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You searched for +publisher:"University of Alabama – Birmingham" +contributor:("Ruppert, J. Michael <br>"). Showing records 1 – 7 of 7 total matches.

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1. Helton, Eric Scott. A role for p63 in the regulation of cell cycle progression and cell death.

Degree: PhD, 2007, University of Alabama – Birmingham

p63 is a member of the p53 family of transcription factors that is a critical regulator of epithelial development. Studies have shown that p63 does… (more)

Subjects/Keywords: Cell Cycle  – physiology <; br>; Cell Death  – physiology <; br>; Transcription Factors <; br>; Tumor Suppressor Protein p53  – physiology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Helton, E. S. (2007). A role for p63 in the regulation of cell cycle progression and cell death. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,279

Chicago Manual of Style (16th Edition):

Helton, Eric Scott. “A role for p63 in the regulation of cell cycle progression and cell death.” 2007. Doctoral Dissertation, University of Alabama – Birmingham. Accessed September 21, 2019. http://contentdm.mhsl.uab.edu/u?/etd,279.

MLA Handbook (7th Edition):

Helton, Eric Scott. “A role for p63 in the regulation of cell cycle progression and cell death.” 2007. Web. 21 Sep 2019.

Vancouver:

Helton ES. A role for p63 in the regulation of cell cycle progression and cell death. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2007. [cited 2019 Sep 21]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,279.

Council of Science Editors:

Helton ES. A role for p63 in the regulation of cell cycle progression and cell death. [Doctoral Dissertation]. University of Alabama – Birmingham; 2007. Available from: http://contentdm.mhsl.uab.edu/u?/etd,279

2. Ko, Rose Marie. The effect of the AML1-ETO translocation on cell cycle tumor suppressor gene function.

Degree: PhD, 2007, University of Alabama – Birmingham

The t(8;21)(q22;q22) AML1-ETO translocation is one of the most frequent translocations in acute myeloid leukemia (AML), occurring in approximately 12% of cases. Our laboratory has… (more)

Subjects/Keywords: Cell Differentiation  – physiology <; br>; Core Binding Factor Alpha 2 Subunit  – physiology <; br>; Cyclin-Dependent Kinase Inhibitor p15  – physiology <; br>; Hematopoietic Stem Cells <; br>; Leukemia, Myeloid, Acute  – physiopathology <; br>; Myeloid Cells  – pathology

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APA (6th Edition):

Ko, R. M. (2007). The effect of the AML1-ETO translocation on cell cycle tumor suppressor gene function. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,375

Chicago Manual of Style (16th Edition):

Ko, Rose Marie. “The effect of the AML1-ETO translocation on cell cycle tumor suppressor gene function.” 2007. Doctoral Dissertation, University of Alabama – Birmingham. Accessed September 21, 2019. http://contentdm.mhsl.uab.edu/u?/etd,375.

MLA Handbook (7th Edition):

Ko, Rose Marie. “The effect of the AML1-ETO translocation on cell cycle tumor suppressor gene function.” 2007. Web. 21 Sep 2019.

Vancouver:

Ko RM. The effect of the AML1-ETO translocation on cell cycle tumor suppressor gene function. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2007. [cited 2019 Sep 21]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,375.

Council of Science Editors:

Ko RM. The effect of the AML1-ETO translocation on cell cycle tumor suppressor gene function. [Doctoral Dissertation]. University of Alabama – Birmingham; 2007. Available from: http://contentdm.mhsl.uab.edu/u?/etd,375

3. Harms, Kelly Lynn. Mechanisms of P53-mediated apoptosis.

Degree: PhD, 2007, University of Alabama – Birmingham

The p53 tumor suppressor is the most commonly inactivated gene in human cancers. The ability of p53, a transcription factor, to regulate genes that mediate… (more)

Subjects/Keywords: Apoptosis  – genetics<; br>; DNA, Neoplasm  – metabolism<; br>; Gene Expression Regulation  – physiology<; br>; Histone Deacetylases  – metabolism<; br>; Insulin-Like Growth Factor Binding Protein 3  – genetics<; br>; Repressor Proteins  – metabolism<; br>; Transcriptional Activation  – physiology<; br>; Tumor Suppressor Protein p53

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APA (6th Edition):

Harms, K. L. (2007). Mechanisms of P53-mediated apoptosis. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,446

Chicago Manual of Style (16th Edition):

Harms, Kelly Lynn. “Mechanisms of P53-mediated apoptosis.” 2007. Doctoral Dissertation, University of Alabama – Birmingham. Accessed September 21, 2019. http://contentdm.mhsl.uab.edu/u?/etd,446.

MLA Handbook (7th Edition):

Harms, Kelly Lynn. “Mechanisms of P53-mediated apoptosis.” 2007. Web. 21 Sep 2019.

Vancouver:

Harms KL. Mechanisms of P53-mediated apoptosis. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2007. [cited 2019 Sep 21]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,446.

Council of Science Editors:

Harms KL. Mechanisms of P53-mediated apoptosis. [Doctoral Dissertation]. University of Alabama – Birmingham; 2007. Available from: http://contentdm.mhsl.uab.edu/u?/etd,446

4. Joo, Heui Yun. Understanding the regulatory mechanisms of UBP-M and H2A deubiquitination in chromatin and cellular functions.

