Advanced search options

Advanced Search Options 🞨

Browse by author name (“Author name starts with…”).

Find ETDs with:

in
/  
in
/  
in
/  
in

Written in Published in Earliest date Latest date

Sorted by

Results per page:

Sorted by: relevance · author · university · dateNew search

You searched for +publisher:"University of Alabama – Birmingham" +contributor:("McDonald, Jay"). Showing records 1 – 7 of 7 total matches.

Search Limiters

Last 2 Years | English Only

No search limiters apply to these results.

▼ Search Limiters

1. Turk, Amy Nicole. Cholangiocarcinoma combination therapy : Tamoxifen and Gemcitabine produce an additive effect in vitro due to differing mechanisms and will be an effective combination in vivo.

Degree: MS, 2010, University of Alabama – Birmingham

Cholangiocarcinoma, a fatal tumor arising from the biliary epithelium, has a very poor 5-year survival rate due to the lack of early diagnosis and effective… (more)

Subjects/Keywords: Apoptosis  – drug effects<; br>; Cholangiocarcinoma  – drug therapy<; br>; Deoxycytidine  – therapeutic use<; br>; Tamoxifen  – therapeutic use

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Turk, A. N. (2010). Cholangiocarcinoma combination therapy : Tamoxifen and Gemcitabine produce an additive effect in vitro due to differing mechanisms and will be an effective combination in vivo. (Masters Thesis). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,639

Chicago Manual of Style (16th Edition):

Turk, Amy Nicole. “Cholangiocarcinoma combination therapy : Tamoxifen and Gemcitabine produce an additive effect in vitro due to differing mechanisms and will be an effective combination in vivo.” 2010. Masters Thesis, University of Alabama – Birmingham. Accessed October 23, 2019. http://contentdm.mhsl.uab.edu/u?/etd,639.

MLA Handbook (7th Edition):

Turk, Amy Nicole. “Cholangiocarcinoma combination therapy : Tamoxifen and Gemcitabine produce an additive effect in vitro due to differing mechanisms and will be an effective combination in vivo.” 2010. Web. 23 Oct 2019.

Vancouver:

Turk AN. Cholangiocarcinoma combination therapy : Tamoxifen and Gemcitabine produce an additive effect in vitro due to differing mechanisms and will be an effective combination in vivo. [Internet] [Masters thesis]. University of Alabama – Birmingham; 2010. [cited 2019 Oct 23]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,639.

Council of Science Editors:

Turk AN. Cholangiocarcinoma combination therapy : Tamoxifen and Gemcitabine produce an additive effect in vitro due to differing mechanisms and will be an effective combination in vivo. [Masters Thesis]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,639

2. Saxena, Ritu. Mechanisms and countermeasures of microgravity-induced bone loss: in vitro and in vivo model systems.

Degree: PhD, 2009, University of Alabama – Birmingham

Rapid bone loss occurs during prolonged periods of weightlessness experienced by astronauts during spaceflights which leads to osteopenia and increased fracture risk upon return to… (more)

Subjects/Keywords: Bone Resorption<; br>; Cell Differentiation<; br>; Osteoblasts  – cytology<; br>; Osteoclasts  – cytology<; br>; Parathyroid Hormone<; br>; Weightlessness

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Saxena, R. (2009). Mechanisms and countermeasures of microgravity-induced bone loss: in vitro and in vivo model systems. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,689

Chicago Manual of Style (16th Edition):

Saxena, Ritu. “Mechanisms and countermeasures of microgravity-induced bone loss: in vitro and in vivo model systems.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 23, 2019. http://contentdm.mhsl.uab.edu/u?/etd,689.

MLA Handbook (7th Edition):

Saxena, Ritu. “Mechanisms and countermeasures of microgravity-induced bone loss: in vitro and in vivo model systems.” 2009. Web. 23 Oct 2019.

Vancouver:

Saxena R. Mechanisms and countermeasures of microgravity-induced bone loss: in vitro and in vivo model systems. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2019 Oct 23]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,689.

Council of Science Editors:

Saxena R. Mechanisms and countermeasures of microgravity-induced bone loss: in vitro and in vivo model systems. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,689

3. Jules, Joel. In vitro elucidation of the role and mechanism of RANKL in TNF- and IL-1-mediated osteoclast formation and function.

