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You searched for +publisher:"University of Alabama – Birmingham" +contributor:("Sztul, Elizabeth S.<br>"). Showing records 1 – 5 of 5 total matches.

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1. Ramanathan, Harish N. Trafficking of hantaviral nucleocapsid proteins.

Degree: PhD, 2007, University of Alabama – Birmingham

 Hantaviruses are enveloped, negative-sense RNA viruses belonging to genus Hantavirus, within the family Bunyaviridae. Hantaviruses include many serious human pathogens and are classified as Old… (more)

Subjects/Keywords: Hantavirus  – drug effects <; br>; Hantavirus  – genetics <; br>; Hantavirus  – metabolism <; br>; Nucleosides  – chemical synthesis <; br>; Nucleosides  – metabolism

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Ramanathan, H. N. (2007). Trafficking of hantaviral nucleocapsid proteins. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,261

Chicago Manual of Style (16th Edition):

Ramanathan, Harish N. “Trafficking of hantaviral nucleocapsid proteins.” 2007. Doctoral Dissertation, University of Alabama – Birmingham. Accessed December 11, 2019. http://contentdm.mhsl.uab.edu/u?/etd,261.

MLA Handbook (7th Edition):

Ramanathan, Harish N. “Trafficking of hantaviral nucleocapsid proteins.” 2007. Web. 11 Dec 2019.

Vancouver:

Ramanathan HN. Trafficking of hantaviral nucleocapsid proteins. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2007. [cited 2019 Dec 11]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,261.

Council of Science Editors:

Ramanathan HN. Trafficking of hantaviral nucleocapsid proteins. [Doctoral Dissertation]. University of Alabama – Birmingham; 2007. Available from: http://contentdm.mhsl.uab.edu/u?/etd,261

2. Parish, Lindsay A. Protein trafficking and 4.1R relocalization in Plasmodium falciparum-infected erythrocytes.

Degree: PhD, 2009, University of Alabama – Birmingham

Malaria is a mosquito-borne infectious disease that is caused by parasites in the genus Plasmodium. There are four species of malaria that routinely infect humans,… (more)

Subjects/Keywords: Erythrocytes  – metabolism<; br>; Plasmodium falciparum  – metabolism<; br>; Protozoan Proteins  – genetics<; br>; Qa-SNARE Proteins  – genetics<; br>; Secretory Pathway  – genetics

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APA (6th Edition):

Parish, L. A. (2009). Protein trafficking and 4.1R relocalization in Plasmodium falciparum-infected erythrocytes. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,503

Chicago Manual of Style (16th Edition):

Parish, Lindsay A. “Protein trafficking and 4.1R relocalization in Plasmodium falciparum-infected erythrocytes.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed December 11, 2019. http://contentdm.mhsl.uab.edu/u?/etd,503.

MLA Handbook (7th Edition):

Parish, Lindsay A. “Protein trafficking and 4.1R relocalization in Plasmodium falciparum-infected erythrocytes.” 2009. Web. 11 Dec 2019.

Vancouver:

Parish LA. Protein trafficking and 4.1R relocalization in Plasmodium falciparum-infected erythrocytes. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2019 Dec 11]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,503.

Council of Science Editors:

Parish LA. Protein trafficking and 4.1R relocalization in Plasmodium falciparum-infected erythrocytes. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,503

3. Matthews, Tori A. Pathological modifications of tau induce toxicity and facilitate cell death.

Degree: PhD, 2009, University of Alabama – Birmingham

lzheimer’s disease (AD) is a progressive neurodegenerative disorder characterized by two major pathophysiological hallmarks, beta-amyloid (A[Beta]) plaques and tau tangles. In AD and other tau… (more)

Subjects/Keywords: Caspases  – metabolism<; br>; Cell Death  – physiology<; br>; Microtubules  – metabolism<; br>; tau Proteins  – physiology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Matthews, T. A. (2009). Pathological modifications of tau induce toxicity and facilitate cell death. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,607

Chicago Manual of Style (16th Edition):

Matthews, Tori A. “Pathological modifications of tau induce toxicity and facilitate cell death.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed December 11, 2019. http://contentdm.mhsl.uab.edu/u?/etd,607.