Degree: PhD, 2009, University of Alabama – Birmingham

Posttranslational modifications of histones regulate important chromatin and cellular functions. Among them, ubiquitination of histone H2A is correlated to transcriptional repression, such as HOX gene… (more)

Subjects/Keywords: Chromatin  – physiology<; br>; Endopeptidases  – metabolism<; br>; Histones  – metabolism<; br>; Ubiquitin Thiolesterase  – metabolism<; br>; Xenopus Proteins  – metabolism<; br>; Xenopus laevis  – embryology

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APA (6th Edition):

Joo, H. Y. (2009). Understanding the regulatory mechanisms of UBP-M and H2A deubiquitination in chromatin and cellular functions. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1101

Chicago Manual of Style (16th Edition):

Joo, Heui Yun. “Understanding the regulatory mechanisms of UBP-M and H2A deubiquitination in chromatin and cellular functions.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed September 21, 2019. http://contentdm.mhsl.uab.edu/u?/etd,1101.

MLA Handbook (7th Edition):

Joo, Heui Yun. “Understanding the regulatory mechanisms of UBP-M and H2A deubiquitination in chromatin and cellular functions.” 2009. Web. 21 Sep 2019.

Vancouver:

Joo HY. Understanding the regulatory mechanisms of UBP-M and H2A deubiquitination in chromatin and cellular functions. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2019 Sep 21]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1101.

Council of Science Editors:

Joo HY. Understanding the regulatory mechanisms of UBP-M and H2A deubiquitination in chromatin and cellular functions. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1101

5. Phipps, Sharla Marion Ostein. Genetic and epigenetic modulation of telomerase activity in development and disease.

Degree: PhD, 2007, University of Alabama – Birmingham

The end replication problem of linear chromosomes leads to the erosion of telomeric DNA with each cell division. A mechanism to counteract telomeric attrition involves… (more)

Subjects/Keywords: Telomerase  – Physiological effect <; br>; Telomerase  – Inhibitors <; br>; Genetic regulation <; br>; Enzymes  – Regulation <; br>; Genetic transcription <; br>; Reverse transcriptase <; br>; Breast  – Cancer  – Genetic aspects <; br>; Tretinoin  – Physiological effect <; br>; Epigenesis.

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APA (6th Edition):

Phipps, S. M. O. (2007). Genetic and epigenetic modulation of telomerase activity in development and disease. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,260

Chicago Manual of Style (16th Edition):

Phipps, Sharla Marion Ostein. “Genetic and epigenetic modulation of telomerase activity in development and disease.” 2007. Doctoral Dissertation, University of Alabama – Birmingham. Accessed September 21, 2019. http://contentdm.mhsl.uab.edu/u?/etd,260.

MLA Handbook (7th Edition):

Phipps, Sharla Marion Ostein. “Genetic and epigenetic modulation of telomerase activity in development and disease.” 2007. Web. 21 Sep 2019.

Vancouver:

Phipps SMO. Genetic and epigenetic modulation of telomerase activity in development and disease. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2007. [cited 2019 Sep 21]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,260.

Council of Science Editors:

Phipps SMO. Genetic and epigenetic modulation of telomerase activity in development and disease. [Doctoral Dissertation]. University of Alabama – Birmingham; 2007. Available from: http://contentdm.mhsl.uab.edu/u?/etd,260

6. Steg, Adam. Analysis of the hedgehog pathway in pancreatic adenocarcinoma.

Degree: PhD, 2008, University of Alabama – Birmingham

Pancreatic adenocarcinoma (PAC) is the fourth leading cause of cancer mortality in the United States. Despite the use of highly aggressive treatment regimens (surgery, chemotherapy… (more)

Subjects/Keywords: Adenocarcinoma  – metabolism <; br>; Adenocarcinoma  – pathology <; br>; Hedgehog Proteins  – metabolism <; br>; Pancreatic Neoplasms  – metabolism <; br>; Pancreatic Neoplasms  – pathology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Steg, A. (2008). Analysis of the hedgehog pathway in pancreatic adenocarcinoma. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,290

Chicago Manual of Style (16th Edition):

Steg, Adam. “Analysis of the hedgehog pathway in pancreatic adenocarcinoma.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed September 21, 2019. http://contentdm.mhsl.uab.edu/u?/etd,290.

MLA Handbook (7th Edition):

Steg, Adam. “Analysis of the hedgehog pathway in pancreatic adenocarcinoma.” 2008. Web. 21 Sep 2019.

Vancouver:

Steg A. Analysis of the hedgehog pathway in pancreatic adenocarcinoma. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2019 Sep 21]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,290.

Council of Science Editors:

Steg A. Analysis of the hedgehog pathway in pancreatic adenocarcinoma. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,290

7. Duffy, Aaron A. Visualizing the productive program of HPV in differentiating squamous epithelial tissue.

Degree: PhD, 2010, University of Alabama – Birmingham

The human papillomavirus (HPV) establishes persistent infections in the basal stratum of squamous epithelia, while productive amplification of viral DNA occurs in differentiated keratinocytes prior… (more)

Subjects/Keywords: Cell Culture Techniques  – methods<; br>; DNA Replication<; br>; G2 Phase<; br>; Genetic Techniques<; br>; Host-Pathogen Interactions<; br>; Human papillomavirus 18<; br>; Papillomaviridae  – pathogenicity

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Duffy, A. A. (2010). Visualizing the productive program of HPV in differentiating squamous epithelial tissue. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,705

Chicago Manual of Style (16th Edition):

Duffy, Aaron A. “Visualizing the productive program of HPV in differentiating squamous epithelial tissue.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed September 21, 2019. http://contentdm.mhsl.uab.edu/u?/etd,705.

MLA Handbook (7th Edition):

Duffy, Aaron A. “Visualizing the productive program of HPV in differentiating squamous epithelial tissue.” 2010. Web. 21 Sep 2019.

Vancouver:

Duffy AA. Visualizing the productive program of HPV in differentiating squamous epithelial tissue. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2019 Sep 21]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,705.

Council of Science Editors:

Duffy AA. Visualizing the productive program of HPV in differentiating squamous epithelial tissue. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,705

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