Degree: PhD, 2010, University of Alabama – Birmingham

The receptor activator of NF-қB (RANK) ligand (RANKL) and its receptor RANK play a critical role in osteoclast biology. RANK has three tumor necrosis factor… (more)

Subjects/Keywords: Amino Acid Motifs<; br>; Cell Differentiation<; br>; Cytoplasm  – metabolism<; br>; Osteoclasts  – cytology<; br>; RANK Ligand  – metabolism<; br>; Receptor Activator of Nuclear Factor-kappa B  – chemistry<; br>; Receptor Activator of Nuclear Factor-kappa B  – metabolism<; br>; Tumor Necrosis Factor-alpha  – metabolism

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Jules, J. (2010). In vitro elucidation of the role and mechanism of RANKL in TNF- and IL-1-mediated osteoclast formation and function. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,917

Chicago Manual of Style (16th Edition):

Jules, Joel. “In vitro elucidation of the role and mechanism of RANKL in TNF- and IL-1-mediated osteoclast formation and function.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 23, 2019. http://contentdm.mhsl.uab.edu/u?/etd,917.

MLA Handbook (7th Edition):

Jules, Joel. “In vitro elucidation of the role and mechanism of RANKL in TNF- and IL-1-mediated osteoclast formation and function.” 2010. Web. 23 Oct 2019.

Vancouver:

Jules J. In vitro elucidation of the role and mechanism of RANKL in TNF- and IL-1-mediated osteoclast formation and function. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2019 Oct 23]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,917.

Council of Science Editors:

Jules J. In vitro elucidation of the role and mechanism of RANKL in TNF- and IL-1-mediated osteoclast formation and function. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,917

4. Swindall, Amanda F. The Role Of St6gal-I Sialylation In Fas (cd95) Death Receptor Function And Tumorigenesis.

Degree: PhD, 2012, University of Alabama – Birmingham

The golgi glycosyltransferase, ST6Gal-I, adds a negatively-charged sialic acid in an alpha-2-6 linkage to N-linked glycans. ST6Gal-I is upregulated in many cancers, and is associated… (more)

Subjects/Keywords: Antigens, CD – metabolism.<; br>; Antigens, CD95 – metabolism.<; br>; Apoptosis<; br>; Tumor Cells, Cultured<; br>; Colonic Neoplasms<; br>; Neoplasm Proteins – metabolism.<; br>; Sialyltransferases – metabolism.

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Swindall, A. F. (2012). The Role Of St6gal-I Sialylation In Fas (cd95) Death Receptor Function And Tumorigenesis. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1392

Chicago Manual of Style (16th Edition):

Swindall, Amanda F. “The Role Of St6gal-I Sialylation In Fas (cd95) Death Receptor Function And Tumorigenesis.” 2012. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 23, 2019. http://contentdm.mhsl.uab.edu/u?/etd,1392.

MLA Handbook (7th Edition):

Swindall, Amanda F. “The Role Of St6gal-I Sialylation In Fas (cd95) Death Receptor Function And Tumorigenesis.” 2012. Web. 23 Oct 2019.

Vancouver:

Swindall AF. The Role Of St6gal-I Sialylation In Fas (cd95) Death Receptor Function And Tumorigenesis. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2012. [cited 2019 Oct 23]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1392.

Council of Science Editors:

Swindall AF. The Role Of St6gal-I Sialylation In Fas (cd95) Death Receptor Function And Tumorigenesis. [Doctoral Dissertation]. University of Alabama – Birmingham; 2012. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1392

5. Pawar, Pritish Subhash. Calmodulin binding to cellular FLICE like inhibitory protein modulates Fas-induced signaling and tumorigenesis in cholangiocarcinoma.