MLA Handbook (7th Edition):

Matthews, Tori A. “Pathological modifications of tau induce toxicity and facilitate cell death.” 2009. Web. 11 Dec 2019.

Vancouver:

Matthews TA. Pathological modifications of tau induce toxicity and facilitate cell death. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2019 Dec 11]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,607.

Council of Science Editors:

Matthews TA. Pathological modifications of tau induce toxicity and facilitate cell death. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,607

4. Ontiveros, Steven J. Intracellular trafficking of the hantaviral nucleocapsid protein and its function in modulation of immune signaling.

Degree: PhD, 2009, University of Alabama – Birmingham

Old World and New World hantaviruses, family Bunyaviridae, mature intracellularly within cellular compartments. Although it is generally accepted they assemble and bud in the Golgi… (more)

Subjects/Keywords: Apoptosis<; br>; Capsid Proteins  – physiology<; br>; Hantaan virus  – pathogenicity<; br>; Hantavirus  – pathogenicity<; br>; Signal Transduction<; br>; Viral Core Proteins  – physiology<; br>; Virulence Factors  – physiology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Ontiveros, S. J. (2009). Intracellular trafficking of the hantaviral nucleocapsid protein and its function in modulation of immune signaling. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,687

Chicago Manual of Style (16th Edition):

Ontiveros, Steven J. “Intracellular trafficking of the hantaviral nucleocapsid protein and its function in modulation of immune signaling.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed December 11, 2019. http://contentdm.mhsl.uab.edu/u?/etd,687.

MLA Handbook (7th Edition):

Ontiveros, Steven J. “Intracellular trafficking of the hantaviral nucleocapsid protein and its function in modulation of immune signaling.” 2009. Web. 11 Dec 2019.

Vancouver:

Ontiveros SJ. Intracellular trafficking of the hantaviral nucleocapsid protein and its function in modulation of immune signaling. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2019 Dec 11]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,687.

Council of Science Editors:

Ontiveros SJ. Intracellular trafficking of the hantaviral nucleocapsid protein and its function in modulation of immune signaling. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,687

5. Gill, Rachel B. Human cytomegalovirus UL97 kinase activity modifies cell cycle checkpoint regulators.

Degree: PhD, 2011, University of Alabama – Birmingham

Human cytomegalovirus (CMV) infection results in destructive infections in neonates and immunocompromised individuals. Being the primary congenital infection in the United States, it can often… (more)

Subjects/Keywords: Benzimidazoles  – pharmacology<; br>; Cytomegalovirus<; br>; Enzyme Inhibitors  – pharmacology<; br>; Phosphotransferases (Alcohol Group Acceptor)  – chemistry<; br>; Phosphotransferases (Alcohol Group Acceptor)  – metabolism<; br>; Retinoblastoma Protein  – metabolism<; br>; Ribonucleosides  – pharmacology<; br>; Virus Replication

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Gill, R. B. (2011). Human cytomegalovirus UL97 kinase activity modifies cell cycle checkpoint regulators. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,867

Chicago Manual of Style (16th Edition):

Gill, Rachel B. “Human cytomegalovirus UL97 kinase activity modifies cell cycle checkpoint regulators.” 2011. Doctoral Dissertation, University of Alabama – Birmingham. Accessed December 11, 2019. http://contentdm.mhsl.uab.edu/u?/etd,867.

MLA Handbook (7th Edition):

Gill, Rachel B. “Human cytomegalovirus UL97 kinase activity modifies cell cycle checkpoint regulators.” 2011. Web. 11 Dec 2019.

Vancouver:

Gill RB. Human cytomegalovirus UL97 kinase activity modifies cell cycle checkpoint regulators. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2011. [cited 2019 Dec 11]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,867.

Council of Science Editors:

Gill RB. Human cytomegalovirus UL97 kinase activity modifies cell cycle checkpoint regulators. [Doctoral Dissertation]. University of Alabama – Birmingham; 2011. Available from: http://contentdm.mhsl.uab.edu/u?/etd,867

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