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You searched for +publisher:"University of Alabama – Birmingham" +contributor:("Sanderson, Ralph D."). Showing records 1 – 7 of 7 total matches.

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1. Fulzele, Keertik. Insulin signaling and function in osteoblasts.

Degree: PhD, 2009, University of Alabama – Birmingham

Insulin and insulin-like growth factor-1 (IGF-1) are evolutionarily conserved hormonal signaling pathways with structurally similar ligands and receptors. Recent studies suggest that that insulin and… (more)

Subjects/Keywords: Insulin  – metabolism<; br>; Osteoblasts  – metabolism<; br>; Osteogenesis  – physiology<; br>; Receptor, IGF Type 1  – metabolism<; br>; Receptor, Insulin  – metabolism<; br>; Signal Transduction  – physiology<; br>; Skull  – metabolism

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Fulzele, K. (2009). Insulin signaling and function in osteoblasts. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,999

Chicago Manual of Style (16th Edition):

Fulzele, Keertik. “Insulin signaling and function in osteoblasts.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed February 27, 2020. http://contentdm.mhsl.uab.edu/u?/etd,999.

MLA Handbook (7th Edition):

Fulzele, Keertik. “Insulin signaling and function in osteoblasts.” 2009. Web. 27 Feb 2020.

Vancouver:

Fulzele K. Insulin signaling and function in osteoblasts. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2020 Feb 27]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,999.

Council of Science Editors:

Fulzele K. Insulin signaling and function in osteoblasts. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,999

2. Higgs, Jerome T. Therapeutic Potential Of Genetically-Engineered Mesenchymal Stem Cells In Multiple Myeloma.

Degree: 2013, University of Alabama – Birmingham

 ABSTRACT Multiple Myeloma (MM) remains the second most common hematologic malig-nancy occurring in adults, which primarily affects the skeletal system causing severe bone destruction, spinal… (more)

Subjects/Keywords: Bone and Bones<; /br>; Mesenchymal Stem Cell Transplantation.<; /br>; Mesenchymal Stromal Cells.<; /br>; Multiple Myeloma<; /br>; Neoplasms – therapy.<; /br>; Osteoclasts<; /br>; Osteoprotegerin.

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APA (6th Edition):

Higgs, J. T. (2013). Therapeutic Potential Of Genetically-Engineered Mesenchymal Stem Cells In Multiple Myeloma. (Thesis). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1651

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Higgs, Jerome T. “Therapeutic Potential Of Genetically-Engineered Mesenchymal Stem Cells In Multiple Myeloma.” 2013. Thesis, University of Alabama – Birmingham. Accessed February 27, 2020. http://contentdm.mhsl.uab.edu/u?/etd,1651.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Higgs, Jerome T. “Therapeutic Potential Of Genetically-Engineered Mesenchymal Stem Cells In Multiple Myeloma.” 2013. Web. 27 Feb 2020.

Vancouver:

Higgs JT. Therapeutic Potential Of Genetically-Engineered Mesenchymal Stem Cells In Multiple Myeloma. [Internet] [Thesis]. University of Alabama – Birmingham; 2013. [cited 2020 Feb 27]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1651.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Higgs JT. Therapeutic Potential Of Genetically-Engineered Mesenchymal Stem Cells In Multiple Myeloma. [Thesis]. University of Alabama – Birmingham; 2013. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1651

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

3. Edmonds, Mick D. Brms1 Coordinately Regulates Microrna To Suppress Breast.

Degree: PhD, 2010, University of Alabama – Birmingham

The majority of cancer related mortality is attributed to complications associated with metastatic disease. Breast cancer metastasis suppressor 1 (BRMS1) suppresses metastasis of multiple cancer… (more)

Subjects/Keywords: Antineoplastic Agents – pharmacology<; br>; Colonic Neoplasms – pathology<; br>; Cyclic Nucleotide Phosphodiesterases, Type 5 – metabolism.<; br>; Phosphodiesterase 5 Inhibitors – pharmacology.<; br>; Sulindac – pharmacology.<; br>; Transcriptional Activation – drug effects.<; br>; beta Catenin – genetics.