Degree: PhD, 2008, University of Alabama – Birmingham

Cholangiocarcinoma, a fatal tumor arising from biliary epithelium, has very poor 5-year survival rate due to lack of early diagnosis and effective therapies. Induction of… (more)

Subjects/Keywords: Antigens, CD95  – metabolism <; br>; CASP8 and FADD-Like Apoptosis Regulating Protein  – metabolism <; br>; Calmodulin  – metabolism <; br>; Cholangiocarcinoma <; br>; Signal Transduction

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Pawar, P. S. (2008). Calmodulin binding to cellular FLICE like inhibitory protein modulates Fas-induced signaling and tumorigenesis in cholangiocarcinoma. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,301

Chicago Manual of Style (16th Edition):

Pawar, Pritish Subhash. “Calmodulin binding to cellular FLICE like inhibitory protein modulates Fas-induced signaling and tumorigenesis in cholangiocarcinoma.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 23, 2019. http://contentdm.mhsl.uab.edu/u?/etd,301.

MLA Handbook (7th Edition):

Pawar, Pritish Subhash. “Calmodulin binding to cellular FLICE like inhibitory protein modulates Fas-induced signaling and tumorigenesis in cholangiocarcinoma.” 2008. Web. 23 Oct 2019.

Vancouver:

Pawar PS. Calmodulin binding to cellular FLICE like inhibitory protein modulates Fas-induced signaling and tumorigenesis in cholangiocarcinoma. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2019 Oct 23]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,301.

Council of Science Editors:

Pawar PS. Calmodulin binding to cellular FLICE like inhibitory protein modulates Fas-induced signaling and tumorigenesis in cholangiocarcinoma. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,301

6. Byon, Chang Hyun. Oxidative stress-stimulated vascular calcification.

Degree: PhD, 2009, University of Alabama – Birmingham

Oxidative stress plays a critical role in pathogenesis of atherosclerosis. However, the effect of oxidative stress-induced molecular signaling in development of vascular calcification, a feature… (more)

Subjects/Keywords: Calcinosis  – metabolism<; br>; Core Binding Factor Alpha 1 Subunit  – metabolism<; br>; Hydrogen Peroxide  – metabolism<; br>; Muscle, smooth, Vascular  – metabolism<; br>; Oxidative Stress<; br>; Proto-Oncogene Proteins c-akt  – metabolism<; br>; Signal Transduction

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Byon, C. H. (2009). Oxidative stress-stimulated vascular calcification. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,662

Chicago Manual of Style (16th Edition):

Byon, Chang Hyun. “Oxidative stress-stimulated vascular calcification.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 23, 2019. http://contentdm.mhsl.uab.edu/u?/etd,662.

MLA Handbook (7th Edition):

Byon, Chang Hyun. “Oxidative stress-stimulated vascular calcification.” 2009. Web. 23 Oct 2019.

Vancouver:

Byon CH. Oxidative stress-stimulated vascular calcification. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2019 Oct 23]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,662.

Council of Science Editors:

Byon CH. Oxidative stress-stimulated vascular calcification. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,662

7. Kim, Junghyun. Transcriptional regulation of the human heme oxygenase-1 via chromatin looping in renal cells.

Degree: PhD, 2010, University of Alabama – Birmingham

Heme oxygenase-1 (HO-1) is a critical enzyme catalyzing the degradation of heme and generating carbon monoxide, iron, and biliverdin. In addition to heme degradation, HO-1… (more)

Subjects/Keywords: Acute Kidney Injury<; br>; Chromatin  – chemistry<; br>; Gene Expression Regulation, Enzymologic<; br>; Heme Oxygenase-1  – biosynthesis<; br>; Kidney  – enzymology<; br>; Mice<; br>; Sp1 Transcription Factor  – physiology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Kim, J. (2010). Transcriptional regulation of the human heme oxygenase-1 via chromatin looping in renal cells. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1100

Chicago Manual of Style (16th Edition):

Kim, Junghyun. “Transcriptional regulation of the human heme oxygenase-1 via chromatin looping in renal cells.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 23, 2019. http://contentdm.mhsl.uab.edu/u?/etd,1100.

MLA Handbook (7th Edition):

Kim, Junghyun. “Transcriptional regulation of the human heme oxygenase-1 via chromatin looping in renal cells.” 2010. Web. 23 Oct 2019.

Vancouver:

Kim J. Transcriptional regulation of the human heme oxygenase-1 via chromatin looping in renal cells. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2019 Oct 23]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1100.

Council of Science Editors:

Kim J. Transcriptional regulation of the human heme oxygenase-1 via chromatin looping in renal cells. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1100

.