance and regulation of CD68 expression in the osteoclast. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1033

Chicago Manual of Style (16th Edition):

Ashley, Jason Waid. “Significance and regulation of CD68 expression in the osteoclast.” 2011. Doctoral Dissertation, University of Alabama – Birmingham. Accessed December 14, 2019. http://contentdm.mhsl.uab.edu/u?/etd,1033.

MLA Handbook (7th Edition):

Ashley, Jason Waid. “Significance and regulation of CD68 expression in the osteoclast.” 2011. Web. 14 Dec 2019.

Vancouver:

Ashley JW. Significance and regulation of CD68 expression in the osteoclast. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2011. [cited 2019 Dec 14]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1033.

Council of Science Editors:

Ashley JW. Significance and regulation of CD68 expression in the osteoclast. [Doctoral Dissertation]. University of Alabama – Birmingham; 2011. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1033

13. Cook, Leah M. (Leah Marie). Understanding molecular mechanisms of breast cancer metastasis using genetically-engineered mice.

Degree: PhD, 2011, University of Alabama – Birmingham

Morbidity and mortality of breast cancer patients are drastically increased when primary tumor cells metastasize to distant organ sites. Effective treatment of metastatic disease has… (more)

Subjects/Keywords: Breast Neoplasms  – pathology<; br>; Mammary Neoplasms, Animal<; br>; Mice, Transgenic<; br>; Neoplasm Metastasis  – pathology<; br>; Tumor Microenvironment<; br>; Tumor Suppressor Proteins  – genetics<; br>; Tumor Suppressor Proteins  – metabolism

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APA (6th Edition):

Cook, L. M. (. M. (2011). Understanding molecular mechanisms of breast cancer metastasis using genetically-engineered mice. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1045

Chicago Manual of Style (16th Edition):

Cook, Leah M (Leah Marie). “Understanding molecular mechanisms of breast cancer metastasis using genetically-engineered mice.” 2011. Doctoral Dissertation, University of Alabama – Birmingham. Accessed December 14, 2019. http://contentdm.mhsl.uab.edu/u?/etd,1045.

MLA Handbook (7th Edition):

Cook, Leah M (Leah Marie). “Understanding molecular mechanisms of breast cancer metastasis using genetically-engineered mice.” 2011. Web. 14 Dec 2019.

Vancouver:

Cook LM(M. Understanding molecular mechanisms of breast cancer metastasis using genetically-engineered mice. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2011. [cited 2019 Dec 14]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1045.

Council of Science Editors:

Cook LM(M. Understanding molecular mechanisms of breast cancer metastasis using genetically-engineered mice. [Doctoral Dissertation]. University of Alabama – Birmingham; 2011. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1045

14. Beck, Benjamin H. (Benjamin Hester). Immunotherapy of cancer employing γδ-T cells: a study examining their utility and feasibility.

Degree: PhD, 2009, University of Alabama – Birmingham

Unlike antigen-specific αβ-T cells, γδ-T cells can recognize and lyse cancerous cells rapidly upon encounter in a manner that does not require the recognition of… (more)

Subjects/Keywords: Adenocarcinoma  – therapy<; br>; Breast Neoplasms  – pathology<; br>; Immunotherapy, Adoptive<; br>; Mammary Neoplasms, Experimental  – therapy<; br>; Receptors, Antigen, T-Cell, gamma-delta  – immunology T-Lymphocyte Subsets  – transplantation

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APA (6th Edition):

Beck, B. H. (. H. (2009). Immunotherapy of cancer employing γδ-T cells: a study examining their utility and feasibility. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1087

Chicago Manual of Style (16th Edition):

Beck, Benjamin H (Benjamin Hester). “Immunotherapy of cancer employing γδ-T cells: a study examining their utility and feasibility.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed December 14, 2019. http://contentdm.mhsl.uab.edu/u?/etd,1087.

MLA Handbook (7th Edition):

Beck, Benjamin H (Benjamin Hester). “Immunotherapy of cancer employing γδ-T cells: a study examining their utility and feasibility.” 2009. Web. 14 Dec 2019.

Vancouver:

Beck BH(H. Immunotherapy of cancer employing γδ-T cells: a study examining their utility and feasibility. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2019 Dec 14]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1087.

Council of Science Editors:

Beck BH(H. Immunotherapy of cancer employing γδ-T cells: a study examining their utility and feasibility. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1087

15. Paik, Jason Chang. A nucleolar specificity factor for E2F1-induced cell death.

Degree: PhD, 2010, University of Alabama – Birmingham

The E2F family of transcription factors are important regulators of cell proliferation, and are often dysregulated in cancers. One member of the E2F family, E2F1,… (more)

Subjects/Keywords: Apoptosis<; br>; Chromosomal Proteins, Non-Histone  – physiology<; br>; E2F1 Transcription Factor  – physiology<; br>; Nuclear Proteins  – physiology<; br>; Transcription, Genetic<; br>; Transcriptional Activation

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APA (6th Edition):

Paik, J. C. (2010). A nucleolar specificity factor for E2F1-induced cell death. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1171

Chicago Manual of Style (16th Edition):

Paik, Jason Chang. “A nucleolar specificity factor for E2F1-induced cell death.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed December 14, 2019. http://contentdm.mhsl.uab.edu/u?/etd,1171.

MLA Handbook (7th Edition):

Paik, Jason Chang. “A nucleolar specificity factor for E2F1-induced cell death.” 2010. Web. 14 Dec 2019.

Vancouver:

Paik JC. A nucleolar specificity factor for E2F1-induced cell death. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2019 Dec 14]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1171.

Council of Science Editors:

Paik JC. A nucleolar specificity factor for E2F1-induced cell death. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1171

16. Fetterman, Jessica L. Mitochondrial Genetics And Function In Cardiovascular Disease Susceptibility.

Degree: PhD, 2011, University of Alabama – Birmingham

While progress has been made in understanding the development and progression of cardiovascular disease (CVD), the mechanisms of CVD risk and initiation are not completely… (more)

Subjects/Keywords: Antigens – metabolism.<; br>; Hematopoietic Stem Cell Transplantation – methods.<; br>; Hematopoietic Stem Cells – metabolism.<; br>; HIV-1<; br>; Immunologic Memory – genetics.<; br>; Lentivirus.<; br>; Neoplasms – immunology<; br>; T-Lymphocytes, Cytotoxic – immunology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Fetterman, J. L. (2011). Mitochondrial Genetics And Function In Cardiovascular Disease Susceptibility. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1395

Chicago Manual of Style (16th Edition):

Fetterman, Jessica L. “Mitochondrial Genetics And Function In Cardiovascular Disease Susceptibility.” 2011. Doctoral Dissertation, University of Alabama – Birmingham. Accessed December 14, 2019. http://contentdm.mhsl.uab.edu/u?/etd,1395.

MLA Handbook (7th Edition):

Fetterman, Jessica L. “Mitochondrial Genetics And Function In Cardiovascular Disease Susceptibility.” 2011. Web. 14 Dec 2019.

Vancouver:

Fetterman JL. Mitochondrial Genetics And Function In Cardiovascular Disease Susceptibility. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2011. [cited 2019 Dec 14]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1395.

Council of Science Editors:

Fetterman JL. Mitochondrial Genetics And Function In Cardiovascular Disease Susceptibility. [Doctoral Dissertation]. University of Alabama – Birmingham; 2011. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1395

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