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APA (6th Edition):

Edmonds, M. D. (2010). Brms1 Coordinately Regulates Microrna To Suppress Breast. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1417

Chicago Manual of Style (16th Edition):

Edmonds, Mick D. “Brms1 Coordinately Regulates Microrna To Suppress Breast.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed February 27, 2020. http://contentdm.mhsl.uab.edu/u?/etd,1417.

MLA Handbook (7th Edition):

Edmonds, Mick D. “Brms1 Coordinately Regulates Microrna To Suppress Breast.” 2010. Web. 27 Feb 2020.

Vancouver:

Edmonds MD. Brms1 Coordinately Regulates Microrna To Suppress Breast. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2020 Feb 27]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1417.

Council of Science Editors:

Edmonds MD. Brms1 Coordinately Regulates Microrna To Suppress Breast. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1417

4. Khotskaya, Yekaterina B. Role of syndecan-1 as key regulator of multiple myeloma pathogenesis.

Degree: PhD, 2009, University of Alabama – Birmingham

Syndecan-1 (CD138), a transmembrane heparan sulfate-bearing proteoglycan, is expressed at high levels on most myeloma cells and is shed into the microenvironment. In patients, high… (more)

Subjects/Keywords: Gene Expression Regulation, Neoplastic<; br>; Melanoma  – metabolism<; br>; Neoplasm Proteins  – biosynthesis<; br>; Neovascularization, Pathologic  – metabolism<; br>; Syndecan-1  – biosynthesis

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APA (6th Edition):

Khotskaya, Y. B. (2009). Role of syndecan-1 as key regulator of multiple myeloma pathogenesis. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,766

Chicago Manual of Style (16th Edition):

Khotskaya, Yekaterina B. “Role of syndecan-1 as key regulator of multiple myeloma pathogenesis.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed February 27, 2020. http://contentdm.mhsl.uab.edu/u?/etd,766.

MLA Handbook (7th Edition):

Khotskaya, Yekaterina B. “Role of syndecan-1 as key regulator of multiple myeloma pathogenesis.” 2009. Web. 27 Feb 2020.

Vancouver:

Khotskaya YB. Role of syndecan-1 as key regulator of multiple myeloma pathogenesis. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2020 Feb 27]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,766.

Council of Science Editors:

Khotskaya YB. Role of syndecan-1 as key regulator of multiple myeloma pathogenesis. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,766

5. Ritchie, Joseph P. Heparanase drives the aggressive myeloma phenotype: preclinical development of a heparanase inhibitor for the treatment of multiple myeloma.

Degree: PhD, 2010, University of Alabama – Birmingham

Heparanase, an endoglycosidase which cleaves heparan sulfate chains at specific sites, is rarely expressed in normal tissues but becomes evident in many human cancers. We… (more)

Subjects/Keywords: Heparin  – metabolism<; br>; Heparitin Sulfate  – metabolism<; br>; Multiple Myeloma  – pathology<; br>; Neoplasm Invasiveness<; br>; Syndecan-1  – metabolism

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Ritchie, J. P. (2010). Heparanase drives the aggressive myeloma phenotype: preclinical development of a heparanase inhibitor for the treatment of multiple myeloma. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,852

Chicago Manual of Style (16th Edition):

Ritchie, Joseph P. “Heparanase drives the aggressive myeloma phenotype: preclinical development of a heparanase inhibitor for the treatment of multiple myeloma.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed February 27, 2020. http://contentdm.mhsl.uab.edu/u?/etd,852.

MLA Handbook (7th Edition):

Ritchie, Joseph P. “Heparanase drives the aggressive myeloma phenotype: preclinical development of a heparanase inhibitor for the treatment of multiple myeloma.” 2010. Web. 27 Feb 2020.

Vancouver:

Ritchie JP. Heparanase drives the aggressive myeloma phenotype: preclinical development of a heparanase inhibitor for the treatment of multiple myeloma. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2020 Feb 27]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,852.

Council of Science Editors:

Ritchie JP. Heparanase drives the aggressive myeloma phenotype: preclinical development of a heparanase inhibitor for the treatment of multiple myeloma. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,852

6. Warram, Jason M. (Jason Morgan). Strategies for combined screening and imaging of breast cancer.

Degree: PhD, 2011, University of Alabama – Birmingham

Successful treatment of breast cancer directly correlates with tumor stage and grade at diagnosis. Early diagnosis leads to a five-year relative survival rate of 96.8%… (more)

Subjects/Keywords: Adenoviridae  – genetics<; br>; Breast Neoplasms  – diagnosis<; br>; Gene Therapy<; br>; Mass Screening  – methods<; br>; Microbubbles<; br>; Neoplasm Metastasis<; br>; Promoter Regions, Genetic  – genetics<; br>; Vascular Endothelial Growth Factor Receptor-2  – immunology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Warram, J. M. (. M. (2011). Strategies for combined screening and imaging of breast cancer. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,881

Chicago Manual of Style (16th Edition):

Warram, Jason M (Jason Morgan). “Strategies for combined screening and imaging of breast cancer.” 2011. Doctoral Dissertation, University of Alabama – Birmingham. Accessed February 27, 2020. http://contentdm.mhsl.uab.edu/u?/etd,881.

MLA Handbook (7th Edition):

Warram, Jason M (Jason Morgan). “Strategies for combined screening and imaging of breast cancer.” 2011. Web. 27 Feb 2020.

Vancouver:

Warram JM(M. Strategies for combined screening and imaging of breast cancer. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2011. [cited 2020 Feb 27]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,881.

Council of Science Editors:

Warram JM(M. Strategies for combined screening and imaging of breast cancer. [Doctoral Dissertation]. University of Alabama – Birmingham; 2011. Available from: http://contentdm.mhsl.uab.edu/u?/etd,881

7. Bodenstine, Thomas Morgan. Molecular Mechanisms Of Breast Cancer Metastasis: Gap Junction Intercellular Communication And The Bone Microenvironment.

Degree: PhD, 2010, University of Alabama – Birmingham

Metastatic disease accounts for the overwhelming majority of cancer related deaths. More specifically, breast cancer remains one of the leading causes of death in women… (more)

Subjects/Keywords: Atherosclerosis – etiology.<; br>; Cardiovascular Diseases – etiology<; br>; Disease Susceptibility<; br>; DNA, Mitochondrial – genetics<; br>; Mice<; br>; Mitochondria – physiology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Bodenstine, T. M. (2010). Molecular Mechanisms Of Breast Cancer Metastasis: Gap Junction Intercellular Communication And The Bone Microenvironment. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1386

Chicago Manual of Style (16th Edition):

Bodenstine, Thomas Morgan. “Molecular Mechanisms Of Breast Cancer Metastasis: Gap Junction Intercellular Communication And The Bone Microenvironment.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed February 27, 2020. http://contentdm.mhsl.uab.edu/u?/etd,1386.

MLA Handbook (7th Edition):

Bodenstine, Thomas Morgan. “Molecular Mechanisms Of Breast Cancer Metastasis: Gap Junction Intercellular Communication And The Bone Microenvironment.” 2010. Web. 27 Feb 2020.

Vancouver:

Bodenstine TM. Molecular Mechanisms Of Breast Cancer Metastasis: Gap Junction Intercellular Communication And The Bone Microenvironment. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2020 Feb 27]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1386.

Council of Science Editors:

Bodenstine TM. Molecular Mechanisms Of Breast Cancer Metastasis: Gap Junction Intercellular Communication And The Bone Microenvironment. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1386

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